INTERIM REPORT
TASK 002 ROAP 10CAR
TOXICITY OF COPPER, CADMIUM AND
ZINC TO PACIFIC NORTHWEST SALMONIDS
Gary A. Chapman
Western Fish Toxicology Station
National Water Quality Laboratory
1350 S.E. Goodnight Avenue
Corvallis, Oregon 97330
June 1975
NATIONAL ENVIRONMENTAL RESEARCH CENTER
OFFICE OF RESEARCH AND DEVELOPMENT
U.S. ENVIRONMENTAL PROTECTION AGENCY
CORVALLIS, OREGON 97330

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INTERIM REPORT
TASK 002 ROAP 10CAR
TOXICITY OF COPPER, CADMIUM AND
ZINC TO PACIFIC NORTHWEST SALMONIDS
Gary A. Chapman
Western Fish Toxicology Station
National Water Quality Laboratory
1350 S.E. Goodnight Avenue
Corvallis, Oregon 97330
June 1975
NATIONAL' ENVIRONMENTAL RESEARCH CENTER
OFFICE OF RESEARCH AND DEVELOPMENT
U.S. ENVIRONMENTAL PROTECTION AGENCY"
CORVALLIS, OREGON 97330

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LIST OF TABLES
Number
I	Water quality characteristics of dilution water
for bioassays.
II	Ninety-six hour mortality data from static
bioassays of coho salmon alevins and buttoned-up
fry. Solutions were made up in Willamette River
water and renewed every 24 hours. Bioassays were
conducted at 10 C.
Ill	Mortality data from copper, cadmium, and zinc acute
lethality tests with adult salmonids.
IV	LC50 values (yg/1) for cadmium to various life
stages of salmonids.
V LC50 values (ug/T) for zinc to various life
stages Of salmonids.
VI LC50 values (yg/1) for copper to various life
stages of salmonids.
VII	Growth and mortality in 4 month, egg-through-fry,
partial chronics with chinook salmon and cadmium,
zinc, and copper. The cadmium, zinc, and copper
bioassays were terminated after 12, 13, and 14
weeks post-hatch respectively.
2

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SECTION I
INTRODUCTION
Active and abandoned mining sites create heavy metal pollution
problems in several areas in the Pacific Northwest (Sceva, 1973).
Most notable pollution problems from these activities are acidity,
copper, iron, zinc, manganese, and copper pollution. In addition,
data for water quality criteria are being developed on numerous
chemicals including copper, cadmium, and zinc, but little data are
available on the salmonids of the Pacific Northwest.
This report covers a series of 50 toxicity tests with heavy
metals, primarily cadmium, copper and zinc, and their effects on
selected species of salmon and trout native to the Pacific Northwest.
The series began in February, 1971, and was completed in March, 1975.
Forty-six of the tests were acute lethality tests lasting from 96 to
390 hours, and four tests were partial (less than full life cycle)
chronic toxicity bioassays lasting from 19 weeks to 21 months. The
studies can be split into five separate groups according to purpose:
GROUP I: COHO SALMON STATIC ACUTE BIOASSAYS.
These tests were conducted to find the approximate lethal
threshold ranges for coho salmon (Oncorhynchus kisutch) for cadmium,
copper, lead, and zinc. Data were collected for 96 hr. LC50
calculation, and changes in metal tolerance were followed closely
through the early larval and juvenile stages.
3

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GROUP II: SOCKEYE SALMON ADULT THROUGH SMOLT PARTIAL CHRONIC BIOASSAY.
This 21-month test was designed to determine the maximum allowable
level of zinc for the sockeye salmon (Oncorhynchus nerka).
GROUP III: ADULT SALMONID FLOW-THROUGH ACUTE BIOASSAYS.
These tests were conducted to determine the levels of heavy
metals acutely lethal to adult coho salmon (Oncorhynchus kistuch)
and steel head trout (Salmo gairdneri), which because of their large
size and relative inavailability, had never been bioassayed.
GROUP IV: JUVENILE SALMONID FLOW-THROUGH ACUTE BIOASSAYS.
This series of bioassays was designed to yield data on the
relative sensitivity to metals of the various juvenile life stages
of coho and chinook salmon and steelhead trout. Ninety-six hour,
200 hours, and longer LC50 values were calculated whenever possible.
GROUP V: CHINOOK SALMON EGG-THROUGH-FRY PARTIAL CHRONIC BIOASSAYS.
These tests were conducted to determine if continuous exposure
to metals through the embryo, alevin and young fry stages would
produce adverse effects at lower metal concentrations than those
causing mortality in the acute bioassays.
4

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SECTION II
METHODS
CHEMICAL
All bioassays were conducted with reagent grade chloride salts
of the metals (CuCl2*2H£0; CdCl2"2 1/2H20; and ZnCl2)• Stock
solution for static bioassays were mixed in distilled or deionized
water, while stock-solutions for flow-through bioassays were mixed
in diluent water. (Cadmium superstocks for dilution to make up flow-
through cadmium stock solutions were also mixed in distilled or
deionized water). Stock solutions were usually acidified with
concentrated nitric acid to about pH 4.
Six static bioassays (GROUP I) were conducted using Willamette
River water for dilution. All other bioassays used Western Fish
Toxicology Station (WrTS) well water for dilution. Both waters are
characterized by low hardness and alkalinity. In some instances
where the well water quality differed significantly from the norm,
reverse osmosis treated well water was mixed with ambient well water
to attain normal water quality using hardness as the index of
normality. A summary of water quality values for the three diluent
waters is listed in Table I.
BIOLOGICAL
All bioassays were conducted on marine anadromous salmonids
native to the west coast of North America. Four species were used:
5

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TABLE I. Water quality characteristics of dilution water for bioassays
Blend:
. Well + Reverse
Parameter	Units Willamette	WFTS Weill/ Osmosis Product
Total Organic
Carbon
mg/1
3.4
l.a
1.4
pH

7.4
7.3
7.2
A1kalinity
mg/1
23
24
22
Hardness
mg/1
22
25
24
Nitrate
mg/1
0.15
0.19
0.18
Ammonia
mg/1
D.06
0.04
0.02
Calcium
mg/1
5.5
6.8
6.1
Magnesium
mg/1
2.1
1.8
1.8
Sodium
mg/1
4.1
5.0
4.4
Potassium
mg/1
0.8
0.6
0.4
Sulfate
mg/1
6.6
4.2
5.8
Dissolved Solids
mg/1
52
59
53
Suspended Solids
mg/1
15
1.5
<1
Chloride
mg/1
4
6
6
Cadmium
yg/l
<1
<1
<1
Chromium
yg/1
<2
<2
<2
Cobalt
yg/l
<1
<2
<1
Copper
yg/l
3.3
3.1
1.8
Iron
yg/1
736
98
36
Lead
yg/l
3.0
4.0
5.5
Manganese
yg/i
30.7
3.5
2.2
Nickle
yg/l
1.9
1.9
1.6
Zinc
yg/l
9.4
4.8
4.0
Mercury
yg/l
0.9
1.2
0.9
•I/Mean values excluding periods of fluctuating hardness and alkalinity.
6

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sockeye salmon, Oncorhynchus nerka; coho salmon, Oncorhynchus kisutch;
Chinook salmon, Oncorhynchus tshawytscha; and steel head trout, Salmo
qairdneri.
Adult salmonids were guided into traps or ponds by water flow,
individually brail led into a refrigerated fish hauling truck, and
transported to the WFTS test facility. Adult coho salmon and steelhead
trout were taken at the end of their spawning migrations at the Fall
Creek and North Fork Hatcheries respectively, on the Alsea River in
western Oregon. Sockeye salmon adults were collected during their
spawning migration at Bonneville Dam on the Columbia.River.
All non-adult fish were reared at the WFTS test facility from
eggs obtained from state fish hatcheries in Oregon. Coho salmon eggs
were obtained from the Fall Creek Hatchery, steelhead trout eggs from
the North Fork Alsea Hatchery, and chinook salmon eggs from the
Willamette Hatchery.
Iodophore disinfectant (Wescodyne or Betadyne) has been used
at WFTS since 1973 for prophylactic treatment of eggs brought into
the laboratory. Solutions of 1:600 to 1:150 have been employed for
from 5 to 15 minutes as a dip-bath treatment. The pH of the
solutions was adjusted to pH 7 by the addition of sodium bicarbonate.
Egg disinfection was the only treatment given any of the fish used
in the bioassays.
Juvenile fish were fed a commercial diet (usually Oregon Moist
Pellet) except for 48 hours prior to and during acute lethality bioassays.
All partial chronic test juveniles were fed an OMP ration based on fish
number, disregarding growth differences between treatments.
7

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APPARATUS AND DESIGN
Coho salmon static lethality tests (group I) were conducted
in 3 liters of water in 1 gallon jars. Solutions were maintained
at 10 C, aerated continuously, and renewed every 24 hours. Two
jars were prepared at each metal concentration and 5 fish were
placed in each jar. Loading factors increased from 0.27 to 1.15
g/1 during the series of tests.
All adult salmon tests were conducted in a series of 5 tanks
3.67 m in diameter and 0.9 m deep. Tank volume was maintained at
5250 liters with a flow rate of 19 liters per minute and a recycle
rate of 150 1/min. Adult tests were conducted at 10 t 2 C. Loading
factors were from 5 to 17 g/1.
Twenty adult sockeye were held at each zinc concentration
during the initial phase of the partial chronic bioassay (group II).
Eggs from 5 females per concentration were used as the source of
fish for the juvenile phase of this bioassay. After hatching, the
eggs from a common treatment were pooled and 400 fry (200 fry per
duplicate aquarium) were exposed to each zinc concentration. During
the adult salmonid acute lethality test series (group III) the number
of test fish per tank varied. The minimum was 10 fish per concen~
tration. The maximum was 33 fish per concentration.
Egg and juvenile test chambers were dosed by continuous flow
diluters with flow rates of 500 ml/min/aquarium except for smolt
8

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tests which had 1.5 1/min/aquarium flow rates. Eggs were incubated
in small incubator chambers, juveniles were exposed in 19 liter
aquaria, and smolts were exposed in 100 liter aquaria. Loading
factors ranged from 0.2 g/1 to 14.6 g/1 and mean temperatures were
12 t 1 C.
All group IY juvenile acute lethality tests utilized 40 fish
per concentration (20 fish per duplicate aquarium). The egg through
fry partial chronic bioassays (group V) were started with about
200 eggs (100 eggs per duplicate incubator).
With the above loading factors, flow rates, and chamber volumes,
mean dissolved oxygen concentrations never fell below 8.8 mg/1 in
any chamber, aquarium, or tank.
9

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SECTION III
RESULTS
GROUP I: COHO SALMON STATIC TESTS
The most definitive data were collected in a series of six weekly
96 hr. lethality tests of Cd, Cu, Pb, and Zn using coho salmon from
two weeks post-hatch through seven weeks post-hatch. Dilution water
was coarse-filtered Willamette River water.
Results of these bioassays indicated that 96 hr. LC50's could
be expected at concentrations near 45 ppb with copper, 200 ppb with
zinc, 600 ppb with lead, and 2 ppb with cadmium (Table-II).¦ The
susceptibility of the fish to copper, zinc, and lead did not vary
appreciably with age, but the susceptibility to cadmium increased
significantly as the fish developed from two week old alevins to seven
week old fry during the series of bioassays.
GROUP II: SOCKEYE SALMON PARTIAL CHRONIC TEST
A 22-month, adult-through-smolt, partial chronic bioassay was
begun in July 1971 with the completion of the adult test facility at
the Western Fish Toxicology Station. Sockeye salmon were chosen as
the test organism because of their relatively long freshwater residence
as prespawning adults (4 to 5 months), their relatively small size,
and their availability. Test exposure concentrations were based on
the available data from juvenile coho and chinook salmon. Zinc
concentrations of 30, 60, 90, and 120 ppb were selected as exposure levels.
10

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Table II. N1nety-s1x hour mortality data from static bloassays of coho salmon alevlns and buttoned-up
fry. Solutions were made up 1n Willamette River water and renewed every 24 hours. Bloassays
were conducted at 10 C.
Date
1000
300
100
60
Cu (ppb)
45
30
10
Developmental"^
Index
'F1sh
Wet Wt.
(nig)
02/02/71
100
100
100


0
0
43.8
159
02/08/71
—
100
100
80

0
0
61.8
225
02/15/71
—
...
100
60
20
0
0
75.0
328
02/22/71
—
—
100
60
70
0
0
83.5
375
03/01/71
— ¦
—
100
70
10
0
0
(100)
474
03/08/71


100
80
60
10
0
(100)
486





Zn (ppb)




Date
30,000
10,000
3,000
1,000
600
400
300
200 140 100
60
02/02/71
02/08/71
02/15/71
02/22/71
03/01/71
03/08/71
100
100
100
100
80
100
100
90
100
100
10
90
60
80
100
100
50
50
80
80
30
0
0
0
40
0
Pb (ppb)
Date
30,000
10,000
6,000
3,000
1,000
600
300
100
60
02/02/71
100
100

90
90

0
_ — —'

02/08/71
	
	
100
100
100
60
100
	
¦ —
02/15/71
	
	
—
—
80
10
0
0
0
02/22/71
	
	
—
70
60
40
0
0
—
03/01/71

—
—
100
100
60
10
0
—
03/08/71
	
—
—
100
100
50
20
0

Cd (ppb)
Date
3,000
1,000
300
TOO
30
15
10
6
4.5
3
2
1
0.5
02/02/71
—
20
...
10
30
...
40


...

...
...
02/08/71
100
100
100
100
100
—
—
—

.— .
	
—. .
...
02/15/71
100
...
100

100
100

100
	
30

—
' .—
02/22/71
100
	
—
—
—

100
90
70
50

10

03/01/71
100
—
—
—
—
—
100.
100
100
100
—
0
...
03/08/71
—
—
—
—
—

—
100
100
100
90
10
0
1/ Index of development of alevlns Is the percent of total alevin weight which 1s not yolV.
This is calculated:
Index = fish weight w/o yolk
weight of fish plus yolk
X 100
11

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No effects were observed through egg-take in October, so one additional
concentration (240 ppb) was employed throughout the remainder of the
bioassay, (i.e. eggs from control fish were subsequently reared through
the smolt stage at 240 ppb zinc).
The adult-to-smolt bioassay was concluded in April 1973, when
smolts from each zinc concentration were successfully held for 96
hours in 30 ppt seawater. No adverse effect of zinc exposure was
noted during the 22 month test. The following parameters were
measured for effect and yielded "no effect" results: adult mortality;
fecundity; spawning time; hatchability; growth; embryo, alevin, parr
and smolt mortality; and ability to survive transfer to 30 ppt seawater.
About mid-way through the bioassay, an acute lethality flow-through
test was conducted to determine if acclimation to zinc may have occurred
and to check the 96 hr. LC50 of control juvenile sockeye. The results
indicated that the 96 hr. LC50 for 9 month old juvenile sockeye'was
about 740 ppb, a concentration at which all the 240 ppb acclimated
juveniles survived.
The data produced from this adult-to-smolt bioassay do not allow
the determination of either a Maximum Allowable Toxicant Concentration
(MATC) or an application factor relating the 96 hr. LC50 with the MATC.
However, the application factor shown to be "safe" from the adult-to-
smolt exposure is ^ 120/740 or 0.16; that based on egg-to-smolt exposure
12

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is > 240/740 or 0.32. Thus, zinc concentrations of about 0.2 of the
96 hr. LC50 appear to be safe for spawning and rearing sockeye salmon,
however no application factor is derivable for an effect or unsafe
level because chronic exposure levels of zinc were too low to produce
an effect.
GROUP III: ADULT SALMONID ACUTE LETHALITY TESTS
The acute lethalities of cadmium, copper, and zinc were determined
for adult coho salmon and steelhead trout in a series of bioassays
from November, 1971 through February, 1972 (Table III). Fluctuations
in hardness and alkalinity occurred during the series of bioassays
and made direct comparisons between bioassays impossible.
GROUP IV: JUVENILE SALMONID ACUTE: LETHALITY TESTS
Full life cycle chronic bioassays with copper and zinc have
indicated that the effect of copper on brook trout (Salvelinus
fontinalis) appears most severe during the very young juvenile stages
(McKim and Benoit, 1971) and that the effect of zinc is manifested by
poor reproductive success in fathead minnows (Brungs, 1969). Since
zinc was shown to have no discernible adult-to-smolt effect at 0.16
of the 96 hr. LC50 (as opposed to an application factor of at least
0.01 in the fathead minnow), I decided to concentrate on the acute
lethality of metals to the various freshwater life stages of salmonids.
13

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TABLE III. Mortality data from copper, cadmium, and zinc acute
lethality tests with adult salmonids.
Species
Exposure Period
Steel head Trout	n	A1kalinity
copper	11	25-45
cadmium	12	25-62
zinc	11	45-59
zinc	11	37-55
96 hr.
57 ppb
1400-2000 ppb
<1265 ppb
400 hr.
5 ppb
Coho Salmon
copper
cadmi um
zinc^
n
33
20
10
A1 kalinity
22
22
25
96 hr.
43 ppb
17 ppb
1250 ppb
200 hr.
3 ppb
y
Static bioassay with recirculation for aeration.
14

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Due to limitations of time and space the study was confined
mainly to two species, the chinook.salmon and the steelhead trout.
The coho salmon was de-emphasized as it inhabits mainly small coastal
streams away from existing population centers and pollution sources.
The sockeye salmon was eliminated from this phase of the study due
to evidence that it is more resistant to metal toxicity than other
salmonids.
Flow-through lethality tests were conducted on newly hatched
alevins, swim-ups, parr, and smolts of steelhead trout and chinook
salmon with cadmium, copper, and zinc. Each test required up to
3 weeks, including a one-week, post-handling acclimitization period
for fish in the test aquaria prior to metal introduction.
The series of tests was begun August 1972 and concluded August
1974, although no tests were conducted between January 1973 and
March 1974 because of inability to control fluctuating hardness and
alkalinity in ambient well water. The completion of a system to
variably blend osmosis treated well water with ambient well water
made it possible to complete the series after March 1974.
The mortality data from these tests are included in Tables IV,
V, and VI. The 200-hour LC50's for steelhead and chinook
(excluding the more resistant alevins) were 0.9 to 2.3 ppb for Cd,
93 to 395 ppb for Zn, and 14.7 to 26 ppb for Cu.
15

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TABLE IV. LC50 values (yg/1) for cadmium to various life stages
of salmonids.
Species
Stage (Age)l^
96-hr.
Time
200-hr.
Terminal^
Steel head
Parr (5 mo.)
1.0
0.9
0.9 (390 hr)
Steel head
Swim-ups (4 wk.)
1.3
1.3
1.3
(216 hr)
Chinook
Parr (6 mo.)
3.5
2.0
1.4
(339 hr)
Chinook
Swim-ups (3 wk.)
1.8
1.6
1.5
(264 hr)
Chinook
Smolts (18 mo.)
>2.9
2.3
1.4
(339 hr)
Steel head
Smolts (13 mo.)
4.1
1.5
1.5
(339 hr)
Co ho
Parr (6 mo.)
2.7
-*•-
2.0
(225 hr)
Coho
Adults
17.5
3.3


Steel head
Adults
•» ^

4.8
(408 hr)
Chinook
Alevins (1 day)
>26.3,
>26.3
>26.3
(261 hr)
Steel head
Alevins (1 day)
>27.0

>27.0
(186 hr)
i/Age (from hatch) at start of test.
2/LC50 value at the time that the test was terminated.
16

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TABLE V. LC50 values (yg/1) for zinc to various life stages of salmonids.
Species
Staqe (Aqe)-^
96-hr.
200-hr.
Terminal-^
Steel head
Swim-ups (4 wk.)
93
93
93
(216 hr)
Chinook
Swim-ups (3 wk.)
97
97
95
(264.hr)
Steel head
Parr (5 mo.)
136
120
120
(344 hr)
Steel head
Smolts (13 mo.)
>651
278
278
(339 hr)
Chinook
Smolts (18 mo.)
701
369
306
(369 hr)
Chinook
Parr (6 mo.)
463
395
374
(276 hr)
Steel head
Alevins (1 day)
815
—
555
(186 hr)
Chinook
Alevins (1 day)
>661
>661
>661
(214 hr)
Co ho
Parr (6 mo.)
>1232

1058
(217 hr)
.1/ Age (from hatch) at start of test.
if LC50 value at the time that the test was terminated.
17

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TABLE VI. LC50 values (yg/1) for copper to various life stages of salmonids.
Species
Staqe (Aqe)^
96-hr.
200-hr.
Terminal 2/
Steel head
Parr (5 mo.)
18.0
14.7
13.8 (387 hr)
Steel head
Swim-ups (4 wk.)
17.1
16.6
—
Steel head
Smolts (13 mo.)
>26.0
21.4
18.4 (339 hr)
Chinook
Swim-ups (3 wk.)
19.1
18.7
18.7 (264 hr)
Chinook
Alevins (1 day)
26.4
20.4
19.7 (235 hr)
Chinook
Parr (6 mo.)
38.0
29.8
21.6 (276 hr)
Chinook
Smolts (18 mo.)
26.0
26.0
22.0 (339 hr)
Steel head
Alevins (1 day)
28.2
—
25.8 (186 hr)
Co ho
Parr (6 mo.)
28.0-38.0

28.0-38.0 (217 hr)
Co ho
Adults
42.9
—
—
Steel head
Adu]ts
57.4
^ «
...
y Age (from hatch) at start of test.


y LC50 value
at the time that
the test was
terminated.

18

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GROUP V: EGG-TO-FRY PARTIAL CHRONIC TESTS.
The finding of acute mortality of swim-up and parr salmonids
at very low levels of Cd, Cu, and Zn raised the question of whether
longer-term exposure of very young stages of these fish would produce
adverse effects at even lower levels of these metals. Accordingly,
a series of 3, egg-through-fry 4-month partial chronic tests were
conducted on Chinook salmon, one test each with Cd, Cu.and Zn.
Exposure was continuous from several hours post-fertilization through
hatch, swim-up and feeding fry stages.
Mortality in these 3 bioassays is summarized in Table VII.
Significant mortality occurred with all 3 metals, but even though
the highest test concentrations of each metal exceeded the 200 hr.
LC50's for chinook parr and/or swim-ups, only-the, 20. ppb Cu concen^
tration produced greater than 50 percent mortality.
Neither 0.96 ppb cadmium nor 272 ppb zinc produced adverse
effects during the four month egg-to-fry partial chronic, even though
these concentrations were near the 200 hr. LC50 value observed in
acute lethality tests. Only copper, which produced significant
mortality at 15.5 ppb and 11.7 ppb appeared to produce adverse effects
at levels below 200 hr. LC50 values.
The effect of cadmium and zinc on growth were small, and indicated
that the metal exposed fish grew as well as or better than control fish.
Copper caused very much lower growth rates at all concentrations
19

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TABLE VII. Growth ami mortality in 4 month, egg-through-fry, partial chronics with chlnook salmon
and cadmium, zinc, and copper. The cadmium, zinc, and copper bioassays were terminated
after 12, 13, and 14 weeks post-hatch respectively.
Cadmium Concentration (pg/1)
Nominal
I
s
2.0
1.88
0.29
1.34
1.30
0.22
0.9
0.96
0.18
0.6
0.71
0.13
0.4
0.51
0.13
Control
0.16
0.11
Z1nc Concentration (pg/1)
Nominal
X
S
500
511
20
280
272
11
157
173
10
88
101
15
49
60
7
Control
9
6
Copper Concentration (yg/1)
Nominal
J
s
20
20.2
1.8
15
*5.5
0.8
11.25
H.7
0.8
8.4
9.4
1.0
6.3
7.4
1.0
Control
1.2
1.7
Mortality (X)	Mean Dry Weight (mg)
27
140
18
131
3
162
6
159
5
127
9
127
Mortality (%)	Mean Dry Weight (mg)
22
143
11
167
13
163
4
175
9
202
9
141
Mortality (2)	Mean Dry Weight (mg)
94	41
49	63
20	99
10	91
10	113
9	177
20

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tested. A similar effect was reported by McKim and Benoit (1971),
where copper levels down to 3.4 ppb reduced growth of brook trout
for up to 23 weeks post-hatch, however the growth rate of copper
intoxicated fish was the same as control fish after 23 weeks. A
similar recovery could have occurred in the chinook bioassay had
it been continued longer.
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SECTION IV
SUMMARY
ZINC
Exposure of salmonids, especially post-swim up juveniles,
to zinc concentrations as low as 93 ppb produced 50 percent
mortality in 96 hours. Extending the exposure period longer (up
to 344 hours exposure) didn't produce appreciably greater mortality.
Steel head trout appeared to be the most susceptible species tested,
followed by Chinook salmon, coho salmon and sockeye salmon,
although data on the latter two species were not as complete.
Partial chronic exposures to zinc produced no evidence of
effect levels below those seen in acute flow-through tests. In
fact, effect levels in Chinook egg through fry tests, (4 month)
were three to five times higher than 96 hr. LC50 concentrations for
Chinook swim-ups, indicating that significant acclimation to zinc
occurred.
No effect was seen in adult through smolt partial chronic
tests at a high concentration of 120 ppb, or in egg-thru-smolt
partial chronic tests at a high concentration of 240 ppb. These
presumably safe concentrations would, when coupled with the 740 ppb
96 hr LC50 for sockeye fry, result in application factors for "safe"
levels of zinc of not less than 0.16 for adult thru smolt exposure
and not less than 0.32 for egg thru smolt exposure.
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Concentrations below the 96 hr. LC50 produced mortality in the
acute exposure tests; 20 percent mortality occurred at 67 ppb with
steelhead swim-ups and 10 percent mortality was seen at 63 ppb with
chinook swim-ups.
Acclimation to zinc accounts for the higher "no effect" levels
seen in partial chronic exposures. Recent communications from NWQL
(McKim, personal communication) revealed similar acclimation of
brook trout to zinc, and showed no effect of 500 ppb Zn on reproduction
in brook trout. Thus, the 0.01-0r-so application factor for Zn,
derived from fathead minnow reproduction effects, appears to have
little relation to salmonids. Lorz (personal communication!/) has
shown that Zn has little effect on saltwater adaptation of salmonids
following acute exposure to zinc. It therefore appears that the
acute toxicity tests reported here represent a surprisingly
sensitive direct estimator of safe and unsafe levels of zinc.
COPPER
Acute lethality tests with copper produced 96 hr. LC50 values
as low as 18 ppb and 200 hr. LC50 values as low as 14.7 ppb (with
steelhead trout swim-upsjL Appreciable mortality, 33 and 18 percent,
occurred at 13.8 and 11.5 ppb respectively. Other stages of steelhead
trout and chinook salmon had 200 hr. LC50 values between 16.6 and 29.8 ppb.
Dr. Harry Lorz, Oregon State Wildlife Commission, Research
Division, Corvallis, Oregon 97330.
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Egg thru fry partial chronic bioassays produced 20 percent
mortality at 11.7 ppb in 5 months. According to McKim and Benoit
(1971) alevin-juvenile mortality was the most sensitive life stage
in brook trout, regardless of previous parental or embryonic exposure.
However, the bulk of the mortality occurred between 3 and 11 months
post-hatch. The lowest effect level in their study (17.4 ppb)
produced double the control mortality at three months post-hatch.
A similar mortality (20 percent Vs. 9 percent in controls) at 3 1/2
months post-hatch occurred at 11.7 ppb in my chinook egg-to-fry
partial chronic.
The effect and no-effect concentrations in the chinook egg
thru fry partial chronic were 11.7 ppb and 9.4 ppb respectively,
and the 96 hr. LC50 for chinook swim-ups (most sensitive stage)
was 19.1 ppb. Therefore the apparent application factors would be
11.7/19.1 and 9.4/19.1 or 0.61 and 0.49 respectively for the effect
and no effect levels. These levels (11.7 and 9.4 ppb) are very
close to what would be predicted from the acute toxicity bioassay
with chinook swim-ups (18 percent mortality at 11.5 ppb).
CADMIUM
Cadmium concentrations as low as 0.9 ppb can produce 50 percent
mortality in acute toxicity bioassays (200 hr. LC50 for steel head
parr). Ten percent mortality occurred at 0.65 ppb. These are the
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lowest cadmium effect levels measured In studies at WFTS. Chronic
egg-thru-fry exposure indicates that acclimation occurs and this
implies that unless reproductive or smoltification problems can be
shown at sub-acute levels, there is little liklihood for direct
effect levels below these acutely lethal concentrations.
GENERAL
There is little doubt that the acute toxicity bioassay tests
conducted on metals at WFTS were very sensitive with respect to
estimating safe levels. None of the partial chronic tests (which
were far fewer than the acutes) yielded evidence of a large application
factor and supposably "safe" levels could be estimated fairly well
at the highest no kill concentration from the acute lethality tests.
These results seem atypical with respect to much of the heavy
metal data developed at other NWQL laboratories. However, on
closer examination, the smaller application factors derived at WFTS,
coupled with the lower LC50's, produce effect and no-effect levels
for these metals which are similar to other NWQL data. This is
especially true with the brook trout data of McKim and Benoit (1971)
which show unsafe and safe levels of copper at 9.5 yg/1 and 17.4 yg/1
respectively (ours are 9.4 yg/1 and 11.7 yg/1).
Much of the variability in LC50 and application factor data
probably derives from the effect of metal complex formation. Natural
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waters of distinctly different chemical composition will yield
different proportions and types of metal forms. Metal uptake rates
will therefore vary and this will affect LC50 concentrations. This
factor alone could produce different application factors. Some
forms of a metal may have significant long term toxic effect but
have little acute lethal effect (or visa-versa). In addition, intra-
and inter-specific fish variability may also affect the application
factor. Considering all these phenomona it is surprising how well
the bioassay data compare.
Further research is planned to compare lethal levels of metals
in the field and laboratory, especially with respect to free cation
to total metal ratios. In addition, the Cu and Cd complexing
capacity of various natural waters will be investigated to ascertain
the variability and predictability of metal complexing.
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BIBLIOGRAPHY
Brungs, William A. 1969. Chronic toxicity of zinc to the fathead
minnow Pimephales promelas Rafinesque. Trans. Am. Fish.
Soc., 98(2J: 272-2797^
McKim, J. M., and D. A. Benoit. 1971. Effects of long term
exposures to copper on survival, growth, and reproduction
of brook trout (Salvelinus foritinalis). J. Fish. Res. Bd.
Canada 28; 655-662.
Sceva, Jack E. 1973. Water quality considerations for the metal
mining industry in the Pacific Northwest. E.P.A. Region X,
Report No. Region X-3, 69 pp.
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