&EPA
          United States
          Environmental Protection
          Agency
           Office of Research and
           Development
           Washington, DC 20460
EPA/600/R-92/147
June 1992
Proceedings of the 4th
Investigators' Meeting for
EPA's Biotechnology-
Biological Control Agent
Risk Assessment
Research Program

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                                          EPA/600/R-92/147
                                                 June 1992
            PROCEEDINGS OF THE 4th
     INVESTIGATORS' MEETING FOR ERA'S
BIOTECHNOLOGY-BIOLOGICAL CONTROL AGENT
    RISK ASSESSMENT RESEARCH PROGRAM
                 APRIL  8-12, 1991
                       Prepared by

                     James E. Harvey
                  Technical Resources, Inc.
          Gulf Breeze Environmental Research Laboratory
                 Gulf Breeze, Florida 32561
                Contract Number 68-03-3479
                      Project Officer
                     Richard B. Coffin
           Microbial Ecology and Biotechnology Branch
          Gulf Breeze Environmental Research Laboratory
                 Gulf Breeze, Florida  32561
     GULF BREEZE ENVIRONMENTAL RESEARCH LABORATORY
          OFFICE OF RESEARCH AND DEVELOPMENT
         U.S. ENVIRONMENTAL PROTECTION AGENCY
               GULF BREEZE, FLORIDA 32561
                                           Printed on Recycled Paper

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r
                                            DISCLAIMER

                  The information in this document has been funded wholly or in part by the U.S.
             Environmental Protection Agency under various cooperative agreements. It has been
             subject to the Agency's peer and administrative review, and it has been approved for
             publication as an EPA document.  Mention  of trade names or commercial products
             does not constitute endorsement or recommendation for use.

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                            TABLE OF CONTENTS
 Acknowlegment

 Preface

 Introduction

 Invited Special Presentations
      European Biotechnology Research Presentations
      Canadian Biotechnology Efforts
      U.S. Department of Agriculture Biotechnology Overview
      U.S. Environmental Protection Agency Presentations

 Research Presentations
      Session I: Environmental Exposure
            Detection/Enumeration
            Dispersal/Transport
            Survival/Colonization
            Gene Transfer

      Session II: Environmental and Human Health Effects
            Ecological Processes
            Higher Organisms
            Human Health

      Session III: Risk Control
            Mitigation
            Field Releases
            Process Containment

Contributor Index
Affiliation Index
Subject Index
Page

   iv

   v

   1

   3
   4
  15
  20
  22
 35
 37
 61
 81
 102

129
 131
 169
 207

 223
 225
 228
 242

247
249
251
                                   111

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                          .  ACKNOWLEDGMENT

      This publication is a result of the patience and cooperation of the researchers
who contributed to the 1991 All-Investigators Meeting. We wish to acknowledge the
word  processing and editing assistance of Ms. Maureen Stubbs, and Ms. Valerie
Coseo, Computer Science Corporation.  We also wish to acknowledge the assistance
of Ms. Nancy Padgett,.Technical Resources, Inc., both at the meeting and in the
completion of this document.
                                    IV

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                                  PREFACE

    The Office of  Research  and Development (ORD)  in the  U.S.  Environmental
Protection Agency is responsible for providing EPA program offices with scientific and
technical information on biotechnology products intended for environmental release.
This includes researchon genetically engineered microorganisms (GEMs) and biological
control agents (BCAs).  To accomplish this, ORD created a Matrix-managed program
for Biotechnology/BCA Risk Assessment and designated the Gulf Breeze Environmental
Research Laboratory as the lead laboratory.  To assure coordination and  promote
multi-disciplinary research approaches, the Matrix manager holds periodic meetings
attended by laboratory and headquarters scientists and  cooperative agreement and
contract researchers.  This document is a compilation of extended abstracts of the
research presented at the 1991 All-Investigators meeting> the fourth meeting of this
type.  The meeting was held at the Holiday Inn Crowne Plaza, Crystal City, Virginia,
April 8-12.  Extended abstracts submitted from specially invited guests representing
biotechnology efforts  in  Canada  and  several European  countries  reflect the
international collaboration theme of the meeting. These invited abstracts are followed
by scientific presentations from EPA laboratory, cooperative agreement and contract
personnel separated into the three program element research areas: exposure, effects
and risk control.

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                                  SECTION 1
                                INTRODUCTION
    During   the  last   two  decades,
 sophisticated  scientific advances  in
 molecular  biology have increased our
 ability  to   change  or  combine the
 inherited   characteristics  of   micro-
 organisms. The power of recombinant
 DNA techniques  to make extremely
 precise  alterations   in the   genetic
 character  (genotype)  and expression
 (phenotype) of organisms  has  created
 both the promise of  enormously useful
 products   as  well  as  concern over
 potential environmental release.
    Among   the   potential  industrial
 products   and  applications  of  this
 technology   are   microorganisms
 designed to degrade toxic pollutants,
 leach minerals, enhance oil recovery,
 produce industrial chemicals, and act as
 pesticides.  These activities fall within
 EPA's regulatory  purview  under the
 Toxic Substances Control  Act  (TSCA)
 and,  for  pesticidal  applications, the
 Federal  Insecticide,  Fungicide  and
 Rodenticide Act (FIFRA) as established
 by  the  Coordinated  Framework for
 Regulation  of  Biotechnology  (EPA,
 1986).      These  regulations   were
 intended to  protect the environment
 from perturbation by industrial products
 regardless  of the scientific process of
their  manufacture, and are therefore
applicable   to  the   products   of
 biotechnology.
   EPA regulators rely on the results of
research and development programs to
provide the scientific  and technical
knowledge  required  for  successful
regulatory  policies.  Risk assessment
methods for predicting  the risk posed
by some chemical pesticides arid toxic
 industrial chemicals, including fate and
 effects  data,   are   reasonably   well
 developed and have been incorporated
 into regulatory policies.
   The   more   dynamic   properties
 presented by microorganisms, including
 replication, mutation and gene transfer,
 coupled   with   the   complexity  of
 molecular biological techniques create a
 high degree of  uncertainty when risk
 assessment  methods  developed  for
 chemicals are  applied to  genetically
 engineered microorganisms (GEMs) or
 biological control agents (BCAs). BCAs
 include microbial pest  control agents
 i.e.  Bacillus thuringiensis  derivatives,
 and  biochemical pest control agents,
 such  as  insect  growth   regulators.
 Concern  over   the   potential   risks
 introduced by the environmental release
 of biotechnological products  and the
 need to reduce uncertainty in assessing
 risk led the Agency to establish the Bio-
technology/Biological  Control Agent
 Risk Assessment Research Program in
 1985.
   Participating  ORD research labora-
tories  and their expertise  areas are
listed below:

   Gulf Breeze, FL., Environmental Research
   Laboratory (GBERL), Lead Laboratory, Fate
  ,and  Effects of GEMs and BCAs in Marine
   and  Estuarine Ecosystems.

   Corvallis, OR.,  Environmental  Research
   Laboratory (CERL),  Fate,  Effects,  and
   Transport of GEMs and BCAs in  Terrestrial
   Ecosystems.

   Duluth,  MN.,  Environmental  Research
   Laboratory (DERL), Fate and Effects of
   MPCAs in Freshwater Environments.

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   RTF,  NC.,   Health  Effects  Research
   Laboratory (HERD, Human Health Effects
   from GEMs and MPCAs.

   Cincinnati, OH., Risk Reduction Engineering
   Laboratory (RREL), Control of Risk through
   Containment and Decontamination.

   Las Vegas, NV., Environmental Monitoring
   Systems Laboratory (EMSU,  Aerosol Dis-
   persal of Biologicals, Model Development.

   Also, the  Office of Environmental
Processes and Effects Research in EPA
Headquarters   at Washington,  DC.,
serves as a liaison between ORD  and
Program Offices.
   ORD scientists have developed a
complementary   extramural  research
program fostering close interaction with
academic  scientists in the  fields  of
genetics,  biochemistry,  ecology  and
molecular  biology.  This cooperative
agreement  process  promotes   col-
laboration  between diverse  disciplines
to  solve   research  problems  often
resulting  in  scientific  advancement.
Over twenty-five universities in the U.S.
and abroad have contributed to  EPA's
program and to  scientific information
exchange.   Scientists at  EPA  labora-
tories  and under  EPA  cooperative
agreements have  further  facilitated
scientific exchange by participating in
national  and   international  scientific
meetings,  publishing journal  articles,
and    participating  in   research
coordination workshops.
   The knowledge base for character-
izing  the  risk posed by  GEMs/BCAs
continues to improve and has potential
to estimate and to reduce those risks.
The use of information developed for
assessing   risk  to  create  predictive
models  and   to   develop  control
strategies   represents a  shift  in  our
capabilities  towards risk   reduction.
Research designed  to  improve  our
understanding  of  problems  and  to
identify  promising solutions to those
problems are important and recognized
tools for reducing environmental risk.
EPA  funded  research  provides  an
important   basis  for   continued
development of  methods  and tech-
niques  needed for reliably  evaluating
the  behavior of  microorganisms and
microbial communities.
   The  ecological processes mediated
by  microorganisms are crucial to the
operation and health of ecosystems.  If
these processes  are  affected  by en-
vironmental stress resulting  from the
introduction of novel microorganisms or
their toxic by-products,  then the po-
tential for affecting the  rest of  the
ecosystem is  significant.   Continued
development of methods for evaluating
and  improving  the quality of  eco-
systems is necessary if strategies for
assessing  and  reducing  deleterious
changes in ecosystems health are to be
effective, timely,  and  relevant.  The
research program's mission  is to  dev-
elop these methods and techniques and
to provide guidance in their appropriate
application  for   characterizing  and
reducing risk to the environment.
   Extended   scientific   abstracts
describing EPA funded research com-
prise most of this report, the  fourth in a
continuing  proceedings series.   The
abstracts  are   grouped   by  broad
research  areas   of   environmental
exposure, environmental  and  human
health  effects,  and  control  of  risk
through  mitigation and  containment
strategies.   These abstracts demon-
strate  the  Scientific  direction  and
accomplishments  of  EPA's Biotech-
nology/Biological  Control  Agent  Risk
Assessment Program.

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                                 SECTION 2
                     INVITED SPECIAL PRESENTATIONS

  Information dissemination and technical information transfer are important facets
of EPA's Biotechnology Risk Assessment Program.  To improve international under-
standing and foster collaborative  research efforts,  representatives from  several
European countries, Canada, USDA, and EPA were invited to present an overview of
their biotechnology efforts. Extended abstracts of those presentations comprise this
section.
M. Hoefle, National Research Center for Biotechnology,
Braunschweig, Germany

P. Kearns, European Organization for Economic Cooperation and Development,
Paris, France                                                          ,

M. Reuss, Ministry of the Environment, Charlottenlund, Denmark

T. Mclntyre, Environment Canada, Hull, Quebec, Canada

D. MacKenzie, National Biological Impact Assessment Program, U.S. Department of
Agriculture, Washington, D.C.

E. Milewski, Office of Pesticides and Toxic Substances, U.S. Environmental
Protection Agency, Washington, D.C.

P. Sayre, Office of Toxic Substances, U.S. Environmental Protection Agency,
Washington, D.C.                      •

W. Schneider, Office of Pesticides Programs, U.S. Environmental Protection
Agency, Washington, D.C.

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     OVERVIEW OF GESELLSCHAFT FUR BIOTECHNOLGISCHE FORSCHUNG
        mbH/NATIONAL RESEARCH CENTER FOR BIOTECHNOLOGY (GBF)

                               Manfred Hoefle
                  Gesellschaft fur Biotechnolgische Forschung
                        Braunschweig, West Germany
   The   GBF  (Gesellschaft   fur
Biotechnolgische   Forschung   mbH/
Research  Institute for Biotechnology
had  its origins  in the  "Institute for
Molecular Biological Research (GMBS)"
started and funded by the Volkswagen
Foundation.   Since  the  inception of
GBF,  it  has  been financed  by  the
Federal Government, represented by the
Federal  Ministry  of  Research   and
Technology (BMFT) on the one hand,
and the State of Lower Saxony on the
other, to a ratio of 90:10.  The articles
of the company incorporated  in 1976
reinforce  the  structure of  GBF as  a
research-oriented  national   research
centre. The statutes governing the GBF
were amended in 1984.  The GBF is a
member of  the  AGF (Association of
National   Research   Centres),    and
employed  a total of 590 staff at the end
of 1989.
   The objective of the company is the
operation of a multidisciplinary  research
centre in the area of biotechnology with
the aim of contributing to the  solution
of public duties and tasks by means of
comprehensive   biotechnological
research and development work. The
research and development activities at
the GBF are basically located within the
framework of the Federal Government's
"Applied  Biology and Biotechnology"
programme.     The  GBF   pursues
exclusively   peaceful   purposes,
publishing the results of scientific work.
   The structure and operation of the
bio-pilot plant based on interdisciplinary
cooperation, facilitates the work on
research  projects within  the  GBF as
well  as in the  form  of cooperation in
working  groups  involving industry,
universities  and  other  institutions.
Accordingly the GBF is devoted above
all to the following tasks:

*  applications-oriented basic research
   in the areas of  microbiology,  cell
   biology, molecular biology, genetics,
   biochemistry,   natural   product
   chemistry, enzyme  technology and
   bioengineering,

*  the   development   of   new
   pharmacological   and   technically
   significant   natural   products  and
   research on  their mode  of action,

*  developmentof new biotechnological
   processes  as  a   contribution  to
   maintaining  an assured supply of
   basic products for  the chemicals,
   pharmaceutical   and   foodstuff
   sectors, as well as processes for the
   reduction of environmental pollution,

*  the  transfer from  the biotechno-
   logical laboratory process  to  the
   semi-industrial  scale  as   a   pre-
   condition for  the development of
   tried industrial processes.

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* the  support of  external  research
  groups  in  the  sectors of biology,
  chemistry  and  medicine  by  the
  provision of natural products from
  enzymes and biomasses, which are
  not available commercially,

* use   of   the   scientific-technical
  equipment  and installations at the
  GBF  as   a  service  to  projects
  performed   by   external   research
  groups and industry,

* participation  in  joint  projects,   in
  particular within the framework of
  the biotechnology programme of the
  Federal  Ministry  of Research  and
  Technology (BMFT); and in addition,
  participation  in  the  main areas of
  research of bioengineering together
  with   the   universities   of
  Braunschweig,   Hannover  and
  Gottingen,

* enhanced interdisciplinary training of
  scientists, engineers and technicians
  in the framework of further training
  courses on a  national and  inter-
  national basis, and

* strengthening of  the national and
  European    biotechnology   infra-
  structure  by  the  provision  and
  performance  of  scientific services
  such as informatics  (data bases), or
  the analysis of biological molecules.
THE MAIN AREAS OF RESEARCH
AND PROJECTS OF THE GBF
                              «
   In 1989 the research activities at the
GBF  were  undertaken  under   the
framework of the following  four main
strategic research areas:

1. Biosynthesis and biocatalytics
2. Biomolecules and molecular design
3. Environmental biotechnology
4. Biochemical engineering

   These involve the screening for new
materials  as  well as method-oriented
developments.   A  balance  between
target-oriented  basic  research  and
industrial development work is sought.
   The   projects   were   essentially
performed   in   the   framework  of
interdisciplinary  cooperation   by
numerous departments/researchgroups
at the GBF. The central facilities of the
bio pilot  plant service  unit  and the
central   computer  group   provided
support to these activities.   Project
participation is clearly presented in the
matrix   below.     The  scientific
organization of  the GBF is detailed in
the organigram at  the  end of this
abstract.

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                                          Scientific Organization of GBF
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                                                              6

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                    THE WORK OF OECD RELEVANT TO
          THE ENVIRONMENTAL IMPLICATIONS OF BIOTECHNOLOGY

                             Peter E.W. Kearns
            Organization for Economic Cooperation and Development,
                 Environment Directorate, Paris, 75017, France
THE OECD IN BRIEF

  The Organization for Economic Co-
operation and Development (OECD) is
an   inter-governmental   organization
which is part of the system of Western
international   institutions  developed
after World War II.
  OECD replaced the Organization for
European Economic Cooperation (OEEC)
which  was  founded  in  1948  to
administer the  Marshall  Plan of the
United States.
  There  are  24-member  countries
which are the'free-market democracies
of North America  (USA  & Canada),
Western Europe (e.g. the EC & EFTA
nations, Switzerland,  etc.)  and  the
Pacific   (Australia,  New  Zealand  &
Japan).    The  Commission  of  the
European Communities also participates
in OECD work, as does Yugoslavia as a
"special status" country.
  It is worth nothing that these account
for only 16% of the world's population
but produce two-thirds of its goods and
services, three-fifths of its exports, and
generate four-fifths of economic aid to
developing countries.           ,
  The  basic aims  of  the OECD, as
described its founding Convention, are:

1) to achieve the highest sustainable
economic growth and employment,

2) to promote  economic and social
welfare throughout the  OECD  area by
co-ordinating the policies of its Member
countries, and

3)  to  stimulate  and  harmonize  its
Members' efforts in favor of developing
countries.

   For those of you with an interest in
history,  you might like to know that
following the signing of the Convention
on  30 September 1960,  OECD .was
established  on 30 September,  1961.
This year is the 30th anniversary.

HOW DOES THE OECD WORK?

   Each   of  the  Member  countries
maintains a  permanent delegation of
OECD which is  headed by an ambas-
sador. The ambassador represents his
country  on  the  Council, the  supreme
body  of the  OECD  which  normally
meets  about once  a  week.   The
Chairman  of  the  Council   is  the
Secretary-General of the OECD.
   Once a year, the Council meets at
Ministerial level under the Chairmanship
of one of the national Ministers.
   The Council, which operates on the
principle  of  consensus,  produces
Decisions, which are legally binding on
Member countries. The Council also
makes  Recommendations which are
more an  expression of political will than
something binding.
   The Council also approves the work
of numerous  specialized  committees,

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 expert  groups  and  working  parties
 which  carry  out  the major  part  of
 OECD's work.
   To give you a flavour of the scope of
 the   work,   a  few  of  the  main
 Committees are:

 Economic Policy,
 Trade,
 Financial Markets,
 Energy  Policy,
 Scientific and  Technological Policy,
 Environment,
 Agriculture,
 and  Management  Committee of the
 Special  Programme on the Control  of
 Chemicals.

   The  common approach of  public
 policy of  the  OECD countries  means
 that  discussions  on  a   given  issue
 usually  reach consensu^. It is then
 possible to  establish  "rules  of the
 game" or codes of behavior, for a given
 topic, to  which the participants can
 bind themselves. The OECD has given
 rise  to  several such agreements, for
 example,   an   agreement  on  the
 movement of  hazardous  wastes over
 borders.

 OECD WORK ON BIOTECHNOLOGY

   OECD work on Biotechnology  really
 began in 1982 with the recognition, by
 the OECD Committee for Science and
 Technology Policy, that Biotechnology
 was  an major  emerging  field  from
 which various policy issues would arise
 with  wide  scientific  and economic
 impacts.
  Consequently, a group of  national
experts  was  convened  to consider
future OECD work  in this  area.   Their
 report on "Biotechnology: International
 Trends and Perspectives (OECD, 1982)"
 is still, in many ways, a forward-looking
 study of many of the issues associated
 with  use of biotechnology.
   The authors recognized the need for
 a unique  multidisciplinary  approach
 integrating   the   development   and
 implementation of policies and technical
 instruments in the areas of science and
 technology  research,  industry,  agri-
 culture,  education, environment,, and
 health and worker safety.
   As a  follow-up to this  activity, a
 study on "Safety  ,and Regulations  in
 Biotechnology" was undertaken.  That
 study led to a Recommendation of the
 OECD  Council   concerning  Safety
 Considerations  for  Applications  of
 Recombinant  DNA   Organisms    in
 Industry,   Agriculture   and  ,the
 Environment which called for further
 research to  improve  the  prediction,
 evaluation and monitoring the outcome
 of application of, recombinant, DNA
 organisms.  This  work also  led to  the
 publication   of  "Recornbinant DNA
 Safety Considerations (OECD, 1986)".
   A new programme of work, began in
 1988, is being carried out by the OECD
 Directorate for Science, Technology and
 Industry (DSTI) in co-oper.ation with the
 Environment  Directorate,  under  the
 supervision  of a  group  of  National
 Experts on Safety in Biotechnology.
   The  Group  of  National  Experts
 decided that part  of the programme
 should be the development  of  general ,
 principles  which   would  identify   a
 generic approach and guidelines for the
design of small-scale field research with
genetically modified plants and micro-
organisms.  As this work evolved,  the,
Group of National Experts recognized ,
                                     8

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the need for developing criteria and test
methods for the environmental monitpr-
ing of genetically modified organisms in
"Good  Developmental  Practices  for
Small   Scale  Field   Research  with
Genetically Modified Plants and Micro-
organisms:  A Discussion  Document
(OECD, 1990)".
  The OECD Secretariat was called  on
to analyze monitoring methods and data
requirements with a view of identifying
the  most  promising  approaches  for
conducting   and  evaluating  the
deliberate  release   of  genetically
engineered   organism   to  the
environment.
  As part of  subsequent  efforts  to
prepare a publication, an "International
Survey  on  Biotechnology   Use and
Regulations   (OECD/    Environmental
monograph no. 991,  1990)",  inform-
ation on monitoring programmes for the
environmental  release  of GMOs in the
Member  countries was   requested.
While this information was not included
in detail in the  survey  report, some
information was annexed.
  Additional information  is summarized
in BIOTRACK  which is  OECD's  com-
puterized Pointer System on the Use of
Genetically   Modified   Organisms.
BIOTRACK contains summarized details
of  over  260  releases of genetically
modified   organisms   in   Member
countries.
  As a further  step on the OECD  work
on  monitoring  releases  of genetically
modified organisms , a workshop was
held in Copenhagen the first week of
December, 1990.  This workshop was
an  opportunity  for experts  from a
variety  of  member    countries  to
exchange views and experiences. The
report of the workshop is currently in
preparation and will include two  main
parts.  The first, "Principal Findings of
the Workshop", is  comprised of three
sections:    General  Approaches  to
Monitoring;  Plants  -   Monitoring
Approaches and Methods; and Micro-
organisms - Monitoring Approaches and
Methods.     The   second  part  is
suggestions  for   future  work  and
priorities.
   I  have  stressed  our  interest  in
monitoring because that is the aspect
of the work that I feel is  most relevant
to a meeting like this.
   However, there  are  other relevant
biotechnology activities  in the OECD
programme; for  example, there is work
underway to elaborate Good Industrial
Large Scale Practices, and this work is
likely to result in a publication before
much  longer.   There  is  also  work
underway  on   Food   Safety  and
Biotechnology.

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               NATIONAL ENVIRONMENTAL RESEARCH INSTITUTE
              DIVISION OF MARINE ECOLOGY AND MICROBIOLOGY
                             M. Reuss and T. Leser
                          Ministry of the Environment
                    National Environmental Research Institute
                   Divison of Marine Ecology and Microbiology
                       Charlottenlund, DK-2920, Denmark
 PERSPECTIVES AND EXPECTATIONS
 OF BIOTECHNOLOGY

   Since genetic engineering started
 accelerating  in the  mid 1970s  there
 have been great expectations of this
 new  technology.   Both industry and
 agriculture   interests   hoped   that
 biotechnology would soon be able  to
 solve a number of problems.  However,
 we now must acknowledge that the
 speed of the development has not been
 quite what we expected.
   In spite  of this,  several genetically
 engineered microorganisms have been
 developed and are now exploited com-
 mercially.  So far, it has mostly been
 industry that  has  benefitted  from
 biotechnology and the new organisms
 have   been  well-known  strains   of
 microorganisms, which have been used
 in  fermentation  industry  for  many
 years.   The  microbes  have  been
 engineered to produce new or modified
 products  in  fermentation.    Several
 Danish  industries have  been able to
 implement the new technology.

 LEGISLATION

  In   June,    1986,   the    Danish
 parliament, passed the worlds first
comprehensive  act  regulating  genetic
engineering.  The act introduced  tight
regulation from the start, partly on the
 basis of previous experience with  the
 regulation  of  pollutants  and  partly
 because this was a new area involving
 possible risks.  The act will be revised
 as we  acquire more insight into  the
 problems resulting from discharge and
 release  of   genetically  engineered
 microorganisms.
   In connection with the administration
 of the act, the National Environmental
 Research Institute's (NERI)  job  is to
 provide  the  necessary  professional
 expertise  needed  by  the   National
 Environmental  Protection  Agency in
 order to evaluate the risks and decide
 on the rules in this area.
   NERI  has participated  in  a  group
 working with the  realization of the
 OECD concept GILSP (Good  Industrial
 Large-Scale Practice).   The idea  of
 GILSP is that certain organisms may be
 categorized as low risk organisms and
 used in  ordinary  production  systems
 where  the  discharge of organisms is
 limited as much as possible.  In 1990,
 two  directives were  introduced  on
 genetically  modified organisms which
 are now being incorporated in Danish
 legislation.  So far, only contained use
 of   genetically   engineered   micro-
 organisms  has  been   approved  in
 Denmark. Approval has been given for
the use of  the organisms  in ferment-
ation plants' production  and restrictions
have been  imposed  on  the number of
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 living cells in the effluents.
 into nature.
   The organisms used so far have been
 strains that do not  survive  in  the
 environment.    They   have  a  very
 reduced frequency  of gene  transfer
 compared to the parental strains and
 they do not produce substances that
 constitute any environmental risks.
   In the future however, an increasing
 number  of  genetically  engineered
 microorganisms will  be deliberately
 released into the environment.  These
 organisms will be designed to carry out
 various tasks in the  environment, e.g.
 biodegradation of  recalcitrant organic
 pollutants or  biological  pest  control.
 These  organisms  will   have a  much
 larger  potential of  survival than the
 organisms   used   for  industrial
 production.  They will be able to persist
 in the  environment at least while they
 perform  the function for which  they
 were designed.  Because of their new
 traits some will interfere with ecological
 processes such as  mineralization and
 nutrient  cycling.    Viewed   in  an
 ecological  perspective  this  type of
 genetically engineered microorganisms
 poses  a  risk of unintended  ecological
 impact on the environment.  We have
 no experience in assessing this kind of
 risk  as  the  release  of  genetically
 modified organisms is a completely new
 activity with no previous examples that
 can help in identifying  the potential
 dangers.
  The  research at  NERI  has  been
focused on the ecology of microbes and
their influence on ecological processes.
 By   experimenting    in   contained
ecological   models   (microcosms)
researchers have tried to elucidate what
happens  to  microorganisms  released
 DETECTION   OF   GENETICALLY
 ENGINEERED MICROORGANISMS

   To  be able to study  the fate  of
 genetically modified microorganisms in
 the environment or in experiments in
 the   laboratory  sensitive   detection
 techniques are needed.
   The  classical   microbiological
 methods are all based on identification
 and enumeration of the organisms on
 selective media. By composing media
 with a very special content of essential
 nutrients it is possible to select the type
 of organisms growing on the  media.
   Very specialized media have been
 made   for   detection   of   specific
 microorganisms  as  well  as  general
 media  for  the  enumeration  of total
 number of  bacteria  in  environmental
 samples.
   Genetically   engineered   micro-
 organisms   have  very  often  been
 endowed with antibiotic resistance that
 can  be used for detection.   These
 organisms   can   be   grown   in  the
 presence   of   concentrations   of
 antibiotics that inhibit the indigenous
 microflora and when samples are plated
 on  media  containing  one  or more
 antibiotics only resistant organisms, i.e.
 the genetically engineered strains, form
 colonies.
   Other  traits   such   as   special
temperature requirements or the ability
to degrade toxic organic  compounds
 may be useful for detection.
   Plating on selective media is still the
 most   frequently  used   method  of
detection  and   the  one   that  new
techniques are compared with.
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   However,   selective  plating   has
several drawbacks. The plating and the
colony   counting  are  very   labor-
intensive. Many microorganisms grow
very slowly on the plates, and often the
results are only obtained after several
days.
   The  most   serious  drawback  of
selective plating is, however, that not
all bacteria from environmental samples
are  able  to  form  colonies. Bacteria
which are stressed, e.g. when released
into waste water or into the environ-
ment, are able to change into a dormant
state.  Bacteria in  this state (viable-but-
nonculturable)  are  not dead as  they
may be  brought  back to  a culturable
form  under  certain  circumstances.
They are however, unable to grow on
the   selective  media  they  would
otherwise grow on. The phenomenon
has first been described for pathogenic
bacterial strains that are stressed and
enter the viable-but-nonculturable state
when  they  are   released  into  the
environment, but  it seems now to be a
very general bacterial characteristic.
   It  is  possible  to   reinduce  the
culturable state for pathogenics again
through injection into a host-organism,
but the mechanisms governing this are
unknown.
   When   organisms  are   in   a
nonculturable state they may be present
in  the environment but undetectable
when using selective plating. Because
of the general problems concerning the
selective plating method and because
the development of biotechnology has
made specific detection of genetically
engineered bacterial strains necessary,
research has been directed towards the
development of alternative detection
methods. What these methods all have
in common  is that they  are  direct,
meaning they do not imply cultivation
of  the organism in the  laboratory.
Direct detection methods are based on
immunological and molecular biological
principles.  At NERI selective plating as
well  as direct  methods  have  been
implemented  in   the   detection  of
microbes.
   Previously,  an ELISA   (Enzyme  -
Linked Immunosorbant Assay) has been
implemented for the detection of bakers
yeast  producing  insulin in industrial
waste water.   This  method has been
used in monitoring the survival of yeast
in the field.
   For  the   validation  of  terrestrial
microcosms (see  below) a method for
detection  of Enterobacter cloacae by
immunofluorescence   has   been
developed.  In this  analysis, specific
polyclonal antibodies react with cells of
E. cloacae. After treatment with fluoro-
chromconjugated secondary antibodies
cells can be  counted  with the aid of an
epifluorescence microscope only.
   Detection of microorganisms based
on  molecular  biological methods has
focused on DNA hybridization. By  the
use of a specific DNA probe for a 2,4-D
degrading Alcaligenes eutrophus strain,
it  has  been  possible  to  study  the
survival  of  the  bacteria  in  soil
microcosm and water samples.
   It is a  necessary step for  several
molecular biological approaches to  the
study of microbial communities and for
the  detection  of  specific  micro-
organisms  in  nature to  be able  to
extract purified  DNA  from  environ-
mental samples.
   Extraction and purification of DNA
from environmental samples is not an
easy task. Lysis has  to be  effective on
                                     12

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all bacteria present in the sample and
once  extracted,  the  DNA  is  con-
taminated   by   various   organic
substances.  At NERI a great deal of
work  has  been put  into, the  imple-
mentation  and the improvement of
methods for recovering  high quality
DNA  from  complex  environmental
samples.

MICROCOSMS

  It   is  internationally   agreed   that
microcosms   are  an   essential
experimental tool  for   research  in
microbial  ecology  and  for  the  risk
assessment of  genetically engineered
microorganisms.
  At the microbiological department of
NERI aquatic and terrestrial microcosm
(ecological   models)   have   been
constructed.  The models have  been
designed, to contain parts of natural
ecosystems  in  such  a   way   that
physical,   chemical  and   biological
conditions  are kept  as  natural  as
possible.  .
 . The philosophy  behind the structure
of the models is that because micro-
organisms in nature are controlled by a
complex set of determinants, complex
ecosystem  models are  essential  to
studies  o-f  the ecology of genetically
engineered  microorganisms  and  their
possible ecological effects.
  Only  little is  known of the factors
controlling  microbes.  Recreating the
natural ecosystems in the laboratory as
far as is possible is the best way to
ensure  that all decisive  factors are
included in the experiment.
  But  experiments  in   microcosms
cannot  stand  alone.    Experiments
designed to uncover causalities  on  a
more detailed level are needed.
   The context for the  design  of the
microcosms has been to create an as
natural as possible model system in the
laboratory and  it has therefore, been
important to investigate how well the
models  simulate nature.    Extensive
validations of  the models  have been
carried out.
   The aquatic models have been used
to   simulate   the  eutrophic   Lake
Bagsvaerd  and  validation  has been
made   by  comparing   important
ecological variables  measured  in the
models to similar measurements in the
field. Microcosms have been run for up
to 4 months and an  array of variables
have been measured:   total  bacterial
numbers determined by microscope and
by colony forming units  on  plates; the
numbers of fluorescent Pseudomonads;
changes   of   bacterial   community
structure; bacterial production; primary
production;  algal  biomass  and  the
concentrations of inorganic nutrients.
   The overall  conclusion is that with
the  actual microcosm  design  it is
possible to simulate important features
of aquatic ecosystems.
   At the bacterial level there is  a very
high concordance between measure-
ments made in  the models  and  in the
lake.  This is important as the models
are intended to be used  for studies of
bacterial ecology.
   The  microcosm  validations also
reveal that the model is able to function
satisfactorily through long experimental
periods  (months) and that  replication
between uniformly treated microcosms
is  very  high.   Therefore,  it will  be
possible to study microbial populations
throughout many  generations "in  the
models  and to  discover  long  term
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 ecological effects  of the organisms'
 presence   and   activities   in   the
 environment.
   The terrestrial microcosms have been
 validated  by  conducting the  s"ame
 experiment  in the  field  and  in  the
 microcosms. A non-genetically engine-
 ered strain of Enterobacter cloacae was
 sprayed on  barley  and  bean plants
 growing in the field and in  microcosms.
   Survival of the bacteria was followed
 for 7 weeks on the leaves of the plants
 and in  the soil.   E.  cloacae survived
 equally well in the two systems and at
 the end of the experiments the numbers
 of organisms present in the field and in
 the models was the same.
   In the field experiment the numbers
 of E. c/oacaethat could be detected by
 selective plating decreased dramatically
 immediately  after  spraying, while  the
 numbers  detected  by  immuno-
 fluorescence were unchanged.   The
 field release took place during a spell of
 drought and  the discrepancy between
 the two detection methods may be
 explained  by  stress  suffered  by  the
 bacteria that were sprayed into the dry
 environment.   This  stress may have
 induced a non-culturable state. After a
 couple of weeks the number of cultur-
 able cells increased and at the end of
 the experiment were indistinguishable
 from the numbers determined by  the
 direct method. In the microcosms none
 of this was seen, probably because the
 models  did not simulate the dry period
 and the rain.  The conclusion about the
 behavior of the terrestrial microcosms is
 that they are able to simulate ecological
 conditions  that are of importance to
 bacterial survival and activity, but that
extreme climatic conditions cannot-be
simulated.
 CONCLUSION

   .Through the extensive validation of
 both types of microcosms we have
 obtained important knowledge of some
 of the factors determining the extent to
 which  ecological  models   resemble
 natural ecosystems.
   Experiments   with   genetically
 engineered microorganisms which will
 be  carried out in the microcosms will
 benefit from the thorough evaluations
 of  ecological  parameters, by making
 extrapolations to  nature possible from
 results   obtained,  in  the   models.
 Extrapolations  will   be  meaningful
 because  the model behavior and the
 model limitations are well known.
   In the near future the models will be
 used in  studies of the fate and the
 possible ecological effects of genetically
 engineered  microorganisms   in  the
 environment. This work will be done in
 collaboration   with   international
 scientific  groups  which work on the
 same topics.
  The molecular biological approaches
 to detection will be an important and
 integrated part of the  future  work and
 one of the techniques that are going to
 be implemented is the polymerase chain
 reaction (PCR). By PCR it is possible to
 improve  the detection limit  for DNA
 hybridization to 1-10 cells per gram of
soil  or millilitre of water.
  Extensive research will be put into
the  study of possible ecological effects
of  the   introduction   of  genetically
engineered microorganisms  into  the
environment.
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     DEVELOPMENT OF REGULATORY AND ENVIRONMENTAL OVERSIGHT
                  FOR BIOTECHNOLOGY IN CANADA UNDER
             THE CANADIAN ENVIRONMENTAL PROTECTION ACT
                       (PRESENTATION TRANSCRIPT)

                                T.C. Mclntyre
             Biotechnology Section, Commercial Chemicals Branch
                  Environment Canada, Hull Quebec K1A OH3
INTRODUCTION

  Environment Canada is currently in the
process of finalizing draft regulations for
biotechnology   products   under  the
Canadian Environmental Protection Act.
This Act, passed in 1988, provides for a
comprehensive  regulatory  scheme to
control toxic substances at each stage in
their life cycles: from development and
manu-facture;   through   transport,
distribution,, use  and storage; to their
ultimate safe disposal  as wastes.  The
purpose of my presentation this morning
is threefold:

i)   Provide an update on the extent of
    organizations currently carrying out
    biotechnology  R&D  activities  in
    Canada,  (Figure 1);

ii)   Share the proposed methodological
    approach  for  the regulation  of
    biotechnology   products   by
    Environment   Canada  under  the
    Canadian Environmental Protection
    Act; and finally

iii)   Highlight ongoing in- house research
    and development activities in support
    of the regulatory oversight initiatives
    of Environment Canada.
  In   discussing   the   environmental
components of the CEPA biotechnology
regulations  this morning, I  will briefly
examine  three  forces   that  greatly
influenced the structure and methodo-
logical approach proposed in our draft
regulations.

1)   CEPA  AND  THE  LEGISLATIVE
FRAMEWORK FOR  ENVIRONMENTAL
PROTECTION

  Our  proposed  biotechnology   reg-
ulations  are  based  on  a number of
elements  underlying  the   legislative
framework for CEPA that features:
 CEPA AND THE LEGISLATIVE FRAMEWORK
                FOR
     ENVIRONMENTAL PROTECTION

  * Strong prevention focus

  * Life cycle approach to management of
   toxic substances (cradle to grave)

  * Shift in onus of proof to manufacturer/
   importer

  * Integration of environmental and health
   reporting

  * An ecosystem approach to environmental
   protection
                                     15

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      N = 420
      FIGURES IN PARENTHESIS ARE FOR 1988

      Source ISTC
CANADA
Figure 1.    Geographical distribution of organizations carrying out biotechnology
           R&D activities in Canada 1990.
                                    16

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       CEPA AND THE LEGISLATIVE
            FRAMEWORK FOR
   ENVIRONMENTAL PROTECTION (CONT.)

  *  National Inspection plan and enforcement
    policy based in criminal law

  *  Extensive public role in the administration
    of the act

  *  Innovative mechanisms for federal-
    provincial partnerships

  *  Enforceable standards for federal lands,
    works and initiatives

  *  Environment economy linkages and
    environmental quality objectives
2)  Our draft  regulations acknowledge
criteria developed at the national level as
essential  components of  a regulatory
framework for  biotechnology.   Some of
these include:
   CRITERIA CONSIDERED AS ESSENTIAL
    COMPONENTS OF A REGULATORY
    FRAMEWORK FOR BIOTECHNOLOGY

  * Engenders public confidence

  * Makes economic sense

  * Transparent to all affected stakeholders

  *  Facilitates industrial planning for
    development

  *  Compatible with international approaches

  *  Flexible and accommodating of new
    approaches

  *  Clarifies jurisdictional responsibilities

  *  Draws upon independent scientific advice
3) Our regulations acknowledge certain
working principles that have evolved in
the  international  community  for  the
regulation of biotechnology products that
include:
     FEDERAL GOVERNMENT WORKING
   PRINCIPLES FOR THE REGULATION OF
  BIOTECHNOLOGY PRODUCTS IN CANADA

  * Use of a standard definition of
    biotechnology

  * Buijd upon existing legislation where ever
    possible

  * Regulate product as opposed to process

  * Assess biotechnology products on a case
    by case basis

  * Build upon internationally developed
    guidelines and harmonization whenever
    possible at both federal and international
    levels
PART II Methodological Approach

  General   provisions   of   the
biotechnology   regulations  under  the
Canadian Environmental  Protection  Act
include:

* Products  assessed  under  the  pro-
  posed  Regulations  include  micro-
  organisms to  be used  in  a  variety of
  applications such as, but not limited to,
  bioremediation,   mineral   leaching,
  degradation   of  chemicals,  mining,
  waste   treatment,   waste  disposal,
  chemical  production,  lignin degrad-
  ation, microbial enhanced oil recovery,
  biosensors, and energy production.
                                         17

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 *   An interactive and staged approach
    to notification  and  provision  of
    information is used, based on the
    stages  of  development  of  the
    product   towards   commercial
    exploitation.  The corresponding
    information   requirements   and
    assessment periods build on those
    of previous stages of  notification.

 *   Notification is   required  prior  to
    manufacture or  importation  for
    contained uses beyond the scale  of
    laboratory research.

 *   Information will be assessed within
    prescribed assessment periods.

 * '  CEPA contains explicit provisions
    for protecting confidential business
    information.

 *   The  waiver provisions  of  CEPA
    provide  flexibility for  dealing on a
    case-by-case basis with specific
    products  where  the   progression
    through the stages  is  either not
    appropriate  or  necessary, or for
    waiving information  requirements
    that may not be applicable for the
    product.

 *   A mechanism is proposed for listing
    specific  uses of microorganisms for
    which  notification would not  be
    required (Schedule XVI).

Categories of Notification

  Five stages   of  development  are
defined for notification.

  For  contained use,  notification  is
required:
 * prior  to  research  and development
  beyond laboratory scale (Schedule XI)

 * prior  to  commercial manufacture
  (Schedule XII)

  For   environmental   introduction,
 notification is required:

 * prior   to   small-scale  field  trials
  (Schedule XIII)

 * prior  to  large  scale  field  trials
  (Schedule XIV).

 * prior   to   commercial   production
  (Schedule XV)

Information Requirements

  Each   of  the   five  information
schedules   specify  the   application
information  requirements.  Categories
of-information to be submitted include:

* identification  and characterization of
  the microorganism(s)

* data   on  environmental., fate  and
  effects on  nontarget  substances in
  the environment

* human health safety testing data

* description   of   intended   uses,
  manufacturing methods and quality
  control   and   quality   assurance
  procedures

* specifics  on  field  trials  including
  location and procedures to be used
  during the trial, where applicable
                                      1.8

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PART III Biotechnology Research

  In   development   of  the   CEPA
biotechnology   regulations,   we
recognized that a  properly drafted and
administered regulatory framework can
be a catalyst and not an impediment to
technology development.
  We  recognize the  need  for flexibility
in addressing the needs of the dynamic
and expanding biotechnology industry
in  Canada.    The  flexibility  we are
building into the regulations is linked to
and   dependent  upon  information
concerning both human health and the
environment.  Environment  Canada  is
funding and continues to fund  basic
research   in    microbial   ecology
addressing such issues as:

*  mechanisms for gene exchange  in
   the environment '

*  development of methods for detec-
   tion, monitoring and determination
   of  environmental  fate  -of
   microorganisms,         •   '
                                i
*  microbial  ecological  profiles  on
   specific   genus  of   organisms
   targeted for environmental release,

*  environmental codes of good  prac-
   tice for contained research and de-
   velopment involving biotechnology

*  characterization  and quantification
   of  emission   streams    from
 •  bioprocessing facilities, and

*  • identification of factors/character-
   istics    that   affect   survival,
   persistence, and dissemination of
   microorganisms in the environment.
  The data generated by our research
programs will not only assist us in our
regulatory program but will be a source
of valuable scientific information on the
behavior of  microorganisms  in  the
environment  for  industry,  academia,
and other government departments.
  We  constantly  seek  to  promote
national and  international cooperation
and  information   exchange  in  bio-
technology through our involvement in
a number of interdepartmental commit-
tees under the National  Biotechnology
Strategy, the OCED, federal provincial
regulatory  consultations,  and  more
recently,  under   a memorandum  of
understanding (MOD) we have with the
Environmental Protection  Agency  in
United  States.
  We believe that we are establishing a
sound   and  responsive  health and
environmental oversight structure for
biotechnology in  Canada under  the
Canadian Environmental Protection Act.

Conclusion

  The design of a regulatory oversight
mechanism for any activity, particularly
one  as novel .and  exciting  as bio-
technology will not be  an easy task.
We  are  however,   guided  in  our
endeavours by previous experiences in
the  regulatory    arena  that  have
demonstrated  unequivically  ,  that  a
consensus building process involving all
stakeholders  can be  a  powerful and
effective tool for  the establishment of
an appropriate regulatory framework.
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  USDA'S BIOTECHNOLOGY RISK ASSESSMENT RESEARCH GRANTS PROGRAM

                                D.R. MacKenzie
                 National Biological Impact Assessment Program
                       Cooperative State Research Service
             U.S. Department of Agriculture, Washington, D.C. 20250
   Section  1668 of  the  1990 Food
Security Act (a.k.a., Farm  Bill) directs
the Secretary of Agriculture to establish
a grants program in biotechnology risk
assessment research.  The program is
to be funded as a 1 % set-aside of the
Department's  biotechnology research
expenditures.     This  Section  also
specifies necessary coordination  with
EPA and consultation with Department
agencies in establishing the direction
and activities of the grants  program.
  The responsibilities for administering
the grants program have been assigned
to  the  National  Biological   Impact
Assessment Program.  At the time of
this writing, discussions are underway
on the dimensions of the program, and
the  mechanisms  for  funding.    A
meeting with representatives from the
appropriate   federal   agencies  and
interested  public groups  provided a
starting point for discussions on  how
the program could best meet the spirit
of Section 1668.
  One of the obvious intentions of the
program is to meet the needs of federal
regulatory   agencies  for   sufficient
information to make informed, science-
based decisions on  regulatory permits
for   field  testing   with   genetically
modified organisms.  It has thus been
proposed that an advisory  committee
solicit   "wish   lists"   each  year  of
researchable questions that could be
incorporated into the annual request for
 proposals.  The received  grant  pro-
 posals  would then  be "flagged"  for
 interest by the regulatory and research
 agencies of the USDA.  Subsequently,
 a panel of independent peer scientists
 would rank the proposals for scientific
 merit. These rankings would then be
 reviewed by the regulatory and research
 agency representatives for interest and
 appropriateness.    The scientifically
 meritorious,  high-priority   proposals
 would  then   be  recommended   for
 funding.
   Through these mechanisms,  a new
 set of questions could be asked each
 year, but  the duration  of the research
 undertaken to answer those questions
 would likely extend for several years to
 obtain the necessary  answers.  For
 instance,  a research proposal  might
 look  at  seed  dormancy  factors  of
 genetically engineered canola. It might
 be necessary to study the seed  buried
 in different types of soil over a  period
of years.  This  reality would not limit
the program from asking new questions
each year, and then funding them for a
period  of  time appropriate to  the
question.
  Another recommendation from the
first   meeting  was  that  an   annual
conference should be  held with  the
scientists  receiving  funding from the
program.  This conference would be a
forum for the  exchange of information,
and for annually reporting results to all
                                     20

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interested  parties.     It  has   been
proposed that this annual meeting of
the   USDA   Biotechnology   Risk
Assessment Research Grants recipients
be linked to the EPA's Biotechnology
Risk  Assessment  Research  annual
meeting, for  mutual  benefit.   This
proposal is  now  under discussion
between the two agencies.
  What has not been decided as of this
writing   is   the  definition   of   bio-
technology,  which  will  be  critically
important for determining the amount
of funds available to support the grants
program in risk assessment research.
A   very broad  definition   of   bio-
technology for the Department of
Agriculture would circumscribe about
$ 140,000,000, yielding at 1 %  $1.4
million for risk assessment research. A
very narrow definition of biotechnology
research being conducted in the field by
the U.S.  Department  of  Agriculture
would  ,yield   .$60,000  -  a  rather
insignificant sum.
  Whatever the final outcome of the
mechanisms used to select and,support
biotechnology   risk   assessment
research, there is a clear and evident
commitment  by 'the  Department  of
Agriculture to launch this program as a
responsible complement to its  ongoing
investment in biotechnology research.
The solicitation for proposals should be
issued early summer 199.1  for funding
in  early  Fiscal  Year  1992  (begins
October 1,  1991).
                                     21

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                 THE EPA PERSPECTIVE ON BIOTECHNOLOGY
                              Elizabeth Milewski
                   Office of Pesticides and Toxic Substances
         U.S. Environmental Protection Agency, Washington, D.C. 20460
   For centuries, people have used living
organisms to produce useful substances
such as bread, cheese, beer, and wine,
and  more  recently to perform useful
functions such as the control of harmful
insects.  Initially, use of the organisms
was  empirical;  however, over the
centuries  people   have  developed
techniques to manipulate the genetic
materials of useful organisms  to select
and  enhance  desired  characteristics.
Recently, powerful new techniques  in
molecular biology have been developed
that permit the manipulation of the
genetic materials of organisms in ways
that heretofore were not possible, such
as  moving  genetic material  between
organisms  that  would  not  normally
exchange  such   material.     Public
awareness  and  concern that use of
these  new  technologies may be as-
sociated  with  dramatic  consequences
have resulted in a call for some type of
oversight to ensure adequate protection
of human health and the environment.

FEDERAL OVERSIGHT

  The  United  States  Environmental
Protection  Agency (EPA) is  one of
several United States agencies involved
in the  regulation  of  biotechnology
products as" part   of  a  coordinated
federal effort.  During the past several
years,  EPA  has  been developing the
specifics of its regulatory approach to
these products.  In order to put EPA's
approach into perspective, it is helpful
to  have an overview  of the federal
concept encompassed  within  a co-
ordinated  federal  effort.   In  1984,
.recognizing   its   responsibilities  to
address issues raised  by the use of
biotechnology,   the  United   States
government formed  an  interagency
working group under the White House
Cabinet Council on Natural Resources
and the  Environment.    This  group
examined  existing laws and concluded
that for the most part,  these laws
would  adequately  address regulatory
needs for  biotechnology.  A regulatory
matrix  describing applicable laws and
responsible agencies was published in
the Federal Register on November 14,
1985 (50  Fed. Reg. 47174).
   Subsequently,   the   "Coordinated
Framework   for    Regulation   of
Biotechnology" was published on June
26, 1986  (51  Fed.  Reg.  23302). As
used in the Coordinated Framework, the
term biotechnology is broadly defined
as  "the  application  of  biological
systems and organisms to technical and
industrial processes" (see "Proposal for
a   Coordinated    Framework   for
Regulation of Biotechnology," 49 Fed.
Reg. 50856, December31, 1984). The
Coordinated  Framework spells out the
basic federal philosophy for regulating
products of biotechnology, details the
network of  agency jurisdiction  over
both research and commercial products,
and includes statements of regulatory
policy  from  the agencies principally
responsible for such regulation.   The
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 United  States  approach to regulating
 biotechnology   is   based  on   the
 recognition   that  biotechnology
 encompasses  a   large  and  varied
 collection of techniques and activities,
 and that the potential products resulting
 from this technology will cover a wide
 spectrum of uses.  The Coordinated
 Framework provides that biotechnology
 products will be regulated in the United
 States  as  are  products  of  other
 technologies -- that is,  by  the various
 regulatory agencies on the basis of use.
 Thus,  many  agricultural  uses  of
 microorganisms, plants,  and animals are
 regulated  by   the  Department  of
 Agriculture;  foods  and  drugs  are
 regulated  by  the  Food  and   Drug
 Administration; microorganisms used as
 pesticides are regulated by EPA under
 the Federal Insecticide,  Fungicide, and
 Rodenticide Act (FIFRA), 7U.S.C. §136
 et  seq.; commercial uses of  micro-
 organisms not covered by other existing
 authorities are regulated by EPA under
 the  Toxic   Substances  Control   Act
 (TSCA),  15  U.S.C.  §2601  et  seq.
 Examples of uses  covered by TSCA
 include  microorganisms used in metal
 mining,  degrading wastes,  conversion
 of biomass  for energy,  production of
 proteins   and   enzymes  for   non-
 pharmaceutical  purposes,  and   non-
 pesticidal agricultural applications such
 as nitrogen fixation.

 EPA REGULATION

  As  indicated above,  an essential
 characteristic of any regulatory scheme
 for  biotechnology is that is promotes
 public confidence that  the oversight
 process will protect human  health and
the   environment.     Similarly,   the
 scientific and  regulated  communities
 must  believe  that  the  process   is
 credible, equitable, and sound.  At the
 same  time,  it is  important that the
 development and commercialization of
 biotechnology products be allowed to
 proceed in  a  reasonable  and timely
 manner.  Accordingly, EPA's goals in
 developing  a regulatory  scheme for
 products  of  biotechnology are to (1)
 protect   human   health   and   the
 environment, (2) meet the public's need
 to  be  assured  that use  of  living
 products does not present unacceptable
 risk,   (3)  expedite   the   regulatory
 process, (4) maintain flexibility in order
 to  respond   to   rapidly  developing
 knowledge and technology, and  (5) to
 accomplish  this end  without stifling
 innovation and  the  development  of
 valuable hew products.
   At this time, EPA's regulatory effort
 involving  these  products is, in  large
 part,  pursuant  to  its  authority under
 FIFRA and TSCA.   FIFRA  creates  a
 statutory framework under which EPA,
 through   a   registration  process,
 regulates   the  development,   sale,
 distribution,  and  use of  pesticides,
 regardless of how these pesticides are
 made or their mode of action.  FIFRA,
 therefore, covers natural and genetically
 altered  microbes that  are  used  for
 pesticide purposes.  EPA has routinely
 reviewed   and   registered   natural
 microbial pesticides, such as Bacillus
 thuringiensis, for years.
   A pesticide can be registered for use
only if  the  pesticide  will  not  cause
 unreasonable   adverse   effects   to
humans  or  the environment.    The
 "unreasonable adverse  effects"  test
involves a weighing of the  risks and
benefits of use of  the  pesticide.  In
                                     23

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order to demonstrate that a  pesticide
will  not cause unreasonable adverse
effects, an applicant seeking to register
the product must submit or cite data on
subjects such as product composition,
toxicity, environmental fate, and effects
on nontarget organisms.
  Some of the information needed to
support registration must be developed
under actual field conditions, and such
preregistration field testing is authorized
under FIFRA through the experimental
use  permit process.   The results of
such early testing are a critical element'
in determining when  and under what
circumstances a full registration may be
issued.
  TSCA gives EPA jurisdiction over the
manufacturing,processing, importation,
distribution,  use,  and  disposal of all
chemicals in commerce (or intended for
entry into commerce)  that  are not
specifically covered by other regulatory
authorities (e.g., substances other than
foods,    drugs,   cosmetics,   and
pesticides).   TSCA  authorized  EPA,
through  a  "screening"  process,  to
acquire    information   on  chemical
substances and mixtures of chemical
substances in order  to  identify and
regulate  potential   hazards   and
substantial exposures.  Under TSCA,
EPA can require testing of any chemical
substance that   may   present  an
unreasonable risk to human health or
the environment or which'is produced
in substantial quantities and may result
in substantial environmental release or
substantial human exposure.
  TSCA's applicability to the regulation
of microbial biotechnology products  is
based on the interpretation that  living
organisms are  chemical substances
under TSCA  - an interpretation that
was embodied  in the development of
the initial chemical inventory in 1979.
The basis for this interpretation is that
all  substances,  living  and nonliving,
have a chemical foundation at the most
fundamental  molecular  level.   As  a
result  of  this   interpretation,
microorganisms  (except for those in
excluded use categories) are subject to
all provisions of TSCA.
   The premanufacturing notice (PMN)
process implements  TSCA's  goal of
screening new substances before they
enter commerce  in order  to  identify
those  that  pose  potential  hazards.
Manufacturers and importers of "new"
chemical substances  are  required to
submit data and other information to
EPA that  will  allow  the  Agency to
evaluate  the  potential  risks  of  the
product.  If a potentially unreasonable
risk  is identified  during  the  PMN
screening process, EPA is authorized to
[exert] comprehensive oversight of the
commercialization  of  the  affected
product.
   Because microorganisms introduced
into the environment have the ability to
replicate,  increase  in number,  and
disseminate  from the  test site,  EPA
feels  that  it is prudent  to  address
certain questions before some of these
microorganisms  are  released  to  the
environment.    Accordingly,   EPA's
regulatory   approach   encompasses
procedures   designed  to  provide
sufficient oversight of the initial stages
of  environmental  testing   of  certain
microbial products of biotechnology. In
its   section   of  the   Coordinated
Framework, EPA indicated that, under
FIFRA and TSCA, ^intended to focus
regulatory emphasis on three categories
of microorganisms:
                                     24

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 1.     microorganisms   with   "new"
       characteristics or that are new to
       the  environment in  which they
       are  intended  to be  used (and
       whose behavior is therefore less
       predictable),

 2.     microorganisms   that   have
       hazardous  components  or
       characteristics (and  therefore a
       recognized  potential  to  cause
       adverse   effects   in   other
       organisms), and

 3.     microorganisms that are used in
       the  environment (and therefore
       have the potential for widespread
       exposure).

  Through   its   statement  in   the
 Coordinated  Framework,  EPA  made
 certain provisions immediately effective
 and indicated that rulemaking would be
 required before other provisions could
 be  formally  implemented.    Where
 provisions    were  not   immediately
 effective,   EPA   invited   voluntary
 compliance in the interim.
  One  of  the  most  complex  (and
 contentious)   issues   for   the   EPA
 biotechnology rulemaking process has
 been  identification of  the  scope  of
 organisms  to be subject to  regulatory
 oversight.   Because this is a cross-
 cutting  issue  for  all  the  agencies
 included in the Coordinated Framework,
 development of a "scope, definition"
 was undertaken as a joint effort.  The
 result  of this  effort,  "Principles for
Federal Oversight of  Biotechnology:
 Planned   Introduction   Into   the
 Environment   of   Organisms   With
Modified   Hereditary  Traits",   was
published on  July 1,  1990,  and serves
 as guidance  for  EPA  and the  other
 agencies  during   their  rulemaking
 processes.
   In  developing the  Principles  for
 Scope,  the  agencies   relied   upon
 "familiarity"   as   the   basis    for
 determining   whether   oversight   is
 appropriate, and emphasized the role of
 information  in  the  determination of
 "familiarity".  (The appropriateness of
 using   familiarity   based  on   solid
 information as the basis for  a flexible
 regulatory process is clearly supported
 by the  1989 National  Academy of
 Sciences  report,   "Field   Testing
 Genetically    Modified   Organisms".)
 Specifically, the Principles for Scope set
 forth the general directive that "to the
 extent  permitted by  law,   planned
 introductions  into the  environment of
 organisms with  deliberately  modified
 hereditary traits should  not be subject
 to   oversight...   unless  information
 concerning  the  risk  posed  by  the
 introduction indicates that oversight is
 necessary."     Also   included   are
 examples of  organisms  that would
 "generally be  excluded from oversight
 because their introduction is considered
 to be  similar to previous safe intro-
 ductions, or because other information
 available regarding the  risk  posed  by
 such introductions (including knowledge
 that  existing  practices or regulations
 adequately address possible risk posed
 by   the   introduction)  makes  such
 oversight unnecessary."   With   the
 Principles for Scope  as guidance, and
 utilizing provisions available under both
TSCA  and   FIFRA  that  allow   for
flexibility in the regulatory process, EPA
is [well on its way to establishing the
regulatory program  promised  in  the
Coordinated Framework].  The Agency
                                     25

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is  currently  nearing completion of the
drafting process for its biotechnology
regulations  under  both  TSCA  and
FIFRA, and  it is-anticipated that the
proposed  rules will  be published  for
comment in  the near future.

EPA REVIEW PROCESS
AND EXPERIENCE

   Since  1984,   EPA  has  reviewed
several small-scale tests using products
of  biotechnology,   many  of  which
involved release to the environment.  A
scientific review process for such tests
has been developed that EPA  believes is
credible, sound, and respected by the
scientific community and which allows
the  public  to  feel  confident that
potential   human   or   environmental
impacts have been addressed. To date,
the  review  procedures,  which are
briefly described below, have involved
case-by-case evaluation.
   When a submission  for small-scale
testing is received, TSCA or FIFRA staff
groups evaluate  the submission and
develop   a   coordinated   scientific
position.   During  this process,  hazard
and exposure are addressed, potential
problems,   issues,   or  significant
unanswered questions are  identified,
and  the  likelihood  of  significant risk
from the proposed test is assessed.
   Under both TSCA and FIFRA, intro-
agency workgroups then comment on
the positions developed by  staff.   If
appropriate,  the submission and EPA's
scientific position are  sent to  other
federal   agencies   for  comment.
Appropriate  state regulatory agencies
are also contacted in order to  alert them
to  the submission,  to  discuss EPA's
assessment, and  to ensure that the
federal  and  state  positions  are  as
consistent  as  possible.    For some
submissions, visits to the test sites are
conducted  in order to evaluate actual
field conditions.
   In order to obtain  an independent
peer review of EPA's scientific position,
to address  specific scientific questions
raised  by  staff,  or to  identify  any
additional data that may be needed to
complete  the  risk assessment,  the
submission  and the EPA's scientific
evaluation  may be sent to a group of
independent  scientists.    EPA  has
established  a   specific   advisory
committee, the Biotechnology Science
Advisory   Panel   (SAP)   for  these
independent  peer  reviews.    Several
proposed field trails have been reviewed
by specially convened BSAC, SAP, or
joint BSAC/SAP subcommittees,  and
these   groups   have   consistently
endorsed the EPA assessments.
   Public comment is  considered  an
important aspect of the reviews,  and
for  many  proposals  the  public  is
provided   several   opportunities   to
comment during the review process.
   In some  cases, microorganisms that
are subject to TSCA or FIFRA are also
subject to the Federal Plant Pest Act or
other statutes  administered  by  the
Animal  and  Plant  Health  Inspection
Service  (APHIS) of the United  States
Department of  Agriculture.   In such
situations,  APHIS  and EPA  conduct
coordinated reviews. The two agencies
cooperate closely, alerting each other to
submissions,  and sharing expertise and
information.  This  close cooperation
benefits  both  submitters  and  the
agencies.
   At the conclusion of a review, EPA
determines  whether the microorganism
                                     26

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 may be released into the environment.
 Under  both  TSCA  and  FIFRA,  this
 decision is based on an assessment of
 both   the  potential   risks  and   the
 potential benefits of the proposed  use
 of  the microorganism.   Should  the
 analysis  indicate that  use  of   the
 microorganism may pose unreasonable
 risks,  EPA has authority  under either
 statute to impose  restrictions on  its
 use.
   Between the two programs, TSCA
 and   FIFRA,.   EPA   has  reviewed
 approximately 75 submissions,  some of
'which have been "second-generation"
 variations of previously approved tests.
 While  it is still too  early to speak in
 general  terms   or  draw  definitive
 conclusions about the  reviews, certain
 observations can be made:

 1.     EPA's review  process has been
       effective   in  identifying   and
       addressing risk concerns.

 2.     Evaluation of the proposed tests
       often requires different types of
       background studies, data bases,
       and scientific expertise than are
       required for review of traditional
       chemical  products.   Similarly,
       monitoring   for   living
       microorganisms  once they have
       been  released to  the  environ-
       ment   is   frequently   more
       complicated.

 3.     The  issues giving  rise  to the
       greatest concerns are often not
       related   to   whether   the
       microorganism   performs   its
       intended function, but rather to
       other, nonintended impacts that
       might occur.
 4. Many  (if  not  most) of  the  risk
   concerns   about   these   micro-
   organisms have been related to their
   potential impacts on the environment
   and nonhuman, nontarget species; to
   date,  potential impacts  on human
   health  have not been a significant
   concern.

 5. Monitoring data from some  of the
   tests  have  confirmed that  under
   actual   use  conditions,   physical
   containment  of  the test  micro-
   organisms may not  be feasible or
   possible. Accordingly, issues related
   to spread of the test organisms from
   the   test   site  are  appropriately
   considered   in   most   risk
   assessments.

 6. The small-scale tests reviewed thus
   far  have been found to  pose  no
   significant risks.  However, large-
   scale or commercial use of some of
   these   microorganisms may  raise
   more   significant  concerns,
   particularly regarding environmental
   and ecological impacts, than those
   addressed  for small-scale tests.

   As to nonscientific observations, it
 has  become  increasingly  clear that
 public  involvement  is  an   important
 element   of   most   test   programs
 involving  environmental   release.
AA/ithout such involvement, public fear
 of the unknown could derail the whole
 process and prolong the testing through
 extensive administrative and  judicial
 maneuvers.    The impact  of such
 administrative and judicial activity can
 be seen by contrasting the first and a
 more  recent EUP  review  of two
 genetically   engineered   microbial
                                     27

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pesticides.   In  the first,  the  time
elapsed between  receipt  of  the EUP
application and the test was almost two
years.  In between were two lawsuits
brought in federal and state courts to
stop the test, and numerous state and
local  administrative proceedings.  The
more recent EUP application, submitted
after  more extensive preliminary public
outreach  and  presumably  after  the
public had gained more confidence in
EPA's review  process,  moved   from
submission  to   field  testing approx-
imately five months.  There  were no
lawsuits or supplemental administrative
proceedings involved with this review.
It  appears that the more the  public
knows  and  feels  a  part  of  and
comfortable with the process, the less
likely it is  to obstruct the test.  Hence,
many submitters have elected to shield
from  public scrutiny  only the  barest
essentials about their microorganisms
and proposed tests.
  EPA is  using the experience gained
from  its early test reviews, along with
public comments and advice from our
BSAC,  in  fashioning  its  proposed
regulations.  Based on these  sources,
EPA has bolstered confidence that its
basic  approach  as  set  out  in the
Coordinated Frameworkand procedures
developed  for individual  test reviews
are adequate to accomplish the  goals
for   its  biotechnology   regulatory
program.   Therefore, in  many  areas,
EPA's  proposed   regulations   are
envisioned in much the same light as its
policy statement in the  Coordinated
Framework.
  To  summarize,  EPA's  experiences
over  the  last  few years with the
approach  for regulating  products of
biotechnology first articulated in  1984
and   later  included   in  the   1986
Coordinated Framework, demonstrate
that the  process  has met  its  major
goals:

*  utilization of a systematic approach
   for   identifying   and   assessing
   potential  human or  environmental
   risks,

*  incorporation  of   a  peer  review
   mechanism   that   draws   upon
   independent  scientists  expert  in
   diverse areas of relevant knowledge,

*  establishment of an  open process
   that allows  for and  invites  public
   participation, and

*  approval of field tests, the results of
   which   confirm   EPA's   original
   judgment of  insignificant risk.

   Notwithstanding  these successes,
EPA recognizes that certain  modifi-
cations  to the current  process  are
probably  appropriate   in  order  to
accommodate  in a timely manner the
number of tests anticipated for review
in  the  future.    EPA  is  currently
developing proposed  regulations  that
will implement  its modified approach.
However,  based  on  its  past  experi-
ences, EPA is confident that its ultimate
regulatory program  will protect the
public health and the environment from
unreasonable risks while allowing the
public to reap the benefits of scientific
and technological innovations. "-
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       OFFICE OF TOXIC SUBSTANCES PURVIEWS AND RESEARCH NEEDS
                                Philip G. Sayre
       Health and Environmental Review Division, Office of Toxic Substances,
          U.S. Environmental Protection Agency, Washington, D.C. 20460
   The  Office  of  Toxic  Substances
 (OTS) implements the Toxic Substances
 Control Act  (TSCA), which applies to
 commercial  microbial  products.  The
 microbial  products  reviewed  under
 TSCA are those that are specifically
 excluded by other federal statutes, e.g.,
 pesticides  (which are regulated by the
 EPA's  Office  of Pesticide Programs
 (OPP) under  FIFRA), foods, drugs, and
 cosmetics which  fall under the purview
 of the Food  and  Drug Administration,
 and nuclear materials regulated by the
 Nuclear Regulatory Commission.  The
 products reviewed thus far by OTS fall
 into two categories:  (1) fermentation
 applications  for production  of cellular
 components  such as  enzymes to  be
 used in the production of  ethanol,
 detergents,  and  culture media;  (2)
 microbial  products   intended   for
 intentional environmental release such
 as  recombinant rhizobia for enhanced
 nitrogen fixation  in legumes.  Other
 applications which are subject to TSCA
 include  for example those for degrad-
 ation of toxic  wastes, metal mining,
 desulfurization  of coal,  enhanced  oil
 recovery, and biomass conversion (for
 reduction of solid  waste).  Products for
 bioremediation  of  toxic compounds
-.such as organic  solvents, crude and
 refined petroleum wastes, and creosote
 may account for a significant portion of
 future submissions to OTS.
  There are several  differences in the
 research needs of OTS and OPP, which
 naturally  follow   from  the  current
 interpretation of TSCA as it applies to
 microbial products. First, a potentially
 diverse group of microorganisms and
 uses are subject to TSCA. Second, the
 microorganisms  reviewed by OTS are
 not intended to be toxic or pathogenic,
 as are microbial pesticides. Third, OTS
 examines submissions for fermentation
 applications of microorganisms.   OPP
 does not,  although FIFRA provides for
 ensuring   quality  control   in    the
 manufacturing process.   Fourth,  the
 OTS  risk  assessment  process places
 more   emphasis   on   microbial   fate
 analysis,  in addition  to  examining
 human  health and ecological effects
 concerns.  Concerns for microbial fate
 necessitate  research which  leads  to
 better detection of microorganisms, and
 better estimation of microbial dispersal
 as a result of fermentation releases and
 intentional   environmental   releases.
 Although   interest  in  research  on
 microbial   fate   will   continue,   the
 research program is now shifting  to
 place  a stronger  emphasis on human
and ecological effects.
   Currently, OTS is in the process  of
developing tiered testing  schemes for
both  ecological   effects  and  human
health.  In order to implement these
testing schemes, additional  protocols
beyond those developed for  OPP, will
be needed.   OTS has begun a  joint
project with  ORD to  develop simple
screening-level tests to determine the
survival   and   competitiveness   of
genetically engineered microorganisms
                                     29

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in   various   environmental   media.
Further,   since  exposure  to  OTS-
reviewed products often includes areas
beyond the agricultural  environment,
protocols  for effects testing should be
developed for  different species of
organisms that are not only important
ecologically but are commonly found in
the general environment (such as some
ant species).  Protocols may also need
to be developed which can discriminate
between  effects of  a microorganism
and  its metabolites  to  address both
human health and ecological concerns;
such protocols are particularly relevant
to the evaluation of microorganisms
used   for  bioremediation  of  toxic
pollutants or recalcitrant compounds.
Due to the  fact that many microbial
products reviewed under TSCA are not
designed to  be toxic/pathogenic but
may  play  a  role  in  other  critical
interactions (e.g., mineral cycling), OTS
will  continue  its   interests  in   test
systems  such as microcosms which
examine  a broader range  of  effects
concerns. Finally, OTS will continue to
seek support as it develops computer-
ized risk  assessment tools such as
those  for  gene  sequence analysis,
microbial taxonomy, and microbial fate
modelling.
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           THE OFFICE OF PESTICIDE PROGRAMS: REGULATION OF
                MICROBIAL PESTICIDES AND RESEARCH NEEDS
                             William R. Schneider
                         Office of Pesticide Programs
                     U.S. Environmental Protection Agency,
                           Washington, D.C. 20460
  The  Office  of Pesticide  Programs
(OPP) is responsible for  regulating all
pesticides under the Federal Insecticide,
Fungicide, and Rodenticide Act (FIFRA)
and  the  Federal  Food,  Drug,  and
Cosmetic Act (FFDCA).  FIFRA defines
pesticides as any substance, or mixture
of substances, intended for preventing,
destroying, repelling, or mitigating any
pest,  and  specifically includes  plant
regulators,  defoliants, or desiccants.
Before  the  Agency  can  register  a
pesticide, FIFRA requires the Agency to
have sufficient data to determine that
the pesticide, when used in accordance
with   widespread   and   commonly
recognized practice, will not cause (or
significantly  increase  risk  of)
unreasonable   adverse   effects  to
humans of the environment.
  The   microbial  pesticides  include
bacteria, viruses, fungi,  protozoa, and
algae. The first microbial pesticide was
registered in  1948 (Bacillus popilliae]
and 20  others have been registered to
date.  Of those, 8 have been registered
in the last 2 years. Over one third of
the new pesticides registered  recently
have been microbial pesticides. This is
indicative of  the  overall trend  for
industry to turn to biological pesticides
to address the agricultural problems of
the 90's: insect resistance to chemical
pesticide  and  the   need  for  safer
pesticides. These biologically derived
pesticides   include   the   microbial
pesticides, non-toxic biochemicals such
as pheromones, and the newest and
largest group: plants that are genetically
engineered   to   product   pesticidal
chemicals.   Although  our regulatory
system for transgenic plants is not yet
in place, we have reviewed  over 50
small scale  field  tests  through  a
memorandum of understanding with the
U.S. Department of Agriculture Animal
and Plant Health Inspection Service.
   Part 158 of 40 CFR 158  specifies
the data and  information that must be
submitted to  EPA  to  support  regis-
tration of pesticides.  Subdivision M of
the Pesticide Testing Guidelines  (NTIS
# PB89-211676, July,  1989)  provides
information   relating  to   the   data
requirements listed in 40 CFR  158.690
and   158.740,  including  conditions
under which  each data requirement is
applicable,  standards  for  acceptable
testing,  information that  should be
included in a test report, guidance on
evaluation and  reporting of data, and
examples  of  protocols.   In  addition,
scientific   publications   and   ORD
laboratory  reports  are cited in the
guidelines  as useful information  for
designing test protocols. OPP must be
able to evaluate these  protocols since
40 CFR  160.120, the  regulation for
Good Laboratory Practice  Standards,
specifies that "each study shall have an
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approved written protocol....".
   In  addition to registering microbial
pesticides, the Agency must perform
risk   assessments   to   evaluate
applications   for   Experimental   Use
Permits (EUPs) as described in 40 CFR
172.   Microbial pesticides must have
EUPs before being tested on cumulative
total of not more than 10  acres of land
or not more than one surface-acre of
water, or if to be used for food or feed
uses, or on water containing animals or
plants that my be used for food or feed.
In addition, some microbial pesticides
are  subject  to  an  interim  policy,
published  in  the   June  26,   1986
FEDERAL REGISTER (51  FR  23302).
This policy essentially states that the
Agency  must be   notified  prior .to
conducting small scale field testing of
genetically altered  or  non-indigenous
microbial  pesticides in order to allow
the Agency  to determine if an EUP
should be obtained.  Under this policy,
OPP has reviewed a number of interest-
ing constructs including ice enucleating
gene  deletions in Pseudomonas,  and
Bacillus thuringiensis toxin  genes in
Pseudomonas and in Clavibacter xyli.
Indications  are that most  microbial
pesticide  genetic engineering work in
the immediate future will involve  toxin
genes inserted into  baculoviruses.  In
reviewing these notifications, we have
found  that  the majority  of the  risk
issues are the same as for the  wild-
type,   naturally  occurring   microbial
pesticides.
  OPP   uses  a   risk   assessment
framework for microbial pesticides that
was derived  from traditional chemical
assessment methods, i.e. the overall
risk is a function of both exposure and
hazard  (the   effects  on  non-target
 species, including human).  In order to
 minimize the  data requirements, OPP
 has elected to  require environmental
 exposure data  only if  unacceptable
 hazards are  observed  in  non-target
 species or short-term mammalian tests.
   Although  we  have  recommended
 specific protocols  for many  tests in
 Subdivision M, relatively few have been
 performed and some tests  have  been
 difficult to  perform  and   evaluate.
 Accordingly,   OPP  has  identified
 Subdivision M  protocol development
 and evaluation as its primary  research
 need.   Some  problems with the eco-
 logical  effects  testing  are:  (1)  We
 generally test adult honeybees since
 larvae are difficult to keep alive, yet
 larvae are the most likely to  be sus-
 ceptible to biological insecticidal toxins.
 (2) It  has been difficult  to maintain
 Daphnia for a sufficient time to evaluate
 potential pathogenicity.  (3) Very few
 species  of   beneficial  insects   are
 available for testing. (4) Which aquatic
 species are most at  risk  and are we
 adequately protecting them?  (5) Do the
 protocols adequately reflect a maximum
 hazard approach?
   In  addition to the actual  testing
 protocol development, there is a need
 for  information   that  allows  us  to
 evaluate the study results.   In many
 cases, we do  not have much inform-
 ation on  the  role  of pesticidal gene
 products in the environment, even for
 the most prevalent Bacillus thuringiensis
 toxins.     Bacillus  thuringiensis  is
 ubiquitous in soil but not at  levels that
 appear to affect insects.  Microcosm,
 mesocosm, and/or  field studies could
 be utilized to study the relationship of
Bacillus thuringiensis and/or its many
toxins  to the soil and aquatic  eco-
                                     32

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systems.   Are  there  alternative  soil
hosts?   Do  the toxins  give Bacillus
thuringiensis a competitive  advantage
over other soil microorganisms?
  Potential  human health  effects of
pulmonary  exposure   to   microbial
pesticides  are  one of  our  primary
concerns, since bacterial, fungal,  and
viral pesticides are frequently broadcast
over large areas by aerial applications.
These  extensive  uses present  pos-
sibilities   for   human   epidemiology
studies and for  relating experimental
test results with actual effects.
  Health research has centered on the
tier 1 acute pulmonary exposure tests
for   Subdivision  M  of the  Pesticide
Assessment Guidelines.   These tests
were  developed,  standardized   and
evaluated for  the assessment of  the
fnfectivity   primarily  of    naturally-
occurring agents.  In these studies,
intratracheal and intranasal  challenges
were demonstrated to be equivalent to,
if not better than, aerosol inhalation for
the assessment of pulmonary exposure
to infectious bacterial and viral agents.
In addition, results from EPA-sponsored
studies have  shown that  pulmonary
exposure   of   rodents  to  Bacillus
thuringiensis  have  shown  adverse
toxicological effects at sufficiently high
dose levels.   The  extent  of toxicity
observed appears related to differences
in  the Bacillus thuringiensis  strain
administered.  While this is a significant
first step in methodology and protocol
development  for  testing  guidelines,
relevant   new  technologies    and
innovative   methods  must  be  in-
corporated to  increase the breadth of
coverage  of these tests for a  wider
range of agents in the future. This  can
be  attained   by  obtaining,  through
research, a better understanding of the
basis of these empirical tests. Areas of
additional inquiry should  address the
microbial  agent/cellular  interactions,
factors  influencing the reisolation and
identification of  the microbial  pest-
icides, the  utility  of these tests for
repeated  pulmonary  exposures,  the
equivalence of  intratracheal and intra-
nasal challenge results  with  aerosol
inhalation results when toxicity instead
of infectivity is the endpoint,  and the
scientific  underpinnings of new ap-
proaches  so appropriate tests can  be
developed to address problems such as
assessment of the potential for transfer
of deleterious  genes  to  mammalian
tissues  and cells by  more  innovative
and complex genetically altered agents
which can be expected to be developed
for such problems as assessment of the
potential  for transfer  of  deleterious
genes to mammalian tissues and cells
by   more   innovative   and  complex
genetically altered agents which can be
expected to be developed in the future.
Microbial agents of greater anticipated
use should be identified and studied to
better understand any adverse cellular
responses or effects that might be pro-
duced  in  the  pulmonary  tests.   In
particular, Baci/fusthuringiensis,bacu\o-
viruses, and Beauvaria bassianna have
high  pulmonary  exposure  potential.
Additionally, as successful agents are
more utilized and  exposure becomes
more frequent, components of microbial
pesticides of minor relevance to the
human   health   effects    must   be
reassessed.
                                      33

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                                 SESSION I
                ENVIRONMENTAL EXPOSURE RESEARCH
Detection and Enumeration

  Scientists are using serological, biochemical, physiological, and genetic methods for
improving detection and enumeration of microorganisms under laboratory, microcosm,
and field conditions.  Methods are being developed and refined for their sensitivity,
specificity,  and reliability in a variety of terrestrial, aquatic, and air habitats.  The
following  extended  abstracts summarize continuing research in detection and
enumeration of microorganisms of special interest.

Dispersal and Transport

  Developing a better understanding of the movement of microorganisms through the
environment, either by the natural elements, or through associations with  higher
organisms is crucial.  The mechanisms and dynamics of transport among and within
various environmental components (air, soil, groundwater, plants, insects, and other
animals)  are being studied.- This research includes development  of  mathematical
models and other aids for predicting transport and exposure. The following extended
abstracts summarize continuing research in the potential of microorganisms of special
interest to disperse or be transported from the site of release.

Survival and Colonization

  Understanding the  factors that affect the ability of microorganisms to survive and
colonize habitats under a variety of conditions is  central to this research element.
Cellular,  molecular,  and  environmental factors influencing  survival  or  actual
multiplication are being identified and described.

Gene Transfer

  This research provides test methods that describe conditions for determining the
frequency and probability of genetic exchange between microorganisms  released to
the environment and  their indigenous counterparts. Factors that affect and control
gene stability and rates of transfer in the environment are being evaluated.
                                     35

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  MOLECULAR STUDIES OF MICROBIAL ECOSYSTEM PERTURBATIONS
                                  D.A. Stahl
                              University of Illinois
                                Urbana, Illinois
INTRODUCTION
  A central question in environmental
microbiology is: how do the activities
of microbial communities correlate with
the diversity of populations responsible
for  those  activities?    This  question
remains   essentially     unanswered
because of well recognized limitations
of pure culture isolation and existing
determinativeschemes of classification.
Pure culture  isolation and phenotypic
characterization have  not  served  to
adequately   describe   environmental
diversity.  However, the need for pure
culture  isolation   is    no   longer  a
prerequisite.  Techniques of molecular
biology (including  recombinant DNA,
nucleic  acid  hybridization,  molecular
systematics,  comparative sequencing
and  the  polymerase  chain  reaction
(PCR)) now provide additional tools for
characterizing natural  populations.  In
particular, comparative  sequencing  of
the ribosomal RNAs is serving to unify
microbial systematics and has provided
the   foundation   for   the   explicit
characterization of natural   microbial
diversity.
  We  have  developed a  variety  of
techniques  using  comparative  rRNA
sequencing and hybridization for use in
the   characterization   of    microbial
populations  in natural communities.
These  techniques and  a phylogenetic
framework have  served our ongoing
studies  of  microbial     population
ecology;   addressing   questions   of
community  stability,  competitive
exclusion, succession and  synergism,
community response to  perturbation,
and  the   relationship   between
community structure and  community
function.  These questions are central
to  microbial ecology.  From a more
practical  standpoint,  the   rational
manipulation   of   the  activity   of
environmental populations [e.g. via the
introduction of exogenous organisms
(or selected  DNA  encoded traits)]
should be based on an understanding of
population ecology.  Similarly, rational
risk assessment is dependent upon this
understanding.
  Our research with the  EPA has
focused  upon   two  "model"  com-
munities to further develop comparative
molecular methodologies and  establish
basic  paradigms of microbial  ecology.
These communities  are  the bovine
ruminal  microbial community and those
represented  by   marine  anaerobic
sediments.    Studies of the  ruminal
community  have   focused  on  the
principal  fiber-digesting   populations
whereas   sediment   studies   have
examined the diversity and distribution
of  sulfate-reducing   bacteria   in
relationship to   their  activity.  This
abstract  addresses  only the  ruminal
studies.  Studies of the environmental
diversity and  activities  of   sulfate-
reducing bacteria will be addressed by
Dr. R.  Devereuxand M. Winfrey, whose
work  builds upon our earlier studies of
the sulfate-reducing bacteria.
                                     37

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 METHODS

 Comparative 16S rRNA Sequencing and
 Oligonucleotide Probe Design

  The determination of partial or near-
 complete 16S rRNA  sequences has
 been the foundation for the population
 studies.   Initially we focused on the
 now-recognized  major  fiber-digesting
 populations of the rumen (the anaerobic
 ruminal fungi, Butyrivibrio fibrisolvens,
 species  of   Ruminococcus   and
 Fibrobacter). These studies emphasized
 the  above stated  limitations  of  the
 existing taxonomy; organisms classified
 as strains of Fibrobacter succinogenes
 and   F.   fntestina/is   (previously
 Bacteroides  succinogenes)   and
 Butyrivibrio   fibrisolvens  were
 demonstrated to encompass diversity at
 the levels of genus or greater. This is
 an   observation   common  to other
 natural   communities  . as  well.
 Population diversity (genetic diversity)
 has    generally  been  greatly under-
 estimated.  Thus,   past  studies   of
 environmental   abundance   and
 distribution have most often obscured
 the true population/genetic diversity.
  Comparative 16S rRNA sequencing
 has  served both  to  reveal  genetic
 diversity   and to  provide  sequence
 information  to design  Oligonucleotide
 probes for use in environmental studies.
 Probes are either labeled with 32P or a
 fluorescent dye.   Radioactive probes
 have  been  used   for   determining
 population abundance, either of specific
 species (or subspecies) and of larger,
 phylogenetically coherent,  groups (e.g.
eukaryotes  or  archaea).  Fluorescent
 probes  have  been used  to  identify
single   cells,  either  directly     in
environmental  samples  or  in   pure
culture.

The Fibrobacter Paradigm

  The genus Fibrobacter provides the
paradigm for our general experimental
approach to defining the environ-mental
diversity   of functionally comparable
populations of  micro-organisms.  Our
work with   both the sulfate-reducing
bacteria    and   the   fiber-digesting
populations of  the bovine rumen has
shown  that   many  functionally
comparable groups are phylogenetically
coherent.  In general,  a collection of
microorganisms that is phylogenetically
coherent can also be circumscribed by
regions of  rRNA  sequence (signature
sequences) common to, and unique to,
the group (a group  could  consist of a
subspecies, species, genus or larger
more inclusive    assemblage).  These
signature regions serve as target sites
either for Oligonucleotide probes (used
for  identification and quantification) or
PCR primers (for selective sequencing).
Thus, it has been possible to fabricate
probes (and  primer  sets) to specific
functional target groups.
  We  have  outlined  the   genetic
diversity  within  the genus Fibrobacter
by  several genetic criteria,  including
comparative  16S  rRNA sequencing,
DNA  similarity, restriction  fragment
length   polymorphism  and   Oligo-
nucleotide  probe hybridization to both
pure culture isolates and total rumen
contents. The now recognized diversity
within  the genus  as  inferred   by
comparative 16S rRNA sequencing and
DNA similarity is displayed in Figure 1.
Oligonucleotide  probes  specific for the
genus, species and subspecies have
                                     38

-------
NR9
- C1a
         DR7
                  81 A3C  S85
                   HM2
                                          MM4
                                         MB4
                                   BL2
                                        REH9-1
                                    MC1
                    10%


                 Bactero/des fragllis

                 Flavobacterium heparinum
           JG1
           LH1
            DR7
            C1a
            NR9
          r- MC1
            JB1
          JA3C
          r|ss5
                                              Fibrobacter intestinalis


                                     IGroup 4

                                     > subsp.  succinogenes

                                              Fibrobacter succinogenes
            ,MM4
            MB4   Group 3
              .—   i
                              ilVUVIt   \
                            _IMB4   1(
                              IHM2   )
                   10%
                                         B. fragilis
                                         F. heparinum
Figure 1.   Comparison of genetic relationships among Fibrobacter as  inferred by
comparative 16S rRNA sequencina (bottom) and DMA simiisritw  t+™\
    sequencing (bottom) and DNA similarity (top).
                    39

-------
been  used both for  classification and
natural population studies (below).
  Assurance that natural  population
diversity  has  been   reasonably-well
circumscribed comes from consistency
of quantification between the general
and specific probes: the sum of groups
quantified by hybridization to  specific
probes  (e.g. species  and subspecies)
should  be comparable to the   value
determined,by  use of a  more general
probe (e.g.  genus).   In, addition,  we
have  recently constructed  a "global"
panel of 66  reference organism nucleic
acids (representing  the  diversity  of
archaea,  bacteria  and   eukaryotes)
against  which the specificity of each
probe will be evaluated.

RESULTS AND  DISCUSSION

Community  Stability  and Community
Response to Perturbation

  The overall goals of the research are
to define the major ruminal populations,
their constancy (between animals and
over   time)  and   associations
(succession,  competitive  exclusion,
synergism).  Only with  appreciation of
general   community   structure   and
population  associations  can    more
specific questions be addressed. We
are   establishing   the  -necessary
overview  of  community  structure  via
two interrelated studies.  First, we  are
establishing  normal  variation among
major ruminal populations and.second,
we are  using this baseline of normal
variation  to evaluate changes   'in
abundance  of  specific 'ruminal
populations   following  'perturbation
(altered  diet)  and  with  normal diet
variation.
Long Term Population Stability

  Studies of population abundance and
distribution   among  subspecies  of
Fibrobacter have revealed unexpected
population stability.  Of animals so far
inspected  (five   steer),   only   one
subspecies of Fibrobacter predominates
in a single animal at a  given  time.
Among  the five steer examined over
extended time periods (months), either
the  group  2  ^subspecies  or  F.
succinogenes   subsp.  succinogenes
predominated. Initial population studies
of a single animal demonstrated that F.
succinogenes   subsp.  succinogenes
predominated  in that animal. A  more
recent study of fo\jr animals (Figure 2)
demonstrated  a "different  subspecies
(group  2)  to be stably associated with
each  of  these  animals.    Although
representatives of other  species and
subspecies   of   Fibrobacter   were;
present, their  numbers remained low:
throughout  the   study    period  (2;
months).

Diel Population Variation

  Another approach we  are using to
identify    population  associations and
population successions is to follow
population   changes through  time
following feeding.  A study set of four
animals  was  fed  once daily at 11:00
AM and samples were taken  at four
hour intervals  over a three day period.
Initial populations^studies examined the
diversity and  abundance  of different
subspecies of Fibrobacter. Fibrobacter
numbers were depressed  immediately
following  feeding and  reached their
greatest numbers[almost 20 hours after
feeding (as measured by total rRNA
                                     40

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                  COW 1
                                               COW 3
   7

   6 -

   5


2
•E  3i

   2

   1 -\

   0
20        40
   DAYS
                                 60
                                7

                                6-

                                5 -



                            1   3-

                                2 •

                                1 -
                                               0
20        40        60
    DAYS
                  COW 2
                                              COW 4
    7

    6 .

    5 -

    4-

    3 -

    2
              20        40
                  DAYS
                   60
                                7

                                6

                                5-

                            <  4-



                                2

                                1

                                0
20        40
    DAYS
60
    Figure 2. Long term ruminal population stability study demonstrating the  relative
    constancy and stability of populations of Fibrobactersuccinogenes among a study set
    of four cows.  The species-specific probe and universal probes were used to quantify
    relative abundance of this species. Hybridization with the different subspecies-specific
    probes has demonstrated that the group 2 subspecies (Figure 1} accounts for most of
    the genus-specific hybridization throughout the study period.
                                         41

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abundance or fraction of total ruminal
rRNA).   This is  consistent with this
group  functioning  to  digest  more
refractory plant cell wall polymers (and
retained with  larger  feed particles).
Continued  studies   will  examine the
relationship of this  population to other
major fiber-digesting populations (e.g.
ruminococci and the anaerobic ruminal
fungi) during the diel cycle. This should
point up synergism, competition  and
succession among populations serving
a similar function role in  the  ruminal
community (fiber digestion).

Autecological Studies

  We earlier demonstrated the use of
fluorescent  oligonucleotide probes in
combination   with   fluorescence
microscopy  to  visualize  individual
bacterial cells. Fluorescent probes have
been used  to  identify  species  and
subspecies of Fibrobacter, both in pure
culture  and within  rat cecal contents.
Continued studies will use fluorescent
probes  in  combination  (each  labeled
with  a  different  fluorescent dye and
targeting Fibrobacter  subspecies and
other fiber-digesting   populations)  to
examine the distribution  of individual
fiber-digesting bacteria on the surfaces
of plant material. This analysis should
also serve to address  issues of niche,
competition and synergism.

Genetic Isolation of Catabolic Traits

  An   outstanding    question   in
environmental  microbiology   is  the
fluidity  of    gene transfer  among
populations.  The ribosomal RNA offers
an historical  framework for evaluating
the stability  of specific genes within
microbial populations. Specifically, if a
gene is stably associated with a given
population (i.e.  not subject  to  lateral
transfer), then the genealogy of  that
gene  should   correspond   to   the
genealogy of the microbial populations
that it is associated with.
   In general, catabolic genes  appear to
be more readily transferred  between
populations.   The catabolic  genes of
greatest  importance  to  the ruminal
microbial community are those  that
participate in  the hydrolysis of plant
cell    wall   material   (e.g.   poly-
saccharidases).  We initially  evaluated
the  potential  lateral  transfer of  two
polysaccharidases (an endo-glucanase
and  a xylanase) among subspecies of
Fibrobacter.  Both genes  (as  assessed
by  heterologous hybridization) were
found to be widely distributed among
representatives  of  the  genus.   This
raised the question  of  whether  this
distribution reflected lateral transfer or
conservation of structure.  To address
this   question,  we  sequenced  the
homologous endoglucanse from three
Fibrobacter  isolates  (representing  the
diversity of the genus).
   The endoglucanse genes are highly
conserved relative  to total  genomic
similarity.  Excluding the third codon
position  from sequence  comparisons,
the more highly conserved C-terminus
(ca.  900 nucleotides out of 2000)
shares approximately 75%  sequence
similarity with  the   most  distantly
related  strains  (S85 and DR7)  and
approximately 92%  with the closest
(A3C and S85).  This compares with
total DNA similarity values of 5-7%  and
65% (Figure 1).  Conservation does not
appear to reflect lateral   transfer of
these  catabolic  genes  among sub-
                                      42

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species.   The  relationships of  the
different subspecies,  as  inferred  by
sequence  divergence  of  both  the
endoglucanses and the 16S  rRNAs are
consistent   (Figure  3).    This  data
strongly suggests  that  this   endo-
glucanase is a stable component of the
cell and also  is  a  strong  comparative'
argument that  the  enzymes partici-
pating  in fiber  digestion are closely
integrated.    More   generally,   the
retrospective  approach outlined here
could serve for the assessment of past
gene transfer events  among genetically
distinct populations.

FUTURE WORK

  Continued  studies  will  continue to
use the nucleic acid  samples obtained
from the study set of five animals to
further characterize ruminal population
diversity,    stability,   population
associations  and  response  to  per-
turbation. Initially, major fiber-digesting
populations will be  examined in greater
detail. The long term goal is to establish
the  basis     for  stable  community
structure  (reflected  by  long  term
association of specific populations over
time and following  community per-
turbation).     Given  that the   study
animals all received the same diet, an
initial     hypothesis  is  that specific
populations (each representing a subset
of  a  larger  functionally  comparable
collection of populations) are stabilized
by overall community architecture.  If
so,  this should be reflected by specific
associations between populations,  for
example, between  genetically distinct
populations   of  fibrobacter   and
ruminococci.
REFERENCES (ruminal studies)

Amann,  R.I.,  C.  Lin;  R.  Key,  L.
Montgomery, and D.A. Stahl. Diversity
among Fibrobacter isolates: Towards a
phylogenetic   and   habitat-based
definition  of  species  (accepted  to
System.  Appl. Microbiol.).

Dore, J.  and D.A. Stahl.  Phylogeny of
anaerobic  rumen   Chytridiomycetes
inferred  from small subunit ribosomal
RNA sequence comparisons (accepted
Can. J. Botany).

Krumholz,  L.R.,  M.P.  Bryant,  W.J.
Brulla, J.L. Vicini, J.H. Clark and D.A.
Stahl.  Quinella ova/is gen. nov.,  sp.
nov., a phylogenetic analysis (accepted
Int. J. Syst. Bacteriol.).

Stahl,  D.A. and  R. Amann.   1991.
Development and application of nucleic
acid probes in  bacterial systematics.
in: E. Stackebrandtand M. Goodfellow
(Eds.), Sequencing and  Hybridization
Techniques in  Bacterial  Svstematics.
John  Wiley  and  Sons,  Chichester,
England.                     *

Amann, R.!., I.E. Krumholz, and D.A.
Stahl.     1990.     Fluorescent-
oligonucleotide probing of whole cells
for  determinative,  phylogenetic,  and
environmental studies in microbiology.
J. Bacteriol. 172, 762-770.

Amann, R.I.,  B.J. Binder, R.J. Olson,
S.W. Chisholm,  R. Devereux, and D.A.
Stahl.   1990.   Combination of  16S
rRNA-targeted  oligonucleotide probes
with   flow cytometry  for  analyzing
mixed  microbial populations.   Appl.
Environ.  Microbiol.  56: 1919-1925.
                                     43

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            Endoglucanase
                                                — F. succinogenes S8S
                                                - F. succinogenes A3c
• F. succinogenes REH9-1
                                                     • Fiintestinalis DR7
                                                                             • C. therm. D
                16S rRNA
                                                         70%
                                                                 • DR7
                                                                • S8S


                                                                -A3C


                                                                — REH9-1
                                                                        • C. petfringens


                                                                        — C. botulinum
                                                                            • C. barken
                                                        10%
Figure 3. Comparison of endoglucanaseand 16S rRNA sequence relationships among
Flbrobacter,
                                        44

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Mannarelli, B.M.,  D.A. Stahl, and R.J.
Stack.   1990.   Genetic  relatedness
among    strains   of  Butyrivibrio
determined   from   DNA   analysis,
extracellular    polysaccharide  (EPS)
composition and 16S rRNA sequences.
Abstracts of the Annual Meeting of the
American Society  for Microbiology.

Montgomery, L. Flesher, and D. Stahl.
Transfer  of  Bacteroides succinogenes
(Hungate) to Fibrobacter gen. nov.  as
Fibrobacter succinogenes comb.  nov.
and Fibrobacterintestinalissp. nov. Int.
J.   Syst.  Bacteriol.  38,   430-435
(1988).
Stahl, D.A., B.F. Flesher, H. Mansfield,
L. Montgomery.   1988. The  use of
phylogenetically based  hybridization
probes for studies of ruminal microbial
ecology.  Appl.  Environ. Microbiol. 54,
1079-1084.

Stahl,  D.A.     Phylogenetically-based
studies  of   microbial   ecosystem
perturbations.   1988.   In: American
Chemical Society Symposium Volume:
Biotechnology  in Crop Protection. P.
Hedin (Ed.) pp. 373-390.
                                    45

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  USE OF 16S rRNA PROBES TO CORRELATE SULFATE REDUCER COMMUNITY
                 STRUCTURE WITH MERCURY METHYLATION

                     Michael R. Winfrey and Janet Winfrey
                       University of Wisconsin-La Crosse
                    Department of Biology and Microbiology
                            La Crosse, Wisconsin
INTRODUCTION

  In order  to  assess the  effect of
releasing    genetically   engineered
microorganisms (GEMs) in the environ-
ment on native microbial communities,
it is necessary to be  able  to  detect
changes in the microbial community.  A
potential method to evaluate microbial
ecosystem   changes  is  to  quantify
changes  in  microbial  activities  and
changes   in  microbial   community
structure in response to a perturbation.
Although  methods are  available to
quantify  most  microbial   activities,
severe limitations exist in the ability of
microbiologists to characterizemicrobial
communities. Traditional enumeration
and  identification  techniques  require
cultivation of microorganisms, yet many
native  microorganisms are difficult or
impossible to cultivate.  This dilemma
has precluded any accuratedefinition of
microbial   community   structure   in
natural habitats.
  The use of 16S rRNA-targeted probes
to detect microbial  groups offers  a
method to  overcome this limitation  in
microbial ecology. Since the probes are
used on  total RNA extracted from  a
natural sample,  this method  eliminates
the  need  to  cultivate   the   micro-
organisms  and  has  the  potential to
accurately define microbial community
structure.
  Because  of the wide diversity of
microbial   populations  in   natural
communities, the use of this technique
as a risk assessment tool should target
microbial groups that play a  vital role in
the community.  In addition,  an ideal
target   group   would  change  in
abundance and/or diversity in response
to  any  changes  in  the  microbial
community.     The   sulfate-reducing
bacteria  (SRB)   represent  such  a
microbial group because they act as
terminal organisms in  anaerobic food
chains  and  are essential to  efficient
anaerobic decomposition  in  habitats
containing  sulfate.     By  consuming
products of  bacterial fermentations in
higher   trophic  levels,  they  allow
thermodynamically   unfavorable
fermentations to occur and "pull" the
entire anaerobic food chain.  Thus, a
change in the SRB population would be
detrimental to carbon decomposition in
the environment  they inhabit,  and
changes in other microbial groups that
provide the substrates for the SRB may
also alter the SRB population.
  We used 16S rRNA-targeted probes
to groups of SRB to determine their
community structure  in sediment from
northern Wisconsin lakes and correlated
these   communities    with   mercury
methylation  and lake  pH.   Since SRB
are  known  agents   of   mercury
methylation  and    methylmercury
bioavailability is enhanced  in  low  pH
                                     46

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 lakes, it is possible that sulfate loading
 due to acid-rain may play a role in the
 enhanced methylmercury production by
 stimulating  methylation   by  sulfate
 reducers.  Thus, acidified lakes provide
 an excellent system to evaluate the use
 of rRNA-targeted probes  to  examine
 changes  in  community  structure in
 response to an ecosystem perturbation.

 METHODS

 Sample Collection

   Lake sediment  and water  samples
 were  collected  from selected lakes
 (ranging in pH from 4.6 to 7.1) in the
 Northern  Highlands  Lake district  of
 Wisconsin.   One  of the lakes, Little
 Rock  Lake,  is  particularly useful  in
 studying  SRB   community  structure
 because it has been divided and one
 basin is being artificially acidified with
 sulfuric acid.  Sediment samples (upper
 5  cm.) were either hand collected by a
 diver (for  littoral sediments) or with a
 peristaltic   pump    (for  flocculent
 profundal  sediments).  All sediments
 were placed  immediately  on  ice after
 collection and either frozen or extracted
 within  24   hours  of   collection.
 Methylation assays were set up within
 24 hours of collection. Water samples
 were collected using a peristaltic pump.
 A  Pellicon tangential flow filter system
 (Millipore) fitted with four 0.45 fjm filter
 cassettes   was   used  on  site  to
 concentrate water  down  to  1 liter.
 Lake water and concentrates were kept
on ice to minimize RNase  activity.
Water concentrates were transported to
the lab on ice,  centrifuged,  and the
pellets frozen.   Processing of water
concentrates was done within  24 hours
 of  collection.      Strict   anaerobic
 technique was used during  collection
 and storage of anoxic sediment and
 water samples.

 Methylation Assays

   Mercury methylation was  measured
 by spiking water or  sediment samples
 with 203Hg(ll) (1.0/yg as 203HgCI2) and
 incubating  24   hours   at   in   situ
 temperature.     Methylation   was
 quantified by extracting the radioactive
 methylmercury produced  followed  by
 scintillation counting.

 RNA   Extraction  from  Environmental
 Samples

   RNA  in  sediment  samples  was
 initially extracted by placing  1  ml of
 sediment in a 2-ml  bead  beating vial
 containing 1.2 g baked zirconium beads
 and 0.2  g  acid  washed  polyvinyl-
 polypyrrolidone  (PVPP).   Vials  were
 then   filled  with   50  mM  sodium
 acetate/10 mM EDTA  buffer (pH 5.2)
 containing p-mercaptoethanoland SDS.
 The vial was shaken at high speed on a
 Mini Beadbeater for six 1-min intervals
 with a two minute incubation on ice
 between  each beating cycle.   The
 aqueous  phase  was separated by
 centrifugation and extracted once with
 an equal volume of phenol  (equilibrated
 with 50  mM sodium acetate/10 mM
 EDTA  (pH 5.2),  once with  phenol:
 chloroform and twice with chloroform.
 RNA   was  ethanol  precipitated,
 resuspended in RNase-free water, and
 stored  at -20°C.  All extracts were
 quantified by absorbance  at  260 nm
and  the  purity  was evaluated  by
 260/280 and  234/260 ratios.
                                     47

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  In order to quantify the amount of
RNA  per volume  of  environmental
sample, we modified the procedure so
that the percentage of RNA recovered
in the extraction  was  quantitatively
known.   To  increase  the  aqueous
volume in  the  extraction  vials,  we
decreased the amount of PVPP by  half
(to  0.1  g/vial)  and  decreased  the
amount of zirconium beads from 0.5 ml
(1.2 g) to 0.5 g.  The total aqueous
volume of an extraction mixture in a 2-
ml bead beating vial was calculated by
subtracting the volume of non aqueous
materials (PVPP,  zirconium beads,  and
sediment dry weight) from the total
volume of liquids in each tube. After
bead   beating  and centrifugation  a
constant volume (900 //I) of aqueous
supernatant  was  removed.    After
solvent extraction, we recovered  a
constant volume  (500  fj\)  of  the
aqueous  extract. From  the  initial  and
final aqueous volumes  we  calculated
the percent recovery of the RNA in the
sample.

Nucleic  Acid   Hybridizations  and
Detection

  RNA extracted from  environmental
samples and reference standard RNAs
(from  pure  cultures of  SRB) were
denatured for  10  minutes in  1.5%
glutaraldehyde at 30°C  and applied to
nylon  membrane with a  slot   blotter.
Amounts applied  ranged from 1  to
1000 ng  (based on A260) for environ-
mental samples and from 0.01 to 10 ng
for  reference standards.  Membranes
were baked at  80°C for 1 hour, then
prehybridized at 40°C for 4 hours in a
hybridization bag  containing  0.1   ml
hybridization   buffer  per  cm2   of
membrane. Universal (1400) or SRB-
specific oligonucleotide  probes were
end labelled with polynucleotide kinase
and  (gamma-32P)-ATP.  SRB specific
probes  used  were:    687   (for
Desulfovibrio), 660 (for Desulfobulbus],
129   (for  Desulfobacter],  BTM  (for
Desuffobacterium],   813   (for
Desulfosarcina,  and  Desulfococcus),
and 804 (which  detects  all organisms
detected by the 129, BTM  and  813
probes).    Probes  were   added  to
prehybridized    membranes   at  a
concentration  of 2  X  106  cpm/ml
hybridization buffer.  Each  membrane
was  hybridized at an  optimized probe-
specific temperature  for  16  to  18
hours.  Membranes were washed 30
minutes at  room temperature and 30
minutes at an optimized probe-specific
wash temperature in a 1 % SDS, 1X
SSC wash buffer. Air dried membranes
were placed in X-ray cassettes against
preflashed Kodak X-AR film, exposed at
-70°C (or room temperature for the
universal probe), and  developed  to
visualize   hybridization  signal.
Hybridization signal was  quantified by
scanning densitometry coupled  with a
peak  integration  software  package.
Hybridization signals of environmental
samples were  converted  to ng RNA
based on the  signals from  hybridized
reference standards.

Quality Assurance

  We initiated several quality assurance
protocols to evaluate  factors that  may
affect  the  amount  of  hybridization
signal produced  from RNA extracted
from environmental samples.  These
procedures included: (a) evaluating the
effect  of  varying  degrees   of
                                    48

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 glutaraldehyde denaturation on  the
 hybridization signal from environmental
 RNA,  (b) testing the hybridization of
 SRB-specific  probes  to  non   SRB
 negative  controls   containing  RNA
 equivalent to the amount of total RNA
 present in environmental samples, and
 (c)   evaluating   the   presence   of
 components in environmental extracts
 that alter the hybridization signal by
 performing  standard   addition
 experiments.
   To   evaluate   the   effect   of
 glutaraldehyde denaturation on  the
•hybridization signal, environmental RNA
 samples were  denatured in  1.5, 3.0 or
 4.5% glutaraldehyde and incubated in a
 30°C heat block for intervals from 0 to
 60 minutes.   These  samples were
 applied  to  nylon   membranes   and
 hybridized with  the 1400 (universal)
 probe and the  687 (Desulfovibrio spp.)
 probe.
   The   amount   of   non-specific
 hybridization of SRB-specific probes to
 environmental  RNA was determined by
 preparing   negative   control  RNA
 extracted from  grass,  yellow  perch
 fillets,   Clostridium  perfringens,   and
 Pseudomonas  aeruginosa.    Extracts
 from each species were combined in
 equal amounts (based  on A260)  and
 the mixture  applied to membranes in
 amounts equivalent  to the amounts of
 environmental  RNA applied.
   Matrix  effects   in  environmental
 extracts were examined by the addition
 of reference standards to sediment and
 water extract,  or to extracts that had
 been treated with RNase or with RNase
 plus DNase.  Samples were hybridized
 with the universal probe and the slope
of  standard  curve  in  the standard
addition experiments was compared to
 the  slope  obtained  with  reference
 standards alone.

 RESULTS AND DISCUSSION

 Methods  Development .and  Quality
 Assurance

   We modified the extraction procedure
 to allow quantitative calculation of the
 total amount of RNA extracted from a
 given volume of environmental sample.
 Decreasing the amount of zirconium
 beads  or  PVPP had no effect on the
 recovery  (based  on  A260)  or  purity
 (based on A260/A280) of the extracted
 RNA  and  allowed  us to  routinely
 recover 29% of the aqueous extract in
 sediment  samples.  This  will  allow
 accurate calculation  of the  absolute
 amount of SRB-specific 16S  rRNA in
 addition  to  the   relative  abundance
 (percent  of   the total   populations
 accounted  for  by  SRB).    Absolute
 amount of  16S   rRNA  is  a  better
 parameter  to  describe   community
 structure  since  the  total  microbial
 population  varies  significantly among
 different  habitats.    One  potential
 limitation  of the current  extraction
 procedure is that  the effectiveness of
 bead   beating   on   quantitatively
disrupting  all  cells  in  the  sample
 population is unknown.  This will affect
the accuracy of quantifying  microbial
communities using 16S rRNA probes
and  should  be  investigated  in  the
future.
  Quality   assurance   protocols  are
important in any quantitative assay and
should be  developed and incorporated
into rRNA hybridization studies  if the
results are to be used quantitatively or
semiquantitatively. We have begun to
                                    49

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develop procedures to quantify factors
that may interfere with the hybridization
signal of SRB-specific  probes.  Since
glutaraldehyde denatures by covalently
bonding  to the  RNA   molecule, the
degree of exposure to glutaraldehyde
could conceivably affect the  resultant
hybridization signal. However, varying
the   time   and   concentration   of
glutaraldehyde used to  denature RNA
showed no significant, effect on the
hybridization  signal  obtained  from
sediment extract.  Only the elimination
of glutaraldehyde  resulted in a lower
hybridization signal.
  Matrix interferences were observed in
standard addition experiments when
sediment   extract  was  added   to
reference standards.  The addition  of
10  and 50 ng  (based on A260)  of
sediment RNA to  reference standards
decreased   the   slope  of   standard
addition curves  (compared to curves
prepared with reference standards)  by
about 24%.  This indicates  that the
sediment extract contains material that
interferes  with the detection of  16S
rRNA,  but  the matrix  effect can  be
corrected for with traditional  standard
addition  procedures.    When  RNase
treated  or  RNase  and  DNase treated
sediment   extract  was  added   to
standards,   the  slope   was   not
significantly different from the slope of
the standards alone. This suggests that
the interfering matrix in the extract was
RNA.
  We observed significant non-specific
hybridization with  SRB-specific probes
when amounts of negative control RNA
comparable   to   amounts    of
environmental sample were applied  to
membranes. Since the  hybridization
signal  obtained  from  environmental
extracts is often low, correcting for non
specific  hybridization  by   negative
controls  is  important if  quantitative
estimates of SRB populations is to be
obtained.

SRB Community Structure in Wisconsin
Lakes

  The  relative abundance of the  SRB
groups examined was similar in all lakes
and  Desulfovibrio   spp.   comprised
greater than 66% of the total  SRB
population in each lake.  The total 16S
rRNA (based on  the 1400 probe)  was
positively correlated  with  lake  pH
suggesting that total sediment biomass
decreases at low pH.   Methylation was
active  in  all lake samples  and  was
negatively correlated with lake pH.  The
relative abundance of all SRB groups
detected was negatively.correlated with
lake, pH although the correlation  was
only significant (P < 0.10) with two of
the probes.  This suggests that sulfate
reducers make up a larger portion of the
microbial population  in low  pH lakes,
but should be further evaluated with
larger  sample  sizes.   Since  SRB are
known  to  be active methylators of
mercury,  this  may  account for the
inverse correlation observed  between
sediment methylation and lake pH.
 ' Although  the  use  of  16S  rRNA-
targeted probes  is in its infancy  in
microbial ecology, our results indicate
that they  show  much  promise  in
defining microbial community structure
in natural habitats. Further work needs
to  be  done before truly  quantitative
estimates  of   bacterial  community
structure are available, but the use of
SRB-specific probes provide information
on  the community structure of these
                                     50

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key   microorganisms   in   anaerobic
habitats   that  was   previously
unobtainable.  Thus, determination of
SRB community structure based on 16S
rRNA probes  provides a  useful risk
assessment tool to evaluate changes in
microbial communities.

FUTURE WORK

  In   the  remainder  of the  current
funding period we will characterize the
SRB   communities  in  the   anoxic
hypolimnion  of  Little  Rock  lake.
Samples for this study were collected in
the summer  of  1990  to  follow the
development  of SRB communities as
the anoxic hypolimnion built up, and in
the fall to monitor persistence of SRB
after the fall turnover when the water
column became aerobic. We will also
be   sampling  sediments   from the
acidified  and  reference basin of Little
Rock Lake in a random design to allow
us to show any statistical differences in
SRB  community structure and mercury
methylation between basins.
  Future areas of work involving the
use of, 16S  rRNA-targeted probes to
determine   microbial   community
structure should  focus  on  further
methods improvement to allow more
quantitative measurements to be made,
and further evaluation  of changes  in
SRB community structure and activity
in  response  to   environmental
perturbations.

PUBLICATIONS (abstract)

Winfrey, J., R.  Devereux, and M. R.
Winfrey. 1991.   Use of 16S  rRNA-
targeted   Probes   to   Correlate
Community ,  Structure  of   Sulfate-
reducing  Bacteria   with   Mercury
Methylation in Freshwater Sediments.
American  Society  for  Microbiology
Annual Meeting, Dallas, TX, May 5-9.
                                    51

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              APPLICATION OF DNA HYBRIDIZATION TO COMPARE
                     BULK DNA'S FROM DIFFERENT SOILS

                   L. Arlene Porteous and John L. Armstrong
                     U.S. Environmental Protection Agency
                       Environmental Research Laboratory
                              Corvallis, Oregon
 INTRODUCTION

   The distribution and persistence of
 recombinant DNA  in the  terrestrial
 environment has  brought  molecular
 ecologists  to  develop techniques for
 extracting DNA from soil samples. One
 approach relies on  recovery of intact
 bacteria  from  soil before  the DNA  is
 extracted.  Another approach involves
 direct- extraction of DNA without prior
 removal of the cells from soil. Based on
 these procedures, we developed a new
 direct-extraction method tha| is small-
 scale, rapid, and simple, yielding up to
 20 jjg of DNA from  a gram of soil.
 Furthermore, we have applied  DNA
 hybridization of RFLP's and dot blots to
 demonstrate  differences  between
 DNA's extracted from  different soils.

 METHODS

  Soil  samples  were  collected near
 Corvallis, Oregon: under an Oak tree, in
 a  sheep pen,  from  the  rhizosphere
 under grass.  One gram of soil in a 50
 ml plastic Oakridge tube was combined
 with six ml of mixing buffer (0.5 M D-
sorbitol,  15% PEG 4000,  2% diethyl-
dithiocarbamic acid, 100  mM  EDTA,
and 50 mM Tris-CI, pH  8.0) and vortex-
mixed for one minute.  Then, 500 mg
of polyvinylpolypyrrolidone was added.
Lysis of  bacteria was promoted with
100 jj\ lysozyme solution  (50 mg/ml)
 and fungal  cell  walls were degraded
 with 120//I  Novozym 234 solution (50
 mg/ml). The sample was vortex-mixed
 for 15 sec and incubated on ice for 1 -2
 h.  Next, 3.8 ml  lysis buffer (4% SDS,
 100 mM EDTA, proteinase K at 500
 //g/ml, and  50  mM  Tris-CI,  pH 8.0)
 were added. After mixing the contents,
 the tube was returned to ice for 1-16 h.
 The extract was then centrifuged at
 5000 x g for 5 min. at 4°C and placed
 in a sterile Oakridge tube on  ice.  To
 recover additional DNA,  the pellet was
 resuspended in 3 ml wash buffer (100
 mM EDTA,  50 mM Tris-CI, pH  8.0),
 vortex-mixed,  and centrifuged.  The
 previous  .step  with wash buffer was
 repeated  and both  supernatant liquids
 were combined.  Then, 5 M  potassium
 acetate   was    added   to    a   final
 concentration of  0,5 M. After  1-2 h  on
 ice, the sample was centrifuged at 4°C
 for 10  min. at  15,000 x g.   The
 supernatant  liquid was mixed with two
 volumes   of   95%   ethanol   and
 centrifuged at 10-15°C for 10 min. at
 15,000 x g.  The pellet was dried and
 suspended in 1 ml buffer (0.01 M Tris-
 CI, pH 8.0, and 0.001  M EDTA; TE).
  Ten fj\ of each  DNA  extract was
electrophoresed for 1.5 h at 125 V in
0.7% agarose using a buffer consisting
of 0.04 M  Tris-acetate  (pH 8.0) and
0.001 EDTA (TAE), and stained for 30
min. in TAE containing  0.4//g ethidium
bromide  per milliliter.   Gels  were
                                    52

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photographed  over UV light.    DNA
extracts adjusted to a density of about
1.58  g/ml  were purified by gradient
centrifugation using standard methods.
Extracts  were  further  purified  with
Geneclean  Glassmilk (BIO  101,  Inc.)
according to manufacturer's directions.
  Restriction enzyme  digests   were
performed with approximately 0.5 jug
DNA and 2 fj\ of endonuclease in 20 //I
buffer.  To  determine if impurities in
DNA   extracts   inhibited   restriction
enzyme activity, 0.2//g of pBR322 was
added to some reaction mixtures. After
incubating for 16-24 h at 37°C, more
enzyme was added for  another hour.
DNA  samples  were  electrophoresed,
transferred  to  a  nylon   membrane
(procedure by Schleicher and Schuell),
and hybridized with the following   P-
labeled probes:  nick-translated pBR322
DNA   or   random-primed  labeled
chromosomal DNA from  Pseudomonas
putida,   Enterobacter   cloacae  and
Bacillus cereus,  and the rRNA genes of
Escherichia  coli (Bam   H1  insert  of
plasmid pKK3535).  The final post-
hybridization washes   for  pBR322-
hybridized  filters  were  performed  at
56°C using  0.1X SSPE (18 mM NaCI,
1 mM NaPO4,  pH 7.7,  and 0.1 mM
EDTA)  and  0.5%  SDS.  Southern
transfers of RFLP's hybridized with the
random-prime   labeled   probes   were
washed in 2X SSC  (0.3 M NaCI and 30
mM sodium citrate, pH  7)  and  0.5%
SDS at 65°C.  Autoradiograms  were
prepared by standard methods.
   Dot blots were prepared on nylon
membranes  (Schleicher  and  Schuell)
according   to  the   manufacturer's
instructions.  Concentrations of serial
dilutions  applied through  a filtration
manifold  (Bethesda Research  Labs.)
included: 500 to 3.9 ng/dot for  soil
DNA's;  '10  to   0.08  ng/dot  for
chromosomal DNA's of P.  putida, E.
cloacae,  and B. cereus;  1  to 0.008
ng/dot for  salmon  DNA and  E.  coif
rRNA  genes.     Membranes  were
hybridized  according  to  methods  of
Boehringer  Mannheim  with random-
primed, digoxigenin labeled DNA probes
(P. putida, E. cloacae, B. cereus, and E
coli   rRNA   genes).     Final   post-
hybridization  washes were  performed
under highly stringent conditions using
0.1X SSC  and  0.5% SDS at 68°C.
Autoradiograms were  produced using
the chemiluminescent substrate, Lumi-
Phos 530 (Boehringer Mannheim).

RESULTS AND DISCUSSION

  Soils  from different sources were
used to  demonstrate the effectiveness
of the DNA extraction method.  The
method  reported here yielded up to 20
fjg  DNA from  one gram of various
agricultural  soils.  In some cases (e.g.,
from  the soil of a sheep  pen),  we
recovered up to 35 JJQ  DNA  in one
gram. Determinations were measured
by   spectrophotometric  absorbance
(wavelength,  260  nm).    Accurate
readings depended  on the  complete
elimination   of   Nal  and   glassmilk
following the Geneclean  purification
step. Due to the possible selectivity of
the extraction process, all of the DNAs
from a sample  may not be retrieved.
For example, some species of DNA may
be firmly bound  to soil components.
  The degree of purity  and  restrict-
ability that resulted from the use of this
method was measured on agarose gels.
High molecular weight DNA, 20-25 kb
in  size,   was   routinely  observed
                                    53

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 following  the  extraction  procedure.
 This DNA was  digested by restriction
 enzymes so that the entire DNA sample
 was fragmented into  smaller pieces.
 This observation suggested that  the
 DNA lacked residual, soil constituents
 that inhibit endonuclease activity. This
 was further demonstrated  by  the
 appearance  of  completely restricted
 pBR322 DNA that was added to digests
 containing  purified soil  DNAs.   The
 resulting one band at 4.4  kb clearly
 demonstrated complete  restriction  of
 the   plasmid to   the   linear  form.
 Southern transfers of these  agarose
 gels were probed with pBR322 in  the
 more   sensitive  method   of  DNA
 hybridization.   The  autoradiograms
 verified complete digestion with Eco Rl,
 Sal I, and Bam HI  (Bethesda Research
 Labs.).  These findings emphasized the
 reliability of the method as an effective
 means  to purify DNA from different
 types of soils.
   Bulk  DNA extracts from soils  were
 examined by RFLP analysis to determine
 differences in the observed fragmented
 band   patterns   between  samples.
 Distinguishable,  isolated  bands  were
 not detected in any of the soil extract
 samples analyzed.   The completely
 fragmented  DNA appeared as  a  wide
 distribution of molecules  ranging from
 less than 0.125 kb to approximately 23
 kb.  We suggest  that   the  lack  of
 discernible bands is  indicative of the
 presence of a wide variety of DNAs.
 The work of other researchers confirms
 the diversity of microorganisms found
 in the  soil   and the  resulting  high
 heterogeneity  of  their   DNAs.
 Discernible restriction fragments would
be  seen  if  the DNA   extract  was
relatively homogeneous, e.g., a  sample
 containing  DNA  from a  predominant
 microorganism in large numbers.
   Hybridization of electrophoresed soil
 DNAs  was  observed  when  DNA
 extracts were  digested with Sail and
 Notl (Promega) and probed with the E.
 coli rRNA genes.  In contrast, little  or
 no hybridization  was observed when
 the chromosomal DNA's of P. put/da, E.
 cloacae, and B. cereus were used  as
 probes.  As with  visual analysis of the
 RFLP's,  discernable bands  were not
 seen following  DNA hybridization with
 any of the bulk DNA extracts examined.
 Due to the lack of specific bands, dot
 blots  were  utilized  to  determine
 whether ,  quantitative   differences
 between samples were evident.  Using
 post-hybridization  washes   under
 conditions   of   high   stringency,
 differences  were  detected.  Different
 samples demonstrated different degrees
 of homology to  a probe encoding the E.
 coli rRNA genes.  The extract from the
 soil  of  the  rhizosphere  of  grass
 contained 8 pg of hybridizable DNA and
 that from the sheep pen had 62.5 pg,
 i.e.,  0.0016%  and 0.0125%  of  the
 total DNA, respectively.  When using
 the P. put/da chromosomal DNA probe,
 differences were also detected between
 the two  DNA extracts.  Related DNA
 recovered from the rhizosphere of grass
 and  the  soil  from  a  sheep   pen
 represented  0.06% and 0.25% of the
total DNA.  Hybrids were not detected
 when  E.  cloacae  and  B.   cereus
chromosomal DNA's  were  used as
 probes.  Based on the sensitivity of the
method,  we  estimated  that  both
extracts  contained  <0.06% of DNA
related to E. c/oacaeDNA and <0.12%
of DNA related to B.  cereus DNA,  if
present.
                                    54

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  In summary, we describe a bulk soil
DNA extraction method that is rapid
and small-scale. The DNA extracts are
pure enough to be restricted and used
in hybridization experiments. Coupled
with the use of dot blot techniques,
specific DNA populations in soil DNA
extracts   can  be  compared   and
quantified.

FUTURE WORK

    Plans for this project include: a) use
various probes to identify DNAs from
various   "functional   groups"   of
microorganisms, b) enhance sensitivity
of  detection with PCR  methods, c)
assess efficiency of method for  re-
covery  of  fungal,  protozoan,  plant
rhizosphere, and "microfauna" DNAs,
d)  retrieve  recombinant  DNA  after
introducing  a specific  GEM  into  a
terrestrial   microcosm,   e)   develop
methods to recover bulk DNA from leaf
litter of tilled soil containing transgenic
plants, and f) demonstrate effects of a
GEM on population of DNA molecules
indigenous to soil.

PUBLICATION

Porteous, L. A.,  and  J. L. Armstrong.
1991.  Recovery of bulk DNA from soil
using a rapid,  small-scale extraction
method.  Current Microbiol. (accepted
for publication).
                                     55

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                     COMPARISON OF xy/E GENE ACTIVITY
                   IN DIFFERENT MOLECULAR CONSTRUCTS

               H.M. Abebe1, R.J. King1, S.E. Lindow2, K.A. Short3
                               and R.J. Seidier3
                   ManTech Environmental Technology Inc.1,
                       Environmental Research Laboratory
                               Corvallis, Oregon
                         Department of Plant Pathology,
                            University of California,
                              Berkeley, California2
                      U.S.Environmental Protection Agency
                       Environmental Research Laboratory
                              Corvallis, Oregon3
 INTRODUCTION

   The activity of the xy/E gene product,
 2,3-catechol dioxygenase, is a useful
 colorimetric  tool  for  autecological
 investigations. The yellow color  (2-
 hydroxymuconic  semialdehyde)
 produced  due   to  the  enzymatic
 cleavage  of  catechol,   a colorless
 substrate,   has   been  exploited   to
 enumerate   target   microorganisms
 released into soil, lake water or sprayed
 onto  plant leaves. Thus far however,
 the application of the xy/E  marker gene
 has  been  confined  to   plating  and
 spectrophotometric assays of sonicated
 filter concentrated cells.
   The objectives of the current study
 were: a) to assess the stability and level
 of expression of a plasmid-borne versus
 chromosomally inserted  xy/E  marker
 gene, b) to  examine the effect of cell
 density and  growth phase on catechol
 2,3-dioxygenase   activity,   c)   to
 determine the efficacy of xy/E  gene
 expression for direct determination of
 cell numbers via a spectrophotometric
 analysis   of   2-hydroxymuconic
semialdehyde, and d)  to establish the
 sensitivity of the technique in detecting
 target microbes by HPLC analysis.

 MATERIALS AND METHODS

 Cultures and media

   Luria-Bertani (LB)  agar was used for
 plate counts.  Liquid  cultures  Were
 grown in LB broth. All cultures were
 incubated at 30°C. Broth cultures were
 maintained  on a rotary shaker at 200
 rpm. Appropriate antibiotics were added
 when selective media were needed.

 Construction   of  xv/E  Gene Marked
 Strains                        .:•

   Most molecular biological techniques
 were performed according to standard
 procedures.  A typical  approach used
 was similar to that for inserting the xy/E
 gene  into the iceC structural  gene.
 Plasmid  plCE1.2, which contains the
 iceC gene,  was  subjected  to  partial
digestion   with  sa/1   (Boehringer
Mannheim),   followed   by   dephos-
phorylation    with   .calf   intestinal
phosphatase (Boehringer Mannheim).
                                     56

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Plasmid pR01733 was restricted with
xho\   (Boehringer  Mannheim)   and
electrophoresed   in   low  melting
temperature   agarose  (International
Biotechnologies, Inc.).  The 2.3 kb band
containing the xy/E structural gene was
excised and extracted. The 2,3 kb xho\
fragment from  pR01733 was ligated
with  the  partially  digested  PICE1.2
DNA, and transformed into E. C0//DH5.
Colonies   resistant  to  50  //g  ml-1
ampicillin  were sprayed  with 0.1M
catechol,  and  those  colonies  which
turned yellow were isolated. One of the.
x//E+ colonies that exhibited an Ice-
phenotype  was  selected  and  the
recovered  plasmid   was   designated
pSEL2.

Marker Exchange

  Selection for integration  of  non-
replicating  but  mobilizable  plasmid.
Plasmid pSEL2-4 was mobilized into  P.
syringae Cit7 which.contains the iceC
gene   using  pRK.2013  as , a -helper
plasmid.   Colonies  which  fluoresced
under UV were selected, and tested for
xy/E activity by spraying with catechol*
Single xy/E+ Flu+ (colony growing on
King's B  agar  plates fluoresce  upon
exposure  to  UV  light) colonies  were
restreaked onto KB amended with 100
JJQ   ml"1,,  rifampicin,  30 , VQ  ml"1
kanamycjn,.and 100#g ml"1 ampicillin,
and tested for ice  nucleation activity  to
confirm the lce+  phenotype and thus
ensure that  plasmid  integration  had
occurred but a second  recombination
event had 'not.  Strains designated mer
3,  12, 15,  and  17  were used for
subsequent marker exchange steps.
Identification of Second Recombination
Events

  Four  different  xy/E+  Flu+  lce +
colonies  were  inoculated  into  KB
amended with 100/yg ml"1 rifampicinto
107 cells ml"1, and cultured at 28°C for
about 12 hours to a cell density of over
10? cells-ml"1.  Cultures were diluted
1:50 into fresh medium,  and regrown
to 109 cells ml"1. This cycle of dilution
and regrowth was repeated  10 times.
Dilution series of the final culture were
plated onto  KB amended with  100 //g
ml"1  rifampicin  and grown  2 days  at
28°C.
  Approximately  104  colonies  were
replica-plated onto KB containing 100
fjQ   ml"1  rifampicin,  30   jjg   ml"1
kanamycin, and 100//g ml"1 ampicillin.
About  1%  of  these colonies did not
grow in the presence of kanamycin and
ampicillin and were .thus  hypothesized
to   have   undergone  a   second
recombination   event  in  iceC  distal
relative   to   xy/E  from  the   first
recombinational   event.      Of  the
kanamycin-  and/or ampicillin-sensitive
colonies, about 9% were also  xy/E +
and Ice-, suggesting that excision for
vector-derived   sequences   and
recombinational placement of the native
iceC 'gene with the xy/E-iceC  fusion
construct had  occurred.   Five such
strains, designated hap 18, 45, 49, 69,
and 72 were obtained.

Stability  and  Expression of Genetic
Markers

  Antibiotic  and xy/E gene expression
and stability were assayed by spread
                                     57

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 plating onto selective and nonseiective
 media. All cultures except P. syringae
 Cit7  #69 colonies, were assayed for
 expression of catechol 2,3-dioxygenase
 activity after 48 hours of incubations at
 30°C.   P. syringae  Cit7  #69  was
 assayed after 72 hours of incubation.
 White colonies, on selective and  non-
 selective agar plates, were enumerated
 before they were  sprayed with a 1 %
 aqueous catechol solution for counting
 xy/E gene harboring constructs.

 Determination of Bacterial Growth

   Cell density and  bacterial  growth
 phases  were  determined  by  plate
 counts   and   optical   density
 measurements. A 300 ml  LB broth
 amended   with   an   appropriate
 antibiotic(s) was  inoculated, from an
 over  night  culture,  to a  final  cell
 concentration of less than 106 cells/ml
 and incubated at 30°C. Samples were
 withdrawn  for  plating  and  optical
 density measurements (spectronic 21
 Milton Roy Co., Rochester, NY set to a
 wave length of 600nm) at intervals 0,
 15, 24, 39,  48, 63, 72 and 87 hours.

 Spectrophotometric Enzyme Assays

  Cells were washed twice with 10 ml
 20 mM phosphate buffer, pH 7.2.  The
 pellet  from the  second  wash  was
 resuspended  in  100  mM phosphate
 buffer, pH 7.5'. One milliliter of this cell
 suspension  was  then  treated  with
 catechol  solution   to   a   final
 concentration of 0.05% and incubated
 for  30  to  60   minutes   at  room
 temperature. Catechol 2,3-dioxygenase
activity   was  then  determined
spectrophotometrically (Beckman DU-
 BS)  by   reading  the  increase   in
 absorbance at 375 nm. Where catechol
 2,3-dioxygenase activity resulted in an
 intense  yellow '  coloration  (high   2-
 hydroxymuconic   semialdehyde  pro-
 duction), serial dilutions of the product
 was made to  accommodate ranges  of
 Spectrophotometric  specificity   and
 sensitivity.

 HPLC Enzyme Assays

   For   HPLC   analysis,   cells   were
 washed twice with 20 mM phosphate
 buffer, pH  7.2 and resuspended in the
 same buffer.  Final catechol concen-
 tration and incubation time was 0.05%
 and 30 to 60 minutes,  respectively.
 Following  incubation  however,  the
 samples  were pelleted at  13000 rpm
 for  10  minutes  at  4°C  using   a
 microfuge  (Beckman  Microfuge  11).
 The supernatant was transferred into
 LC automatic sampler 2 ml vials with
 11  mm crimp  caps  (Hewlett-Packard
 Co., Avondale, PA). The samples were
 analyzed  with   an   HP-1090   high
 performance   liquid    chromatograph
 (Hewlett-Packard  Co.,  Avondale, PA)
 coupled to a  diode-array  UV detector
 and  monitored at 375 and 277  nm
 (lambda max for  2-  hydroxymuconic
 semialdehyde  and catechol respect-
 ively). Samples were separated by an
 isocratic mobile phase (35% water with
 0.1% acetic acid, 65% methanoDon an
 HP C18  reverse  phase  micro-bore
 column with C8 guard column at a flow
 rate of 0.40 ml min"1.  External catechol
 reference  standards   (NBS  certified)
were incorporated to  bracket samples'
concentrationsand analyzed in triplicate
every tenth injection.
                                    58

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RESULTS AND DISCUSSION

  Marker  instability,  especially with
markers carried on a plasmid, is a major
problem  in  ecological  studies  since
under nonselective environments where
the trait borne on the plasmid need  not
be expressed, the  antibiotic-resistant
populations  have  been  observed  to
disappear as they are out competed by
the native populations. Because of  the
importance  of  genetic  markers  , as
selectable traits  for tracking/detecting
genetically engineered microorganisms
(GEMs),   measuring   their   stable
expression or maintenance in the host
is a prerequisite for studies prior to field
releases.
  We present here results of our study
in   which   a   comparison  of   the
expression  and  stability  of  genetic
markers, and assay systems is made for
bacteria proposed for release  into  a
microcosm.   Results  of   our   study
suggest that the xy/E marker gene is
stably expressed in some  strains  but
not in others. Expression of xy/E gene
was or approached 100% in all colonies
of the seven strains when  grown  on
selective media. Except  for  E.  coli
W311DHRalpha   (PSEL2-4)  and   P.
aeruginosaPA4-032 (pRO1940::xy/E) all
strains which carried the xy/E gene on
a plasmid  proved highly unstable with
respect to the simultaneous expression
of the  xy/E  and  antibiotic  resistant
phenotypes. Such instability could be a
function     of   the   frequency   of
segregation  of plasmid-free cells  and
the difference in growth  rate between
plasmid-free  and   plasmid-containing
cells.   In   contrast,   chromosomally
inserted marker genes, such as the xy/E
gene   in  the  haploid   (P.  syringae
Cit7#69) and merodiploid  (P. syringae
Cit7#17)   colonies,   consistently
maintained   both   the  xy/E   and
associated   antibiotic   marker  gene
phenotype,  on  various selective and
nonselective growth media.
  The  activity  of the  2,3-catechol
dioxygenase  depends  on   the  cell
density and also on the growth phase.
The activity of the enzyme was optimal
when the cell concentration was greater
than 108 CFU/ml and the cultures were
in  the  stationary  phase.  It  was  also
observed that 2,3-catecholdioxygenase
activity  was strain  specific  and its
activity decreased in the late stationary
phase  of the  culture. This  loss of
activity  may be  a  function of the
depletion of  iron which is  known to
affect  the  specific  activity  of the
enzyme.
  The   spectrophotometric   limit  of
detection  observed    in  this  study
indicates  that  a  sample  needs to
contain  a population  density greater
than 107 CFU/ml of the  marked bacteria
for  direct   enumeration   without
cultivation on plates. This condition is
unlikely to occur in soil or in lake water.
Even under  laboratory  conditions and
with the use of a strain with vigorous
catabolic activity of catechol such as in
the merodiploid, the  limit  of detection
of the  assay system   could  not be
improved.  Additional  limitation noted
was the  instability  of  the  enzyme
product,  2-hydroxymuconic   semi-
aldehyde. Where  large numbers of
samples are  involved, the short half life
characteristic of the  product limits its
application.
  Although   catechol   dioxygenase
activity   could   not   be   detected
spectrophotometrically  at whole cell
                                     59

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 concentrations below  107   CFU/ml,
 HPLC  analysis revealed that activity
 was detectable with cell density as low
 as 103 CFU/ml. The sensitivity attained
 via HPLC analysis is comparable to the
 spectrophotometric detection limit (103
 cells ml"1 } observed with cell extracts
 (supernatant of sonicated cells) of filter
 concentrated   lake   water   samples
 reported by others.
   Of  the  seven strains studied, five
 carried the  xy/E gene on a plasmid.  Of
 the five assayed for expression and
 stability of  the xy/E  gene, only one
 consistently expressed  both  xy/E and
 antibiotic  resistance  phenotype.  All
 constructs   bearing   chromosomally-
 inserted  xy/E  gene   and  antibiotic
 markers  expressed   catechol   2,3-
 dioxygenase activity and resistance to
 antibiotics. The merodiploid (P. syringae
 Cit7#17) showed  superior  catabolic
 activity  of the  substrate and produced
 the  most   2-hydroxymuconic semi-
 aldehyde.   The   apparent   hyper-
 expression  of xy/E gene in this strain
 facilitated HPLC mediated detection  of
 very low  concentrations  of  marked
 bacteria. Although our study suggested
 that the xy/E-gene  tagger system has
 limitations for spectral assay of whole
 cells (107  cells/ml),  it also  demon-
 strated that the xy/E gene assay is very
 useful for  plate assays  and is highly
 sensitive (103 cells/ml) for detection of
 merodiploid  xy/E  activity  by  HPLC
 analysis. Our study also  confirmed the
 viability of the assay system as a stable
 and sensitive  analytical tool for  the
 study  of anthropogenic  strains.  The
 high degree of sensitivity afforded  by
 HPLC analysis presents an opportunity
 useful in the autecological investigation
of released microbes.
 FUTURE WORK

   Future work will include a) increasing
 the sensitivity of the assay system by
 concentrating  and  stabilizing   the
 product,  b)  determining the  factors
 responsible   for   the   differential
 expression of xy/E gene in the  haploid
 versus the merodiploid, and c)  testing
 the  assay  system  in  a  microcosm
 setting using plant, lake water or soil.

 PUBLICATIONS

  This study was initiated this year and
there are no publications at this time.
                                      60

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      POTPOURRI OF BIOAEROSOL RESEARCH AT ERL-CORVALLIS IN 1990
           Bruce Lighthart1, Brenda T. Shaffer2, and Balkumar Marthi2
                  U.S.Environmental Protection Agency1, and
                   ManTech Environmental Technology Inc.2,
                      Environmental Research Laboratory
                              Corvallis, Oregon
INTRODUCTION

  Viable   microbial   bioaerosols
generated by many human (and natural)
activities   are   dispersed    by   the
atmosphere to  unwanted  downwind
locations.  In order to estimate the near
source downwind risk associated with
the deposition  of  those  viable  bio-
aerosols,   the   following  areas  of
research are being pursued:  increasing
bioaerosol   sampling  efficiency,
developing better tools to  evaluate
bioaerosol  generation  potential,  and
developing predictivesimulation models
of bioaerosol  dispersal and deposition.
The models act as integrators of new
information generated with better tools
and sampler efficiencies.
  The remainder of this report will
describe the progress in the past year
in:  (1)  increased bioaerosol  sampling
efficiency,  (2) tool development of an
automated  dynamic  aerosol torroid
(auto-DAT),  (3)  development of  an
Environmental   Bioaerosol    Research
Chamber (EBARC), and (4) bioaerosol
simulation models.   In the following,
bioaerosol specifically means bacterial
bioaerosols.

PROGRESS DESCRIPTIONS

Bioaerosol Sampling

  The atmospheric environment is more
or less inhospitable for maintenance of
viability in bioaerosols.   However, at
least some organisms in bioaerosols are
only partly damaged while airborne and
will not grow when cultured,  although
they  are  viable.    In  an  effort to
resuscitate some bioaerosols, betaine
has  been  added  to  culture  media
increasing sampling efficiency in terms
of viable counts by up to 30% (Marthi
and Lighthart,  1990)
  Catalase is  another compound that
has been used to resuscitate stressed
bacteria. When we add catalase to our
bioaerosot recovery media for both pure
cultures  and  wastewater  treatment
plant bioaerosols, sampling  efficiency
increased up to 113% (Table  1).
        Table 1. Resuscitation of aerosolized Pseudqmonas svringae or of wastewater
        treatment plant (WWTRP) bacteria in 1000 units of catalase containing collection fluid
        (as percent of 0 min. non-catalase control).
Catalase Contact
Time (minutes)
A. WWTP
0
30
60
B, Ps. syringae
0
30
60
Catalase
4

0.0
98.4
112.9

0.0
110.9
112.1
-

0.0
10.1
8.2

0,0
-8.5
•28.0

-------
  A  speculative  explanation  of the
catalase resuscitation mechanism is as
follows:

1.    Many  bacteria  produce  toxic
      hydrogen peroxide.

2.    Some   bacteria   produce
      membrane associated catalase to
      dissipate hydrogen peroxide.

3.    On  aerosolization, the  bacterial
      membrane is disrupted affecting
      catalase function, peroxide ac-
      cumulates,  and nonculturability
      occurs.

4.    Exogenous  catalase  penetrates
      damaged cells, catalyzes

      dissipation  of  toxic hydrogen
      peroxide,  and  subsequent
      culturability occurs.
 5.    Upon  addition of  betaine  (an
      osmoprotectant)   to   aerosol
      suspension  fluid,  the catalase
      effect is reduced, indicating that
      the   betaine   may   negate
      membrane   disruption   and
      consequent need for exogenous
      catalase.

Tool Development:  Auto-DAT

  Large, slowly rotating airtight drums
have been used to evaluate the survival
of airborne  microbes (Figure  1).  .In.
order to increase the  precision  of the
drum system, we have automated it
using a microcomputer. The auto-DAT
has just been used for  the  first time
with some of the resuscitation experi-
ments above. A protocol describing the
construction of the auto-DAT audits use
to evaluate the survival of bioaerosol is
forthcoming.
                                              Drum
                                          Bioaerosol
                                          Input/Output
           Figure 1.  An Automated Dynamic Aerosol Torroid (Auto-DAT).

                                    62

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Tool Development:  EBARC

  In an effort to determine some of the
conditions that would lead to bioaerosol
entrainment   from   plant   surfaces
sprayed with microbial suspensions, the
EBARC was designed to give a  set of
plants  from one to many puffs of air.  It
is  thought that the accelerated force
(drag  and thrust)   occurring at  the
leading edge of a  puff wind field would
be  the most  effective  mechanism to
force  microbial  release  from  plant
surfaces.   Figure 2 (next page) is  a
diagram  of  the EBARC  showing  the
rotating "lazy susan" with plants
passing through  influent air streams
emitted from the influent air manifold.
Samples downwind  from  the ports
collect the entrained bioaerosol.
  To date, we have used the EBARC to
evaluate   the   variation   between
Andersen samplers (Table 2)  and the
particle size distribution of bacteria size
fluorescent spheres sprayed onto plants
and subsequently blown off (Figure 3).
  Preliminary  data  in  Table  2 shows
that  there  are significant differences
between Andersen  sampler   stages.
Figure  3 (page 65) shows that many
bacteria-sized  particles  are   released
from plants on larger rafting  particles.
This  is also  found  in nature,  where
approximately 60% of the bacteria are
particles of  _>.  7  //m  aerodynamic
diameter.
   Table 2.  Comparison of 8 randomly selected Andersen samplers used in the EBARC to evaluate
   BACILLUS SUBTILIS var. NIGER spores blown off oat leaves.
Andersen
Sampler
1*
2
3
4
5
6
7
8
Average
(CV)
6-
(0.65 Mm)
1.0
2.6
9.9
5.1
3.2
1.1
9.3
1.0
4.2+1.0
(23.8)
Stage Number-(Particle
5- 4-
(I'.Owm) (2.1 Mm)
16.2
14.3
5.0
12.6
13.1
13.8
10.8
15.3
12.6 + 0.9
(7.1)
0.4
11.8
10,3
7.4
10.7
11.0
10,4
9,7
9.0 + 0.5
(5.5)
Size), Percent
3-
(3.3 Mm) (4
20.7
10.8
12.3
8.8
10.1
9.7
13.7
9.5
12.0 + 0.6 8.
(5,0)
Total
2-
.7 Mm)
6.8
8.5
6.7
9.4
5.6
7.1
11.8
8.3
0 + 0.4
(5.0)
1-
(7.0 Mm)
54.8
51.9
55.7
56.7
57.3
57.3
44.0
56.2
54.3+1.5
(2.8)
Total Colonies
(Log 10)
2.97 + 0.04
2.78 + 0.15
2,84 + 0.14
3.04 + 0.05
2,96 + 0.04
3.01 +0,04
2.96 + 0.04
2.97+0.05
2.95 + 0.03
(1.0)
        * 10 Replicates per sampler
                                      63

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                                      Roof
                                                                      Roof
              in
                       Influent
                       air i
                                          Sampler         Sampler

                                                 Stationary
                                         ,  Rotating T platform

                                                 Side view
Figure 2.  Environmental Bio-Aerosol Research Chamber (EBARG).
                                         64

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          ea
          I
          •s
          ^
              100

              90 -

              80 -

              70

              60

              50

              40

              30

              20

              10
                                                      1 pro spheres
                                                      6
                                   Particle Size (pin)
Figure 3. Comparison of the particle size distribution of Bacillus subtilis var. niger spores and 1//m
fluorescent microspheres blown off oat plants after multiple wind puff treatments using the EBARC.
                     General  Survival
Simulation  Models:
Model
  Using  DAT determined death  rate
values from ca. 70 literature cited cases
involving 17 bacterial species in which
gram  reaction,   relative  humidity,
temperature, and aerosol age were the
experimental variables  (Figure 4b),  a
regression  model  was  fit having R2-
value 0.94 (Figure  4a)(Lighthart, ,B.,
1989, Aerobiol.  5:138-144). Death
Rate  Constant (log^g)  =  Constant -
Log10 (Aerosol Age) "+ temperature -
Gram  Reaction   *   RH.     A;   less
demanding model with an R2  if  0.90
was fit to: Death Rate Constant (Iog10)
= Constant - Log10 (Aerosol Age)
  These  models may be  inserted  into
the dispersion model to give  realistic
survival properties of the bioaerosol as
a  function  of  temperature,   relative
humidity, gram reaction over time in the
atmosphere.
Simulation Models: Trajectory Model

  In an effort to obtain a more realistic
characterization how a  polydispersed
bioaerosol  might   wander  in  the
atmosphere,  the trajectory  of various
size  droplets   containing  various
numbers   of   microorganisms   in
atmospheres   of   different  relative
humidity,   temperatures,  and   wind
speeds  was, prepared (Lighthart  et al.,
in press).  The results of this effort are
shown in Figure 5a (simulated) where it
is seen that  droplets  of  .<.  80  fj
diameter in a 50% relative humidity and
20  °C  atmosphere, evaporate to the
bacterial residue of about 1  jjm before
they  can  fall  to  the  ground  and
therefore remain airborne becoming part
of the  aeroplankton.   Those greater
then  80   /ym   fall  to  the  ground.
Observations   confirm   the   model
(Figures 5b,c observed).
                                      65

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Figure 4.  a) Response surfaces for aerosol age, evaporation function and bacterial
death rate constants for Gram positive (lower surface) and Gram negative (upper
surface) bacteria using the general death rate equation model.  The two surfaces
result to be not parallel; b) enlarged picture of insert shown in fig. 4A.
                                       66

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                              Simulated
                              if. it, it, n, H > M in '.si
                                               Wl.ll
                                               11(1.«
                                               Ml',ii
                                   MO.II.
I        I
     Sourc* PROXIMAL
                              Observed
                                                        iM («*>
                                                    8ourc» DISTAL
Figure 5. Trajectory model
                                   67

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Simulation Models:  Deposition Model

  A bioaerosol dispersion model  has
been developed and crudely compared
with observations made at the Tulelake,
CA, ice-minus bacterium   spray-out
(Lighthart and Kim, 1989). recently, a
controlled spray of bacterial spores has
been carried out by the U.S. Army at
Dugway Proving Ground to provide
data to quantitatively evaluate reliability
of the bioaerosol dispersion model with
a good set of observations.  Presently,
the model has  been  run  using  the
observed  meteorological data (Figure
6);  the statistical comparison between
simulated  and   observed   will   be
completed in the near future.

FUTURE RESEARCH

* Evaluate  the   effects  of  genetic
  construct  on   airborne   microbial
  survival.

* Evaluate the  airborne  survival  of
  insect pathogens including MPCAs.

* Prepare a protocol  to evaluate the
  survival of airborne microorganisms.

* Modify  the  bioaerosol   dispersion
  model for use in a microcomputer.
REFERENCES

Marthi,  B., and B. Lighthart.   1990.
Effects  of betaine on enumeration  of
airborne bacteria.    Appl.  Environ.
Microbiol. 56(5):1286-1289,

Lighthart, B. 1989. A statistical model
of laboratory death rate measurements
for airborne bacteria. Aerobiol. 5:138-
144.

Lighthart, B., B.T. Shaffer, B.  Marthi,
and L. Ganio.   In press. Trajectory  of
aerosol   droplets  from   a  sprayed
bacterial suspension.   Appl.  Environ.
Microbiol. 57(4).

Lighthart, B.,   and J. Kim.    1989.
Simulation of airborne microbial droplet
transport.  Appl.  Environ. Microbiol.
55(9):2349-2355.
                                     68

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                                    69

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      MEASURING ENTRAPMENT OF BACTERIA FROM THE PHYLLOSPHERE

          Michael Walter1, Valerie Fieland1, Lisa Ganio1, and Ray Seidler2
                      ManTech Environmental Technology1
                     U.S. Environmental Protection Agency2
                       Environmental Research Laboratory
                               Corvallis, Oregon
 INTRODUCTION
   Monitoring the dispersal patterns of
 genetically engineered microorganisms
 (GEMs)  from  a  release site  is  an
 important aspect of understanding what
 impact   GEMs  may  have  on   the
 environment. The possibility exists that
 after application, GEMs  could become
 resuspended (entrained) from soil  or
 leaf   surfaces   and   transported
 downwind  to colonize other locations.
   Plant canopies as well as soil have
 been shown to be sources of entrained
 bacteria.  The   number  of  bacteria
 entrained is influenced by the season of
 the year, moisture content of the soil,
 and type of plant canopy. In addition,
 activities such as harvesting have been
 known  to  influence  the number of
 entrained bacteria detected.
   Measurements taken on days 1,2 and
 4   following   the   application   of
 recombinant strains of  Pseudomonas
 indicated that low numbers of the GEM
 were   being   entrained   into   the
 atmosphere. The number of entrained
 bacteria  decreased  with time  after
 application.   In   addition,  entrained
 bacteria  were  detected  primarily  on
 gravity settling plates (GSPs), which are
 150   mm   petri   dishes containing
 selective agar.  Mechanical air samplers
that proved very sensitive during the
actual applications of the GEM  either
failed  to detect  entrained GEMs  or
 detected very few of them compared to
 the GSPs.
   It has been demonstrated that under
 greenhouse conditions, bacteria can be
 injured during, aerosolization and  that
 this injury can adversely effect their
 subsequent survival.   Entrainment of
 bacteria  results  in  their  resuspension
 into the atmosphere,  therefore it  is
 possible that this process could cause
 injury or death to GEMs after release, or
 render them incapable of surviving in
 competition   with    indigenous
 microorganisms.
   The objectives of  this study were to:
 A)  develop a  method of artificially
 inducing bacterial entrainment from the
 phyllosphere,  B) compare  sampling
 efficiencies of GSPs, all glass impingers
 (AGIs)  and  Andersen  samplers  to
 determine which method of sampling
 would be most sensitive in  detecting
 entrained  bacteria,  C)  compare  the
 magnitude of entrainmentfrom different
 species  of  host   plants,   and,   D)
 determine if the viability of bacteria is
 affected during entrainment.

 METHODS

 Media and Cultures

  All  experiments  were conducted
 using  a   nonrecombinant  strain  of
Pseudomonas syringae  TLP2, resistant
to  100  //g/ml  of  rifampicin  (RIF).
                                     70

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Several studies were conducted using
spores of Bacillus subtilis var. niger.  P.
syringae were cultured and enumerated
using 100//g/ml of RIF in Luria-Bertani
broth and agar (LB-RIF) respectively.  B.
subtilis  spores  were obtained from
spread plates on casitone agar (CAS)
incubated  at 30°C for  seven  days.
Spores were recovered by scraping 10
plates with 10  ml of sterile distilled
water and  suspending them in 100 ml
of distilled  water.

Plant Application and Sampling

  P.  syringae were applied to three-
week-old  beans  (Phaseolus vulgaris
humilus cv. Bush  Blue Lake) and three-
week-old oats  (Avena  sativa  L. cv.
Cayuse)  planted in 61 X  30 X 13 cm.
pulp  trays  grown  in a greenhouse.
Plants were sprayed until run-off with
twice washed  P.  syringae  following
incubation  for 18  hours  at 30°C.   A
population  of 1 x  108/ml was applied
using  a   CO2   backpack   sprayer
pressurized to 36 psi and equipped with
a Tee-Jet  nozzle  #8004-SS.   Plants
were allowed to dry a minimum of two
hours before sampling  was  initiated.
Bacterial  populations were determined
by randomly sampling three bean leaves
or five oat  stalks.  Leaves were diluted
with 20  ml of  phosphate buffer and
blended  for one min. in  a  stomacher
blender.  Samples were serially diluted
and  plated on  selective media  and
incubated  for  48  hours  at  30°C.
Counts  were standardized on  a per
gram basis.  Spore populations were
determined by heating at 80°C for 20
min.  to   heat-shock   spores  into
germination and kill undesired bacteria.
Samples  were filtered using 0.45 /vm
 filters  and filters placed on CAS agar
 plates.

 Aerosol Samplers

   Six-stage   Andersen   samplers
 containing six agar plates filled with 20
 ml of  LB RIF were  operated  at 28.3
 L/min. The AGIs were loaded  with 20
 ml of sterile 10 mM phosphate buffer
 and run  at  12.5 L/min.   Following
 sampling,  the  buffer   was  filtered
 through  37  mm diameter, 0.45 //m
 cellulosic filters, then transferred to the
 appropriate agar. GSPs used  in  these
 experiments consisted of 150 mm petri
 dishes filled with 75 ml of LB-RIF agar.
 Plates  were uncovered  immediately
 prior   to   sampling  and   covered
 immediately after sampling events.  All
 plates  were incubated for 48 hours  at
 30°C and colonies counted.

 Bacterial Entrainment

   Bacterial entrainment was induced by
 placing a 20 inch  electric fan 0.5  m
 from sprayed plants. Air flow  through
 the plants was  measured at 4 m/sec
 and entrainment was measured for 10
 min. In all experiments except  sampler
 efficiency, plants were mounted on two
 9 cm2 wood blocks. Experiments were
 conducted in a 10 X 30 m greenhouse.

 Sampler Efficiency

  A two-level 122 X 183 cm. platform
 was constructed to adjust the  intakes
of  the three samplers  to the same
 height. The upper level of the platform
consisted of three parallel rows  of three
circular holes located 0.25, 0.6,  and
 1.0 m  from sprayed  plants. The AGI
                                     71

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and Andersen samplers were placed on
the lower level of the  platform with
intakes  protruding through  the holes
level with the  height  of  the  gravity
settling   plates.     Entrainment  was
measured at 2, 24, and  120 hours after
application of the P. syringae using a
separate set of  plants for each time
point.   Results were compared using
Iog10 transformations of the number of
CPU observed on a per sampler basis.

Factors  Affecting Bacterial Entrainment

  Eight  AGIs or two Andersen samplers
were placed  at 0.25, 1.0, 2.0, and 3.0
m from  inoculated plants.  AGIs were
mounted in two groups of 4, at 26 cm.
and  53  cm. above the ground, each
group of four separated  by 35  cm.
Andersen samplers were placed 35 cm.
apart on the soil surface at each of the
four distances.

Effect of Host Plant

  The   effect  of  host  plant   was
measured by comparing the magnitude
of  bacterial entrainment  from  beans
with that of oats.  Experiments were
conducted to  measure total bacterial
populations  being entrained from each
plant using  AGIs,  and difference  in
particle  size   distributions   were
measured with Andersen samplers.

Dilution vs Die-off

  To   determine   if   decreases   in
entrained bacterial counts  were due to
dilution  or loss of viability, the number
of entrained vegetative bacteria were
compared with the number of entrained
spores of B. subtilis.  Decreases in the
spore populations were due to dilution,
while  decreases in  the  vegetative
bacterial populations were due to loss
of viability.

Time After Application

  The effect of  time after application
was measured by sampling bean plants
sprayed with P. syringae at 2, 24, and
120 hours after application using only
AGIs.  In  these experiments, all four
distances were sampled simultaneously,
since the aim of the experiment was to
detect the total number being entrained.
The number of bacteria entrained were
compared  over time and related to the
population of bacteria found on the leaf
surface.

RESULTS AND DISCUSSION

Sampler Efficiency

  In general Andersen samplers, AGIs
and GSPs  had  the same pattern  of
response throughout the  experiments.
In  all  cases, there  were  significant
effects  due  to  both   time   after
application  and distance  from  the
sprayed plants. On a per  volume basis
(CFU/m3),  there was no  difference
between  the  number  of  entrained
bacteria detected in AGI and Andersen
samplers.    AGIs exhibited the  most
variable data over both  distance and
time.  However, variability tended to
increase for all three samplers as the
distance from the sprayed plants and
time  after  inoculation increased.  No
entrained  bacteria  were  detected  on
GSPs beyond 1 m from plants.
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 Factors Affecting Bacterial Entrainment

   Over a distance of 3 m from sprayed
 plants,   both   AGI  and   Andersen
 samplers  exhibited  similar  dispersal
 patterns  of  entrained bacteria.  Both
 samplers detected substantial numbers
 of  bacteria  3   m    from  plants.
 Significantly  higher   numbers  of
 entrained bacteria were  detected 0.25
 m from plants than at the other three
 sampling distances.  There was  no
 significant difference in the number of
 entrained bacteria detected  1 to 3 m
 from plants.
   Graphical  examination of the data
 collected  from  AGI  and  Andersen
 sampler?  demonstrated   that   the
 distributions of data  were not the same.
 This difference indicates that the two
 populations  being  sampled  are  not
 equivalent, and rendered it impossible
 to make a direct comparison between
 sampler types. It appears that the AGI,
 which  is a  liquid  based  sampler, is
 sampling actual  numbers  of bacteria
 entrained, while the  Andersen sampler,
 which impacts bacteria directly onto an
 agar plate, is sampling the number of
 infective particles being entrained.

 Effect of Host Plant

   An average of 79% more P. syringae
 were entrained from beans than from
 oats.  For each distance sampled,  the
 number of  entrained  bacteria  from
 beans was significantly higher than  the
 number of bacteria detected from oats.
, A significantly higher population  of P.
 syringae were detected on  bean leaves
 as compared with oat leaves.  If the
 number of bacteria entrained from each
 plant is compared as a frequency of the
 relative population  on  the plant, then
 the amount of entrainment was actually
 higher from oat plants than bean plants.
 Bacteria entrained from oats followed
 the  same  dispersal pattern as  from
 beans  but  with  significantly  higher
 numbers of entrained bacteria detected
 at 0.25 m from plants than from 1 to 3
 m. Again, no difference was found in
 the number of bacteria detected 1-3 m
 from  plants.  There was a significantly
 different particle size distribution from
 each  plant.    The  size  of particles
 entrained from bean plants tended to
 decrease  in size over distance while
 those from oats  tended  to  have  a
 particle size of 7 //m or greater.

 Dilutions vs. Loss of Viable Count

   The decrease in entrained spores was
 significantly greater than the decrease
 in vegetative P. syringae.  At 0.25 m,
 virtually the same number of spores and
 vegetative   cells   were   detected.
 However, as the distance increased, the
 number  of  vegetative  P.  syringae
 decreased  below  the  number of  B.
 subtf/is.  This indicates that differential
 loss of viable count was occurring. By
 dividing the number of  vegetative cells
 by the  number of spores detected at
 each  distance,  it  was   possible  to
 generate a survival ratio over distance.
 If  the  number  of vegetative  cells
 decreased  relative to the  number  of
 spores at each distance, then the ratio
 would become smaller over distance,
 which  is  what   was   observed.
Therefore,  loss of  viable  count  is
 occurring beyond a distance of 0.25 m
 from plants.
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Time After Application
  Significantly   more  bacteria  were
entrained 2 hours after application than
24  and  120 hours  after application.
However,  the  number  of  bacteria
entrained 24  hours after application
was not  significantly different than the
number  entrained  120  hours  after
application.   Bacterial  populations on
plants reflected  the decline  in  the
number  of  bacteria  entrained  to a
limited extent. The number of bacteria
detected 2 hours after application was
significantly higher than  the number
detected  at 24  hours,  and   these
populations  were significantly  higher
than those detected after 120  hours.
However,  while  plant  populations
continued to decline from 24 to 120
hours, the numbers  of  bacteria  being
entrained did not.
  Results  from  these   experiments
suggest  that: A) GEMs are entrained
following leaf applications in response
to moderate amounts of wind,  B)  the
type of sampler used to detect bacterial
entrapment must be based on the type
of data desired, C) the magnitude of
bacterial entrainment may be dependent
on the host plant, D) the process of
entrainment may cause death or  injury
to vegetative bacteria,  and  E)  the
magnitude of bacterial entrainment may
not decrease over time.
  Future research should  be directed
toward using this method to  measure
the colonization  of plant  surfaces by
entrained   bacteria,    identifying
environmental   conditions    most
influential on bacterial entrainment, and
conducting field experiments to validate
the greenhouse data.
PUBLICATIONS

M.V. Walter, V.J. Prince, B. Marthi, B.
Shaffer,  L.  Ganio,  and  R.J.  Seidler.
1990.   Measuring aerosol survival of
sprayed bacteria. Review of progress
in the  biotechnology-microbial  pest
control agent risk assessment program.
USEPA   Office   of  Research   and
Development, November 5-9, Corvallis,
OR.

M.V. Walter, V.  Prince,  L. Ganio, and
R.J.  Seidler.  1990.  A method to
measure bacterial entrainment from the
phyllosphere.    Abstract.     Annual
meeting of the  American Society for
Microbiology  May  13-17, Anaheim,
CA.

M.V. Walter, V.J. Fieland, L.M. Ganio,
and  R.J.  Seidler.   1991.     Factors
affecting bacterial entrainment from the
phyllosphere.  Abstract.  Annual EPA
Biotechnology   All   Investigators
Meeting. April 8-11,  Arlington, VA.

M.V. Walter, V.J. Fieland, L.M. Ganio,
and R.J. Seidler.  1991.  Measuring
entrainment  of   bacteria from  the
phyllosphere. Manuscript submitted to
Applied and Environmental Microbiology
3/91.
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           UPDATE ON THE ASSESSMENT OF BACTERIAL DISPERSAL
                           FROM FOLIAGE BY RAIN
                     H. A. McCartney and Julie Butterworth
                  A.F.R.C. Institute of Arable Crops Research,
                       Rothamsted Experimental Station,
                           Harpenden, Hertsfordshire
                               United Kingdom
INTRODUCTION
  Recent   advances   in   genetic
engineering  techniques  have   made
possible the modification of bacteria for
use as novel pest and disease control
agents in crop plants, or as potential
replacements  for  chemicals  currently
used in agriculture.  However,  before
such  agents  are released   into the
environment, possibly on a large scale,
it is important to know if they are likely
to have  deleterious effects  on non-
target organisms or plants.
  The release of genetically engineered
microorganisms   requires   their
containment within the area of release,
and  thus  an  assessment   of  their
potential   for  spread  is   of   great
importance. Rain splash as a factor in
the spread of disease in crops has long
been recognized and it has been shown
that  it can  play  a major part  in the
spread of many  fungal and  bacterial
plant  pathogens.  However, little  is
known of  the efficiency of  rain  in
removing and  dispersing bacteria from
leaf surfaces.  A project to study the
potential  of rain  splash for  removing
and  distributing   bacteria  from  the
foliage of crop plants is  underway  at
the Institute of Arable Crops Research,
Rothamsted Experimental Station.
METHODS

  All experiments were done  in  the
Rain  tower/wind  tunnel  facility  at
Rothamsted. Mono-sized water drops
(simulating rain drops) were allowed to
fall  11m  onto  target leaves carrying
populations of bacteria.   Rain drops of
between 2 and 5mm diameter could be
produced.    Droplets,  dispersed   by
splash,   were  collected  at  different
distances from the targets  and their
bacterial  content  assessed.    The
bacterial  content of  water  which
collected on, and subsequently ran off,
the leaves was also assessed.
  Isolates  of Pseudomonas syringae,
Klebsiella planticola and Bacillus subtilis
var.  niger,  all resistant to  100//g/ml of
the antibiotic rifampicin  were used in
these experiments.
  Two  species of glasshouse  grown
crop  plants  were used:  Phaseolus
vulgaris cv. Prince, and Brassica napus
cv.  Cobra.  The  plants  were chosen
principally because of their different leaf
surface characteristics: P. vulgaris has
a hairy surface and no pronounced wax
cuticle,  while  B.  napus has a waxy
cuticle, and is not hairy.
  Bacteria  were  grown  overnight  in
nutrient broth (Oxoid) at 30°C to a
concentration of between 107 and 108
CFU/ml.    The  target   plants were
sprayed,  inside  a  closed  perspex
                                     75

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chamber,  with  the  nutrient  broth and
left for about 22  hours to  allow the
bacterial population to stabilise.  The
plants were removed from the chamber
about 6 hours before  each experiment
to allow surface moisture to dry from
the leaves. Leaves were removed from
the sprayed  plants and  placed in an
overlapping layer on a horizontal nylon
mesh covered frame at the base of the
rain tower. The population of antibiotic
resistant bacteria on the target leaves
was assessed by taking a sample of
four leaves before exposing the rest to
splash.  Each sample leaf was weighed
and  placed  in  a  sterile  glass conical
flask containing 50ml  of sterile quarter
strength Ringer's solution (Oxoid) and
washed on a flask shaker for  1 hour.
Samples of the wash water were plated
out  on  selective  medium   (N + R)
consisting of nutrient agar (Oxoid) with
100^/g/ml  actidione to suppress fungal
growth, and  100//g/ml rifampicin as a
selective  agent  for  the  inoculated
bacteria. The plates were incubated at
30°C  overnight  and  the   bacterial
colonies counted.
  The  target leaves were exposed to
splash  from artificial rain drops for 30
minutes. Immediately after exposure, a
further  sample  of  four leaves was
removed and  washed to assess the
numbers   of   inoculated   bacteria
remaining  on the leaves. A  sample of
water from the rain generator was also
plated  out onto N + R as a  check for
contamination by  antibiotic  resistant
organisms.
  Splash  droplets  were collected in
either sterile  9cm  plastic Petri  dishes
containing  20ml of N + R or in sterile
9cm polypropylene funnels supported in
sterile  glass  universals.   The Petri
dishes  were  used  to  estimate the
numbers of bacteria-carrying droplets
splashed and the funnels to estimate
the total  number  of cells dispersed.
The collecting vessels were supported
at the same height as the leaves, at  8,
24, 40,  56, 72 and 88cm from the
edge  of  the target area.   During an
experiment the two Petri dishes closest
to the target were replaced with fresh
plates at three minute intervals and the
third closest plate was  replaced after
15 minutes.  Four pre-weighed funnel/
universal  assemblies were  arranged
underneath  the leaf  support frame  to
collect samples of the water running off
the leaves.   Run off water was  also
collected in  plastic trays placed under
the target.  The sum of  the volume  of
water  collected  in   the  trays  and
funnel/universal assemblies was taken
as the total amount of run off water.
   The Petri dish droplet  samplers were
incubated at 3Q°C overnight and the
colonies growing on the plates counted.
The  droplet collecting  funnels  were
rinsed with  a known volume of sterile
distilled water which  was added to the
collected  water  and samples of the
resultant  bacterial suspension  plated
out onto  N + R medium.  Samples  of
run-off   water   were   diluted   as
appropriate  and also plated out onto
N + R.   The  plates  were  incubated
overnight   at  30°C  and   colonies
counted.

RESULTS

  Twenty-five experiments were done
using the six different combinations  of
two  plant species, P. vulgaris and  B.
napus  and  three bacteria species  P.
syringae, K. planticola and B. subtilis.
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For all experiments the rain drop size
was 3mm and  air  temperature  was
about  18°C with about 85% relative
humidity.  The effect of rain drop size
was studied  in a  further 24 tests.
Three  different  rain drop sizes were
used: 2.4, 2.9 and  4.7mm diameter.
Eight experiments were done with each
drop size using B. subti/is on B. napus
as the target.  Experiments were done
at  about  21  °C and   75%  relative
humidity.

Proportion   of  Bacterial  Populations
Removed by Splash

  There was  a  large variation in the
estimate of the  proportion of  bacteria
removed,  by   splash   for   replicate
experiments   and   no   statistical
differences were found either between
bacteria  species or between  plants.
The  variation  in  bacterial  numbers
between  individual  leaves,  used  to.
assess pre-  and post-splash  bacterial
populations,   was  often  large  and
frequently the differences between pre-
and post-splash bacterial numbers were
not statistically significant.  However,
the results suggest that up to  90%  of
bacteria may be washed off the leaves.
Surprisingly, there was little effect  of
drop size  in removing B. subtilis cells
from B. napus  leaves. It was estimated
that, for all drop sizes,  about  75%  of
the bacteria were removed during each
experiment.

Bacteria Removed bv Run-off

  In   all   experiments  substantial
numbers of bacteria were found in the
run-off  water.   In  the first set  of
experiments the numbers of  bacteria
recovered per unit area of target was
frequently (70% of experiments) greater
than the estimated  population density
at  the  start   of   the  experiment,
suggesting  that  the  method of leaf
washing may have underestimated the
initial  populations.    There  was no
significant   difference  between  the
proportion  of bacteria  in the run-off
water (Pr) for the two plant species and
the same organism.  However, Pr for P.
syringae and K. planticola, was nearly
always greater than for B. subtilis.  In
the second  series of  experiments the
value of Pr increased with drop size
(mean  values of 0.27, 0.44 and 0.84
for  2.4,  2.9  and   4.7mm   drops
respectively).

Dispersal by Splash

  The  numbers of inoculated bacteria
recovered on exposed agar  plates  or
from the funnel assemblies decreased
rapidly  with distance from the target
leaves.    The   number  of  bacteria
deposited per square centimetre, D,
decreased exponentially with distance
from the target: D = A exp(-ax) where
A, a are constants. The equation has
the property that D decreases by half at
regular  distances, d1/2  (  = 0.693/a),
from the source.  The half  distance,
dy2, is  an easily visualised measure of
the deposition gradient.
  Deposition gradients for both plates
and funnels for all tests were steep: dy2
ranged  between about 5 and 17 cm.
For a given organism the values of d,/2
for funnels were similar for each plant
species: for  P.  syringae  and  K.
planticola the average value of dy2 was
about  7cm  for B. napus but slightly
larger (»9cm) for P.  vulgaris.  Because
                                     77

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the numbers of B. subtilis cells caught
in the funnels were small at distances
greater  than  about  20cm from  the
target dy2 values were unreliable. The
values of dy2 for plates were similar
(di^«7cm)  for K. planticola  and  B.
subtilis  inoculated leaves,  but, for P.
syringae  on  both   plant   species
deposition gradients  were significantly
shallower (di/2=12cm). For both funnels
and   plates  mean   (of  8   replicate
experiments) d i/2 values were largest for
4.7mm  rain drops and smallest for the
2.4mm  drops: 8.2, 7.9 and 5.1cm for
the funnels  and 10.3, 7.5 and 7.2cm
for the plates for 4.7, 2.9 and 2.4mm
rain drops respectively.

Proportion Dispersed by Splash

  The  total  numbers   of   bacteria
dispersed  in.  splash  droplets   per
centimetre width  of the target  was
estimated by numerically integrating the
deposit  with distance from the edge of
the target.  The numbers of  bacteria
dispersed  depended  on  the  initial
population  present on the leaves.  In
nearly  all  cases,  (80%),   it  was
estimated that less  than 5% of  the
initial inoculated bacterial population
was  splashed. The average fraction of
bacteria  splashed was  about  1.5%
(range 0.02 - 8%). There appeared to
be   little   systematic  difference  in
proportions  splashed  between either
bacteria  species  or  plant   species,
except for B. subtilis where the mean
values for B. napus  were significantly
larger   than   for  P.   vulgaris  (2%
compared to 0.3%).  In the second set
of experiments a greater proportion of
B. subtilis  cells  were splashed from
target leaves by large rain drops than
the  smaller  ones:  about  1.7%  for
4.9mm drops compared  to 0.5 and
0.2% for 2.9 and 2.4mm drops.
,  The number of cell carrying droplets
splashed was much smaller than  the
number of cells for both P. syringae and
K. p/antico./a: between 5 and 200 times
as many P. syringae cells and between
2  and  50  K.  planticola  cells were
splashed than droplets.  For B. subtilis
the  numbers of  droplets  and cells
splashed were similar.  This difference
between organisms was probably due
to differences in initial populations. The
initial populations  of both P. syringae
and  K.  planticola  were usually  larger
than those of B. subtilis.  The number
of cells carried in  each  splash droplet
will  depend  on  the leaf population
density:  larger densities  will  lead  to
more cells per drop.   Thus  at high
densities the number of cells splashed
will be  larger than the number of cell
carrying droplets splashed. As with the
cells larger rain  drops splashed more
bacteria  carrying  drops than smaller
ones.  About twice as  many  cells  as
bacteria  carrying   droplets   were
splashed for the three drop sizes.

Dispersal Rates with Time

  The pattern of dispersal with time, as
indicated by  the change in deposition
rate  on the plates nearest the  target,
was   substantially  different for th'e
experiments with P.  syringae than for
the other two organisms.   With  P.
syringae the deposition rates decreased
almost linearly with time and for both
plant species were reduced by about
20%. over the 30 min. exposure period.
Deposition  rates  of  the   other  two
organisms  decreased roughly linearly
                                      78

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with  time  when  splashed  from  P.
vulgaris plants:.rates were reduced by
about 70% during the experiments. For
B.  napus the decrease in  deposition
rates.followed a concave pattern  .with
rates at the end of the test only about
10%  of  those  at  the  beginning,
suggesting that the available bacteria
were washed off B. napus leaves more
readily.  There were also differences in
the changes in deposition rate with time
for the rain drop experiments.  For the
4.7mm  drops   deposition   rates,
decreased in  a similar manner to the
first set of experiments, except that,the
differences between beginning and end
rates  were   smaller;   about a  60%
reduction.  With the other drop sizes
deposition   rate   initially,  increased
reaching a maximum about  5 minutes
and 1-0. minutes from the start of ,the
experiment for the  2.9  and  2.4mm
droplets respectively.  Deposition rates
then decreased and at the  end of the
tests were about 45% of the maximum
value for the 2.9mm drops and about
60%'for,the 2.4mm drops.

DISCUSSION

   Our   experiments  show  that  rain
splash can be an efficient mechanism
for  removing   bacteria   from   leaf
surfaces. Patterns of dispersal and the
distances travelled by bacteria in splash
droplets were similar to  those which
have been reported for both fungal and
bacterial pathogens. Although we only
examined the effect of splash on the
removal of three species of bacteria
from  two  plant  species, the. results
suggest that  splash dispersal patterns
and splash processes may be similar for
a wide range of organisms and plants.
The proportions of the leaf populations
dispersed in splash droplets were small
(usually < 10%) as were the distances
travelled by  most of  the  bacteria.
However, large numbers of cells were
found  in the run-off  water  collected
from underneath the target area. Thus,
rain water running off leaves to the soil
around  crop  plants   could  be  an
important source of bacteria for further
spread through travel in ground water,
or by secondary splashing.
  Large rain drops appear to be more
effective in removing  and  splashing
bacteria from leaves than  small ones.
However,  bacteria were more quickly
removed by the large drops, suggesting
that  for longer periods of rainfall drop
size may not be critical for the removal
(but  not necessarily the  dispersal) of
bacteria from leaves. Indeed, when the
proportions of bacteria removed during
the experiments were  adjusted for the
volumes of rain falling on the target,
the small drops were just as efficient at
removing bacteria as the large ones.
  These experiments show that rainfall
has  the  potential to  remove  and
disperse bacteria  from leaf surfaces.
However, as studies of the dispersal of
fungal pathogens  show,  the splash
process is  complex and its effective-
ness depends  upon the nature of the
rainfall or irrigation  and on the nature of
the crop or vegetation canopy.  More
work is needed to answer  questions
such as how does crop structure effect
bacteria spread? Which rainfall events
favour the  dispersal of  bacteria? Are
natural bacteria dispersed in  a similar
manner to  inoculated bacteria? How
efficiently  do  bacterial  cells survive
dispersal? Wind may also have a role in
the  spread  of  bacteria  from  leaf
                                     79

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surfaces.  Thus,  an understanding  of
the effects of the combination of wind
and rain to the removal and spread  of
bacteria is needed before the potential
for dispersal of genetically manipulated
organisms can be quantified.

FUTURE WORK

  As   the   co-operative   agreement
terminates in July 1991 there is little
scope for expanding the studies much
further.   However,  experiments,  to
examine the influence of environmental
factors  such  as relative humidity  on
bacterial spread are planned.  We also
intend  to  examine the potential for
longer distance transport by very small
airborne droplets.  Funding to expand
these studies is  being sought  from
other sources.
PUBLICATIONS

Butterworth,  J. and  McCartney, H.A.
(1990) Dispersal of foliar bacteria by
rain  splash  (abstract).   Journal  of
Applied Bacteriology, 69, xix (meetings
supplement).

Butterworth,  J. and  McCartney, H.A.
The  dispersal of  bacteria from  leaf
surfaces by water splash.  Submitted to
Journal  of   Applied   Bacteriology
(January 1991).
                                    8.0.

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      GENETIC AND MOLECULAR ANALYSIS OF SURVIVAL MECHANISMS
                            OF PSEUDOMONADS

    Anne J. Anderson, Robin Buell, Jirasak Katsuwon, Chris Heck and Rob Zdor
                 Biology Department, Molecular Biology Program
                            Utah State University
                                Logan, Utah
INTRODUCTION

  Colonization of plant roots by certain
isolates of fluorescent pseudomonads
promotes plant growth and suppresses
pathogens.  These  beneficial  effects
have raised interest in the agricultural
value of crop inoculation using native or
genetically  engineered   strains  to
improve quality and yield. In order to
assess some of the potential hazards
associated with  release of  genetically
engineered  pseudomonads we  have
researched basic processes involved in
colonization   of  plant   roots   by
pseudomonads.  Colonization  is
important  in the attainment of effective
populations of the bacteria and  in the
persistence and spread of organism in
the environment.
  We  have concentrated  upon two
aspects of pseudomonad colonization in
the  plant   root   environment:  1)
mechanisms of pseudomonad attach-
ment to the plant root involving plant
agglutinin,  and  2) the role of catalase
and  superoxide dismutase (SOD)  in
pseudomonads in  protection of the
bacteria against root produced activated
oxygen species.

METHODS, RESULTS and DISCUSSION

Mechanism of Attachment to the Plant
Root Involving Agglutinin
  Certain  isolates  of  Pseudomonas
put/da are agglutinated by plant surface
glycoproteins  termed   agglutinins.
Purification of the agglutinin has been
initiated to  determine structure and as
a primary step in isolation of the plant
genes  encoding   agglutinin-active
components. We have examined wild
type  agglutinable   isolates,  mutants
which lack the agglutination phenotype
(Agg") and agglutination complemented
mutants  to  probe  the molecular and
genetic   basis  of   the   agglutination
phenotype in the bacterium.

A)   The  agglutinin  is  a   IS-lectin
arabinogalactan protein

  Agglutinin  activity  is detected in
water washes  of  intact plant  roots.
Components in bean root washes have
been  purified  by  passage  through  a
sizing filter to remove molecules of less
than   10,000  Mr,   followed   by
Concanavalin A and  DEAE-Sephadex
chromatography.  These approaches
demonstrate that agglutinin activity is
associated with multiple  fractions. The
majority of the agglutinin activity elutes
without adsorption to both  ConA and
DEAE-Sephadex.  This   nonadsorbed
material retains ability to react with
Yariv's reagent: a reaction indicative of
fc-lectin   activity   associated   with
arabinogalactan proteins. IEF  of the
nonadsorbed  material  fractionates the
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 preparation into four bands of agglutinin
 -active components with pi 7-8 and a
 larger agglutinin-active  heterogenous
 fraction with a more basic pi.
   A second  line of evidence supports
 the agglutinin to be an arabinogalactan
 protein. Both crude and more purified
 preparations react with a  monoclonal
 antibody MAC 207, which is derived to
 an arabinose epitope of a  membrane-
 associated arabinogalactan protein from
 carrot. This antibody was generated in
 the  laboratory of Dr. Keith Roberts,
 John  Innes   Institute  of  Norwich
 England,  with  whom  collaborative
 studies   are  being  continued.
 Agglutinin-active fractions which elute
 without adsorption to ConA, but which
 are  retained  or  adsorbed   to  DEAE-
 Sephadex react differently to two other
 mononclonals derived by Robert's et al.
 to pectin : thus these studies indicate
 that   differences   in   carbohydrate
 structure   between   agglutinin-active
 components exists. Data from studies
 of Robert's et al. indicate that MAC
 207  antibody-reactive materials  are
 widespread in the plant kingdom: these
 data agree with our previous observ-
 ations that extracts from a wide array
 of  plant  species  possess  agglutinin
 activity.
   In collaboration with Dr. Schippers at
 the  University of  Utrecht we  have
 surveyed several pseudomonad isolates
 from  wheat,  tomato  and  potato for
 agglutinability.  Certain   of   these
 European strains display agglutinability
 with  our crude  and  more  purified
 preparations of agglutinin from bean, as
 well   as  with  crude  root   wash
 preparations  from wheat, tomato and
 potato prepared at Utrecht.  These data
support   that   the   agglutination
 phenotype   in   pseudomonads   has
 relative little plant species specificity.
 Additional collaborativestudies with our
 Agg + wild type and Agg" mutants of P.
 put/da   have   revealed  that   the
 agglutination  phenotype  has   little
 influence   on whether  the   bacteria
 become endorhizosphere colonists  or
 remain on the rhizoplane.

 B) Genetic  and molecular studies of the
 bacterial agglutination phenotype.

   Genetic  and  molecular  techniques
 have been used to explore the nature of
 the  bacterial  phenotype  which  is
 responsible "for agglutination. Genetic
 analysis has  revealed two distinct loci
 associated    with  the   agglutinable
 phenotype. One  locus, termed  aggA
 has been analyzed in mutant Tn5 5123.
 A second locus aggB is detected in Tn5
 mutants 1104 and 6000.
  The aggA  locus is present in a 2.7
 kbp EcoRI-Hind III subclone of parental
 DNA.      Deletion   analysis   using
 exonucleases revealed only  1.45 kbp
 was needed for complementation of the
 Agg"  mutant 5123   to  the  Agg +
 phenotype.  The  presence   of  the
 subclone in transconjugants  of  5123
 restored the agglutinability of the cells.
 Adherence to intact bean roots surfaces
 in a  15 minute  exposure was  also
 restored  to  wild  type  level in the
transconjugants.  Thus,  a role of the
 locus in binding,  which was suggested
 by previous studies with Agg" mutants,
was confirmed.
  Sequence analysis  of  the  2.7 kbp
fragment reveals an open reading frame
on  one strand of  1356 nucleotides
encoding a  predicted 50,509 Da protein
and pi of 5.24- A consensus sequence
                                     82

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for a ribosome binding site (AGGGAG)
was  located 8bp upstream of  the
projected ATG translational start site.
Portions  of  the  canonical sequences
recognized by the 6 54 and 5 70 RNA
polymerase holoenzymes were present
5' of the ATG. The predicted protein
contains  one  membrane associated
helix, from residues 2-17, and there is
consensus for a prokaryotic cleavage
site located  between residues 22 and
23.
  Another reading frame  was present
on the  opposing strand of the 2.7 kbp
subclone but  it  lacked  a  ribosome
binding site, promoter sequences and
would   use  GTG  as   a  potential
translational  initiation   site.   No
translational  stop   codons   were
observed in  86 bases 5  to the GTG
site.    These data suggest  that  the
second ORF may be incomplete and not
be   involved  in  the   agglutination
phenotypes. A search of the NBRF and
GenEMBL data bases has revealed no
homologous sequences with either of
the predicted ORFs.
  Hybridization analysis with the aggA
locus  to  the   genomes  of   plant-
associated bacteria indicated that with
thirty  isolates examined  from  bean,
wheat, tomato  and potato,  only the
agglutinable  isolates  demonstrated
strong  to moderate hybridization. Thus
this  locus  has  good  specificity for
detection   of   the  agglutinable
phenotype.
  The second locus aggB from P.putida
also  is involved  in the  agglutination
phenotype.  Complementation of  the
phenotype  has  not been achieved in
trans using cosmid clones from a wild
type P. put/da library. Marker exchange
studies with  the locus   aggB:  Tn5
disrupted   the  Agg+   wild   type
chromosome  and  the  recombinants
were nonagglutinable. Preliminary data
suggest  the  presence  of  repetitive
sequences in the region of the insertion.

  Molecular  studies  have  demon-
strated that ceil envelope proteins are
associated  with  agglutination pheno-
type.  Agg'mutants  Tn5  5123,  EMS
1202  and  EMS  1236  possessed a
16,000 Mr protein band which was less
apparent in the Agg+  parental strain.
Fractionation of the cells indicates this
16,000  Mr protein  is located in the
periplasm.  Agg"  mutants Tn5 4312,
Tn5 5123 and  EMS 1236 displayed an
outermembrane protein  band  of Mr
42,000 which was not observed in the
parent. Transconjugants  of Agg" 5123
which were restored to  agglutinabilty
(Agg + )   by the  presence  of  clone
pRKAGG201 bearing a wild type 2.7
kbp EcoRI  -Hind III  fragment  did not
produce the 42,000 Mr protein but still
expressed  the 16,000   Mr  protein.
These data suggests that the 42,000
Mr  protein  is associated  with  the
agglutination phenotype.

Catalase and SOD Activities from Root
Colonizing Pseudomonads

  The plant root is potentially a  toxic
environment for  microbes because of
the production of  activated  oxygen
species   from   the  root   surface.
Consequently,  we have  examined the
role  of  catalase and  SOD  in  root
colonizing  pseudomonads   as  a
protective mechanism to permit survival
in   the   rhizosphere.  Each  of  the
pseudomonad  isolates examined  have
catalase  and SOD activities. We  have
                                     83

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 surveyed five beneficial isolates  from
 wheat, six isolates from sugar beet, the
 bean colonizing isolate of P. putida and
 isolates from  plants grown  at a  mine
 spoil site  with  acidic pH  and  high
 copper content. The mine spoil site was
 examined because of the potential of
 heavy   metals   to  catalyze
 interconversions of activated oxygen
 species  which  would  perhaps   put
 oxidative  stress  on the bacteria. The
 pseudomonads from  the agricultural
 soils  had  similar  levels  of catalase
 whereas   three  of four  mine  spoil
 isolates displayed three to ten fold more
 activity. Multiple isozymes of catalase
 of differing mobilities between isolates
 were observed  for  cells  grown to
 stationary  phase  in  rich   medium,
 whereas one or two isozymes of SOD
 were obtained.
   Studies  with  P.  putida  and  P.
 fluorescens  isolates  with   beneficial
 properties demonstrate that SOD  is
 rapidly  increased  in  activity  upon
 exposure to bean roots and remains at
 high  level  during  root  colonization.
 Catalase activity is low in cells  that
 haveestablished colonization. However,
 upon  short term  exposure in  the
 bacteria   the   activity   transiently
 increases, in part because of production
 of  a  second  isozyme  and  enhanced
 levels  of   the  normally  expressed
 isozyme.   We  are   exploring   the
 differential functions of these catalase
 isozymes.  Catalase isozyme  A is  the
 first   isozyme  produced   during
 logarithmic  growth   and   has   a
 cytoplasmic   location.  Additional
 isozymes,  which  are less anodic on
 nondenaturing gels and are termed B,C
etc.,  are  produced later  in  growth
phase. Catalase B  is  located in  the
 membrane  envelope  of  P.  putida.
 Increased activities of catalase A and
 production of catalase B and C occur in
 logarithmic  phase cells  of P.  putida
 after exposure to H202.  Thus  these
 changes in isozyme expression may be
 important  for  the  survival   of the
 bacterium  to  exogenous H2O2,  a
 situation occurring upon exposure to
 plant roots.
   It seems likely that catalase A and B
 are products of different genes, rather
 than being  derived from  one  gene
 product. The isozymes are differentially
 inhibited by aminotriazole and organic
 solvents.  Also  we  can  separately
 mutate  the activities  in  P.  putida.
 Mutant   J1,   obtained   by   EMS
 mutagenesis  of the  parental strain,
 lacks catalase A activity under liquid
 growth   conditions.   Nondenaturing
 electrophoresis  indicated that  catalase
 A was absent, although catalase B and
 C were  produced  during late growth
 periods.  However, mutant J1 was able
 to  colonize  plant roots at  wi|d  type
 rates.  This   ability  may  relate  to
 production of catalase A in J1  after
 exposure to external sources of H202.
 A similar induction  of catalase A in J1
 occurs on contact of  J1  with  plant
 roots.  Cells of J1  examined from the
 root surface  after  twelve  hours of
 exposure displayed,both catalase B and
 catalase  A isozymes.
  A further mutant J6 obtained by Tn5
 insertion  into J1  lacked all detectable
 catalase activities when grown on liquid
 growth media even to stationary phase.
Thus J6  appears to have a mutation
which  prevents  the  production  of
catalases B  and C. This mutant only
colonized roots at levels much reduced
from that of the wild type. Also J6
                                     84

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was reduced in its ability  to  survive
H202 treatments although it was able
to  produce catalase A, after H202
exposure.  These  data  suggest that
regulation of catalases B and C as well
as  catalase A  is  important  in  the
survival of bacteria to H202  and  on
plant roots.
  We are exploring  the  altered gene
expression of SOD upon  root coloniz-
ation  by  the   generation  of  SOD
deficient  mutants,  through  marker
exchange of a  mutated gene into the
parental cell. Currently we have isolated
a fragment from a  cosmid library of P.
putida which will complement the SOD
deficiency   in   a   SOD-mutant   of
Escherichia  coli.  The  complemented
cells produce  a SOD  activity  which
migrates   on.  nondenaturing  gel
electrophoresis  to the same location as
the Fe SOD from the pseudomonad cell.
This migration pattern is  quite  distinct
from that of the £.. coli SODs.  We are
currently subcloning the fragment and
will perform transposon mutagenesis to
delete the SOD  activity.

CONCLUSION

  We  have demonstrated that root
colonizing  pseudomonad cells have
several  levels of  interaction with  the
plant.    Interaction  between: a root
surface arabinogalactan  protein  and
surface features  of  root  colonizing
bacteria   is    involved   in   strong
attachment and improved colonization
potential. The agglutination phenotype
in the bacteria is  related.to discrete
genetic loci and we have identified one
locus as being a specific marker for the
agglutinability trait. Other genes in the
bacteria are also  regulated upon root
colonization.  These include genes for
catalase  and   SOD  which   encode
proteins involved in the survival of the
bacteria on the root surface.

FUTURE STUDIES

  Our goals are to:
1) continue the  purification  of - the
agglutinin so that amino acid sequences
and  glycosylation  structure  can  be
determined. We will initiate progress
towards  isolation  of  plant   genes
involved in  agglutinin synthesis.
2) isolate genes encoding catalase A
and  FeSOD from  P. putida. Mutated
genes  will be  inserted  into  the
chromosome  of  pseudomonads   by
marker   exchange  to   probe  their
function. We  also  will  examine  the
regulation  of  the  promoters  of  the
genes through  gene fusion constructs.
3) examine the function of the promoter
regions  for the  two  loci  that  are
involved in the agglutination phenotype
in the bacteria. We will  use  promoter
fusions to  determine if expression of
the agglutination genes is regulated as
the bacteria colonize root surfaces.

PUBLICATIONS

Tari, P.M. and A.J. Anderson.  1988.
Fusarium  wilt  suppression   and
agglutinability of Pseudomohas putida.
Appl.  Environ.  Microbiol  54:2037-
2041.

Katsuwon and A.J. Anderson.  1989.
Response  of  plant-colonizing
pseudomonads to hydrogen peroxide.
Appl.  Environ.  Microbiol  55:2985-
2989.
                                     85.

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 Anderson, A.J., and  Katsuwon,  J.
 1990.    Catalase  and  superoxide
 dismutase   of   root-colonizing
 saprophytic pseudomonads.   Applied
 and  Environmental   Miicrobiology.
 56:3576-3583.

 EPA Investigators  Meeting,  Corvallis,
 November 13-17, 1989. Presentation:
 Bacterial traits involved in root surface
 colonization.

 A.J.  Anderson.    1990.   Biological
 Control  of Sugar Beet Diseases.  161-
 163. In: Proceedings of the University
 of  Idaho  Winter Commodity Schools
 Burley, Idaho Feb  15 1990,  Caldwell
 Idaho Feb 16 1990.

 UCLA Conference April 1990. R.  Buell
 and A.J. Anderson. Genetic analysis of
 adhesion  of  P.  put/da.   J. Cellular
 Biochem.  Suppl. 14E p. 314

in:  G.Defargo,  ed.,  Plant  Growth
 Promoting Rhizobacteria. 1991.
 i)    R.E.  Zdor  and A. J. Anderson,
 Influence of root colonizing bacteria on
the defense responses of beans.
 ii)   J. Katsuwon and A. J. Anderson,
Survival strategies of root colonizing
pseudomonads
iii)  C. R.  Buell  and A.J.  Anderson,
Genetic  analysis of  agglutination  in
Pseudomonas put/da
i-iii) presented at Rhizosphere  meeting,
Interlaken October 1990.
 EPA Investigators meeting April 8-12
 1991   Presentation   Genetic  and
 molecular  analysis   of   survival
 mechanisms of pseudomonads.

 Buell, R. and A.  J. Anderson. Genetic
 analysis   of   a   locus  involved   in
 agglutination of pseudomonads   (in
 review J. Bacteriology)

 Katsuwon,  J.  and  A.J.  Anderson.
 Characterization  of  catalases  from
 fluorescent   root   colonizing
 pseudomonads  (in   review  Appl.
 Environ. Bacteriol)

 Zdor, R. and A. J. Anderson. Defense
 responses in bean activated by root
 colonization  by  fluorescent
 pseudomonads  (in review Plant  and
 Soil)

 B. Lovic, C. Heck, J.J. Gallian and A. J.
 Anderson. Biological control of sugar
 beet   pathogens      (in  review
 Phytopathology)

 Anderson, A. J., R. Buell, R. Whetten
 and P. Tari. Cell surface properties and
 agglutinability of Pseudomonas put/da
 (in   review   Canadian  Journal   of
 Microbiology)

 C. R. Buell and A.J. Anderson.  1990.
Genetic  analysis  of  agglutinability  of
 beneficial  root  colonizing
pseudomonads.  J. Cellular Biochem.
Suppl. 14E, p.314.
                                    86

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                        STABILITY OF FOREIGN DNA
                IN THE FUNGUS Colletotrichum gloeosporioides
                  John L. Armstrong1 and Deborah L. Harris2
                    U.S. Environmental Protection Agency1
                   ManTech Environmental Technology Inc.2
                      Environmental Research Laboratory
                              Corvallis, Oregon
INTRODUCTION
  Progress in the genetic engineering of
fungi  has  been rapid in recent years.
With these advances, researchers are
observing that the foreign DNA is often
unstable in the transformed fungi. It is
typical for the introduced  DNA  to  be
lost   or   rearranged   within  the
chromosome.  Also, transformants are
often  relatively sickly as demonstrated
by  their   poor  survival,   abnormal
pigmentation,   and  strange  colony
morphology.  We selected the fungus
Colletotrichum  gloeosporioides  var.
aeschynemone  (CGA)   as  a  model
organism  to  study  the factors that
determine the stability  of transformed
marker genes in CGA sprayed on plants
and in microcosm chambers.

METHODS

Transformation of Protoplasts
and Detection of Foreign DNA

  The fungus was cultured for 4 days
at 30°C on  Torula yeast  agar  (15 g
Torula yeast, 15 g starch, 1 g K2HPO4,
0.5 g MgSO4'H20, 15 g agar/L) or in
YpSS broth.   To prepare  protoplasts,
spores were harvested from plates with
sterile water, centrifuged for 5 min. at
1000xg,  suspended  in  a  solution
consisting of 2 parts  1.2 M MgSO4-
Tris-CI (10 mM, pH 5.8) and 1 part 0.6
MgS04-Tris-CI  (10  mM,  pH  5.8.)
containing Novozym 234 (50 mg/ml),
and  incubated for 5 hours at  32°C.
Protoplasts were centrifuged for 1 min.
at 2000xg and washed  five times by
centrifugation in 1.2 M sorbitol-Tris-CI
(10 mM, pH 7.0)  to remove nucleases
present  in the Novozym.  Cells were
resuspended in the sorbitol solution for
DNA transformation.
  Cells  were transformed with  pBT.
This plasmid was made  by inserting a
2.6 kb Sal I fragment that encodes the
p-tubulin  gene  from  a  Neurospora
crassa mutant (gene confers resistance
to  benomyl)  into the  Sal I  site  of
pBS( + /-) (Stratagene). Twenty //g pBT
DNA  were added to about 106  proto-
plasts. After adding an equal volume of
30% PEG  in  10 mM  CaCI2,  the
suspension was incubated 30 min. at
25 °C. Protoplasts were centrifuged for
3 min. at SOOOxg, resuspended  in 1.2
M sorbitol-Tris-HCI (10 mM; pH 7), and
spread  on  agar  containing  0.8  M
sucrose. After 24 h, four ml volumes
of 1 % YpSS overlay agar containing 10
jjg benomyl/ml were layered on the agar
surfaces.  These were  incubated at
30°C   for   5   days.     Putative
transformants  were restreaked  three
times on the selective medium.
   To extract DNA, fungi were cultured
in  YpSS  broth   containing  10  /vg
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 benomyl/ml and collected by filtration,
 dried, and ground with a mortar-pestle.
 The  powder  was  suspended  in  a
 hexadecyltri-methylammoniumbromide
 (CTAB)  solution  (10% CTAB,  0.7 M
 NaCl), heated  1 hour at 55°C, mixed
 with   an   equal   volume  of
 chloroform:isoamyl  alcohol  (24:1),
 centrifuged    10   min.,   and   the
 supernatant liquid collected.  An equal
 volume of 1% CTAB-Tris-CI (50 mM,
 pH 8.0)  was added. After 30 min. at
 25°C, the suspension was centrifuged
 at 5000xg for 5 min.  The  pellet was
 dissolved in  cesium chloride solution
 and  the  DNA  purified   by   ultra-
 centrifugation.
   Presence  of the  p-tubulin gene in
 transformants was confirmed by DNA
 hybridization.   DNA was  restricted,
 electrophoresed, transferred to a Nytran
 membrane, and probed with the Sal I
 fragment carrying the  (J-tubulin gene.
 The probe was labelled with 3^P by
 random  primer extension.   The final
 wash in  the  hybridization  step was
 performed for 15  min. at 65°C in 2 X
 SSC and 0.1 % SDS. Hybridization was
 detected by autoradiography.

 Construction of Plasmid pBC

  Plasmid pBC was  prepared by first
 restricting the Sph I site in the multiple
 cloning  site (MCS)  of  pBS( + /-)  and
 inserting a 2.2 kb Sph I fragment which
 encoded  the  cutinase  gene  from  C.
gloeosporioides. The direction of the
 cutinase reading frame in this construct
 was away from the single Eco Rl site in
 the MCS. This DNA was then doubly-
digested  with Eco Rl  and Eco RV (site
about 1  kb from the Eco Rl site in the
MCS) and blunt end-ligated to eliminate
 the 1 kb RI-RV fragment.  This step
 removed the Sal I  site in the MCS and
 shifted the unique Nar I  site in  the
 cutinase gene  to  a position  near  the
 middle of the fungus DNA insert.  Next,
 we restricted the  Nar I site,  removed
 the tails, and  inserted  Sal I  linkers.
 Finally, the Sal I fragment carrying the
 N.  crassa p-tubulin gene was  inserted
 into the new Sal I site of the cutinase
 sequence.

 Microcosm Studies

   Microcosm studies were performed in
 a glass chamber (1 .Ox 0.75 x 0.75 m)
 that was irradiated with a 1000-watt
 Sylvania metal halide lamp  (18 h/6
 hours light/dark cycle). Crimson clover
 plants were  raised in wooden  trays
 filled  with 6-7  cm. of soil.  To spray
 plants, CGA  spores were washed  off
 Torula yeast agar plates with sterile
 water, centrifuged  twice in  water, and
 suspended   at  2-5x107  spores/ml.
 Plants and soil were sprayed with about
 100 ml suspension/tray using a plastic
 misting bottle. Three plant samples (2-
 3 g/sample) were  aseptically taken at
 random from each chamber on  each
 sampling day, put in sterile plastic bags
 with 20 ml water, and  blended for 1
 min. in a Stomacher blender (Model 80,
 Tekmar Co.).   Using  a spatula, three
 soil  samples   (1-2  g/sample) were
 aseptically collected at  random from the
 top centimeter of soil and vortex-mixed
 for  1  min.  in 18  x 150 mm  screw-
 capped tubes containing 5 ml water and
 glass  beads.  To enumerate colonies,
 samples were diluted in water, spread
 in duplicate on Martin's agar amended
with 100//g benomyl/ml, and incubated
for  36 hours at 30°C.  Colony  counts
                                    88

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from a pair of plates were averaged for
calculations  of colony forming  units
(CFU).

RESULTS AND DISCUSSION

Transformation of Protoplasts
and Detection of Foreign DNA

  We obtained  benomyl®  strains of
CGA  following  transformation  with
plasmid  pBT (A/,  crassa p-tubulin gene
on Sal I insert in pBS(+ /-)). To date,
three putative transformants have been
analyzed.  They tolerate up to  100 /yg
benomyl/ml and  hybridize with the p-
tubulin gene probe.  However, in the
presence  of   the  antibiotic,  only
transformant   II-4  has  the  orange
pigmentation  of  the parent strain and
does not have retarded growth.  This
isolate also grows without significant
inhibition  in the presence  of  300 //g
benomyl/ml.   Based on  DNA  hybrid-
ization  of Southern  transfers,  the
transforming DNAs in the  three isolates
inserted  into  sites  other  than the
genomic P-tubulin sequences.  We are
currently determining if the foreign DNA
changes  the   pathogenesis   of  the
transformants by inoculating northern
joint  vetch  plants   (susceptible  to
invasion by CGA) with  spores from
transformants.
  We are at the early stages of the
transformation studies and have not yet
transformed CGA with pBC (pBS(+/-)
carrying N. crassa (i-tubulin gene with
flanking  C.  gloeosporioides  cutinase
sequences).   As  we acquire  these
transformants, they will  be compared
with  isolates  containing  DNA  from
plasmid   pBT  (pBS( +  /-)   carrying
Neurospora   p-tubulin   without  the
flanking  C.  gloeosporioides  cutinase
sequences).    This  will allow .us to
determine  the   influence   of   the
associated, homologous cutinase DNA
on  a)  frequency of  p-tubulin  gene
transformation, b) site of integration, c)
stability of the transforming DNA in the
chromosome, and d) persistence of the
benomyl phenotype.

Microcosm Studies

  In the second aspect of our research,
soil/plant microcosms are being used to
compare survival of CGA transformants
with the nonrecombinant CGA parent.
We have already developed methods for
spraying  spores   on   plants   and
processing soil and plant samples.  This
preliminary   work  was   done   by
inoculating crimson  clover plants  with
106 CFU/g of leaf. Populations dropped
about 100-fold by day 22.  During the
same period,  populations in  the soil
decreased from  about 1O4  to  10
CFU/g of soil.
  Currently,  microcosm experiments
are being used to compare populations
of  the  parent and  transformant II-4
strains.  When the CGA populations in
the microcosms drop to near-threshold
levels, we will culture isolates from leaf
and soil samples  and extract the DNAs.
RFLPs  of these  DNAs will  then be
analyzed by DNA hybridization with the
P-tubulin gene as a probe.  This will
allow us to determine if the location of
the transformed  gene changes during
exposure of the fungus to leaf surfaces
and soil.
                                                                                  v
                                     89

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ACKNOWLEDGMENT

   Dr. George Rohrmann's expert advice
on cloning strategy was crucial for the
successful construction of pBC.

FUTURE WORK

   Plans for  this  project include: a)
determine   genetic  factors   (e.g.,
homologous   versus   heterologous
integration)   related to  stability  of
recombinant DNA in transformed fungi;
b) assess survival recombinant fungi in
the  field;  c) study  persistence  of
recombinant fungi in microcosms that
simulate on-going field conditions (RH,
air temperature, light intensity, and soil
moisture); d)  use recombinant fungi to
enhance bioremediation and restoration
of polluted environments.
                                    90

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                   PERSPECTIVES ON PLASMID STABILITY:
                A STUDY OF THE EPA BENCHMARK PLASMIDS
                     Toai T. Nguyen and Richard E. Lenski
                Department of Ecology and Evolutionary Biology
                         University of California, Irvine
                               Irvine, California
INTRODUCTION
  Microbial   degradation   of   toxic
pollutants released into the environment
by various  human activities proceeds
through    co-oxidation   and   /  or
mineralization  (Alexander,   1981).
Often  resulting   from  non-specific
degradation  by enzymes of common
pathways,    co-oxidation   produces
intermediates which can no  longer be
broken down  by  enzymes with high
substrate specificity further down the
pathway  and  which accumulate in the
medium.      In   some  cases,  the
intermediates are even more toxic than
their   precursors,   which   makes
co-oxidation  quite  undesirable.    It
follows that co-oxidation is rather slow,
fails   to  completely   degrade  toxic
pollutants    and   cannot   provide
microflora with energy or metabolites.
In  contrast,  through  mineralization,
toxic pollutants are completely broken
down by microflora which can derive
energy and   metabolites  from  them.
Thus microbial mineralization appears to
be a more attractive process to detoxify
the environment.
  Although,   in  several  documented
instances, microbial mineralizationcould
be shown to result from the  combined
action  of chromosomal and  plasmid-
borne genes in the same strain (Ghosal
et al.; 1985), it is seldom observed that
a naturally occurring pure culture can
completely  break  down  a   target
compound.   Rather,   mineralization
usually  results from  complementary
action of members of a  mixed culture
(Chatterjee et al.,  1981).   Through
intensive selection  in the laboratory,
one can isolate a  pure strain that can
use a target compound as sole energy
and  carbon source.  Often, such a
strain could subsequently be shown to
harbor a plasmid carrying all the genes
necessary   for   the   mineralization
process.
  By  using selection to  engineer a
strain that  can metabolize a  target
compound,  one  is  faced  with  the
problem that the resulting strain might
require a much higher concentration of
substrate  for  growth than could  be
found  in   the  worst  polluted  site.
Compounding that problem, alternative
carbon sources readily available in the
environment might be used in prefer-
ence to the target compound,  thus
defeating the purpose of the engineered
strain.  Also, a strain developed in a
laboratory environment might lose its
competitiveness over indigent flora of a
polluted site,  thus  would  be  rapidly
outgrown   before  it   has  time  to
complete  its  intended  detoxification
task.
  While making them more amenable to
various studies the plasmid location of
those genes of interest also increases
their likelihood to  be lost. Our current
                                     91

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 research focuses  on the problem  of
 plasmid  loss  from  a  population  of
 genetically engineered microorganisms.
 With plasmid copy number restored to
 its pre-division state shortly after cell
 division, plasmid loss could result from
 random unequal partitioning of copies
 to  daughter cells, or segregation.  It
 could also  be accelerated by a higher
 growth rate of the plasmid-free cells, as
 compared to  that of plasmid-bearing
 ones, or selection.
   Most often, plasmid loss results from
 a  combined effect of segregation and
 selection. Knowing which phenomenon
 plays a major role in plasmid loss helps
 one decide  on the most .appropriate
 course of action  to  enhance plasmid
 stability.  If segregation is the  major
 cause of  plasmid loss,  one   could
 consider ways to increase copy number
 to lessen the chance of loss by unequal
 partitioning or to incorporate a function
 that ensures equal partitioning.   If a
 plasmid reduces the growth rate of its
 host, increasing its  copy number  or
 incorporating  a  partitioning  function
 might have  the   opposite effect  of
 making plasmid-bearing cells even less
 competitive,  thus  exacerbating the
 problem.
   As model plasmids, we used a series
 of EPA benchmark plasmids (pR02313,
 pRO2317,  pRO2318, pRO2320  and
 PRO2321;  Zylstra et al.,  in  press),
 maintained   in  two  bacterial  model
 backgrounds, Pseudomonas aeruginosa
 strain  PAO1c  and P.  putida  strain
 PRS2015. We determined that all the
above benchmark plasmids are more or
 less unstable in either host background.
Whether segregation or selection is the
major cause of plasmid  loss depends
very much  on the plasmid, the host
 background and the growth medium.

 METHODS

 Bacterial Strains and Plasmids

   Dr.  R. Olsen (Medical School, Univ.
 of Michigan) kindly provided plasmid-
 free P. aeruginosa PAO1c and PAOIc
 strains bearing benchmark  plasmids
 PR02313,   pR02317,   pR02318,
 PR02320  and  pRO2321.    Dr.   S.
 Cuskey  (Environmental   Research
 Laboratory, Gulf  Breeze,  FL)  kindly
 provided   plasmid-free   P.   putida
 PRS2015 and PRS2015 strains bearing
 the same set of the above benchmark
 plasmids.  To ensure true isogenicity,
 plasmid DMA was isolated from plasmid
 be.aring strains by  the rapid isolation
 technique (Maniatis et al.,  1982) and
 used   to  re-transform  plasmid-free
 strains by the CaCI2 technique (Mandel
 and Higa, 1970). Transformants were
 isolated,  streak-purified on antibiotic
 plates, grown  to saturation in L broth
 with antibiotic,  mixed with an equal
 volume of glycerol and frozen  at -80
 °C.  Glycerol stocks of plasmid-free
 strains were made in a similar way
.except no antibiotic  was  used.   All
 experiments were initiated with single
 colonies streaked on  antibiotic  plates
 from such glycerol stbcks.

 Media  and Antibiotics

   LB  broth contains per  liter  10  g
 Tryptone, 5 g  Yeast Extract and 10 g
 NaCI.  LB agar is LB broth With 14 g
 Bacto  agar per liter.  PG contains per
 liter 30.7  mmoles  K2HP04,   14.7
 mmoies KH2PO4, 18.7 mmoles NH4CI,
 1 mmole MgSO4, 2 micromoles FeSO4,
                                     92

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2 mg thiamine hydrochloride and 1 g
glucose.  Anhydrous ampicillin (Ap) is
dissolved in dimethyl sulfoxide at 100
g/l, stored  at  -80 °C, and  used at a
final  concentration  of  500   mg/I.
Carbenicillin (Cb) and piperacillin (Pp)
are dissolved in water at 600 g/l and
100 g/l, stored at -80 °C and used a
final concentrations of 600 mg/I and 20
mg/1, respectively. Tetracycline (Tc) is
dissolved in methanol at 10 g/l, stored
at  -80  °C,  and  used  at  a  final
concentration of 80 mg/I for PAOIc or
10  mg/I for PRS2015 strains bearing
Tc- resistant plasmids.
                  Competition-type
Stability-   and
Experiments
  Stability-   and   competition-type
experiments  were   conducted  in  a
pairwise   fashion.      Essentially,
plasmid-bearing  cells were  grown ^to
saturation in  LB supplemented  with
antibiotic, harvested by centrifugation,
washed  three  times with  an  equal
volume of 8.5 g/l NaCI.   Plasmid-free
cells  were  grown   up  similarly but
without antibiotic, and harvested in the
same  manner.    For a  stability-type
experiment,  plasmid bearing cells were
inoculated on day 0 as pure culture at
,QD550 - 0.05. A competition-type
experiment started with a culture of the
same OD550 but consisting of a 1:1
mixture   of   plasmid-bearing , and
plasmid-free  cells.    Cultures  were
incubated at 32 °C,  aerated with a 240
RPM   gyratory  motion.      Every
subsequent   24  hours,  cells  were
subcultured  at  1:100,  as  described
above,  into fresh  medium.    This
regimen  resulted in  an equivalent  of
Iog2(100)=6.64 generations per 24 h.
                                         Each  day,  saturated  cultures  were
                                         serially  diluted in 8.5  g/l  NaCI and
                                         plated  on  LB  agar  to  obtain single
                                         colonies;  the  daily  frequencies  of
                                         antibiotic   resistant   cells   were
                                         determined by toothpick streaking 100
                                         or 200  colonies on plates of  LB agar
                                         supplemented  with antibiotic.

                                           Data analysis.  According to Lenski
                                         and; Bouma (1987), the rate of plasmid
                                         loss could be described by the following
                                         diff.erential equation:
                                          [1]
                                                 dP
dt
                                                      = - uP- sP(1  - P)
                                         where P is the frequency of plasmid-
                                         bearing cells, u. the segregation rate and
                                         s the selection coefficient.  Equation [1]
                                         can be integrated to yield:
[2]   P =
• .   .  [U  H
                                                     (u + s)Pr
       -P0)]e
                                                              lu
                                                                      sP
                                         where P0 is the initial frequency  of
                                         plasmid-bearing  cells.  We  computed
                                         estimates of the segregation  rate,  u,
                                         and  selection coefficient, s, by least
                                         squares  non-linear  regression  (Dixon,
                                         1985) fit to  equation [2]. Through the
                                         use  of a  dummy variable (Kleinbaum
                                         and  Kupper, 1978), the intercept, P0,
                                         could be varied according to  whether a
                                         particular set of points came from a
                                         stability-   or   competition-type
                                         experiment.     This  allowed   paired
                                         stability-   and   competition-type
                                         experiments "to be used simultaneously
                                         in the  estimation  of  either u  alone
                                         (model I, in which s  is constrained to  be
                                         zero) or both u and  s (model II). Partial
                                     93

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E-tests were conducted to determine if
model  II  significantly improves  the fit
over model I  with the null hypothesis
being rejected only at p<0.05.

RESULTS AND DISCUSSION

  For  each  pair of  stability-  and
competition-type experiments, we used
partial F-test to determine whether the
data points fit model II (segregation and
selection)  significantly   better  than
model I (segregation alone). For each
combination  of  plasmid,  host  and
growth  medium, we  tabulated  how
many pairs of experiments fall into the
above category.  If over 5% of the total
pairs of experiments does, we conclude
that for  that  combination, selection
plays a significant role in  plasmid loss.
The results are shown in Table T. The
                 TABLE 1.  PROPORTIONS OF
             SIGNIFICANT CONTRIBUTION OF
    process  leads   to   assembling  the
    host-plasmid-mediumcombinationsinto
    two groups: Group A comprising those
    with a  pattern  of   instability  fitting
    model I (segregation alone); group  B
    comprising  those with  a pattern  of
    instability fitting  model II (segregation
    .and selection).
      The  above arrangement facilitates
    comparison among member combina-
    tions  of  a   same  group  for  their
    instability. This could readily be done
    with group A since it involves direct
    comparison of the segregation rates.
    However, the instabilities of member
    combinations in group B result from an
    interaction between  segregation and
    selection.   To   arrive  a  meaningful
    comparison, we define a new quantity,
    1, a loss factor in  equation [3]:

EXPERIMENTS SHOWING
SELECTION IN PLASMID LOSS
Plasmid


PRO2313
PRO2317
pRO2318
pR02320
PR02321


pRO2313
PR02317
PR02318
pRO2320
pRO2321


pRO2313
PR02317
pRO2318
PR02320
PR02321
Experiments
Total with selection
as major cause
host: PRS2015
medium: PG
6
6
6
6
6
host: PRS2015
medium: LB
5
6
18
20
6
host: PAO1c
medium: PG
4
10
4
15
4


0
0
0
1
0


1
2
7
0
5


1
3
0
9
0
Percentage


0.0%
0.0%
0,0%
16.7%
0.0%


20.0%
33.3%
38.9%
0.0%
83.3%


25.0%
33.3%
0.0%
60,0%
0,0%
                                    94

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       dp
[3] I = -lim	 = -lim-u-s( 1 -P) = u + s
     P->0 Pdt  P->0

where   u,   s.  and  P   are defined
previously.
  Since  the segregation  rates  and
consequently the loss factors as well,
are not normally distributed, we chose
non- parametric methods to conduct
our statistically  analyses.   We used
Kruskal-Wallis method to determine if
there   was  a  significant   difference
among the segregation rates of group A
and the  loss factors of group  B.   In
both cases, the  null  hypothesis  was
rejected  at   p<0.0005.    We then
proceeded   to   carry   out  pairwise
two-tailed Mann-Whitney test  within
each group  to  determine the  relative
ranking in stability.  In  this case,  the
null  hypothesis   was   rejected   at
p < 0.05. The results are summarized in
Table 2 (page 96).
  Our analyses of the EPA  benchmark
plasmids indicate that  stability of a
plasmid-bearing strain is the result of a
complex interaction among plasmid,
host and growth medium.   It is thus
very difficult to  generalize and  to
predict if a particular background would
support two plasmids equally well even
if they were derived from  a common
precursor.      For   each   particular
combination, it is therefore necessary
to determine empirically the  pattern by
which a plasmid-bearing strain behaved.

FUTURE WORK

  From  our  studies,   we  identified
several host-plasmid-medium combina-
tions  with  minimal instabilities.   We
plan to use them in experiments to test
for the destabilizing effects of gene(s)
of environmental  utility.    We  also
identified  several  combinations with
high instabilities. These would be ideal
backgrounds to test for the stabilizing
effects of various genetic functions.

REFERENCES

Alexander, M.  1981.  Biodegradation
of Chemicals of environmental concern.
Science 211:132-138

Chatterjee, O.K.,  S.T. Kellog, D.R.
Watkins and A.M.  Chakrabarty. 1981,.
Plasmids  in  the  biodegradation  of
chlorinated aromatic compounds.  |Q:
S.B.   Levy,, R.C.  Clowes  and  E.L.
Koenig   (eds.)  Molecular   Biology,
Pathogenicitv, and  Ecology of Bacterial
Plasmids. Plenum  Press, London.

Dixon,   W.J.   (ed.).   1985.   BMDP
Statistical  Software.   University of
California Press, Berkeley. Ghosal,  D.,
I.-S.  You,  O.K.  Chatterjee and A.M.
Chakrabarty.  1985.  Plasmids in  the
degradation of  chlorinated  aromatic
compounds,   in:  D.R. Helinski, S.N.
Cohen, D.B. Clewell, D.A. Jackson and
A.  Hollaender  (eds.)  Plasmids   in
Bacteria. Plenum Press, New York and
London.

Kleinbaum, D. G., and L. L.  Kupper.
1978.  Applied regression analysis and
other  multivariable methods.  Duxbury
Press, North Scituate, Mass.

Lenski,  R.E. and J.E. Bouma.  1987.
Effects of segregation and  selection on
instability of  plasmid  pACYC184  in
Escherichia  coli   B.    J.  Bacteriol.
169:5314-5316.
                                     95

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             TABLE 2. HOST-PLASM ID-MEDIUM COMBINATIONS3
               WITH DIFFERENT PATTERNS OF PLASMID LOSS
Host
Plasmid
Medium
(Generation-1)b
Group A
(segregation alone)
PRS2015
PRS2015
PRS2015
PRS2015
PAO1C
PRS2015
PAO1C
pRO2313
pRO2317
pRO2321
PRO2318
PRO2321
pRO2320
pRO2318
PG
PG
PG
PG
PG
LB
PG
0.002
0.002
0.002
0.003
0.044
0.053
0.15
Group B
(segregation and selection)
PRS2015
PRS2015
PRS2015
PRS2015
PAO1C
PRS2015
PAO1c
PAO1C
pRO2321
pRO2317
pRO2318
pRO2320
pRO2317
pRO2313
pRO2313
PRO2320
LB
LB
LB
PG
PG
LB
PG
PG
-0.011
-0.002
0.006
0.017
0.037
0.055
0.069
0.15
a Combinations showing no statistical difference in segregation rates or loss
 factors are clustered together.
b Median segregation rate u for Group A and median loss factor I for Group B.
                                  96

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Mandel,  M.  and  A.  Higa.   1970.
Calcium-dependent bacteriophage DNA
infection.  J. Mol. Biol. 43:159-162.

Maniatis,  T.,  E.F.  Fritsch  and   J.
Sambrook. 1982.  Molecular Cloning.
Cold Spring Harbor Laboratory, Cold
Spring  Harbor, New York.

Zylstra, G.J.,  S.M. Cuskey and R.H.
Olsen.     In  press. Construction  of
plasmids for  use in  risk assessment
research,  in:  M. Levin, R. Seidler and
P.  Pritchard  (eds.)  Classical  and
molecular  methods   to   assess
                 applications   of
environmental
microorganisms.

PUBLICATIONS
Lenski, R. E. Fitness and gene stability.
in:  M. Levin, R. Seidler, M. Rogul and
H.  Pritchard  (eds.),   Classical  and
Molecular   Methods   to   Assess
                                 of
Environmental   Applications
Microorganisms. In press.
Nguyen, T.T., M. Patel and R.E. Lenski.
1991.  Reduced growth  rates due to
plasmid carriage  cause  instability  of
pACYC184   and   derivatives   in
Escherichia coli K12. Submitted to J.
Bacterioi.

Lenski, R.E. 1991. Quantifying fitness
and gene  stability in microorganisms.
in:   Ginzburg, L.R. (ed),  Assessing
Ecological Risks of Biotechnology, p.
173-192.  Butterworth-Heinemann.

Nguyen,  T.T.,  J. Lanners and  R.E.
Lenski. 1990.  How evolution and the
pSC101 par locus stabilize plasmids, p.
115-118.  in: R. Seidler (ed.), Review
of  Progress  in  the  Biotechnology-
Microbial  Pest   Control  Agent  Risk
Assessment    Program.   U.S.
Environmental Protection Agency.

Lenski, R.E. and T.T. Nguyen.  1989.
Fitness  and  the  fate  of genetically
engineered microorganisms,  p. 108-
116.  in: P.H. Pritchard, B.L.  Jackson,
J.E.  Harvey and  S.M.  Martin  (eds.),
Integration of Research and Predictive
Model Development for Biotechnology
Risk Assessment. U.S. Environmental
Protection Agency.

Nguyen,  T.T.,  J.  Lanners and  R.E.
Lenski. 1989.  How evolution and the
pSC101 par locus stabilize plasmids.
An  abstract  submitted  to  the  EPA
Biotechnology All-Investigators Meeting
14-16   November  1989,   Corvallis,
Oregon.

Nguyen, T.T. and R.E.  Lenski. 1989.
Stability of EPA benchmark plasmids:
Effects of host background and growth
medium, Submitted In  Proceedings of
the   Review.   U.S.     Environmental
Protection Agency.

Lenski, R.E. and T.T. Nguyen.  1988.
Stability of recombinant DNA and  its
effects on fitness, p. S18-S20. in: J.
Hodgson  and  A.M. Sugden  (eds.),
Planned   Release  of   Genetically
Engineered  Organisms   (Trends   in
Biotechnology/ Trends in  Ecology and
Evolution Special Publication). Elsevier
Publications, Cambridge.
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           MODELING THE FATE OF BACTERIA IN SURFACE WATERS
           John P. Connolly1, Richard B. Coffin2 and Robin E. Landeck1
                 Environmental Engineering & Science Program1
                              Manhattan College
                             Riverdale, New York
                     U.S. Environmental Protection Agency2
                       Environmental Research Laboratory
                             Gulf Breeze, Florida
 INTRODUCTION

   Potential ecological and public health
 impacts resulting from  the release of a
 genetically engineered microorganism
 (GEM)   to   the   environment   are
 dependent on the fate of the organism
 and the relationship between organism
 density or  activity  and  a  particular
 effect. A fundamental consideration in
 this regard is the ability of the organism
 to  survive and  compete in  a natural
 setting  and  possibly  transfer   the
 engineered   genetic  trait  to  other
 organisms. Prediction of the fate of the
 organism  and its  engineered  trait in
 natural systems  is a major component
 of a quantitative risk assessment.
   Surface  water   systems  are   an
 important environmental compartment
 with regard  to organism fate because
 they typically provide a suitable habitat
 for a  variety of organisms  and they
 provide a means  of rapid transport.
 Predicting the  fate  of a  GEM  in a
 surface water  system  requires  an
 analysis  of  substrate  and  nutrient
 inputs, the response of the GEM  and
 the indigenous  community  to these
 inputs and the impact of predation as a
 population control factor.  A necessary
 component  of  this analysis  is  the
competition for resources that  defines
the potential of the GEM to invade the
 community.  The analysis must  also
 describe the  transfer of  engineered
 genetic material between the GEM and
 indigenous organisms.
   The overall objective of the research
 being conducted  in this project is the
 step-wise  development   of   the
 components of a  framework to model
 the   movement   and   growth   of
 genetically-engineered  bacteria  that,
 either purposely or inadvertently, have
 been  introduced  to  a surface water
 system.  A primary goal has been to
 develop a modeling framework capable
 of predicting the long-term behavior of
 bacteria in surface water systems.  We
 have made significant progress towards
 this   goal.    We  have   completed
 development of a framework which has
 been   successfully   applied  to   a
 microcosm study of bacterial dynamics.
  The  second goal is to  extend  the
 framework  to   include  competition
 between bacterial populations.   This
 involves the division of bacteria into a
 minimum of two  groups  (indigenous
community and introduced population).
Recombinant bacteria  will be  included
as a third  group if  gene  transfer is
considered  to   be   a    potentially
significant  process.   Achieving this
objective requires the definition and
quantification of  the  mechanisms  of
resource competition.  Existing theory
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and past laboratory studies will be used
to establish working  hypotheses  that
will  be evaluated through laboratory
experimentation to be conducted at the
Gulf Breeze EPA.
   Ultimately the  framework  must be
expanded to incorporate  mechanistic
descriptions  of  gene transfer.    In
ongoing   and   recently   completed
research, models  have been proposed
to  describe  this process in  simple
laboratory systems. These formulations
provide the basis for development of a
complete biotechnology  risk  assess-
ment modeling framework.   Such  a
framework must  then  be evaluated
based on its ability to predict the fate of
engineered  genetic  material.   This
would  be accomplished by application
to laboratory  systems in  which  the
organisms have been introduced. Such
laboratory  data are  being generated
through the biotechnology program at
the Gulf  Breeze EPA and evaluation of
the full modeling  framework could be
con-ducted  in  a future  cooperative
agreement.

RESULTS AND DISCUSSION

  Bacterial  Dynamics   Modeling
Framework.   The  primary  goal  of
developing a .mathematical model of the
microbial   food  web   capable   of
predicting bacterial dynamics in surface
water  systems  has  involved  the
following tasks:

1)    a  review of   bacterial  growth
      kinetics in laboratory systems,
2)    development of a methodology
      for defining bacterial substrate,
3)    formulation  of the equation set
      describing the  dynamics  of the
      microbial food web, and
4)    application  of  the  modeling
      framework to a natural system.

  We have completed the first three of
these tasks and are nearing completion
of the fourth. The major conclusions of
this work follows.
  The utilization of dissolved  organic
carbon  (DOC) by  pelagic  bacteria is
dependent on the both the compounds
comprising the DOC and environment
specific growth  characteristics of  the
bacteria.    Laboratory  studies  have
shown that growth  yield coefficients
are related both to the energy  content
of the compound and the availability of
other  required  nutrients   such   as
nitrogen.   Half-saturation   constants
describing   substrate   or  nutrient
limitation of  growth rate appear to be a
function of environment,  higher values
being associated with  higher nominal
substrate levels.
  To  make  a  modeling  analysis   of
bacterial   growth   tractable   it   is
necessary to  define a  scheme  for
classifying DOC according to its ability
to be  used  as substrate.   We have
proposed three categories of DOC:  L1,
L2 and R carbon, in order of decreasing
lability. The fractionation of carbon into
these categories  may be accomplished
through a BOD assay in which breaks in
the oxygen utilization curve are  used to
signify the exhaustion of first  the  L1
and then the L2 component.
  Development  of the  BOD  assay
procedure is continuing  at the Gulf
Breeze EPA.  A methodology has been
established which appears to have the
needed  sensitivity  and  replicability.
Samples  (unfiltered  and  innoculated
sterile filtered) are  incubated at in situ
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temperature in the dark and  oxygen
utilization is  monitored  at  close  (1-2
hour) intervals.  A change  in slope of
the oxygen utilization curve is  used to
define exhaustion of L1  carbon.  Total
oxygen  utilization is used to establish
the degradable (L1  +L2) carbon. DOC
measurements   establish   the
oxygen:carbon  stoichiometry   and
bacterial counts (AODC) confirm  the
exhaustion of substrate and cessation '
of growth. This methodology  is being
applied   to  natural   water samples,
samples  spiked  with various  carbon
sources (e.g.,  algal exudate, sea grass
detritus)   and  will   be  applied  to
terrestrial carbon in non-point runoff
and to treatment plant effluent.
  Preliminary  laboratory assay data
suggest that the L1  substrate pool is
small.  The bacteria are  able to readily
use this substrate at maximum growth
rates calculated  to  be about  4/d.
Because  of  the  small   size  of  this
substrate pool and  its rapid use  it is
exhausted in the assays within a time
scale of hours or even minut'es.
  A review of experimental studies of
the fate  of  carbon  produced through
primary  production  indicates  that a
significant fraction of the DOC released
from phytoplankton through exudation,
cell lysis and sloppy feeding by grazers
would  be classified  as  L1  substrate.
Further, a portion of the detrital POC
generated through  grazing  appears to
be rapidly solubilized to L1 DOC.
  A  comprehensive   review   of
zooplankton  bioenergetics  has  been
used  to  establish  equations defining
trophic transfer  and loss of biomass
carbon.   Allometric   relationships
between consumption rate and  body
weight and  between respiration  rate
 and body weight are  being  used to
 define parameter values for the three
 grazer   levels   (nano-,   micro-   and
 mesozooplankton)  included  in   the
: framework.
   Use   of  the   L1-L2   substrate
 categorization, routing  phytoplankton
 carbon to these classes in accordance
 with published experimental results and
 specification of  bacteria  and grazer
 dynamics  according   to  a  Monod
 formulation successfully modelled the
 microbial dynamics  of a  microcosm
 study.

 Competition

   The  expansion  of  the modeling
 framework  to  include  GEMs  entails
 quantification of competition between
 the GEM and the indigenous population
 and-gene transfer  between them.  We
 have focused on modeling competition
 between bacterial  groupings.  Growth
 of each group  is defined  using  the
 Monod equations.  Competition results
 through  interaction with the substrate
 pools as defined by growth rates, yield
 coefficients   and   half-saturation
 constants.  In addition, differences in
 predation, as defined 5by group specific
 grazing  rates,  are  also   considered.
 Coexistence of an introduced organism
 and the  indigenous community or the
 isogenic  counterpart   of  the  GEM
 depends on  each of  the coefficient
 values and substrate specificity. It may
 be  necessary  to   model   multiple
 substrates to adequately  define  the
 potential of the GEM to survive and to
 invade the community.
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 FUTURE WORK

   We are continuing to  review the
 literature on zooplankton growth  and
 grazing.   We have found  that  the
 allometric  relationship  we used  for
 maximum ingestion rate underestimated
 rates  reported for microzooplankton.
 We  are  now  using   an  alternate
 relationship with published growth rate
 data   to  estimate  the   assimilation
 efficiency  of  ingested carbon.  Once
 this  has  been  completed  we  will
 attempt to recalibrate the Bagsvaerd
 microcosm  model   with  the   new
 parameters.
   A model of bacteria in the Delaware
 River estuary has  been formulated.  It
 consists   of  .the  bacterial   kinetic
 equations    which   we   previously
 developed and empiricalspecificationof
 phytoplankton dynamics.   Observed
 primary   production   rates   define
 substrate   input  to   the   system.
 Observed   phytoplankton  biomass
 concentrations  define   algal   prey
 concentrations for  the  zooplankton
 grazers. Segmentation of the estuary
 and bay has  been completed  and we
 are currently calibrating transport over
 the four month period being modeled
 (2/18/85 to  5/25/85).   Salinity data
 from  the five cruises conducted during
 this  period  are  being  used  as  the
 transport  tracer.     When   this  is
 completed we will begin to model the
 bacterial  dynamics  that   have  been
.observed in this system.
   The   laboratory  experiments   to
 examine bacterial uptake  of  various
 carbon  sources  (i.e.,  the substrate
 utilization  assay)   has begun to yield
 data.  We will analyze these  data to
 determine  the distribution of carbon
between operationally defined substrate
categories  and  bacterial  growth  on
these.
  We   will  continue  to   examine
competition between bacteria to better
define the processes controlling survival
and growth of an introduced organism.
  We   will  continue   to  prepare
manuscripts   covering   research
conducted during this project. The first
manuscript has  just been completed
and   a   manuscript  describing  the
Bagsvard  microcosm  modeling  study
will be started shortly.
  An   abstract • of   a   platform
presentation of our modeling work has
been  submitted  for consideration for
inclusion  in,  the- -Fifth-International
Workshop  oh  the  Measurement of
Microbial Activities in the Carbon Cycle
in Aquatic  Environments  "Microbial
Ecology of Pelagic Environments" to be
held   from  August  18-23,   1991  in
Helsingor, Denmark;

PUBLICATION

Connolly, J.P., R. B. Coffin  and R. E.
Landeck.   1991.  Modeling  Carbon
Utilization  by   Bacteria   in  Natural
Waters.   To be published  in Modeling
the Metabolic and Physiologic Activities
of Microorganisms  (Christen Hurst,
Ed.), John Wiley, New York.
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           MODELING TRANSDUCTION IN AQUATIC ENVIRONMENTS
            Robert V. Miller1, Tyler A. Kokjohn2, and Gary S. Sayler3
            Department of Microbiology1, Oklahoma State University
                             Stillwater, Oklahoma
         Environmental Research Division2, Argonne National Laboratory
                              Argonne, Illinois
        Center for Environmental Biotechnology3, University of Tennessee
                             Knoxville, Tennessee
INTRODUCTION

  With the recent advances of genetic
engineering have come new questions
about genetic  stability and exchange
among microbes in natural ecosystems.
We are using  Pseudomonas aeruginosa
as a  model organism to  study  viral-
mediated gene transfer (transduction) in
freshwater microbial populations.  Our
studies  have revealed  a  significant
potential  for  transduction  of  both
plasmid and chromosomal DNA in these
environments.  Initially, we explored
three   models  for  the  source  of
transducing   particles  in  freshwater
environments:  (a) cell-free lysates of
bacteriophages  grown   on   an
appropriate DNA  donor,  (b)  environ-
mental  induction  of bacteriophages
from a lysogenic DNA donor bacterium,
and  (c)  environmental  induction  of
bacteriophages   from   a  lysogenic
recipient bacterium.   The transfer of
plasmid and  chromosomal DNA was
documented in  each of these  systems
in  situ.    The highest   number  of
transductants were routinely recovered
from  systems  where the  recipient
bacterium was a lysogen, probably due
to   the  immunity  imparted  by  the
resident prophage. Transduction was
observed  in  both  the  absence  and
presence  of the  natural  microbial
community.   Reciprocal chromosomal
transduction was observed in chambers
inoculated   with  two   lysogens.
Apparently, both primary infection of a
non-lysogen  and  prophage induction
from a lysogen can generate sufficient
numbers of  transducing particles  to
allow gene exchange to be observed.
  For transduction to take place in this
system  a  unique  sequence of events
must take place, (a) Phage virions  must
be produced  through  spontaneous  or
stress-stimulated  induction  of  the
prophage from the lysogen.  (b) These
viral  particles must  infect, propagate,
and lyse the plasmid-containing donor.
(c)   Transducing particles produced
during this lytic infection  must absorb
and  transfer  DNA  to  the  remaining
lysogens.     Hence,   environmental
lysogens serve as both efficient sources
of transducing phages and as viable
recipients for  transduced DNA.

  The  finding that  transduction can
occur   in   natural  environments  is
significant as this mechanism  of  gene
transfer has been virtually  ignored  in
both the design and preliminary testing
of   genetically   engineered   micro-
organisms for environmental  release.
However, the ultimate question which
must be addressed in  determining the
effects of environmental transduction is
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whether   phage-mediated   horizontal
gene transfer alters the probability or
equilibrium frequency of an introduced
genetic sequence in a natural bacterial
community. We are now turning  our
attention   to   developing   a  model
transduction   system   whose
components are derived directly from
the environment and  to  determining
whether  transduction  is  effective in
altering the makeup  of the gene pool
available   to  natural   populations   of
bacteria.   These studies will allow a
more accurate assessment of the  real
potential for genetic exchange to affect
natural microbial populations.

METHODS

  Our investigations of horizontal gene
transfer   are   conducted   in   both
laboratory and field formats and  can be
divided into three stages.   First,  the
potential  for transmission is  evaluated
by standard genetic protocols. Second,
microcosm studies are conducted in the
laboratory to evaluate  protocols to be
tested  in  situ.   Third, field  trials  are
conducted in  biological  containment
chambers incubated at the freshwater
field site.  This stage is  used to validate
predictions made from the laboratory
simulation experiments. The potential
for  transduction  in   freshwater   P.
aeruginosa populations was  evaluated
using   a   variant  of  the  generalized
transducing  phage  F116  originally
designated  DS1.     For  studies  of
plasmid transfer, the Tra- Mob" plasmid
Rms149 is used.  Plasmid transduction
was confirmed  by molecular analysis.
Detailed methods have been  published
(see below).   Natural  bacteriophages
were isolated from lake water collected
at several of our test sites.  Samples
were  used   either directly  or  con-
centrated.  Phages were identified by
their ability to plaque on one or more
indicator strains.   Phages selected for
further studies were purified by glycerol
gradient  ultracentrifugation.     P.
aeruginosa strains were isolated from
lake water by means of selective media
and Pseudomonas Isolation Agar (Difco,
Detroit,  Ml) and further characterized as
appropriate.   In  some  experiments,
mixed populations  of either  bacterio-
phages  or  bacteria isolated from the
field sites were used.  Lysogens were
identified  by  their ability  to  release
phage either spontaneously or following
exposure to UV radiation and by their
superinfection-immunity phenotype.
  Continuous culture experiments are
conducted   in  a   New  Brunswick
Scientific    (Edison,   NJ)    BioFlo
Chemostat.   Culture medium consists
of Pseudomonas Minimal Medium Salts
without  citrate  supplemented with
yeast extract (10~5 to 10"7 g/ml).

RESULTS AND DISCUSSION

Developing a
Natural Transduction Model

  While  our  research  has  demon-
strated the potential for transduction in
natural  freshwater  habitats, it  is still
artificial as  its components  are  well
characterized laboratory strains.  We
are currently attempting to develop a
model system based  on components
isolated  directly from the environment.
  To    begin   this   study,  we
demonstrated  the   occurrence  of
bacteriophages,  potential  hosts,  and
lysogens in  the aquatic environment.
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We monitored these parameters in our
field sties for an eight month period.
During  this  period, we  found   total
bacterial counts rangee  from 105  to
107 CFU/ml and Pseudomonas counts
from 5 x 102 to 5 x 104 CFU/mi. Titers
of bacteriophages capable of forming
plaques  on laboratory  strains of  P.
aeruginosa ranged  from  101  to  104
PFU/ml.  When lysogeny was evaluated
using  a  laboratory  strain  of   P.
aeruginosa as a indicator, between 1
and  7%  of the Pseudomonas isolates
tested positive.   However, we found
that  approximately   45%   of
Pseudomonas isolates  from  our  field
site  tested   positive   in   colony
hybridization when probed with  DNA
from  a   naturally   occurring  phage
isolate.
  Second,  we  isolated  transducing
phages   from  our  environmental
samples.  One of these, UT1,  has been
studied in some detail. This phage was
isolated from Fort Loudoun Lake  near
Knoxville, Tennessee where  we  have
carried out the majority of our in situ
transduction  experiments.  Although
this  phage is virulent under laboratory
conditions,  it  establishes  pseudo-
lysogeny in the  environment. It  is a
generalized transducing phage capable
of  mediating  the  transfer  of  both
plasmid  (Rms149)  and  chromosomal
DNA among P. aeruginosa.  As might
be expected with a virulent phage, the
highest  levels  of   transduction  are
observed  at  very  low   MOIs (10"3
PFU/CFU).   Using standard laboratory
protocols,   both   chromosomal   and
plasmid transduction was detected  at
frequencies   as   high   as   10~4
transductants/PFU depending  on  the
MOI used.
   Third, we have used UT1  to initiate
 studies to determine the potential of the
 natural microbial community  present at
 our field sites to act as recipients of
 transduced plasmid DNA.  Cells from
 10  liters of lake water  were concen-
, trated and used as a  recipient pool for
 transduction using cell-free  lysates of
 UT1 grown  on a>P.  aeruginosa strain
 containing Rms149.  Significant levels
 of  transduction were observed  and
 confirmed   molecularly.     These
 observations have been made using
 samples  from  several different field
 sites  collected at  different  times of
 year.  They suggest that the natural
 microbial community at  our  field sites
 contains organisms capable of acting as
 hosts  and   recipients   for  plasmid
 transduction.
   For  transduction  to  take  place,
 interaction  between  the transducing
 phage and  its host  must take  place.
 We examined, the ability of bacterio-
 phages to interact with  their hosts at
 low cell densities and under starvation
 conditions.     The   attachment   and
 replication  of  three P.  aeruginosa
 bacteriophages were investigated under
 conditions similar to those  found in
 nature.  Attachment and replication of
 bacteriophages were  not impaired at
 host-cell densities  equal to  or  lower
 «105  CFU    ml"1}  than   those
 frequently found in  aquatic environ-
 ments  when  the  host cells  were
 physiologically  competent   to  allow
 phage growth.  .Attachment to  either
 actively  growing or starved  cells was
 not impaired in river  water,  indicating
 that attachment is efficient  in natural
 freshwater  habitats.    However,  the
 replication  of   bacteriophages  was
 significantly  altered .in. starved cells in
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river water;  the  latency  period  was
extended  (broth,  70-110 min.;  river
water,  110-240 min.), and  the burst
size was reduced (broth,  27-65;  river
water 5-7).  The findings of this study
indicate that phages are likely to affect
microbial   ecology  significantly   in
freshwater ecosystems.
  We isolated strains of P.- aeruginosa
which were sensitive and resistant to
UT1 from Fort Loudoun Lake and used
them to monitor host-phage interactions
over 45 days in lakewater microcosms.
Temporal  changes  in  host  density,
phage-to-bacterium ratio (PBR), and the
appearance   of  apparent  prophage
carriers within the host population were
analyzed.    About 45%   of  sensitive
bacteria  incubated  with   phage  UT1
were pseudolysogenic within  12 hours
of incubation in  natural  lake  water.
Phage  UT1  appeared to   stabilize  the
density x>f host bacteria in lake water at
a  level  of  104  CFU/ml.   Bacterial
coexistence  with  a  mixed  phage
population isolated from Fort Loudoun
Lake   resulted   in   an   oscillating
equilibrium with the PBR  stabilizing at
about 3.  The presence of extraneous
homoimmune phages  appeared to be
detrimental  to the  stability  of  the
pseudolysogens,   which  were
maintained  at a  lower  population
density than prophage-free cells in lake
water  containing  the mixed  phage
population.

A Mathematical Model
for Transduction

  We  are  currently  developing  a
mathematical model for transduction.
We  have  convincingly  shown  that
transduction  can  act to stabilize  (and
even  increase)  the  frequency  of a
genotype  in a population from which
that genotype would otherwise be lost.
Our research to date suggests that, in
populations  of   bacteria  in  which
transduction is occurring, the change in
the  frequency  of  a   transducable
genotype  (Pht)   as   a  function  of
generation of  growth   "g"  can  be
described  by the formula:

          dPht/dg = t - s

Where "t" is the fraction of new cells
added  to  the  "Pht"  genotype due to
transduction and "s" is the fraction due
to selection.

FUTURE WORK

  In the future, we plan to evaluate our
natural  model  system  in  situ  by
introducing a  plasmid donor  (either a
lysogen or pseudolysogen) and allowing
the  natural  community  to   act  as
recipients. We are currently developing
an appropriate contraselection for the
introduced donor which will allow us to
identify transductants  in the natural
population.    In  addition,  we  are
determining the physiological, environ-
mental, and   genetic  factors  which
regulate "t."  Preliminary analysis of our
data  suggest  that  "t"  cannot  be
described  simply  as  a   mass-action
phenomenon but contains  parameters
which  are highly  dependent  on the
physiological state of the hosts.  We
will continue to explore these factors in
the immediate  future.            '
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 PUBLICATIONS

 .Kokjohn,  T.A., G.S. Sayler, and  R.V.
 Miller.   1991.     Attachment  and
 Replication of Pseudomonasaeruginosa
 bacteriophages   under   conditions
 simulating aquatic  environments.   J.
 Gen. Microbiol. 137:661-666.

 Kokjohn, T.A., and  R.V. Miller.  1991.
 Gene  transfer  in  the  environment:
 transduction.  JnJ.C. Fry and M.J. Day
 (eds.),   Release   of  Genetically
 Engineered and Other Microorganisms.
 Edward Arnold, London, in  press.

 Miller, R.V.  1991.  Genetic Stability
 and Fate  Concerns on  Environmental
 Release,   in: A.P.  Hugenjoltz (ed.),
 Contemporary  Issues  in  Toxicology.
 Proc.   Twenty-Third   Symp.,   Soc.
 Toxicol. Canada. Montreal,  in press.

 Miller, R.V.  1991.  Methods  for the
 evaluation of  gene transfer  in  the
 environment;  general  considerations.
 in:  M. Levin, R. Seidler, and M. Rogul
 (eds.),  Microbial  Ecology:  Principles.
 Methods,   and   Application   in
 Environmental Biotechnology. McGraw-
 Hill, N.Y., in press.

 Miller, R.V.  1991.  Transduction in
 natural environments,  in: M. Levin, R.
 Seidler, and M. Rogul (eds.), Microbial
 Ecology:  Principles,  Methods,   and
 Application   in   Environmental
 Biotechnology.  McGraw-Hill, N.Y., in
 press.

 Saye,  D.J.,   and   S.B.  O'Morchoe.
 1991.  Evaluating  the potential  for
genetic exchange in  natural  freshwater
environments, in: M. Levin, R. Seidler,
 and M. Rogul (eds.), Microbial Ecology:
 Principles, Methods, and Application in
 Environmental Biotechnology. McGraw-
 Hill Pub.,.N,Y,, in press.

 Miller, R.V.,  and G.S. Sayler.  1991.
 Bacteriophage-host   interactions'   in
 aquatic systems,  in:  E.M. Wellington
 and  J.D.  VanElsas  (eds.),   Genetic
 Interactions Between Microorganisms in
 the  Microenvironment.     Univ.
 Manchester  Press,   Manchester,   in
 press.

 Ogunseitan,  O.A.,  G.S/  Sayler,  and
 R.V. Miller. 1990.  Dynamic interaction
 of   Pseudomonas  aeruginosa   and
 bacteriophages in lake water.  Microb.
 Ecol. 19:171-185.

 Simonson, C.S., T.A. Kokjohn,  and
 R.V. Miller. 1990.  Inducible UV repair
 potential of Pseudomonas aeruginosa
 PAD. J.  Gen.  Microbiol.  136:1241-
 1249.

 Saye,  D.J.,  O.A.  Ogunseitan,  G.S.
 Sayler,  and   R.V. Miller.    1990.
 Transduction  of linked  chromosomal
 genes   between  Pseudomonas
 aeruginosa during incubation in situ in a
 freshwater habitat.   Appl.  Environ.
 Microbiol. 56:140-145.

 Miller, R.V. 1990.  Increasing levels of
 environmental mutagens:  potential fbr
 affecting   viral   evolution   and
 pathogenicity-a  speculative  review.
 Environ. Carcinogen. Rev. 08:89-137.

 Miller, R.V., and T.A. Kokjohn.  1990.
 Microbiology and evolution of the recA
gene. Annu. Rev. Microbiol. 44: 365-
394!
                                    106

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Miller, Robert V., Tyler A. Kokjohn, and
GaryS. Sayler. 1990. Genetic Transfer
in Freshwater Environments, p. 72-77.
in:   Review   of  Progress   in   the
Biotechnology-Microbial  Pest Control
Agent Risk Assessment Program. U.S.
EPA, EPA/600/9-90/029.

Levy, S.B., and R.V. Miller.  1989.
Gene Transfer  in the  Environment,
McGraw-Hill, NY.
Saye, D.J.,  and R.V. Miller.   1989.
Gene transfer in aquatic environments.
p.  223-254.  In S.B. Levy and  R.V.
Miller (eds.), Gene  Transfer  in  the
Environment. McGraw-Hill, NY.

Miller, R.V.,  and  S.B.  Levy.   1989.
Horizontal gene transfer in. relation to
environmental release  of genetically
engineered microorganisms,  p. 405-
420.  In: S.B.  Levy and  R.V.  Miller
(eds.),   Gene   Transfer   in   the
Environment* McGraw-Hill, NY.
                                    107

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    ENVIRONMENTALLY INDUCED GENETIC INSTABILITY IN MICROORGANISMS
                     Tyler A. Kokjohn1 and Robert V. Miller2
  Environmental Research Division1, Argonne National Laboratory, Argonne, Illinois
 Department of Microbiology2, Oklahoma State University, Stillwater, Oklahoma
 INTRODUCTION

   Organisms   must   maintain  the
 integrity  of their  genetic  material  if
 species survival is to be assured.  It is
 now  apparent that  microorganisms
 possess  DNA  repair systems which,
 although  differing  in  capabilities and
 potentials,   act  to   repair  damage
 resulting   from  cell  metabolism  or
 environmental insult.
   Most of the data available concerning
 DNA  repair has  utilized  the enteric
 bacterium  Escherichia   coli  under
 conditions which probably will rarely, if
 ever,  occur in  natural environments.
 However,  some important  principles
 have been revealed by these studies.  It
 is clear  that  bacterial  cells suffering
 DNA  damage  will  undergo  repair
 processes.  In some cases, these repair
 systems are mutagenic.  Further, the
 efficiency and  activity of DNA  repair
 systems will be different under differing
 growth conditions.
   From work already performed, there
 can be no doubt that genetic systems
 exist   in  the  eubacteria  which  are
 induced   by   environmental  stress
 resulting in enormously increased rates
 of mutagenesis of the genetic material.
 The  existence  of such potentials for
 genetic instability complicates the task
 of risk assessment for  environmental
 releases   of  genetically   engineered
 microorganisms  (GEMs)  and  raises
questions  concerning the stability of
introduced   genotypes   in   natural
 ecosystems.  We  have been utilizing
 Pseudomonas aeruginosa as  a  model
 system to study stress-induced genetic
 alterations in bacterial cells.  The use of
 this model will allow a more  accurate
 assessment  of  risk  associated with
 GEM releases in the future.

 METHODS

   The frequency of mutation-containing
 cells in a population of P. aeruginosa
 was determined for cells  incubated in
 growth medium in  the  laboratory and
 for cells placed in
 in biological containment chambers and
 incubated either in the laboratory or in
 situ at our established field sites. The
 chambers utilized in these studies are
 permeable to gases  and allow the
 transmittance of solar  UV  radiation.
 They were  filled with  sterilized lake
 water   from  our  field  sites   and
 inoculated   with   genetically   well
 characterized  P.  aeruginosa  strains.
 Chambers incubated in situ were placed
 at locations receiving solar UV radiation
 for significant periods of the day or in
 locations  substantially shielded  from
 sunlight.
   Cells were recovered from  chambers
 by plating on one-tenth strength  YEPG
 agar.  Viable counts were determined
 for each sampling period.  In order to
 determine the frequency of  mutation-
containing  cells  in the  population,
samples (0.1 ml) were plated on 0.1  X
YEPG agar and incubated for 48 hours
                                     108

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to recover cells.  The cells were then
replica plated onto  various  selective
media.    Cells  were  screened  for
streptomycin  resistance,   rifampicin
resistance   and,  in   some   cases,
resistance to infection by the virulent
phage  UT1.    The   prptocols  for
determination of drug resistance have
been modified to include another round
of  replica  plating  on nonselective
medium  before  recovered  cells  are
plated on  media  containing antibiotics.
Individual drug resistant clones used for
further experimentation  were carefully
streaked for isolation.   Extra replica
plating  steps are  required  to  avoid
artifactual contributions to the apparent
mutation  rate  of   cells   that  are
physiologically  adapted  to  starvation
conditions but,  contain no  genetic
mutations to drug resistance.
  The ability of cells  existing  under
starvation conditions to resist  stress
was determined by  laboratory experi-
mentation. Cells were grown  in 0.1 X
YEPG at room temperature to stationary
phase.  The cells were diluted 1:10 in
sterile water from  a  field  site  and
incubated at room   temperature for
various periods of time.   After a  period
of  such  starvation, the cells  were
irradiated  with UV, and  the percentage
survival was determined for several
doses of UV  radiation.
  Induction of therecA gene product of
P. aeruginosa and £.  co//was examined
under several growth conditions using
the Western  Blotting technique.  Cells
were grown in Luria Broth, low-strength
medium (0.01 X YEPG) or starved as
described  above.    They  were then
irradiated with UV light.  Subsequent to
UV exposure, the cells were incubated
in the dark and samples were taken
periodically  to  be used  for Western
Blotting.      These   samples   were
electrophoresed  on  denaturing
polyacrylamide  gels and the proteins
transferred   to   nitrocellulose
membranes.  These membranes were
subjected to Western  analysis using a
biotintylated-streptavidin-horseradish
perox id ase-conju gated    antibody
detection system.  Anti-rec/4 E. coli
antibody  was  used  as  the  primary
antibody.
  Fusions   of   a  promoterless   P-
galactosidase gene to  stress-inducible
genes  of P.  aeruginosa PAO  were
constructed  using  a  modified  Tn3
transposon.  This transposon will insert
into target DNA yielding transcriptional
fusions  of the disrupted gene and  P-
galactosidase.
  For prophage induction experiments,
F116L or D3 lysogens of P. aeruginosa
were placed  in chambers and incubated
in the laboratory or  in  situ at sites
receiving solar UV irradiation. The ratio
of  infectious  centers  (1C)  to  total
colony-forming    units   (CFU)   was
determined to  detect  induction of the
prophages.  Cell  concentrations were
determined  as described above.  ICs
were enumerated by dilution in 0.85%
NaCI and plating  with  phage-sensitive
indicator strains.  In  some  laboratory
experiments, lysogens of E. coli were
treated in a similar manner.
  For starvation induction experiments,
a D3 lysogen of P.  aeruginosa was
grown to early  exponential  phase  in
Luria broth.  The cells were harvested
by  centrifugation  and  suspended  in
sterile river water to induce a putative
stringent response.  1C determinations
were  made  at   various  times  after
medium shift.
                                     109

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RESULTS AND DISCUSSION

Mutation Frequency

   Our   original   data  suggesting
increased rates of mutagenesis in situ
were   based   on  the   reversion
frequencies of amino acid auxotrophs.
Although no apparent selection for the
prototrophs could be detected, we were
concerned that unknown factors were
acting  to  increase   the   reversion
frequency.  Therefore, we have spent
some time  evaluating our procedures
for   determination   of   mutation
frequency. The experimental protocols
which we  are  now  utilizing  reflect
experience  gained   from   our  field
experiments.   Specifically  we  have
found   that expression  time   after
sampling the cells is required to detect
mutation events.  It has proven best to
recover cells on low-strength medium
(0.1 X YEPG) and apply selection later
by replica  plating  onto  appropriate
media.  The use of the replica  plating
technique allows a one-to-one mutation
to resultant colony detection and allows
testing of identical populations for the
frequency  of several different mutant
phenotypes.
   We  have  modified  our  screening
procedures  for  detection   of   drug
resistant mutants to  include a  longer
recovery  time  before  selection  is
applied.   Cells undergoing  starvation
stress apparently adapt physiologically
in such a way that their apparent drug
resistance is increased.  This change is
not necessarily genetic  in  nature.  We
have  clearly  been  able  to  isolate
Rifampicin-resistant mutants of at least
two distinct classes. By transductional
analysis we have determined one class
is resistant only to Rifampicin, the other
class  is  resistant  to  several  drugs
simultaneously. The exact frequency of
production   of  Rifampicin-resistant
mutations  subsequent   to   stress
application is unknown at present. Our
previous estimates were confounded by
artifactual contributions from adapted
cells   not  containing  true  genetic
alterations.
  Reversion  of  amino  acid   auxo-
trophiessubsequentto starvation stress
has proven to be reproducible.  Under
conditions of  no apparent  selection
advantage for revertants, we find rapid
production of prototrophic cells.  It is
unknown  if these are true revertants,
second site suppressor mutations,  or
expression of normally cryptic genes.
  Several different mutant phenotypes
have  been examined.   Screening for
rifampicin resistance (Rifr) has proven
to  be  ideal  since  Rifr  bacteria  are
infrequent at our field sites. The use of
streptomycin   resistance  is   also
convenient.    Mutation   to   phage
resistance is also an excellent marker.
For these studies, we have used the
extremely virulent environmental phage
UT1.   This phage is  ideal- since it is
absorbed  by and lyses even starved P.
aeruginosa  cells.    Other   virulent
laboratory phages were  unacceptable
since   changes  in  growth  condition
resulted   in  declined  virulence  or
reproduction potential.

Enhanced  Resistance to UV Stress

  The effects  of starvation  on the
response  of  bacterial cells  to  DNA
damage are  dramatic.   The  physio-
logical adaptations required for viability
maintenance result  in a tremendously
                                     110

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enhanced   ability  to resist  UV ir-
radiation.    Preliminary  experiments
suggest  that the genes  required  for
DNA repair  (recA, uvrABC, etc. in E
co//)  are  expressed  at  enhanced,
constitutive  levels.   Western Blotting
experiments performed on both  E. coli
and   P.   aeruginosa  support  this
conclusion.

Induction of Prophages

  Laboratory  and  field  experiments
have also demonstrated that starvation
of E. coli and  P.  aeruginosa lysogens
greatly affects DNA-damage induction
of  resident  prophages.  Preliminary
experiments using  the temperate P.
aeruginosa bacteriophage F116 have
suggested that conditions encountered
in natural aquatic environments  may
lead to enhanced activation of  lytic
phage  production in  these cells.  One
important question is whether this is a
general characteristic  of all  phages.
Our initial experiments have used three
UV-inducible temperate phages: D3 and
F116 of  P. aeruginosa and of E.  coli.
Field experiments have demonstrated
no increased induction of D3 prophages
from lysogens of P. aeruginosa exposed
to significant, long term solar irradiation
as measured by the frequency of ICs in
the  population.    Laboratory  investi-
gations have  confirmed  that starved
cells lose the ability to support  UV
induction   of   prophage.     This
phenomenon has been  observed  for
both E. coli and P. aeruginosa and has
been   termed   "aptitude"  by  Andre
Lwoff.    Significantly, we  find  that
starved lysogens  of P. aeruginosa are
capable of the  spontaneous production
of phage particles for long periods.  In
contrast, lysogens  of  E.  coli rapidly
become inviable under the same growth
conditions.

Stress-lnducible Expression
of P. aeruginosa Genes

   To aid in the quantification of levels
of expression of DNA-damage inducible
(din]   genes   under   environmental
conditions  such  as  starvation  and
exposure  to  solar UV,  we  have
constructed and partially characterized
transcriptional fusions  of various P.
aeruginosa PAO  genes to (5-galacto-
sidase (/acZ) which are inducible by
exposure  to  DNA  damaging  agents.
These genes are  clearly expressed at
greatly increased  levels subsequent to
UV irradiation of  Rec+  P. aeruginosa
and are inducible by  other agents which
damage DNA as  well in this species.
They are not inducible when transferred
into E. coli.  The molecular mechanism
of this anomaly is currently under
investigation.

Plasmid-Encoded UV Resistance

  Laboratory  studies   of  the   UV-
resistance plasmid  R2 have  revealed
that it encodes a true /-ec/4-dependent,
DNA  damage-inducible  repair system
which is highly mutagenic.  Laboratory
and field studies of  P. aeruginosa  cells
containing the  UV-resistance  plasmid
R2  have  demonstrated an  enhanced
mutagenesis  activity under  environ-
mental  conditions   which  result  in
exposure  to  solar-UV  radiation.   An
understanding of the contribution of
these plasmids to genetic instability of
natural populations of bacteria and  their
effects  on  novel genetic sequences
                                     111

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 introduced into these environments by
 the release of GEMs is absolutely vital
 in risk and fate assessment.

 FUTURE WORK

   We will continued to investigate the
 existence  of a hypermutable state in
 cells  undergoing  starvation  stress.
 Auxotrophic revertants will be mapped
 to better characterize their nature.  We
 will continue to examine the expression
 of UV resistance plasmids exemplified
 by R2.

 PUBLICATIONS

 Kokjohn,  T.A., G.S. Sayler,  and R.V.
 Miller.     1991.     Attachment  and
 Replication of Pseudomonas aeruginosa
 bacteriophages   under  conditions
 simulating aquatic  environments.   J.
 Gen. Microbiol'. 137:661-666.

 Kokjohn, T.A., and  R.V. Miller.  1991.
 Gene  transfer in  the  environment:
 transduction. In: J.C. Fry  and  M.J. Day
 (eds.),    Release   of  Genetically
 Engineered and Other Microorganisms.
 Edward Arnold, London, in press.

 Miller, R.V. 1991.  Increasing levels of
 environmental mutagens:  Potential for
 affecting   viral   evolution  and
 pathogenicity -- a speculative review.
 Environ. Carcinogen. Rev. C8:89-137.

 Miller, R.V.   1991.  Genetic Stability
and  Fate Concerns on Environmental
Release,   in:  A.P. Hugenjoltz (ed.),
Contemporary Issues  in  Toxicology.
Proc. Twenty-Third Symp.  Soc. Toxicol.
Canada, Montreal, in press.
 Simonson,; C.S.,  T.A. Kokjohn,  and
 R.V. Miller.  1990. Inducible UV repair
 potential of Pseudomonas aeruginosa
 PAO. J.Gen.Microbiol. ,136:1241 -1249.

 Miller,  R.V., T.A. Kokjohn,  and  G.S.
 Sayler.  1990.    Environmental   and
 molecular  characterization of systems
 which  affect  genome  alteration  in
 Pseudomonas, pp. 252-268.  in:  S.
 Silver, A.M. Chakrabarthy, B. Iglewski,
 and S.  Kapland (eds.), Pseudomonas:
 Biotransformations. Pathoaenesis. and
 Evolving  Biotechnology.  Amer. Soc.
 Microbiol., Washington, D.C.

 Miller,  R.V. ,1990.   Estimating  the
 stability of gene  inserts:    Microbial
 systems, p. 61-72. in: D. Mahon (ed.),
 First  Consultative   Workshop   on
 Assessing  Safety in  Foods Derived
 Through Biotechnology.   Health  and
 Welfare Canada, Ottawa.

 Miller, R.V., and T.A.  Kokjohn.  1990:
 General Microbiology of recA:  Environ-
 mental and Evolutionary  Significance.
 Annu. Rev. Microbiol.  44: 365-394.

 Kokjohn, T.A., and R.V. Miller.  1990.
 Environmentally   induced   genetic
 instability in microorganisms, p. 110-
 114.  in: Review  of  Progress  in  the
 Biotechnoloav-Microbial  Pest Control
 Agent Risk Assessment Program. U.S.
 Environmental   Protection   Agency
 A/600/9-90/029

 Kokjohn, T.A.  1989.  Transduction:
 mechanism  and  potential  for  gene
transfer in the environment, p. 73-99.
in:  S.B.  Levy  and  R,V. Miller (eds.),
Gene Transfer in  the  Environment.
McGraw-Hill, N..Y.
                                    112

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            GENOMIC PLASTICITY AND CATABOLIC POTENTIAL OF
                           Pseudomonas cepacia

        T.G. Lessie, A. Ferrante, A. Byrne, M.S. Wood, and H.-P. Cheng
                         Department of Microbiology
                         University of Massachusetts
                          Amherst, Massachusetts
INTRODUCTION       ,

  The   genome   of   the   bacterium
Pseudomonas cepacia contains a large
number of  insertion sequences.  The
abilities of these elements to  mediate
genomic rearrangements and  activate
the expression  of neighboring genes
have been implicated in the evolution of
novel catabolic functions and appear to
underlie  the extraordinary  nutritional
versatility  and   adaptability'  of  this
bacterium.  We have been particularly
interested in the roles of IS elements in
the recruitment of foreign  genes for
new degradative pathways  as well  as
their capacity to be transferred to and
transpose and activate gene expression
in other bacteria.
   In order to gain information about the
relationship of P.  cepacia IS elements
and  insertion  sequences  from  other
bacteria  we  have  determined  the
nucleotide sequences of \S401, \S402,
\S406, and \S407 and compared them
to elements listed in. the GenBank and
EMBL data bases.  The four elements
examined had been shown to  promote
rearrangements of a  cryptic  plasmid
present in P. cepacia 249 or to activate
the  expression of the lac genes  of
Tn357  when   this  transposon  was
introduced into P. cepaciaon the broad-
host-range  plasmid 6GC91.14.  The
data obtained  has also been  used to
construct \S402  and  IS407  variants
carrying the  trimethoprim  resistance
(TpR) gene of pR388, which are being
used to examine factors  influencing
transpositional activity.
  To   gain   information   about  the
organization of the P. cepacia genome
we have undertaken the construction of
a physical  map of the chromosome of
strain 249.  Our aims are  to examine
the distribution  of  IS  elements and
genes encoding key catabolic enzymes.
Such   information   should   provide
insights into the bases for the unusual
catabolic potential of this bacterium.

METHODS

Analyses Of the Nucleotide Sequences
of Selected IS Elements

   Insertion  sequences  were  cloned
from element-containing derivatives of
pRP1    and    pGC91.14   into   the
sequencing   vector  pBLUEKSP  and
inserts of suitable length for sequence
analysis were generated by subcloning
DNA fragments or by creating  nested
deletions.   Nucleotide  sequence data
was obtained by the dideoxynucleotide
procedure of  Sanger using ccc-DNA as
template.  The data were compiled and
analysed on  a  MicroVAX computer
using  programs   provided  by  the
Genetics  Computer  Group  at  the
University of  Wisconsin.
                                    113

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 Construction of Marked IS Elements

   The nucleotide seque/ices of \S402
 and  \S407 were examined to  identify
 unique restriction enzyme sites located
 outside  open  reading  frames  and
 terminal inverted repeat sequences that
 might be  important for transposition.
 Two such sites were identified: a Dra\\
 site within \S402 and a Dra\ site within
 \S407.     A  0.7-kb  trimethoprim
 resistance (TpR) cassette from pR388
 was ligated into these sites to form the
 derivatives \S402.2 and \S407.1, which
 each carry the dihydrofolate reductase
 (fol)  gene of pR388, and  accordingly
 confer  high  level   resistance   to
 trimethoprim.

 Physical Mapping of the Chromosome
 of P.  ceoacia 249

   Bacteria were immobilized in agarose
 plugs   and   lysed   under   alkaline
 conditions by treatment with SDS and
 proteinase K.  The plugs were washed
 in TE buffer and the chromosomal DNA
 was digested in  situ with restriction
 enzymes such as Pac\, Afl\\, Xba\, and
 Dra\,  which  cleaved the  P. cepacia
 chromosome  into a  relatively  small
 number  of  high  molecular  weight
 fragments.  The  agarose plugs were
 transferred into wells on agarose gels,
 and the DNA fragments were resolved
 by pulsed-field gel electrophoresis using
 a CHEF gel apparatus  obtained from
 Owl  Scientific  Co.,  Cambridge, MA.
 Yeast chromosomes  and concatamers
 of coliphage lambda DNA were used as
 size  standards.   Neighboring  Pad
 fragments were identified by Southern
 hybridization experiments using as DNA
probes Af/ll and Xba\ fragments  which
 overlapped the pertinent Pac\ sites.

 RESULTS AND DISCUSSION

 Relationship between Elements from
 P. ceoacia and other Bacteria

   The   results  of  our   nucleotide
 sequence analyses indicate that \S401
 and  \S407 are members of  the IS3
 family of insertion sequences, whereas
 \S402  and  \S406 are  distinct from
 previously described  elements.   The
 results  indicate that at least some of
 the IS elements present in P.  cepacia
 have been widely dispersed  among
 bacteria.  Table 1  compares some of
 the characteristics of \S401 and \S407
 and other IS3-like  elements.  \S407,
 \S476, and  IS/?7 comprise a subgroup
 of the IS3 family.  All three of these
 elements produced 4-bp duplications of
 target DNA upon insertion. IS407 was
 most closely  related to  \S476.  The
 deduced amino acid sequences of the
 major open reading frames of these two
 elements  exhibited between 72 and
 82% similarity.  \S407 was  typical of
 the majority of IS3-like elements in that
 it  generated 3-bp target duplications.
 \S40J  exhibited  a  high degree  of
 homology with IS57 and with a region
 of the Ti plasmids of  Agrobacterium
 tumefaciens   important  for  tumor
 induction in plants.  In this context it is
 of  interest  that certain  Ti  plasmids
 confer ability  to  utilize  octopine,  a
 compound which supports rapid growth
 of P. cepacia.  It is possible that \S401
 might have been involved in acquisition
of octopine utilization genes.
                                    114

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  Table 1.  Characteristics and sources of IS3-like elements
Element
IS407
\S476
\SR1
\S401d
\S51
\S3411
IS3
\S911
Size
(bp)
1236
1225
1260
1316
1311
1309
1258
1250
IRsa
(bp)
12
26
13
26
26
27
39
27
Target
repeats'3
4
4
4
3
3
3
3
3
Percent0
Hornology
100
62
55
44
40
39
38
38
Source
Pseudomonas cepacia
Xanthomonas campestris
Rhizobium lupini
P. cepacia
Pseudomonas syringae
E. coll
E. coli
Shigella disenteriae
    Terminal inverted repeats
    Directly repeated duplications of target site DNA
    % Overall nucleotide sequence homology with IS407
    \S401 was most closely related to IS57 (65% homology)
Transposition  of  \S402  and  \S407
Variants in E. coli

  Donor strains containing pBluescript
vectors carrying \S402.2 and \S407,1
(variants   conferring   resistance   to
trimethoprim) as well as IncPI plasmids
conferring  resistance to  tetracycline
were mated with E. coli and P. cepacia
recipients, Transconjugants containing
cointegrate  plasmids  were  obtained
readily.  \S402.2, a variant  with the
TpR gene of  pR388 inserted into the
unique Drall site of \S402, was ligated
into   pTG.LI 33,  an  IncPI   plasmid
temperature sensitive with respect  to
its replication to form pTGL154. E. coli
derivatives bearing  pTGL154, when
propagated at 42°C,  gave rise to TpR
transposants  lacking the TcR marker of
the donor plasmid. The results suggest
that \S402 and \S407 can transpose in
E.  coli,  We are analysing the putative
tranposants to confirm that this is the
case.

MgcrQrgstriction Analysis of the
P.  cepacia Genome

  The  restriction  enzymes Pac\, Dra\,
and  Ase\   cleaved  the  P.   cepacia
chromosome into  6,   41,   and  35
fragments, respectively.  The sums  of
the molecular weights of the fragments
indicated that the  size of the P. cepacia
chromosome is  4.9 Mb.   Thus the
unusual  degradative abilities  of  this
bacterium are not a consequence of its
having a large genome than that of less
versatile  bacteria.   Prototrophic  and
auxotrophic derivatives were identified
in  which  large deletions resulted  in
                                     115

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 fusion of neighboring Pad fragments.
 Afl\\  and  Xba\ fragments  containing
 Pad sites have been used in Southern
 hybridization  experiments  to define
 linkages between  Pad fragments.  All
 six  Pad  fragments  were shown  to
 comprise a single linkage group.   We
 have carried out Southern hybridization
 experiments  to   determine   the
 distribution of \S401, \S406,  \S407,
 \S408, and \S415. All copies of these
 elements   were  clustered  within  a
 limited region of the genome comprised
 by the two smallest Pad fragments.
 Analyses   of  the   distributions  of
 additional  elements  and  of  essential
 genes  should  reveal whether  such
 clustering   reflects  the   location  of
 essential genes in  other regions of the
 chromosome or perhaps organization of
 elements  into  domains   where  their
 transposition  can  be  regulated by
 localized changes  in DNA topology --
 e.g.   alterations  in   extent    of
 supercoiling.

 FUTURE WORK

   Our long term goal is to understand
 the  factors  underlying  the  unusual
 adaptability of P.  cepacia.   We  are
 particularly interested in examining the
 roles of IS elements in the evolution of
 catabolic pathways.  Towards this end
 we will extend the  Pad restriction  map
 we have generated  to include more  data
 about the distribution of IS elements as
 well as the locations of genes encoding
 various   biosynthetic  and  catabolic
 pathway  enzymes.  A major aim  is
 understand how P. cepacia maintains
 relatively stable phenotypes despite its
 potential   to  undergo   frequent  IS
element dependent rearrangements of
 its genome.  Our working hypothesis is
 that transpositional activity is triggered
 in  response to environmental stress --
 e.g. prolonged starvation for nutrients
 and exposure to  toxic agents.    To
 examine   whether  transposition  -is
 regulated and  gain information about
 functions required for transposition we
 are  constructing   strains  whose
 chromosomes  contain  IS   elements
 carrying  a  TpR  resistance casette.
 These will be used to obtain data about
 the influence of various physiological
 conditions on  transposition.    (The
 transposition assay entails  transfer of
 broad-host-range  plasmids  from  the
 pertinent  strains  and  screening  of
 transconjugants for acquisition of the
 TpR  determinant  of  the   marked
 elements). It should also be possible to
 use such strains to gain information
 about the factors governing exchange
 of  P.  cepacia elements in  complex
 bacterial populations.

 PUBLICATIONS

 A.  Ferrante, and T.G.  Lessie.  1991.
 Nucleotide sequence  of  \S402,  a
 transposable  gene-activating element
 from Pseudomonas cepacia.  Gene.  In
 press.

 Wood, M.S., A. Byrne, and T.G. Lessie.
 1991.   Characteristics of \S406  and
 \S407,  two   lac  gene   activating
 elements from Pseudomonas cepacia.
 Submitted for publication.

 Wood, M.S., C. Lory, and T.G. Lessie.
 1990.  Activation of the lac genes of
Tn951  by insertion  sequences from
Pseudomonas  cepacia.  J.   Bacteriol.
 172: 1719-1724.
                                    116

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Lessie, T.G., M.S. Wood, A. Byrne, and
A. Ferrante. 1990. Transposable gene
activating  elements in  Pseudomonas
cepac/a. • p. 279-291.  in:  S.  Silver,
A.M. Chakrabarty, B. Iglewski, and S.
Kaplan   (ed.),    Pseudomonas:
Biotransformations. Pathogenesis, and
Evolving  Biotechnology.   Am.  Soc.
Microbiol., Washington, D.C.
                                    117

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       STUDIES ON CONJUGAL TRANSFER OF PLASMIDS FROM GEMs TO
                       INDIGENOUS AQUATIC BACTERIA
                                Tamar Barkay
                     U.S. Environmental Protection Agency
                       Environmental Research Laboratory
                              Gulf Breeze, Florida
 INTRODUCTION
   The horizontal spread of recombinant
 genes  from  genetically  engineered
 microbes may magnify ecologicalrisks
 because their stability and expression
 are likely to increase in microorganisms
 indigenous to  aquatic and terrestrial
 environments.     Two  experimental
 approaches  have   been  used   for
 evaluation of  gene  transfer  in   the
 environment:     i.  A  retrospective
 approach comparing clonal identity with
 plasmid  patterns  of  bacterial  pop-
 ulations  (Selander and Levin, Science
 210:545-547(1979)).  ii.  The addition
 of  genetically  marked   donor   and
 recipient  strains   to  environmental
 samples and the scoring of recombinant
 strains.   Due to problems posed  by
 background indigenous microbes  this
 last approach employs either environ-
 mental  enclosures   or  sterilized
 environmental  samples.    Both  ap-
 proaches indicate that gene exchange
 does occur, albeit at low frequencies, in
 the environment (Levy and Miller (eds.),
 Gene   Transfer  in  the  Environment.
 McGraw-Hill  Publishing  Co. (1989)).
 However, the use of marked laboratory
 strains may underestimate frequencies
 of transfer because the strains are not
 adapted to life in the test  environment.
Thus, methods are needed to detect in
situ horizontal gene transfer between
indigenous microbes.  Here, I describe
 an  experimental  approach  for  the
 detection of  gene transfer from  an
 engineered   organism  to  microbes
 indigenous to aquatic environments.
   The  approach  is  based  on  the
 assembly of catabolic pathways when
 the  donor  and  recipient  genes are
 brought together through the formation
 of  recombinant  strains.    If  genes
 encoding part of  the  pathway are
 placed on a  genetic element whose
 transfer is followed, and the indigenous
 community is acclimated to carry out
 the  remaining reactions of the same
 pathway,  then  strains  with  new
 catabolic capabilities would  emerge
 when indigenous microbes receive and
 replicate the transferred genes. I have
 used the assembly of merB (encoding
 organomercurial  lyase) on broad host
 range  conjugal  plasmids,  and  merA
 (encoding  mercuric  reductase);  to
 detect gene  transfer from a donor
 pseudomonad   strain    to   Hg(ll)
 acclimated aquatic  microbes  by  the
 ability  of transconjugants  to grow in
 presence  of   an   organomercury
 compound,  phenyl-mercuric  acetate
 (PMA).    This  is  based  on  a  prior
 demonstration that Hg(ll) acclimated
 microbial communities are enriched for
 microbes 'that reduce Hg(il)  by the
activity  of  the  mercuric  reductase
enzyme and carry merA genes (Barkay
et  al.,  Appl.  Environ.   Microbiol.
56:1695-1701 (1990)).
                                    118

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MATERIALS AND METHODS

  Construction of conjugal broad host
range plasmids containing a /we/B-plant
DNA cassette: Plasmids, pGTE15 and
pGTE22, containing the merB gene of
the broad spectrum mercury resistance
plasmid R831 and a 350 base pairs (bp)
sequence  of the  plant  napin  gene,
bracketed by multi cloning sites of the
pUC  plasmids  were  prepared.  The
/nerB-plant  DNA  cassette was excised
and  cloned into  appropriate cloning
sites of the following broad host range
plasmids, R388 (IncW), RP1  (IncP) and
pKM101  (IncN)   to  create  plasmids
pGTE16, pGTE26 and  pGTE25,  res-
pectively   (Figure  1,  page   120).
Conjugal transfer  of  parental  and
recombinant plasmids between E. coli
and pseudomonad strains was tested
by  the  filter  conjugation  test  as
described   by Simon   et  al.,   (Bio-
technology  1:784-791 (1983)).
  Conjugal  transfer of merB-plant DNA
plasmids to  aquatic microbes:  The
experimental procedure employed is
outlined in Figure 2 (page 121). Fresh-
water bacteria were acclimated to Hg(ll)
using conditions described by  Barkay
(Appl.   Environ.   Microbioi.   53:
2725-2732  (1987)).   P.  aeruginosa
containing  the test  plasmid (approx-
imately 107 cells/ml) were  added to
acclimated water samples, and samples
were  filtered  through  0.2  jj  nitro-
cellulose filters   using  low  vacuum
pressure. This treatment did  not injure
either donor or  acclimated  microbes.
Following a 4 hour incubation (a period
demonstrated  to allow for  conjugal
transfer), the cells were suspended in 1
ml broth, Hg(ll) was  added to 0.1 //M
and suspensions were incubated for 30
minutes prior to  plating  on selective
media. This incubation in presence of
a sublethal concentration of Hg2+ was
essential  for  expression  of mer and
subsequent selection of PMA resistant
transconjugants.   Conjugation   with
microbes  from control communities
(incubated  in  the  laboratory  in  the
absence  of  Hg   )  were  used  as
negative controls.  Colonies grown on
PMA plates (10//g/ml) were confirmed
as transconjugants by i.  transfer  to
medium containing antibiotics (Tp - tri-
methoprim (1000//g/ml), Cb - carben-
icillin (600 //g/ml), and Kn - kanamycin
(600//g/ml), for pGTE16, pGTE25 and
pGTE26,  respectively), and ii.  hybrid-
ization with  the  plant  DNA  probe
(described by Barkay ibid).

RESULTS AND DISCUSSION

  Evaluation of enrichment (Table  1)
indicates that whereas Hg(ll) resistant
bacteria were selected during  the  2
days acclimation  period, selection for
PMA  resistance did  not occur. Such
selection was anticipated because merB
is  a  part  of   the  mer operon that
includes  merA,  the  gene  enriched
during acclimation (Barkay et al., 1989.
Appl.   Environ.   Microbiol.  55:
1574-1577).  Coselection  would  have
resulted in a high  background of PMA
resistant   bacteria  and  a  possible
masking of transconjugants.
  Results   of   conjugation  between
PA01(pGTE16)  and indigenous Hg(ll)
are presented in (Table 2).  Presumptive
transconjugants were present at 10  to
103 CFU/ml whereas background PMA
resistance was below detection. Thus,
there   is   at   least  two  orders  of
magnitude difference between the
                                    119

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                                                                             Hlndill
                                                                                EcoRl
                                                           Hlndill
                                                         EcoRl dlgttt
                                                                  Hlndill
                                                          Km
                                                  2.3kb
                                          _  _.x  B«lhHI
                                          EcoRI\frigrn»nt   «"8««<
                                                     .,  ,: E!
Figure 1.      Construction of Broad Range Plasmids with a mer B-Plant DMA Cassette
                                          120

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                    Freshwater samples
                      £        ~3-£
                    +25Q|Lig/L
                                 Control
                                  NoHg2*

                            y 2 days Incubation at 30°C
             Enumeration of total, HgR and PMAR CFU/ml.
20 ml water sample 2x106 cells of P. aeruginosa PA01 (merS-plant DNA plasmid)
                      : filter - 0.22^ Type GS filter

                               1 4 hours incubation at 30°C    ;


                    Cells suspended in 1ml 1/2PCB
                        HgCI2 added toO.

         .'       •'-•    --''"   'i
                              incubation at 30°C for 30 minutes
        Plating on:  1/2PCA with PMA (25 \iM) -+* Transconjugants
                   172PCA with Hg2+ (25 ^M) -»- Potential recipients
                   1 /2PCA with antibiotics •'  r^- Donors
                             2 days • 30°C
        Confirm conjugation by:
         i.  Growth in presence of antibiotics
         ii.  Hybridization with a eukaryotlc DNA probe
Figure 2.    Conjugal Gene Transfer From GEMs to Aquatic Bacteria
                                 121

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 Table 1. Enrichment of Hg(ll) and PMA resistant heterotrophic bacteria after exposure
 to 250 M9/L Hg(ll) for two days at 30 • C.
                                    Exp.1
                                                CFU/ml
Exp. 2

Day 0


Day 2



Total
Hg2 + -R
PMA-R
Total
Hg2+-R
PMA-R
+ Hg(ll) Control
3.3x1 04
4.8x1 02
2.X10'2
4.0x1 05 3.2x1 04
8.6x1 04 ' 1.0x102
<10.0 <0.01
+ Hg(ii)
5.4x1 04
1.2x101
<0.01
4.7x106
8.0x1 02
0.9
Control



6.7x1 04
<1.0
<0.01
Table 2.  Results of conjugation experiments between PA01(pGTE16) and indigenous
Hg(ll) resistant indigenous bacteria.

Donor
"Recipient"
(Hg2+-R)
Transcon.
Background
(PMA-R,
no donor)
Efficiency
(trans./donors)
+ Hg(ll)
3.1x106
ND2
6.0x1 02
<1.25
1.9x10'4
(< 4.0x1 0'7)3
Control
4.0x1 07
ND
<1.25
<1.25

+ Hg(ll)
7.8x1 06
9.5x1 03
3.5x1 02
<1.42
3.6x1 Q-2
Control
1.2x107
ND
ND
ND
recipient
1  Hg(ll) - indigenous flora from exposed microcosms; Control - indigenous flora from
microcosms not exposed to Hg(ll).
2  ND - not determined
3  appearance of background PMA-R/donor
                                     122

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number   of   transconjugants   and
background PMA resistant organisms,
allowing  for  unmasked detection of
transconjugants. Efficiency of transfer
was at the  range of  10~4  to  10~5
transconjugants/donor. In comparison,
the efficiency of pGTEt 6 transfer to an
E.  coli   HMS174  that  contained
pACYC184::Tn501   (encoding  Hg(II)
resistance   and   compatible   with
pGTE16) was 3x10"3 per donor.  When
the  number   of  transconjugants  is
related  to  the  number  of  potential
recipients  (in  Exp.  II,  Table  2),  it
appears that 3% of the Hg(ll) resistant
strains  received  pGTE16.     Thus,
experiments using  standard laboratory
strains may overestimate efficiencies of
transfer.      All   PMA  resistant
transconjugants  were  found  to  be
resistant to Tp at 1000 //g/ml and they
hybridized with the plant DNA probe,
confirming that they were formed by
conjugation   between   PA01   and
indigenous bacteria. When the number
of transconjugants is  related  to the
number of potential recipients (in Exp.
II, Table 2),  it appears that 3% of the
Hg(ll)   resistant   strains   received
pGTE16, indicating  that  there  is  a
substantial potential for transfer'of this
IncW plasmid to indigenous flora.
  In  summary, the utility of catabolic
gene assembly as a tool for detection of
conjugal   transfer   of   recombinant
plasmidsto indigenous micro-organisms
was   demonstrated  under   optimal
conditions.   This approach is currently
applied  to   bacterial populations  in
environmental samples.   This  experi-
mental system will allow determinations
of   the   effect   of  environmental
parameters  on  transfer  of conjugal
plasmids.
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    THE FATE, STABILITY & MOVEMENT OF FOREIGN DNA IN FILAMENTOUS
                     FUNGI: AN ENVIRONMENTAL STUDY
                     Martin B. Dickman1 & John F. Leslie2

                        Department of Plant Pathology
                            University of Nebraska
                              Lincoln, Nebraska1
                        Department of Plant Pathology
                            Kansas State University
                             Manhattan, Kansas2
INTRODUCTION
  Genetically engineered fungi (GEF)
offer great potential as tools with which
to study the fate of recombinant DNA
{rDNA)  in  the  environment.   While
techniques have been  developed to
detect   and   monitor   genetically
engineered bacteria, progress has been
less rapid with fungi.  Presently there is
no eukaryotic model for studying the
effects resulting from the introduction
of rDNA into an ecosystem via a fungal
vector.     As  simple,  but   true,
eukaryotes, GEFs  provide a  unique
opportunity  for  studies   of  strain
development  and   use  under  field
conditions.   Prior  to   environmental
release,   however,   detailed   risk
assessments  are needed  to  design
rational strategies for safe release.
  We have been using  both molecular
and genetic techniques to study the
impact of GEFs in the ecosystem.  In
order to predict the potential impact of
a GEF following introduction  into the
environment, it is critical to evaluate the
consequences of foreign DNA insertion
into the organism.   We have focused
our studies on the  stability  of such
foreign  sequences following  meiosis
and mitosis, and are using sequences
with and without significant homology
to the recipient's genome to determine
if the origin of the foreign sequences
plays a  significant role in determining
stability.
  For our studies we have been using
fungal strains identified as Gibberella
fujikuroi  mating   population   "A"
(Sawada)  Ito  in  Ito  &  K.  Kimura
(asexual stage Fusarium moniliforme
Sheldon).  This organism can be readily
manipulated   under   laboratory
conditions,    and   sexual   crosses
completed with ease in a 4-6 week time
frame.   Numerous markers,  including
resistance  to   hygromycin   B  and
benomyl®, as  well  as  auxotrophs and
spore color  variants,  are available in
strains that we have developed.  Also,
this  fungus  can be divided into  over
1000 different vegetative compatibility
groups  (VCGs).  At  least  10 genes
underlie this VCG system and  they
determine whether stable heterokaryons
can be formed and appear to  limit the
degree  to which asexual  transfer  of
genetic  material can occur.  We have
also developed a reliable DNA-mediated
gene transfer system for this  fungus.
These advantages make this organism
a natural candidate for studies of the
fate of foreign DNA in the environment.
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  Our  overall objectives  are:  a.) to
determine   conditions   for   stable
maintenance of  rDNA in  filamentous
fungi and identify factors that influence
genetic stability, and b.)  to determine
parameters affecting genetic transfer of
rDNA  within  and  between   fungal
populations.
  Specifically we will:  a), determine if
copy number affects the stability of the
foreign  DNA through meiosis  and
mitosis, and b). Determine if rDNA can
be   passed   within   and  between
populations of  G.  fujikuroi through
asexual or sexual genetic exchange.

METHODS

Strains and Plasmids

  All Gibtrerella fujikuroi strains used in
this study belong to mating population
"A"  (variety moniliformis) and were
fertile as males.  Most of the  strains
were also female fertile.  Strains were
stored  in  15%  glycerol  at  -80°t:;
vegetative cultures were maintained on
complete medium.    For  hygromycin
resistance studies, the vector pBHM-1
containing   the   hygromycin   B
phosphotransferase  resistance  gene
from   E.   coli  fused  to  promoter
sequences   from    Cochliobolus
heterostrophus was used.  For  studies
of benomyl  resistance the Neurospora
crassa  altered  p-tubulin  gene  in the
vector  pBT6, was used.   In addition,
we have cloned as mutant P-tubulin
gene from G. fujikumi (see below).

Cultural Practices  ,-.    .

  .Resistance to  the  amino glycoside
antibiotic hygromycin  B  was   scored
three  days following  transfer  from
complete medium  to  minimal medium
plus 100  mg/L  of the  drug.   Plates
containing  hygromycin  B were  made
fresh and  not  kept for more than two
weeks because  the drug's  inhibitory
properties in the media  decrease with
time.  Resistance to  benomyl®  was
done at a concentration of  1  mg/L.
Strains carrying  the  nicl  mutation
require nicotinic  acid and were grown
on  MM supplemented with 10  mg/L
nicotin-amide.    Strains  carrying the
pdxl mutation require pyridoxine and
were grown on MM supplemented 10
mg/L pyridoxine  H.CI.   Sexual crosses
were made on carrot  agar, and single
random ascospores recovered using a
micro-manipulator. Mutants and trans-
formants  were  purified  through the
isolation   and  subculturing  of   uni-
nucleate microconidia.,

Mutant Induction

  A benomyl  resistant  mutant of G.
fujikuroi was obtained by UV irradiation
of fungal spores.        ,

DNA Manipulations

  Fungal DNA was extracted using a
"rniniprep"  procedure  that  we  have
modified for G.  fujikuroi.  Large and
small scale plasmid preparations  were
made using the alkaline lysis procedure.
For Southern  analyses,  DNA samples
were  digested  to completion  with
appropriate   restriction  enzymes,
separated   on  agarose  gels,   and
transferred  to  nitrocellulose  mem-
branes. DNA probes were isolated and
purified from agarose gels and  radio-
labeled by nick translation. Blots were
                                    125

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 autoradiographed   with   Kodak   X-
 (OMAT)  film  and   Dupont  Cronex
 intensifying screens at -70°C.

 Transformation Procedures

   Cells  were  transformed to  either
 hygromycin or benomyl resistance via a
 whole  cell  transformation  procedure
 adapted for G. fujikuroi. This procedure
 is relatively inefficient with respect to
 the number of transformants per //g of
 DNA,   however   it   is   rapid  and
 technically simple.

 RESULTS AND DISCUSSION

 Recovery of Benomyl Resistant Mutants
 and  Cloning of G. fuiikuroi /?-Tubulin
  To isolate  the p-tubulin  gene, a
plasmid   library   in   pUC119   was
constructed from the benomyl resistant
mutant.   Using   highly   conserved
sequences for (5-tubulin, primers were
designed  and the  polymerase  chain
reaction (PCR) was used to generate a
probe from total genomic  DNA of the
mutant. Following the screening of the
library a 2.1  kb clone was selected and
mapped.  Constitute  expression of the
gene was  indicated by RNA blots and a
transcript  of  1.9  kb  was observed.
Validation of the functionality of this
gene was  demonstrated  by  trans-
formation  of wildtype  G.  fujikuroi to
benomyl resistance.

Meiotic Stability of Transformed Strains
of G. fuiikuroi

  To date,  stability  studies  have
focused on the hygr gene under meiotic
 conditions.  Crosses with single copy
 transformants segregated hygr: hygs in
 a  1:1  manner  consistent  with  that
 expected  for  a  Mendelian locus  in a
 haploid   organism.     Multiple-copy
 transformants,  however,   segregated
 hygr: hygs in  a  1:2 manner that was
 not   consistent   with   Mendelian
 expectations  for   a   chromosomal
 marker, even though  two unrelated
 auxotrophic   nuclear   genes   were
 segregating  normally.     Segregation
 ratios  in  crosses in which  hygr  was
 introduced via the male parent did not
 differ  significantly  from  crosses  in
 which the transformed strain served as
 the  female parent.   Some  of the
 sensitive progeny from the crosses with
 the  multi-copy  transformants carried
 hygr sequences.  When these pheno-
 typically  sensitive   progeny   were
 crossed with  a  wild-type  strain  that
 carried no hygr sequences, some of the
 progeny  were  phenotypically hygr.
 Some progeny from some crosses were
 more resistant to hygromycin than were
 their sibs  or the transformant strains
 that   served   as   their  parents.
 Transformants  passaged  through  a
 maize  plant  only  rarely  segregated
 progeny with high levels of resistance.
 The   mechanism   underlying  these
 genetic instabilities  is  not clear, but
 may  involve  unequal  crossing-over
 and/or methylation.

 FUTURE WORK

  Foreign  DNA  can be inserted  into
fungal chromosomes in three basic
patterns: a single copy at a single site,
multiple copies at a single  site,  and
multiple copies at multiple sites.  We
are presently constructing  G. fujikuroi
                                    126

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multiple copies at multiple sites.  We
are presently constructing  G.  fujikuroi
strains with  each  of  these different
patterns  with  respect to  the  hygr,
Neurospora benr, and G. fujikuroi benr
genes. We will analyze these different
genomic configurations with respect to
fitness   characters,   stability   and
transmission capability. We will cross
these  transformed, strains with  our
auxotrophically   marked   strains   to
determine if the origin of the sequence
and/or  the   amount   of  sequence
homology between the genomic DNA
and  the  foreign  sequence  affect  the
stability of the  DNA.    We will also
further examine the structure of  the
second  generation   progeny   with
"reactivated" resistance to determine if
the configuration of the foreign DNA
has  been altered  in these  strains  as
compared to th'e original transformant.
  We will begin studies to determine if
strains that belong to the same or dif-
ferent VCGs are capable of  exchanging
foreign  DNA  ihtroduced  via  trans-
formation.     Strains   marked   with
traditional genetic markers (auxotrophs
and spore color mutants) will be paired
with the foreign DNA  included in the
auxotrophic strain.   Selection will  be
based on  the  concurrent recovery of
resistant  prototrophic  colonies   fol-
lowing growth under  conditions  that
favor the  formation of heterokaryons.
Strains that differ at 0,  1, and 10 of the
loci underlying the VCG phenotypes will
be used to provide a range of possible
inhibitions  to  the  asexual  genetic
transfer process.

PUBLICATIONS

Leslie, J.  F., and  Dickman,  M.  B.
1991.      Fate  of   DNA  encoding
hygromycin   resistance  following
meiosis   in transformed  strains  of
Gibberella  fujikuroi.    Applied  and
Environmental Microbiology 57{5): in
press.
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                                 SESSION II
                           EFFECTS RESEARCH
Ecological Processes
  Researchers are evaluating the effects of introduced microorganisms on
structural/functional aspects of freshwater, marine and terrestrial ecosystems.
Improvements in the procedures that are developed in these ecological
investigations may be incorporated into new testing methods.  Further work is
being conducted on smaller components of aquatic and terrestrial systems not
directly related to ecosystem-level effects.  This work includes pesticide degrading
microorganisms and biological control agents such as insect growth regulators and
microbial pest control agents.

Higher Organisms

  A wide variety of microorganisms may be used in biotechnology and information
on their lexicological, behavioral, pathogenic, and histopathological effects on
higher organisms is unavailable. This research investigates the genetic and
molecular basis of infectivity and pathogenicity, and is developing methods that
determine the host range of microbial pest control agents..  Experimental results will
be used to develop protocols for testing effects of microorganisms on beneficial
invertebrate and vertebrate species.

Human Health

  Researchers are investigating the toxicological, pathological and cytogenic
effects of exposing humans to selected recombinant and non-recdmbinant
microorganisms using mammalian cells and surrogate organisms. Different
exposure routes are evaluated in terms of measurable effects.
                                    129

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  USE OF RIBOSOMAL rRNA SEQUENCES TO CHARACTERIZE DIVERSITY AND
                   STABILITY OF MICROBIAL POPULATIONS

                               Richard Devereux
                           Technical Resources, Inc.
                      Environmental Research Laboratory
                                 Sabine Island
                              Gulf Breeze, Florida
INTRODUCTION

    An  explicit  characterization  of
natural  microbial community diversity
has been elusive. In large part, this has
been due to the constraints pure culture
methodologies   have  imposed  upon
environmental  microbiology  -   pure
cultures needed to be  representatively
obtained from  the  natural population
for  subsequent laboratory  identifi-
cation   and   biochemical  character-
ization.  This approach was recognized
to be  limited in  that only 10% (at best)
of  the  bacteria  observed  in   an
environmental   sample   by   direct
counting   could   be   obtained   in
laboratory  pure  cultures.    Further,
biochemical identifications of such pure
cultures may have been imprecise and
have  lead  to  erroneous  misclass-
ifications in the past.  Assessing the
potential risk   imposed upon  natural
microbial populations and the ecological
processes  they  mediate requires a
greater  degree  of  resolution   than
offered  by isolation of pure cultures.
    Recent advances in the application
of  molecular techniques now  permit
long-standing   issues   in   microbial
ecology to be addressed.  In  particular,
small  subunit ribosomal RNA (16S-like
rRNA) sequence comparisons facilitate
the direct measure of natural microbial
population   diversity.      Population
f diversity  of  sulfate-reducing  bacteria
  (SRB) in anaerobic estuarine sediments
  is being explored through  16S rRNA
  sequences.   SRB  have direct roles in
  the cycling of sulfur and the  terminal
  oxidation of organic matter.  Currently,
  SRB  population  diversity  is  being
  measured  with   16S   rRNA-targeted
  hybridization probes. These studies are
  directed towards defining populations
  of  SRB  and   assessing   population
  stability  in   relation   to   ecological
  processes.

  METHODS

  Phvlogenetic Probes

     Oligonucleotide   probes   were
  redesigned such  that  occurrence  of.
  mixed,  or multiple  nucleotides at  a
  common  nucleotide   position,  were
  eliminated. This was found  to provide
  more precise measurements  of relative
  rRNA abundance for hybridizations  to
  membrane bound nucleic acids.  Probes
  for specific SRB groups were labeled at
  their 5' ends using gamma 32P-ATP and
  polynucleotide  kinase.   Additionally,
  fluorescent  dye-conjugated  bacterial
  domain- and  delta subdivision-specific
  Oligonucleotide probes  were prepared
  for  direct   fluorescent  microscopic
  identification of bacterial cells obtained
  from marine sediments.
                                     131

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Analysis of Natural SRB Populations

    Nucleic   acids   were   extracted
directly  from fractions  of sediment
cores by mechanical disruption (bead-
beater lysis) in the presence of sodium
acetate/EDTA buffer pH 5.2,  sodium
dodecyl  sulfate,   polyvinylpoly-
pyrollidone, and beta mercaptoethanol.
Lysates were extracted  with  phenoU
phenol:chloroform,   and  chloroform.
Nucleic acids were  precipitated with
ethanol.  These  were then applied to
nylon membranes and hybridized with
the SRB rRNA specific probes. Rates of
sulfate reduction in the  sediment core
fractions were determined by isotopic
assay.  Measurements of sulfate and
sulfide in the sediment porewater was
by standard methods.

Preparation  of a  Sediment  Bacterial
Fraction

    Sediment bacterial  fractions were
obtained for  whole cell hybridizations
with   fluorescent  dye-conjugated
oligonucleotide   probes.      Sandy
sediment samples (3.0 g wet weight)
were placed into 50  ml  flasks with  3
mm glass beads and 0.15 M NaCI. The
contents of the flasks were vigorously
shaken  for 30 min.  then allowed to
settle for 15  min.  A 3 ml aliquot was
withdrawn from  the upper portion of
the settled suspensions.  DAPI stained
cells and cells  hybridizing  with  the
rRNA   probes   were   determined.
Efficiencies  of cell  recoveries were
obtained by adding known amounts of
sulfate-reducing  bacteria to washed,
sterile  sand  and to  natural sediment
samples.  For enumeration of sulfate-
reducing bacteria,  preparation of cell
fractions  were  prepared  under an-
aerobic  conditions.    The  bacterial
fractions were passed through 8.0 //M
filters   to   remove   large  particles.
Filtrates  were treated with combina-
tions  of  SRB  basal  salts  medium,
nutrients, and nalidixic acid to stimulate
production of ribosomes  and  enhance
the detection of SRB.

RESULTS AND DISCUSSION

SRB Population and Function Profiles
in Sediment Cores

    Concentrations of sulfate  were 15
mM in  the surface  water   and  in
sediment porewater to a depth of 4 cm.
Porewater  concentrations  of  sulfate
decreased from 10 mM at the 4-5 cm.
depth interval to less than 5 mM in the
14-16  cm, depth  interval.  Porewater
concentrations of sulfide  were 0.1 to
0.2 mM in the 4-10 cm. depth  intervals
and increased markedly to 0.8 mM  12
to 16  cm. below  the water-sediment
interface.    The  peak in  activity  of
sulfate  reduction  (3.5  mmol/ml/hr)
occurred in the 2-3 cm. interval.
    Desulfovibrio  16S  rRNA was the
most abundant SRB rRNA and peaked
at the 3-4 cm. interval. Stratification of
the SRB  population  with sediment
depth was apparent.  Amounts of 16S
rRNA  of fatty  acid-  and/or  acetate-
utilizing SRB (encompassing four SRB
phylogenetic groups)  decreased in the
sediment fractions haying the  highest
amounts of Desulfovibrio 16S  rRNA.
    The  peak, in   activity  of  sulfate
reduction did not occur within the same
fraction as  the predominant  peak in
Desulfovibrio rRNA.     Acetate   has
previously   been   shown  by  other
                                    132

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investigators  to   be  the   primary
substrate   for  sulfate  reduction  in
estuarine sediments.  Thus, the  peak
sulfate reduction .rate  was expected to
coincide  with  a peak  in the  rRNA
abundance  of  acetate-utilizing  SRB.
This relationship was not seen in the
results and  may be accounted for  by
several possibilities. Additional groups
of SRB may exist in nature that are not
represented in  the culture  collection
that not are being accounted for by the
probes. Also, since the data represent
one sampling time, it is possible that a
shift in the activity of  sulfate reduction
had occurred and that the population
had yet to respond by an increase in
rRNA abundance.  Finally, accounting
for energy released by the reduction of
sulfate  by  different  electron  donors
should be considered in evaluation of
the   community   structure-function
relationship. Hydrogen, for example, as
an electron donor for sulfate reduction
yields considerably more  energy  than
acetate.

Whole Cell Hybridizations
of Natural Populations

    Laboratory grown SRB cells were
recovered   from   washed,   sterile
sediment  samples  with  efficiencies
approaching 90%.   For fixed sediment
samples,  cells   hybridizing  with  the
bacterial domain probe  were  16% of
the cells observed  by DAPI staining.
Cell counts hybridized with the delta
subdivision-specific probe were 42% of
the counts hybridized with the bacterial
domain probe.   Delta subdivision  cell
counts increased to 59%  and  49% of
the  bacterial  domain counts when
samples  were  incubated  in  medium
with or with nalidixicacid, respectively.
When  samples  were  incubated  in
medium  containing  molybdate,  and
inhibitor  of  sulfate  reduction,  delta
subdivision cell counts were only 11 %
of the bacterial domain counts.  The
results demonstrate that  fluorescent
dye labeled rRNA probes could be used
with natural samples and that specific
bacterial populations can be stimulated
to enhance their detection.

FUTURE WORK

    Research planned will address the
community structure-function relation-
ship of SRB populations.  PCR amplifi-
cation  and sequencing of  16S rRNA
genes from natural populations will  be
used to determine natural diversity not
represented in the culture  collections.
Natural populations will be character-
ized  during   temporal  sampling   to
determine  base  line   variations  in
populations with  respect  to sulfate
reduction  rates and turnover rates  of
substrates.     Laboratory  sediment
systems   will  be  used   to  control
parameters,  such as  availability  of
specific electron donors,  to alter  SRB
populations.     Hybridization    of
fluorescent labeled probes sediment
bacteria as a means  to  increase the
number of  samples  which can   be
analyzed will  be explored.

PUBLICATIONS

Amann, R.I.,. B.J. Binder, R.J,  Olsen,
S.W. Chisholm, R. Devereux, and D.A.
Stahl.  1990.  Combining  16S  rRNA-
targeted  oligonucleotide  probes with
flow cytometry for  analyzing  mixed
microbial populations.  Appl. Environ.
                                     133

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 Microbiol. 56:2012-2020.

 Devereux, R., S.-H. He, C.L. Doyle, S.
 Orkland, D.A. Stahl,  J.  LeGall,  and
 W.B. Whitman.   1990.  Diversity and
 origin   of   Desulfovibrio   species:
 phylogenetic definition of a  family.  J.
 Bacteriol. 172:3609-3619.

 Stahl, D.A., R. Devereux, R.I. Amann,
 B. Flesher,  C. Lin, and J.  Stromley.
 1990. Ribosomal  RNA based studies of
 natural microbial diversity and ecology.
 pp. 669-673. In: T. Hattori et al. (eds.),
 Recent Advances in Microbial Ecology.
 Proceedings  of  the  5th International
 Symposium   on  Microbial   Ecology.
 Japan Scientific Societies Press.

 Devereux, R. and D.A. Stahl.  1991.
 Phylogeny of sulfate-reducing bacteria
and  a perspective for  analyzing  their
natural communities,   in: J.M. Odom
and  R.  Singleton, Jr.  (eds), Sulfate-
Reducinq Bacteria:  A   Contemporary
Perspective.  Brock-Springer series  in
Contemporary Bioscience. (in editorial
review).
                                    134

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      HABITAT SPECIFIC DIFFERENCES IN PERSISTENCE AND EFFECTS OF
  INTRODUCED CELLULOLYTIC BACTERIA USED AS SURROGATES FOR GEMS

                     Thomas L. Bott and Louis A. Kaplan
                        Stroud Water Research Center
                        Academy of Natural Sciences
                           Avondale, Pennsylvania
INTRODUCTION

   These  studies have three primary
objectives:    a)  to  determine  the
persistence of introduced bacteria in
natural benthic aquatic communities, b)
to  test  several  community   and
ecosystem level  response variables in
order to pinpoint those most useful for
detecting  effects of  the  introduced
organism,  and c) to evaluate the utility
of mesocosms for assessing  the  fate
and effects  of genetically engineered
microorganisms   (GEMs)   in   natural
systems.    We   have worked  with
surrogates for a GEM with enhanced
cellulosedegradingcapability. Cellulose
"superdegrader"  GEMs   and   their
products are  of industrial interest and
may find application in the conversion
of biomass  to fuel.  The use  of  a
surrogate   expedites   field  studies,
although we recognize that a surrogate
will   not   mimic   a   GEM   in   all
physiological  and ecological respects.
Our   research  focuses   on   stream
ecosystems  since they  often receive
waste discharges and non-point source
inputs.  White Clay Creek, the study
stream,  drains   a   protected  rural
watershed in southeastern  (Chester
Co.) Pennsylvania.
   We  screened  over 20  isolates of
aerobic cellulolytic bacteria from culture
collections by  assaying  their   lytic
activity  on pure  cellulose, ability to
degrade algal detritus and leaf litter,
and their growth rates and ranked them
according to their performance in these
tests.  Fluorescent antisera (FA) were
produced  against  the  most   active
isolates, Cellulomonas flavigena (NRC
2403), C. fimf (NRRL  B402),  C.  sp.
(NRC 2406), and C. uda (NRRL B404)
to enable their detection in  benthic
communities. Preliminary studies of the
population dynamics of C. sp. (NRC
2406), C. uda (NRRL B404), and C. sp.
CS1-1 (an organism that cross  reacted
with C. uda antiserum) in streambed
sediments were conducted in  2.95 L
microcosms.
    C. sp.  (NRC 2406) was selected for
initial studies in  larger (35 L) mesocosm
streams.    The    first   experiment,
conducted during  the summer  months
indicated that the  isolate could persist
for several weeks  in sediments  and
benthic  algal   growths,   although
densities declined  fairly rapidly from
high post-inoculation levels. Effects on
benthiccommunity metabolism (primary
productivity  and   community  respir-
ation), associated changes in DOC,  and
bacterial  productivity   were   not
pronounced,    High densities  of   FA
stained  cells   were found  in algal
growths in both an inoculated and a
control  mesocosm 3  months after
introduction.  We have also found cells
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 that stained with C.  uda (NRRL B404)
 and C. sp.  (NRC 2406) antisera in leaf
 packs and streambank soil from White
 Clay Creek.   While there  may  be a
 natural source  of  FA stained  cells  in
 White  Clay Creek,  our  observations
 suggested habitat specific differences in
 persistence  deserved  study.     The
 experiment described below focused on
 population dynamics of the introduced
 organism  in  three  different  micro-
 habitats; algae, sediments, and leaves,
 and  on  alteration  of  the  rate  of
 degradation of leaf litter.

 METHODS

    Six stream  mesocosms  (2.23 m
 long x 0.203 m wide x 0.127 m deep),
 designed to simulate a slow run habitat
 in White Clay Creek, were constructed
 of vinyl coated steel and Plexiglas end
 plates.  They were filled with 2 cm. of
 gravel and coarse sand from White Clay
 Creek,  over which 40  plastic trays
 (0.10  m  square  x  0.038  m deep)
 containing   surface  sediments  from
 White Clay Creek  were  placed.  The
 bottoms of the trays were removed and
 replaced with  400 jim  mesh nylon
 screening to allow exchange of water,
 nutrients,    dissolved   gases  and
 organisms   within   the   sediments.
 Stream water (35 L) was recirculated in
 each  system;  900  ml/min  of  new
 stream water was added continuously
 to   replace  the   entire   volume
 approximately every 40  min.   Water
 depth approximated 1.5 cm.   Water
 returned to White Clay Creek was first
 filtered through cartridge filters  (which
 were autoclaved before discarding) and
 irradiated  with   ultraviolet   light,  a
treatment found to effectively kill the
 study bacteria.  The streams, housed in
 a greenhouse, were immersed in water
 jackets continuously replenished with
 White  Clay  Creek stream  water to
 maintain   ambient   stream   water
 temperatures.   The   systems  were
 separated  by  clear  plastic  sheeting
 suspended from a wooden frame to
 hinder  spread of organisms  between
 microcosms.
     Population and community struct-
 ural   and  functional  parameters
 (including densities of FA stained cells,
 total bacteria,  primary productivity,
 community respiration, ATP, chlorophyll
 a, and cellulasesactivity) were measured
 by sampling sediments and filamentous
 algae (dominated  by Cladophora sp.)
 prior to  addition of the isolate.  Just
 before inoculation of the isolate, packs
 (approximately 5 g each) of tulip poplar
 (Liriodendron tulipifera) leaves that had
 been previously  leached and redried to
 obtain weights,  were  placed  in  the
 mesocosms. The surrogate was placed
 in  the   water  in  two mesocosms,
 maintaining two  others as uninoculated
 controls.   Cells for inoculation were
 grown at 20°C  on  Solka-Floc in basal
 salts medium for 5 days with agitation.
 After allowing the  cellulose to settle,
 cells were harvested, resuspended  in
 autoclaved stream water, and added to
 duplicate  mesocosms.      After
 recirculating    the   inoculum  for
 approximately 72  h with no stream
 water  addition,   the   systems  were
 returned to flow-through mode and 4 h
 later, an intensive (3-5 day) study  of
 densities  of the surrogate  and total
 bacteria  in surface sediments,  leaf
 packs, and algae was initiated.  Then,
at approximately 10-14 day intervals
the response  variables  noted  above
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were measured, and samples for  leaf
pack chemical composition and weight
loss   were   collected.     Light,
temperature,   and   selected   water
chemical   characteristics   were
monitored.   Samples of sediment  and
Cladophora sp. were obtained by filling
plastic  rings (1.15 cm. i.d. x 0.2  cm.
deep) with  sample.  Leaf packs were
sampled  by coring replicate samples
(1.10 cm. i.d. x approximately 2 cm.)
with a  cork borer.
   Primary productivity and community
respiration  of  sediments    and
Cladophora sp.   were estimated from
dissolved oxygen changes in flowing
water  microcosms  from  which  eco-
system P/R  status was  determined.
Leaf  pack   respiration  rates  were
determined in the same systems.  For
bacterial  enumeration, formalin fixed
samples were sonicated for 45 sec. at
30 W  with 0.1   M dibasic ammonium
phosphate as a  surfactant, centrifuged
once (412  x g  for 5 min at 4°C) in
30% glycerol, and twice more in 60%
glycerol,  Aliquots of supernatant fluids
were filtered onto 0.2  |im pore  size
Nuclepore filters and stained with FA or
DAP! ,for epifluorescence  microscopic
counts  of  the  target  cell or  total
bacteria on from 6-10  or 3 replicate
samples, respectively.   Algal biomass
was  assessed   from  chlorophyll  a
determination, and total viable biomass
from   ATP,  each   on  5  aliquots.
Cellulase activity was determined using
the Cellulose Azure (CA)  assay; data
were corrected for color contributed by
samples  using   a  two  wavelength
correction procedure for leaf packs or a
regression  of  color  against  sample
weight  for  sediments.   Leaf  pack
decomposition  was  determined  from
weight  loss  and  cellulose content.
Cellulose was obtained by combustion
at 450°C for 4 h following sequential
extraction  of  non-cell-wall  material,
hemicellulose,   and   lignin   using
detergent solutions and permangenate
oxidation.

RESULTS AND DISCUSSION

    Epifluorescence      microscopic
counts indicated that the C.  sp. (NRC
2406) inoculum contained 1.97  ± 0.15
x 109 cells/ml, and viable counts from
spread plates indicated 4.77  ±  0.68 x
109 colony  forming units/ml.  Using the
average  of these estimates, we  cal-
culated  that the initial  density  in  the
inoculated  mesocosms  was approxi-
mately 4.57 x 107 cells/ml of water.
    Six  water changes after exposure,
the  densities  (ce!ls/cm3)  associated
with sediments,  Cladophora sp.,  and
leaf  packs  in the duplicate inoculated
mesocosms averaged 1.06 ±  0.16 x
106, 4.32 ±  2.32 x 106, and 2.07 ±
0.97 x  106,  respectively.   Based  on
sample  weights, the densities  (cells/g
dry weight) were 8.06  ± 4.16 x 105,
3.52 ± 2.83 x 107, and 1.30 ± 6.34
x  107,  for  those  sample   types,
respectively.   Given  the densities of
total bacteria in these samples, which
ranged between  1 x 108 and 1  x 1010
in all three sample types, the  isolate
composed   maximally   0.17%   and
0.44% of the total bacterial community
in  Cladophora  in  each  inoculated
stream, 0.35 % and 0.49 % of in  leaf
packs, and  only 0.04% and 0.04% of
the sediment bacterial communities in
each  of  the   duplicate  inoculated
mesocosms.  FA stained cell densities
(cells/g  dry weight)  in  the duplicate
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 control mesocosms averaged only 3.8
 ± 17.1  x 104 and 4.58 ± 4.20 x 106
 in  sediments  and   Cladophora  sp.
 respectively  and  the organism was
 never detected in leaf packs.
     Densities decreased  rapidly  from
 post inoculation  maxima in sediments,
 and  slowly  in  leaf  packs,  with an
 intermediate   rate   of   decline   in
 Cladophora.   Regression analyses of
 cell densities on both a dry weight and
 a volumetric basis indicated half lives
 (T1/2) of  13.5 (r2 = 0.829,  0.778),
 1.5 (r2 = 0.599, 0.602), and 8.8 (r2
 = 0.875, 0.989) days for the isolate in
 those habitats,  respectively.    While
 densities   were   near  background
 (control)   levels   in   sediments  and
 Cladophora  after  10  and  30  days
 respectively,  FA  stained  cells  were
 found in a leaf pack at 70 days.
    Effects  on community  respiration
 and primary productivity of sediment
 were   not   statistically   significant
 (Before-After,   Control-Impact  test,
 BACI, a  =  0.05).   These  measures
 were made on Cladophora samples less
 frequently,  and although  rates  were
 much  higher the  isolate  had  no
 pronounced effect on either respiration
 or primary productivity. Similarly, there
 were no strong effects on chlorophyll a
 concentrations and assimilation ratios
 and  hourly P/R ratios were in a similar
 range  in both inoculated and  control
 mesocosms throughout the study. Leaf
 pack respiration per gram dry  weight
 sample was higher on packs exposed to
 the isolate  (0.450 ± 0.205  vs. 0.367
 ± 0.181, n = 6), but the difference was
only statistically significant at the a =
0.10 level (paired sample t-test). There
was overlap in ATP  values  between
streams  and  no  striking  difference
 between data collected before and after
 exposure.
     Cellulase  activity  tended  to  be
 slightly higher overall in  the inoculated
 streams   for   each   sample   type.
 However,  the difference was statist-
 ically  significant only  for  sediment
 samples (paired t test, a = 0.05) and
 since it occurred both before and after
 inoculation  the  effect  cannot  be
 attributed to introduction of the isolate.
 Correlation  analyses   including  all
 sample  types  indicated that isolate
 densities  whether  expressed  on  a
 volumetric or dry weight  basis,  were
 weakly but significantly correlated with
 cellulase activity (r= 0.40  and 0.32, a
 =  0.01  and 0.05 respectively).  The
 relationship was highly  significant for
 sediment samples (r  = 447; a = 0.0.1,
 n  = 32) although not for leaf packs,
 perhaps  because  other  cellulolytic
 bacteria and fungi were more numerous
 there.
    Regression analyses  indicated that
 leaf packs  lost weight at a similar rate
 in both the experimental (T1/2  = 99
 days) and  control (T1/2  = 115 days)
 systems,  and  predicted,slightly less
 cellulose (68.9%) in exposed leaf packs
 than in unexposed (75.6%).  Sample
 sizes were small however, and these
 indications require confirmation. While
 other cellulolytic  bacteria  and  fungi
 were present in the packs, correlation
 analyses showed that both weight loss
 and  cellulose content of  leaf packs
 were weakly correlated (r= 0.45 and
0.50, a = 0.08 and 0.10)  with FA + cell
densities and those densities expressed
as a percentage of the total bacterial
flora (r = 0.64, a = 0.05, n =  11).
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FUTURE WORK

   A  more  detailed  study  of  FA
population dynamics in leaf packs and
effects on leaf pack decomposition has
been performed, including a comparison
of those parameters in mesocosms and
the field.  Samples from that experi-
ment are still being processed.  More
work with Cladophora or  other  algae
with a  cellulose cell wall is needed.
   Obviously,   for   an   introduced
organism  to  affect  community  or
ecosystem properties the impact must
be either immediate and acute or the
organism must be able  to persist and
exert an effect over time. Work to date
indicates the  need to  understand in
greater detail the factors  controlling
bacterial population dynamics in natural
systems,  including the physiological
and ecologically relevant characteristics
of the  organism as they interact with
ecosystem properties such as  nutrient
supply  and   grazing   rates.     The
application of rRNA probes in  those
studies is desirable, to increase the
specificity of identification of the isolate
in  natural  samples.    Finally,  we
anticipate the  extension   of  these
studies to work with a GEM.

PUBLICATIONS

Bott, T.L. and L.A. Kaplan. In revision.
Selection of surrogates for a genetically
engineered    microorganism  with
cellulolytic capability  for   ecological
studies in streams. Canadian Journal of
Microbiology.

Bott,  T.L., and  L.A.  Kaplan.   1991,
Persistence  of introduced  cellulolytic
bacteria and cellulose decomposition in
leaves, algae, and sediments measured
in mesocosm streams. Abstracts of the
91st Annual Meeting of the American
Society for  Microbiology, Dallas  TX,
May 5  - 9, 1991.

Bott, T.L.,  and  L.A. Kaplan.   1990.
Cellulolytic bacteria as surrogates for a
GEM:     Microcosm   studies   of
persistence and  effects in  streambed
sediments.    Abstracts  of the  90th
Annual  Meeting of  the  American
Society for Microbiology, Los Angeles,
CA, May 13 - 17, 1990.

Bott, T.L.,  and  L.A. Kaplan.   1990.
Cellulolytic  bacteria  as surrogates for
genetically engineered microorganisms:
Microcosm  studies of persistence and
effects in streambed sediments,  pp.
139 - 143.  jn; Review of Progress in
the   Biotechnology-Microbial   Pest
Control  Agent  Risk   Assessment
Program.  EPA/600/9-90/028.
EPA, Washington.
U.S.
Bott,  T.L.  and L.A. Kaplan.   1990.
Beyond chemical  toxicity:    Stream
microcosms as systems for the study of
persistence and effects of  genetically
engineered  microorganisms  (GEMs).
Technical    Information   Workshop-
Experimental Ecosystems: Applications
to Ecotoxicology.  Publications of the
North American Benthological Society.

Bott,  T.L., and L.A. Kaplan.   1989.
Selection of a surrogate for a GEM with
enhanced cellulose degrading capability
for studies in streams. Abstracts of the
89th Annual Meeting of the American
Society for Microbiology, New Orleans,
LA, May 14- 18,  1989.
                                     139

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      FIELD CALIBRATION OF SOIL-CORE MICROCOSMS FOR EVALUATING
     FATE AND EFFECTS OF GENETICALLY ENGINEERED MICROORGANISMS
                        IN TERRESTRIAL ECOSYSTEMS

                  Harvey Bolton, Jr., and James K. Fredrickson
                      Battelle, Pacific Northwest Laboratory
                             Richland, Washington
 INTRODUCTION

    Microcosms are an attractive option
 for obtaining  preliminary  data  on the
 fate  and  ecological  effects  issues
 associated   with  the   release   of
 genetically engineered microorganisms
 (GEMs) into the environment, because
 tests  and  microorganisms  can   be
 contained   within   the   laboratory.
 Historically,  microcosms  have been
 used to assess the fate, transport, and
 toxicity of chemicals and pollutants.
 Recently/ intact soil-core  microcosms
 incubated in the laboratory were used
 to  evaluate the  fate  and ecological
 effects  of  transposon   mutants  of
 Azospirillum  lipoferum  and a  wheat
 (Triticum  aestivum) -root colonizing
 Pseudomonas sp.   However,  before
 microcosms can be  used as a standard
 tool for biotechnology risk  assessment,
 they must be compared with the field to
 ensure that they are effective  field
 models.
    The  principal  objective  of  this
 project  was  to  calibrate  soil-core
 microcosms with their field  counter part
for  microbial   fate  and  ecosystem
structural and functional properties and
the  resultant   effect -of  introduced
microorganisms on these properties.  In
the first year of this project (October
 1988 to September  1989), the fate of
rhizobacterium Pseudomonas sp. RC1
in soil and on the wheat rhizoplane was
 determined as a function  of time in
 intact soil-core microcosms at ambient
 temperature  (22°C) and in a growth
 chamber with temperature fluctuations
 that simulated average conditions i.n the
 field, field lysimeters,  and field plots
 (Bolton et al.,  199la).  The  effect of
 the  introduced RC1  on  ecosystem
 structural and functional properties as
 well as a comparison between the four
 systems was also conducted the first
 year (Bolton et al., 1991 b').
    In the second year of this project
 (October  1989 to  September 1990)
 two experiments were conducted with
 soil-cores as microcosms located in a
 growth chamber and as  lysimeters in
 the  field.   First,  RC1  was  used  to
 determine ho.w year-to-year field vari-
 ability influenced  the  calibration  of
 microcosms with the field. Second, the
 Gram-positivebacterium Streptomyces
 lividans TK24 was used "to allow'us  to
 compare microcosms as field  'surro-
 gates for both Gram-negative (RC1) and
 Gram-positive (TK24) bacterial survival
 and ecological effects.  The complete
 results of this project are contained in
 the final report (Bolton et al. 1991c).

 MATERIALS AND METHODS

    The bacterial strains  used in this
 project   included   a   spontaneous
 rifampicin  resistant mutant.(100 mg/L)
of Pseudomonas sp. strain RC1 (RC1)
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grown  on  Sands  rif50  medium and
Streptomyces  lividans TK24  (TK24),
which  was  selectively   grown  on
starch-casein mineral salts medium with
carbenicillin  (25 nng/L),  streptomycin
(50 mg/L), nystatin (50 mg/L), Benomyl
(50  mg/L),  and  cycloheximide  (500
mg/L).
    Intact  soil  cores   for   use  as
microcosms  and field lysimeters (17.5
cm.  diameter,  60 cm.  length)  were
obtained   from   the  field   site  in
southeastern Washington state using a
steel coring  device.  The  surface soil
(top 15 cm.)  was removed  from  all
cores and combined, mixed, and sieved
to pass a 2-mm screen. The RC1 study
had  two  soil  treatments  including
inoculation  with  2.3 x   107  colony
forming units (CPU) of RC1/g dry soil
and uninoculated.  The surface soil for
the separate TK24 study was amended
with   1.0%  cellulose    and   either
inoculated with 2.5 x 106 CPU of TK24
spores/g dry soil or left uninoculated.
All treatments were brought  to a final
soil moisture content of  16% with tap
water.
    The microcosms were  incubated  in
a  growth  chamber  under a  cycling
temperature regime that approximated
the average daily minimum temperature
during the dark cycle and  the average
daily high  temperature  during  the
photoperiod at the Hanford Site.  The
lower temperature limit  in the growth
chamber was 5°C. The field lysimeters
consisted  of intact soil cores within
polyethylene pipe placed back into the
soil at the field  site.  The bottoms of
the  microcosms  and field lysimeters
were   covered   with  polyester
monofilament cloth (160 mesh)  to
prevent root  (for RC1) and  soil (for
TK24) loss from the core.
    The mineralization of 14C-labelled
cellulose in microcosms and lysimeters
was determined in the TK24 study in
minitubes  (4.3 cm. diameter, 24 cm.
length  PVC  pipe)  with the  14CO2
evolved determined as a function  of
time.
    All treatments were replicated six
times. The RC1 study was seeded with
five  "Daws"  winter wheat  seeds  in
each soil  core on October 25, 1989
that were later thinned to two wheat
seedlings. The TK24 study was started
on  November 2,  1989.  Microcosms
and  field  lysimeters  were  routinely
watered with tap water to a final soil
moisture content of 16%.
    The survival of RC1 and TK24 in
surface soil  was  determined  as  a
function of time by plating onto their
selective media.
     For the RC1 study, the populations
of  RC1,  Pseudomonads,  and  total
aerobic heterotrophs  on  the  wheat
rhizoplane were  enumerated  at  the
three-leaf  stage of growth on seedlings
and at  the boot  stage of growth  on
roots from three sections (top: 0 to  15
cm., middle: 15 to 35 cm., and bottom:
35  to 55 cm.). The biomass of wheat
shoots and soil dehydrogenase activity
in surface (0 to 15 cm. depth) soil were
determined at the three-leaf and boot
stage of wheat growth.
     For the TK24 study,  surface soil
samplings occurred in late November,
early March,  early  June,  and  early
September.    The  surface  soil  was
sampled  at   these  four  times   for
populations  of total  aerobic  hetero-
trophs  and  Actinomycetes,  the  soil
enzyme activities of dehydrogenase and
b-glucosidase,    and   14C-labelled
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  microbial biomass.

  RESULTS AND DISCUSSION

     The decline in the population of RC1
  in  surface  soil  was  similar  in  the
  microcosms and the field.  However,
  the population of  RC1  in surface soil
  was   significantly   higher   in   the
  microcosms  than  the  field  at  the
  three-leaf stage of wheat growth (5.9
  log CFU/g > 4.5 log CFU/g), while the
  population decreased to the same value
  at the  boot stage  (4.2 and  3.6 log
  CFU/g for microcosms and  the field).
  The rate of decline was similar to the
  rate  found  in  the  first  year study
  (Bolton  et al. 1991 a), suggesting that
 small differences in inoculum level (7.4
 and 7.7 log CFU RC1/g in this  and the
 previous  study)   and   year-to-year
 climatic variability in the field  did  not
 greatly influence the rate of decline of
 RC1 in soil. There was no difference in
 the colonization of wheat roots  by  RC1
 at the three-leaf stage in microcosms or
 the field, similar to first  year  results.
 The  population  of   RC1  on  the
 rhizoplane at the boot stage of wheat
 was different from that of the first year
 with  significantly higher  populations
 associated with the microcosms than
 the field.  Also, the distribution  of RC1
 on the rhizoplane as a function of depth
 in the  microcosms was different from
 the field with the population of RC1
 generally higher in the microcosms than
 the field.
    Pseudomonad and  total  aerobic
 heterotrophrhizoplanepopulationswere
 very similar  in  the  microcosms and
field, and similar to the previous year's
results.   Shoot  biomass and dehydro-
genase   activity  of  soils  were  no
  different in the microcosm and the field
  at the three-leaf stage of wheat growth,
  but  were lower  and higher,  respect-
  ively, in microcosms versus the field at
  the  boot  stage.   These results were
  different from those of the previous
  year  where the  growth  chamber and
  the field had  a  similar  final  biomass.
  The  previous  year the soil  dehydro-
  genase activity was significantly higher
  in the microcosms than the field at the
  three-leaf stage of wheat growth.  Soil
  dehydrogenase activity  at  the boot
  stage sampling was not determined the
  first year. Year-to-year variability was
  found in comparing this study with
  previous  years (Bolton et al., 1991 a,
  1991b)  including  differences in  the
 colonization of the rhizoplane by RC1 at
 the  boot  stage  and  wheat  biomass
 production at the boot stag. However,
 for the majority of parameters, results
 from microcosms and the  field were
 similar in both years.
    Soil populations of TK24 declined
 less than 2 log units over the ten-month
 study.  This slow decline in the  soil
 population of TK24 compared to RC1 is
 not  surprizing because  TK24 is a
 gram-positive spore-forming bacterium
 and was added to soil as spores.  There
 were  no  differences  between  the
 microcosms and the field for the soil
 population of TK24 until after 45 weeks
 when  the population was 1 log unit
 higher in the microcosms.  At the initial
 sampling in the fall, soil dehydrogenase
 activity was greater in the microcosms
 than  the  field  and inoculation with
 TK24  depressed  activity.  Dehydro-
 genase activities  were   higher  in
 microcosms than in the field during the
 winter and summer, but there were no
differences at the spring sampling. Soil
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B-glucosidase  activity  was  higher  in
microcosms than the field  during the
winter and summer, but were the same
during the fall and  spring.  Aerobic
heterotroph and Actinomycete popula-
tions were higher in microcosms than in
field soils during  winter and summer,
while the field  had larger populations in
spring.    The  rate  of    C-cellulose
mineralization   was   similar  in  the
microcosms and  the field  until early
May when  there was a flush of 14C02
in  the  field.   The  14C-labelled soil
microbial  biomass   was significantly
larger in the microcosm than the field,
while inoculation with  TK24  had no
effect-.
    In   summary,    the  microcosms
simulated  the field  quite  well  for
microbial fate  and effects.  Exceptions
include  a flush of 14C02 from the field
soils  in early May  and  the  higher
populations of TK24 in microcosm soils
at the summer sampling, both pf which
could be  attributed  to differences  in
temperature and water regimes.  The
differences in microbial populations and
enzyme  activities  at the  winter and
summer sampling points were attribut-
ed  to  the inability  of the  growth
chamber   to   mimic   temperature
extremes  in   the  field during  these
periods.
    The results from studies conducted
the second year of this project suggest
that  microcosms  can   accurately
simulate the  field  with  respect to the
fate  and   effects   on   ecosystem
structural and functional properties  of
both  gram-negative and gram-positive
introduced micro-organisms. However,
better  controls   of   environmental
variables  including  temperature and
moisture will  be  necessary to more
closely simulate the field for future use
of microcosms for risk assessment.

FUTURE WORK

    This project was completed and a
final report submitted to EPA-Corvallis
in March 1991.

PUBLICATIONS

Bentjen, S.  A.,  H.  Bolton, Jr., J. K.
Fredrickson,  and   D.  J.  Workman.
1990.    Field   calibration  of  intact
soil-core  microcosms inoculated  with
Streptomyces lividans TK24.  American
Society for Microbiology Abstracts, pp.
315.

Bolton, Jr., H., J. K. Fredrickson, S. A.
Bentjen, D. J. Workman, S. W. Li, and
J.  M.  Thomas.    1991 a.     Field
calibration  of soil-core microcosms  :
Fate   of   a   genetically  altered
rhizobacterium.  Microb. Ecol. in press.

Bolton, Jr., H., J. K. Fredrickson, J. M.
Thomas, S.  W. Li, D. J. Workman, S.
A.  Bentjen,  and J.  L. Smith.  1991b.
Field   calibration   of   soil-core
microcosms: Ecosystem structural and
functional comparisons. Microb.  Ecol.
(in press).

Bolton, Jr., H., J. K. Fredrickson,  D. J.
Workman, S. A. Bentjen, and S. W. Li.
1990.  Field calibration of microcosms
to assess the fate and ecological effects
of a genetically altered  rhizobacterium.
American   Society   for  Microbiology
Abstracts pp. 314.
                                     143

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Bolton, Jr., H., J. K. Fredrickson, S. A.
Bentjen,  D. J. Workman, S. W. Li,  and
J.  M.  Thomas.     1991c.    Field
calibration of soil-core microcosms for
evaluating   fate   and   effects   of
genetically engineered microorganisms
in terrestrial ecosystems. Final Report.
PNL-7667, UC-403.  National Technical
Information Service, Springfield, VI.

Fredrickson, J. K., S. A. Bentjen,  H.
Bolton, Jr., S. W. Li, and P. Van Voris.
1989.  Fate of Tn5  mutants of  root
growth-inhibiting Pseudomonas sp. in
intact soil-core microcosms.   Can. J.
Microbiol. 35:867-873.

Fredrickson, J.  K.,  H. Bolton, Jr., S. A.
Bentjen, K. M. McFadden, S. W. Li, and
P. Van Voris.  1990.   Evaluation  of
intact  soil-core   microcosms   for
determining   potential   impacts   on
nutrient   cycling   by   genetically
engineered microorganisms.   Environ.
Toxicol. Chem.  9:551-558.

Fredrickson, J. K.,  H. Bolton, Jr., S. A.
Bentjen, D. J. Workman,  and S. W. Li.
1990. Fate and ecosystem effects of a
root-colonizing  Pseudomonas sp.  and
Streptomyces/ividansTK.24 in soil-core
microcosms and field trials.  European
Environmental Research Organization -
Molecular Microbial Ecology Workshop,
May 1, 1990.
 Fredrickson, J. K., H. Bolton, Jr., D. J.
 Workman, and S. A.  Bentjen.  1989.
 Field  calibration  of microcosms  for
 evaluating the  fate  and  effects  of
 genetically  engineered  rhizobacteria.
 American  Society   of  Agronomy,
 Agronomy Abstracts pp. 215.

 Fredrickson, J. K., P. Van Voris, S. A.
 Bentjen,  and  H.  Bolton,  Jr.   1990.
 Terrestrial  microcosms  for evaluating
 the  environmental  fate   and  risks
 associated  with   the   release  of
 chemicals  or  genetically engineered
 microorganisms  to the environment.
 Haz. Assess. Chem. 7:157-202.

 Fredrickson, J. K. H. Bolton, Jr., and G.
 Stotsky.     1991.     Methods  for
 evaluating  the effects  of GEMs  on
 nutrient cycling  processes.  Methods
for Microbial Ecology, McGraw-Hill, NY,
 in press.

Fredrickson, J. K. and  C. Hagedorn.
 1991.   Methods  for  evaluating the
effects  of  GEMs  and  MCPAs on
ecological processes.    Methods for
Microbial Ecology, McGraw-Hill, NY, in
press.
                                    144

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            THE EFFECT OF A GENETICALLY ALTERED BACTERIUM
     ON NITROGEN TRANSFORMATION RATES, INDIGENOUS M1CROBIOTA
                 AND MICROBIAL BIOMASS IN A XERIC SOIL

        L. K. Gander1, E. R. Ingham2, J. D. Doyle1, and C. W. Hendricks3.
                  ManTech Environmental Technology, Inc.1,
                    U.S. Environmental Protection Agency3,
                     Environmental Research Laboratory
                    Dept. of Botany and Plant Pathology,
                          Oregon State  University2
                              Corvallis,  Oregon
INTRODUCTION

   Genetically   engineered   micro-
organisms  (GEMs) released into the
environment could influence microbial
populations and processes in soil, by
altering  nutrient  availability  through
competition, removal of substrate(s),
and/or addition of metabolites.  The
presence  of  GEMs  may   also  alter
predation  patterns and  immobilization
and mineralization processes, affecting
soil  tilth  and  fertility.    Thus,  an
understanding  of  the interrelationship
between GEM  function, differences in
environment  (e.g.,  soil  type  and
microbial   community),   and  'soil
processes is critical.
   We chose to  study Pseudomonas
putida PP0301(pR0103), engineered to
degrade  the  herbicide  2,4-dichloro-
phenoxyacetate (2,4-D); an  ability not
found in its plasmidless, parental strain
(PP0301). Previous work with PP0301
(pR0103)   demonstrated  that   total
fungal  propagule   numbers   were
depressed (both  in the absence and
especially in the presence of 2,4-D) in a
low  carbon soil inoculated with this
GEM. Our study sought to both repeat
this  effect, and   to  examine  other
ecological  endpoints  not   previously
investigated.
    In this  study, we determined the
dynamics of the indigenous populations
of bacteria  (active and total; nitrifying
and  denitrifying  populations);  fungi
(active  and  total);   protozoa,  and
nematodesover an 89-day period in the
following treatments:  (a) unamended
and  uninoculated  soil,  (b)   2,4rD-
amended, uninoculated soil, (c) PP0301
inoculated,   unamended  soil,   (d)
PP0301  inoculated,   2,4-D-amended
soif,  (e) PP0301(pR0103) inoculated,
unamended  soil,  and  (f)   PPO301
(pR0103)  inoculated,  2,4-D-amended
soil.  Additionally, biomass estimates,
concentrations of  inorganic  nitrogen
species, respired carbon (CO2), and pH
were determined.

MATERIALS AND METHODS  '

Sail

    The Xeric/Aridic Frigid soil used in
this study was collected in March 1990
from the same site (Millican Limited Use
Area, near  Bend,  OR),  and character-
ized and stored in the same manner as
that  (obtained in  July  1987) used  in
previous work.
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 Bacterial Cultures

    Pseudomonas  putida   PP0301
 (pR0103)   and   its   parental  strain
 PP0301 were derived previously from
 P.  putida  PP0300  (ATCC  17514).
 These strains are resistant to nalidixic
 acid  (500  jug/ml). Plasmid  pR0103
 encodes resistance to both tetracycline
 (25 //g/mL) and  mercury (25 /vg/mL),
 and the constitutive degradation of 2,4-
 D to chloromaleylacetate. Both strains
 were maintained on either brain heart
 infusion  or  tryptone  nutrient  agar
 supplemented  with  the  appropriate
 selective agents.  The 'presence of the
 plasmid in the GEM was confirmed  by
 DNA  extraction   and   agarose  gel
 electrophoresis prior to the start of the
 experiment.  Inocula of both  bacterial
 cultures were prepared  as in previous
 work.

 Soil Preparation and Microcosms

    Sieved  (2mm-mesh)   soil   was
 adjusted to the -33 kPa  water tension,
 and left to equilibrate at 4°C, with daily
 mixings for 72 hours.  On Day 0, the
 soil was inoculated with ca. 10^ CFU/g
 oven-dry  equivalent  (ODE)  soil  and
 amended  with 2,4-D  (500 //g/g ODE
 soil) where  appropriate.    Following
 ample  mixing, 60 g ODE soil  was
 placed into each of 144 pint-size Mason
 jars and incubated at  25°C in a  light-
 free chamber.   Each  Mason jar was
 closed with an air-tight lid  fitted with a
 rubber septum to  permit headspace
 sampling  for  C02 analysis.  Carbon
 dioxide measurements were conducted
 by  gas  chromatography  (Hewlett-
 Packard  model   5840;   thermal
conductivity   detector;   Porapak   Q
 (80/100  mesh)  column;  helium  gas
 mobile phase).

 Sampling Description

     During  the  89-day  study  period,
 soils were sampled eight times and C02
 analysis was performed  23 times. To
 prevent the accumulation of metabolic-
 ally inhibitory amounts of CO2 (i.e., _>.
 3-4%), all jars were periodically allowed
 to  off-gas for 1-2 minutes in a moist
 chamber. At each soil-sampling period,
 three replicate jars per treatment were
 processed for: % moisture; pH; 2,4-D
 and dichlorophenol  (DCP)  concentra-
 tions  (by  HPLC); inorganic  nitrogen
 analysis; enumeration of the indigenous
 bacteria (nitrifying, denitrifying, active,
 and total), the introduced bacteria, the
 active and total fungi (including hyphal
 length  measurements),  the protozoa,
 and the  nernatodes;  and  microbial
 biomass estimations.  Only  one jar per
 treatment was subsampled  for C02
 analysis, and enough jars  were used
 such that, except for the  last three
 sampling periods, none were sampled
 for C02 on more than one occassion.

 Enumeration of Indigenous Bacterial and
 Fungal Populations

    Bacteria and fungi were enumerated
 both by viable plate counts (soil extract
 agar + cycloheximide (100//g/mL), and
 Martin's medium, respectively) and by
direct counts. Nitrifying and denitrifying
bacterial populations were estimated by
standard MPN techniques.    Ten-fold
(v/v) serial dilutions of each  replicate
were  prepared   in   either  1  mM
potassium phosphate  buffer,  pH 7.2
(for nitrifier and denitrifier enumerations
                                    146

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only) or sterile tap water (for all other
estimates,   including   protozoa).
Aliquots of 1-5 mi_ were removed from
the first soil dilution and prepared as
suspension-agar films for estimates of
active  (fluorescein  diacetate stained)
and total (phase contrast microscopy)
fungi   and   for   hyphal   length
determinations by standard  methods.
Active bacterial numbers and  diameters
were determined by the same method,
under oil immersion.  Total numbers of
bacteria were  determined using  fluo-
rescein  isothiocyanate direct counts.
Diameters  of  fungal  hyphae   and
bacteria were measured  to convert
estimates   of  bacterial  numbers  or
fungal volumes into biomass, using the
average values of 0.41 g biomass/cm3
hyphae and 0.33 g/cm3 bacteria.
                               and
Enumeration   of   Protozoa
Nernatodes
   Protozoa were enumerated  by an
IVIPN  technique  and characterized as
flagellates,  amoebae,   or   ciliates.
Nematode numbers were determined by
extracting 5 gram subsamples of soil in
Baermann extraction funnels.

Inorganic Nitrogen Determinations

   Soil  aliquots of 15  grams/sample
were  extracted  in  30  mL of 0.5 M
K2SO4  and filtered by  gravity-flow.
Filtrates were brought to final volumes
of 30 mL, preserved  in  0.2%  (v/v)
H2SO4,  and  stored at  4°C  prior to
colorimetric analysis for ammonium and
nitrate  species  by  an  autoanalyzer
(Alpkem Rapid  Flow  Analyzer:  RFA-
300;  methods A303-S021 and A303-
S170, respectively). Nitrite,  unstable
under these conditions, was oxidized to
nitrate, and  expressed  as part of the
sum of the two species.

Statistical Comparisons

    Statistical  comparisons  (ANOVA
and   correlation  coefficients)   were
performed using the Statistical Package
for  the Social Sciences (SPSS; Nie et.
al.  1975.  2nd  ed., McGraw-Hill, NY).
Two-way  analysis  of  variance was
performed using time and treatment as
the main effects.  Only those main or
interactive effects found significant by
the F-test at P <  0.05  were consider-
ed.  The mean separation test used was
Least Significant Differences calculated
for  a significance level of P < 0.01.

RESULTS

Concentrations of 2.4-D and  DCP

    During  the  89-day  study,   no
reduction  of 2,4-D concentration was
observed in any of the amended soils.
Extraction efficiencies of 2,4-D  were
ca.  36% of the added material.  Also,
no  DCP  was  detectable  in  2,4-D-
amended soil inoculated with  the GEM.

Soil Moisture. pH, and Respiration

    In spite of the periodic opening of
jars to relieve accumulating CO2 levels,
soil moisture did not vary significantly
during the study.  Soil pH values did
not vary widely over time within the
treatments, although all of the 2,4-D-
amended  soils  were  more alkaline
(perhaps   reflecting   concurrent
reductions  in   nitrifying   bacterial
populations  and  increases  in  NH4 +
                                    147

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 levels).  Furthermore, within the 2,4-D-
 amended soils, those inoculated with
 either the  GEM or • the  parent  were
 significantly  higher  in  pH  than the
 uninoculated  soil.  All 2,4-D-amended
 treatments showed lower rates of CO2
 evolution.

 Funaal  Activity. Hvohal  Length  and
 Number of Prooagules

    During the first week of the study,
 some  contrasting changes in bacterial
 and  fungal  numbers  occurred  in
 treatments  with introduced  bacteria,
 i.e.,  as  fungal  numbers increased,
 bacterial numbers decreased, and vice
 versa.   However,  these  population
 changes were transient, and in no case
 was a prolonged GEM-specific effect
 observed.     Contrary  to  previous
 findings  with  PPO301(pR0103), no
 decline in fungal numbers was seen in
 soil inoculated with the GEM.  In  fact,
 the numbers of fungal propagules were
 significantly higher in  2,4-D-amended
 soils inoculated with either the GEM or
 the parent than in any other treatments,
 when enumerated by the viable plate
 count procedure. Counts of the active
 fungi  suggested, however, that these
 increases were only transient (possibly
 reflecting sporulation).  Both  direct
 counts of total fungi and  total hyphal
 length   measurements' revealed   no
 strong differences between any of the
 treatments (2,4-D-amended or not). In
 general,  the hyphal lengths of active
 fungi   were  decreased  in  all  soils
amended with  2,4-D. .

Introduced Bacteria

   The numbers of both the GEM  and
 parent strains ranged between approx-
 imately 106 to 107 CFU/g ODE soil for
 most of the study. Occassion-ally there
 was a transient decrease in the number
 of both strains to 105. On Day 89, the
 numbers of both  of the introduced
 bacteria in  the unamended treatments
 were undetectable.   The numbers of
 introduced  bacteria in soil  amended
 with   2,4-D   remained   constant,
 suggesting  that the  presence of  the
 herbicide  gave  these  organisms  a
 selective advantage.   At  each  soil
 sampling period, the GEM was pheno-
 typically  distinguishable  from   the
 parental strain.

 Indigenous  Bacteria.  Protozoa,  and
 Nematodes

     No   GEM   effects,   including
 remediation of  2;4-D  effects,  were
 observed.  Bacterial biomass estimates
 for total bacteria and (especially) active
 bacteria, as  well as direct counts, were
 reduced in the presence of 2,4-D. The
 numbers  of  total,  nitrifying,   and
 denitrifying bacteria were also reduced
 in the presence of 2,4-D.
    Nematode  numbers  were  only
 affected by 2,4-D on Day 89.  Neither
 amelioration of this latent 2,4-D effect
 nor significant selection for nematode
 species were  observed  in  GEM  (or
 parent) inoculated treatments.
    The   community  structure   of
 protozoa  was  modified   in   soils
 inoculated with the GEM.  Two species
of  amoebae  appeared  in   greater
numbers  in  GEM-inoculated   soils
relative  to soils inoculated  with  the
parental  strain,  regardless  of 2,4-D
amendment.     However,  protozoal
numbers were  reduced in  all 2,4-D-
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amended soils.

Nitrogen Pools

   Nitrogen transformations  in  both
parent- and GEM-inoculated soils were
affected  by the  presence  of  these
organisms; however, no GEM-specific
effects  were seen.   Relative to the
uninoculated,   unamended   control,
NH4 + levels were higher in both parent-
and GEM-inoculated soils, regardless of
2,4-D amendment. After Day 10, this
was also true for (NO2~ + N03") levels
in unamended soils. The former might
be attributed to nitrogen supplied by
the introduced populations of  bacteria,
but more  probably  reflects  reduced
nitrification in the 2,4-D treatments as
nitrifying  bacteria were  suppressed.
The  levels of  (NO2~  +  N03")  were
consistently  reduced  in  all  2,4-D-
amended soils.  However, the levels of
NH4+ were consistently increased by
2,4-D in these soils.

DISCUSSION

   Contrary  to  earlier  work  with
PPO301(pRO103), we saw no  GEM-
mediated   influences  on   2,4-D
degradation or numbers of fungi.  Yet,
the GEM  maintained  its  phenotypic
traits  throughout  the experiment, and
the pR0103 plasmid  has  been shown
to  be stable in PP0301. Given the
similarities  (e.g., the  same soil source
and handling; manner of both addition
and extraction/analysis from  soil, as
well as concentration of 2,4-D; size and
preparation of inocula, etc.)  between
these  experiments, these findings are
surprising.  In  earlier studies,  addition
of  500   ppm  2,4-D  resulted in  the
recovery  of  66%   of   the   added
chemical, while we recovered only 36%
of the added 2,4-D on Day 0 and in
subsequent  samplings.  Short et,  al.
(1991) found a steady decline of 2,4-D
from  500 ppm to <100  ppm  in  53
days.   They  attributed  this to the
degradation  of 2,4-D (and  subsequent
appearance of  the metabolite DCP)  by
the added GEM.
    We believe our findings may reflect
changes in Millican soil, which caused
the   extraction   of   2,4-D  to   be
compromised  in our  study.  Of the
differences  that  we  are  aware  by
chemical analysis between Millican soil
collected in 1987 and in 1990, perhaps
two are  most significant.   Grazing
pressure,  or  at  least  cattle   usage
patterns,  on  this  land  may   have
changed.  Inorganic nitrogen (i.e., NO3"
and NH4 + ).levels were 2-3 times lower
in soil collected in  1990.  Also, this soil
had a  higher organic  content (1.8  ±_
0.2%  in  1990 vs. 1.3 ±_ 0.1%  in
1987).
    An increased  (albeit low) level of
organic matter may have reduced the
extraction efficiency (and availability to
the  GEM)  of  2,4-D  by  promoting
increased soil binding and incorporation
of the compound (e.g., to humic acid).
We never recovered  more than 200
ppm 2,4-D.  Short et al. (1991)  found
that as 2,4-D  decreased  below 150
ppm,   the  concentration  of   DCP
decreased as well. Although this may
reflect the instability of DCP, another
explanation could be that a minimal titer
is required to activate the catabolism of
2,4-D  by  PPO301(pRO103).   In our
study, no DCP was produced, no 2,4-D
was removed, and no DCP effects were
observed.    We  speculate that  not
                                    149

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 enough  2,4-D was available for the
 GEM to be activated.

 FUTURE WORK

    We hypothesize that if 2,4-D were
 available (i.e.,  extractable) in Millican
 soil  to PPO301(pRO103) in  sufficient
 concentrations,  the   GEM-mediated
 effects of  2,4-D  degradation,  DCP
 accumulation,  and  decreased fungal
 populations would occur.  We plan to
 test  this  in  the  series  of  short
 experiments   (using   Millican   soil
 collected  in  1990),  detailed  below.
 Unfortunately, our reserves of Millican
 soil  collected in  1987 are  depleted.
 However, prior to the initiation of this
 work,  we will verify that our present
 strains of PPO301(pRO103)  have the
 ability to degrade 2,4-D in culture.
 (a) Titration study of 2,4-D iri Millican
   soil.
   Objective:    To  determine  that
   amount of 2,4-D recoverable as 500
   ppm.
 (b) Comparative  extraction  of 2,4-D
   (amount recoverable Millican soil as
   500  ppm)  from  three  different
   "soils": low carbon (Millican);  high
   carbon (Thatuna -Naff); no carbon
   (silica sand).
   Objective:    To  verify  that   soil
   composition affects  the extraction
   efficiency of 2,4-D.
(c) Small scale repeat of present study,
   but using the  2,4-D  amount which
   can be recovered as  500 ppm.
   Objective:  To determine  if higher
   levels  of  2,4-D  amendment  will
   result in the GEM-mediated effect of
   depressed fungi.
 PUBLICATIONS   .

 Ingham, E. R., Gander, L. K., Doyle, J.
 D.  and C.  W.   Hendricks.   1991.
 Assessing   interactions   between   a
 genetically engineered microorganism,
 target  toxic chemicals and the soil
 foodweb.  (manuscript in progress).

 Doyle, J. D., Short, K. A., Stotzky, G.,
 King, R. J. and R. H. Olsen.  1991.
 Ecologically  significant   effects   of
 Pseudomonas put/da PPO301 (pR0103),
 genetically engineered to degrade 2,4-
 dichlorophenoxyacetate on  microbial
 populations and processes in soil. Can.
 J. Microbiol. Accepted.

.Short, K. A., Doyle, J. D., King, R. J,,
 Seidler,  R.  J., Stotzky, G. and R. H.
 Olsen.    1991.    Effects  of   2,4-
 dichlorophenol,  a  metabolite  of  a
 genetically  engineered bacterium and
 2,4-dichlorophenoxyacetate on some
 microorganism-mediated    ecological
 processes  in  soil.   Appl.  Environ.
 Microbiol.  57: 412-418.

Short, K. A., Seidler,  R. J., and R. H.
Olsen. 1990. Survival and degradative
capacity  of  Pseudomonas  put/da
induced  or  constitutively  expressing
plasmid-mediated  degradation of 2,4-
dichlorophenoxyacetate in soil. Can. J.
Microbiol. 36: 821-826.
                                    150

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      EFFECTS OF A LIGNIN-DEGRADING RECOMBINAIMT Streptomyces ON
            MICROBIAL ACTIVITY AND NUTRIENT CYCLING IN SOIL
      Don L. Crawford1, Zemin Wang1, Jack D. Doyle2, Harvey Bolton, Jr.3,
      James K. Fredrickson3, Steve A. Bentjen3, and Charles W. Hendricks4
                 Department of Bacteriology and Biochemistry1
           Institute for Molecular and Agricultural Genetic Engineering
                      University of Idaho, Moscow, Idaho
                   ManTech Environmental Technology, Inc.2
              Environmental Research Laboratory, Corvallis, Oregon
          Battelle Pacific Northwest Laboratory3, Richland, Washington
                    U.S. Environmental Protection  Agency4
              Environmental Research Laboratory, Corvallis, Oregon
 INTRODUCTION

   This research was a collaborative
effort between the University of Idaho,
the   Battelle  .Pacific   Northwest
Laboratory,  and EPA's  Environmental
Research  Laboratory-Corvallis.   The
research  was  designed  to  provide
information on  the magnitude, extent,
and   mechanisms   by   which   a
recombinant   lignin-degradation
enhanced  Streptomyces strain effected
the integrity of a soil ecosystem,  the
need  for  the  study stemmed  from
previous findings  in  D.L. Crawford's
laboratory at the  University of  Idaho
(EPA   Cooperative  Agreement  CR-
815300-01-05)   showing  that
recombinant   S.   lividans  strains
expressing  a   plasmid-encoded  gene
coding  for bacterial lignin  peroxidase
(1), when  released into soil, transiently,
but significantly affected the  rate  of
carbon  mineralization  in the  soil  as
measured  by an increased rate of C02
evo.lution from the soils (2).  The effect
wtas particularly significant in nonsterile
soils  containing  recently   deposited
lignocelulosic residues.   It  was  found
that this  carbon mineralization  effect
 resulted from increased production of
 lignin peroxidase by the recombinant in
 soil.   It  was  hypothesized that  this
 resulted in  a transient  increase in the
 rate of lignin depolymerization, which in
 turn  more  quickly opened the lignin
 barrier   and  temporarily   stimulated
 lignocellulose  decomposition  by  the
 native cellulolytic soil microflora.  The
 most  affected  lignin  was likely' the
 relatively undegraded  lignin  recently
 deposited in the  soil.   In  effect, the
 recombinant transiently relieved a rater
 limiting, step in carbon  cycling in soil,
 that is lignin depolymerization. This in
 turn temporarily made  more cellulosic
 polysaccharides available to the non-
 ligniaolytic,  cellulolytic  soil microbial
 flora.   This resulted in a  short term
 spike in microbial  metabolic activity.
     In addition to carbon mineralization
 effects,  this  genetically   engineered
 microorganism (GEM) might have other
 effects on  the soil ecosystem.  The
 objective of the just completed' multi-
 investigator project was to determine
"what  those  effects,  if  any,  were.
 Ecological   parameters   examined
 included   1)   degradation   of  low
 molecular  weight  aromatic   and/or
                                     151

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 chloroaromatic compounds in the soil,
 2) incorporation of organic carbon into
 soil   humus,   3)   effects  on  other
 biogeochemical cycles  including  the
 nitrogen,  sulfur,   and  p'hosphorus
 cycles,  and,  4)  effects   on  total
 microbial biomass,  specific  microbial
 populations,  and the activities of soil
 microbial  enzymes   within  the  soil
 environment.    Results  showed that
 while  no  long  term  environmental
 effects on these parameters apparently
 resulted  from the release of the GEM,
 certain  statistically  significant,  but
 transient  effects   were  measured.
 These findings confirmed the previous
 observations of Crawford's group, but
 extended the findings  of measurable
 transient effects   to  several  other
 components of the soil ecosystem.

 METHODS

   The  soil  used  was from a  single
 batch  of a Palouse  silty  loam  rich in
 organic matter^(3).   This  soil was air-
 dried, sieved,  and split into equal lots,
 which were distributed to investigators
 and  then stored at  4°C until  used.
 14C-lignin  and  14C-cellulose labeled
 lignocelluloses were  prepared  from
 poplar and  chemically characterized as
 previously described (4). The wild-type
 microbial strain used was Streptomyces
lividans TK23, a prototroph expressing
chromosomally encoded resistance to
spectinomycin. The recombinant strain
was   Streptomyces  lividans TK23.1
which expresses plJ702.LP encoding a
chromosomal lignin peroxidase gene of
Streptomyces viridosporus T7A.  this
plasmid  also  encodes   thiostrepton
resistance  as  a  selectable marker.
Maintenance and characterization of the
 strains was as  described  previously
 (1,2).     Enumeration   of   the
 Streptomyces in soil was monitored via
 plate counts onto selective media, using
 procedures  similar to those described
 previously  (2,3).   Maintenance  and
 expression  of   plJ701.LP  by   the
 recombinant cells  in  soil  was  also
 monitored  as  previously  described
 (2,5).  C02 evolution from sterile  and
 nonsterile control soils and from soils
 inoculated  with  the  recombinant  or
 wildtype  strain  was   monitored   as
 described previously  (2,3).    14C02
 evolution  and incorporation of 14C into
 soil  humic   and  fulvic  acids  was
 measured as described previously (6,7).
 In   humification   experiments,
 incorporation   of  14C-lignocellulose
 derived 14C  into the different fractions
 of soil  organic matter was examined by
 extracting and analyzing the soil for14C
 labeling of the soil fulvic acids, humic
 acids,  and humans (7).  Soil microbial
 biomass   (C  and  14C-labeled) was
 quantified   by   the  soil   chloroform
 fumigation technique  (8), while total
 soil carbon (C and 14C) was determined
 by soil  oxidation.  Soil inorganic (NH4-N
 and NOg-N) determinations were by  the
 method of Keeney and Nelson (9), soil
 biomass nitrogen by the soil chloroform
 fumigation technique (8), and total soil
 nitrogen by  Dumas combustion.  Soil
enzyme assays included  those for acid
and  alkaline  phosphatase,  dehydro-
genase,   (J-glucosidase,   peroxidase,
arylsulfatase,  and cellulase.    These
assays were carried out  as described
previously (1,10-13).   In  addition,  in
some experiments soluble phenolics,
pH, and water content of the soils were
monitored.    In  some  experiments
involving  unamended soils or  soils
                                     152

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amended   with  lignocellulose   (un-
inoculated  or  inoculated   with   the
wildtype or recombinant Streptomyces
strains),  specific  cultivable,  aerobic
microbial populations were  monitored
using plate counts, and included total
bacteria,  total   fungi,   cellulose
decomposers, chitin  utilizing bacteria,
total spore-forming bacteria, nitrifying
bacteria, and  denitrifying bacteria. The
media  used  for  these enumerations
have  been described (3,9,10,13-16).
The  experiments  were carried out in
"Master  jar"   microcosms  (17).   All
experiments were done in  3 or  more
replicates  with  values  reported  as
averages   ±   standard   deviations.
Additional statistical analyses were also
performed  on all   data,  and  each
experiment was repeated at least two
times.

RESULTS

   The objective  of this research was
to evaluate the effects of release of S.
//V/tfansTK23.1 (expressing plJ702.LP)
on   microbiological   activities   and
biogeochemical cycling or carbon and
nitrogen  in soil.   While some  repro-
ducible, statistically significant transient
effects were  indeed  observed, among
the environmental parameters examin-
ed, this GEM appears to have had no
detrimental long term (> 60-90 days)
effects on the soil ecosystem.
   The GEM  specific enhancement'of
short term CO2 evolution  rates from
soils inoculated with  strain TK23.1, as
compared to  control soils  and those
inoculated with strain TK23 (2),  was
confirmed.
   For nonsterile  soils inoculated with
the wildtype or recombinant strain, and
supplemented with 14C-lignin labeled
lignocellulose,  the  rate   of   14C02
evolution  from  the  soils  was  not
significantly  different  from the ' rate
observed  for  nonsterile  uninoculated
soils over a  30 day  periods.  Thus,
neither the parent or GEM measurably
altered the net rate of lignin mineral-
ization.   14C02 evolution  rates from
soils supplemented with  14C-cellulose
labeled lignocellulose were significantly
greater for soils inoculated with TK23.1
as compared to TK23; however, the
rate  for TK23.1 inoculated  soils  was
not  significantly  different  from  the
uninoculated control soil.
    After 30 days, incorporation of 14C
into the humus fractions of these same
soils  was " determined.     In  soils
supplemented   with   14C-lignih
lignocellulose, the amount of 14C-humic
acid  recovered was  greater for soils
inoculated with the GEM (TK23.1) than
with the parental strain (TK23), and the
value was higher for  TK23-inoculated
soil than for nonsterile,  uninoculated
soil.    However,  by  60  days  this
difference had disappeared,  such  that
the control soil value was greater then
the  TK23.1  value,  which  was  still
greater than the TK23 value. After 30
days, the amount of 14C recovered in
fulvic acid fractions  was  somewhat
lower for  soils inoculated with TK23
and TK23.1 as compared to the control
soil;  however, after 60 days the soils
inoculated  With   TK23.1   contained
somewhat more 14C-fulvic acids  that
either   the   TK23-inoculated   or
uninoculated   soils.     For   soils
supplemented   with  14G-cellulose
labeled lignocellulose, after 30 days the
amount of 14C incorporated into soil
humic acids was significantly greater
                                     153

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for TK23.1-inoculated soil as compared
to TK23-inoculated soil, and the value
for TK23-inoculated soil was greater
than   for  the   uninoculated   soil.
However, after 60 days there were no
significant differences  between the
three soils.  After 30 days the level of
14C incorporation into fulvic acids was
higher for TK23.1-inoculated soils than
for the TK23-inoculated or control soils.
Again, however, the values were not
significantly different after 60 days.
   In  the water   content, soil  pH,
soluble   soil   phenolics, and  TK23/
Tk23.1   population  studies,  it  was
observed that the water content of the
soil  remained stable  throughout the
experiments.     The   presence   of
lignocellulose in the soil'affected soil
pH, which rose about 0.5 units over the
first  35 days of the experiment before
stabilizing.  In unamended  soil  there
appeared to be a transient  increase in
pH due to presence of the GEM.  In
lignocellulose amended  soil, the GEM
appeared to significantly increase soil
pH relative to controls.   Thus, the
effects of  lignocellulose ori the  other
measured variables  could be related to
this  pH effect.  The survival of  both
TK23 and TK23.1 was similar through-
out the study. The numbers of the GEM
in unamended  soil remained  greater
than those in lignocellulose amended
soil.  After 14 days, the numbers of the
GEM  in  lignocellulose  amended soil
approximated  those of  the parental
strain in both unamended  and ligno-
cellulose  amended  soil.   Thus, the
presence of lignocellulose did not give
the GEM a selective advantage over the
parental  strain.  There was no  signif-
icant  effect of the GEM or parental
strain on the total phenolics content of
the  soil  in  either amended  or  un-
amended  soils over the time of  the
experiment (90 days).
    The enzyme studies showed some
transitory   GEM   specific  effects.
Greater  arylsulfatase   activity   was
present in soil  amended with  ligno-
cellulose as compared to unamended
soil, regardless  of  whether the soils
were  inoculated or  not  inoculated.
There   was   a  transient  (3  day)
enhancing  effect   of  the  GEM  on
arylsulfatase   activity  in   both
unamended and amended soils.   There
was   an   initial  increase   in   acid
phosphatase activity in all of the soils,
but the activity than leveled off for the
remainder of the study.  There was no
measurable effect of the GEM on acid
phosphatase activity in either amended
or  unamended  soils.     In  general a
greater amount alkaline phosphatase
activity was detected in soils amended
with lignocellulose than in unamended
soils,  regardless of whether the  soil
was  inoculated  or not inoculated.
There was typically a small  increase in
activity which then leveled  off for  the
remainder  of  the  study.    Alkaline
phosphatase  activity  was,  however,
transiently  enhanced during the first
week of incubation in  GEM-inoculated
soils   amended  with   lignocellulose.
Similarly,  soil   dehydrogenase  was
transiently increased in GEM inoculated
soils.  Soil peroxidase activity was  not
significantly  affected   by  the   GEM,
although it is likely that peroxidase was
tightly bound to lignin-derived organic
matter in  the soil  and  therefore  not
                   f
easily  measured by  the techniques
employed.
    In  all  soils,  over time  there was
typically  a  slight  increase  in  total
                                     154

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bacterial   numbers   followed  by   a
decline.   By day  91, however,  the
numbers  of  culturable  bacteria had
generally  stabilized.    There  were
measurable   increases  in  bacterial
numbers,  as compared  to the other
soils,  in  soils  amended  with  ligno-
cellulose and inoculated with either the
GEM  (TK23.1)  or  the parental strain
(TK23). However, bacterial numbers in
the soil amended  with  lignocellulose
and  inoculated  with TK23.1, were
transiently  lower than  for  those  in
lignocellulose amended soil inoculated
with TK23.  When the numbers  of
added  bacteria  (TK23  and TK23.1)
were subtracted from the  numbers of
total bacteria,  the  numbers of  total
bacteria in soils inoculated with TK23.1
were  transiently lower than those in
uninoculated, unamended  soils or un-
amended soils inoculated  with TK23.
As expected, bacterial numbers in soils
amended   with  lignocellulose were
generally  higher than  in  unamended
soils.
    In all soils from time zero there was
typically an initial  10-fold increase in
culturable fungal propagules, followed
by a decline and stabilization over time.
Relative to  controls,  the  numbers of
fungal propagules   were  transiently
depressed in the presence of the GEM
in soils amended with  lignocellulose.
This may  be seen  in  the  result  of
increased  competition form the more
active bacterial microflora.
    In general, from time zero there was
an  initial increase  in  the  numbers of
spore-forming bacteria, followed by a
decline, subsequent recovery, and then
stabilization of numbers.  In unamended
soil  inoculated  with the  GEM,  the
numbers   of  spore-formers   were
significantly greater then those in soil
inoculated with the parental strain, but
only between days 14 and 35. In soil
amended   with   lignocellulose   and
inoculated with the GEM, the number of
spore-formers  were fewer  than  in
amended  soil  inoculated  with  the
parental  TK23  strain, but  only on
sampling days 14. 35, and  63.  By day
63, the numbers of spore-formers were
lower  in  soil amended  with  ligno-
cellulose than in the unamended soil.
The numbers of added  bacteria (TK23
or   TK23.1)   had   essentially  no
measurable effect on the numbers of
spore-formers.
    As would be expected, there were
some general differences in number of
cellulose  decomposing  bacteria  in
unamended soils versus those amended
with  lignocellulose.   However,  in
general no differences were seen in the
numbers of cellulose-degraders in soil
amended   with   lignocellulose   and
inoculated with the GEM as compared
with those that were uninoculated or
inoculated  with  the  parental strain.
There  was, however,   a long  term
difference in the numbers  of cellulose
degraders  in   unamended   soils
inoculated with the GEM as compared
to  unamended uninoculated soil  or
unamended  soil  inoculated  wi  the
parent.   When the total numbers of
added bacteria  were subtracted  from
the numbers of cellulose-degraders, the
effect due to the GEM  in uninoculated
soil disappeared,  suggesting that the
addition of TK23.1  to unamended soil
may affect the numbers  of  cellulose-
degrading   bacteria   in   that   one
treatment.
    Again, there were some differences
between populations of chitin utilizing
                                    155

-------
 bacterial populations in  soil amended
 with  lignocellulose  as  compared to
 unamended soil.  There was a transient
 effect of the GEM in unamended  soil,
 but only on one sampling day (day  14).
 This effect remained statistically valid
 when the  number of added  bacteria
 were subtracted  from the  numbers of
 chitin utilizing bacteria.
    In general, the numbers of nitrifying
 and   denitrifying  bacteria  were  not
 significantly affected by introduction of
 the GEM or parental strain into any of
 the soils, and no appreciable effects on
 nitrogen cycling were observed.
    Overall, the results show that a few
 statistically significant effects on  the
 measured  environmental  parameters
 were observed during the course of this
 research.    In  all cases,  the effects
 tended  to  be  small  and transient.
 These results  generally  support  our
 hypothesis  that  the  GEM  transiently
 alters the rate of lignin depolymerization
 in the soil  and,  thereby,  temporarily
 stimulates  the  saprophytic soil micro-
 flora. The  data,  particularly from the
 microbial populations and soil  enzyme
 activity studies, support this conclusion
 and  extend our  original observation
 concerning  the  CO2  mineralization
 effect (2).  The transitory nature of the
 measurable effects, and also the humif i-
 cation study,  support the conclusion
that  these  effect are of  little, if any,
significance to  the long term stability
and health  of this soil ecosystem.

REFERENCES

 1)  Wang,   Z.,  B.H.  Bleakley,   D.L.
Crawford,  G.  Hertel, and F.  Rafii.
 1990.   Cloning and  expression of a
lignin  peroxidase   gene   from
 Streptomyces   viridosporus   in
 Streptomyces lividans.  J. Biotechnol.
 13:  131-144,

 2)   Wang,  Z.,  D.L.Crawford,  T.S.
 Magnuson,   B.H.   Bleakley,   and  G.
 Hertel.   1991.  Effects of  bacterial
 lignin  peroxidase  on organic carbon
 mineralization in soil using recombinant
 Streptomyces  strains.     Can.   J.
 Microbioi. 37: In Press.

 3)  Wang,   Z., D.L.  Crawford,  A.L.
 Pometto III,  and  F.  Rafii.    1989.
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4) Crawford, R.L. and D.L. Crawford.
1988.  14C-[lignin]-lignocelluloses and
14C-milled  wood  lignins:  preparation,
characterizations and  uses.
Enzymol. 161B:  18-31.
Meth.
5) Rafii, F. and D.L. Crawford,  1988.
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mobilization   of   a   nonconjugative
plasmid between Streptomyces strains
on  agar and in soil.   Appl. Environ.
Microbiol;  54:  1334-1340.

6) Crawford, D.L., R.L. Crawford, and
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7) Paul,  E.A.  and  F.E. Clark.   1989.
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10)  Tabatabai,  M.A.   1982.    Soil
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11) Bartha, R. and L. Bordeleau. 1969.
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12)  Sichak, P.S. and A.L. Dounce.
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13) Martin, J.P.  1950. Use of  acid,
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14)  Hsu,  S.C.  and J.L.  Lockwood.
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actinomycetes in water and soil.  Appl.
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15)  Scales,  P.M.    1916.   A  new
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Madison, Wl.

ANTICIPATED FUTURE RESEARCH

    It would be of interest to  examine
the long term (e.g., a year or more) fate
of the GEM (TK23.1) and  its plasmid in
several  different  soil types.   More
importantly,  since  humification is  a
slow process, it would be of interest to
examine the effects of the GEM  on the
humification process over much  longer
periods of time than could be done in
the present project.  Effects not seen
over 30 days might  show  up over
longer  period  of  time.   Finally, this
project has shown that the procedures
employed  are  sufficiently sensitive to
detect effects if they occur.  Thus, it
would  be  of value to   utilize  these
procedures to evaluate environmental
effects  of   other  GEMs   in   soil
ecosystems.

PUBLICATIONS

Crawford,  d.L., Z. Wang, H. Bolton, Jr.,
J.D.  Doyle,  J.K.  Fredrickson,  S.A.
Bentjen, and  C.W. Hendricks.  1991.
Effects  of   a  recombinant   lignin-
degrading  Streptomyces  on  microbial
activity and nutrient cycling in soil.
                                    157

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          MECHANISMS OF EFFECTS OF RECOMBINANT Streptomyces
                        ON THE CARBON CYCLE IN SOIL

              Don L. Crawford, Zemin Wang, and Bruce H. Bleakley
                  Department of Bacteriology and Biochemistry
            Institute for Molecular and Agricultural Genetic Engineering
                              University of Idaho
                                Moscow, Idaho
 INTRODUCTION

    In  our  recently  completed  EPA
 cooperative agreement CR-815300-01 -
 05, we carried out research to elucidate
 the mechanism by which a recombinant
 Streptomyces   strain,   genetically
 engineered   to  overproduce   an
 extracellular   lignin   peroxidase,
 transiently enhanced the rate of carbon
 turnover in soil. The gene encoding the
 lignin peroxidase was obtained from the
 chromosome of the lignin-solubilizing
 actinomycete   Streptomyces
 viridosporus T7A and was cloned into
 Streptomyces lividans TK64 in plasmid
 plJ702 (1).  This enzyme catalyzes the
 oxidative depolymerization  of  lignin
 during lignocellulose degradation and is
 involved  in  the  ligning  solubilization
 process of S. viridosporus (1,2).  The
 need  for the present study stemmed
 from  our previous  observation  that
 certain   genetic  variants   of   S.
 viridosporus temporarily enhanced the
 rate of  carbon  turnover in  soil,  as
 measured by CO2 evolution rates from
 soils inoculated with the variants (3).
 The   enhancement   was  particularly
 significant  when the  variants  were
 released  into nonsterile soils amended
 with lignocellulose. Evidence implicated
 overproduction of lignin peroxidase by
the variants as a possible mechanism
for the effect, since they characteristic-
ally, but transiently overproduced lignin
peroxidase as the result of spontaneous
chromosomal  gene   amplifications.
However, because the variations were
unstable,   the  mechanisms  of their
effect on  carbon turnover rates-could
not be easily studied.  The availability
of a recombinant  lignin  peroxidase-
overproducing S. lividans TK64 strain
(1),  however,  made  such  a  study
feasible, and  it also raised the question
of how such a genetically  engineered
bacterium might effect the soil carbon
cycling  if  it  were  released  into the
environment.  The  objectives of this
past year's reseach were to elucidate
the genetic and chemical bases for the
carbon mineralization effect, using the
recombinant  S. lividans strains  [strain
TK64.1  expressing lignin  peroxidase
gene ALip-P3 in plasmid plJ702.LP (2)].
TK64.1 was inoculated into soil, and its
effects  on  carbon  turnover   were
monitored  and compared  to  control
soils and  soils  inoculated  with  the
nonrecombinant  parental  S.  lividans
TK64 strain.  In addition to  monitoring
carbon mineralization effects, we also
examined   how  release   of  the
recombinant strain affected  the rate of
incorporation of  lignocellulosic carbon
into soil humus fractions.
                                     158

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METHODS

   The parental S.  lividans TK64  and
recomblnant S. lividans TK64.1  strain
expressing lignin peroxidase-encoding
plasmid plJ702.LP have been described
previously (1,2).  A gene encoding the
ALiP-P   lignin   peroxidase  of   S.
viridosporus (2) was cloned on a 4.1 kb
fragment  of  chromosomal  DNA  into
multi-copy vector PIJ702 to construct
plJ702.LP(1).
   Peroxidase   activity   and   H202
concentrations  were   measured   as
described by Ramachandra et al.  (2).
Plasmid   isolations   and   character-
izations, Southern hybridizations,  and
other  molecular analyses  of  DNA
preparations   were   performed   as
described by Wang eta/. (3,4). Growth
of   wild-type   and   recombinant
Streptomyces   strains   in   soil,
Streptomyces inoculation procedures,
selective   enumeration   procedures,
measurement of carbon mineralization,
and the Palous silt loam soil used in the
study have been described by Wang et
al. (3,4). . Preparation of 14C-lignin  and
14C-celluiose  labeled   lignocelluloses
was  as  previously   (5,6),  and  incor-
porated of  14C  into soil  humic  and
fulvic   acids   was   monitored   by
extraction of the acids from soil  and
analysis by liquid scintillation counting
after combustion to C02.

RESULTS

   When recombinant strain S, lividans
TK64.1,  which  expresses  a  lignin
peroxidase gene in  plasmid  plJ702.LP
(1), was released into a rich organic soil
in, flask scale laboratory experiments,
the rate of CO2 evolution from the soil
was temporarily  enhanced  (4).  The
enhancement was shown to be lignin
peroxidase specific, and significant only
in  nonsterile soils still containing  an
active microbial flora.  The increase in
carbon  mineralization  resulted from
increased  production  of  lignin  per-
oxidase by  the recombinant strain in
soil.   This  resulted  in a  transitory
increase in the rate of lignin depolymer-
ization,  which  in  turn   temporarily
stimulated lignocellulosedegradation by
the native soil microflora. The affected
lignin was  likely the  relatively  un-
degraded  lignin that  had been recently
deposited  in the  soil.  In  effect,  the
recombinant strain- transiently relieved
a rate-limiting step in carbon cycling in
soil, that  is lignin  depolymerization.
The increase in lignin depolymerization
rate in  turn temporarily  made more
cellulosic  polysaccharides available to
the  celluloytic,  non-ligninolytic   soil
microbial population. This resulted in a
temporary  spike  in  the  metabolic
activity  of the  microflora.   While the
effect was transitory,  this  is the first
report  of  a  genetically  engineered
microorganism (GEM) having a measur-
able effect  on  a  soil  biogeochemical
cycle.  Studies  of the incorporation of
carbon into  the humus fractions of the
soil showed that  the GEM-caused en-
hancement  of  soil  organic  carbon
mineralization    probably   did   not
significantly effect the soil humification
process.   The  rate of incorporation of
organic  carbon  into soil  humic and
fulvic acids was not significantly altered
by  introduction of  the GEM.    This
conclusion  was  based upon  results
showing that when a rich organic soil
inoculated with the recombinant strain
was amended  with 14C-Iignin or  14C-
                                     159

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cellulose   labeled   lignocellulose,
incorporation of 14C into soil humic and
fulvic  acid  fractions  over a  60 day
incubation period was not significantly
different as compared to control soils or
those   inoculated  with   the   non-
recombinant parental strain. Thus, the
overall effect of this GEM  on the soil
ecosystem,  at  the  level  of carbon
turnover, appears to be measurable, but
short term.   The observed temporary
spike in carbon  mineralization probably
has little if any  long term effect on soil
humification or  fertility.

REFERENCES

1)   Wang,  Z.,  B.H.  Bleakley,  D.L.
Crawford,  G.  Hertel, and  F.  Rafii.
1990.   Cloning and  expression  of a
lignin   peroxidase   gene   from
Streptomyces   viridosporus   in
Strepomyces lividans.  J.  Biotechnol.
13: 131-144.

2)  Ramachandra, M.,  D.L. Crawford,
and G. Hertel.  1988.  Characterization
of an extracellular lignin peroxidase of
the   lignocellulolytic   actinomycete
Strep torn yces  viridosp or us.     A p p I.
Environ. Microbiol.  54: 3057-3063.

3)   Wang,  Z., D.L.  Crawford,  A.L.
Pometto   III, and F.  Rafii.    1989.
Survival  and   effects of  wild-type,
mutant, and  recombinant Streptomyces
in a soil ecosystem. Can. J. Microbiol.
35: 535-543.

4)   Wang,  Z., D.L.  Crawford,  T.S.
Magnuson,  B.H.  Bleakley,  and  G.
Hertel.   1991.   Effects  of  bacterial
lignin peroxidase  on  organic carbon
mineralization in soil using recombinant
Streptomyces  strains.
Microbiol.  In press.
Can.   J.
5)  Crawford, R.L. and D.L. Crawford.
1988.   14C-[lignin]-lignocelluloses and
14C-milled  wood  lignins:  prepration,
characterization,  and  uses.    Meth.
Enzymol.  161B: 18-31.

6) Crawford, D.L., R. L. Crawford, and
A.L. Pometto III. 1977.  Preparation of
specifically labeled 14C-lignin and 14C-
cellulose labeled   lignocelluloses  and
their decomposition by the microflora of
soil.  Appl. Environ.  Microbiol.  33:
1242-1251.

ANTICIPATED FUTURE RESEARCH

    It is not currently anticipated that
additional research will  be carried out
on this project. It  would be of interest
to study the long  term (e.g.,  a year)
fate of the plasmid  bearing recombinant
S. lividans  TK64.1 in  soil, particularly
with  regard  to  maintenance  and
expression  of the lignin peroxidase-
encoding plasmid.  It  would  also be
valuable -to extend  the humification
effects  study out  to a  year or more.
Humification   is   a   relatively  slow
process, and  statistically  significant
effects  of  the GEM  might  not  be
observed over the short periods of time
covered in the present research.

PUBLICATIONS

Wang, Z., D.L. Crawford, A.L. Pometto
III, and  F. Rafii.   1989.  Survival and
effects  of   wild-type,  mutant,  and
recombinant  Streptomyces in  a  soil
ecosystem.   Can.  J.  Microbiol.  35:
535-543.
                                     160

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Wang,Z., B.H. Bleakley, D.L. Crawford,
G. Hertel, and F. Rafii.  1990.  Cloning
and expression  of a  lignin peroxidase
gene from Streptomyces viridosporus in
Stfepomyces lividans.  J.  Biotechnol.
13: 131-144.

Wang,  Z.,   D.L.    Crawford,   T.S.
Magnuson,  B.H.  Bleakley,  and  G.
Hertel.  1991.   Effects of bacterial
lignin peroxidase  on  organic carbon
mineralization in soil using recombinant
Streptomyces   strains..    Can.   J.
Microbiol.  |n press.   (April).

Bleakiey,  B.H.  and  D.L.  Crawford.
1991.;Plasmid  exchange and hetero-
trophic   activity   associated  with
colonization of artificial soil aggregates
by  Streptomycetes.    Submitted  for
Publication (April 1991).
Abstracts and Presentations

Crawford, D.L.   1989.   Effects  of
recombinant Streptomyces  oh a soil
ecosystem.    Presented,   EPA
Biotechnology  Risk  Assessment  All
Investigators Meeting and Peer Review.
EPA Corvallis Environmental Research
Laboratory, November 14-16.

Crawford, D.L. 1989. Mechanisms of
effects of recombinant Streptomyces
on the carbon cycle in soil. Presented,
89th Annual Meeting, American Society
for  Microbiology.  New Orleans, LA.
May 14-18.

Crawford, D.L.   1990.   Effects  of
recombinant  Streptomyces  in  soil.
Presented,   90th  Annual   Meeting,
American  Society  for Microbiology,
Anaheim, CA.  May 13-17.
                                    161

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  LABORATORY OBSERVATIONS ON THE INHIBITION OF SOIL FUNGI BY MPCAs
                         OF THE GENUS Pseudomonas

                  H.M. Abebe1, V.P. Fieland1, and R.J. Seidler2
                    ManTech Environmental Technology Inc1
                     U.S. Environmental Protection Agency2
                       Environmental Research Laboratory
                              Corvallis, Oregon
INTRODUCTION

    Biotechnological   methods   have
stimulated   interest  in  the  use  of
microorganisms  to  ease the  environ-
mental impact posed by anthropogenic
toxic chemicals.  One  aspect of the
studies  conducted  in  our laboratory
over the last several years, involved an
examination  of  the   genetics,  ex-
pression, and metabolic controls of 2,4-
D and phenoxyacetic acid degradation
in fluorescent pseudomonads.  In one
xenobiotic model system, the  use of a
deregulated   operon    (continuously
expresses the  ability  to  biodegrade)
has  been proposed  to stimulate the
bioremediation of phenoxyacetic acid
and   2,4-Dichlorophenoxyacetic  acid
(2,4-D). However, before these kinds of
modified strains (genetically engineered
microorganisms) can be field tested, it
has been proposed that a series of test
methodologies be developed to evaluate
the  ecological consequences  of  their
releases. Such methodologies need to
be accurate, reliable, economical and
easily applicable for appropriate  risk
assessment studies.
   In a  series of ecological studies
designed to evaluate the consequences
of  2,4-D biodegradation on microbial
ecosystems, we demonstrated an un-
expected accumulation of a metabolic
intermediate (2,4-dichlorophenol) that
reduced cell numbers of indigenous soil
fungi.  Similarly, in control experiments
where 2,4-D  amendments were not
made, we found evidence that the fluo-
rescent pseuddmonad,  Pseudomonas
putida PP0301  also caused a decline in
the  number  of  fungal  propagules
present in  nonsterile soil.  Although
fluorescent    pseudomonads   are
important (agronomically) in the control
of  fungal  plant  diseases such  as
damping off of cotton arid take  all of
wheat, very few  attempts have  been
noted to study the  effects  of these
bacteria on beneficial fungi.
    The  present  study  represents  a
follow up of our initial observations and
involves a more general assessment of
the interactions between bacteria and a
variety of  fungi,  including beneficial
fungi  of  economic importance.  The
initial  series of  fungal inhibition assay
experiments have been conducted using
agar plates  and  sterile soil. In the  latter
case,  bacteria-fungi antagonism in the
sterile  soil  was assessed by replica
plating onto agar plates.

METHODS

Bacterial Cultures and Media

    Cultures of P. putida PPO301, with
and without plasmids, P. fluorescens
Pf-5  and   VT2KTR21),  and   P.
                                    162

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aeruginosa PA01C, with and without
plasmids,   were used in this  study.
Luria-Bertani  (LB)  agar was used  for
plate  counts.  Liquid  cultures  were
grown in  Brain Heart Infusion  (BHI)
broth.  Broth  cultures were propagated
on  a  rotary  shaker  at  200  rpm.
Appropriate antibiotics   were  added
when selective media were needed.
Unless otherwise stated, cultures were
incubated  at 30°C.

Fungal Cultures and Media

   Thirty   nine   fungal   isolates
representing  free-living  soil  forms,
mycorrhizae,  microbial   pest  control
agents, and biodegraders of xenobiotics
were used in this study.  Except for the
ectomycorrhizae where  MMN  slants
were  used, all  fungal cultures  were
maintained on   potato-dextrose agar
slants at 4°C.   Fungal liquid cultures
were grown in  MMN broth on a rotary
shaker (200 rpm) at 25-30°C.

Fungal Inhibition Assays

   In vitro bacteria-fungus plate assays
were   performed   on   agar   plates
consisting  of   media   of  different
compositions   and  included  potato
dextrose  agar   (PDA)   (Difco
Laboratories,  Detroit,  Mich.),   MMN,
medium 523  and  Kanner's medium.
Fluorescent  pseudomonads   were
patched along the perimeter of the agar
plates  and   incubated  overnight  at
30°C. Ten fj\  of fungal suspension or a
plug  from  the leading edge  of a 5 day
old  fungal culture, grown  on  MMN
broth or PDA, was  placed at the center
 of  the  agar plate previously  patched
 with the test bacteria. The plates were
 then incubated at 25-30°C for 5 to 14
 days.  For  mycorrhizal fungi,  longer
 incubation  time  (30-40  days)  was
 necessary before scoring for inhibition
 could be  made. The degree of fungal
 inhibition by fluorescent pseudomonads
 was expressed relative  to  a control
 (fungus   grown  in  the  absence  of
 fluorescent   pseudomonads)   by
 measuring  the diameter   of  fungal
 growth over time.

 Assay of Fungal Inhibition in  Sterile Soil

    Sixty five grams of Willamette River
 bottom soil  was placed in a  glass petri
 dish and autoclaved three  times  (by
 tyndallization) each for 2 hours at 121
 C and  15  psi. The sterile soil was
 inoculated with 20 ml of a washed cell
 suspension  ( 24-48 old culture) such
 that the final bacterial density was  108
 CFU/gram of sterile soil. Controls were
 not inoculated with the test bacteria. A
 free-living fungal (S4) inoculum was
 prepared by culturing in MMN broth and
 incubating for 5 days at 30°C and 200
 rpm on   a  rotary  shaker.  After  the
.bacteria were inoculated into sterile soil
 in petri dishes and were incubated for
 60-75 hours at 30°C, an aliquot of 0.1
 ml of 8.4x103 CFU/ml of the fungus S4
 was centrally inoculated per  petri dish.
 Each treatment was  replicated at least
 in triplicate. Fungal  spread  or growth
 was monitored by replica  plating onto
 Martins medium agar plates. Bacterial
 growth in the soil  was  assessed  by
 serial dilution  plating on selective  LB
 agar plates.
                                    163

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RESULTS AND DISCUSSION

    All 39 isolates of fungi representing
diverse origins  and  properties  were
evaluated for potential sensitivity  (in
plate assays) to eight strains of Gram
negative bacteria. A scoring system of
minus (-) to four plus (4 + )  range was
used to denote the extent of inhibition.
A complete inhibition was assigned a
four plus where as insensitivity was
denoted with a minus sign.
    A large variation was  noted in the
extent of fungal  inhibition induced  by
the  various  bacterial  strains  and
different media.  However, on medium
523,  virtually  all  bacterial  strains
exhibited  maximum inhibition  (4 + ) of
fungal growth, i.e.,  no  fungal  growth
occurred over the 7-14  days of incub-
ation.   The  antagonism  was  due  to
diffusible substances expressed in both
the  presence and absence of added
ferric chloride.
    Susceptibility of ectomycorrhizaeto
the bacteria was evaluated on medium
MMN. Great differences were noted in
the patterns of inhibition.  In general,
Pf-5  and  PA013 demonstrated  the
greatest levels of inhibitory effects.  P.
fluorescens VT21(TR21) was the least
inhibitory.  This diversity in the extent
of inhibitory activities suggests that the
different  bacterial  strains  produce
different   type(s)   of   inhibitory
substances.
    Antifungal activity was detected in
sterile  soil inoculated with  the  free-
living  soil  fungus  (S4) and  various
strains  of  P.  fluorescens and  P.
aeruginosa. Several significant features
were derived from these experiments.
P.  aeruginosa strongly inhibited  the
growth of the fungus  in sterile soil
 medium.  Growth inhibition was noted
 over  the  25 day  observation  period.
 Maximum  growth of the fungus oc-
 curred in the control soil (without the
 bacterium) and reached the perimeter of
 the plate within 8 days of incubation.
 PA013 (pR0103) exhibited significantly
 less  fungal  inhibition  implying  the
 plasmid   somehow   interfered   with
 production   of   the  inhibitory
 substance(s).   Numbers of  the Pf-5
 inoculum declined  10-fold by day 15.
 This  decline,  in  bacterial   numbers
 coincided with slightly greater spread of
 the S4 fungus.  On the other hand, S4
 growth in the presence of VT21  (TR21)
 was  only  slowed  and by  day .21
 covered  as much  of the spi'l.  as the
 uninoculated control following a 50-fold
 reduction of bacterial numbers in the
 soil.

 FUTURE WORK

    Studies are  underway to develop
 assays using nonsterile soil and plants
 to   evaluate   the   interaction  of
 pseudomonads  with  indigenous soil
 microflora, including fungi. Studies will
 be conducted to ascertain the type(s) of
 chemicals produced in the rhizosphere
 environment that alter the composition
 of the ecosystem. Fungus-bacterium
 antagonism  and   its  autecological
 significance will, be evaluated  using
 fungus and fluorescent pseudomonads
 both  marked  with stably  expressed
 antibiotic labels.

 PUBLICATIONS

    This study was initiated  this fiscal
 year and there  are no publications at
this time.
                                     164

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    EFFECTS OF ENVIRONMENTAL FACTORS ON BACTERIAL CONJUGATION
                  M. A. Gealt, T. A. Khalil and S. Selvaratnam
                               Drexel University
                  Department of Bioscience and Biotechnology
                            Philadelphia, PA 19104
INTRODUCTION
   The intentional release of bacteria to
foreign  habitats  is  common.    The
agricultural  use  of  bacteria,  e.g.,
Rhizobia to enhance nitrogen fixation,
has resulted in the introduction of high
concentrations   of   reactor   grown
bacteria  directly  into   the  natural
environment.   The  enhancement of
bioremediation capability of indigenous
bacte'ria by augmentation with bacteria
containing specific gene sequences has
been proposed.  Pathogens  contained
within  the  effluents  from sewage
treatment facilities  are  used for soil
emendation. There are many proposals
for, and have been some tests of, the
release of bacteria modified by genetic
engineering techniques.  In addition to
scheduled releases  of bacteria,  many
possibilities  exist for the unscheduled
release  resulting  from  breaches in
industrial reactor vessels or transport
from release sites by foraging animals.
   Many  bacteria  from scheduled or
unscheduled releases will end up in
either   agricultural   or   industrial
wastewater.   Wastewater  also is  a
likely place for  genetic interactions to
take place because it contains  a high
bacterial titer, and, during the treatment
process, the bacteria will be exposed to
different   environmental  conditions,
including  aerobic,   anaerobic,  etc.,
which  may enhance or curtail gene
transfer.  Conjugation may occur using
wastewater-resident  conjugative
plasmids;  it is also possible  for non-
conjugative plasmids to participate in
conjugal transfer by the  mobilization
process.
    Conjugation  requires the  presence
of specific genes, e.g., the tra genes of
conjugative  plasmids  and  the  mob
genes  of  mobilizable non-conjugative
plasmids.  In addition to the  presence
of these genes needed  for conjugation
or mobilization, bacteria must be able to
produce functioning  gene  products in
their environment.  In order to predict
the  possible escape of gene sequences
from released bacteria  to indigenous
populations  it is necessary to know
how  specific   components   of   an
environment affect the  transcriptional
(and translatio'nal) control mechanisms
for  the production of these  products.
We  report here the effect of different
factors on transcription of tra and mob
as might occur in wastewater.

METHODS

Bacteria and Plasmids

    E.  coli chi1784 that contains  the
plasmid R100-1  was used to determine
inhibition of conjugation by exogenous
compounds,  with   E.   coli  chi1997,
which  is  plasmidless,  serving as  the
recipient.  E.  co//HB101 was used as
host for plasmids F, R100-1, RP4, R6K,
N3, RA1,  R144, R27,  and colEI-amp.
                                     165

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 £. co// DH5alpha was used as host of
 plasmid pTK31.  This reporter plasmid
 contained traP promoter sequences of
 plasmid pFL114  fused to the facZ of
 plasmid pMC1403. This construct en-
 abled us to monitor the effect on the
 transcription through the traP promoter
 of  chemicals  coincubated  with  the
 bacteria.
    The  bacteria  were  grown  on
 primarily on L-broth supplemented with
 appropriate   antibiotics.      Some
 conjugation   experiments    were
 performed   in   sterile   synthetic
 wastewater.   Compounds  tested for
 their  effect on conjugation or  beta-
 galactosidase  activity were  added to
 actively  growing  cultures.     Beta-
 galactosidaseactivity was monitored by
 method of Miller (1972) in which cells
 were perme'abilized, then induced with
 o-nitrophenol-beta-D-galactoside
 (ONPG).  The developing yellow color
 was monitored at A420-

 Transcription of the mob Genes

    In  order  to  determine  if   the
 mobilization specific mob genes  were
 being  transcribed,  total  RNA   was
 extracted from the cells and probed by
 slot blot with oligonucleotides specific
 for  either the mob2 or the mob3  (and
 mob5) gene sequences. Comparisons
 were  made  of extracts from bacteria
 containing either a conjugative plasmid
 (F, R100-1, RP4, R6K, N3, R144,  RA1
 or R27), the colEI-amp plasmid,  or both
 of these plasmids.  Prior to binding to
the  membrane, extracts treated  with
either DNase or RNase to destroy one
nucleic acid or the other. The absence
of DNA or RNA was confirmed by gel
electrophoresis.
 RESULTS AND DISCUSSION

 Effect of  Environmental  Factors  on
 Conjugation

    The   chemical  composition  of
 wastewater can have significant impact
 on the probability  of gene transfer by
 conjugation.  Significant inhibition (>3
 logs)  was exhibited in the presence of
 millimblar  concentrations  of  Zn2 + ,
 Fe3 + , and SDS,  which are relatively
 common (especially of Fe3 + , which is
 often  added to enhance precipitation).
 It has been proposed that the effect of
 zinc is directly on  the receptor of the
 pilus  while SDS affects the structural
 integrity  of  the   pilus.    Significant
 inhibition, but at much  higher concen-
 trations,   was  exhibited  by  Ca2 + ,
 Mg2 + , Na+and K + .  No inhibition was
 observed with Triton X-100.
    To see if any of these inhibitors
 specifically affected the transcription of
 traP, the major regulatory gene of the
 tra operon of the derepressed IncFII
 plasmid R100-1, a  fusion was made of
 the promotor  of traP and the /acZ  beta-
 galactosidase reporter gene.   In this
 way the effect of external compounds
 on the  transcription  of  traP  was
 monitored.  Comparisons were made
 between  the  effect of compounds  on
lacZ.  genes  located on this  fusion
 plasmid with the  lacZ.  located other
 plasmids or on the  chromosome (using
ONPG as an  inducer).  All constructs
showed  similar inhibition  of  beta-
galactosidase  transcription    'as  a
function  of  temperature  and Zn2 +
concentration. Fe  +, however, showed
decreased induction  of  beta-galacto-
sidase from the traP fusion,   but  an
increase  in  the amount of  enzyme
                                    166

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induced  when facZ  was  under  its
natural promoter.  Therefore, at least
part of the decrease  in conjugation
frequency which was observed when
ferric ions were present was probably
due   to   the  inhibition   of  traP
transcription.

Induction of mob Genes

   Some of the dissemination of genes
in natural environments is due to the
mobilization  of   non-conjugative
plasmids.    It  is possible that some
environmental  elements will specifically
affect  mobilization  of  these  non-
conjugative plasmids,   but  not  the
functions on conjugative plasmids. We
used ColEI, a common non-conjugative
plasmid, to analyze these interactions.
Becausethisnaturally-occurringplasmid
had no easily selectable gene, we used
a modified ColEI that contains the beta-
lactamase  gene  of plasmid  pBR322.
This ColEI-amp plasmid is mobilizable
because it contains mob genes as well
as a bom  site, the origin of transfer
during transfer to  a recipient cell.  In
particular, ColEI has the mob2, 3 and 5
genes.   The  mob5 gene  is  wholly
contained  within  the  mob3  gene
sequences.  Under  conditions in which
ColEI is the sole  plasmid in a strain
grown in monoculture, there was only
trace synthesis of  RNA from the mob
sequences.    When   a  conjugative
plasmid, however,  was introduced into
this cell containing  the mob sequences,
transcription  initiated.      This
transcription depended  solely  on the
presence of the conjugative plasmid and
was not dependent on the presence of
a potential recipient cell. The induction
was  found to  occur with  several
plasmids from different incompatibility
groups  (F [IncF],  N3  [IncN],  RA1
[IncA/C], RP4 [lncP]I, R6K  [IncX], R27
[IncH],    R100-1   [IncFII],   R144
[Inclalpha]).  The  induction by such a
variety   of  conjugative   plasmids
suggests that there is  a common, and
perhaps  universal, product  of  the
conjugative plasmids which induces the
synthesis of the mob gene  products.

FUTURE WORK

Effect of Environmental Factors on mob
Induction

    In a manner similar to the effect of
Fe3+  on transcription  at  the traP
promoter, it  is possible that various
environmental components would have
an effect on induction of mob. We will
analyze   the  effect  of   various
compounds common to wastewater on
the mobilization of ColEI and on mob
transcription.    This  would  aid  in
predicting probability  of  conjugative
plasmids  mobilizing  non-conjugative
plasmids.

Isolation  and  Characterization of mob
Effector

    Although  eight  conjugative
plasmids have shown the capability of
inducing mob function, the universality
of this  induction  by plasmids from
different incompatibility groups has yet
to be  completely determined.  We will
analyze frequency of colEI mobilization
for conjugative plasmids studied and
correlate  this mobilization  with  the
inducton of mob gene  transcription.
    The  induction of  mob genes  by
plasmids   from   several   different
                                    167

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incompatibility  groups  suggest  that
there  may  be  a  common  effector
molecule. It would be of great interest,
therefore, to know  whether there is
similarity in sequence between all such
inducing  plasmids.     A   common
sequence for  such  a regulator gene
would be of interest to (a) quantitatively
and specifically monitor by hybridization
analysis  the  presence of  the total
population  of  conjugative  plasmids
without knowing which plasmids were
present and (b) assess the amount of
conjugative activity which is going on in
wastewater  and   other   natural
environments by the amount of active
product (RNA or protein) which cells
are synthesizing in situ.

Evaluation   of   Metabolic  State
Necessary for Conjugation

   The synthesis of proteins necessary
for conjugation may be a function  of
the metabolic  state of the cell.  It'is
unknown  whether   all  conjugation
occurs in the initial period following
release.   It may  be  possible that
following  24 hours  in wastewater no
more conjugation will occur.  In  this
case the risk of  release is  immediate,
but has a  short half life.  Alternatively
the  intention  of  releasing plasmid
containing bacteria with the  possibility
of the gene transfer into  the  native
population would be limited.
PUBLICATIONS

Gealt, M. A.   1991.  Gene transfer in
waste water,  in:  M. A. Levin,  R.  J.
Seidler and M. Rogul (eds.), Microbial
Ecology:  Principles,  Methods   and
Applications.   McGraw-Hill:  NY  (in
press).

Gealt, M. A. 1988. Recombinant DNA
plasmid  transmission  to  indigenous
organisms  during   waste  treatment.
Water Sci. Tech. 20: 179-184.

Khalil, J. and M.   A.  Gealt.   1987.
Effect of exogenous compounds on the
mobilization of plasmids in synthetic
wastewater.   Can. J.  Microbiol. 33:
733-737.

Mancini, P., S. Fertels, D. R. Nave, and
M. A. Gealt.   1987.  Mobilization of
pHSV106 from Escherichia coli in a
laboratory  waste  treatment facility.
Appl. Environ. Microbiol. 53: 665- 671.
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              TEST PROCEDURES FOR ASSESSING HAZARDS OF
          MICROBIAL PEST CONTROL AGENTS TO FRESHWATER FISH

                             Virginia M. Snarski
                     U.S. Environmental Protection Agency
                      Environmental Research Laboratory
                             Duluth, Minnesota
INTRODUCTION

   Widespread  concern   over   the
environmental  safety   of  chemical
pesticides has stimulated interest in the
development and use of microbial pest
control  agents (MPCAs).   Selected
because  of  their  host  . specificity,
MPCAs (bacteria, fungi,  viruses,  and
protozoa)  are generally  regarded  as
safer  alternatives  to   some environ-
mentally-hazardouschemicalpesticides.
However, as  living organisms, MPCAs
pose concerns not shared by chemicals,
ie., their survival, multiplication,  and
dissemination in the environment and
the  potential  to  cause infection  or
disease in nontarget species.
   As mandated  under  the  Federal
Insecticide, Fungicide, and Rodenticide
Act  (FIFRA),  EPA requires testing as
described  in   Subdivision  M  of the
Pesticide  Assessment  Guidelines  to
address potential ecological and human
health effects prior to the registration of
a MPCA.  These  requirements include
determining  the safety of MPCAs to
nontarget aquatic  species, including
fish.
   While test protocols are available for
assessing effects of microbial agents on
fish, basic information is needed on the
factors influencing the extent and kinds
of interactions between microbes and
fish to better understand and predict
potential effects  and  environmental
exposure of nontarget fish populations,
The studies presented here investigated
the interactions between  fish and the
registered MPCA, Bacillus thuringiensis
subsp. israelensis (Bti) under laboratory
conditions.

METHODS

    Single  species  exposures  were
conducted  with two  commercial Bti
spore-crystal formulations, Vectobac-G
(Abbott  Laboratories)  and  Mosquito
Attack (Reuter Laboratories).  Exposure
concentrations used ranged from 104 to
106  CFU/ml,  based on spore counts,
and  were at and above recommended
field application rates.
    Four species of  freshwater  fish
were  used:      fathead  minnows,
Pimephales promelas,  bluegill sunfish,
Lepomis   macrochirus,   and  two
salmon ids,   rainbow   trout,
Oncorhynchusmykiss, and brook trout,
Salvelinus fontinalis.
    Bti  formulation was added to the
water and  fish were exposed under
static or static renewal conditions for
periods  of from one hour to 30 days.
Following exposure fish were removed,
rinsed in clean water, and sampled to
determine the extent of Bti accumu-
lation or transferred to clean water to
study clearance of Bti.
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    Fish samples,  either whole  body
 (WB)   or   dissected   tissues,   were
 homogenized,   generally  pasteurized
 (65°C  for  20 min.) to reduce back-
 ground vegetative cells, and plated into
 tryptic  soy agar  (TSA).   Typical .Bti
 colonies were counted  following a 24
 hour incubation at 32°C.
    To  determine if  germination  and
 colonization occurred within the fish,
 homogenates  were  subdivided  and
 plated   with   and   without   prior
 pasteurization   onto   a   selective,
 differential media, PEMBAC (developed
 by Dr.  Allan Yousten, VA Polytechnic
 Institute and State Univ., Blacksburg,
 VA).    Comparison of total Bti (non-
 pasteurized samples)  and  Bti  spore
 (pasteurized)  counts  was   used  to
 determine if germination occurred.

 RESULTS AND DISCUSSION

    Concentration-dependent dissolved
 oxygen  depletion with  both  formu-
 lations,  believed  to   result  from
 stimulation  of   indigenous  aquatic
 bacteria by nutrients  in the  formu-
 lations, resulted in mortality of all fish
 within 24  hours at and above  water
 concentrations  of  2-3x106  CFU/ml;
 partial mortality of trout occurred at
 5x105  CFU/ml.    No  direct  adverse
 effects of Bti on fish were observed.
    Rapid accumulation of Bti spores by
 all fish tested was measured following
 addition of either formulation  to their
 water.   No differences  in  responses
 were  detected to the two Bti formu-
 lations.  Plate counts on dissected guts
 were nearly identical to  WB  counts in
 all species showing that ingestion was
the major exposure route.  Skin swabs
and   gills   had   counts  orders  of
 magnitude lower than gut samples. For
 example, fathead minnows whole body
 and gut samples contained 105 to 10
 CPU/fish while gill samples were 102 to
 103 CFU/gill.
     Differences in relative accumulation
 of Bti spores by species was related to
 feeding  habits and  food size prefer-
 ences.     The  omnivorous  fathead
 minnows actively ingested the small
 formulation particles resulting in  WB
 counts that were two to four orders of
 magnitude higher  than counts in the
 bluegill  and trout.
     Upon transfer to clean water, rapid
 elimination  of  Bti  spores  occurred
 through  fecal elimination.   Maximum
 loss occurred within the first 24 hours
 after transfer; near complete elimination
 occurred in 4 to 10 days if reingestion
 did not occur.
    A decrease in  WB spore  counts
 with increasing exposure time  and the
 presence of spores in feces for over a
 week after spore  counts  on tissue
 homogenates  were  negative,  led to
 studies   to  investigate   germination
 within the fish.  Comparisons  of total
 Bti  counts  and  spore  counts  of
 subsamples of  tissue  homogenates
 using PEMBAC agar demonstrated that
 germination of spores occurred within
 the  fish  gut.    Attempts to  identify
 vegetative cells  within these samples
 were not successful.  It  may  be  that
 even  though spores germinated  (as
 shown by loss of heat-resistance), they
 did not continue  through outgrowth to
 form vegetative cells but formed spores
 in  feces via the process of microcycle
sporulation.
    No evidence of increases  in total
counts with time were shown suggest-
ing that colonization of the fish guts did
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not occur.  Results of these studies
showing  rapid accumulation of spores
directly from the water and short-term
retention of Bti suggests that fish may
have   signficant   infuence  of  the
persistance and  dissemination of Bti,
and possibly other microbes, within an
aquatic ecoysystem.

FUTURE WORK

   We  are  planning   additional
laboratory   studies   on   fish-MPCA
interactions,   including  work  with
fungus (possibly Lagenidiumgiganteum)
and other bacterial species,  including
non-sporeformers (possibly with several
species of Pseudomonas).   Plans are
underway   to   evaluate   additional
endpoints   ( h istolog ical   and
physiological/immunological)  for  our
effects testing procedures.   Studies
comparing laboratory-derived data with
that from small scale field studies will
also be conducted in conjunction with
other  members   of   ERL-Duluth's
research  program.
   In addition, studies will be begun to
develop   procedures   to   study  the
influence of stressors of environmental
and   physiological   origin   on  the
susceptibility  of  fish  to   microbial
exposure. As part of these procedures,
positive   controls   for   infectivity/
pathogenicity  endpoints    will   be
developed  by including exposures to
known fish pathogens.
PUBLICATION^

Snarski, V.  M.   1990.   Interactions
between Bacillus thuringiensis subsp.
israelensis   and  fathead  minnows,
Pimephalespromelas Rafinesque, under
laboratory conditions.  Appl.  Environ.
Microbiol., 56: 2618-2622.
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             INTERACTIONS OF NON-TARGET INVERTEBRATES WITH
             Bacillus thuringiensis var. israelensis IN NATURAL PONDS

 Richard L. Anderson1, Eric Mead2, Lyle J. Shannon3, and David M. Janssen3,
                     U.S. Environmental Protection Agency1
                        American Scientific International2
                    Environmental Research Laboratory-Duluth,
                               Duluth, Minnesota
                              Biology Department3
                         University of Minnesota-Duluth
                               Duluth, Minnesota
 INTRODUCTION

  .  A  goal  of the  MPCA  research
 program at  ERL-D is  to  improve the
 confidence  of  predicting effects in
 natural  systems  using   laboratory
 derived data. Our research has three
 projects. Each of these is represented in
 this section. One project is to develop
 and  evaluate  single  species   test
 systems. Research has been conducted
 with fish and invertebrates exposed to
 either  fungi or bacteria  spore-based
 microbial pest control agents (MPCAs).
 The second  project is  to  develop  and
 evaluate  multiple species/microcosm
 tests. The third project is to conduct
 fate and  effect  studies  in  natural
 systems treated with  the  MPCA. The
 natural system study is the topic of this
 presentation.
    Few   studies  exist  which   are
 specifically   designed   to   evaluate
 non-lethal  interactions  of non-target
 invertebrates with MPCAs in natural
 aquatic systems. Most studies  were
 designed to  measure  only effects on
 either target or  non-target  animals.  We
 propose to develop general information
on  how  an  added   microorganism
interacts with  both the  target  and
non-target  animals. The objective of
 this  project  was  to  determine,  in
 temporary  pools  treated  to control
 mosquitoes, whether or not non-target
 animals accumulated and retained the
 spores of Bti.                •

 METHODS                    ,  ,.

 Site and Application

  •  The study was  conducted in three
 pools located  in a  wooded area  near
 Duluth, MN. Two pools were located in
 an  open grassy area, while the third
 pool was more enclosed by trees. The
 area contains  many small depressions
 (2-10 m in diameter) that fill with snow
 melt and rain in the spring and usually
 remain wet for up to 2 months. During
 the summer the pools were occasionally
 filled with run-off from rain. All pools
 contained  emergent cattails and  all
 were heavy mosquito producers.
    Three  pools  were  divided  with
 Scrimweave,   a   rip-stop  -woven
 polyolefin material. Wooden walkways
 were constructed to provide access to
 inner  pool  areas to enable sampling
 without disturbing the pool sediments.
    One side of each pond was treated
 with Vectobac, a formulation consisting
of  corncob particles  covered  with
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Bacillus thuringiensis  var  israelensis
(Bti). The amount added to each pond
was specifically calculated for each site
with a target of 104 Bti spores per ml
of  pond water.  The  formulation  is
designed   to   disperse   the   agent
throughout  the  water. The particles
float for a short time before sinking.

Sampling

    Samples of water and invertebrates
were  collected  from both sections  of
each pool. The  aquatic animal samples
were  collected by sweeping the water
with a fine  mesh net and transferring
the animals to a sterile glass jar to be
transferred  to  the   laboratory  for
analysis.   Emerging   insects   were
captured   and   transferred  to   the
laboratory in sterile  glass jars.
    At  the  laboratory,  invertebrates
from the aquatic samples were removed
from the jar and separate species were
placed in sterile glass  test-tubes. The
animals were washed  to remove any
loosely held Bti by  removing the pool
water with  a pipet, then rinsed with
sterile water. Each  sample was rinsed
twice,  after  the  water  had  been
removed.  A sample may consist of a
single large individual, or  for  smaller
animals such as daphnids, a pool of up
to  10 animals.  After rinsing twice, all
samples were frozen in 5 ml of sterile
water.  In all cases, the values were
adjusted to reflect  the number of Bti
spores (as Colony Forming Units, CPU)
per individual.
    Each sample to be processed was
thawed and poured  into a sterile plastic
bag. The sample tube  was rinsed with
5 ml of sterile water which was added
to the bag and the sample was blended
in  a Stomacher™. The  sample was
removed  from the bag with  a  sterile
pipet,  heat  shocked,   cooled  and
pour-plated at dilutions of  10~2  and
10~4 with Tryptic Soy Agar.
    The Bti content of emerged insects
was  measured  after  returning  the
insects to the laboratory. Individuals are
placed .in  a Stomacher  bag,  frozen,
thawed and blended in  sterile water
with  the   Stomacher.  The  blended
sample is heat-shocked and pour plated
on Tryptic Soy Agar.

RESULTS AND DISCUSSION

    The concentration of Btim samples
of water, and in insects,  zooplankton,
snails  and  emergent  insects  were
measured. Six taxa of insects, 3 taxa of
zooplankton and 1 taxon of snails were
collected with sufficient  frequency to
provide data throughout  the  summer.
Four  hundred and forty-nine samples
were processed.  Of that total,  104
samples were adult insects. Accumu-
lation differed for each taxa and the
extent of the accumulation might have
been affected by pool characteristics.

Insects

    Uptake of Bti is rapid  in insects.
Insects  sampled   soon   after  the
application   showed  the   highest
concentrations. Within several days,
many of the insect taxa had lost most
of  their  accumulated  ,Bti.  However,
there  were   some   samples   that
measured  uncharacteristically  higher
than samples taken before or after that
specific sample. The insects  collected
from  the  pools  were  predaceous,
therefore   ingestion   of  prey  that
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 contained Bti  might account for the
 higher values.

 Zooplankton

    Zooplankton   also  rapidly   ac-
 cumulated  Bti.  The  highest  concen-
 tration in  any  of the animal  samples
 was found in Lynceus, (Conchostraca:
 clamshrimp) which rapidly accumulated
 up  to  30,000   CPU/  animal.  The
 daphnids in  pool 1  contained only a
 small amount of Bti when the four-hour
 sample was taken but by 24 hours the
 average count was about 2000 spores
 per animal. The  amount of Bti in the
 daphnids decreased  through the  next
 two sampling periods until at day 7 the
 content  was about  300 spores  per
 animal. At 14 days post-treatment the
 count had  decreased to about  100
 spores per animal. The pool dried up
 before the  next  scheduled sampling
 date.
    Pool 3 was  deeper and  larger than
 pool  1 and  samples could be taken
 throughout the season. The rate and
 extent of uptake by  daphnids in this
 pool  was  much  different  than   was
 measured in pool  1. The accumulation
 did not show a  peak but remained, flat
 throughout the  monitoring period. The
 content never did exceed 300 -  400
 spores per animal in this pool.
    The differences in uptake between
 daphnids in each  pool may be due to
 pool structure. Pool 1  was shallow, less
 than 1 m in depth, while pool 3 was
 deeper and contained about  3-times as
 much   water  volume. In  both pools,
 although the  Bti was measured in the
 water it ultimately accumulated on the
surface  of   the   sediment.  In   the
shallower pool the animals would have
 been exposed both in the water and by
 contact with  the sediment.  In  the
 deeper pool,  those  daphnids  in  the
 water column would have less contact
 with   the   higher  concentrations
 associated  with  the  sediment.  The
 consistency in values after 7 days may
 indicate  a   minimum   value   for
 zooplankton which accumulated Bti.
     Lynceus and other zooplankton are
 detritus or filter feeders. These forms
 would easily collect Bti either from the
 water or from feeding on the surface of
 the sediment. Within 6 to 8 days most
 zooplankton had lost over 80% of the
 accumulated Bti. A low concentration
 of Bti was found in these animals the
 remainder of the season.
    The last  of  the  aquatic  forms
 analyzed in this study were Planoribidae
 snails. The concentration of Bti found in
 these animals  did  not increase  rapidly
 soon after the application and did not
 decrease as was demonstrated in other
 animals. This may be related to feeding
 practices  or  contact with  sediment
 surfaces. If feeding was on or  near a
 cob  particle, high Bti concentrations
 would be expected.

 Adult Insect

    Adult  insects were also collected
 and analyzed. Early in the season, the
 samples were largely chironomids  and
 mosquitoes.  Later in the season, most
 adults were odonates, either dragonflies
 or damselflies. A few caddisflies were
 also  collected and analyzed. Only one
 chironomid sample contained  Bti and,
 as  was  expected,  none   of  the
 mosquitoes contained Bti.   Bti was
found in a few damselfly adults but the
greatest concentrations were found in
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dragonflies.  Dragonfly content ranged
from less than 100 to more than 1000
CPU per individual. Most of the samples
contained between 100 and 500 CPU
per individual.  The  highest  concen-
trations  were   found   in  dragonfly
exuviae which  were up  to  10 times
higher than in the emergent adult.
   The adult insect  collections show
that the Bti spore could move from the
site of application either  in or on the
bodies of some emerging insects. The
data also show that the amount carried
by each  insect is highly  variable and
that not all emerging insects contain the
spore.  Odonates  may  contain  Bti
because  of their predatory  feeding
habits.   Direct ingestion  of particles
may also result in Bti accumulation. A
study this year will begin to evaluate
some  of these issues.

FUTURE

   An  integral  part  of the  risk
assessment  of  microbial  pest  control
agents  must   be to  evaluate  the
predictive value of laboratory data. This
data begins to  describe how inverte-
brates can  affect distribution of an
MPCA in natural systems. This year's
uptake and  distribution research will
include  another  application  to  the
ponds. Special emphasis will be placed
on the movement of  the  spore out of
the  system  by  emerging  insects.
Long-term plans are to use the divided
pond  method to evaluate other MPCAs
and to understand how the invertebrate
and vertebrate  populations affect the
MPCA survival and distribution.
PUBLICATIONS

Nestrud,   Lori  B.  and   Richard  L.
Anderson.   1990.   Protocols  for
Exposing   .Freshwater   Fish  and
Invertebrates to Fungi used  as Pest
Control   Agents.  Environmental
Research  Laboratory-Duluth,   Duluth,
MM.  (Report  describes techniques for
exposing and measuring effects on  8
species of  invertebrates and one fish
species)
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           FIELD VALIDATION OF LABORATORY MICROCOSMS USING
                      Bacillus thuringiensis var. israelensis

               L.J. Shannon1, D.M. Janssen1, and R.L. Anderson2
                             Biology Department1
                         University of Minnesota-Duluth
                     U.S. Environmental Protection Agency2
                    Environmental Research Laboratory-Duluth
                               Duluth, Minnesota
 INTRODUCTION

    The   potential   for  negative
 environmental effects resulting from the
 release  of   genetically   engineered
 organisms   (GEMs)  is a  source  of
 concern  for  both regulators and  the
 public.  Unfortunately,  it is not  yet
 possible  to  predict  the  survival  or
 ecological  effects  of an  introduced
 microorganism   released   into   the
 environment. Single species laboratory
 exposures can measure direct toxicity
 or  pathogenicity  of  a  new  micro-
 organism toward a given species,  but
 they provide little information on  the
 critical  questions  of  survival  and
 ecological effects. More complex tests
 are needed  to predict effects  at  the
 community and ecosystem level.
    Microcosms,   contained   model
 ecosystems, should be important tools
 for  answering   these   ecological
 concerns.  A variety of terrestrial and
 aquatic microcosms have been used in
 chemical testing  but  their  use  for
 evaluating GEMs has been limited. This
 reflects  a   shortage   of  validated
 protocols.  Although many microcosm
 procedures  have  been proposed  by
various   investigators,    lack  of
information  about sensitivity,  repeat-
ability, and similarity to the "real world"
has precluded  their widespread use.
 Indeed,  none  of  these  procedures
 should  be used until we can demon-
 strate that (1) they are sensitive enough
 to  detect  effects,  (2) they  give  the
 same answer twice, and (3) they ac-
 curately  mirror  events  in  natural
 systems.
    We have  developed two aquatic
 microcosm  protocols   of  differing
 complexity. Both have performed well
 in lab tests with Bacillus thuringiensis
 var. israelensis (Bti).  We are now in the
 process of duplicating lab tests in  the
 field.  Preliminary analyses of field data
 are encouraging.  Although  the two
 microcosms showed differences in their
 ability to  model the  natural  ponds
 (calibration), both  correctly predicted
 that Bti would  survive  for  months,
 would kill target organisms (mosquito
 larvae) but would cause little ecological
 impact.

 METHODS

 Microcosms

    MFC  microcosms:   Our  original
 microcosms were   modifications  of
systems   originally   developed   for
chemical testing by John  Leffler.  After
extensive testing of these "mixed flask
culture"   (MFC)   microcosms    we
developed  a  protocol  for  evaluating
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 survival   and  ecological  effects  of
 microorganisms  added  to  aquatic
 communities.  These microcosms are
 small   (1-Liter),  easily   maintained
 systems that can support a variety of
 bacteria,  protozoa,  algae, zooplankton
 and  insect  species.    The  biotic
 community  in  these  systems  is  a
 species assemblage collected from the
 wild  and allowed to co-adapt in the
 laboratory.
-    Core microcosms:  While the MFC
 protocol performed adequately in tests
 with  Bti,  it was not a close surrogate
 model of a natural pond. Among other
 differences it lacks a complex organic
 sediment,  is  highly  eutrophic  and
 wholly aerobic.
    As an alternative,  we constructed
 new  microcosms from intact sediment
 cores. Cores were collected from a dry
 temporary pond known to contain large
 populations  of  zooplankton,  insects,
-and  molluscs.    We anticipated that
 resting  stages  of  most  of  these
 organisms would be present in the dry
 cores. Cores 75mm in diameter and 40
;mm deep were removed with a PVC
 corer and transferred to wide-mouthed
 glass  canning  jars (Ball Corp.).   The
 rehydrated   jars  were  held  in   a
 Percivaltm Growth chamber at 20°C on
 a 12/12 hour - light/dark cycle.

 Field Sites

    Field  tests  were conducted  at a
 remote location owned by the Duluth,
 MN  International -  Airport.     Three
 temporary ponds  (vernal pools)  were
 used in  the  field tests.  These three
 were  selected  on  the  basis  of their
 similar   water  chemistries   and
 populationsof zooplankton, protozoans,
snails,   and   insects   (including
mdsquitos).    Each  pond  was sub-
divided   with   a   1-M   high   barrier
                             "T"IV A
constructed   of   Scrimweave  ,   a
rip-stop  woven  polyplefin  material.
Wooden  walkways were constructed
over each pond to provide better access
and permit sampling without disturbing
the sediments of the pond. One side of
each divided pond was  treated with
Vectobactm (corn cob grits treated with
Bti) to yield a water concentration of
approximately  104 Bti spores  per ml.
This material was broadcast across the
pond with a  spreader constructed from
a one-gallon plastic bottle.

Test Endpoints       . '

   The same test end points were used
in both lab and field tests.

Fate of Bti

   Survival of Bti was monitored daily
for the first two  weeks after appli-
cation,  and weekly thereafter.   Bti
density was determined  in both water
and sediment by  heat-shocking 65°C
and plating  appropriate  dilutions  on
Tryptic  Soy Agar  (TSA).   Both total
colonies  and typical Bti colonies were
counted. Plates that had suspicious Bti
colonies  were  bioassayed  after  the
plates were exposed at 25°C for three
days to allow germination of spores.
   In addition to water and sediment
samples,  animals  were  periodically
collected and analyzed for Bti content.
For large organisms (e.g.  tadpoles,
insects, snails) only one individual was
used per sample.  Smaller organisms
(e.g.,  zooplankters) were analyzed in
groups  of  five, so  that Bti concen-
                                     177

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 trations   would   be   sufficient  for
 detection. These groups were washed
 by suspending them in  distilled water,
 shaking vigorously and filtering through
 a sterile membrane filter. Tadpoles and
 snails were then homogenized in 5 ml
 of  sterile  water using  a  Tissumizer
 (Tekmar Corp., Cincinnati, OH).  Other
 organisms were ground in 5ml of water
 using  a  Ten  Broek  manual  tissue
 grinder.  All ground samples were then
 pasteurized for 20 minutes at 65 °C.
 Cooled samples were then  diluted, on
 TSA agar and  incubated overnight at
 30°C.
    The  duration  of Bti toxicity to
 mosquito larvae was determined with
 daily  bioassays  on  water  samples
 collected from microcosms and ponds.
 Bioassays were done in the lab in a
 series of small glass vials containing 5
 ml  sample  and 5  Aedes  atropalpus
 larvae. The bioassays were read  at 18
 hours and held for another 24 hours to
 observe  any delayed killing activity.
 Bioassays  for  each pond were  run a
 week prior to treatment  and  daily until
 the samples were no longer toxic.

 Ecological Effects

    A suite of physical, chemical and
 biological  measurements were  made
 both to characterize the systems and to
 detect effects. These are grouped into
 functional (related to processes within
 an   ecosystem)   and  structural
 (populations) variables.    Functional
 variables   included   the   following
 measurements:

Carbon Cycling
   Dissolved Organic Carbon (DOC)
  Total Organic Carbon  (TOC)
 Nitrogen Cycling
    Ammonia (NH3)
    Nitrate + Nitrite
    Total Nitrogen

 Phosphorus Cycling
    Ortho-Phosphate
    Total Phosphate

 Primary Production
    Diel Oxygen Gain

 Community Respiration
    Diel Oxygen Loss

    Structural   variables   included
 measurements   of    populations  of
 bacteria,  protozoa,   rotifers,   algae,
 cladocerans, copepods, ostracods, and
 macroinvertebrates.  Temperature and
 pH were also measured routinely.

 RESULTS AND  DISCUSSION

 Calibration

    MFC vs Core Microcosms: The core
 microcosms  developed   into   fully
 functioning,  .biologically   diverse
 systems.  They showed several differ-
 ences from  typical MFC  microcosms.
 For comparative purposes we contrast-
 ed events in the developing cores to
 events in  a typical  MFC microcosm
 experiment.  The MFC nutrient data are
 from experiments run at ERL-D by Frank
 Stay.
    Communities  were  slower  to
develop for cores than in MFC  micro-
cosms, probably due to the lower initial
nutrient levels. The MFC protocol used
a nutrient  medium to  "jump-start" the
systems, while  in the cores, nutrients
were initially tied up in organic matter
                                    178

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and  gradually  released  by  decom-
position   processes.  Nitrates  were
reduced to near zero in MFC systems
within  four weeks, but phosphates
remained high. Primary production was
initially very high in MFC systems and
decreased  over  time,  while  primary
production in the cores continued  to
increase over time. In response to the
initial algal bloom  in MFC systems a
largezooplanktoncommunity developed
but later  crashed.   The  zooplankton
community  developed  much  more
slowly  in  core  microcosms.     As
organisms  died  off  there  was   an
increase in ammonia in MFC systems
that did not occur in the cores. Finally,
the pH of the two systems was quite
different, ranging between 8 and 9 in
MFC systems and between 5 and 6 in
core systems.
   Laboratory vs.  Field:  Temporary
pools are biologically and  physically
more variable habitats than  laboratory
microcosms.   Species  composition,
seasonal  changes  and recurring dry
periods are some obvious differences.
The  water levels in these  pools  are
often low and are sensitive to daily and
diel temperature changes. At all sites,
daily  low  water  temperatures were
within a  few degrees of 10°C while
high temperatures ranged from 15°C to
32°C depending on the air temperature.
This   is  in  contrast  to   laboratory
microcosms  where  water  level  is
carefully   controlled  and   water
temperature is maintained at 20°C.
   Despite these fluctuations, pH in the
pools showed little day-to-day change.
 Since the diel change was so small we
measured  only morning  pH in 1990,
and found a mean value of 6.4. This
compares with mean pH values of 8.75
and  9.6 for  morning and  afternoon,
respectively, in MFC microcosms. Core
microcosms behave much more like the
ponds  and  showed  little  difference
between morning and evening.
    Core microcosms were much closer
surrogates of natural ponds than MFC
systems.    Nutrient  levels,  primary
production, and community respiration,
and the diversity of invertebrate biota in
core systems were close to those seen
in   natural   ponds.    Although  the
temporary pond fauna  were far more
diverse  than  those of the microcosm
communities,  the  major  taxa were
similar   in  both.    The  zoopiankton
community of the microcosms and field
sites contained cladocerans, cyclopoid
and   harpacticoid  copepods,  and
ostracods. Both microcosms and field
sites also supported several species of
coleopterans, chironomids, and aquatic
oligochates, as well as snails (Lymnaea
sp.)  and clams (Sphaeriidae}.   Some
predaceous insect taxa (e.g.,  Dytiscid
beetle larvae and  Chaoborus larvae)
were  deliberately  removed  from
microcosms   to   prevent  excessive
predation   on   the   zooplankton
community.   Other insect taxa (e.g.,
dragon  fly  nymphs),  Eubranchipoda
(conchostracans) and  tadpoles (Hyla
crucifer) were present in ponds but not
in microcosms.

Validation

    Bti Survival: Btispores survived for
extended periods   in both  laboratory
microcosms and field sites. While most
spores   disappeared from the  water
column within a few days, levels in the
sediments remained stable for most of
each test.
                                    179

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    Although spores persisted, toxicity
 rapidly disappeared in both  lab  and
 field.  In the field,  sediment samples
 lost toxicity to mosquito larvae between
 9  and  12  days  after   application.
 Similarly  samples  from  both micro-
 cosms lost toxicity between 7 and 12
 days after application.  This loss of
 larvacidal  activity  is  due  to  the
 inactivation or microbial degradation of
 the   parasporal   protein  crystal
 associated with Bti spores.  Despite the
 loss   of  toxicity,   spores  from
 microcosms  and  field  sites  could
 produce  new  toxic  crystals  when
 cultured on TSA agar.
    The  patterns  of  Bti  uptake  by
 invertebrates were similar in lab  and
 field.  The amount of Bti accumulated
 varied among the organisms tested.
 Filter  feeding  planktonic  organisms
 accumulated   the smallest  amounts
 while   the  bottom-feeding  snails
 accumulated very large amounts.  While
 there were some differences in rates of
 uptake  and losses  between  lab  and
 field, the most important finding is that
 we could confirm in  both lab and field
 that the organisms had been exposed to
 the test agent.
    Ecological  Effects:  The effects on
 the target organism (mosquitoes) were
 obvious in both lab and field tests.  In
 each  case they were killed within 24
 hours after application.  The effects on
 non-target  organisms  and  functional
 variables were not so obvious. In fact,
 our first task in  comparing effects on
 microcosm and field communities  has
 been to develop appropriate statistical
 procedures for analysis of field data.
    We   detected  no   significant
ecological effects in either lab or field.
We did  become  more  aware of  the
 difficulty  of detecting  effects .in the
 field.  In  the  laboratory  we  are  can
 control environmental conditions  and
 manipulate   microcosms   to   reduce
 variability. Since this is not possible in
 the  field,  replicate  field  sites  show
 much  greater variability than  replicate
 microcosms. This makes it much more
 difficult to detect effects in the field.
 We are currently investigating several
 procedures for analyzing such  data.
    In an attempt to deal with this high
 variability we investigated the use of an
 analysis of covariance (ANCOVA).  In
 this  procedure  we   adjusted  experi-
 mental means for the influence of the
 covariate factors. In this case,  depth of
 the pond and  water  temperature were
 the most significant  covariates.   We
 conducted ANCOVAs for  each day of
 the exposure,  and found no consistent
 effects that could be attributed to Bti
 treatment.
    We have  not yet  completed  all
 summation  and  analysis  of   data
 collected  during  the   1990  pond
 exposures.  All  but one set of ponds
 dried up by mid-July.  With heavy rains
 in August and September all the ponds
 re-filled, however, and we  were able to
 collect additional samples in October.
 We are still analyzing these and a few
 earlier  samples   so  our  conclusions
 remain incomplete at  this time.  At this
 point  we  do  not see any significant
 effects caused by Bti in the field.

 SUMMARY

    Microcosm  tests  provide  fate,
direct effects and ecological data that
can be used as a surrogate for  outdoor
testing.   In  this  report we  have
discussed the  strength  of the  link
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between   microcosms   and   field
performance.  On the basis of current
data we can draw several conclusions:

1.  Both  the  MFC  and  the  core
microcosm   model   events   were
observed in  natural  systems. The core
microcosm is  more closely correlated
with the field than the MFC.

2. We were able to monitor the fate
and effects of Bti in both lab and field
and recorded similar results:
   a.  Bti  spores   persisted  for the
   duration of the test.
   b. Bti toxicity persisted for 7  to 12
   days.
   c. Non-target animals accumulated
   Bti. The extent of the accumulation
   and rate of loss of Bti were species-
   specific.
   d. Target animals were killed in both
   systems.
   e.  Non-target  animals  were not
   sensitive in either system.

   Tests in a single laboratory or field
ecosystem will not provide definitive
answers  to  the  questions of survival
and  ecological effects of a  particular
introduced microorganism.    Survival
and  effects  are  determined  by  a
complex  series of  abiotic (e.g.,   pH,
temperature, redox potential, structure
and composition of  the substrate) and
biotic factors  (e.g.,   the presence or
absence of competitors, predators, prey
or host species).  Clearly, tests in one
type  of  ecosystem  cannot provide
results generalizable to all ecosystems.
This  presents  a dilemma, since it  is
obviously not  possible to run all the
tests  needed  to evaluate  a  micro-
organism under  every combination of
abiotic and biotic factors.
    The core procedure may provide an
alternative to  running  multiple tests.
The system is flexible  enough to use
biota and sediment from any local site.
Survival and  effects questions could
then  be   focused  on  a  particular
ecosystem of concern, rather than on a
generalized  generic  aquatic  system.
The questions of field  calibration and
validation  will be further clarified as we
complete additional tests.

PUBLICATIONS

Shannon,   L.J.,   T.E.   Flum,   R.L.
Anderson,  and  J.D.  Yount,  1989.
Adaptation of  the mixed flask culture
microcosm for testing the survival and
effects of introduced microorganisms.
pp. 224 -  242. in:  U.M. Cowgill and
L.R.   Williams,   (eds.),   Aquatic
Toxicology and  Hazard  Assessment:
12th Volume. ASTM STP 1027.  Amer.
Soc.   for  Testing  and  Materials,
Philadelphia, PA.

Shannon,  L.J.  and R.L. Anderson. Use
of the  mixed flask culture microcosm
protocol to estimate the survival and
effects  of microorganisms added to
freshwater ecosystems,  in: M. Levin
(ed.),  Methods in  Microbial  Ecology.
Am. Soc. of Microbiology (in press)

Stay,  F.S., T.E.  Flum,  L.J. Shannon,
and J.D. Yount. 1989. An Assessment
of the precision and accuracy of SAM
and  MFC  microcosms  exposed  to
toxicants, in:  U.M. Cowgill and  L.R.
Williams,  (eds.),  Aquatic Toxicology
and Hazard Assessment: 12th Volume,
ASTM  STP 1027.   Amer.  Soc.  for
Testing and Materials, Philadelphia, PA.
                                     181

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Shannon,  L.J., T.E. Flum, and  J.D.
Yount.  1987a  Draft  protocol for  a
mixed flask culture aquatic microcosm
toxicity test. Output No. 7667A 36 pp.

Shannon,  L.J.,  M.C.  Harrass,  J.D.
Yount,  C.T.  Walbridge.  1986.    A
comparison of mixed flask culture and
standardized   laboratory   model
ecosystems  for  toxicity testing, pp
135-157.  in: John  Cairns, Jr.  (ed.),
Community  Toxicity Testing,  ASTM
STP 920. American Society for Testing
and Materials, Philadelphia, PA.
                                   182

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       CURRENT INVESTIGATIONS ON MICROSPORIDIAN TEST SYSTEMS
       W.S. Fisher, J.W. Fournie, C.L. McKenney, Jr. and D.P. Middaugh
                     U.S. Environmental Protection Agency
                       Environmental Research Laboratory
                              Gulf Breeze, Florida
INTRODUCTION

    Since  1982, Gulf Breeze Environ-
mental  Research Laboratory  (GB/ERL)
has supported a request by the Office
of  Pesticide  Programs  to   develop
testing  protocols for  fate and effects
testing of microbial pest control agents
(MPCAs). This has included description
of single and multi-species test systems
and toxicity and pathogenicity testing
protocols  for  freshwater and marine
nontarget organisms.
    It  was recognized that  MPCA test
systems must be capable of containing
the MPCA and providing reproducible
data,  must be reasonable in  cost and
size,  must  maintain  nontarget  test
organisms  from a variety  of  phyletic
groups, and should represent as closely
as possible the conditions of  a natural
system.  Additionally, it should have
the capacity for testing all  microbial
groups  of  MPCAs, including viruses,
bacteria, protozoa and  fungi.  Multi-
species  test    systems   have  the
advantage  of  testing several species
with one  exposure and  may demon-
strate interactions among nontargettest
species.
   Single  and  multi-  species  test
systems employed at GB/ERL quickly
evolved  into   the  present  totally-
enclosed aquarium that incorporates an
undergravel filter system using artificial
substratum and an external ultraviolet
light.    Several  different marine  and
fresh water nontarget organisms, includ-
ing  fishes,  grass  shrimp,  bivalve
molluscs and aquatic plants, have been
held in  these systems and challenged
with a variety of MPCAs.  Represent-
atives of all  four MPCA groups were
initially  tested,  including a  baculovirus
Autographa  californica, a  bacterium
Bacillus  thuringiensis,   a   protozoan
Nosema cuneatum  and  a  fungus
Lagenidium   giganteum.      Positive
controls  demonstrated characteristic
infections and  pathogenicity in target
species; however, no signs  of infection
or pathogenicity  were  found  in non-
target  species   using   histology,
serology, and electron microscopy.
    More recently, GB/ERL research has
focused on   microsporidian  MPCAs.
Microsporidians are small  unicellular
organisms, all  of which are .obligate
intracellular  parasites  with a unique
mode of infecting host cells. Currently,
there is  one registered microsporidian,
Nosema locustae, developed for control
of rangeland grasshoppers and  other
requests have  been received by the
Office of Pesticides Programs.  Future
registration requestsfor microsporidians
will probably increase as their pesticidal
capabilities  become  better  known.
There is a relative abundance of new
information on  the  biology of micro-
sporidians:  Many are now known to
have  indirect  life   cycles,  multiple
sporulation sequences, vertical as well
as  horizontal  transmission  and  a
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broader host specificity than previously
believed.
   One  of  the first microsporidians
tested at Gulf Breeze was the mosquito
pathogen Nosema algerae, which has a
relatively  simple  direct   life  cycle.
Development of N. algerae as an MPCA
was  originated in the  1970's  when
limited field tests were performed for
it's use as a mosquito  control agent.
Currently,  Edhazardia  aedis  is  being
investigated in collaboration  with  the
USDA's  Insects  Affecting' Man and
Animals   Research   Laboratory   for
possible use in the control of container-
breeding  mosquitos.    This  micro-
sporidian   is  an  interesting  model
because  it has a  more complex  life
cycle  than  N.  algerae,  exhibiting
multiple sporulation sequences and the
capability of both vertical and horizontal
transmission. Nontarget testing for this
MPCA has been expanded to include
fish  embryo/larval assays  and mysid
shrimp life-cycle assays.

METHODS

Nosema algerae

   A series of single  species assays
were conducted  on  freshwater and
estuarine grass shrimp  (Palaemonetes
kodiakensis and  P.  pugio),  marine
rotifers   (Brachionus  plicatilis),  and
inland  silversides  (Menidia beryllina)
employing   the   aquarium   system
described above. Exposure was water-
borne except for grass shrimp, which
were also exposed by intrahemocoelic
injection or gavage.
Edhazardia aedis

    Single  species  tests  with  grass
shrimp, P. pugio, and mosquito fish,
Gambusia affinis, were conducted  in
the aquarium systems described above.
Grass shrimp were exposed via water
or gavage.  Gambusia affinis were fed
infected or uninfected mosquito larvae
once a day for 7 days then uninfected
artificial food for 7 days. Samples were
taken eight hours after the first feeding
and then at  the end of the experiment
to examine  gill,  liver,  stomach  and
reproductive tissues for presence of E.
aedis.
    Exposure systems were developed
to test estuarine mysids Mysidopsis
bahia to E. aedis through an entire life
cycle.   Newly released young mysids
were exposed to E. aedis spores (1000
mL"1} at optimal (20 ppt)  salinity for 1
week and monitored for survival. Adult
mysids  were  exposed for  4 weeks
under   low-salinity   (7    ppt)  test
conditions and monitored for survival
and reproductive effort.
    Fish embryo/larval  toxicity assays
were  adapted to  MPCA  testing for
water-borne exposures.    Embryonic
inland  silversides> Menidia  beryllina,
were  exposed to E. aedis spores  in
moderately-hard freshwater (hardness
100mg/L as CaCoS).  Single  blastula-
stage fish embryos were placed in each
of 30  individual  tissue culture tubes
with spore  concentrations of 0, 10,
100  and  1000  mL"1.   Tubes  were
sealed with teflon  lined  caps and
embryos incubated at 25°C  with  a
14:10   photoperiod  until  death  or
hatching occurred.
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RESULTS AND DISCUSSION

Nosema algerae

   Infections  did  not  develop   in
gavaged grass shrimp, but did develop
in those that received intrahemocoelic
injections. Infected tissues included the
gills,  antennal  gland,  eyes, skeletal
muscle, heart, and  gonads.   Proof  of
infection   was  demonstrated   ultra-
structurally by the presence  of mature
spores and  developmental  stages  in
infected tissues.   Infections did not
occur  in  the   marine   rotifer  after
ingestion  of  spores  or   in  inland
silversides   fed   marine   rotifers
containing ingested spores.  Due to the
extreme exposure measures, infection
of nontarget organisms in nature is not
indicated by these results;  however
they do demonstrate that infections can
be identified and characterized  using
the diagnostic techniques available.

Edhazardia aedis

   Exposure of E. aedis spores to grass
shrimp did not cause any mortalities nor
were  any microsporidian life stages
detected  in  sampled  tissues.   Eight
hours after feeding  of infected larvae,
E. aedis was found  in the guts of fish,
G. affinis, demonstrating ingestion with
the infected mosquito larvae. However,
none were detected at the end  of the
experiment,  one week after  fish were
removed from the mosquito larvae diet.
This indicated that colonization and/or
infection of mosquito fish did  not occur.
   Initial exposures of newly released
young  mysids  to  E.   aedis spores
indicated no  mortality (> 90% survival)
after 1  week under optimal salinity and
temperature conditions. Exposures of
adult  mysids  for  four weeks under
conditions of low salinity stress (7 pptj
showed reduced mysid survival (from
80% to 54%) and inhibited reproduc-
tion (the total young produced by three
females was reduced  from 9 to  3).
Based  on these results,  a series of
studies have been initiated to examine
the interaction of stress conditions  (10
p.pt salinity and temperatures of 20,  25,
and 30°C)  survival  and reproductive
capacity of M: bahia exposed through
an entire life cycle.
    Results from a. preliminary test with
M.  beryllina indicated that E. aedis
spores cause  an increase in  embryo
mortality   (3%   control   vs.   27%
exposed)  and   deformed  larvae  at
hatching.  This effect did not appear to
be concentration dependent; rather, all
exposure  levels, of E.  aedis showed
approximately,  the   same   result.
Preliminary  observations  of  exposed
embryos  indicated  that the chorion of
developing embryos  may  serve as an
attachment surface for E. aedis spores.
The  mode  of  pathogenicity/toxicity
remains unknown.

FUTURE WORK

    Research  at GB/ERL  on micro-
sporidians will continue to expand  the
number of MPCAs tested and determine
toxicity, infectivity and pathogenicityon
nontarget aquatic organisms. Emphasis
will be placed on microsporidian species
with different and more complex  life
cycles,  such  as  Amblyospora  and
Parathelohania,   which   have
intermediate hosts in their  life cycles.
    The results obtained with E. aedis
on  fish larvae will  be pursued  and
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 mechanisms of toxicity or pathogenicity
 will  be elucidated.  The  adaptation of
 this  fish embryo/larval toxicity assay to
 MPCA   testing  appears   extremely
 productive and  other MPCAs will  be
 examined using this procedure.
   Development of  the mysid  test
 system is highly appropriate for MPCAs
 developed   for  mosquito   control.
 Mysidopsis  bahia   is  an   estuarine
 arthropod that  is closely related  to
 mosquitos and can be reared through
 it's entire life cycle in the  laboratory. It
 is a  representative  of  the dominant
 aquatic, non-insect  arthropods  (the
 Crustacea),  and  plays a crucial role in
 the  trophodynamics of  the  highly
 productive estuarine ecosystem.
   Mysids are the first nontarget test
 organism to be  used at  GB/ERL  to
 explore the effects  of environmental
 stress on pathogenicity and toxicity of
 MPCAs.  Eventually,  exposure condi-
 tions will be  altered to determine MPCA
 effects under stressful environments for
 several  nontarget species.  Stressors
 will include both natural environmental
 factors  (salinity and  temperature) and
 chemical pollutants. It is expected that
 these types  of stress are prevalent in
 nature  and  more accurately  reflect
 conditions of susceptibility.

 PUBLICATIONS

 Couch,  J. A., T. W. Duke, S. S. Foss,
 and   K.  T.  Perez  1986.  Enclosed
systems for  testing microbial  pest
control  agents.  Proc.  workshop  at
 ERL/Gulf Breeze, sponsored by U. S.
 EPA,  Office of  Pesticides Programs,
Washington, D. C.
 Couch, J.  A.,  S.  S.  Foss, and  L. A.
 Courtney 1985. Evaluation for risks of
 an   insect   virus,   bacterium,   and
 protozoan .to a  nontarget, estuarine
 crustacean. Report EPA 600/X-85/290,
 U. S. EPA,  Gulf Breeze FL.

 Couch,  J.  A.,  S.   M.  Martin,  G.
 Tompkins,  and J. Kinney  1984.  A
 simple  system for   the   preliminary
 evaluation   of  infectivity   and
 pathogenesis  of  insect  virus  in  a
 nontarget   estuarine  shrimp.  J,
 Invertebr. Pathol. 43:351-357.

 Couch, J.  A.  and K. R.  Rao  1983.
 Biorational  Workshop.  Report  EPA-
 600/X-83-054,  Gulf Breeze ERL,  Gulf
 Breeze FL.

 Foss,  S. S.,  L. A. Courtney, J.  W.
 Fournie  and  D.  V.   Lightner  1989.
 Nontarget  testing of  microbial   pest
 control agents  in  aquatic  systems.
 Report EPA/GOO/x-89/387, 21pp.

 Fournie,  J.  W., S. S.  Foss and J. A.
 Couch 1988. A  multispecies system for
 evaluation  of  infectivity   and
 pathogenicity of microbial pest control
 agents in nontarget  aquatic  species.
 Dis. Aquat. Org. 5:63-70.

 Fournie,  J.  W.,  S.  S. Foss,  L.  A.
 Courtney and  A.  H.  Undeen 1990.
Testing  of  insect   microsporidians
 (Microspora: Nosematidae) in nontarget
aquatic  species.  Dis.  Aquat.  Org,
8:137-144.
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   DEVELOPMENT OF TEST METHODS TO ASSESS FATE OF MICROBIAL PEST
                 CONTROL AGENTS AND THEIR EFFECTS ON
                     NONTARGET AQUATIC ORGANISMS

          Fred J. Genthner, Geraldine M. Gripe and Doug P. Middaugh
                     U.S. Environmental Protection Agency
                      Environmental Research Laboratory
                                Sabine Island
                             Gulf Breeze, Florida
INTRODUCTION

   Microbial   pest  control  agents
(MPCAs) are regulated by the Environ-
mental Protection Agency. One respon-
sibility of EPA is to prescribe tests for
determining the  potential   ecological
risks of MPCAs to nontarget organisms.
   To fulfill  the Agency's  need  for
ecosystem effects testing of  MPCAs, a
fully  contained,  single species  test
system was  developed to  determine
whether  exposure  of  a   nontarget
aquatic invertebrate to an MPCA will
result  in   infectivity,   toxicity  or
pathogenicity.  This system  was field
calibrated  to  assess whether MPCAs
will colonize, persist or germinate in the
animal.    MPCAs  representing   a
vegetative bacterial cell, Pseudomonas
fluorescens;  and   both  a  fungal,
Colletotrichum gloeosporioides (Cga),
and   a   bacterial   spore,   Bacillus
sphaericus,   were   used   for  field
calibration.
   In addition, we have successfully
adapted standard chemical toxicity test
methods   for   assessing   nontarget
effects  by  exposing  the  estuarine
crustacean,  Mysidopsis  bahia,   and
embryos  of the  inland  silverside fish,
Menidia beryllina to Beauveria bass/ana,
an entomopathogenic fungus.
METHODS

Single Species Test System

    American   oysters,   Crassostrea
virginica, were exposed in enclosed 57
liter aquaria filled with filtered (2 /vm)
seawater  from  Santa  Rosa  Sound.
Oysters were fed with a mixture of 3
marine algae 3 times a week.
    To  field calibrate the  elimination
rates of MPCAs, exposed oysters were
thoroughly cleaned and  divided into 2
groups. One group was placed in wire
cages suspended in 6-8' of water (6
inches above the bottom) in Santa Rosa
Sound.   The remaining oysters were
placed in an enclosed 57 liter aquarium
equipped  with  aeration, and  a pump
driven,  recirculating system (1  £/min)
which  allowed  water  to  pass  first
through an external cartridge filter of
crushed  coral  then   through  a  UV
sterilization unit.   Both the exposure
and UV-clearance  tanks were  sealed
with a plexiglas lid.  Sampling ports and
inlets  for  air and  water were  sealed
with silicon stoppers.
    Viable counts of the MPCAs were
performed on aquaria water and oyster
samples at regular  intervals. Sections
of whole oysters were taken for histo-
logical examination. Tissue samples
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were   fixed   in  Bouin's   solution,
dehydrated, embedded in paraffin, and
sectioned at  6  //m.   Sections were
stained  with  Harris'  hematoxylin and
eosin, Brown and Brenn tissue Gram
stain or  periodic  acid shift.

Nontarget Effects

   ASTM recommendations for acute
exposures were  used in toxicity tests
exposing M. bahia with viable spores of
B. bassiana.   Spores were  obtained
from cadavers of infected Heliothis zea
to assure pathogenicity. Inoculum was
prepared  by  scraping  conidiospores
from the surface of 11 day old  plates.
Spores were either treated with Triton
X-100 (0.03%) or left untreated before
exposure of the  animals. Animals (10
per bowl, 2 bowls per spore density)
were  exposed for 96  hours  at spore
densities of 2 X 106,  1 X 106, 1  X 105,
1 X 104, 1 X 103 per ml.  Autoclaved
controls   treatments  were  also
performed.   Beauvericin (Sigma), an
secondary metabolite produced  by this
fungus,  was also tested with M.  bahia.
   Single blastula stage embryos of the
inland  silverside, M.  beryllina, were
placed in each of 120 Leighton tissue
culture  tubes   and   exposed  to   B.
bassiana at densities of .1  X 106, 1  X
105 and 1 X 104 conidiospores per ml.
Embryos were incubated at 25 C under
a 14L:10D photoperiod. Observations
for effects were  made daily.

RESULTS AND DISCUSSION

Single Species Test System

   Neither toxicity,  pathogenicity,  or
infectivity  was  observed  in  oysters
exposed  to  B. sphaericus, Cga or P.
fluorescens.      Germination  of B.
sphaericus of Cga  spores  was  not
observed  in the oyster tissue.   The
rates of clearance of these MPCAs from
the oyster were  compared in exposed
oysters relayed  in the UV-tank or  in
Santa  Rosa  Sound.  With all MPCAs
tested  the  rates of clearance  from
oysters in the sound versus the  UV-
clearance tank were similar.
    The  rates  of clearance,  however,
between   MPCAs   varied   widely.
Oysters  were exposed to  either B.
sphaericus and  P.  fluorescens for  2
weeks.  B. sphaericus spores were
undetectable in  oyster tissue at  5
weeks. In contrast, P. fluorescens was
still detectable in oyster tissue at 49
days.  Oysters, exposed to Cga spores
for 3 days, contained 1.7 X 105 spores
per gram dry wt.  These spores were
rapidly cleared from the  oyster  and
were undetectable in oyster tissue  at 9
days of clearance.
    Exposure plates, placed  in various
locations during  the laboratory tests,
indicated a completely sealed system.
The  clearance rates of MPCAs  from
oysters in the fully contained system
were similar to the ''clearance rates  in'
the field.  Thus,  this system  will be of
value before an  environmental release
has been deemed safe in  determining
whether,  or  how   long,  genetically
engineered  MPCAs  or other micro-
organisms  will  persist in  nontarget
aquatic  invertebrates  and   whether,
exposure   will  result   in   toxicity,
infectivity or pathogenicity.

Nontarget Effects                   '.

    In  an initial  toxicity test with M:
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 bahia, B. bassfana spores were treated
 with  Triton X-100  to aid in dispersal.
 This  test demonstrated no toxicity of
 the spores up to and including 1 X 106
 per ml test water.  However, a sub-
 sequent exposure  of  test  animal to
 these spores without the wetting agent
 indicated that with this particular strain,
 mortality  was  associated with  high
 particulate density of the spores (as low
 as 1  X 106 per ml) an not with spore
 viability.  Testing  of beauvericin, an
 antibiotic  produced by  some other
 strains of  this fungus, has  indicated
 some toxicity.
   While   responses   of the inland
 silverside  embryos  to B. bassiana has
 been  variable among the three tests
 performed  to  date, in  all  instances
 pathogenicity has been observed in the
 form  of  rupture of  the chorion  of
 developing embryos followed by death,

 FUTURE WORK

   The fully contained exposure  and
 clearance system will continue to  be
 field  calibrated  using   a viral   and
 protozoan MPCA. Testing B. bassiana,
 its secondary metabolites, and other
 MPCAs will proceed using M. bahia.
 Additional exposures of inland silverside
 embryos are planned to determine the
 reason(s)  for variability of responses
 among tests.

 PUBLICATIONS

 Yousten,  A.A.,  E.F.  Benfield,  R.P.
 Campbell,   S.S.    Foss,   and   F.J.
 Genthner.  1991.   Fate  of   Bacillus
sphaericus  2362   spores  following
 ingestion by nontarget invertebrates. J.
Invert. Pathol. (In  Press).
Genthner, F.J.,  S.S.  Foss, and  R.P.
Campbell.   An  enclosed system for
testing effects of microbial pest control
agents   on   nontarget   aquatic
invertebrates. (In Preparation).

Genthner, F.J. and D.P.  Middaugh.
Effects   of   the   entomopathogenic
fungus, Beauveria bassiana, on inland
silversides, Menidia beryllina, embryos
(In Preparation).
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                 FATE OF Bacillus sphaericus MICROBIAL PEST
                   CONTROL AGENT IN THE ENVIRONMENT

                     Allan A. Yousten and Ernest F. Benfield
                              Biology Department
                Virginia Polytechnic Institute and State University
                              Blacksburg, Virginia
INTRODUCTION

   Some  strains  of  the  bacterium
Bacillus sphaericus have been found to
be  pathogenic  for  mosquito  larvae.
This pathogenicity  is  caused  by  the
production by the bacteria  of  protein
toxin which is lethal  upon ingestion by
larvae  of  many  species  of  mosquito.
Unlike  the toxin produced by Bacillus
thurfngiensis subsp.  israelensis (Bti),
the toxin of B. sphaericus is not active
against black fly larvae.  The gene for
the B. sphaericus toxin has been cloned
and  sequenced from several strains of
the  bacteria and has been found to
differ  among  strains  by only a  few
amino  acids.  However, the strains do
differ  somewhat in  their toxicity for
different  species of mosquitoes  and
these  differences  probably  lie in  the
slight   differences  in "  amino   acid
sequence in the toxins.   The toxin  is
synthesized in the bacterial cell at the
time of sporulation and it accumulates
in the  cell  as a parasporal inclusion
body or "crystal". The inclusion body
contains  primarily two  proteins,  and
both of these are required for toxicity to
larvae. Thus, the toxin of B.  sphaericus
is a  "binary toxin".
    In  its  role as a mosquito larvicide,
viable  B. sphaericus spores  along with
the attached parasporal bodies will be
delivered  into  the aquatic environment.
Since the metabolism of B.  sphaericus
is  different  than  that  of  Bti,  the
ecological niche in which it may survive
or grow in the aquatic environment may
also differ. One salient feature that has
attracted  attention  is  the  apparent
ability of B. sphaericus to persist in the
mosquito larval feeding zone for longer
than Bti.   This  effect  has not been
observed  in all field trials, but it  has
been reported often enough to make
this bacterium an attractive larvicide in
situations where frequent application is
not practical.  The reasons for  this
larvicidal persistence are unclear.
    Our studies  are examining  fate of
the  bacterial  spores   during  their
interaction with the aquatic environ-
ment  as  well as  the  fate  of  spores
ingested   by   certain   non-target
invertebrates.

METHODS

Bacterial Growth

    B.   sphaericus  2362-7,  a  spon-
taneous rifampicin-resistant mutant of
B. sphaericus was used in  all  experi-
ments.    Spores  were  produced by
growth in nutrient  broth-0.05% yeast
extract-mineral salts  (Mn2 + ,  Mg +,
Ca2 + ) broth (NYSM) with shaking at
30°C  for 48  hours.    Spores were
washed  and held  as  a  distilled water
suspension  at 4°C.  Spore numbers
were determined by heating 2 ml  of a
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 sample at 80°C for 12 min. and plating
 on NYSM agar containing 50  yt/g/ml
 rifampicin and 0.002% cycloheximide.

 Midge Larvae

    Seventy   five,  fourth    instar
 Chironomus riparius larvae were placed
 into 200 ml  sterile distilled water in a
 23 cm. X 23 cm. dish at 23°C. Three-
 tenths  ml Tetramin suspension was
 added along with B. sphaericus 2362-7
 spores at a final concentration  of 9.6 X
 I06/ml. After 5 hours of feeding, the
 larvae were  rinsed 3 times in 100  ml
 sterile distilled water.  Five larvae were
 weighed,  homogenized  in  a  ground
 glass tissue grinder, and a spore count
 performed  as  described.   Forty five
 spore-fed larvae were placed into 200
 ml water  containing  Tetramin  but
 without spores.  Groups of five were
 homogenized at  intervals to obtain
 spore counts.
    To collect larval feces, twenty five
 spore-fed larvae were  placed into  a
 funnel blocked at the narrow end with
 screen.  The funnel outlet led into a
 latex rubber tube which dipped into an
 ice water bath.   A small amount  of
 Tetramin  was added  to  the  sterile
 distilled water  in the funnel.   After 17
 hours, the 50-ml of water in the funnel
 and tube were  removed and the funnel
 and tube were rinsed with 50 ml water.
 The combined volumes  were centri-
 fuged  (27,200 x g,  15  min),  the
'resulting  pellet   containing   2362-7
 spores  suspended in  sterile  distilled
 water, and a spore count performed.
   Thirty-one snails, Helisoma trivo/vis,
were placed into 200 ml sterile distilled
water in a 23  cm. X 23 cm. dish at
23°C.  B. sphaericus 2362-7  spores
were added to  a final concentration of
9.6 X I06/ml. After grazing for 5 hours,
the snails were rinsed 3 times with 50
ml sterile water and placed into 300 ml
sterile water  containing  a  piece  of
lettuce.  Two snails were immediately
removed, the bodies separated from the
shell, the foot dissected away, and the
remaining tissue homogenized. A spore
count  was   performed   on   the
homogenate.   The remaining  snails
were placed in fresh sterile water daily
for the first three days after feeding on
spores and twice  weekly thereafter.
Lettuce   was   provided   as   food
throughout  the experimental period.
Three  of the   spore-fed  snails  were
placed in a funnel apparatus described
above for fecal collection.  These snails
were also moved to fresh water daily
for  three  days  and  twice  weekly
thereafter to  prevent reingestion of
feces that might not pass through  the
screen at the   bottom of the funnel.
Feces were collected for 17 hours,  the
collection water centrifuged (27,200 x
g, 15  min.), and  the  resulting  pellet
suspended in sterile distilled water for
spore counts and bioassay.

Oysters

    Four,  112-liter glass  aquaria were
used for  spore  feeding tests; two  for
inoculation  and  two  for  controls.
Aquaria were   equipped  with  under-
gravel filters covered with crushed coral
and filled  with  artificial seawater at a
salinity of 10 parts per thousand.  Sixty
cleaned oysters were placed into each
test  aquarium.  B.  sphaericus spores
                                     191

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 were added and the  aquaria sealed.
 Oysters were fed with a mixture of 3
 marine  algae every third  day.   To
 determine the spore burden in oysters,
 samples of digestive, gill and remaining
 whole oystertissues were homogenized
 as previously described.
    To  determine  spore  elimination
 rates, 20 oysters were removed from
 each inoculated  aquarium  after  28
 days,  cleaned,  and   placed  into  a
 flow-through  seawater  tank.   Four
 oysters were removed every week for 5
 weeks  and  a   spore  count   was
 performed on the whole oyster tissue.
 Fecal  material was  obtained directly
 from  the   shell  surface.     When
 recognizable,   fecal   material    was
 collected from the tank bottom using a
 pipette. Fecal material was placed into
 a test tube, and the excess liquid was
 removed from the surface.  The tube
 was centrifuged  (7,700 X g, 10 min.),
 and the supernatant discarded.

 Toxicity of Snail and Oyster Feces

    Feces from snails and oysters were
 collected as described above.   Fecal
 suspensions   were  bioassayed  for
 toxicity to second instar larvae of Culex
 quinquefasciatus.

 Stonefly Larvae

   Paragnetina media (Walker)  larvae
 were placed individually into chambers
 containing 400ml tap  water at 20°C.
 Midge larvae were fed on/ B. sphaericus
 spores overnight,  rinsed 3X,  and  3
 larvae   placed   into  each   stonefly
chamber.    Only  stoneflies  that  had
eaten 3 midges within 4 hours  were
used  in  the depuration experiment.
 Immediately after  feeding  on midges
 and at intervals thereafter, the stonefly
 larvae  were  rinsed,   appendages
 dissected  away, bodies weighed, and
 homogenized.    Homogenates  were
 plated for spore counts.  Beginning 24
 h  after having  ingested spore-laden
 midges, the  water  in  each stonefly
 chamber was changed  and this  was
 repeated on  alternate days.   On the
 days  on  which the  water was not
 changed, each stonefly larva was fed 1
 midge larva  (not  containing spores).
 The water changes alternating with
 midge  feeding   were   continued
 throughout the experiment.

 Crane Fly  Larvae

    Tipula  abdominalis  larvae   held
 individually in 400 ml chambers in the
 dark at  I3°C  were each  fed 1 yellow
 poplar conditioned leaf  disc that had
 been  soaked  in  B. sphaericus spore
 suspension.  Each disc  had approxi-
 mately  5xl06  spores adhering to it.
 Twenty  four hours after consuming the
 spore-laden disc,  the water in  each
 larval  chamber was changed and  a
 fresh  leaf disc lacking spores was fed.
The water was changed and a leaf disc
fed at 48 h intervals  thereafter for the
duration of the experiment. At intervals
groups of  3 larvae were removed, the
guts dissected out, weighed, homogen-
 ized,  and  spore counts  performed on
the homogenates.

RESULTS AND DISCUSSION

Indigenous Microflora

    None of the animals or their feces
used in this study contained microflora
                                    192

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which survived  heating  at 80°C and
grew on NYSMRC selective medium in
numbers  which   interfered  with
enumeration of B. sphaericus,

Midge Larvae

    Elimination  (depuration) of spores
from fourth instar larvae was followed
for 4  days   following  ingestion  of
spores.  Larvae  examined immediately
after   feeding  averaged  1.2  X  10°
spores/mg larval dry  weight.   This
number   declined  to   7.7   X   I02
spores/mg after 4 days at which time
the larvae began to pupate.  The total
viable bacterial count on the fourth day
was approximately equal to the spore
count.   The  spore  content  of feces
declined  in parallel  to   body  spore
content.
    Larvae that were reared, allowed to
pupate, and emerge as adults in water
initially containing I X I07 or 4.3 X I03
spores/ml, had an average of 8.2 X I03
and 68 spores in/on the bodies (15 and
18  flies,  respectively).     At  the
conclusion of  the  experiments, the
culture water contained 9.5 X I06 and
8.4 X  I02 spores/ml.   Control  flies
reared in  water  lacking added  spores
averaged 4 spores/fly (14 animals) and
no spores/fly (10  animals), respectively.

Snails

   Depuration of spores from snails
was followed for 49 days.  There was
a rapid decline  (about 2  logs) in the
body burden of spores during the first 7
days  followed   by. an  approximately
constant number of spores (between 2
X I01 and 2 X I02/mg) for an extended
period.   At 49  days  the  body spore
content  declined  to  less  than  IOO
spores/mg for the first time.  As with
midge larvae, the total viable  bacterial
count approximated the spore count.
The spore content of feces  declined
even more rapidly than  that of  the
body, but it also leveled out/and feces
containing low numbers of  spores
continued  to  be  produced  for  the
remainder of  the experiment.
    To eliminate the possibility that the
persistence of  spores in snails was a
result of reingestion of feces containing
spores, snails were allowed to feed in
9.6 X I06 spores/ml for 23 hours which
resulted in a body content of 4.9 X I06
spores/snail.  These snails were placed
in a sterile fecal collection funnel which
was  drained  and  rinsed  with sterile
water twice daily before being refilled
with sterile water.  After 7 and 14 days,
the snails were transferred to another
sterile funnel. After 21 days, the feces
from the snails  were collected and the
body burden of spores determined. The
snails averaged 1.7 X I04 spores/snail
and  the  feces  contained  2.3 X  I03
spores/snail.
    The   LC50  of  the  spore/toxin
preparation fed  to snails  was 1.2 X I02
spores/ml.  This declined 66% to 3.6 X
I02 spores/ml in feces.

Oysters

    Oysters placed in spore-containing
water and then transferred to flowing
seawater, decreased in spore  Content
from 2.5 X I06 spores/g dry tissue to
5.0 X I03 in 21 days and had eliminated
the  spores in 37  days.   Histological
examination showed no  indication  of
spore  germination  or of vegetative
bacilli in  the  gut.  Oyster feces were
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non-toxic when fed to mosquito larvae,
apparently  having  been detoxified by
passage through the oyster.

Stonefly Larvae

   Ingestion and clearance of spores in
a   predator-prey   relationship   was
examined   with   stonefly   larvae
(predator)  and spore-carrying  midge
larvae (prey). The midges were rapidly
consumed  by the  stoneflies  and B.
sphaericus spores were found to be
present  in  the stoneflies  at levels
approximately equal to the sum of the
spores present in the midges.   The
spore  number declined  rapidly  and
spores  were  below  the  level  of
detection by 7 days.

Crane Fly Larvae

   In contrast to rapid depuration seen
in midge and stonefly larvae, crane fly
larvae retained a large number of spores
for an extended period of time.  The
animals had a mean number of 4.5 X
I05 spores/gut immediately after eating
a leaf disc.  This number decreased to
8.5 X I04 after 4 weeks.  This was the
highest  level   of  spore  retention
observed  among any  of the  animals
studied.   Spores were present in  the
feces for the entire  period,  but  the
content dropped more rapidly than did
the spore content of the animal  gut.
Total viable plate counts done at 1  day
and  2 weeks after feeding  on spores
approximately equaled the spore count.
This suggests that at least on those
times there  was not a large population
of  vegetative  cells  growing  in   the
animal. Such a population could have
continuously  produced   spores   to
replace those .lost in  the  feces.  We
cannot exclude the presence of a small
vegetative  population approximately
equal to the  spore  population  that
would  not  be  detected  given  the
sensitivity of plate counts.  The crane
fly has an unusually alkaline foregut and
a  possible  relationship  between  this
phenomenon and  spore  retention or
production is being investigated. Also,
more a exact location  of the spores in
this anjmal is being determined.

Summary

    Spores of the MPCA, B. sphaericus,
were consumed but largely or complete-
ly  eliminated from the gut of 2 insects
(midge,,  stonefly)  and   1   mollusk
(oyster).   A second mollusk (snail)
eliminated most  of  the  spores  but
retained a low number for an extended
period  of time.   A third insect, crane
fly,  retained a  significant  number of
spores for up to I month.   All  animals
tested  passed viable  spores  in their
feces.    Midges  reared  in   spore-
containing water emerged as adults
carrying spores in/on their bodies.  The
mosquitocidal toxin of  B.  sphaericus
was partially  destroyed by  passage
through the snail  and completely (or
nearly   completely)  destroyed   by
passage through the oyster.

FUTURE WORK

    We will continue to examine the
effect  of different gut  conditions on
survival of B. sphaericus spores.  This
will focus on the highly alkaline gut of
the crane fly where extended  survival
was observed and move to another leaf
shredder, Pteronarcys proteus, which
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has a slightly acidic gut. We will also
examine a micro-crustacean, Daphnia,
for   the first  time.    The possible
proliferation  of   B.   sphaericus  in
nontarget cadavers produced when the
animals died of other (non B.
sphaericus  toxin)   causes  will   be
investigated.   These  interactions  of
spores with nontarget animals will be in
addition to  other  studies  of  the
interaction of  spores  with physical
factors  in the aquatic environment.

PUBLICATIONS

Yousten,  A.A.,  E.F.  Benfield,  R.P.
Campbell,   S.S.   Foss,   and   F:J.
Genthner. Fate of Bacillus sphaericus
2362' spores  following  ingestion  by
nontarget invertebrates. J.  Invertebr.
Pathol.  (in press).

Genthner, F.J., R.P.  Campbell,  S.S.
Foss, E.F. Benfield, and A.A. Yousten.
1990. Fate of Bacillus sphaericus 2362
spores following ingestion by nontarget
invertebrates.  Abst. Annu.  Mtg. Am.
Soc. Microbiol. p. 254.
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           PROTOCOL DEVELOPMENT FOR TESTING THE EFFECTS OF
                BACTERIAL PESTICIDES ON BENEFICIAL BEETLES
                     Rosalind James1 and Bruce Lighthart2
                   ManTech Environmental Technology, Inc.1
                     U.S. Environmental Protection Agency2
                       Environmental Research Laboratory
                               Corvallis, Oregon
INTRODUCTION
    Microbial  pest   control   agents
(MPCAs) are increasing in use because
of their specificity and low mammalian
toxicity and pathogenicity;  however,
they  are  a  potential hazard to non-
target, beneficial insects.  MPCAs could
be  used more effectively in  integrated
pest  management if their impact on
non-target insects was better  under-
stood.   Non-target  insects are eco-
logically and economically important in
a variety of ways, including:  as natural
predators and parasites  of  pests, as
introduced  biological  pest  control
agents of both insect and plant pests,
as   food   for   wildlife,   and  as
decomposers.  Subdivision  M  of the
Pesticide Testing Guidelines calls  for
assessing  the  potential hazard  of
MPCAs on several orders of terrestrial
non-target insects including Coleoptera
(beetles).    Such  tests  need  to  be
standardized because the  results of the
tests may be affected by the conditions
of the bioassay.  To develop a standard
assay for testing  the effects of bacterial
pesticides on beneficial  insects, the
effect  of  temperature  and  dietary
stress,  and  of  larval instar on the
susceptibility of Hippodamia con vergens
(the convergent lady beetle) to a weak
bacterial pathogen  was  tested.   H.
convergens  is  a  major  predator  of
aphids and occurs throughout most of
the United States and as such  is ah
important biological control agent.

METHODS

    H. convergens was reared on pea
aphids, Acyrthosipon pisum, at 25°C,
70%  RH.  Pseudomonas fluorescens
was used  as  the pathogen (i.e. the
surrogate MPCA). It is a bacterium that
was isolated from the haemolymph of
dead adult beetles during a time when
there  was  high  morality  in   the
laboratory  colony.     The  relative
susceptibility of different larval instars
of H. convergens to P. fluorescens was
compared  by  determining the  dose
response of each  instar at 25°C on an
aphid diet.  First instar larvae were used
to determine  the effects of diet and
temperature stress  on  susceptibility.
Since temperature and the insect's diet
can  potentially  affect  the  bacterial
virulence as well  as  insect suscept-
ibility, the treatments were given to the
insects in 24  hour  pulses before the
insects were exposed to the pathogen.
Three  different  diets   (water,   5%
sucrose, and aphids) were used at each
of two temperatures (25° and 30°C),
thus there were six treatments.
    The  experiment was set  up to
determine the probit dose response of
the pathogen on  the  beetle.  Insects
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were exposed  to  the  pathogen  by
dipping them in the appropriate dose of
the bacteria.  Five or  six doses were
used per treatment with twenty insects
per dose for a total of 100-120 insects
per  treatment.     There  were  two
replicates of  each treatment.  After
insects   were  dosed,   they  were
maintained at 25°C, 70% RH, 14:10
hours   light:dark.     Mortality   was
recorded on  day six.  Both replicates
were combined for the probit  analysis
of mortality.

RESULTS AND DISCUSSION

    The mean LC50 for the first, second
and third instars was 7.9x109 cfu/ml.
There were no significant differences in
the slopes  or intercepts  of the probit
lines for  these age groups.  However,
the  last instar  was  40  times  less
susceptible with an LC50 of 3.2x1011
cfu/ml. The slope of the probit  line was
not significantly different from the other
instars  but  the  intercept was  (p.
-------
convergens, and to develop protocols
for beneficial Diptera,  such  as the
Tachinidae.   The  methods used for
laboratory testing of the protocols will
be similar to that used for this protocol.
However,  future work is  planned for
laboratory and  field studies  used to
determine the appropriateness of the
LCg0 endpoint.  Some of the questions
we would like to address are:  What
does a laboratory LC50 mean when it
comes to field  applications?   Might
some other endpoint be more approp-
riate?  We would also like to monitor
the effects of specific MPCAs on non-
target  insects under field conditions.
The results of laboratory and field tests
could then be incorporated into a model
for predictive value.
PUBLICATIONS

James, R.R. and Lighthart, B.   1990.
Bioassay for testing the lethal effects of
bacterial  pathogens on the predatory
beetle  Hippodamia  convergens  Gue.
(Coleoptera:  Coccinellidae). National
Technical ,  Information  Service
publication   no.   PB91-127795.
Springfield, VA.
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             ASSESSING HOST SPECIFICITY OF FUNGAL MPCAs
              TO THE BENEFICIAL WASP Trichogramma pretiosum

                        O.K. Sewall1 and B. Lighthart2
                   ManTech Environmental Technology Inc.1
                    U.S. Environmental Protection Agency2
                      Environmental Research Laboratory
                              Corvallis, Oregon
INTRODUCTION

   The  registration  process   for
Microbiological  Pest Control Agents
(MPCA's)  requires data on nontarget
effects  to  representatives  of   key
beneficial   insect   species,  e.g.,
Trichogramma  pretiosum  Riley,   a
lepidopteran  egg  parasite.
Trichogramma spp.  are recognized as
important, both as naturally occurring
and  commercially  released  natural
enemies  of  pest Lepidoptera.    The
laboratory evaluation  of  MPCAs  for
nontarget   effects   is   generally
recognized   as  the  first  step   in
determining  the  risk  a pest control
agent may pose to  natural enemies.
Although methods have been developed
to evaluate chemical pesticide effects
on  Trichogramma  spp.,   bioassay's
evaluating   entomogenous  fungal
pathogenicity to  Trichogramma spp.
have reported only negative effects.
   This  study  was  undertaken  to
develop   bioassay   procedures   to
evaluate  pathogenicity  of   entomo-
genous  fungi, compare specific  and
nonspecific fungi, and evaluate factors
that may affect the expression of fungal
virulence  to   T.  pretiosum  or  the
susceptibility of T. pretiosum to fungal
pathogens.
METHODS

Insects Used

    A  colony of  T.  pretiosum was
maintained  on  U-V  killed  Cabbage
Looper eggs,  Trichoplusia ni (Hubner),
at 25 ± 1C0, 75% RH and with continu-
ous light (40w fluorescent).

Fungi Tested

    The life form of the  fungi tested
was conidia.  Pure stocks  of Beauveria
bass/ana and  Metarhizium anisopliae
conidia, without  mycelia or agar, were
obtained by   inoculating  Sabouraud
Maltose  Agar + 1 % yeast extract
(SMA +Y)  plates,  lined with a sterile
disc of  200  mesh nylon  screening.
After  21  days incubation,  plates were
dried  and conidia were scraped from
the screening with a sterile spatula and
stored dry (20-40mg portions) at-70°C
until needed.  B.  bass/ana was passed
through  T. pretiosum by inoculating
ten  wasps  with  enough  conidia
(108/ml) to  ensure  100%  mortality by
72 hours when incubated  at 100% RH
and 25°C.  As wasps died, they were
moved to a fresh vial,  incubated until
covered with  conidia, seven days, and
ten new wasps were added to the vial.
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 These steps were repeated five times.
 Conidia on  cadavers from the  last set
 were washed with 1.0% T-80, and a
 stock   of   conidial   inoculum  was
 generated  as  described above.   To
 ensure maximum pathogenicity of the
 host specific fungus  Nomureae rileyi,
 conidia were collected  directly from
 infected   T.  nf  cadavers and used
 immediately.

 Inoculum Preparation

    Approximately  20mg  of  conidia
 were  suspended in  3  ml  of sterile
 0.10% (v/v) Tween 80 (T-80) with  an
 all glass  tissue grinder and processed
 until clumps of conidia were no longer
 visible.  A   dilution   sequence  was
 prepared  with a final  T-80 concentra-
 tion   =   0.01%.  Concentrations  of
 conidia were based on viable conidia
 and estimated with drop plate counts.

 Exposure  to pathogen

   Because  of  the  small  size  and
 relative delicateness of Trichogramma
 spp. special techniques were developed
 to  handle  and  expose wasps   to
 pathogens which minimized  risk  of
 pathogen    escape   while   allowing
 efficient handling  of the large numbers
 of  T.  pretiosum needed  for  dose
 response  tests.  To sort and  handle,
 wasps were anesthetized on a cold
 plate   (3-10°C).   Medium  to  large
 females (0.5-0.75mm  long) were used
 for   the   bioassay.   Wasps   were
transferred  in  groups  of  75    to
confinement chambers of an immersion
apparatus. Wasps were exposed to
 pathogens  by  filling  the  immersion
 apparatuses with conidial suspensions.
 Apparatuses containing wasps were
 then dried on a vacuum manifold with
 down  stream  air filtered  to remove
 entrained conidia.  Wasps from each
 apparatus  were  divided  into  three
 groups of 20 wasps each, transferred
 to clear one ounce plastic cups supplied
 with a streak of 50%  fructose syrup,
 sealed with snap over  plastic lids, and
 incubated   inverted   in    humidity
 chambers.  To  evaluate the  effect of
 relative humidity on the expression of
 virulence  by  B.  bass/ana  and  M.
 anisopliae, cups with wasps were either
 supplied with four droplets (0.010 ml
 ea.) of distilled water and incubated in
 a  humidity  chamber  with  saturated
 KH2PO4  (= rapid saturated air)  or
 water  droplets  were omitted and cups
 were  incubated in   chambers  with
 distilled  water (=  delayed saturated
 air).
    Mortality was evaluated on days 3
 and  4   after   exposure  to  fungi.
 Estimates   of   the   reported   dose
 response  parameters   and  pairwise
 comparisons of dose  response slope
 functions  were  made  with  .probit
 software (SAS Version  6.06).
    Conidia adhering to wasps  were
 enumerated by using drop plate counts
 on  SMA  +  Y  plates. Counts  were
 prepared using ten wasps  from each
 dilution in a ten-fold dilution sequence
 and homogenized in 1 ml of 0.1 % T-80
 with tissue grinders. Duplicate  plates
 were  prepared   directly  from   each
 dilution with no further dilution.  Plates
 were counted  at 48   hours for  B.
bass/ana and at 72 hours for N. rileyi.
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 RESULTS AND DISCUSSION

    The  bioassays   differentiated
 between host specific and nonspecific
 pathogenic fungi. Of the three species
 of fungi with which T. pretiosum was
 challenged,  only N.  rileyi  failed  to
 produced mortality.  B. bassiana levels
 of virulence were significantly different
 for the isolates tested. Virulence  of a
 given isolate of B. bassiana maintained
 continuously  on artificial  media  was
 significantly increased  when  passed
 through T.  pretiosum.  Estimates  of
 conidia  adhering   to   wasps  after
 immersion indicated that the lack of A/.
 /•//ey/pathogenicity to T. pretiosum was
 not due to rate of conidia attachment to
 wasp cuticle but to other factor(s).  M.
 anisopliae and B. bassiana while both
 pathogenic  to T. pretiosum, expressed
 different  levels  of  virulence  to   T.
 pretiosum when saturation of air in the
 test vials was rapid versus  delayed.
 When saturation of air was delayed the
 virulence of these fungi were nearly
 identical. When saturation of  air  was
 rapid the  virulence  (LC50)   of  both
 pathogens  increased (= decrease  in
 LC50)  with  M.  anisopliae having  a
 significantly  lower   LC50   ( = more
 virulent).  However, the dose response
 slope function for B. bassiana increased
 by  64%  while  the  slope  for  M,
 anisopliae  decreased  by  74%.    T.
pretiosum longevity  and susceptibility
 to B. bassiana  was  not affected  by
 withholding food, withholding water, or
 by exposure to temperature extremes
 (5°  or  30°C  for  24  hours).  These
 results  indicate  that  the  nontarget
effects of entomogenous fungi on T.
pretiosum are largely affected by innate
differences of the fungi tested and the
 environmental factors, relative humidity
 and  temperature,  which  affect the
 expression of virulence by the fungus.
 For  quantitative   comparisons  of
 different species, isolates, or strains of
 entomopathogenic  fungi the  culture
 history,  original isolate source  and
 environmental conditions adequate, for
 expression of the pathogen's virulence
 must be  rigorously documented  and
 controlled. The shifts and differences in
 the  dose  response  slope  functions
 observed  when  saturation of air  was
 achieved   rapidly   suggest   that
 comparisons  between  pathogens for
 relative virulence and host susceptibility
 must  go  beyond  the  classic  point
 comparisons (LC50 fiducial  limits)  and
 include  the  other  dose   response
 functions e.g., slope. This bioassay will
 discern between  host  specific  and
 nonspecific   pathogenic  fungi   and
 produce quantitative results to compare
 nonspecific  fungal    MPCAs   for
 pathogenicity to T. pretiosum.

 FUTURE WORK

    Future research is to be focused on
 developing test procedures to evaluate
 pathogenicity of MPCAs to adult  and
 larval honey bees (Apis mellifera L.) and
 effects on colony health and product-
 ivity.  Our goals are to (1)  develop a
 microbiologically defined  test system
 using  microcosms and  mini-hives to
 evaluate  the  effects  of  MPCA's on
 specific  life  stages,  e.g.,  workers
 versus larvae, and the colony and (2)
evaluate the  effects of biotic and abiotic
environmental factors on bee suscept-
 ibility. Parameters proposed to evaluate
effects include, but are not limited to,
worker longevity, larval  survivorship,
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food consumption and storage, and the
growth  rate of  the  colony. Pending
development  of  the microcosm,  the
system will be validated for detecting
effects using known life stage specific
honey  bee pathogens.  This  will be
followed  by  evaluations of environ-
mental factors on susceptibility of bee
life stages to pathogens.
PUBLICATION

Sewall, O.K. and  B. Lighthart. 1990.
Standard practice for conducting fungal
pathogenicity tests on the Lepidopteran
egg parasite Trichogramma pretiosum
(Hymenoptera: Trichogrammatidae).
NTIS Acces. # PB90-263849/AS. 40
PP-
                                    202

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              AN OVERVIEW OF PROTOCOL DEVELOPMENT FOR
                       AVIAN PATHOGENICITY TESTS
                       A. Fairbrother1 and P. Buchholz2
                    U.S. Environmental Protection Agency1
                      Environmental Research Laboratory  „
                   ManTech Environmental Technology, Inc.
                            Corvallis, OR 97333
               2
 INTRODUCTION
   Microbial   pest  control   agents
(MPCAs) are microorganisms applied to
agricultural  and   silvicultural
environments to  control proliferation
and dissemination of insect or plant
pests.  During the past four years, we
have been working to develop a set of
standardized protocols that can be used
to  determine the  pathogenicity  of
microorganisms   in  nontarget  avian
species.   The   mallard   (Anas
platyrhynchos)   and   the   northern
bobwhite  (Co/fnus  virgin/anus) were
chosen as the test species  since both
are required by the U.S. Environmental
Protection Agency for  routine  wildlife
toxicity  testing during the registration
process of chemical  pesticides. These
protocols  are used  primarily  by  the
Office  of  Pesticide  Programs  when
prospective registrants propose new
microbial pest control agents {MPCAs).
The protocols are written  such that
could be  used  to determine  patho-
genicity of any microorganism, either
wildtype  or  genetically engineered;
however,  they lack  requirements for
determining potential gene exchange
between the introduced microbe  and
endogenous gut microflora.
    Three  routes  of  exposure  of the
birds to MPCAs  were evaluated: oral,
intravenous  (I.V.),  and  respiratory.
Three  classes of  microorganisms,  a
virus,  fungus,  and  bacterium, were
used to test protocols developed for
oral  and  I.V.  exposure  tests.   A
bacterium and a  virus were  used to
evaluate the respiratory protocol. This
report  reviews our development of the
protocols with emphasis on respiratory
protocols developed during the last year
and  a  half and  a study by  our  col-
laborator, Dr.  Matsumoto, at Oregon
State Universtity to develop a sensitive
method for determining  if birds  are
being  exposed   to  Bacillus
thurfnginensis.

METHODS

Oral and  Intravenous Protocols

    Ten-day-old  chicks and ducklings
were used in  the tests because they
lack full immunocompetency until  20-
25  days  of  age,  and  in  order to
maximize the number of replicates.  The
test microbes used were Autographa
californica nuclear polyhedrosis virus as
the viral agent; Metarrhizium anisopliae
as   the   fungal   agent;  Salmonella
pullorum as the bacterial agent. For the
oral exposure route, birds were given a
single  oral inoculation by  insertion  of a
gavage needle down their esophagus,
and injecting  a  known  volume  and
concentration  of the organism.  The
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 intravenous route is similar,  with  the
 microbial injection inoculated into  the
 right   jugular  vein.     The   dose
 administered to the  birds  was  104
 times the insect  LC50 which is  100
 times the commercial application rate
 and represents a potential worst case
 scenario.  Forty birds were inoculated
 with live  test pathogen, forty with
 inactivated pathogen, and another forty
 with diluent only. Birds were monitored
 for  28  days  post  inoculation with
 weekly  recording  of body   weights,
 weekly collection of blood samples for
 serum chemistry analyses and antibody
 response assays, collection  of fecal
 samples  at  specified   intervals  to
 determine if organism is shed  in the
 feces,   and  histopathological
 examination of  all  tissues   showing
 gross lesions or that are collected from
 birds that die during the experiment.
    Salmonella pullorum is known to be
 pathogenic in poultry and was chosen
 as  a positive  control.   LD50 studies
 were conducted for both species.

 Respiratory Protocol

    In collaboration Dr. Crystal  Driver
 and  colleagues  at  Battelle  Pacific
 Northwest Laboratories,  a head-only
 exposure chamber was developed for
 aerosol exposures of 10-day-old quail.
 An   initial  study was conducted  to
 determine the deposition of particles in
 the  respiratory tract  of  quail after
 intratracheal  instillation  and  aerosol
 exposure of polystyrene beads ranging
 in  size  from  0.4  to 20  microns.
 Subsequent tests  were conducted  to
test the procedures for  either  intra-
tracheal  (I.T.)  or  aerosol exposure
routes.  A  non-motile,  non-pathogenic
 E.  coli was chosen  to  represent the
 bacterial agent.  Fifteen minutes after
 exposure,  birds were euthanized and
 bacterial  reisolation  attempted  from
 nasal   turbinates,   trachea,   lungs,
 syrinxes, and air sacs. A similar study
 was   conducted   with  Autographs
 californica NPV, but technical problems
 with bacterial contamination interfered
 with attempts to reisolate the organism
 from tissues.               --..,-.

 ELISA   Development   For  Mallard
 Antibodies

    A   commercial  suspension   of
 Bacillus  thuringiensis var.  israelensis
 (Bti)  was  purchased  and  the  toxin
 proteins were purified.   Bacteria were
 sonicated and  incubated with trypsin
 and proteinase K to  solubHize  the
 proteins. The supernatant was washed
 twice  with  ammonium  sulfate  and
 purified on a SephadexG150 column.
 Four  protein  peaks  were  produced.
 Peak 4 was purified on DEAE Bio-Gei A,
 and separated  on  SDS-PAGE  electro-
 phoresis into  three  peaks: , A  58
 KiloDalton (KD) peak, and 19 or 20 KD
 peak, and another 19 KD  peak. The 19
 KD peak, previously shown to cause
 hemolysis of mouse erythrocytes/ was
 used for the ELISA and coated onto the
 plates at 0.2 ng/ml concentration.
    Experimental   mallards   were
exposed to Bti by aerosolization.  Birds
were placed inside the carrier crate and
covered  with  a plastic  bag.    The
bacteria was nebulized into the bag for
one hour which resulted  in 145 ml of
the suspension  being used.  Ten birds
received  a   1:10  dilution  of  the
commercial   preparation,  ten   birds
received a 1:100 dilution, and five birds
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served as negative controls. Birds were
allowed to inhale the contaminated air
for 45 minutes. They were exposed six
times at 14-day  intervals with  blood
samples collected every 28 days during
this period.  Antibodies to the Bti toxin
were measured by the  ELISA.

RESULTS  AND DISCUSSION

Oral and Intravenous Protocols

   The   baculovirus,   Autographs
californica  NPV,   and  the  fungus,
Metarrhizium   anisolpiae,   were
nonpathogenic  to the   birds  when
administered orally or  I.V. inoculation.
They caused no detectable alterations
in   the   physiological   parameters
measured.    The  studies   with  S.
pullorum showed that  10-day-old quail
are  very   susceptible  to   pullorum
disease,  with   65-100%  mortality;
however, no mallards died or exhibited
signs of  morbidity.  Blood  chemistry
values and antibody response assays of
the quail were inconclusive due to  high
mortality  rate, small volumes of serum
obtained  and few  birds  that survived
long enough to seroconvert. Mallards
inoculated   orally  had  significant
alterations in serum chemistry values;
antibody  titers were detected as early
as 1  week post  inoculation.  Bacteria
were isolated from ail tissue of quail at
necropsy and  from feces collected  6
days post  inoculation.   Attempts to
isolate the organism from mallard fecal
samples   were    unsuccessful   and
bacteria was isolated only  from  liver
tissue of four birds.   Tissue samples
examined histopathologically showed
the quail to be severely affected. Gross
and histopathological examination of
mallard tissues were normal. Use of S.
pullorum   as   a  positive  control
demonstrated that: 1) the protocols are
written such that lethal and sublethal
effects can be detected; 2) there can
be  significant differences in suscept-
ibility between mallards and bobwhite;
3) S. pullorum  is an effective positive
control for bacterial tests with bobwhite
but not for mallards.

Respiratory Protocol

    Particle  size distribution  in  the
respiratory tract of young quail differed
significantly  between I.T. and aerosol
inoculation methods.   The I.T. route
deposited  almost  twice   as  many
particles  in  the  respiratory  tract
compared to the aerosol exposure, and
particles were much larger in size.   No
particles  greater than  0.8  microns
lodged in any air sacs following aerosol
exposure  whereas  I.T.   instillation
deposited 1O micron  particles  in  the
clavicular air sac and 5 micron particles
in  the   thoracic  air  sacs.    The
intratracheal inoculation method caused
sneezing and coughing by the birds
which resulted in contamination of the
workers and the exterior of the bird.
From these preliminary studies,  it was
concluded that  aerosol exposure is a
safer, more realistic  exposure route
than intratracheal instillation.
 ELISA   Development   for  Mallard
 Antibodies

     Antibody production was observed
 when antibodies were measured against
 the 19 KD  protein but the  test lacked
 the required specificity.  Preinoculation
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 samples and control birds also showed
 positive antibody response.  When an
 ELISA was developed using the 58 KD
 protein as  the antigen, the specificity
 increased significantly. Antibody titers
 decreased  rapidly following  a single
 exposure to Bti.   These experiments
 demonstrate the potential for use of the
 ELISA as a tool  to monitor  sentinel
 mallards exposed  to Bti applications.

 FUTURE STUDIES

    The oral and intravenous exposure
 protocols   require  testing  with  a
 protozoan to determine if  and where
 difficulties  might  arise when working
 with this class  of microorganism.  In
 addition, we plan  to develop a positive
 control system with a protozoan that is
 pathogenic in birds.
    The  respiratory protocol  requires
 further  testing   with  a   pathogenic
 bacterium (e.g.  S. pullorum), a virus,
 fungus,   and protozoan.    Mallards
 should  be   incorporated  into  the
 respiratory tests to determine if there
 are  differences  in particle  deposition
 patternsdueto anatomical/physiological
 differences in respiratory tract.
    Work will continue with the mallard
 ELISA test.  It is anticipated to employ
 this test  with free-ranging  birds  this
 summer  or fall  once  an  area  is
 established where  Bti is being applied.
    The   oral,   intravenous,   and
 respiratory protocols developed test for
 lethality and pathogenicity.  The next
 step is to develop protocols that  test
 whether an MPCA  would cause reprod-
 uctive impairment.  The  studies  will
 include a  chronic feeding  exposure
study, and evaluation of contamination
of embryonated eggs via eggshell pene-
 tration of microorganisms.   Fertility,
 teratogenesis,  hatchabiltiy,  and  egg
 shell quality will  be major endpoints
 measured.

 PUBLICATIONS

 Buchholz> P. and A. Fairbrother. 1991.
 Pathogenesis of Salmonella pullorum in
 northern bobwhite and mallards. Avian
 Diseases (in press).

 Driver, C., L. Smith, J. Briant, P. Van
 Voris, A. Fairbrother, and P. Buchholz.
 1990.   Laboratory  test  methods  of
 exposure  of  microbial  pest  control
 agents by  the respiratory  route  to
 nontarget   avian   species.      U.S.
 Environmental   Protection   Agency,
 Environmental  Research  Laboratory,
 Corvallis, OR. EPA 600/3-90/007.

 Fairbrother, A. and P. Buchholz. 1990.
 Laboratory test methods of exposure by
 oral and intravenous routes of microbial
 pest control agents to nontarget avian
 species.    U.S.  EPA   Environmental
 Research  Laboratory,  Corvallis,  OR.
 EPA 600/3-90/002.

 Fairbrother, A. and P. Buchholz. 1990.
Synthesis   Report:  Laboratory  test
methods for exposure  of  birds  to
microbial pest  control  agents.  U.S.
EPA   Environmental   Research
Laboratory, Corvallis, OR.  EPA 600/3
90/079.
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                   ROUTE SPECIFICITY OF THE TOXICITY OF
     THE Bacillus thuringiensis subsp. israelensis 28 KILODALTOIM PROTEIN

                       C. Y. Kawanishi and M. E. Mayes
                     U.S. Environmental Protection Agency
                       Health Effects Research Laboratory
                     Research Triangle Park, North Carolina
INTRODUCTION

   Repeated human exposure to elevat-
ed  levels of  natural and  genetically
altered   microbial   pesticides  and
biotechnology  agents  increases  the
probability of occurrence of detrimental
human health effects. This is partic-
ularly true of agents that are broadcast
or applied  on a  large scale  in  the
environment. Thus,  agents or  factors
with  such  potential must  be investi-
gated to aid  in the  risk  assessment
process.
   The polypeptides of the parasporal
crystals of Bacillus thuringiensis subsp.
israelensis (Bti) have been  reported to
produce  a variety of undesirable bio-
logical  effects  such  as  cytolysis,
hemolysis, neurotoxicity, etc., as well
as mammalian toxicity. Our previous
studies  implicated the  solubilized 28
kDa   polypeptide  of the   parasporal
crystal as  the cause of   mammalian
effects including toxicity and hemolysis
of  various  mammalian erythrocytes
(Mayes   et  al.,  1989).  Monoclonal
antibody  affinity-chromatography
purified   28  kDa  polypeptide  was
demonstrated to cause a profound and
lethal hypothermia and  bradycardia in
mice  challenged intraperitoneally with
this protein.  The 28 kDa polypeptide
was found to  possess minimal insect-
icidal activity (Held et al., 1986).
    To more fully assess the potential
for detrimental effects  in  humans,
studies  were carried out to  ascertain
the  effects  of different  routes  of
challenge  with the   solubilized  Bti
parasporal crystal 28 kDa polypeptide in
rats.  Additionally,  changes in serum
parameters  after intraperitoneal chal-
lenge with  the  protein mixture were
monitored. Results are presented in this
report.

METHODS

    Rats   were   challenged   intra-
peritoneally (IP) and intratracheally (IT)
as described for mice by Mayes et  al.
(1989)  and Sherwood et al.  (1988),
respectively. Sprague Dawley rats were
dosed intravenously (IV) by injection
into   a   lateral  tail  vein  and  sub-
cutaneously (SC) into the area of the
back.   Peroral  treatment  (PO)  was
accomplished with the use of a gavage
needle.   A range  of  dosages were
utilized  for the'various routes.  Mice
were  observed for  14 days for clinical
signs  and mortality.
    Studies of the effects of solubilized
Bti parasporal  crystal  polypeptides on
blood parameters  were undertaken.
One hundred gram rats were challenged
IP with  an 8 //g/g  mixture  of  crystal
polypeptides and,  two  hours  later,
blood was collected by cardiac
                                     207

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 puncture.  Serum  was  obtained  by
 allowing the blood to clot in Eppendorf
 tubes  for  20 min.  at 0°C and then
 centrifuged. Serum was analyzed with
 a Centrifichem400 automated analyzer.

 RESULTS

     Results showing the effects  of the
 maximum administered dose by various
 routes {Table  1)  indicate that only IP
 injection of rats caused mortality. An IP
injection LD50 of 1.95 //g £?/solublized
parasporal  crystal   protein/g  body
weight   was   estimated  from   the
complete data set.  Preliminary results
with  a  limited  number  of  animals
challenged IV, IT, SC and PO indicate
that  there  was  no  mortality.  SC
injection,  however,  caused  localized
cutaneous  necrosis at  the  site  of
administration.   The   area   of  skin
involved   was   proportional   to   the
magnitude of the dose.
 Table 1. Toxicity to rats of solubilized Bacillus thuringiensis subsp. israelensis crystal
 polypeptides by different routes of administration.
Route of
Administration Maximu
intratracheal 10 p
m Dose Mortality
g/g o/e
intravenous ' 21 Mg/g 0/6
per os
9 M$
3/g o/e
subcutaneous8 9 ^g/g 0/12
intraperitoneal g ^c
]/g 21/39b
a An increased lesion size was correlated with increased dosage.
b LD50 = 1.95 0/17 »g/g (p.0.05).
                                     208

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   The changes  in blood  parameters
indicate  effects  in  numerous organ
systems  (Table 2).  Serum levels  of
albumin,  blood  urea  nitrogen,
creatihine,  creatinine phosphokinase,
glucose;  and phosphate were  signifi-
cantly  elevated in rats challenged  IP
with Bti.  Serum levels of sodium were
sigificantly decreased  in the same rats.
Parameters which were not significantly
affected  were: alkaline  phosphatase,
alanine transaminase, total  bilirubin,
total protein,  amylase,  total  CO2  ,
lactate  dehydrogenase,   albumin/
globulin, anion gap, calcium, potassium,
and chloride.
DISCUSSION

    The  28  kDa  protein of.  the Bti
parasporal  crystal  is  toxic  IP  to  a
number of  vertebrate species (Mayes,
et al., 1990). Consequently, it is all the
more surprising that the present results
indicate that it is lethal to  rats  only
when administered IP. The  estimated
median lethal dose of 1.95y/g/g by this
route is close to that reported for Swiss
Webster mice (2.3 //g/g) (Mayes et al.,
1989).   SC  injection, however,  pro-
duces localized cutaneous necrosis at
the site of  administration. The lack of
effects by the IV, IT and PO routes
Table 2.  Changes of serum chemistry parameters in rats injected intraperitoneally with
solubilized polypeptides from Bacillus thuringiensis subsp. israelensis;

                             Target Organ System
Parameter Heart Kid
ney Pancreas Other
Albumin f
BUN t

Creatinine T
CPK f
Glucose
Phosphate
Sodium

t
t
: : :"." ...".*. . ":
                                     209

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 remain unexplained.  It is interesting to
 note  that   although   Bti  produces
 dramatic  hemolysis  and  cytolysis in
 vitro,   preliminary  evidence  in our
 studies show no measurable hemolysis
 in vivo after IP challenge (Mayes et al.,
 1989).

 FUTURE WORK

    Future work will attempt to explain
 the mechanisms involved  in the  route
 specific toxicity  of Bti.  Factors that
 may effect the activity of the 28 kDa
 protein  when challenged  by routes
 other than IP will be investigated.  This
 should help to clarify the risk to human
 health of the Bti 28 kDa protein.

 PUBLICATIONS

 Held,   G.A.,  Huang,   Y-S.,   and
 Kawanishi,  C.Y.   1986.   Effect of
 removal  of  the  cytolytic  factor of
 Bacillus thuringiensis subsp. israelensis
 on   mosquito  toxicity.     Biochem.
 Biophys.  Res.  Comm.,   141   (3),
 937-941.

 Mayes,  M.E., Held,  G.A.,  Lau, C.,
 Seely, J.C.,  Roe,  R.M.,  Dauterman,
 W.C.  and  Kawanishi,  C.  Y.  1989.
 Characterization  of  the  mammalian
toxicity of the crystal polypeptides of
Bacillus thuringiensis subsp. israelensis.
 Fund. Appl. Toxicol.  13, 310-322.
Mayes,   M.E.,   Kallapur,   V.L.,
Dauterman,  W.C., Roe, R.M., Edens,
F.W.,and   Kawanishi,   C.Y.   1990.
Comparative   toxicology   of   the
delta-endotoxin of Bacillus thuringiensis
subsp. israelensis in the mosquito, rat
and  quail.   Abstract  for   the
Entomological Society of America, New
Orleans, Louisiana, Decembers, 1990.

Sherwood,   R.L.,   Thomas,  P.T.,
Kawanishi, C. Y.  and  Fenters,  J.D.
1988.  Comparison  of  Streptococcus
zooepidemicus  and  influenza  virus
pathogenicity   in   mice   by  three
pulmonary exposure routes. Appl.  and
Environ. Microbiol. 54:1744-1751.

Sherwood,   R.L.,   Byrne,  M.J.,
Kawanishi, C.Y. and Sjoblad, R. 1991.
Comparison  of hemolytic  and  toxic
components of  Bacillus  cereus,  B.
thuringiensis variety israelensis (Bti) and
B. thuringiensis variety kurstaki (Btk).
Abstract for the American Society of
Microbiology, Dallas,  Texas, May 5-9,
1991.
                                    210

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          CLEARANCE OF ENVIRONMENTAL PSEUDOMONADS FROM
               CD-1 MICE FOLLOWING INTRANASAL EXPOSURE

         S. E. George, M. J. Kohan, D. A. Whitehouse, and L. D. Claxton
                     U.S. Environmental Protection Agency
                      Health Effects Research Laboratory
                     Research Triangle Park, North Carolina
INTRODUCTION

   Even  though   environmentally
released microorganisms are considered
safe from a health standpoint, there is
always a potential for the occurrence of
adverse health effects. Exposure may
occur through ingestion, inhalation, or
dermal  contact   with  the  microbial
product.    Previous  research  in  our
laboratory has investigated the ability of
engineered  microorganisms to survive
in the intestinal tract and compete with
the normal flora of mice. Translocation
of these microbes to the spleen  and
liver,  which  can be indicative of a
possible systemic infection,  has also
been  examined.   In these previous
studies,  the  microorganisms  were
introduced by gavage.
   The  current  study  investigates
potential health effects associated with
intranasal (i.n.) exposure to engineered
microorganisms.    Effects  monitored
include  a) morbidity, b) mortality,  and
c)  survival of the dosed strains  in the
lungs,  nasal  washing, and  intestinal
tract.

METHODS

Bacterial Strains  and  Preparation  of
Dosing Suspension

   Overnight  (16-hour)  yeast  ex-
tra ct-trypton e cultures of Pseudomonas
aeruginosa strain ACS69 (Chatterjee et
al., 1982, Mol. Gen. Genet., 188,279)
and P. cepacia strain  AC1100 (Kilbane
et al., 1982, Appl. Environ.  Microbiol.,
44,72) were concentrated in phosphate
buffered  saline  (PBS).   Subsequent
dilutions  were   made  in PBS where
indicated.

Intranasal Exposure

    Fasting (16 hour) strain  CD-1  male
mice were anesthetized with methoxy-
fluorane and dosed i.n. with 50 fj\ of
the mic'robial suspension. At 3 hours,
and 1, 2, 5, 7, 10, and 14 days after
treatment, animals were sacrificed and
the lungs and intestinal tract (small and
large intestines,  cecum) removed  and
placed in buffer. The nasal  cavity was
washed with PBS. Animals and tissues
were concurrently weighed.

Enumeration of Dosed Strains

    Homogenized tissues were diluted
and plated onto Pseudomonas isolation
agar (PIA)  with  (AC869) or without
(AC1100) 50 jjg/ml kanamycin. Plates
were  incubated  at   30 °C  for   48
(AC869)   or  72  (AC1100)  hours.
Colony  forming  units   (CPU)  were
determined and  representative colony
types isolated  for antibiotic sensitivity
determinations  to  insure   source
identification.
                                    211

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Statistical Analysis
Pulmonary Survival
    A two-way analysis of variance was
performed to determine what effect the
treatment  had  on lung  and  animal
weights.   A  value  was  considered
significant if P<0.05.

RESULTS AND DISCUSSION

Morbidity and Mortality

    P. aeruginosa strain AC869 had an
LD50 of 2.7 x  107 CPU.  When mice
were dosed with ~109 CPU  of strain
AC869, mortality occurred within 24 to
36 hours. Slight morbidity occurred in
animals treated with ~107CFU.  These
animals had ruffled fur,  weight loss,
and were  lethargic.   A statistically
significant  (P<0.05)  increase in lung
weights occurred in these animals as
well.  No effects were observed when
mice were administered ~ 103 CPU.
    When mice were treated with ~ 108
CPU of P. cepaciastrain AC1100, slight
morbidity was observed. The  decrease
in body weights was accompanied with
an increase in  lung weights.  Animals
also were lethargic and had ruffled fur
but  recovered  within 5  days  after
treatment.
    The slight  morbidity  observed in
treated  animals   is probably due to
endotoxin    (lipopolysaccharide),  a
component of  gram negative  bacterial
cells.      Mortality   in     strain
AC869-treated   animals   may  occur
through  endotoxin   shock  possibly
coupled with pathogenicity factor(s).
Strain  AC869 also may  have more
endotoxin than strain AC1100.
    Strain  AC869 was detectable at
•high levels in the nasal wash and lungs
of animals treated with ~ 109 CPU prior
to animal death.   When  treated with
~ 107 and  ~ 103 CPU, clearance of the
dosed strain  was dose  dependent.
When administered ~103 CPU, strain
AC869 was not detectable in the lungs
after  3 hours,  but was present in the
nasal washings  intermittently for  14
days. Even though there are no overt
health effects  associated  with  this
dose, the nasal cavity may serve as a
reservoir   for   future  intestinal   or
pulmonary inoculations.  The  ~107
CPU dose was cleared from the lungs
after day 7 but was evident in the nasal
washings at day 14.
 •   Unlike  strain  AC869,  the  ~108
CPU dose of P.- cepacia strain AC1100
was cleared from the  lung after day 7
and from the nasal cavity after day 2.
Even  though  strain  AC1100 is  not
detectable  in the nasal wash, it may
still be present at  numbers below the
detection limit of the method used.

Intestinal Survival

    Strain AC869 was easily  detected
in  the Gl  tract  within  3  hours  of
treatment.  In the mice treated with the
highest-  dose (~109  CPU),  recovery
values remained  constant until  100%
mortality occurred.  At the end of 14
days, strain  AC869 was still detectable
in mice treated with ~ 107 CPU in all 3
sections of the Gl tract. However, the
strain was cleared from the cecum  and
large intestine after 3 hours and never
                                    212

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detected in  the  small  intestine when
mice were administered ~ 103 CPU.
   P.  cepacia  strain  AC1100 was
detectable in all three regions of the Gl
tract at 1  day post treatment, cleared
from the small intestine by day  2, and
not detectable in the cecum and large
intestine after day  2.  When dosed by
gavage, strain AC1100 is not recovered
from the Gl  tract even at  3 hours after
treatment.   When dosed i.n.,  mucus
may coat the bacteria and protect it
from adverse conditions in the stomach
and small intestine.
   The Gl  tract  is  a  reservoir  for
potential pathogens.   Endotoxin can
promote translocation from the Gl tract
to the mesenteric lymph nodes, spleen,
and liver, causing a systemic infection.
The treated  strains may be harbored in
the Gl tract or  alter the normal flora
producing future adverse health effects.

FUTURE WORK

   Intranasal   exposure  to   other
biotechnology   agents   will   be
investigated in endotoxin  sensitive and
resistant mice.  Translocation  to the
mesenteric  lymph  nodes, spleen, and
liver will be determined.  Alterations in
normal   flora   will   be  examined.
Morbidity,   mortality,  clearance,  and
translocation   in    antibiotic-treated
animals  also  will  be  investigated.
Pathogenicity factors associated with
Pseudomonas spp. will be studied.
PUBLICATIONS

George;  S.E.,   M.J,  Kohan,   D.A.
Whitehouse,   J.P.   Creason,   C.Y.
Kawanishi,  R.L.  Sherwood,  and L.D.
Claxton.  Distribution, clearance,  and
mortality  of  environmental
pseudomonads in mice upon  intranasal
exposure. Appl. Environ. Microbiol.,
submitted.
                                     213

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    EFFECT OF ORGANOCHLORINE COMPOUNDS ON THE BIOACTIVATION OF
                  2,6-DINITROTOLUENE IN FISCHER-344 RATS

            S.E. George, R.W. Chadwick, M.J. Kohan, and J.C. Allison
                     U.S. Environmental Protection Agency
                       Health Effects Research Laboratory
                     Research Triangle Park, North Carolina
 INTRODUCTION

    Exposure  to   environmentally
 released  microorganisns  may  occur
 during production, application, or use of
 biotechnology products.  There also is
 a potential for exposure  to the target
 environmental  contaminant  and  its
 microbial  metabolites that arise during
 bioremdiation.     Therefore,   it   is
 important to understand the effects of
 exposure to these hazardous chemicals
 and their metabolites  in  conjunction
 with microorganism use.
    The   intestinal  tract  contains
 enzymes  that  can  transform  pro-
 mutagens and procarcinogens to their
 mutagenic and carcinogenic  metabo-
 lites.   Key enzymes involved  in this
 process  include  nitroreductase and
 p-glucuronidase.  For example,  2,6-
 dinitrotoluene   (DNT),   a   hepato-
 carcinogen,  has   been   reported  to
 require Gl tract bioactivation to express
 genotoxicity.
    In this  study, Fischer 344 rats were
 orally treated with the organo-chlorine
 compounds, pentachlorophenol (PCP),
 2,4,5-trichlorophenoxyacetic    acid
 (2,4,5-T),   or   Aroclor  1254.
 2,6-DNT-induced  urine mutagenicity,
 DNA adduct formation, and key Gl tract
enzymes were assayed to determine if
alterations  in  Gl  tract  flora  and/or
metabolism occur due to  exposure  of
these compounds.
 METHODS

 Animal Exposure

    Weanling  male Fischer  344  rats
 were dosed p.o. with 20 mg/kg PCP,
 54 mg/kg 2,4,5-T, or 25 mg/kg Aroclor
 1254 daily for 5 weeks.  At 1,2,4, and
 5  weeks  of treatment,  rats   were
 administered 75 mg/kg 2,6-DNT  and
 placed into metabolism cages for urine
 collection.
 Urine  ^Collection.   Preparation.
 Mutagenicitv Bioassav
and
    Twenty-four-hour  urine  samples
from animals treated with the peanut oil
vehicle, the organochlorine compound,
2,6-DNT plus peanut oil, or the organo-
chlorine compound plus 2,6-DNT were
collected over dry  ice.    Urine .was
treated  with  p-glucuronidase  and
sulfatase and then concentrated on
C-18 columns. The resulting methanol
extract was dried under N2 and stored
at -80°C until bioassay. Samples were
diluted  in   DMSO  and   tested   for
mutagenicity  in   a .microsuspension
bioassay using Salmonella typhimurium
strain   TA98  without  metabolic
activation.   At week  5,  urine  was
fractionated   by   HPLC  (C-18)  and
mutagenicity   bioassay   performed
directly  on the  fractions  that  were
solvent  exchanged into DMSO.
                                    214

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Gl Tract Enzyme Analysis

    Six  rats   treated   with   the
organochlorine compound and  6 rats
that received the peanut  oil vehicle
were sacrificed at 2 and 4 weeks  of
treatment  and  the  small  intestine,
cecum,  and   large  intestine   were
removed  and  placed  into  reduced
buffer.  The tissues were homogenated
under CO2 and placed into an anaerobic
chamber where aliquots were pipetted
into the reaction vessels.  The reaction
vials were stoppered, removed from the
anaerobic chamber, and 25 //I of the
enzyme substrate mixture (in DMSO,
20  mg/ml  p-nitro.phenyl-p-D-glucur-
onide,3,4-dichloronitrobenzene)added.
The reaction  mixtures were  incubated
for 1 hour at 37°C with gentle mixing.
The  reaction  was  terminated  and
products  (yo-nitrophenol,  p-glucuron-
idase;  3,4-dichloro-aniline,   nitro-
reductase)  extracted, derivatized, and
analyzed.

DNA Adduct Analysis

    DNA was isolated from frozen livers
and digested to mono-nucleotides with
micrococcal endonuclease and spleen
phospho-diesterase.  P1 nuclease was
used to select for adducted nucleotides.
The DNA was labeled with (y-32P) ATP
and  T4 polynucleotide  kinase.   The
labeled DNA adducts were resolved on
PEI-cellulose TLC  plates.

RESULTS AND DISCUSSION

Pentachlbrophenoj Effects

    By  week  4 of PCP treatment,  a
decrease  in   nitroreductase  activity
coincided  with  toxic  urine  in  the
bioassay.    Five  weeks  after  PCP
treatment,  urine again  was collected
and fractionated by HPLC to separate
the   toxic   components   from  the
mutagens.  Animals that received both
2,6-DNT and PCP had more mutagenic
urine   than  animals  that  received
2,6-DNT alone. Peanut oil controls and
PCP  only  treated  rats  voided non-
mutagenic  urine.  PCP potentiated the
formation  of 2,6-DNT-derived  DNA
adducts whereas none were observed
in peanut oil controls or rats treated
with PCP only.

Arochlor 1254 Effects

    2,6-DNT  induced   urine  geno-
toxicity was elevated in Aroclor-treated
rats at 1,2,4, and 5 weeks  after treat-
ment.  -This finding corresponded with
either  a  decrease in small intestinal
nitroreductase activity or an elevation in
small intestinal (J-glucuronidaseactivity.
Again, at week 5, elevated DNA adduct
formation  corresponded with   urine
genotoxicity.

2,4,5-TrichloroDhenoxyacetic   Acid
Effects

    In  contrast  to  PCP and Aroclor
1254  effects,   2,4,5-T  treatment
reduced urine 2,6-DNT-induced geno-
toxicity 1 week after treatment with
levels  returning  to  control  values at
weeks 2 and 4.  No differences in DNA
adducts were observed  after 5  weeks
but  small   intestinal   nitro-reductase
activity- was decreased at week 1  and
p-glucuronidase  activity reduced  at
week 2. Unlike PCP and Aroclor 1254,
2,4,5-T is not an inducer of the hepatic
                                    215

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P450 monooxygenase enzymes. From
our  studies,  it  appears  that  the
following  criteria  must  be  met  to
elevate   2,6-DNT-induced   urine
genotoxicity and hepatic  DNA adduct
formation:   a)  small   intestinal
nitroreductase activity reduced and/or
p-glucuronidase activity elevated, and
b) the compound must induce hepatic
monooxygenase enzymes.

FUTURE WORK

   Future studies include examining the
effects  of  a biodegradative micro-
organism with its target compound  on
the key Gl  tract enzymes.    Similar
studies involving microbial metabolites
of the  compound of interest may  be
included.
   Other   future    work   includes
evaluating the. effect of  cresote and
petroleum   products   on  2,6-DNT-
induced urine genotoxicity, Gl enzymes,
and  hepatic  DNA  adduct formation.
Genotoxic metabolite identification will
be done as well.

PUBLICATIONS

Chadwick, R.E., S.E. George, J. Chang,
M.J. Kohan, J.P. Dekker, J.E.  Long,
and  M.C.  Duffy.  1990.  Comparative
enzyme activity and  activation of the
promutagen 2,6- dinitrotoluene in male
CD-1  mice  and  Fischer 344 rats.
Cancer Lett.  52:133-119.
Chadwick, R.E., S.E. George, J. Chang,
M.J. Kohan,  and J.P. Dekker. 1991.
Potentiation of the genotoxicity of 2,6-
dinitrotoluene   by  pretreatment   of
Fischer  344   rats   with  penta-
chlorohpenol.     Pesticide  Biochem.
Physiol. 39:168-181.

Chadwick,  R.W.,  S.E.   George,   J.
Chang,,M.J. Kohan, J.P. Dekker, J.C.
Allison, J.E. Long, M.C. Duffy,  and L.R.
Forehand. 1991. Some effects of age,
species  difference,  antibiotics  and
toxicant exposure, on intestinal enzyme
activity and   genotoxicity.  Environ.
Toxicol. Chem., submitted.

George,  S.E., R.W.  Chadwick, M.J.
Kohan,   and   J.P.   Dekker.
Pentachlorophenol   effect  on  the
activation  of  2,6-dinitrotoluene   to
genotoxic urinary metabolites  in CD-1
mice:  A comparison  of  Gl  enzyme
activities   and   urine  mutagenicity.
Environ. Molec. Mutagen., submitted.

George,  S.E., R.W. Chadwick,  J.J.
Chang, M.J.  Kohan, J.P.  Dekker, and
Y.   Hayes.  2,4,5-Trichlorophenoxy-
acetic  acid influence  on 2,6-dinitro-
toluene-induced  urine  genotoxicity  in
the  Fischer  344  rat:  Effect on   Gl
microflora and enzyme activity. Fund.
Appl. Toxicol., submitted.
                                    216

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               ENVIRONMENTALLY RELEASED MICROORGANISMS
                        AND THE ENTERIC MICROFLORA

                       W. Dobrogosz,1 Y. Lin,1  M. Fiuzat,1
                          E. George,2 and L. Claxton2
                          Department of Microbiology1
                         North Carolina State University
                             Raleigh, North Carolina
                     U.S. Environmental Protection Agency2
                       Health Effects Research Laboratory
                     Research Triangle Park, North Carolina
 INTRODUCTION

    The release of natural occurring or
 genetically engineered microorganisms
 for  bioremediational  purposes   has
 prompted questions  concerning  their
 effects on the environment and human
 health.  The Health  Effects  Research
 Laboratory (HERL) of the Environmental
 Protection Agency (EPA) is responding
 to  these  questions.   The long  term
 objective of our collaborative research
 in this regard  is  to  determine what
 effects bioremediational microorganisms
 (pseudomonads  in particular) have on
 the "normal microflora." of the mouse.
 If bioremediational microorganisms are
 able to disrupt the "normal microflora"
 to any significant extent,  they could
 compromise  it's   regulatory  and
 protective role  in  the gastrointestinal
 tract,  thereby   enhancing the  host
 animal's susceptibility to an assortment
 of diseases.
    Attempts will be  made to  induce
 pseudomembraneous  colitis  in CD-1
 mice   by   oral   administration   of
 Clostridium  difficile   coincident with
alterning  their   protective  "normal
microflora"  by  antibiotic  tratments.
Using this in vivo model as a guideline,
we  will  determine if the protective
 effects of the "normal microflora" can
 be   altered  or  disrupted   by   oral
 administration  of '   bioremediational
 microorganisms.
    The first phase of this research has
 involved development of appropriate
 analytical systems andmethodologies.
 Described  in  this report are (a)  bur
 attempts to determine if an Oxyrase™
 technology  for anaerobic incubations
 can be use instead of anaerobic hoods,
 (b)  development of culture  media for
 enumeration of  C.   difficle and C.
perfringens   using   the  Oxyrase™
 system,,   and  (c)    isolation   and
 enumeration of Lactobacillus reuteri
 (purportedly a member of the  protective
 "normal microflora")  in the  mouse Gl
tract.

METHODS

Growing,   Enteric  Anaerobes  Using
Oxvrase         '
        ™
    Oxyrase  is  a  partially  purified,
sterilized suspension (0.2 microns or
smaller)  of  a  microbial   (E.  coli)
membrane system that, in the presence
of a suitable hydrogen donor, removes
dissolved  oxygen  rapidly  and  com-
pletely from  aqueous  and  semisolid
                                    217

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environments.    It  is  commercially
availablefrom OXYRASE, INC (Ashland,
OH 44805) and was used according to
the manufacuter's instruction.  Broth
cultures were supplemented with 0.1
unit of Oxyrase  ml"1  and  30  mM
sodium lactate. Agar pour plates were
supplemented with 0.3 units  ml"1 of
Oxyrase, 30 mM lactate and overlayed
with 15-20 ml of 2%  agar to slow
diffusion of oxygen in the air to the
nutrient layer containing the  culture.
For agar spread plates the 2% overlay
agar  contained   0.3  units  ml"1  of
Oxyrase, 30  mM  lactate, and  20 mM
phosphate  buffer at ,pH  of 7.5.   All
incubations were carried out at 37°C in
a conventinal incubator.

Growth of Clostridium difficile (Strains
7698 and 10463) and C. oerfrinaens
(NCTC 8798)

    Stock cultures wee maintained in
Cooked  Meat  Medium  and  inocula
prepared  using   BHI   broth.    C.
perfrlngens  was  enumerated using
Trypticase Soy Cycloserine (TSC) agar
counting black colonies after 8 h at
37°C.   C.  difficile was  enumerated
using TSC  agar supplemented with
Cefoxitin (16 //g ml)  and counting
black colonies after 18 h at 37°C.  C.
perfrlngens does not grow in the latter
medium, and C. difficile colonies do not
appear in 8 h in the former medium.

Determination of Total Anaerobes in the
Mouse Gastrointestinal (Gl) Tract

    Tissue extracts were macerated and
diluted in a sterile gelatin-salts solution.
The preparations  were then spread on
Brucella  Agar  (supplemented  with
sheep blood, vitamin K, and hemihKfor
incubation in the anaerobic hood, or
pour-plated in the same medium when
using the Oxyrase.

Isolation   and   Enumeration   of
Lactobacillus reuteri

    Lactobacillus Selection  (LBS) agar
medium (pH   =  5.4)  was used  to
enumerate the total gut population of
lactobacilli.  The sub-population of L.
reuteri colonies on appropriately diluted
plates  is  idenified and  enumerated
based  on  the ability  of  L.  reuteri
colonies  to  convert  glycerol  under
anaerobic  conditions to  a metabolic
intermediate,   3-hydroxypropion-
aldehyde,   which  has  potent  anti-
microbial activity.

RESULTS AND DISCUSSION

    Results obtained to date indicate
that Oxyrase can be  used  as  an
alternative methodology to  isolate and
grow enteric anaerobes. Pure cultures
of a relatively oxygen-tolerant anaerobe
(C.   perfringens)   and  an  oxygen-
intolerant anaerobe (C. difficile) were
observed  to  grow  in   comparable
manner using either  the   Oxyrase™
method an anaerobic chamber.  This
was observed for both  liquid and agar
cultures.
    Similar results were obtained when
the  two   methods  were  used  to
determine   the   total   number   of
anaerobes  present in  (a)  the  entire
mouse (Gl) tract or (b) the cecum/colon
segments of the tract.  Also, we were
able to determine that  the  CD-1  mice
which will be used  later in this research
have very low background populations
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of   either  C.   difficile-like  or   C.
perfringens-tike bacteria resident in their
Gl tract.
    L. reuteri has been found in the Gl
tract of all animals examined to date.
This species  is  believed to play  an
important role as  a  member  of the
regulatory  and  protective  "normal
microflora."   Our  initial  attempts  to
isolate this  species from  CD-1  mice
have  been   unsuccessful.      An
examination of "pet store  purchased"
mice, on  the other had revealed the
expected complement of L. reuteri ce\\s
in their Gl tract.  These results indicate,
not surprisingly, that rearing methods
can  effect  the  composition  of  an
animals "normal microflora."
FUTURE WORK

    The Oxyrase anaerobic technology
will be applied toward  accomplishing
the goals of  the  proposed  research
which are:
(a)  to develop an in vivo model disease
system, (i) to  study  and  monitor  the
protective   role   of   the   "normal
microflora" against disease, and  (ii)'to.
evaluate  the  influence  of  environ-
mentally  released  microorganisms on
this protective role.
(b)  to determine if an  in vitro model
can be developed  and used for  these
purposes.
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  In Vitro SURVIVAL AND COMPETITION OF ENVIRONMENTAL PSEUDOMONADS
                   WITHIN A HUMAN FECAL FLORA CULTURE

                 G.M. Nelson1, L.D. Claxton2, and S.E. George2
                 Environmental Health Research and Testing, Inc.1
                     U.S. Environmental Protection Agency2
                       Health Effects Research Laboratory
                     Research Triangle Park, North Carolina
 INTRODUCTION

    Investigation  of potential  adverse
 health effects due to the environmental
 release of microorganisms is  needed.
 Previous  work in this laboratory  has
 examined the colonization and competi-
 tion   potentials    of   environmental
 pseudomonads with the intestinal flora
 of CD-1 mice both in vivo and in vitro.
 The current research  is  a  comple-
 mentary study  investigating the ability
 of some of these same Pseudomonas
 strains  to  survive  and  compete  in
 culture with human fecal microbiota.

 METHODS

    Veal infusion  broth  enriched with
 yeast extract, vitamin K1, and hemin
 served as the in vitro culture medium.
 Human fecal   flora,  obtained  using
 Anaerobic  Culturette  Collection and
Transport Systems  (Becton Dickinson
 Microbiology  Systems), were grown
overnight in enriched veal infusion broth
and  used  as  the  culture  inoculum.
Pseudomonas aeruginosa strains BC16
 (isolated  from  a  product  for  PCB-
degradation), PAMG (a mouse intestinal
isolate), and AC869 (a 3,5-dichloro-
benzoate   degrader)  were  grown
overnight   in  yeast extract-tryptone
broth and  concentrated in  VPI  dilution
buffer for culture inoculation. A 0.1 ml
 aliquot of one competitor strain  was
 used to inoculate the culture.
     At 12 hours, a 0.06 ml aliquot of
 the culture was removed and used to
 inoculate a  new culture.  The  new
 culture was incubated for 1.2 hours and
 the process repeated for a total of 4
 transfers (5 cultures). The competitor
 strain and fecal microbiota populations
 from the  first, third, and fifth cultures
 were enumerated by spread-plating 0.1
 ml  of  the  appropriate  dilutions onto
 selective  media at pre-selected time
 intervals.

. RESULTS AND DISCUSSION

     All three competitor strains grew
 anaerobically in pure culture to a level
 of  108-109  organisms  per milliliter.
 Although   Pseudomonas  spp.   are
 aerobes,  they can  use   nitrate as  a
 terminal electron acceptor.  In  culture
 with human fecal flora, all three strains
 declined  in  number,  but were  still
 present in low numbers  in culture 5.
 P.  aeruginosa AC869 appeared to be
 the best survivor of the strains tested.
 Survival of the competitor strains was
 comparable in culture with human or
 mouse flora.
     Differences between the  human
 and mouse  enumerated flora   popu-
 lations were minimal. The total aerobic
 and anaerobic counts were higher in
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mouse  fecal  cultures  than  in  the
corresponding human fecal cultures for
the 6 hour timepoint, perhaps indicating
faster growth of the mouse cultures.
    No effect of strain addition on the
human  fecal  flora  populations was
detected for  strain BC16.   Statistical
tests have not yet been completed to
determine if strains PAMG or AC869
alter  the   balance  of   the   flora
populations.

FUTURE WORK

    Flora   populations   and  enzyme
activities for single-stage, 2-stage, and
2-stage with cell-recycling continuous
cultures will be compared to determine
which most closely mimics the in vivo
situation.  Survival and competition of
environmentally relevant  strains will
then be tested  in continuous culture.
Rodent and human in vitro results then
can be combined with rodent in vivo
results in a parallelogram approach for
extrapolation of potential human health
effects,

PUBLICATIONS

Nelson,  G.M.,  L.D.  Claxton,  J.P.
Creason, and  S.E. George. 1991. A
toxicological screen  to determine in
vitro  survival  and   competition  of
environmentally applied pseudomonads
with   mouse   fecal  microbiota.
Environmental   Toxicology  and
Chemistry, 10:597-608.
                                   221

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                                 SESSION III
                         RISK  CONTROL RESEARCH

 Decontamination and Mitigation

    Investigators are exploring a variety of risk reduction strategies,  including the
 development of genetically altered strains for conditional lethal control of survival and
 gene exchange and procedures  for physically and chemically decontaminating field
 sites. These procedures will be evaluated under a variety of environmental conditions.

 Field Releases

    ORD  scientists  develop  criteria for evaluating  containment  and  monitoring
 strategies.  Test  methods are based on the actual application of recombinant and
 surrogate organisms to specially designed experimental  release sites representing
 different environmental habitats.  Methods for detection,  monitoring distribution,
 dispersal,  and   dislodging  characteristics,   and  for  proper  containment   of
 microorganisms during field releases are being evaluated.

 Process Containment

   Researchers produce the appropriate scientific information to develop protocols for
 evaluating the causes of emissions  from  biological  process facilities, and control and
 reduction of risks associated with these emissions.  Engineering and cost models for
 assessing risks  associated  with  the manufacture,  and  use in manufacture of
 genetically engineered  microorganisms in large-scale fermentation facilities will be
 produced.    Issues  dealing  with  process equipment  design,  decontamination
technology,  worker exposure  and protection, and loss prevention techniques will be
considered.                        ,
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        THE USE OF LETHAL BACTERIAL GENES TO LIMIT THE SURVIVAL
  OF INTENTIONALLY RELEASED GENETICALLY ENGINEERED MICROORGANISMS

                    Wade H. Jeffrey1, Stephen M.  Cuskey2,
                    Richard B. Coffin2, and Michael Reagin1
                        Technical Resources, Inc.1 and
                    U.S. Environmental Protection  Agency2
                      Environmental Research Laboratory
                             Gulf Breeze, Florida
 INTRODUCTION

   Concerns  about  the  release  of
genetically engineered microorganisms
to the environment may be assuaged if
the released strains contain a mechan-
ism whereby they may be killed after
completion of their appointed task(s).
This mechanism  should,  ideally,  be
contained  within  the  organism and
should not hinder the survival of the
released strain prior to the application
of the lethal  signal, i.e.  the  strains
should have  conditional lethality.  A
potentially   lethal  gene   putatively
involved with plasmid maintenance, kilA
from plasmid RK2, has  had  its regu-
lation altered  so  that  expression  is
dependent on the presence of an added
compound.    The  native  upstream
regulatory  sequences were  removed
from kilA  and replaced with the TOL
plasmid   lower   pathway  operator-
promoter OP2 to form plasmid pEPA88.
Cells containing  this  plasmid  and  an
appropriate regulatory gene should  be
killed in the presence  of an added OP2
inducer such as benzoic acid.
   This report contains the results  of
tests  on   Pseudomonas  aeruginosa
PA01 containing pEPA88.  Effects on
growth,  lethality, and the previously
uncharacterized mechanism of action of
kilA were examined.
 METHODS

   P. aeruginosa  PA01   was  trans-
 formed with pE.PA88 and incubated in
 a minimal salts medium in the presence
 and  absence  of  benzoate.   Several
 parameters were  examined  including
 increases in  culture  turbidity,  cell
 survival (CPU's), 3H-glutamate uptake,
 and the differences in these parameters
 in different ceil  size  fractions.   A
 second lethal plasmid was constructed
 which placed kilA  down stream  of the
 inducible tac promoter (pWHJ63). This
 plasmid  was transformed  into  E.  coli
 JM109 and the effect of kilA expres-
 sion on DNA synthesis was determined.

 RESULTS

   Microscopic examination of  PA01
 (pEPA88)  cultures   incubated   in
 benzoate minimal medium showed two
 distinct  morphological   forms.
 Numerous long, filamentous cells were
 observed  as  well  as  a significant
 number of normal size cells. Filaments
 appeared after a 2 hour incubation with
 benzoate  and  remained until  the
termination of  the  experiments  at 24
 hours.  Filaments  were not formed in
samples   which   did  not   contain
 benzoate. Filaments could be separated
from single cells by differential filtration
                                   225

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through   3   ;/m   pore-size   filters.
Microautoradiography of cells labelled
with   3H-glutamate  indicated   that
filaments  were  metabolically active.
After 4 hours, 5 % of the biomass (jjg
protein/ml) was  in the greater than 3
//m fraction (filaments). By 8 hours and
12  hours,  60%  and  70%  of . the
biomass,. respectively,   was  in  the
greater than 3 //m fraction.   By 24
hours,   50%  of  the  biomass  was
filaments. At 4 hours, 17% of the 3H-
glutamate was taken up by the greater
than 3 //m fraction.  At 8 hours, 45%
of the uptake was by filaments. By 12
hours,  87% of the uptake was in the
larger fraction and 83% was taken up
by filaments after 24 hours exposure to
benzoate.   Biomass specific rates of
glutamate uptake  were 3  to 5  times
greater for filaments at all time points.
In  contrast  to  metabolic  activity
measurements,  all   of   the   colony
forming organisms were found to be in
the  less than  3 /jm fraction.   This
indicates that while the filaments were
more active, they were incapable of cell
division   and   subsequent  colony
formation.
   Filaments and single cells were also
observed  in E.  coli  cells containing
pWHJ63  after  induction  of kilA by
IPTG.  Filaments began forming within
an hour.   Microscopic  examination
indicated that the number of filaments
remained  constant  after  a  2  hour
exposure  to IPTG.  3H-Thymidine in-
corporation by these cells indicated that
filaments continued to synthesize DNA.

DISCUSSION

The  use of kilA has proven inapprop-
riate as a biological control  agent for
Pseudomonas aeruginosa PA01.  Two
types of response have been observed
in cells containing induced kilA genes.
The   first   is   a   biostatic   effect
characterized by normal  sized  cells
which,  while  not  actively  growing,
remain  metabolically  active.     The
addition of an alternative carbon source
provides  growth substrate and these
cells begin to grow and divide and will
form colonies when plated on nutrient
agar.   The second  lethal  response is
noted  by  the  production  of  long
filamentous  cells.   Metabolic  activity
remains very high in these  cells, yet
these cells  do  not grow and  form
colonies  when  presented with  alter-
native growth substrates.   The lethal
mechanism  of  kilA  has  not  been
previously characterized.  Our results
indicate that lethality is not caused by
an  inhibition  of  protein  nor  DNA
synthesis. The production of filaments
and the inability  of them to divide and
form   colonies  indicates  that   kilA
functions by inhibition of daughter cell
separation.  The reasons for the two
types  of  response  are  not  known.
Plasmid minipreps  indicate that those
cells  which  do  grow maintain intact
pEPA88 plasmids so that loss of the
plasmid construct cannot  account for
loss of activity.

PUBLICATIONS

Cuskey,  S.M.,  and  A.B.  Sprenkle.
1988.  Benzoate dependent induction
from the OP2 operator-promoter region
of the TOL plasmid  (pWWO) in the
absence of known regulatory genes.  J.
Bacteriol. 170: 3742-3746.
                                     226

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Cuskey, S.M.  1990.  Lethal genes in
biological  containment   of  released
microorganisms,  in: M.J. Day and J.C.
Fry (eds.), Environmental Release of
Genetically  Engineered   and   Other
Microorganisms.
Publishers.
Edward   Arnold
Cuskey,  S.M.    1990.    Biological
containment of genetically engineered
microorganisms.   in:  M.  Levin,  R.
Seidler, and P.  Pritchard (eds.), Guide
to  Environmental   Microbiology.
McGraw-Hill
Jeffrey,  W.J.,  S.M.  Cuskey,  R.B.
Coffin, and  M.  Reagin.   Conditional
expression   of  RK2  kilA  gene  in
Pseudomonas aeruginosa can cause a
non-lethal inhibition of growth. To be
submitted to J. Bacteriol.

Jeffrey,  W.J.,  S.M.  Cuskey,  P.J.
Chapman, and  R.H. Olsen. Cloning and
characterization of Pseudomonas genes
involved   in   benzoate   catabolism:
Isolation  of   a   chromosomal   DNA
fragment able to  substitute for xylS in
the  activation  of  the  TOL  lower
pathway promoter. To be submitted to
J. Bacteriol.
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          FIELD RELEASE OF A GENETICALLY-ALTERED BACULOVIRUS
                     WITH A LIMITED SURVIVAL CAPACITY
                      H. Alan Wood and Patrick R. Hughes
                           Biological Control Program
                           Boyce Thompson Institute
                               Ithaca, New York
INTRODUCTION
   Although baculovirus epizootics are
often the major factor in natural control
of lepidopterous insects, there has been
limited commercial usage of these viral
insecticides.    One  of the  primary
problems  with  using  baculoviruses as
microbial  pesticides   is  that target
insects  are  not  killed  quickly  and
continue to damage crops for 3 to  10
days.   To  overcome this   problem
baculoviruses can now be genetically
engineered    to  enhance   their
pathogenicity.    New genes  can  be
inserted into  viral genomes which will
effect a fast death.
   The  insertion  of  new  genes into
baculoviruses is technically easy, and
many commercial and  academic labora-
tories  are  currently   conducting
programs  aimed   at   producing
genetically-enhanced  viral  pesticides.
However, a major problem which must
be addressed prior to the release  of
these products  is  the environmental
consequences,   e.g.,   persistence,
spread,  genetic stability, gene transfer
potential,  and displacement of natural
virus  populations  (Wood,  in press;
Wood and Granados, • in  press - see
publications list).  In order to  address
these issues, we have developed and
tested a release strategy by which the
engineered baculovirus pesticides could
have enhanced pathogenicity  but low
survival and displacement potential in
nature.
   The strategy for this construction is
to  delete  the 'polyhedrin gene and
replace it with a foreign  gene whose
product  will  enhance  pathogenicity.
The polyhedrin gene product is required
for occlusion  of virus particles within
polyhedra and is therefore essential for
survival  and  transmission .in nature.
Therefore, the infectivity of the altered
virus  is stabilized  by occlusion within
polyhedra produced by wild-type virus.
This is accomplished by co-infection of
cells  with  both  the  wild-type and
altered  virus.   Late in the replication
cycle, the polyhedrin produced by the
wild-type  virus  occludes  both  virus
types. Persistence of the  altered virus
in the virus population is determined by
subsequent levels  of co-infection and
co-occlusion.
   This field release project  is  being
conducted to test our laboratory  based
model under natural conditions.  The
release was conducted with an altered
Autographs   californica   nuclear
polyhedrosis virus  (AcMNPV) which
had a deleted polyhedrin gene but no
foreign gene inserted. The altered virus
was co-occluded  with  the wild-type
AcMNPV.   Investigations are  being
performed  to  evaluate the spread of
virus in nature and the survival potential
of the altered virus  within the wild-type
virus population.
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METHODS AND RESULTS

   During the field application phase of
this project (1989), three applications
of polyhedra containing 49% genetic-
ally-altered  and  51%  wild-type
Autographs   californica   nuclear
polyhedrosis virus (AcMNPV) particles
were made on a quarter  acre cabbage
plot  in  Geneva,  NY.   The  cabbage
plants  were seeded with  a total  of
4500 Trichoplusia ni (cabbage looper)
larvae which were all infected and killed
by the virus. Polyhedra samples were
purified from,individual larvae. The viral
DNA was purified,  digested  with the
restriction enzyme £coR1 and fraction-
ated, by  agarose gel  electrophoresis.
Based on molar ratios  of the £coR1 I
fragments (altered virus genome has a
smaller I  fragment), it was determined
that  the  progeny virus  produced  in
these larvae contained 42% genetically-
altered  virus.    In  addition,  it  was
estimated that approximately  1 x 1014
progeny  polyhedra  were deposited  in
the field.
   The questions asked in 1990 were:
1) how much of biologically-active virus
(which contained 42% altered virus)
was present in the soil  in the  spring  of
1990,  2)  with  this as  the starting
inoculum how much occluded, altered
virus would be produced and 3) what.
was the distribution of virus in the field
plot.  The  two approaches  taken  to
answer   these   questionswere  soil
sampling and larval seeding.
   In 1990, the test site was replanted
with cabbage in precisely the  same
location and manner as in 1989.  The
cabbage  plants were seeded with T. ni
larvae  4  times  during  the  growing
season.  Several thousand larvae were
inspected in the field without evidence
of viral  infections.  When half of the
larvae started to  pupate,  a total of
1600 larvae were brought into the
laboratory  and  examined   for  virus
infections.   Less than 1 % of the test
larvae became infected, indicating that
only a small amount of virus in the soil
was deposited on the plant tissues.
   As a second evaluation  procedure,
soil samples were taken from the same
97  locations in the application area as
were sampled in  the spring of  1989
prior to the  release.   In  order to
quantitate the amount of virus in the
test site, polyhedra were extracted from
the soil samples using a combination of
differential centrifugation and detergent
treatments.  The amount of biologically
active virus was determined by feeding
one-gram soil  extracts to each of 30
larvae   from  each   location   (2910
samples).  Based  on control data, the
soil  extraction efficiency  was  25%.
Therefore each  insect  was fed the
amount  of virus present  in 0.25 grams
of soil.  The percent death from these
bioassays was used to  determine the
number  of biologically-active polyhedra
per gram of soil.
   Based  on  infectivity of the soil
extracts,  the  average  number  of
polyhedra detected in soil samples from
the quarter-acre application area was
1219 _+_ 2269 polyhedra per gram of
soil. The average number of polyhedra
in the soil samples from the buffer zone
(1.75   acre   area   surrounding  the
application area) was 274 +_ 404. The
high standard deviations are the  result
of high  variation between samples not
within replicated samples.
   Based on laboratory studies, it was
predicted that the biological stability of
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polyhedra  containing  wild-type  arid
altered  virions  would   be  equal  to
polyhedra containing only the wild-type
virus.   In order to evaluate this,  soil
samples were taken from a control  plot
which had been treated identically in all
respects to the application site except
that the polyhedral  inocula contained
only wild-type  virions.   The  average
number of polyhedra in this control  site
in the  spring of 1990 was 1151  +_
1942 polyhedra  per gram of soil. This
value is very close to the number of
active    polyhedra  found   in   the
application  area.    It  is,  therefore,
concluded that  occlusion of  altered
virions does  not effect the stability of
polyhedra  under  laboratory  or field
conditions.
   Having established the level of active
virus deposition in the  field  and  the .
percentage of altered virus  (based on
introduction  data),  we   are  now
determining  how  much  altered virus
replication has resulted from this level
and  type of  inoculum.   To this end,
infected larvae from the quantitation
procedure discussed above are being
further analyzed.  The polyhedra from
the individual larvae have been purified
and the percentage of occluded, altered
virus is  being  determined by DNA
restriction  analysis.   As  previously
demonstrated,  the  concentration  of
inocula  as well as the percent altered
virus present in  the  inocula determine
the amount of altered virus in  progeny
polyhedra. It is  considered that larvae
feeding on  leaf tissue  contaminated
with soil would  ingest less than 0.25
grams   of soil,   and  therefore  the
inoculum  level  in  this  test  can  be
considered high in  terms of  natural
contamination processes.
   We  are  now  in  the  process  of
completing our analysis of the amount
of altered virus present in the polyhedra
isolated  from  the  individual  larval
samples.  The present data show that
approximately   one-third   of   the
polyhedra samples contain altered virus.
The range in concentration is 12-57%
altered  virus.     The  data  set  is
incomplete  at  present, however  the
average amount  of occluded, altered
virus is approximately 20%.

FUTURE INVESTIGATIONS

Questions for 1991:

A. How   many   biologically-active
   polyhedra remain  in the test site
   after one year?

B. Using soil extracts  as inocula, what
   quantity  of altered  virus  will   be
   occluded in progeny polyhedra?

   The original prediction was that from
1990 to  1991  there  would  be a
decrease in both values.  If a decrease
occurs,,  then   the   co-infection/co-
occlusion  process used in  this  study
would  be  of  use to  ensure that a
released engineered baculovirus would
slowly be removed from natural virus
populations(nonpersistent). According-
ly,  the  engineered virus  could  not
displace natural virus populations based
on its competitive disadvantage.
   If the number of biologically-active
polyhedra do not decline between 1990
and 1991 and the amount of occluded,
altered virus remains  the  same  as in
1990,   the   co-infection/co-occlusion
strategy does not provide the environ-
mental  attributes  for  which  it  was
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designed.     Accordingly,   new
approaches will  be needed to ensure
environmental safety following releases
of genetically-enhanced viral pesticides.
In the absence of  biological  contain-
ment,  engineered  baculoviruses may
pose a multitude of potential problems.
   In the spring of 1991, we will again
take soil samples from the same 97 test
site locations as used  in  1989 and
'1990.   As performed in  1990, thirty
larvae  will  be fed  with extracts  from
one-gram soil samples (1/4 gram based
on extraction efficiency) from each site.
Based  on  the percent  infection  of
larvae, the concentration of biologically-
active polyhedra will be determined and
compared with the 1990 data.
   The dead larvae from the soil assays
will then be processed to determine the
amount  of occluded,   altered  virus
present in  polyhedra.  The polyhedra
from each larva will be  processed and
analyzed  separately.    The  percent
altered virus and standard deviation will
be compared with the 1990 data.
PUBLICATIONS

Wood, H.A., P.R. Hughes, N. van Beek
and M. Hamblin. 1990. An ecologically
acceptable strategy  for  the  use  of
genetically  engineered   baculovirus
pesticides, in: A.B. Borkovec and E.P.
Master (eds.),  Insect Neurochemistry
and Neurophysiology  1989.  Humana
Press, New Jersey, p. 285.

Hamblin, M., N.A.M. van Beek, P.R.
Hughes and H.A. Wood.  1990. Co-
occlusion   and   persistence  of   a
baculovirus  mutant   lacking  the
pblyhedrin gene. Applied and Environ.
Microbiol.  56:3057-3062.

Wood, H.A. and R.R. Granados. 1991.
Genetically engineered baculoviruses as
agents for pest control.   Ann.  Rev.
Microbiol.  45:69-87.

Wood,  H.A.      Development   of
genetically-enhanced  faaculovirus
pesticides, in: K.M. Maramorosh (ed,),
Biotechnology for Biological Control  of
Pests  and  Vectors. CRC  Press,  Boca
Raton,  FL.  In press.
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          EPIPHYTIC FITNESS GENES AND PHENOTYPIC ADAPTATION
                               Steven E. Lindow
                         Department of Plant Pathology
                            University of California
                              Berkeley, California
INTRODUCTION
   The  fate and  effects of  micro-
organisms  introduced  into  the open
environment are usually predicted from
an understanding of their biology and
ecology   studied   under   controlled
conditions, usually in culture.  Studies
we have made of introduced recombi-
nant  bacteria reintroduced  into  field
plots indicated that the strains we used
exhibited    considerable    phenotypic
plasticity. That is, the behavior of Ice"
Pseudomonassyringae strains grew and
survived  with  different efficiencies
depending  on  the  conditions  under
which  they  had  originated.    For
example, we had observed that cells
harvested from broth cultures were less
capable of surviving on leaves surfaces
after inoculation than were cells grown
under solid agar conditions.  If we are
to accurately  predict the growth and
survival  of  bacteria  under  natural
conditions we must be  aware of the
factors  such as  phenotypic plasticity
which may effect the behavior of cells
while in nature.  Similarly, we need to
know more of the nature of the habitats
occupied by bacteria after introduction
into  the open environment  such  as
plants since  the growth and survival of
the introduced bacteria will be dictated
by how well their  adaptations match
the physical and chemical environment
presented to them  by  the plant. We
have  therefore   initiated studies  to
determine the range of differences in
epiphytic fitness exhibited  by a given
genotype of bacterium that is exposed
to  different  environments  prior  to
inoculation onto plant surfaces.  Since
bacteria that occupy  specific  habitats
such as leaf surfaces are rather unique
and exhibit superior ability to grow and
survive in such habitats we  expect that
they posses  unique traits  that allow
them to exploit such habitats.  Little is
known of the fitness traits that bacteria
employ to exploit natural habitats such
as leaf surfaces.  If such fitness traits
were   better known,   it  should be
possible to better define the range of
habitats that an introduced bacterium
would colonize based on its  possession
such attributes.  Similarly, it should be
possible  to  reduce  the  fitness  of
introduced  organisms by  eliminating
habitat-specific fitness traits.  We have
therefore initiated studies to determine
novel   fitness  determinants  in  the
epiphytic   bacterium,   Pseudomonas
syringae.

METHODS

Phenotypic Plasticity and
Nature of Epiphytic  Habitats

  ' Pseudomonas syringae strain MF714
was grown for 12 hours either in Kings
Medium B (kB)  broth  or agar at 28°C
and harvested or inoculated onto bean
plants 24 hours prior to use.  Bacterial
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cells were removed from the surface of
plant by sonication  for 5  minutes in
distilled water and  concentrated  by
centrifugation.    Bacterial  cells form
each of these sources were suspended
in distilled water at a concentration of
107  cells/ml and sprayed  onto bean
plants under field conditions  or  in a
growth   chamber   with   different
temperature   and  relative  humidity
conditions.  Bacterial populations were
determined every 2 to 8 hours for up to
4  days  after  inoculation.    Viable
bacterial populations were  determined
by dilution plating onto KB containing
100 /yg/ml rifampicin.  The proportion
of the viable cells that were culturable
was determined by microscopic exam-
ination of cells  recovered form  leaf
surfaces at different times that were
exposed for 12 hours to solutions of
dilute yeast extract containing 50 //g/ml
nalidixic acid.  The relative exposure of
bacteria of different genotypes and of
cells of a given genotypes  exposed to
different environmental conditions on
leaf   surfaces  was  determine   by
quantifying the ratio of cells surviving
exposure to a topical treatment of 15%
hydrogen peroxide or exposed to a flux
of UV irradiation of 2000 erg/mm2.

Determination  of   Epiphytic   Fitness
Determinants

   Erwinia herbicola strain  BRT89  was
subjected  to  transposon mutagenesis
with  Tn5 delivered on the  plasmid
PDGS61 for which  negative selection
could  be  imposed.    This  plasmid
consisted  on  pLAFRS containing  Tn5
and  the  levan  sucrase  gene  from
Bacillus subtilis.  Strains containing the
levan sucrase gene are  killed when
grown  on   media   containing  5%
sucrose.    Tn-5  mutants were then
inoculated   onto   bean   plants  and
subjected  to  alternating periods  of
wetness   and  drying.     The   final
population size of individual mutants on
leaves after  73  hours  of alternating
environmental conditions was estimated
by  a  measurement  of  the freezing
temperature of individual  leaves cooled
slowly after immersion in tubes of ice
nucleus-free  water.   The relationship
between population size of lce+ strain
BRT98  and  freezing temperature  of
leaves  was  determined.    Genomic
regions of Pseudomonas syringae strain
B728a identified  by insertion of Tn-5
that  confer epiphytic   fitness  were
identified in a library of genomic clones
of this strain using the flanking regions
of cloned DNA  containing Tn-5 from
the mutants.  Subcloning experiments
on the genomic clones were followed
by   complementation    studies   to
determine the minimum  size of  DNA
necessary to confer the stress tolerance
phenotype   identified   using   these
mutants.

RESULTS AND DISCUSSION

   Pseudomonas  syringae   strains
differed greatly in their behavior  after
application  onto  leaf surfaces as  a
function of the physiological condition
of the cells prior to inoculation.  Cells
harvested  from  broth   cultures  died
much more rapidly upon application to
plant surfaces than cells harvested from
agar  plates.  Broth-grown   cells also
required up to twice as long to  resume
growth  after   inoculation   on   leaf
surfaces.    In  contrast, cells of  P.
syringae that were recovered from the
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 surface of bean leaves exhibited up to
 1000-fold  greater survival than  cells
 harvested  from   broth  culture  after
 application to leaves.  Cells recovered
 from  leaves  also  resumed  growth on
 leaf surfaces as much as twice  as fast
 as  even  cells  harvested  from  agar
 surfaces.   These  results indicate that
 bacteria exhibit considerablephenotypic
 plasticity.  It is  likely the  pattern  of
 gene  expression  in  cells  in   natural
 habitats  such  as leaves  are  much
 different   than   that   in   culture.
 Predictions of microbial behavior upon
 field releases must take into account
 the great deal of phenotypic plasticity
 that microbes appear to possess.
    Bacterial survival in natural habitats
 such  as  leaf surfaces appears  to be
 greatly influenced by their modification
 of the habitat.  The fraction of cells of
 P.   syringae  that  survived  on  leaf
 surfaces  was directly "proportional to
 the  concentration of  cells initially
 applied to the leaf. While up to 10% of
 the cells applied as a suspension  of 109
 cells/ml survived  during the  first 48
 hours  after inoculation on leaves, only
 about  0.005 of the cells applied as a
 suspension of  103  cells/ml survived
 during this period.  Application of  109
 dead cells in conjunction with 103 live
 cells increased the survival of the  103
 live  cells to about 10%.  The rate of
 immigration of cells from sites of field
 releases thus could greatly  effect the
 efficiency with which they survive the
 transport from the field site and become
 established in an adjacent site.
   The  natural  habitats  of similar
 bacteria appear  to differ  greatly.  For
 example, the ability of cells of different
P.    syringae   strains   to   become
established  in  "protected  sites"  on
 leaves  differed greatly and  differed
 greatly form other phylloplane residents
 such    as   Erwinia   herbicola   and
 Xanthomonas maltiphilia. The  fraction
 of P.  syringae cells that could not be
 killed  by surface sterilization of leaves
 ranged from less than 0.2% to as much
 as 24% of the total bacterial microflora
 on  a   leaf.    Most  non-pathogenic
 bacterial species such as E.  herbicola
 and X. maltiphilia did not exhibit more
 than 1 % of the cells in such "protected
 sites".   These great  differences in
 habitat preference are unexpected and
 indicate that such preference should be
 carefully established  for recombinant
 organisms to be released into the open
 environment for which a prediction of
 survival  and subsequent behavior is
 desired.  The eradication  of'bacteria
 having   greatly    different    habitat
 preferences also may differ  greatly  and
 should be further investigated.
    A novel leaf freezing assay is being
 used to  assess the epiphytic fitness of
 Tn-5 -induced mutants of E. herbicola.
 Bean plants  are being inoculated with
 individual transposon  mutants  of  this
 species  and subjected  to  alternating
 period of wetness  and dryness.  The
 final population  size  of  the  lce+  E
 herbicola strain used  in this  study is
 proportional to the freezing temperature
 of  leaves colonized  by this  strain.
 Mutants deficient in their ability to grow
 or survive on leaf surfaces are being
 identified  by   a  reduction  in   the
 temperature at which leaves colonized
 by such a mutant strain freeze.
   Transposon mutants of P.  syringae
 which  exhibit  a  reduced  ability  to
survive the stresses of drying on leaf
surfaces have been  identified and  are
being characterized.  Most of  these
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mutants do not exhibit a deficiency in
any phenotype such as osmosensitivity
that  can  be associated  with  stress
tolerance.  The genomic determinants
of one such mutant that conditions the
ability to survive on  leaves as surface
moisture drys from the leaf has been
localized to a  6.5kb region. This region
will compliment the reduced survival
exhibited by the mutant strain. Fusions
of this gene  with  a promoterless ice
nucleation  gene are being  made  to
further localize the position and size of
the genes involved in stress tolerance
and to permit the assessment of  the
transcriptional activity of this  region
under  natural conditions and  under
conditions  of specific  environmental
stresses.  The identification of such
novel stress-tolerance  genes should
permit  their   identification  in   other
bacterial to be released into the open
environment  and thus to indicate their
potential for  tolerating the stresses at
field  sites;  Alternatively, such genes
could  be specifically inactivated  to
biologically contains  organisms at field
sites  by making  them  incapable  of
surviving  specific   environmental
stresses. ,

PUBLICATIONS

Kinkel, L.L.,  and  S.E.  Lindow.  1989.
The role  of competitive interactions in
bacterial survival and establishment on
the leaf  surface,  pp.634-638.  in:  T.
Hattori, Y.  Ishida,  Y. Maruyama,  R.Y.
Morika, and A. Uchida, (eds.), Recent
Advances in  Microbial Ecology. Japan
Sci. Soc. Press.
Lindow, S. E. 1990. Design and results
of  field   trials  of  Ice" recombinant
Pseudomonas syringae strains, pp. 61-
69.   In:  J.  Marois  and J.  Bruhning
(eds.), Risk Assessment in Agricultural
Biotechnology:   Proceedings  of  the
International  Conference.  U. of  CA.,
Oakland, CA.

Kinkel,-L.L..&  Lindow, S.E.,  1990.
Spatial distributions  of Pseudomonas
syringae  strains on   potato  leaves.
Phytopathology 80:1030.

Wilson, S.E.  and S.E.  Lindow. 1990.
Phenotypic plasticity affecting epiphytic
survival  in   Pseudomonas  syringae.
Phytopathology 80:1058.

Kinkel,  L.L.  ,  M. Wilson,  and  S.E.
Lindow.  1990.  Sampling phylloplane
populations:  Distributional  effects on
sample   design.   Phytopathology
80:1030.

Lindow,  S.E.  1991. Determinants of
epiphytic fitness in bacteria, in.:  S.S.
Hirano   and  J.   Andrews   (eds.),
Microbiology  of  the  Phyllosphere.
Springer-Verlag, New York, (in press).
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       COMPARISON OF SURVIVAL ON THE PHYLLQPLANE OF BACTERIA
            RELEASED IN GREENHOUSE AND FIELD ENVIRONMENTS
              Katharine Donegan1, John Armstrong2, Carl Matyac1,
                             and Ray Seidler2

                  ManTech Environmental Technology, Inc.1
                    U.S. Environmental Protection Agency2
                      Environmental Research Laboratory
                              Corvallis,  Oregon
INTRODUCTION

   Because  of   current   concerns
regarding the  release of  genetically
engineered microorganisms (GEMs) into
the environment, the fate, survival, and
effects of many GEMs will  need to be
evaluated   in   small-scale  releases
performed   in  controlled,   contained
environments. In our research, the use
of  greenhouses  for   predicting  the
results  of  field   releases,  and  the
influence  of bacterial species,  plant
species and environmental conditions
on bacterial survival in the greenhouse
and the field were investigated.
   Small-scale  releases in  controlled
environments have been recommended
to  evaluate  the  effectiveness  of
genetically engineered microorganisms
(GEMs) and to detect any potential
adverse effects prior  to  their  release
into the environment. Microcosms have
been used  as controlled environments
in  which   to   conduct   small-scale
releases for the evaluation of the fate,
survival and effects of GEMs in  aquatic
and terrestrial ecosystems.  The use of
microcosms in such studies has  been
criticized, however, because of  their
limited  size and  lack of  biological,
chemical and physical  complexity
relative to the natural environment.
   Greenhouses   can  provide   an
environment intermediate between the
microcosm and the field for studying
the survival and effects  of GEMs prior
to field  release.  Greenhouses allow a
larger scale rele'ase into a more complex
environment  than a  microcosm yet
retain some of the containment and
control advantages of  a microcosm.
   As has been done  with microcosm
studies, greenhouse releases need to be
evaluated for their ability to predict the
results of field releases. In addition., the
influence of bacterial species and plant
species,   and   also   meteorological
conditions, on the ability of greenhouse
releases to simulate field releases needs
to be investigated. The goal of our
research was to address these issues.

METHODS    :

Plant   Growth   Conditions.. and
Experimental Design

   The experiment was conducted once
in the greenhouse and  twice in the
field.  In  each  trial, .Erwinia  herbicola
(NalR), Klebsiella planiicola (RifR), and
Pseudomonas  syringae  (RifR)   were
sprayed 'on bean plants  (Phaseolus
vulgaris]  and  on oat  plants (Avena
sativa) for a total of six plant-bacterium
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treatments.  Treatments were arranged
in a split-strip plot experimental design.
Five replicate plots of beans and of oats
were randomly assigned and planted in
the greenhouse and in the field. In the
greenhouse,   seeds   were   planted
directly in the soil floor. Each oat and
bean replicate plot was divided in thirds
and  randomly  assigned  a  bacterial
treatment for  a  total of  five  plot
replicates per  plant-bacterium treat-
ment.  The area of each treatment plot
that was sprayed  with  a  bacterial
species was 5 x 10 ft in the field and 2
x 5 ft  in the greenhouse.  Unsprayed
areas were maintained between plant-
bacterium treatment plots to  prevent
cross-contamination.

Application of Bacteria to Plants

   All   bacterial   suspensions   for
greenhouse  and  field sprays  were
prepared from 18 h cultures grown in
Luria-Bertani  (LB)   broth   with  the
appropriate antibiotics. The cells were
washed 3 times in 0.01  M phosphate
buffer  (pH 7.2) by centrifuging for  10
min. at 5000 rpm and resuspended in
sterile  water to ca.  2x10' CFU/ml.
Bacteria were applied when bean plants
were beginning to flower and oat plants
were forming seed head.  Plants were
uniformly sprayed until run-off with a
CO2 pressurized backpack sprayer.

Sample Collection and Processing

   Plant  samples were   collected  in
sterile bags on days 1,3,7, 10, 14 and
21  post-application.  In the greenhouse
and field II experiments, samples were
also collected two hours post-
application. Bean samples consisted of
3 leaves per plot that were collected at
•low, middle and high levels of the plant
canopy.  Oat samples consisted of 3
plants per plot,  with  seed heads and
roots removed.  A total of 15 samples
per  plant-bacterium  treatment  was
collected  per sample  day.   Samples
were   immediately   taken  to   the
laboratory where they  were weighed
and processed by adding 20 ml of 0.01
M phosphate buffer  (pH  7.2)  and
treating 1 minute in a Stomacher Lab-
Blender. Samples were diluted in 0.01
M phosphate buffer (pH 7.2) and plated
on  LB agar containing  500 //g/ml
nalidixic acid or  100  //g/ml rifampicin
and   100  //g/ml  cycloheximide  and
incubated at  30°C for 24  hours.

Monitoring Environmental  Conditions

   Meteorological data  were collected
during  the   greenhouse  and   field
experiments  with a 21 x  Micrologger.
Temperature, relative  humidity,  and
incident light were  measured  every
minute and averaged per hour.

Data Analysis

   Plate  counts  were converted  to
logarithmic colony forming units  per
gram of plant tissue  (log CFU/g) and
used in the Statistical Analysis System
(SAS Institute) in a stepwise backward
elimination   regression   procedure.
Separate  regression curves  for each
experiment with each bacterial species
were fit in the  same model  by using
classification variables.  Patterns  of
bacterial  survival were compared  by
testing the hypothesis that the intercept
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 and slope of the regression line for each
 bacterial genus was equal for the green-
 house, field 1, and field 2 experiments.

 RESULTS AND,DISCUSSION

    Despite the application of equivalent
 bacterial concentrations in the trials,
 bacterial populations after only one day
 post-application weresignificantly lower
 in  the field  1  and field  2  trials  as
 compared to the  greenhouse trial.  In
 the greenhouse and field 2 trials, two
 hour post-application samples were also
 collected; bacterial  populations in the
 field were on the average 10-fold lower
 than in the greenhouse.  Substantially
 lower   light  levels   occurred   in  the
 greenhouse trial than in the field trials.
 The initial large  decrease in  bacterial
 populations  in the field 1  and  field 2
 experiments may have been caused by
 exposure to  ultra-violet light.   Solar
 radiation is an important environmental
 factor in the field that is not accurately
 represented  in most greenhouse and
 microcosm simulations of field releases.
    Because  of  the  greater  decline
 during the first day in bacterial levels in
 the  field  trials as  compared  to  the
 greenhouse trial, the intercepts of the
 regression lines for the greenhouse trial
 were all significantly greater than those
 of the field  trials.   The slopes  of the
 regression lines,  however, were not
 statistically   different   between  the
 greenhouse trial and field trial 1 for the
 oat P.  syringae treatment and the oat
 and bean E.  herbicola treatments.
   Survival was very different among
the three bacterial species.  In addition,
the extent of agreement in  results
between the greenhouse and field trials
 was influenced  by  bacterial  species.
 The  K. planticola  treatment  always
 showed a  decline  in  population  and
 reached  the  lowest  bacterial  level,
 whether on oat or bean plants or in the
 greenhouse or field.  In contrast, the P.
 syringae  and  E. herbicola treatments
 displayed variable responses. Because
 E. herbicola and P. syringae commonly
 occur on the oat and bean phylloplane,
 unlike K. planticola which is primarily a
 soil   inhabitant,   their  potential  for
 survival and growth was higher than K.
 planticola but  more variable due to  a
 greater influence from meteorological
 conditions.
   In   the   greenhouse   experiment,
 populations  for  all  three  bacterial
 species were  significantly  greater on
 oat plants than on bean plants.  In the
 field  1   and  field  2   experiments,
 however, no significant differences in
 bacterial survival between plant species
 were   observed.     These  results
 emphasize  the  need   in   simulated
 releases to  evaluate  survival  of a
 bacterial species on the particular plant
 species on which it  will be  released in
 the field.  Due to the initial decrease in
 bacterial-levels in the field trials, the
 greenhouse experiment accounted for
 the maximum bacterial population levels
 observed in the field experiments in all
 but one treatment. Our results suggest
 that  greenhouse  experiments will be
 more successful in predicting trends in
 bacterial populations for field releases
than actual population levels and  that
the predictive success of greenhouse
experiments  will  vary  with   plant
species,   bacterial   species,    and
environmental conditions.
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FUTURE WORK

   Our  results  indicate   that
meteorological conditions significantly
influence the survival of bacteriaapplied
to  plants   in   field   environments.
Consequently, the use of environmental
chambers   or   "mesocosms"  that
successfully  reproduce  the different
meteorological   factors   of   field
environments  will  be  required for
accurate prediction of field releases.
   Additional research evaluating the
individual and collective  influence of
such variables as temperature, relative
humidity,   incident   radiation,   leaf
moisture and rain fall  on survival of
applied  microorganisms   will   be
necessary.   A  statistical and experi-
mental assessment of conducting such
experiments is under discussion.

PUBLICATIONS

Donegan, K., J. Armstrong, C. Matyac
and R. Seidler.  1990.  Comparison of
survival on  the  phylloplane of bacteria
released  in  greenhouse  and  field
environments.     U.S.  EPA  Report
600/3-90/085.
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              NATIONAL ECOLOGICAL FIELD RESEARCH STATION
          Linda D. Stetzenbach, Mark P. Buttner, and James R. Meldrum
                        Environmental Research Center
                    University of Nevada, Las Vegas, Nevada
   The  U.S.  Environmental Protection
Agency  environmental  biotechnology
research program is  charged  with the
development of methods to  measure
the impact of microorganisms released
to the environment.  The techniques
and  data generated  by this  research
program are used by the Office of Toxic
Substances and  Office  of  Pesticide
Programs in  making decisions on the
human   and   ecological   safety  of
microorganisms under consideration for
release  into  the  environment.   EPA
sponsored    biotechnology   research
programs  have   focused   on  the
detection,  fate,  transport,  survival,
genetic   exchange,   effects,  and
monitoring methodology and design for
field-released   microorganisms   in
terrestrial ecosystems.  The techniques
and   methods  developed,  to  date
however, are products of  experiments
conducted  in laboratory,  greenhouse,
and microcosm environments.  Many of
the methods  now being proposed for
use   in  tracking  and  enumerating
microorganisms   such   as   DNA-
hybridization    (probes),   polymerase
chain reaction (PCR), and bulk  recovery
of DNA directly from soil samples, have
yet to be tested over long periods of
time  under field  conditions.    While
useful to the program offices and the
biotechnology industry, none of  the
results have been validated under field
conditions. The lack of a field  research
facility has rendered it impossible  to
integrate the  many facets involved in
the survival of microorganisms in the
environment  and  has  hindered  the
development   of   predictive   risk
assessment expertise.   The  develop-
ment of permanent National Ecological
Field Research Stations at which long-
term experiments could be conducted
would be a direct  benefit to  EPA  and
would  be  useful  to  EPA-funded
researchers and biotechnology industry
research scientists  as well.
   A  potential  site  for  a  pilot field
research facility is available at the EPA
experimental  farm on  the  northeast
corner of the Nevada Test Site (NTS) in
southern Nevada.  The  NTS is approx-
imately  15,000   square  miles   of
dedicated land  used by the Department
of Energy located 90 miles north of Las
Vegas with  a  commute  from  the
Environmental   Monitoring    Systems
Laboratory (EMSL-LV) on U.S. Highway
95 of under two hours.  The farm was
originally used  for radiation monitoring
studies on plants  and  cattle but  has
been idle for several  years.   The farm
site is comprised  of  16 acres of  flat
agricultural fields with a gentle slope to
the south.  Electrical power, telephone
service, and a well are in operation and
only  one  small laboratory  space is
currently used on an infrequent basis by
the Nuclear Radiation Division of  the
EMSL-LV for monitoring of range cattle
and deer. The fields, remaining building
space, and hookup site  for  research
trailers would   be  available  to bio-
technology researchers.    The farm
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offers  a secure  site,  removed from
encroachment  by   development   or
intrusion by vandals.  Low cost living
and dining facilities, machine shop, and
field engineering  by the  NTS   on-site
contractor are available within 1.5 miles
of the  farm.  Extensive meteorological
data are available indicating a climate
with a full range  of seasons.  The
maximum summer daytime temperature
is customarily 95-96°F (approximately
10-15°F cooler than Las Vegas). Light
snows are not uncommon in the winter
although the average maximum daytime
high temperature is above 60°F. High
winds  may blow across the site during
the year but calm periods are common
in the early morning.
   Initial  experiments  at  this  pilot
facility could include field validation of
laboratory, greenhouse, and microcosm
studies using non-recombinant bacteria
and fungi.   Such  experiments would
provide useful  data to  the  program
offices on the survival, dispersal, and
fate of field-applied microorganisms and
on the effectiveness of  field monitoring
methods.  These experiments at a pilot
site would also illustrate the advantages
of the field research site concept.
   While no one site could be expected
to meet every  possible environmental
conditions, the  NTS farm offers  the
unique opportunity of an existing EPA
facility  located  in   a  remote  yet
accessible area within easy commuting
distance  to   an   EPA   monitoring
laboratory.  The facilities at the farm
are  both  generic  and  flexible  to
accommodate a wide range of testing
parameters and conditions.  This pilot
facility would provide  a baseline from
which additional field research stations
could be patterned.
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                OVERVIEW OF BIOTECHNOLOGY ENGINEERING
                        RISK MANAGEMENT PROGRAM

                                  J. Burckle
                     U.S. Environmental Protection Agency
                     Risk Reduction Engineering Laboratory
                               Cincinnati, Ohio
BACKGROUND

   The biotechnology engineering sub-
program is a small component of the
Agency's Biotechnology Risk  Asses-
sment Research Program, which seeks
to characterize risk through  methods
development,   data  collection  and
analyses, and  mathematical modeling.
It is  comprised of  research projects in
three broad categories:  environmental
exposure, environmental effects, and
risk  control.   The  engineering  risk
management  research  activities,  a
major  component  of  risk  control,
addresses three major concerns:
   Mechanisms   of  accidental   or
   deliberate release of the modified
   genome or organism from the site of
   production  (e.g., as  in  spills  or
   effluents),
*  Availability  and  effectiveness  of
   containment controls or destruction
   techniques, and
  Worker exposure, particularly due to
  aerosols.
    Potential commercial applications of
 biotechnology  are divided into two
 loosely   defined   categories;   (1)
 contained   product   manufacturing
 processes and  (2)  uncontained and
 semi-contained    processes.     Our
 approach has been  to  address these
 two applications individually, with our
 first and primary  efforts  devoted  to
 contained  product manufacturing  as
 requested by the OTS.
    In  1988, the  first  phase  of the
 biotechnology assessment effort was
 completed, providing a state-of-the-art
 review  entitled,  "Final   Report  on
 Biosafety   in   Large-Scale   rDNA
 Processing Facilities." This effort was
 undertaken   to   document  existing
 information  regarding (1) the potential
 source of  hazards  associated with
 large-scale fermentation facilities based
 on  recombinant  DNA,  and  (2)  to
 identify biosafety practices to manage
 such risks. The report consists of four
 volumes  which deal with the following
 subjects:   a summary  of applicable
 regulations,   a   review  of  biological
 characteristics of  a  large  variety of
 microorganisms,  unit  operations  and
 equipment used in production facilities,
 releases   and   containment,   and
 methodologies for assessing worker
exposure.
                                    242

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CURRENT PROJECTS

Assessment of Filter Containment of
Bioaerosols

K.  Willeke,  Aerosol  Research  and
Respiratory   Protection   Laboratory,
Department of Environmental Health,
University of Cincinnati; Cincinnati, OH,
M.   Lehtimaki,  Technical  Research
Center of Finland;  R.  Wyza,  Battelle
Memorial Institute, OH.

   The   OTS   has  requested   field
   verification of information used to
   estimate the effectiveness of filters
   to   remove   bioaerosols   from
   fermenter off-gas.  Bioaerosols are
   generated in significant numbers in
   fermenters  by the combination of
   Theological  properties of  fermenter
   broths,  mechanical agitation,  and
   introduction  of   air/oxygen   for
   growth   and  CO2  by  microbial
   activity.    Techniques  are being
   developed  to employ off-the-shelf
   equipment   to    characterize   the
   particle size distribution (by number
   frequency)   using light-scattering.
   When operation is satisfactory, the
   instrumentation  will  be  used  in
   conjunction  with  the   previous
   project in the field to: (1) assess the
   concentration of  bioaerosol in the
   fermenter headspace and attempt to
   relate the concentration to operating
   conditions; (2) assess the amount of
   aerosol challenging the control filter;
   (3) determine if the apparatus has
   sufficient  sensitivity  to  directly
   measure the filter efficiency; and (4)
   test the apparatus for its ability to
   identify  sources  of   bioaerosol
   leakage into the workplace.
Containment Technology and Validation
Techniques

G. Leaver, G.A.L. DeCosemo, and I.W.
Stewart, Warren Springs Laboratory,
U.K.;   R.H.   Cumming,   Teeside
Polytechnic; A.M. Cottam, Health and
Safety  Executive,  U.K.; AI-Hassan,
Birmingham   University,  Birmingham,
Alabama.

   In the United Kingdom, the Warren
   Spring   Laboratory  under  the
   sponsorship of the Department of
   Trade and  Industry, is developing
   techniques  to  evaluate   process
   equipment  containment.  We have
   completed an agreement to support
   this  work  which  addresses the
   following areas:     . :
   a.   Development  and Validation of
   Biological   Fugitive  Emission
   Measurement. This project invest-
   igates  techniques  for   sampling,
   storage, and rapid assay of samples
   for   biologically   active   materials.
   Procedures are being developed for
   optimum positioning and frequency
   of sampling for several techniques.
   b.    Containment Aspects of Bio-
   process Equipment.  Research  is
   being conducted to develop standard
   methods for measuring containment
   of bioprocess unit  operations and
   equipment   components   using
   various leak detection techniques so
   that a breach of containment can be
   detected before the introduction of
   viable  biological  materials.   Lab-
   oratory studies are being conducted
   to evaluate specific methodologies
   and equipment. In-plant studies will
   be used to evaluate applicability to
   real work-place situations.
                                     243

-------
 Evaluation  of  Alternative  Methods  of
 Assessment of Bioaerosols in  Large-
 Scale rDNA Manufacturing Facilities

 K. Martinez and  P.  Jensen, National
 Institutes of Occupational Safety and
 Health, Cincinnati, OH.

    In  preparation for conducting  in-
    plant  tests,  we   have  evaluated
    commercial  bioaerosol  samplers
    designed for  workplace  sampling.
    This   effort   has  provided  the
    information needed to  select the
    most cost-effectivesampling system
    and design of  a  quality assurance
    program.   This   work  is  being
    performed   for EPA   by  NIOSH
    experts   who   have   conducted
    laboratory experiments on a variety
    of commercial samplers to determine
    sampling efficiency and influence on
    bacterial viability during sampling.

Evaluation of the Release of Bioaerosol
from a Fermenterin a Larae-ScalerDNA
Processing Facility

K, Willike, Kettering Institute, University
of Cincinnati, OH.

   Bioaerosols were sampled at the gas
   off-take in the top of a fermenter to
   determine approximate particle size
   distribution  and   droplet
   concentrationsexiting the fermenter.

FUTURE ACTIVITIES

Formation of Bioaerosols

   Research  into  the  formation  of
bioaerosols is a continuing need.  The
investigation  of   the formation  of
 bioaerosols needs'to be extended to the
 formation during separation processes
 to describe the formation of bioaerosols
 in the fermenter headspace based on
 typical operating conditions,

 Efficiencies  of  Processes to Inactive
 Microorganisms

    Research  into the  kill/inactivation
 efficiencies  of  various physical  and
 chemical treatments commonly used for
 both sporeforming and nonsporeforming
 microorganisms  is needed. Although
 limited data on inactivation efficiencies
 are  available,  experiments  were
^conducted in demand-free water and do
 not represent the typical conditions in
 fermenters.

 Release Applications of Microorganisms

    As more  biotechnology PMN's are
 reviewed,    information   concerning
 release scenarios for field  tests  and
 other non-contained  uses covered by
 TSCA will be needed. An investigation
 of the  potential  use  scenarios  for
 release-applications of microorganisms
 should  focus  on  the  method of  appli-
 cation,  potential   occupational
 exposures,  protocols for  monitoring
 workers during non-contained uses, and
 efficiencies of methods of controlling
 releases to  minimize  release to non-
 target areas of the environment.

 PUBLICATIONS

 Cottam, A.N.  Risk  assessment and
 control in biotechnology. In Preparation.

 Gumming, R.H., arid D.  Brown. 1991.
 Process  validation   and   safety   in
                                    244

-------
biotechnology. Institution of Chemical
Engineers, Symposium Series No. 124.

DeCosemo, G.A.L., I.W. Stewart, W.D.
Griffiths,   and   J.S.   Deans.   The
assessment   of  airborne   micro-
organisms. In Preparation.

Jensen, P.A., W.F. Todd, G.N.  Davis,
and  P.V.  Scarpino.  1990. Sampling
efficiencies of eight bioaerosol sampling
instruments challenged with an aerosol
of free  bacteria. National Institute for
Occupational  Safety   and   Health,
Division  of  Physical  Sciences  and
Engineering, Murray  State  University,
Department of Occupational Safety and
Health,  Murray,  KY;   University of
Cincinnati, Department of  Civil  and
Environmental Engineering;  and  U.S.
EPA, Cincinnati,  OH.

Leaver,   G.,  et  al.   Integrity  of
fermentation equipment - containment
and  sterility. In Preparation.

Leaver,  G.  1991.  Measuring  and
monitoring containment in  bioprocess
equipment.  Institution  of  Chemical
Engineers, Symposium Series No.  124,
pp. 349-361.

Martinez,  K.F.,  and  W.F.  Todd. The
sampling   efficiencies  of  alternative
bioaerosol   sampling   devices   as
determined using a physical surrogate
aerosol.   National   Institute   for
Occupational Safety and Health, and
U.S. EPA  Report, Cincinnati, OH.

Pilacinski,  W.,   M.J.  Pan,   K.W.
Szewczyk, M. Lehtmaki,and K. Willeke.
1990.  Aerosol  release from  aerated
broths.    Biotechnology   and
Bioengineering 36:970-973

Pan,   M.J.,   W.   Pilacinski,  K.W.
Szewczyk, U. Krishnan, and K. Willeke.
1991  Size  characteristics of  particles
released   from   broths.    Applied
Occupational Environmental Hygiene 6,
(7): 604-607.

Stewart,  I.W.,   and   J.S.   Deans.
Containment testing of cell disrupters.
In Preparation

Stewart, I.W., and  T.T.  Salusbury.
Evaluation   and   comparison   of
environmental samplers and  particle
monitors for bioprocessing plants. In
Preparation.

Wickramanayake,C.B., B.W.  Vigon, D.
Evers,  M.F.  Arthur,  J.  Kasler,  A.
Gavaskar, S. Clark, N.G. Reichenback,
and V. Kogan, 1988. Biosafety in large-
scale   rDNA   processing   facilities.
Battelle Institute-Columbus Division and
U.S. EPA Report.

Williams,    R,   and   D.    Keir.  A
methodology for hazard assessment of
high  integrity containments.  Atomic
Energy Agency  Technology, Safety
Reliability   Directorate  Report.   In
Preparation.
                                     245

-------
          BIOTECHNOLOGY QUALITY ASSURANCE GUIDELINES MANUAL
                    Amy Smiecinski and Linda D. Stetzenbach
                         Environmental Research Center
                        University of Nevada, Las Vegas
    The   Environmental   Protection
 Agency requires every monitoring and
 measurement project to have a written
 and approved quality assurance (QA)
 plan.   The purpose of  the  QA plan
 requirement is to ensure that the data
 will  yield  scientifically  sound   and
 unbiased conclusions  related  to  the
 projects principle hypothesis.   Quality
 assurance principles and  practice have
 been previously published and guidance
 documents have been written to assist
 the  physical   science  resegrcher- in
 preparation  of QA  project plans  but
 these  publications  were  written with
 the  emphasis  on  chemical  research
 projects and do not specifically address
 many  of the  QA  issues  unique to
 biological  and  biotechnology-based
 research  projects.     Sampling,
 measurement,  and   quality   control
 methods  for   biological  and
 biotechnological parameters  become
 increasingly more  complex  due  to
 concerns  for   the   viability  of  the
 organisms under study and biological/
 biotechnological scientists  are  often
confused  by the terminology used in
QA  discussions   owing  to   their
unfamiliarity with this emerging area.
    The purpose of this document is to
 describe  and clarify QA and  quality
 control (QC) responsibilities prescribed
 by   the   EPA  for   biotechnology
 researchers. This document is intended
 to be used as a guidance document and
 is not to serve as  a template  by the
 research   scientist.     Nor  is' this
 document intended to be an exclusive
 source   of  information  on   quality
 assurance  considerations  for  bio-
 technology researchers. It is designed
 to provide a logical connection between
 EPA QA  policy requirements and the
 research   scientist   focusing   on
 biotechnological projects. It is intended
 to assist the scientist in achieving an
 understanding of QA  and successfully
 implementing   a  quality  assurance
 project plan (QAPP).
   This  manual will  include discussion
 of  the  elements  of a  QAPP with
 particular   emphasis  on  issues  en-
 countered with GEMs  monitoring inthe
 environment  and  incorporating  bio-
 logical  and  biotechnolgical research
 activities not discussed in the available
 QA guidance literature. Comprehensive
disucssions of QA principles, practical
applications,  and definitions are also
included.
                                    246

-------
                           CONTRIBUTOR INDEX
Abebe, H.M., 56, 162
Allison, J.C., 214
Anderson, Richard L., 172, 176
Anderson, Anne, 81
Armstrong, J.L., 52, 87, 236
Barkay, Tamar, 118
Benfield, Ernest F,, 190
Bentjen, Steve A., 151
Bleakley, Bruce H., 158
Bolton, Harvey, Jr., 140, 151
Bott, Thomas L., 135
Buchholz,  Phyllis S., 203
Buell, Robin, 81
Burckle, John, 242
Butterworth, Julie, 75
Buttner, Mark P., 240
Byrne A.,  113
Chad wick, R.W., 214
Cheng, H.-P., 113
Claxton, Larry D., 211, 217, 221
Coffin, Richard B., 98, 225
Connolly,  John  P., 98
Crawford, Don L., 151, 158
Gripe, Geraldine M.,  187
Cuskey, Stephen M., 225
Devereux, Richard, 131
Dickman,  Martin B.;  124
Dobrogosz, W., 217
Donegan,  K., 236
Doyle, J.D., 145, 151
Fairbrother, Anne, 203
Ferrante, A., 113
Fieland, Valerie, 70, 162
Fisher, W.S., 183
Fiuzat, M., 217
Fournie, Jack, 183
Fredrickson, James K., 140, 151
Gander, L.K., 145
Ganio, Lisa, 70
Gealt, Michael A., 165
Genthner, Fred, 187
George, Elizabeth, 211,214, 217, 221
Harris, D., 87
Heck, Chris, 81
Hendricks, C.W., 145,  151
Hoefle, Manfred, 4
Hughes, Patrick R.,  228
Ingham, E.R., 145
James, Rosalind D., 196
Janssen, David M.,  172, 176
Jeffrey, Wade H., 225
Kaplan, Louis A., 135
Katsuwon, Jirasak,  81
Kawanishi, C.Y., 207
Kearns, Peter E.W., 7
Khalil, T.A., 165
King, R.J., 56
Kohan, Michael J., 211, 214
Kokjohn, Tyler A., 102, 108
Landeck, Robin, 98
Lenski, Richard E., 91
Leser, T., 10
Leslie, John F.,  124
Lessie,T.G., 113
Lighthart, Bruce, 196,  199
Lin, Y., 217
Lindow, Steven E.,  232
MacKenzie, David R., 20
Marthi, Balkumar, 61
Matyac, C., 236
Mayes, M.E., 207
McCartney, H. Alastair, 75
Mclntyre, T.C., 15
McKenney, C.L., 183
Mead, Eric, 172
Meldrum, James R., 240
Middaugh, D.P., 183,  187
Milewski, Elizabeth, 22
Miller, Robert V., 102, 108
Nelson, Gail M,, 221
Nguyen, Toai T., 91
Porteous, L.A., 52
                                    247

-------
Reagin, Michael, 225
Reuss, M., 10
Sayler, Gary S., 102
Sayre, Philip G., 29
Schneider, William R., 31
Seidler, Ramon J., 56, 70, 162, 236
Selvaratnam, S., 165
Sewall, O.K., 199
Shaffer, Brenda, 61
Shannon, Lyle J., 172, 176
Short, K.A., 56
Smiecinski, Amy, 246
Snarski, Virginia M., 169
Stahl, David A., 37
Stetzenbach, Linda D., 240, 246
Walter, Michael V., 70
Wang, Zemin, 151, 158
Whitehouse, Douglas A., 211
Winfrey, Janet, 46
Winfrey, Michael R., 46
Wood, M.S., 113
Wood, H. Alan, 228
Yousten, Allan A., 190
Zdor, Rob, 81
                                   248

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                            AFFILIATION INDEX

A.F.R.C. Institute of Arable Crops Research, Rothamsted   Experimental   Station,
Harpenden, Hertsfordshire, United Kingdom, 75

American Scientific International, Duluth Environmental Research Laboratory, 172

Argonne National Laboratory, Illinois, 102, 108                       •    '•-,..

Battelle Pacific Northwest Laboratory, Washington, 140, 151          ,        *  '.

Boyce Thompson Institute, New York, 228                    •     .-  •

Denmark Ministry of the Environment, 10                     '           ,

Drexel University,  165

Environment Canada, 15                         ;

Environmental Health  Research and Testing, Inc., U.S. Environmental Protection
Agency (EPA) Health Effects Research Laboratory, 221

European Organization for Economic Cooperation and  Development, 7

German National Research Center for Biotechnology, 4

Manhattan College, 98

ManTech Environmental Technology, Inc., U.S. EPA Corvallis Environmental Research
Laboratory, 56, 61, 70, 87, 145, 151,  162, 196, 199, 203, 236

North Carolina State University, 217

Kansas State University, 124

Oklahoma State University, 102, 108

Oregon State University, 145

Stroud Water Research Center, Academy of Natural Sciences, Pennsylvania, 135

Technical Resources, Inc., U.S. EPA Gulf Breeze Environ. Research Lab, 131, 225

Teeside Polytechnic Health and Safety Executive, U.K., 242


                                    249

-------
 University of California-Berkeley, 56, 232

 University of California-Irvine, 91

 University of Cincinnati, 242

 University of Idaho-Moscow, 151, 158

 University of Illinois-Urbana, 37

 University of Massachusetts, 113

 University of Minnesota-Duluth, 172, 176

 University of Nebraska, 124

 University of Nevada Environmental Research Center, 240, 246

 University of Tennessee, Center for Environmental Biotechnology, 102

 University of Wisconsin-La Crosse, 46

 U.S. Department of Agriculture, National Biological Impact Assessment Program, 20

 U.S. EPA, Corvallis Environmental Research Laboratory, 52, 56, 61, 70,  87,  145,
 151,162,196,199,203,236

 U.S. EPA, Duluth  Environmental Research Laboratory, 169, 172, 176

 U.S. EPA, Gulf Breeze Environmental Research Laboratory, 98, 118,  183, 187, 225

 U.S. EPA, Health Effects Research Laboratory, 207,  211, 214, 217,  221

 U.S. EPA, Office of Pesticides and Toxic Substances, 22

 U.S. EPA, Office of Pesticides Programs, 31

 U.S. EPA, Office of Toxic Substances,  29

 U.S. EPA, Risk Reduction Engineering Laboratory, 242

 Utah State University, 81

Virginia Polytechnic Institute and State  University, 190


                                    250

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                                    SUBJECT INDEX
16SrRNA, 38, 46, 131
2,4-dichlorophenoxyacetate (2,4-D), 12, 145, 162
2,4,5-trichlorophenoxyacetic acid (2,4,5-T), 214
2,6 dinitrotoluene, 214
32P, 38, 48,  88, 131
Actinomycete, 141
Acyrthosipon pisum, 196
Agglutinins,  81
Alcaligenes eutrophus, 12
Algae, 135
Amblyospora, 185
Anas  platyrhynchos, 203
Ancova (analysis of covariance), 180
Animal and Plant Health Inspection Service (APHIS), 26
Apis mellifera, 201
Aroclor 1254, 214
Autographa californica, 183, 203, 228
Avena sativa, 236
Azospirillum lipoferum, 140
Bacillus cereus, 53
Bacillus popilliae, 31
Bacillus subtilis, 71, 75,  233
Bacillus sphaericus, 187, 190
Baculovirus, 183, 228
Beauveria bassiana, 33, 187, 199
Bioaerosols, 61, 243
Biosafety, 242
Biotrack System, 8
Bovine Rumen Microbial Communities, 37
Brachionus plicatilis, 184
Brassica napus, 75
Butyrivibrio fibrisolvens, 38
Canadian Environmental Protection Act, 15
Carbon Cycling, 100,  151, 158, 178
Cellulomonas fimi, 135
Cellulomonas flavigena, 135
Cellulomonas uda, 135
Chaoborus (larvae), 179
Chironomus riparius, 179,  191
Cladocerans, 179
Cladophora sp., 136
Clavibacter xyli, 32
Clostridium difficile, 217
Clostridium perfringens, 49, 217
Cochliobolus heterostrophus, 125
Coleoptera,  179, 196
Colinus virginianus, 203
Colletotrichum gloeosporioides, 88, 187
Conchostraca, 174
Conjugation (bacterial), 118, 165
Coordinated Framework for Regulation of Biotechnology, 22
                                            251

-------
 Copepods, 179
 Core Microcosm, 177
 Crassostrea virginica, 187
 Culex quinquefasciatus, 192
 Daphnia sp., 32, 195
 Desulfobacter, 48
 Desulfobulbus, 48
 Desulfococcus, 48
 Desulfosarcinia, 48
 Desulfovibrio, 48, 132
 Diptera, 198
 Dissolved Organic Carbon, 99, 135
 Dytiscid (beetle larvae), 179
 Edhazardia aedis, 184
 Enterobacter cloacae, 12, 53
 Enzyme Linked Immunoabsorbant Assay (ELISA),  12, 206
 Erwinia herbicola, 233, 236
 Escherichia coli, 53, 85, 108, 165, 204, 217, 225
 Eubranchipoda, 179
 Federal Insecticide, Fungicide and Rodenticide Act (FIFRA), 23, 31, 169
 Fibrobacter sp., 38
 Fungi, 87, 124, 145, 162
 Gambusia affinis, 184
 Gibberela fujikuroi, 124
 Heliothis zea, 188
 Helisoma trivolvis, 191
 Hippodamia convergens, 196
 Hyla crucifer, 179
 Insertion sequences (IS), 113
 kilA, 225
 Klebsiella  planticola,  75, 236
 lac, 111
 Lactobacillus reuteri, 217
 Lagenidium giganteum, 183
 Lepidoptera, 199
 Lepomis macrochirus, 169
 Liriodendron tulipifera, 136
 Lynceus, 174
 Menidia beryllina, 184, 187
 Mer genes, 118
 Mercury , 46, 118
 Mesocosm, 135, 172, 176
 Metarhizium anisopliae, 199,  203
 Methylmercury, 46, 118
 Mice (CD-1), 207, 211,217, 220
 Mixed Flask Culture (MFC) Microcosm, 176
 Mysidopsis bahia, 184, 187
 National Biological Impact Assessment Program (USDA), 20
 National Ecological Field Research Station, 240
 National Environmental Research Institute (of Denmark) NERI, 10
 Nematodes,  147
 Neurospora crassa, 87, 125
Nitrogen Cycling, 149, 178
Nomureae rilryi, 200
                                           252

-------
Nosema algerae, 184
Nosema cuneatum,  183
Nosema locustae, 183
Oncorhynchus mykiss,  169
Organization for Economic cooperation and Development (OECD), 7
Ostracods, 179
Oxyrase, 217
Palaemonetes kodiakensis, 184
Palaemonetes pugio, 184
Paragnetina media, 192
Parathelohania, 185
Pentachlorophenol (PCP), 214
Phaseolus vulgaris, 71, 75, 236
Phosphorus Cycling, 149,  178
Pimephales promelas, 169
Planoribidae, 174
Plasmids (benchmark), 92
Polymerase Chain Reaction (PCR), 14, 37
Primary Production, 178
Protozoa, 147
Pseudomonas aeruginosa,  49, 59, 102, 108, 118, 164, 211, 220, 225
Pseudomonas cepacia,  113, 211
Pseudomonas fluorescens, 85, 164, 187, 196
Pseudomonas putida, 53, 81,  145, 162
Pseudomonas syringae, 57, 69, 70, 75, 232, 236
Pteronarcys proteus, 194
Quality assurance, 246
Rats (Fisher 344, Sprague Dawley),  207, 214
Recombinant DNA processing  facilities, 242
Ruminococcus, 38
Salmonella pullorum, 203
Salmonella typhimurium, 214
Salvelinus fontinalis, 169
Scrimweave, 172, 177
Streptomyces viridosporus, 152, 158
Streptomyces lividans,  140, 151, 158
Siilfate-reducing bacteria (SRB), 37, 46, 131
Tachinidae, 198
Tipula abdominalis,  192
Toxic Substances Control Act (TSCA), 23
Transduction, 102
Transformation,  127
Trichogramma pretiosum, 199
Trichoplusia ni, 199, 229
Triticum aestivum, 140
UV (stress), 110
Vectobac, 169,  172, 177
Vegetative compatibility group (VGC), 124
Worker exposure, 242
Xanthomonas maltiphilia, 234
xylE, 56
Zooplankton, 100, 173, 179
                                          253

                         *U.S.COVERNMEVTPRINTINGOFFICE:1992 -ste -003/60061

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