United States
              Environmental Protection
              Agency
                    Office of Pesticides and
                    Toxic Substances TS-793
                    Washington D C 20460
Arsenic—
what is  it?
              Toxics  Information
              Series
             Arsenic
Any discussion of  arsenic  usually  prompts  visions
of murder and  intrigue  reminiscent of  "Arsenic and Old
Lace".    Indeed,   the   chemical  has  been  extensively
employed  as   a   poison  due  to  its   extreme   toxic
properties.   However,  it is also an active ingredient  in
an  array  of  commercial  products,   a  by-product   of
numerous industrial processes, and occurs  naturally  in
various chemical forms.   As a result, human exposure  to
arsenic at various  levels is widespread.
     This exposure, coupled with recent medical findings
which  indicate  continued  exposure  to  small  amounts  of
arsenic may produce adverse health effects, have  raised
serious  questions  about  the  continued  use of   the
chemical.  This Information Bulletin  discusses the risks
of  arsenic  and what  the  U.S. Environmental Protection
Agency  (EPA) and others are doing to control arsenic  in
the environment.

Arsenic is a greyish-white  element  common in 'the
earth's crust  where  its compounds  occur  in association
with  many  minerals.   The commercial use  of these  min-
erals,  especially  non-iron metals  ores such as copper,
lead  and  gold  necessitates removal of  the arsenic.   In
the United States  arsenic is  largely generated as a  by-
product of copper  smelting.   Domestic production  of  the
compound arsenic trioxide is  approximately 12,000  metric
tons  every  year,   and   another  9,400  metric tons  of
arsenic  and  arsenic  compounds  are  imported  yearly.
Easily  combined  with  other chemicals,  arsenic trioxide^
is  used as a  raw  material in the  formulation of other
arsenic compounds which are used commercially.
      Agricultural  products account for about 81 percent
of  the  U.S.  consumption  of arsenic.    Arsenic-based
insecticides,  herbicides,  desicants  and  defoliants  are
the  major  agricultural uses,  with  cotton  production
considered  to be  the  top agricultural  consumer.   Low
concentrations of  arsenic are also found  in  an-imal feed
additives  used to increase the rate of  weight gain in
hogs  and poultry.
      The   major   nonagricultural    use   of   arsenical
compounds  (9.5 percent of the total U.S.  consumption) is
for wood  perservatives.  Other applications  for  arsenic
include hardeners  for metal  alloys,  semiconductors,  and
veterinary  drugs.    Once  widely  used as  a  clarifying

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              agent  for  glass, arsenic  has been  largely  replaced by
              other ingredients.

Why  is       Arsenic  has  been identified  as  a public  health
arsenic  a    problem  because  of  its  serious toxic  effects  at  low
problem?      exposure  levels and its  pervasiveness  in  the environ-
              ment.   Arsenic  is  an  element  and  therefore  cannot be
              eliminated  from  the   environment.     Like  all  other
              elements,  it   goes   through  geological  and  biological
              cycles  which  redistribute  it  into  different  compounds
              and  to different  places.   Natural  processes  such as
              oxidation, methylation and demethylation may concentrate
              it  in  hazardous  proportions,  or  may  dissipate  it,
              thereby   decreasing   its   hazardous   concentrations.
              Because  arsenic  easily   changes   forms   and  readily
              combines with  other  chemicals, the  end  product of  such
              processes cannot  always  be anticipated.   Human activi-
              ties  cause arsenic  contamination   of   the  environment
              where  none  previously  existed  by putting  arsenic  into
              the  environment  faster  than  it can  be  dissipated by
              natural processes.
                   Energy production,  pesticide  production  and  use,
              and  copper  smelting  are  all   significant  sources of
              arsenic  discharges   to  the air.    Presently coal-fired
              plants are the most significant energy related emissions
              source.    New  technologies  of  energy  production  have
              important  arsenic implications,  also.    Early  data on
              coal  gasification   indicate   that  two-thirds  of  the
              arsenic present is volatilized (passed off as a vapor in
              the  gasification  process).  Oil  shale  exploitation and
              geothermal  energy development  may  also  release  large
              quantities of  arsenic  gases.   Arsenic is emitted  to the
              air  from  several other  commercial  sources:    lead  and
              zinc smelters,  iron  and  steel production, and arsenical
              compound production.
                   Pesticide  production  and   use  is  probably  the
              largest single  man-made  source of arsenic* entry to the
              total  environment.     Although  pesticides  add   to  the
              atmospheric burden of arsenic, five  times that amount is
              deposited in  the  soil  through pesticide manufacture and
              use.  Other releases of arsenic to the soil due to human
              activity  include fallout of  air  emissions  and  solid
              waste from smelters and coal-fired plants.
                   Arsenic discharges to water are much less than air
              and  soil  releases.    These releases are  the  result of
              energy  production from coal,  boron  production and  use,
              zinc  and  lead production,  non-iron  metal mining  and
              milling,  phosphorus  production and  use, pesticide  use,
              and municipal water and waste water  treatment.
                   Human  exposure   to   arsenic  occurs  through   >11
              environmental   media—air,   water,   food.      Smelter
              emissions result  in  the most arsenic exposure by  air to
              the  greatest  number  of people.   Pesticide  use  is  also
              thought  to expose  a 'large number  of people  to  atmos-
              pheric  arsenic  during  application  and  through   wind
              drift.    At  the  present  time,   there  are  no^a-creurarte
              measures  of the number of people exposed to,---arsenic Trf
              drinking  water nor  of the  degree  of  contamination of

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either  surface  or ground  waters in  the  United States.
Although  arsenic  concentrations in  waters  which  have
been  tested  vary  widely,  the average  concentration  is
close  to natural  levels.    Plants  and  animals contain
differing   amounts   of   arsenic   depending  on   the
concentrations  in soil  and  water,  thus  adding  to the
human body burden of arsenic when the plants  and animals
are  eaten.   However,  the bioccumulation of  arsenic  in
the  food chain  is  normally  not significant.   Although
marint   fish  and  other  seafood   (especially  oysters)
contain  larger  amounts  of  arsenic  than other foods, the
arsenic  is  in  an organic  form  which  is relatively non-
toxic.
     In  humans  arsenic  may be absorbed by the  lungs and
the  gastro  intestinal  tract.    Chemical  species  differ
widely   in  their  toxicity  with  the  inorganic  forms,
especially  trivalent arsenites, being more  toxic  than
organic  arsenic  species.
     Arsenic  causes  both   acute   and  chronic  health
effects  in  humans.   Acute arsenic  poisoning occurs most
commonly from accidental pesticide  ingestion.. ,  The. early
symptoms  of acute  poisoning include  intense -v.omLting,
diarrhea,  and  cramps,   accompanied  by  burning . .of, the
mouth  and  throat,  inability  to   swallow  and , extreme
abdominal,  pain.   Victims often have bloody  urine and
shock can result  from dehydration.
     A  large  body of scientific research  exists  on the
chronic  health  effects  of  arsenic  exposure.   Chronic
arsenic    poisoning,    called   arsenicism,    is   most
recognizably  characterized  by  effects  to  the  skin.
Arsenic  exposure can also produce   neurologic,  vascular,
and  liver  damage.  In addition, arsenic is a recognized
human  carcinogen.   Experiments  with  laboratory animals
suggest   that    certain   arsenic   compounds  may   also
contribute  to  the  risk  of  birth  defects  and genetic
mutations in humans.
     Cancer;    EJpidemiologic  studies  have  shown  that
arseniccauses  cancer  of the  skin.    This  finding has
been  made  in  studies of  people exposed to  arsenic  in
drinking  water,   medications,    and   the    workplace
environment.   In addition, there are  strong  suggestions
that   occupational  exposure  to  arsenic  causes  lung
cancer.   Hints of  association  with  other  tumors  have
also been reported.
     Neurological effects:   Arsenic exposure  can produce
peripheral  nerve damage and altered  electrocardiograms.
Abnormal neurological test  findings  have resulted from
exposures   to   arsenic   that  are   too low   to produce
symptoms of classical  arsenicism.   Exposure to amounts
of  arsenic in  drinking  water,  much  too  low to produce
classical  arsenicism,  has been associated with abnormal
electromyograms,  and  in  one  case, children  exposed  to an
arsenic  compound emitted  by a  coal-burning  power  plant
have  been  found to have an increase  in the  frequency of
a  specific  hearing loss.

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What is the
government
doing about
this hazard?
     Skin effects;   Arsenic ism is characterize.: '.:•/ horny
growths on the skin of  the  hands and feet and c :.rko-nir.g
of the skin of the torso, as  well  as skin cancer.   Skin
symptoms are good indicators of massive arsenic exposure
because  they  appear   in  most   people  with  very  high
exposure.
     Birth defects;   Injections  of a particular arsenic!
compoundintopregnant  animals  have  caused  specific
birth defects  in hamsters,  rats and  mice.   However,  at
the present time  there is  no evidence of arsenic-related
birth defects in  humans.
     Mutations;    Animals  and  humans  who  have  been
exposed to sodium arsenite (a trivalent form of arsenic)
have  shown chromosomal  defects  (as long  as  20  years
after exposure in  humans).    Further  research  is  needed
to determine  the exposure  levels at which these effects
are produced and  the implications for human development.
     Other effects;   High  and  prolonged  exposure  also
cause's   cirrosis   of    the   liver   and   impairment   of
peripheral veins.

There are over two hundred individual regulations        !
controlling exposure to arsenic  and arsenical exposure
of workers, consumers, and the environment through waste
discharges into air,  water  and  land.  Major regulations
actually  limiting  arsenic exposure  are  administered by
six Federal agencies.
     EPA has listed arsenic as a hazardous air pollutant
because  of  its   suspected  carcino-  genicl'ty.    This
listing,  under the  authority of the Clean Air Act, will
enable the Agency  to  determine which stationary sources
of arsenic pose signifi- cant risks  to the public and to
assign  priorities  for   the  development  of  emission
standards.   Using  the  authority  granted by  the  Clean
Water   Act,   EPA  has   set  or  proposed  regulations
controlling the  discharge of  arsenic and its compounds
into waterways from a^variety of industries.
     The  Agency  has  also  set  a  maximum contamination
level for ground water, and has  proposed a water quality
criteria  based on  the  latest  scientific  information.
The  criteria  can  be  used  for  further  regulation  of
arsenic  by EPA  or the individual States.   Under pro-
visions  of the  Safe  Drinking   Water  Act,  EPA  has  set
standards from maximum arsenic content in  drinking water
and promulgated regulations  for  the  protection of ground
water sources.
     In   1978,  EPA   issued   a   "rebuttable   presumption
against   registration"    (RPAR)   notice   on   inorganic
arsenical pesticides.  Each  compound  is  being  considered
in  individual risk/benefit  analysis.   Several, organic
arsenical  pesticides  are  being  considered for the RPAR
process, also.
     EPA  has  listed arsenic  as  a  hazardous waste under
the  Resource  Conservation and  Recovery  Act  regulations
controlling  the  management  of  hazardous  wastes   from
point of manufacture  to ultimate disposal.

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     The Occupational  Safety and  Health Administration
(OSHA)  promulgated  a   workplace   standard  for  arsenic
content in  the  air but the standard has been challenged
in court and the case has not been decided yet.
     The  Food  and Drug  Administration  (FDA)  monitors
food  for  arsenic  content and  enforces  food  tolerance
levels established by EPA.
     The Department of Transportation (DOT), the Nuclear
Regulatory  Commission   (NCR)  and  the Department  of the
Army also regulate arsenic.
     In Summary;   Arsenic, although naturally present in
all parts of  the environment, poses a human health risk
because of  increased levels  in  particular areas caused
by human activity.  Government agencies have taken steps
to   minimize    human   exposure   to   arsenic,   and  are
considering  additional  methods  to  further reduce  the
arsenic risk  in this country.
ENVIRONMENTAL EFFECTS

     In  addition  to  human  health  effects,   increased
arsenic  in  the  environment can have  profound effects on
the environment  itself.   Arsenic compounds are toxic to
all  plants  and  animals.    The  result  of  high-level
arsenic   contamination   is  an  essentially  sterilized
environment.      Orchards  where   inorganic   arsenical
pesticides  have been  heavily used,  for instance, must
have the  contaminated soil  removed  before  replanting can
take place.   Otherwise,  the  new trees will die because
of  the toxic concentrations  of arsenic in the treated
soil.

     When  arsenic  compounds  enter  soil  or  water they
bind to  soil and sediment  components.   Binding greatly
decreases the amount of  arsenic  available  to cause toxic
effects,  but as more arsenic  is  released it becomes more
readily  available because  because  the bound arsenic is
at  equilibrium  with  available  arsenic.    Arsenic  is
continuously cycling  in the environment due to chemical
and biological  transformations.   Also,  man's activities
can  significantly alter  the  cycle  by causinu  localize^
high concentrations.

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