United States
                    Environmental Protection
                    Agency
Health Effects Research
Laboratory
Research Triangle Park NC 2771 1
                    Research and Development
EPA-600/S1-83-007  Sept. 1983
&EPA         Project  Summary
                    Fetotoxic  Effects  of  Nickel  in
                    Drinking  Water  in   Mice
                    Ezra Berman and Blair Rehnberg
                      Nickel chloride was administered in
                    drinking water to pregnant mice from
                    the 2nd through the 17th day of ges-
                    tation at nickel doses of 0, 500, or
                    1000 ppm. Fetal or maternal toxicity
                    was not seen after administration of
                    500 ppm of nickel. However, the higher
                    dose  caused spontaneous abortions,
                    loss of fetal mass in survivors, and loss
                    of maternal  mass. The oral route of
                    administration via drinking water was
                    at least 2.7 times less effective than
                    parenteral routes in  producing  fetal
                    effects.
                      This Project Summary was developed
                    by EPA's Health Effects Research Lab-
                    oratory, Research Triangle Park, NC, to
                    announce key findings of the research
                    project that is fully documented in a
                    separate report of the same title (see
                    Project Report ordering information at
                    back).

                    Introduction
                      This report summarizes the work done
                    to examine a soluble nickel salt for its
                    efficacy to cause fetotoxic effects in mice.
                    Administering the compound in drinking
                                                Off
dam was killed by suffocation with C02
gas and weighed. The uterus was removed
and counts were made of dead (resorbed
and newly dead) and live fetuses. The live
fetuses were removed, examined for gross
abnormalities, blotted dry,  and weighed.
Two of every three fetuses were fixed in
Bourn's solution, and the third fetus was
preserved in alcohol.
  Pregnancy rates and  other incidental
data were analyzed using contingency
tables. Unless stated otherwise, all data
were analyzed using the litter as the ex-
perimental unit.
  The  rate of water consumption in mice
was 1  60  ml/kg body mass/24  h  esti-
mated by observations of bred mice gang-
caged from the 14th through the 1 8th day
of gestation. During 24 h of this study, the
consu mption of nickel at concentrations of
500 and 1000 ppm  in water was  esti-
mated to be 80 and 1 60 mg/kg body
mass,  respectively.  For the period  of
administration (2nd through 1 7th day  of
gestation), the total consumption of nickel
at concentrations of 500 and 1000 ppm
was estimated  to be 1280  and 2560
mg/kg body mass, respectively.

-------
live for producing nickel toxicity  A wide
range and high incidence of terata  in
fetuses of mice administered 4.6 mg  of
NiCli/kg body mass IP once between the
7th and 1 1 th day of gestation has been
observed.  On  the  basis of the  ratio  of
effectiveness of an oral vs. an injected
dose, a 4.6 mg IP dose would be equiva-
lent to a 60 mg oral dose.
  In our study, we could not induce a wide
range and incidence  of terata after we
administered nickel in  1 650 mg/kg orally
daily (1 000 ppm in drinking water).  Only
decreased fetal body mass  was observed.
Any higher dose was so toxic for both dam
and conceptuses that the  frequency did
not continue  Therefore, our 1 60 mg/kg
dose is  the closest  response to a 4.6
mg/kg IP dose that we could achieve. As
the calculated oral equivalent of 4.6 mg/kg
IP is 60 mg/kg, our 1000 ppm dose is 2.7
times less effective. Therefore, such cal-
culations do not adequately account for
the observed differences.
  The variation in results may depend on
some other factor in the route of adminis-
tration which decreases the effectiveness
of low but continuous oral  intake com-
pared to high  single parenteral route. It
appears, therefore, that the fetal effects of
nickel poisoning may be overestimated by
experiments using parenteral methods.

Conclusions
  The oral  dosage of  nickel required  to
produce fetal toxicity  or teratology may
have been overestimated when based on
studies using parenteral administration. A
factor as large as 2.7 times may be needed
to correlate the effects of  parenterally
administered nickel with those of orally
administered nickel.
   The EPA authors Ezra Berman (also the EPA contact) and Blair Rehnberg are with
     the Health Effects Research Laboratory, Research Triangle Park, NC 27711.
   The complete report,  entitled "Fetotoxic Effects of Nickel in Drinking  Water in
     Mice."  (Order No. PB 83-225 383; Cost: $7.00. subject to change) will be
     available only from:
          National Technical Information Service
          5285 Port Royal Road
          Springfield, VA 22161
          Telephone: 703-487-4650
   Ezra Berman can be contacted at.
          Health Effects Research Laboratory
          U. S. Environmental Protection Agency
          Research Triangle Park, NC 27711
                                                 US GOVERNMENT PRINTING OFFICE 1983-659-017/7176
                                                                      -659-017/7

-------