THREAT A SIMPLE SOLUTION ------- Introduction A PHYSICIAN'S GUIDE TO RADON This booklet ori radon has been developed for physicians by (he U.S. Environmental Protection Agency in consultation with the American Medical Association (AMA). Its purpose is to enlist physicians in the nation- al effort to inform the American public about the serious health risk posed by indoor radon gas. Lung cancer's very high associated mortality rate is even more tragic because a significant portion of king cancer is preventable. While smoking remains the number one cause of lung cancer, radon presents a significant second risk factor. That is why, in addition to encouraging patients to stop smoking, it is important for physicians to inquire about and encourage patients to test for radon levels in their homes. One way to do this is for physicans to join those health care professionals and organizat ions who have begun to include questions about the radon level in patients' homes on standardized patient history forms. Because the public views physicians as advisors on health and prevention of disease, physicians are in a unique position to play a vital role in informing the public about the common and serious risk of radon, and in educating their patients in testing procedures and remediation methods for correcting elevated indoor radon levels, thereby helping to reduce the number of lung cancer deaths. Free radon information materials (posters, pamphlets, videos) are available from your state radon office (see page 16) to assist you in educating your patients about radon. ------- Table off Contents Executive Summary .... . 2 Wliat is Radon? . . 3 Characteristics and Sources of Radon , 3 The Health Risk 4 How does Radon Induce Cancer?., 4 What is the Evidence? ,v. .. 4 Is Occupational Exposure to Radon Comparable to Residential Exposure? 7 What About Smoking and Radon Exposure?..,, 7 The Solution.. 9 Why Should Every Home be Tested? 9 How Do You Obtain a Reliable Test Result? ,..., 9 Radon Testing Methods . 9 Radon Test Devices...... ......10 How to Test..... ... 10 Interpreting Radon Test Results II Basis for the 4 pCi/L Radon "Action Level".. 11 Radon Reduction Methods .... 11 Other Indoor Air Pollutants..., 13 Environmental Tobacco Smoke (ETS) 13 Biological .Air Pollutants .......... ................ 14 Volatile Organic Compounds (VOCs)...,.....,.. 14 Other Combustion Products... ,.......,.14 Most Commonly Asked Questions about Radon 15 State Radon Contacts....... 16 Additional Information Sources and Suggested Reading List 17 O ------- \ Executive Summary According to the OFFICE OF THE SURGEON GENERAL: Indoor radon gas is o serious health problem in our nation that con be addressed by individual action. Unless people become aware of the danger radon poses, they will not act. Millions of homes are estimated to hove elevated radon levels. Fortun- ately, the solution to this problem is straight-forward. Like the haz- ards from smoking, the health risks of radon can be reduced. Radon Causes Thousands of Preventable Lung Cancer Deaths Each Year Each year in the United States exposure to indoor radon gas causes thousands of preventable lung can- cer deaths. In fact. the Surgeon General has warned that radon is the second leading cause of lung cancer in the United States. Extensive epidemiological evi- dence from studies of underground miners, comple- mented by animal data, indicates that radon causes lung cancer in both smokers and nonsmokers, although malignancy is especially likely to occur in cigarette smokers. Exposure to both smoking and radon greatly enhances the risk of lung cancer. The carcinogenicity of radon is supported by a consensus of opinion among national and international health organizations. By informing "patients about the health risk posed by radon exposure and providing practical advice about radon testing and mitigation, physicians can have a tremendous positive impact on the nation- al effort to prevent radon-induced lung cancer. 'Radon is estimated to cause about 14,000 deaths per year—however, this number could range from 7,000 to 30,000 deaths per year. The numbers of deaths from other causes are taken from the 1990 National Safety Council reports. 25000 20000 15000 10000 5000 - RADON DEATHS EACH YEAR Radon is Easy to Detect and Reduce in a Home The danger posed by radon can be detected rather easily through inexpensive do-it-yourself testing or through a trained radon contractor. Radon test kits can be purchased hy mail order or in hardware stores and other retail outlets. Because of the serious heal! h risk posed by radon, the U.S. Environmental Protection Agency (EPA) recommends that all homes be tested for radon below the third floor. If an elevated radon level is discovered in a home, il can be corrected. It is recommended that a confirmed radon level of 4 picoeuries per liter (pCi/L) of air or higher be reduced to decrease the risk of developing lung cancer. The cost of radon mitigation in a typical home ranges from about $500 to about $2,500. Your state radon information office (see page 16) can pro- vide general advice about radon testing and mitiga- tion, as well as specific information about qualified radon contractors in your state. "Physicians are often the only science professional known to their patients and are almost always a trusted source of information about science in general and health in particular. Radon does increase the risk of lung cancer, and physicians have an obligation to educate their patients about the health risk associated with radon." Jerod M. Loeb, Assistant Vice-President for Science, Technology and Public Health of the American Medical Association Drunk Driving RADON* Drownings Fires Airline Crashes O ------- Radon-222 is a radioactive gas released during the natural decay of thorium and uranium, which are common, naturally occurring elements found in vary- ing amounts in rock and soil. Odorless, invisible, and without taste, radon cannot be detected with the human senses. Outdoors, where it is diluted to low concentrations in the air, radon poses significantly less risk than indoors. In the indoor air environment, however, radon can accumulate to significant levels. The mag- nitude of radon concentration indoors depends pri- marily on a building's construction and the amount of radon iit the underlying soil. The soil composition under and around a house affects radon levels and the ease with which radon migrates toward a house. Normal pressure differences between the house and the soil can create a sliglu vacuum in the home that can draw radon gas from the soil into the building. Radon gas can enter a home from the soil through cracks in concrete floors and walls, floor drains, sump pumps, construction joints, and tiny cracks or pores iti hollow-block walls, Radon levels are generally high- est in basements and ground floor rooms that are in contact with the soil. Factors such as the design, con- struction, and ventilation of the home affect the path- ways and forces that can draw radon indoors. Another source of radon indoors may be air released by well water during showering and other household activities. Compared to radon entering the home through soil, radon entering the home through water will in most cases hr a small source of risk. What is Radon? Radon-222 decays into radioactive elements, two of which—polonium-218 and poIonium-214—emit alpha particles, which are highly effective in damaging lung tissues. These alpha-emitting radon decay products have been implicated in a causal relationship with lung cancer in humans. Characteristics and Sources off Radon HOW RADON ENTERS YOUR HOUSE A Cracks in concrete slabs B Spaces behind brick veneer walls that rest on uncapped hoi low-block foundation C Pores and cracks in concrete blocks D Floor-wall joints E Exposed soil, as in a sump F Weeping (drain) tile, if drained to open sump G Mortar joints H Loose fitting pipe penetrations I Open tops of block walls J Building materials such as some rocks K Water (from some wellsl o ------- The Health Risk How does Radon Induce Cancer? If inhaled, radon decay products (poionium-218 and polonium-214, solid form), unattached or attached to the surface of aerosols, dusts, and smoke particles, become deeply lodged or trapped in the lungs, where t hey can radiate and penetrate the cells of mucous membranes, bronchi, and other pulmonary tissues, The ionizing radiation energy affecting the bronchial epithelial cells is believed to initiate the process of carcinogenesis. Although radon-related lung cancers are mainly seen in the upper airways, radon increases the incidence of all histological types of lung cancer, including small cell carcinoma, adenocarcinoma, and squamous cell carcinoma. Lung cancer due to inhala- tion of radon decay products constitutes the only known risk associated with radon. In studies done on miners, variables such as age, duration of exposure, time since initiation of exposure, and especially the use of tobacco have been found to influence individ- ual risk. In fact, the use of tobacco multiplies the risk of radon-induced lung cancer enormously. What is the Evidence? More is known about the health risk of radon expo- sure to humans than about most other human car- cinogens. This knowledge is based on extensive epi- demiological studies of thousands of underground miners, carried out over more than 50 years world- wide, including miners in the United States and Canada. In addition to the miner data, experimental exposures of animals confirm that radon and its decay products can cause lung cancer. The research on lung cancer mortality in miners exposed to radon progeny is substantial and consis- tent, Studies of thousands of miners, some with fol- iow-up periods of 30 years and more, have been con- ducted in metal, fluorspar, shale, and uranium mines in the United States, Canada, Australia, China, and Europe. These studies have consistently shown an increase in lung cancer occurrence with exposure to radon decay products, despite differ- ences in study populations and methodologies. The mirier studies produced some interesting findings, ¦ At equal cumulative exposures, low exposures in the range of EPA's 4 pCi/L action level over longer periods produced greater lung cancer risk than high exposures over short periods. ¦ Increased lung cancer risk with radon exposure has been observed even after controlling for, or in t he absence of, other mine exposures such as asbestos, silica, diesel fumes, arsenic, chromium, nickel, and ore dust, ¦ Increased lung cancer risk has been observed in miners at relatively low cumulative exposures in the range of EPA's 4 pCi/L action level (Sevc O ------- Kunz, Tomasik et al, Health Physics 54(1):27-46,1988; Millies Wheeler et al, Proceedings of International Conference on Occupation Radiation Safety in Mining, Vol. 1, Canadian Nuclear Association; Radford and .St. Clair Renard, New England Journal of Medicine 310(23): 1485-1494, 1984; Woodward, Roder el al, "Cancer Causes and Control" 2:213-220, 1991). ¦ Nonsmoking miners exposed to radon have been observed to have an increased risk of lung cancer. The following table lists seven of the major epi- demiological studies of underground miners and their reported relative risk coefficients. ESTIMATES OF LUNG CANCER RISK FROM EPIDEMIOLOGICAL STUDIES OF UNDERGROUND MINERS EXPOSED TO RADON Study Population Average Exposure |WLM)° Relative Risk Coefficient (%/WLM)b References Czech Uranium Miners 313 226 1.92 1.5 Thomas el al. 1985 Sevc et al 1988 Ontario Uranium Miners 40-90 0.5-1.3 1.4C Muller 1 984 NAS1988 New Mexico Uranium Miners 111.4 1.8 Samet et al. 1991 Swedish Iron Miners (Malmberget) 81.4 3.6 1,4C Radford & St. Clair Renard 1984 NAS 1988 Colorado Plateau Uranium Miners 834 .45 .06c Thomas et al. 1985 NAS 1988 Eldorado (Beaverlodge) Uranium Miners 20,2 3.28 2.6C Howe et al. 1 986 NAS 1988 Newfoundland Fluorospar Miners 382.2 0.9 Morrison et al. 1988 "The excess relative "sk coeWieaent used m EPA's risk assessment (1 3WWIMJ is that derrved by the NAS BElR IV report based on their anafysis of studies of underground miners e o Working level month (WLM) is the cumulative exposure equivalent to one working level (WL) for a working month (170 hours). A WL is any com- bination of short-lived radon dough ters in one liter of air that will result in the emission of 1.3 x 105 MeV of potential alpha energy. A home exposure of 4 pCi/L for 70 years would approximately equal a cumula- tive exposure of 54 WLM (assuming 75% occupancy). fa The relative risk coefficient is the fractional increase above the baseline lung cancer incidence or mortality rate per WLM, For example, the Czech Uranium Miner demonstrated o 1.92% increased lung cancer risk for every WLM of exposure. Exposure to 5 WLM would therefore increase lung cancer risk by 9.6% over base- line. c Estimate based on reanalysis of the data by the NAS with the cooperation of the principal investigators. ------- A detailed discussion of the strengths and weak- nesses of the various miner studies can be found in the EPA's Technical Support Document for the 1992 Citizen's Guide to Radon, available from your state radon office, or the BEIR IV Report (National Academy of Sciences (NAS) 1988). Animal experiments conducted ill the United States and France also have confirmed the carcinogenicity of radon and have provided insight into the nature of the exposure-response relationship, as well as the modifying effects of the exposure rate. To date these animal studies have produced several relevant find- ings. ¦ I-fealth effects observed in animals exposed to radon and radon decay products include lung car- cinomas, pulmonary fibrosis, emphysema, and a shortening of life-span (U.S. DOE/Office of Energy Research 1988a), ¦ The incidence of respiratory tract tumors increased with an increase in cumulative expo- sure and with a decrease in rate of exposure (NAS 1988). ¦ Increased incidence of respiratory tract tumors was observed in rats at cumulative exposures as low as 20 WLM (NAS 1988). ¦ Exposure to ore dust or diesel fumes simultane- ously with radon did not increase the incidence of lung tumors above that produced by radon proge- ny exposures alone (DOE/Office of Energy Research 1988a). O ¦ Lifetime lung-tumor risk coefficients that have been observed in animals are similar to the life- time lung-cancer risk coefficients observed in human studies (DOE/Office of Energy Research 1988a), ¦ In a study of rats exposed to radon progeny and uranium ore dust simultaneously, it. was observed that the risk of lung cancer was elevated at expo- sure levels similar to those found in homes. The risk decreased in proportion to the decrease in radon-progeny exposure (Cross et al. 1991). In 1988, a panel of world experts convened by the World Health Organization's International Agency for Research on Cancer unanimously agreed that there is sufficient, evidence to conclude that radon causes cancer in humans and in laboratory animals (IARC, 1988). Scientific committees assembled by the National Academy of Sciences (NAS, 1988), the International Commission on Radiological Protection (ICRP, 1987), and the National Council on Radiation Protection and Measurement (NCRP, 1984) also have reviewed the available data and agreed that radon exposure causes human lung cancer. Recognizing that radon is a significant public health risk, scientific and professional organizations such as the American Medical Association, the American Lung Association, and the National Medical Association have developed programs to reduce the health risks of radon. The National Institute for Occupational Safety and Health (NIOSH) reviewed the epidemiological data and recommended that the annual radon progeny exposure limit for the mining industry be lowered (NIOSH 1987). ------- Is Occupational Exposure to Radon Comparable to Residential Exposure? Because questions have been raised about the appropriateness of using the epidemiological studies of underground miners as a basis for estimating the risk radon poses to the general population, the EPA commissioned the National Academy of Sciences (NAS) to investigate the difference between under- ground miners and members of the general public in the doses they receive per unit exposure due to inhaled radon progeny. The NAS report, published in 1991 (National Academy of Sciences, 1991, Comparative Dosimetry of Radon in Mines and Homes. National Academy Press, Washington, DC.), concluded that it is reason- able to extrapolate from the miner data to a residen- tial situation, but that the effective dose per unit of exposure for people in their homes is approximately 30 percent less than for the miners, in its analysis, NAS considered variables such as the amount and type of dust to which the radon decay particles would attach, the breathing rate of working miners com- pared to that of people at home, and the presence of women and children in the homes. EPA has adjusted its residential risk estimates accordingly. The result is still considerable- EPA now estimates that approximately 14,000 lung cancer deaths in the United States per year are due to resi- dential radon exposures, with an uncertainty range of 7,000 to 30,000. As more data are gathered about res- idential radon exposures, the risk estimates may be adjusted further. Enough data exists now, however, to be able to say with certainty that thousands of pre- ventable lung cancer deaths annually in the United States are attributable to indoor resi- dential exposure to radon. More information about residential exposure to radon is needed to answer important questions about radon's effect on women and children—groups not included in the occupational studies of miners. Although children have been reported to be at greater risk than adults of developing certain types of cancer from radiation, currently there is no conclu- sive evidence that radon exposure places children at any greater risk. Some miner studies and animal stud- ies indicate that for the same total exposure, a lower exposure over a longer time is more hazardous than short, high exposures. These findings increase con- cerns about residential radon exposures. Epidemiological case control studies are underway in the U.S. and Europe, the pooler! results of which should enhance the understanding of the risk of resi- dential exposure to radon. What about Smoking and Radon Exposure? Some people ask whether the lung cancer deaths attributed to radon exposure actually may be the result of smoking. A 1989 study by researchers from the National Institute for Occupational Safety and Health, the Centers for Disease Control, the Harvard School of Public Health, and the University of California at Davis demonstrated a greatly increased lung cancer risk in nonsmoking uranium miners exposed to high radon concentrations: compared to typical non-smoking populations, these miners had 9 to 12 times the risk of developing lung cancer (Roscoe et al, JAMA 262(5): 629-633, 1989). Evidence from some of the epidemiological studies of underground miners, primarily U.S. uranium min- o ------- ers, indicates that radon exposure and smoking may risk of lung cancer; however, exposure to both have a synergistic relationship. Either smoking or greatly enhances that risk, radon exposure can independently increase the Radon Risk comparison for Smokers and Nonsmokers Radon Level If 1,000 people who smoked were exposed to this level over a lifetime... If 1,000 people who never smoked were exposed to this level over a lifetime... 20 pCi/L (740 Bq/m')* About 1 35 people could get lung cancer Abou-t 8 people could get lung cancer 10 pCi/L (370 Bq/m3) About 71 people could get lung cancer About 4 people could get lung cancer 8 pCi/L (296 Bq/m ) About 57 people could get lung cancer About 3 people could get lung cancer 4 pCi/L (1 48 Bq/m3) About 29 people could get lung cancer About 2 people could get lung cancer 2 pCi/L (74 Bq/m ) About 15 people could get lung cancer About 1 person could get lung cancer 1.3 pCi/L (48.1 Bq/m) About 9 people could get lung cancer Less than 1 person could get lung cancer 0.4 pCi/L (14.8 Bq/m) About 3 people could get lung cancer Less than 1 person could get lung cancer * Bq/m3= Bequerel/meter3 o ------- Why Should Every Home be Tested? The EPA and the U.S. Surgeon General recommend testing all homes below the third floor for radon. Data gathered by the EPA national radon survey indicate that elevated radon levels are present in about six million homes throughout the United States. In every state there are homes with dangerously high radon levels. Because the radon concentration inside a home is due to factors relating to its structure and geographic location, each individual home must be tested to determine its radon level. Tvvo adjacent houses may have radically different radon levels. And any kind of home can have elevated levels—new or old, drafty or well-sealed, and basement or non-base- ment. How do You Obtain a Reliable Test Result? Although radon cannot be seen or smelled, with the proper equipment its presence is relatively easy to detect. The EPA operates a voluntary Radon Measurement Proficiency (RMP) Program that evaluates radon measurement companies and the test services they offer. EPA recommends that testing services be pur- chased from one of the organizations listed by EPA or certified by the state. Most companies indicate U.S. EPA or RMP approval on the test kit box by dis- playing the phrase, "Meets EPA Requirements." A list of radon measurement companies and individu- als the EPA has determined to be "proficient" can be obtained from the State Radon Office. (See page 16.) A test kit obtained from a qualified company, if used according to directions, should provide accurate results. Tke Solution Radon Testing Methods The quickest way to test for radon is with a short- term "do-it-vourself radon test kit, available by mail order and in many retail outlets or by hiring an EPA qualified or state-certified radon tester. Common short-term test devices are charcoal canisters, alpha track detectors, liquid scintillation detectors, eleetret ion chambers, and continuous monitors. A short-term testing device remains in the home for 2 to 90 days, depending on the type of device. Because radon lev- els tend to vary from day-to-day and season-to-sea- son, a long-term test is more likely than a short-term test to measure the home's year-round average radon level. If results are needed quickly, however, a short- term test followed by a second short-term test may be used to determine the severity of the radon prob- lem. Long-term test devices, comparable in cost to devices for short-term testing, remain in the home for more than three months. A long-term test is more likely to indicate the home's year-round average radon level than a short-term test. Alpha track detec- tors and elect ret ion detectors are the most common long-term test devices. O ------- i?flladon Test Devices Charcoal canisters and liquid scintilJation detectors contain small quantities of activated charcoal. Radon and its decay pmducts arc adsorbed onto the charcoal and are measured by counting with a sodium iodide detector or a tiqvid scintilla- tion counter. Alpha track detectors contain a small sheet of plastic that is exposed for a period of one to three months. Alpha particles etch the plastic as they strike it. These marks are then chemi- cally treated and counted in the laboratoi~y to determine the ixidon concentration. Electret ion detectors contain an electroslically charged Teflon disk. Ions generated by the decay of radon strike and reduce the surface milage of the disk. By measuring the voltage reduction, the radon concentration can be calculated. Continuous monitors are active devices which need power to function. They require operation by trained testers and work by continuously measuring and recording the amount of tvdon in the home. Charcoal Canister Eleetret Ion Detector © :!f$w To Test During a short-term test, doors and windows are closed 12 hours prior to testing and throughout the testing period. (A short-term test lasting two or three days should not be conducted during unusually severe storms or periods of unusually high winds.) The test kit is placed in the lowest lived-in level of t he home, at least 20 inches above the floor, in a room that is used regularly, but not in the kitchen or bath- room where high humidity or the operation of an exhaust fan could affect the validity of the test. At the end of the test period, the kit is mailed to a laboratory for analysis; result s are mailed back in a few weeks. In some cases, such as real estate transactions, trained EPA-listed or state-certified contractors con- duct the radon test. The EPA's pamphlet Home Buyer's and Seller's Guide to Radon, which addresses issues during real estate transactions, is available from state radon offices. ------- Interpreting Radon test Results A. if the short-term test result is 4 pCi/L or higher, conduct a follow-up test to confirm the results. B. Follow-up with either a long-term test or a second short-t erm test. For a better understanding of the home's year-round average radon level, take a long- term test. If results are needed quickly, take a second short-term test. The higher the initial short-term result, the more certain the homeowner can be to con- duct a short-term rather than a long-term fol- low-up test. If the first short-term test result is several times the action level—for example, about 10 pCi/L or higher—a second short- term test should be taken immediately. C. If the long-term follow-up test result is 4 pCi/L or more, fix the home. If the homeowner followed up with a second short-term test: the higher the short-term results, the more certain the homeowner can be that the home should be fixed. The homeowner should consider fixing the home if the average of the first and sec- ond test is 4 pCi/L or higher. Basis for the 4 pCi/L Radon "Action Level" No radon level is considered "safe." The risk of developing lung cancer is directly proportional to the levels and duration of exposure to radon: the higher the radon concentration, the higher the lung cancer risk. The 4 pCi/L "Action Level" is based on current mitigation technology. Today mitigation technology can almost always reduce high radon concentration levels to below 4 pCi/L and to 2 pCi/L or below 70-80 percent of t he time. The average radon level in homes is about 1.25 pCi/L. Although Congress passed legisla- tion in 1988 establishing a national goal that indoor radon levels not exceed ambient outdoor radon levels (0.2-0,7 pCi/L), this goal is not yet technologically achievable. Radon Reduction Methods Radon in soil is drawn indoors by the differential between the relatively low air pressure in the house and the higher air pressure in the soil. Therefore, radon reduction strategies fall into two basic cate- gories: those that prevent the entry of radon gas into the home, and those that attempt to remove the radon once it has entered the home. In most, situa- tions, the first approach—preventing radon entry—is the most effective. Although sealing cracks and other openings in the foundation is a basic part of most approaches to radon reduction, sealing alone is not recommended; it is best done in conjunction with other mitigation techniques to enhance their effectiveness. The most popular radon mitigation technology is called "sub-slab depressurization" or "slib-slab suc- tion," The "sub-slab depressurization" technique © ------- removes radon-laden air from beneath the foundation and vents it outside the home by installing a fan and inserting a pipe through the foundation into the aggregate below, running it to a point outside the shell of the house. A similar technique, "sub-mem- brane depressurization," which is effective in build- ings with earth-floored crawlspaces or basements, uses a plastic barrier over the soil as a collection cover. Another depressurization technique for pre- venting radon entry, "blockwall depressurization," Seal Around •Entry Points Warning TYPICAL RADON MITIGATION SYSTEM EXHAUST 4 Radon Sxhaust Pipe (typically hidden in closet or between walls!. System Failure Device © uses a fan and duct work to draw suction on the hol- low interior cavities of a concrete block wall. By keeping the air pressure within the block wall lower than the air pressure in the basement, the soil gas is removed before it can enter the basement . Reducing radon levels requires technical knowledge and special skills. The EPA operates a Radon Contractor Proficiency (RCP) Program that evaluates radon reduction contractors. As with t he Radon Measurement Proficiency (RMP) Program, the EPA sends a list of RCP contractors to state radon offices {see page 16). Selecting a radon contractor is much like choosing a contractor for other home repairs; it makes sense to get references and more than one estimate. The average cost to correct a radon prob- lem in a home is about $1,200, although it can range from about $500 to about $2,500. A free copy of A Consumer's Guide to Radon Reduction is available from state radon offices. People who choose to fix their homes themselves should refer to the EPA's technical guide, Radon Reduction Techn iques for Detached Houses, available from state radon offices. After the radon reduction procedure is complete, the home should be ret est ed Most radon reduction systems include a monitor that will alert the home- owner if the system needs servicing. ------- Other Indoor Air Pollutants Preventive Measures that can be taken ta reduce your exposure to indoor air pollutants include the following: Environmental Tobacco Smoke (ETS) • Don'f smoke around others, particularly children. • Every organization dealing with children should have a smoking policy that effectively protects children from ETS. • In the work place, prohibit smoking indoors or provide separately ventilated smoking areas. • If smoking is permitted in restaurants and bars, placement of smoking areas should be designated fa minimize nonsmoker exposure. Biological Air Pollutants • Provide adequate outdoor qir ventilation. • Keep equipment water reservoirs clean. • Maintain relative humidity below 50 percent. • Eliminate standing water, wash bedding and soft toys frequently inhol water. • Vacuum carpets and upholstered furniture regularly. Volatile Organic Compounds (VOCs) • Remove the source. • Avoid use. • Increase ventilation when using products. Other Combustion Products • Vent furnaces, water heaters ond clothes dryers to the outdoors. • Periodic professional inspections and maintenance of major appliances. • Regularly clean Fireplace and waod/coal stove flues. Environmental Tobacco Smoke (ETS) The U.S. Environmental Protection Agency has classified environmental tobacco smoke (ETS) as a Group A (known human) carcinogen. EPA estimates that approximately 3,000 lung cancer deaths in the United States per year among nonsmokers are due to ETS. The U.S. Surgeon General, the National Research Council, and the National Institute for Occupational Safety and Health have all concluded that passive smoking can cause lung cancer in other- wise healthy adults who never smoked. Children's lungs are even more susceptible to harm- ful effects from ETS. In infants and young children up to three years, exposure to ETS causes an approxi- mate doubling in the incidence of pneumonia, bron- chitis, and bronchiolitis. There is also strong evidence of increased middle ear effusion and reduced lung function and lung growth. Several recent studies link ETS with increased incidence and prevalence of asth- ma and increased severity of asthmatic symptoms in children of mothers who smoke heavily. Environmental tobacco smoke is a complex mixture of more than 4,000 chemicals found in both vapor and particle phases, many known toxic and carcinogenic agents. ETS consists of both "sidestream" smoke, the emission from the burning erici of the cigarette, and exhaled "mainstream smoke," the smoke inhaled by the active smoker. 0 ------- Biological Air Pollutants Dust mites, molds, animal dander, and other biolog- icals are found in some degree in every home and workplace. High relative humidity is the primary fac- tor encouraging biological agents to grow and be released into the air. Biological agents are known to cause three types of human diseases: infections, where pathogens invade human tissues; hypersensi- tivity diseases, where specific activation of the immune system causes disease; and toxicoses, where biologically produced chemical toxins cause direct toxic effects. Volatile Organic Compounds (VOCs) Volatile Organic Compounds (VOCs) are emitted as vapors or gases at ordinary temperatures from a vari- ety of sources. Many are toxic, including benzene, carbon tetrachloride, and formaldehyde. VOCs can be found in paint, upholstery, spray cans, copy machine toners, clothing, and other sources. Health effects range from irritation of the eyes and respiratory sys- tem to kidney or liver damage, cancer, or birth defects. © Other Combustion Products Aside from ETS, the major combustion pollutants— carbon monoxide (CO), nitrogen dioxide (N02), and sulfur dioxide (S02)—that may be present at harmful levels in t he home or workplace stem chiefly from malfunctioning or misusing heating devices. An addi- tional source of exposure may be motor vehicle emis- sions as a result of a garage or loading dock located near air intake vents. Symptoms may mimic influenza and include fatigue, nausea, dizziness, headaches, cognitive impairment, and tachycardia during the heating season. For more information on these and other indoor air pollutants, call EPA's indoor Air Quality Information Clearinghouse (1-800-438-4318) for Indoor Air Pollution: An Introduction for Health Professionals and The Inside Story: A Guide to Indoor Air Quality. ------- Most Commonly Asked Questions About Radon Where does radon come from? A, Radon is a naturally occurring gas that results from the breakdown of uranium commonly found in soil. How does radon enter my home? A. Radon comes up through the soil and rocks surrounding your home and seeps through cracks in concrete walls and floors, floor drains, sump pumps, joints, and hollow block walls. Why haven't I heard of the radon danger until recently? A. Radon has always existed. However, it was not until the 1980s that dangerous radon levels were found inside homes across the U.S. What are the health risks? A, Radon is the second leading cause of lung cancer. If I have a radon problem, cam It he corrected? A, Yes. The use of trained personnel is recom- mended. State radon offices can recommend qualified contractors. In some cases, the problem can be treated by the homeowners if they have experience with other kinds of home repair. Will my neighbor's radon measure- ment indicate whether or not 1 have a radon problem? A. No. Radon levels vary from house to house. The only way to know if you have a radon prob- lem is to conduct, a test- How can I get a reliable radon test kit? A. Kits can be purchased through the mail or from your local grocery or hardware store or other retail outlets. Look for a test kit from a company that is State-certified or EPA-listed. How do I know if I have radon m my home? Am By testing with an EPA-listed or State-certi- fied easy-to-use, inexpensive test kit as soon as possible, or by hiring an EPA-listed or State-cer- tified contractor to test your home for you. © ------- State Radon Contacts Alabama 800/582-1866 205/242-5315 Alaska 800/478-4845 907/465-3018 Arizona 602/2554845 Arkansas 501/661-2301 California 800/745-7236 916/324-2208 Colorado 800/846-3986 303/692-3057 Connecticut 203/566-3122 Delaware 800/554-4636 302/739-3028 DC 202/727-5728 Florida 800/543-8279 904/488-1525 Georgia 800/745-0037 404/657-6534 Hawaii 808/586-4700 Idaho 800/445-8647 208/334-6584 Illinois 800/325-1245 217/786-7127 Indiana 800/272-9723 317/633-0150 Iowa 800/383-5992 515/242-5992 Kansas 913/296-1561 Kentucky 502/564-3700 Louisiana 800/256-2494 504/925-7042 Maine Maryland Massachusetts.... Michigan Minnesota Mississippi Missouri Montana Nebraska Nevada New Hampshire. New Jersey New Mexico New York North Carolina... North Dakota Ohio Oklahoma Oregon 0 800/232-0842 207/287-5676 800/872-3666 410/631-3301 800/445-1255 413/586-7525 517/335-8190 800/798-9050 612/627-5012 800/626-7739 601/354-6657 800/669-7236 314/751-6083 406/444-3671 800/334-9491 402/471-2168 702/687-5394 .800/852-3345 x4674 603/271-4674 800/648-0394 609/987-6396 505/827-4300 800/458-1158 518/458-6451 919/571-4141 701/221-5188 800/523-4439 614/644-2727 405/271-5221 503/731-4014 Pennsylvania 800/237-2366 717/783-3594 717/783-3595 Rhode Island 401/277-2438 South Carolina 800/768-0362 South Dakota 800/438-3367 605/773-3351 Tennessee 800/232-1139 615/532-0733 Texas 512/834-6688 Utah 801/538-6734 Vermont 800/640-0601 802/865-7730 Virginia 800/468-0138 804/786-5932 Washington 800/323-9727 206/753-4518 West Virginia 800/922-1255 304/558-3526 Wisconsin 608/267-4795 Wyoming 800/458-5847 307/777-6015 Guam 617/646-8863 Puerto Rico 809/767-3563 Virgin Islands 800/468-0138 National Radon Hotline 1 800/SOS-RADON ------- Additional Information Sources and Suggested Reading List The following free materials are available from your state radon office. • Reducing Radon Risk • A Citizen's Guide lo Radon (1892 Edition): The Guide to Protecting Yourself and Your Family /row Radon (available in Spanish) » Tech n ical Support Docu men t for the 1992 Citizen's Guide to Radon • Home Buyer's and Sellers Guide to Radon • Radon it/ Schools • Consumer's Guide to Radon Reduction For additional indoor air publications, call EPA's Indoor Air Quality Information Clearinghouse (1-800-438-4318). • The Inside Story:A Guide to Indoor Air Quality • Indoor Air Pollution: An Introduction for Health Profess io n a Is • Secondhand Smoke Your state radon office has other promotional materials, including free posters, to display in your office. Suggested Reading List: Council on Scientific Affairs. 1987. American Medical Association (AMA). Radon in Homes. Journal of t lie American Medical Association. 258: <568-672. Council on Scientific Affairs. Health Effects of Radon Exposure. Archives of Internal Medicine. 151: (574-677. Lubin, J.M., Samet and Weinberg, C. 1990. Design Issues in Epidemiologic studies of Indoor Exposure to Rn and Risk of Lung Cancer. Health 1'hvsics. 59(0): 807-817. National Academy of Sciences. 1991. Comparative Dosimetry of Radon in Mines and I ionics. National Acedemy Press, Washington. DC. National Academy of Sciences. 1988, Healt h Risk of Radon and Other Internally Deposited Aloha Emitters: BE1R IV. National Academy Press, Washington. DC. Roscoe. R.J.. et al. 1989. Lung Cancer Mortality Among Non-Smoking Cranium Miners Exposed to Radon Daughters, Journal of the American Medical Association. 262(5): (>29-0-33. Samet. J.M., Stojwijk, J. and Rose, S. 1991a. Summary: International Workshop on Residential Rn Epidemiology. Health physics, 00(2): 223-227. Samet, J.M. and Hornung, R. 1990, Workshop on Indoor Air Quality: Review of radon and Lung Cancer Risk. Risk Analysis. 10(1): 65-75, U.S. DOE/Offiee of Energy Research, 1989. Internaional Workshop on Residential Radon Epidemiology: Wo r k sh on 1 *roc c e d in gs. Commission of European Communities, Radiation Protection Program. CONF- 8907178. © ------- vvEPA United States Environmental Protection Agency September 1993 Washington,D.C. 20460 402-K-93-008 Air and Radiation (6604J) Official Business Penalty for Private Use S300 CKX Recycled/Recyclable r\ \\ Printed wilh Soy/Canola Ink on paper tnat XnCS contains at least 50% recycled liber ------- |