EPA/600/A-92/081
Monitoring of the Estrous Cycle in the Laboratory Rodent
by Vaginal Lavage
Ralph L. Cooper1, Jerome M. Goldman1 and John G. Vandenbergh2
1	Endocrinology/Gerontology Section
Reproductive Toxicology Branch, MD-72
Developmental Toxicology Division
Health Effects Research Laboratory
U.S. Environmental Protection Agency
Research Triangle Park, N.C.
27711
and
2	Department of Zoology
Box 7617
North Carolina State University
Raleigh, North Carolina
27695
Running Title: Monitoring the Estrous Cycle
Correspondence to: Ralph L. Cooper, Ph.D., Chief
Endocrinology /Gerontology Section
Reproductive Toxicology Branch, MD-72
DTD, Health Effects Research Laboratory
U. S. Environmental Protection Agency
Research Trianele Park, NC 27711

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Introduction
Ovarian cyclicity in a number laboratory species can be monitored easily
and noninvasively by observing changes in the vaginal cytology. This process
has been successfully employed by numerous researchers for more than 70
years, since the initial studies published by Stockard and Papanicolaou (1) in
the guinea pig, Allen (2) in the mouse and Long and Evans (3) in the rat.
Information obtained from the daily vaginal smear not only provides a simple
means to determine the normal pattern of ovarian activity present in the
different species, but also allows one to determine the extent to which normal
function may be disrupted by toxicant exposure.
The readily identifiable and predictable changes in the cytological
make-up of the vaginal smear occur as the consequence of the patterned and
dramatic fluctuations of blood estradiol concentrations that are initiated at
puberty and continue (unless interrupted by pregnancy, environmental or
experimental insult) until reproductive senescence. In fact, reproductive
senescence in the rat is characterized by the vaginal smear pattern that
predominates in later life (e.g., constant estrus or repetitively pseudopregnant).
This relationship between ovarian estrogen and vaginal cytology, as it
exists in the 4-day cycling female rat, is outlined in Figures 1 and 2. During the
periods of diestrus (as well as pregnancy and pseudopregnancy) blood
estrogen concentrations are low and the vaginal smear contains a mixture of

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cell types with a predominance of leukocytes and a few scattered cornified
epithelial cells (figure 2a). The increase in serum estradiol concentrations
(originating from the maturing follicles) has a proliferative action on the
vaginal epithelium. On proestrus, this action of estrogen on the epithelium
produces a smear with a predominance of round, polynucleated cells that may
be dispersed or clumped (figure 2bl and 2b2). As serum estradiol levels fall
(coincident with ovulation and formation of the corporal lutea), the
proliferation of the vaginal epithelium declines (figure 2c). At this time,
cornified epithelial cells are predominant in the smear. Not depicted in Figure
1 is the transitional stage between the period of estrus to diestrus in which
there is a large number of both cornified cells and leukocytes. This period,
identified as metestrus by many investigators, is brief and different forms of
cornified cells may be recognized, ranging from the typical jagged shape to a
highly rounded form such as that depicted in figure 2d. However, the
presence of leukocytes usually indicates the transition out of estrus.
Identification of these different cell types and the pattern of change occurring
during the estrous cycle are best determined by following individual females
for a number of consecutive days.
The relationship between ovarian cyclicity and vaginal cytology is
similar in the house mouse to that in the rat (figure 3). However, some
differences should be noted. Vaginal opening, which occurs about day 23 in
the mouse, does not represent puberty because several days or even weeks can

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pass before a fully cornified smear appears, signalling first ovulation. The
cycle, once underway, averages about 5 days, but is far from regular. Some of
this irregularity may be ascribed to social conditions. Female mice housed in
the same room with males tend to cycle more regularly, while females housed
in all female groups can remain anestrous for weeks (4, 5). Nevertheless,
under a controlled environment, regular daily lavages of the vagina will
harvest cells indicative of the stages of the ovarian cycle and will signal
ovulation (6, 7).
It has been suggested that the ovarian cycle of the female can be
monitored by changes in the external appearance of the vagina (8), e.g., the
vaginal orifice has a gaping appearance at estrus. While these changes can be
generally correlated with the various stages of the estrous cycle, by themselves
they have not proven to be a useful measure in the mouse (Vandenbergh,
unpublished observations).
Procedures
Consistent ovarian cycles can be detected in female rodents only if they
are housed under regular lighting conditions. Standardized photoperiods have
defined by either a 14h:10h or 12h:12h light:dark schedule. Major deviations
from these photoperiods are known to lead to alterations in the regularity of
the female's cycle. For example, constant light will lead to" a disruption of
ovulation and the appearance of a persistent or constant vaginal estrus. Under
standard lighting conditions, estrous cycles will be observed and these cycles

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will continue in a rhythmical fashion throughout adulthood (until
approximately one year of age) unless the female is mated or treated
experimentally. In addition to maintaining a proper lighting schedule, the
smears should always be collected at the same time of day.
Studies with Peromyscus mice (9) and hamsters (10, 11) suggest that
even brief flashes of light during the dark phase of the day can reset internal
pacemakers and can have an effect on reproductive rhythmicity. To be
cautious, laboratory colonies of rodents should not be transiently exposed to
brief periods of light (e.g., door opening or lights on briefly) during the dark
portion of the photoperiod. The animals respond to differences in light
intensity, not the absolute presence or absence of light. Consequently, if
nocturnal inspection is required by the experimental protocol, very dim or red
light can be left on at night.
Obtaining a smear is a simple process. Using a clean microscope slide
(e.g., serological ring slide, Scientific Products, Catalog number M6229-1) and
an eye dropper containing approximately 0.25 ml of distilled water or
physiological saline, the rat's vaginal cavity is thoroughly lavaged and the
fluid is drawn back into the eye dropper (figure 4). If the initial flush is
devoid of cells, the lavage is repeated. Caution should be exercised to ensure
that only the tip of the eye dropper is inserted into the vaginal cavity, so that
stimulation of the uterine cervix is avoided. The collected fluid is then
expelled evenly onto the microscope slide in a thin layer. The smear may be
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viewed immediately under low magnification (e.g., 100 x).
Because of the smaller size of the mouse, a Pasteur pipet (e.g., Fisher
catalog no. 13-678-6A, 15 cm long) rather than an eye dropper should be used.
To protect the vagina of the mouse from injury, the tip of each pipet should be
briefly fire polished to smooth jagged edges. The pipet is fitted with a rubber
bulb and a small quantity of saline is flushed 2-3 times in the vagina to harvest
the contents.
Daily vaginal smears should be collected from each female for a period
of at least two weeks, so that the cycling pattern can determined accurately.
Most investigators do this by recording the smears as they are taken on a daily
basis and keeping continuous records on each female. This approach also
provides the novice with the opportunity to observe ongoing fluctuations in
the various cell types as they occur. An alternative approach is to obtain the
daily smears and read them at a later time. In this case, the smears are
arranged in order on the microscope slide, allowed to dry, and periodically
fixed and stained. One technique used by Everett (12) is to stain at the end of
each week in 1% toluidine blue O (Fisher Scientific, Cat # T-161) after fixation
in 95% ethanol and removal of salt by washing in deionized water. The dye
solution must be neutral or slightly alkaline, or the cornified cells will remain
colorless. Nuclei and mucous are stained metachromatically pink. The
cytoplasm appears in various shades of blue ranging from very dark, in case of
small epithelial cells, to pale blue in the squamous cells, whether on not the
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latter are cornified. While staining is not essential to evaluate the smear, for
training purposes application of a dilute vital stain such as methylene blue to
the smear is helpful in initially characterizing the cell types. Once noted, they
are easily recognizable and staining is no longer necessary.
COMMENTS
Evaluation of Cyclicity in Toxicological Studies
Monitoring the vaginal smear of the nonpregnant female provides a
useful ancillary measure in toxicology studies. Properly used, this information
can serve to reduce much of the variance associated with a variety of measures
that fluctuate over the cycle. Furthermore, characterization of the vaginal
smear pattern offers information about ovarian status prior to treatment and
provides a way to make certain that only regularly cycling females are
assigned to control and experimental groups (e.g., usually defined as the
presence of consecutive cycles for 2-3 weeks prior to study). Regardless of the
strain of rat or mouse, regular cycling may not necessarily occur in all young-
adult animals. The incidence of aberrant vaginal smear patterns (indicative of
altered ovarian activity) may be as high as 20-30% with certain commercial
shipments, while at other times it may be as low as 5%. In either case,
including non-cycling animals in an experiment, even when randomly assigned
to treatment groups, could well increase the variance for many measures.
Knowledge of the vaginal smear prior to treatment also provides a point
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of comparison immediately after the initiation of treatment and for any
subsequent changes that may ensue. In chronic studies, an awareness of how
ovarian cycling is altered with age is important, as most strains show an
age-related disruption of cyclicity at approximately 12 months of age. For
example, female Long Evans hooded rats assume a pattern of constant vaginal
estrous at this age. Thus, treatment-induced alterations in this normal,
age-associated alteration in cycling would be important.
Another reason for keeping a daily record of cycling status would be to
synchronize the time of data collection. By killing the females at the same
stage of the cycle, variability inherent in the ovarian cycle would be minimized
and the probability of detecting an effect would be enhanced. For example,
euthanasia at specific times on the day of vaginal proestrus would yield
information about the preovulatory rise in circulating levels of estrogen,
progesterone and luteinizing hormone and/or the status of the preovulatory
follicle. An examination of the females during late estrus/early diestrus
(metestrus) would allow one to assess the presence of fresh corpora lutea and
whether or not ovulation has occurred (e.g., see Perreault and Mattson, this
volume).
Daily vaginal smear data also provide the following types of infor-
mation: (a) presence of a copulatory plug or sperm after mating. In the
mouse, for example, a copulatory plug is found by daily inspection of the
vagina in approximately 80% of breeding females, (b) determination of
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pregnancy (or continuation of cycling after mating), (c) distinguishing
pregnancy from pseudopregnancy, based on the number of days smear
remains leukocytic (e.g., pseudopregnancy = approxximately 14 days,
pregnancy = 21-22 days), and (d) indications of fetal death and resorption by
the presence of blood in the smear after day 12 of gestation.
In studies evaluating the effect of a single treatment on female
reproductive function, knowledge of the animal's cycling pattern is critical, as
a toxicant may affect the reproductive process differently at any of the
numerous stages of follicular development, ovulation, egg transport and
implantation. For example, ovulation may be advanced by treatment with
estrogen on diestrus I (13). On the other hand, treatment with various
compounds on the day of vaginal proestrus, administered during the so-called
critical period, have been shown to affect the characteristics of the LH surge,
and subsequently ovulation (see chapter by Goldman and Cooper, this
volume). Anesthetics (12), pharmacological compounds that alter brain
noradrenergic or cholinergic neurotransmission (12, 14) and pesticides (15, 16)
have all been shown to block the LH surge and delay ovulation (Goldman and
Cooper, this volume). Single treatments with the same dose of these
compounds at other times during the estrous cycle are without effect on the
LH surge or ovulation. In the cycling female, the vaginal smear is used to
determine when the female's oviduct can be examined for the presence of
oocytes (i.e., on the morning of vaginal estrous). Oocytes may not be detected
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in cycling animals in whom (a) the timing of the LH surge has been altered
(17), (b) follicular rupture has been blocked without affecting luteinization (18),
and (c) the rate of oocyte transport has been altered by treatment with
compounds such as the weakly estrogenic pesticide, methoxychlor (19).
Compound-Induced Disruptions in Cyclicity
A compound that disrupts ovarian cycling (and thus the female's
fertility potential) could induce a pattern of constant vaginal estrus, repetitive
pseudopregnancies or anestrus conditions. Constant vaginal estrus usually
indicates that the female cannot achieve an ovulatory surge of LH. The
ovaries of such females are polyfollicular and contain no corpora lutea (20).
Serum estradiol concentrations are appreciable and progesterone concentration
is minimal in the CE female (21). A pattern of constant vaginal estrus may be
induced by compounds that interfere with the neuroendocrine control of
ovulation (22). the delayed anovulatory syndrome (23) is typified by a
constant estrus pattern that develops after puberty following neonatal
treatment with estradiol, diethylstilbestrol or a variety of estrogenic pesticides
(24).
Interestingly, the constant estrous female may be sexually receptive and
ovulation may be induced upon mating (25), but the fertility of such matings
has not been thoroughly evaluated. In our laboratory, we found that although
young-adult, spontaneously constant estrous females rats did mate readily,
pregnancy outcome was significantly reduced, in that only 25 % became
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pregnant and those that did had reduced litter sizes (N=15, mean = 5.57 ± 1.43
vs. control N = 22, mean = 13.4 ± 0.9, Cooper unpublished).
Long-term exposure of adult female mice to estrogen has many
deleterious effects on vaginal cytology, as well as neuroendocrine function (26).
Chronic estradiol treatment of C57BL/6J female mice selectively impairs the
ability of the vagina to produce cornified epithelial cells. Vaginas of female
mice exposed for 3 to 5 months to either high or low levels of estradiol fail to
cornify in response to new estradiol implants. Vaginal cornification was also
significantly reduced in 23 month-old mice. These results suggest that both
age and chronic exposure to estradiol impair estradiol-induced vaginal
cornification.
Successful pregnancy in the female rat and mouse depends upon two
sets of physiological events: (a) transport of gametes through the reproductive
tract so that fertilization can be effected and (b) establishment of an
appropriate hormonal environment (progestational state), through cervical
stimulation, so that the fertilized egg can implant in the uterus and be
maintained during subsequent gestation. Pseudopregnancy is actually an
endocrine pregnancy that can be experimentally induced by stimulating the
uterine cervix of the female (normally a consequence of male intromission
behavior) on diestrus II, the day of vaginal proestrus or estrus (27). The
pseudopregnant female's ovaries contain several prominent corpora lutea and
the uterus is well- developed. The vaginal smear of such females is
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predominantly leukocytic for 12-16 days.
Anestrus is indicated when the vaginal smear remains leukocytic
indefinitely. The ovaries of the anestrus female are atrophic, with few primary
follicles and an unstimulated uterus (20). Serum estradiol and progesterone
are minimal. Anestrus, or prolonged vaginal diestrus, may be indicative of
compounds that interfere with follicular development or deplete the pool of
primordial follicles (28, 29).
The persistence of regular vaginal cycles following treatment does not
necessarily indicate that the compound is not a reproductive toxicant, since
such cycles may be anovulatory. Thus, the follicles may be luteinized without
rupturing, such as those observed following treatment with anti-inflammatory
agents (18). A compound may adversely affect the oocyte itself, the transport
of the oocyte once it is released, or the processes involved in fertilization,
implantation and pregnancy maintenance (30). Irregular cycles may reflect
impaired ovulation, as delayed ovulation may extend the period of vaginal
cornification (e.g., 2-3 days). Such cycles would be typical in animals exposed
to anesthetics (i.e., phenobarbital or pentobarbital) or noradrenergic blocking
compounds during the critical period for the neural trigger of the LH surge
.(12, 15, 31, 32). .
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References
1.	C.R. Stockard, and G.N. Papanicolaou, Am. J. Anat. 22, 225 (1917).
2.	E. Allen, Am. J. Anat. 30 297 (1922).
3.	J. A. Long, and H. M. Evans, The oestrous cycle in the rat and its
associated phenomena. Memoirs. University of California, 6, 1 (1922).
4.	M. K. McClintock, In "Pheromones and Mammalian Reproduction", (J.
G. Vandenbergh, ed.), p. 113, Academic Press, N. Y. (1983).
5.	J.G. Vandenbergh, In "Pheromones and Mammalian Reproduction", (J.
G. Vandenbergh, ed.), p. 95, Academic Press, N. Y. (1983).
6.	R. Rugh, In "The Mouse. Its Reproduction and Development." Burgess
Pub Co., Minneapolis MN (1968).
7.	J.G. Vandenbergh, Endocrinology 84, 658 (1969).
8.	A. K. Champlin, D. L. Dorr, and A. H. Gates, Biol Reprod 8, 491,
(1973).
9.	H. Underwood, J. M. Whitsett, and T. G. CXBrian, Biol. Reprod. 32, 947
(1985).
10.	J. A. Elliott, In "Biological Clocks in Seasonal Reproductive
Cycles", B. K. & D. E. Follett, eds.), p. 203, Academic Press,
N.Y. (1981).
11.	K. Hoffmann, J. Comp. Physiol. 148, 529 (1982).
12.	J. W. Everett, "Neurobiology of Reproduction in the Female Rat."
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Springer-Verlag, New York (1989).
13.	L. C. Krey and J. W. Everett Endocrinology 93, 377 (1973).
14.	S. V. Drouva, E. Laplante, and C. Kordon, Eur. J. Pharm. 81, 341 (1982).
15.	J. M. Goldman, R. L. Cooper, T. L. Edwards, G. L. Rehnberg, W. K.
McElroy, and J. F. Hein, Pharmacol. Toxicol. 68, 131 (1991).
16.	R. L. Cooper, R. W. Chadwick, G. L. Rehnberg, J. M. Goldman, K. C.
Booth, J. F. Hein, and W. K. McElroy, Toxicol. Appl. Pharmacol. 99, 384
(1989).
17.	P. van der Schoot, J. Endocrinol. 69, 287 (1976).
18.	R. F. Walker, L. W. Schwartz, and J. M. Manson Toxicol. Appl.
Pharmacol. 94, 266 (1988).
19.	A. M. Cummings and S. D. Perreault, Toxicol. Appl. Pharmacol. 102
110 (1990).
20.	H. H. Huang, and J. Meites, Neuroendocrinology 17, 289 (1975).
21.	H. H. Huang, R. W. Steger, J. F. Bruni, and J. Meites,
Endocrinology 103, 1855 (1978).
22.	R. F. Walker, R. L. Cooper, and P. S. Timiris, Endocrinology 107,
249 (1980).
23.	R. A. Gorski, Endocrinology 82, 1001 (1968).
24.	L.E. Gray, Jr. In "Aging and Environmental Toxicology: Biological
and Behavioral Perspectives," (R.L. Cooper, J.M. Goldman and T.J.
Harbin, eds.), p. 183. John Hopkins Univ. Press, Baltimore, (1991).
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25.	K. Brown-Grant, J. M. Davidson, and F. Grieg, Endocrinology 57, 7
(1973).
26.	A. J. Adler, and J. F. Nelson, Biol. Reprod. 38, 175 (1988).
27.	J. Terkel, J. A. Witcher, and N. T. Adler, Eur J. Pharm 81, 341
(1990).
28.	J. J. Heindel, P. J. Thomford, and D. R. Mattison, In "Growth
Factors and the Ovary," (A.N. Hirshfield, ed.), p 421. Plenum, New
York, (1989).
29.	R. L. Dobson, and J. S. Felton, Am. J. Ind. Med. 4, 175 (1983).
30.	A. M. Cummings, Fund. Appl. Toxicol. 15, 571 (1990)
31.	N. W. Fugo, and R. L. Butcher, Fertil. Steril. 17, 804 (1966).
32.	P. F. Terranova, Biol Reprod 23, 92 (1980).
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Figure Legends
Figure 1. Schematic of the rat four-day estrous cycle depicting relationship
among vaginal cytology, serum estradiol, and serum LH ( m = midnight).
Figure 2. Photomicrographs (100X) showing major changes in the vaginal
smear during the estrous cycle in the rat. The absolute number of cells may
vary from sample to sample. A. Diestrus, typically represented by many
leukocytes which may or may not be mixed with varying numbers of larger
comified epithelial cells. B. Proestrus, is signalled by the appearance of
rounded polynucleated epithelial cells which may initially occur as wispy or
stringy aggregates (bl) and later as numerous clumps Cb2). C. Estrus,
represented by the predominance of comified epithelial cells. D. Metestrus,
represented by dispersed, round, non-nucleated cells. Leukocytes are
frequently present during this period and show considerable variations in
number.
Figure 3. Photomicrographs (100X) showing major changes in vaginal
exfoliative cytology during the estrous cycle in the female house mouse. The
lavage in the mouse is taken with a fire polished Pasteur pipette rather than
eye dropper in the rat. The absolute number of cells may vary from sample
sample, and a female may remain in phase of the cycle, usually diestrus, for
several days. A. Diestrus, represented by leukocytes, often in large number,
16

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variable number nucleated epithelial cells and few to no comified epithelial
cells. B. Proestrus, represented by a variable number of leukocytes and a large
number of nucleated epithelial cells with few to no cornified cells. C. Estrus,
represented by the absence of leukocytes, few or no nucleated epithelial cells
and many flat cornified epithelial cells. D. Metestrus, represented by the
infiltration of leukocytes, little or few nucleated epithelial cells, and a variable
number of cornified cells, often beginning to "roll".
Figure 4. Common technique used for vaginal lavation. An alternative
method is to elevate the female's hindquarters by holding the base of her tail
and lifting slightly.
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Disclaimer
The material described in this article has been reviewed by the Health
Effects Research Laboratory, U.S. Environmental Protection Agency and
approved for publication. Approval does not signify that the contents
necessarily reflect the views and policies of the Agency, no does mention of
trade names or commercial products constitute Agency endorsement or
recommendation for use.
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Acknowledgements
We would like to thank Tammy E. Stoker and Michelle Barrett,
ManTech Environmental Technology, R.T.P., N.C. and Ms. Kimberly A.
Higgins, North Carolina State University for their excellent technical assistance
in preparing the photomicrographs.
19

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TECHNICAL REPORT DATA
(Please read Instructions on the reverse before complet
1. REPORT NO. 2.
EPA/600/A-92/081
3
4. TITLE AND SUBTITLE
Monitoring of the Estrous Cycle in the Laboratory
Rodent by Vaginal Lavage
5. REPORT DATE
6 PERFORMING ORGANIZATION CODE
7. AUTHOR(S)
RL Cooper, JM Goldman, JG Vandenbergh
8. PERFORMING ORGANIZATION REPORT NO.
9. PERFORMING ORGANIZATION NAME AND ADDRESS
Reproductive Toxicology Branch
Developmental Toxicology Division, HERL,EPA,RTP, NC;
North Carolina State Univ, Dept Zoology, Raleigh,NC
10. PROGRAM ELEMENT NO.
ANNA1E CWGH1A ACSL1A
1 1. CONTRACT/GRANT NO.
68-02-4450
12. SPONSORING AGENCY NAME AND ADDRESS
Health Effects Research Laboratory
Office of Research & Development, USEPA
Research Triangle Park, NC 27711
13. TYPE OF REPORT AND PERIOD COVERED
14. SPONSORING AGENCY CODE
EPA/600/10
15. SUPPLEMENTARY NOTES
16. ABSTRACT
^Ovarian cyclicity in a number of laboratory species can be monitored easily and
noninvasively by observing changes in the vaginal cytology. This chapter describes
the techniques used to collect data in the laboratory rat and mouse and how to
interpret the lavages as they are obtained. The relationship between the various
cell types and the fluctuation in serum hormones as they exist over the cycle is
described. Finally, there is a discussion of how to interpret various changes in
vaginal smear patterns as they occur in response to toxicant treatment.
17, KEY WORDS AND DOCUMENT ANALYSIS
a. DESCRIPTORS
b. IDENTIFIERS/OPEN ENDED TERMS
c. cosati Field/Group
Ovary
Vaginal Cytology
Reproductive Toxicology
Female

18. DISTRIBUTION STATEMENT
RELEASE TO PUBLIC
19. SECURITY CLASS (This Report)
UNCLASSIFIED
21 NO. OF PAGES
24
20. SECURITY CLASS (This page)
UNCLASSIFIED
22. PRICE
EPA Form 2220-1 (Rev. 4-77) previous edition is obsolete

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