Valuing Environmental Health Risk Reductions to Children
OPENING REMARKS BY
MARIANNE L. HORINKO
ACTING ADMINISTRATOR OF THE U.S. ENVIRONMENTAL PROTECTION AGENCY
A WORKSHOP SPONSORED BY THE U.S. ENVIRONMENTAL PROTECTION AGENCY'S NATIONAL
CENTER FOR ENVIRONMENTAL ECONOMICS (NCEE), NATIONAL CENTER FOR ENVIRONMENTAL
RESEARCH (NCER), AND OFFICE OF CHILDREN'S HEALTH PROTECTION; AND THE UNIVERSITY OF
CENTRAL FLORIDA
October 20-21, 2003
Washington Plaza Hotel
Washington, DC
Prepared by Alpha-Gamma Technologies, Inc.
4700 Falls of Neuse Road, Suite 350, Raleigh, NC 27609

-------
ACKNOWLEDGEMENTS
This report has been prepared by Alpha-Gamma Technologies, Inc. with funding from the National Center for
Environmental Economics (NCEE). Alpha-Gamma wishes to thank NCEE's Kelly Maguire and Project Officer Nicole
Owens for their guidance and assistance throughout this project.
DISCLAIMER
These proceedings are being distributed in the interest of increasing public understanding and knowledge of the issues
discussed at the workshop and have been prepared independently of the workshop. Although the proceedings have been
funded in part by the United States Environmental Protection Agency under Contract No. 68-W-01-055 to Alpha-
Gamma Technologies, Inc., the contents of this document may not necessarily reflect the views of the Agency and no
official endorsement should be inferred.
2

-------
Remarks for Marianne L. Horinko
Acting Administrator of the U.S. Environmental Protection Agency
at the
Valuing Environmental Health Risk Reductions to Children Forum
Washington, D.C.
October 20, 2003
Thank you, Jessica (Furey), for that introduction. It is a pleasure to be here with you for
the 9th Economy and Environment workshop - again highlighting a great partnership between
EPA's Office of Policy, Economics and Innovation, Office of Research and Development, and
our co-sponsors, the Office of Children's Health Protection and the University of Central
Florida. For the second time, this series will focus on the importance of children's health
protection - a topic that we are taking very seriously at EPA, especially as we recognize
Children's Health Month during October. This month - and this forum - are excellent
opportunities for us to discuss how we can use economic analyses to improve our decision
making in the areas that affect children's health.
I'm not an economist, but those at EPA who are tell me that "environmental economists"
approach the problem of valuing health risk reductions differently than "public health
economists." I suspect that is true for children's risks as well, and it brings to mind what George
Bernard Shaw once said: "If all economists were laid end to end they would not reach a
conclusion." While they may not agree on a conclusion, economists provide vital information
that informs policy decisions throughout the agency.
At EPA we consider economics a science, just as biology, toxicology, and chemistry are
sciences. And, it is critical that we produce sound science that will help lead to quality
decisions. Part of the scientific process is to engage in debate and disagreement. That is how
progress is made, and it is why we are here. I encourage you to continue to engage on these
critical issues, even when serious disagreement exists among - and within - branches of
economics.
I will admit that I am frustrated when our economists say we lack the methods and
research necessary to value specific benefit categories. Often times we use proxies or no
valuation at all. We have to change that, and your work is crucial to gaining the information we
need to do so. Your research provides the underlying information needed to support sound
analyses and decision making. I am proud that our STAR grants program in the Office of
Research and Development provides support to some of you to help us fill these critical
knowledge gaps.
In the end, of course, there is far more agreement than disagreement. Indeed, the
underpinnings of benefit-cost analysis, discounting, willingness-to-pay, and other economic
3

-------
concepts are now fundamental principles in textbooks. So it seems clear to me that tackling the
problem of valuing health risk reductions to children is a situation where better communication
among the various fields of economics will lead us to the quality decisions we want and need for
our children. This conference will certainly get us closer to that goal. In fact, it is a goal that we
have already been working toward for several years at EPA.
Protecting children is an important part of EPA's mission, and finding ways to value risk
reductions for this vulnerable segment of society is critical to this work. As you may know, EPA
does not account for age differences when estimating benefits for policy-making. However, we
are committed to furthering our understanding of both the science and the valuation components
of our decisions - which is why in 2003, EPA spent $18.4 million on children's health risk
research.
We do know that children are not just little adults. As you know, their neurological,
immunological, and digestive systems are still developing. They eat more food, drink more
fluids, and breathe more air than adults in proportion to their body mass. And, children's
behavioral patterns - such as crawling and placing objects in their mouths - may result in greater
exposure to environmental contaminants. Because of these characteristics, children may not be
sufficiently protected by regulatory standards that are based on risks to adults.
That does not mean that we have not made real progress in protecting children. For
example, largely due to removing lead from gasoline, the median concentration of lead in blood
of children under the age of 5 dropped by 85 percent between 1976 and 2000. This is important
because lead exposure can result in lowered intelligence, impaired hearing, hyperactivity, and
other adverse health effects. Building on the progress made by removing lead from gasoline,
EPA spends $2.5 million annually on regulation development and public education to further
reduce children's exposure to lead. Clearly, all of our efforts over the last three decades are
working.
I hope that several years from now, the same will be said for our ongoing efforts to
reduce childhood asthma. Between 1980 and 1995 the percentage of children with asthma
doubled from 3.6 percent to 7.5 percent - today more than 6.3 million children under the age of
18 have this disease that forces them to miss 14 million days of school each year. The Clean
School Bus USA initiative - which EPA launched last spring - will help clean up emissions
from buses and reduce asthma symptoms by eliminating unnecessary idling, replacing older
buses with new ones, and equipping buses with advanced emission control technologies. This
program seeks to make sure that every public school bus on the road in all 50 states will be a
clean school bus by 2010. They will emit less pollution, contribute to cleaner air, and - most
importantly - keep our kids safe and healthy on the way to school.
Clean School Bus USA is the newest in a long line of EPA initiatives designed to
improve the lives of children suffering the health - and social - impacts of asthma. Tools for
Schools, Smoke Free Homes, and the President's Clear Skies Initiative - along with a request for
a $3 million increase in the President's FY 04 budget for children's health research - will
4

-------
continue to help us combat this epidemic.
The cases of lead and asthma show that when it comes to environmental health risks,
children need special attention. We have targeted children by focusing on things like school
buses, ventilation systems, and educational initiatives for families - but we must go further.
During my time as Acting Administrator - and in my former role as Assistant Administrator for
OSWER - I have seen that it is tremendously difficult to reflect children's issues in our
regulatory process - but I think we can all agree that we must continue to try.
As we work to develop better scientific data on how pollutants affect children, and better
regulatory analysis addressing children's risks, it is important to keep track of how well
children's health protection efforts are working. Earlier this year, EPA published the second
edition of America's Children and the Environment - a ground breaking report that shows trends
in children's environmental health. Today, I am pleased to announce the launch of the online
version of America's Children and the Environment which will house new information and data
as it becomes available and will be a valuable resource for researchers, concerned citizens, and
policymakers. I am also pleased to announce the release of the Children's Health Valuation
Handbook, which is a companion to EPA's Guidelines for Preparing Economic Analyses that
was released in 2002. The Handbook is a reference tool for those conducting economic analyses
of EPA policies that are expected to affect children's health, providing information on how we
might incorporate the unique risks to children into our analyses.
This is where you come in. Your research and the work you are presenting over the next
two days will be invaluable for our ability to further understand this process and ensure that we
develop policies that are protective of everyone - not just adults. It is a true measure of our
society how well we protect those who are the least able to protect themselves. I fully anticipate
that your presentations will challenge and inspire us to continue tackling these difficult issues in
an effort to improve public policy - and the lives of our children.
Thank you.
5

-------
Valuing Environmental Health Risk Reductions to Children
PROCEEDINGS OF
SESSION I: THE STATUS OF CHILDREN
A WORKSHOP SPONSORED BY THE U.S. ENVIRONMENTAL PROTECTION
AGENCY'S NATIONAL CENTER FOR ENVIRONMENTAL ECONOMICS (NCEE),
NATIONAL CENTER FOR ENVIRONMENTAL RESEARCH (NCER), AND OFFICE
OF CHILDREN'S HEALTH PROTECTION; AND THE UNIVERSITY OF CENTRAL
FLORIDA
October 20-21, 2003
Washington Plaza Hotel
Washington, DC
Prepared by Alpha-Gamma Technologies, Inc.
4700 Falls of Neuse Road, Suite 350, Raleigh, NC 27609

-------
ACKNOWLEDGEMENTS
This report has been prepared by Alpha-Gamma Technologies, Inc. with funding from
the National Center for Environmental Economics (NCEE). Alpha-Gamma wishes to
thank NCEE's Kelly Maguire and Project Officer Nicole Owens for their guidance and
assistance throughout this project.
DISCLAIMER
These proceedings are being distributed in the interest of increasing public understanding
and knowledge of the issues discussed at the workshop and have been prepared
independently of the workshop. Although the proceedings have been funded in part by
the United States Environmental Protection Agency under Contract No. 68-W-01-055 to
Alpha-Gamma Technologies, Inc., the contents of this document may not necessarily
reflect the views of the Agency and no official endorsement should be inferred.
11

-------
TABLE OF CONTENTS
Session I: The Status of Children
Changes in the Lives of U.S. Children: 1990-2000
Kristin Smith, U.S. Census Bureau	1
America's Children and the Environment: Measures of Contaminants,
Body Burdens, and Illnesses
Dan Axelrad, U.S. EPA, National Center for Environmental Economics	29
The National Children's Study
Peter Scheidt, NIH, National Institute of Child Health and Human
Development	53
Environmental Health Valuation for Children: Research in Europe
Nick Johnstone, Organisation for Economic Cooperation & Development	73
NOTE: Although Mr. Johnstone was unable to deliver this scheduled
presentation at the workshop, we are including his slide files in the
proceedings.
Summary of Q&A Discussion Following Session 1	87
in

-------


Changes in the Lives of U.S. Children:
1990-2000
Kristin Smith
Robert Kominski
Julia Overturf

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
1

-------
Introduction
M
ii
\a|C
¦ Once every ten years, the decennial census
provides the opportunity to generate snapshots of
the population for very small geographic units.
Much more than a complete count of the nation's
population, the census provides important social,
economic and housing detail about the population,
allowing policy-makers and planners to see how
characteristics have changed over time in cities,
towns and neighborhoods.

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002

-------
ffrfm

¦ Over the past decade there has been renewed
interest on the well-being of children.
Spearheaded by federal activities such as the
Federal Interagency Forum on Children and
Families, much of the focus has been on
identifying a variety of indicators of children's
well-being.

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002

-------
M
ii
¦ In this presentation, I present nine indicators of
child well-being from the 1990 and 2000
decennial Censuses. What is unique about this
presentation is that data for the items highlighted
here are shown for all fifty states and the District
of Columbia. Doing so allows one to see the
variability that exists across the Nation, as well as
providing details of change during the past decade.

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002

-------

Population and Family Characteristics

1
¦	Children living in married-couple families
¦	Children with difficulty speaking English
¦	Children who are foreign-born

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002

-------
Children Living in Married-Couple Families
1990-2000 Change
Percentage point change
~ Decrease of up to 4.2 (16)
Decrease of 4.2 to 5.6 (19)
Q Decrease of 5.6 or more (16)
National average: decrease of4.2
NOTE: Includes children under 18 in households who are not householders, subfamily reference persons or their spouses. Children in married-c
biological, adopted, and step sons and daughters of a married householder or a married subfamily reference person.
SOURCE: U.S. Census Bureau, 1990 and 2000 Censuses.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
6

-------
Children Living in Married-Couple Families
In 2000, 68 percent of children under 18 years old lived in
married-couple families, down from 72 percent in 1990.
The decline in children living in married-couple families
occurred in all states in the nation, with several states
showing a decrease of about 7 percentage points. New
Jersey had a small decrease (1.7 percentage points) from
74 percent in 1990 to 72 percent in 2000.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
1

-------
Children With Difficulty Speaking English
1990-2000 Change
Percentage point change
| Increase of 1.4 or more (14)
~	Increase of up to 1.4 (31)
~	No significant change (6)
National average: increase of 1.4
NOTE: Includes children under 18 in households who are not householders, subfamily reference persons or their spouses. Children with difficulty speaking English
speak a language other than English at home and speak English less than 'very well.' This includes those who speak English 'well,' 'not very well,' or'not at all.'
SOURCE: U.S. Census Bureau, 1990 and 2000 Censuses.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002

-------
Children With Difficulty Speaking English
Nationally, the proportion of children ages 5 to 17 with
difficulty speaking English increased from 5 percent in
1990 to 7 percent in 2000.
Most states experienced such an increase, with the largest
percentage point increase occurring in Nevada, where it
rose from 4 percent in 1990 to 9 percent in 2000.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
9

-------
Children Who Are Foreign-born
1990-2000 Change
F
Percentage point change
~	Increase of 1.1 or more (28)
| Increase of up to 1.1 (20)
~	No significiant change (2)
~	Decrease	(1)
National average: increase of 1.1
NOTE: Includes children under 18 in households who are not householders, subfamily reference persons or their spouses.
Foreign-born children were not born in the 50 states, the District of Columbia, U.S. outlying territories, or abroad to American parents.
SOURCE: U.S. Census Bureau, 1990 and 2000 Censuses.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
10

-------
Children Who Are Foreign-born
In 2000, 4 percent of children living in the United States
were foreign-born, up from 3 percent in 1990.
Five states had increases of 2.5 percentage points or more
in the proportion of foreign-born children: Arizona,
Colorado, Nevada, Oregon and Washington - all of which
were also states that saw their percentages of children with
difficulty speaking English increase.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
11

-------



	RM
Children in families in poverty
Children in crowded housing
Children living with a full-time employed parent

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
12

-------

Percentage point change
~	Increase	(6)
~	No significant change (6)
~	Decrease of up to 1.7 (12)
~	Decrease of 1.7 or more (27)
National average: decrease of 1.7
NOTE: Includes children under 18 in households who are not householders, subfamily reference persons or their spouses. Child poverty includes
children living in households who are related to the householder and whose family income and family size put the child
below the poverty threshold. Poverty data collected in the 1990 and 2000 censuses refers to poverty in calendar year 1989 and 1999, respectively.
The average poverty threshold for a family of four was $12,674 in 1989 and $17,029 in 1999.
SOURCE: U.S. Census Bureau, 1990 and 2000 Censuses.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
13

-------



	I'vii
\iljj
Child poverty decreased for the nation as a whole from 18
percent in 1990 to 16 percent in 2000.
Louisiana and Mississippi had large declines in child
poverty over the 1990s, even though they had the highest
levels among the states in 2000. Despite a decrease in
child poverty for the nation as a whole and for many
individual states, child poverty increased significantly over
the decade in five states and the District of Columbia.
|
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
14

-------
Percentage point change
|_J Increase of 2.8 or more (13)
I n c re a s e of u p to 2.8 (26)
|_| No significant change (4)
~ Decrease	(8)
National average: increase of 2
NOTE: Includes children under 18 in households who are not householders, subfamily reference persons or their spouses.
Children living in crowded housing live in a house where the number of persons per room is greater than 1.
SOURCE: U.S. Census Bureau, 1990 and 2000 Censuses.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002

-------
In 2000, 19 percent of children lived in crowded housing,
up from 16 percent a decade earlier.
¦	In Nevada, a rapidly-growing state, the proportion of
children living in crowded housing increased 7.6
percentage points over the decade, from 19.7 percent in
1990 to 27.3 percent in 2000, the largest increase in the
nation.
¦	However, the largest decrease was found in Texas, where
the rate decreased from 25 percent in 1990 to 15 percent in
2000.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
16

-------

Percentage point change
¦ Increase of 7.0 or more (9)
~	Increase of 5.7 to 7.0 (17)
O Increase of up to 5.7 (24)
~	Decrease	(1)
National average: increase of 5.7
NOTE: Includes children under 18 in households who are not householders, subfamily reference persons or their spouses. Children living with an employed parent areth
never-married biological, adopted, and stepsons and stepdaughters of a householder or a subfamily reference person, living with one or two parents who are employed
and working at least 35 hours per week.
SOURCE: U.S. Census Bureau, 1990 and 2000 Censuses.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
17

-------
In 2000, 83 percent of children lived in families with at
least one parent employed full time, up from 77 percent in
1990.
The largest gain in parental employment was found in
Michigan where the rate of children living in families with
an employed parent rose from 73 percent in 1990 to 84
percent in 2000.
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
18

-------

Education

	I'vii
3- to 5-year olds enrolled in school

18- to 24-year olds who have completed high
school

16- to 19-year olds not working nor enrolled in
school

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002


-------
3- to 5-Year Olds Enrolled in School
1990-2000 Change
Percentage point change
| Increase of 22.0 ormor^10)
¦ Increase of 19.4 to 22.0(18)
| Increase of up to 19.4 (23)
National average: increase of 19.4

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
20

-------

3- to 5-Year Olds Enrolled in School

	I'vii
\iljj
Nationally, the proportion of children ages 3 to 5 enrolled
in early education rose from 42 percent in 1990 to 61
percent in 2000, representing an increase of 19 percentage
points.
The figure clearly shows the geographic variation in early
education among children ages 3 to 5, with most of the
smaller increases clustered among the Western states.
Georgia, a Southern state, had the largest increase from 41
percent in 1990 to 67 percent in 2000.

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
21

-------
18- to 24-Year Olds Who Have Completed
High School
1990-2000 Change
Percentage point change
¦ Increase	(13)
~	No significant change (5)
~	Decrease of up to 1.2 (8)
~	Decrease of 1.2 or more (25)
National average: decrease of 1.2 I
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
22

-------
18- to 24-Year Olds Who Have Completed
High School
Nationally, the percentage of people ages 18 to 24 who had
completed high school dropped from 84 percent in 1990 to
82 percent in 2000. Part of this decrease was related to
changes in the demographic composition of this age group,
particularly with respect to the Hispanic population.
Declines occurred in many states where the proportion of
young Hispanics in the population increased, such as in
Arizona, Colorado, Nevada, and North Carolina.
Twelve states and the District of Columbia experienced
increases in high school completion rates. The rates in
California and West Virginia increased about 3 percentage
points from 1990 to 2000.	
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
23

-------
16- to 19-Year Olds Not Working Nor Enrolled
in School
1990-2000 Change
Percent point change
| Increase	(6)
~ No significant change (14)
H Decrease of up to 0.9 (10)
Q Decrease of 0.9 or more (21)
National average: decrease of 0.9
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
24

-------
16- to 19-Year Olds Not Working Nor Enrolled
in School

	 —l!(ii
In 2000, 9 percent of youth ages 16 to 19 neither worked
nor attended school, representing a decrease from 10
percent in 1990.

Several states experienced decreases of around 2
percentage points. In contrast, the rate significantly
increased in only six states (Colorado, Delaware, Hawaii,
North Carolina, South Carolina, and South Dakota).

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002

25

-------
Additional Data
M
ii
¦	Future U.S. Census Bureau reports on child well-being will
build upon the data presented here today and incorporate
other data to more fully portray changes in the lives of
U.S. children.
¦	Census report to be published by end of 2003,
"Children and the Households They Live In: 2000"
using Census 2000 data.
¦	Census report to be published in 2004 comparing 1990
and 2000 Census data on more indicators.
¦	Many more indicators of child well-being are available for
analysis at many geographic levels (Nation, State, MSA,
county, tract, etc.).

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
26

-------
Other available data
Hispanic origin
Foreign language spoken at home
Recent immigrant
Citizenship
Metropolitan residence
Home ownership
Labor force status of parents
Household utilities (incomplete plumbing or kitchen, no
telephone or vehicle)
Teen marital status
Family structure and living arrangements
USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
27

-------
Other available data
M
ii
¦	Receipt of government assistance
¦	Multi-generational household
¦	Foreign-born parent
¦	Parent's educational status
¦	Living with a parent who is disabled
¦	Parent is a recent immigrant
¦	Living with a parent who speaks English less than very
well

USCENSUSBUREAU
Helping You Make Informed Decisions • 1902-2002
28

-------
America's Children and the Environment:
Measures of Contaminants,
Body Burdens, and Illnesses
US Environmental Protection Agency
Office of Policy, Economics and Innovation
Daniel A. Axelrad
29

-------
America's Children and the
Environment	
•	First edition - December 2000
•	Second edition - February 2003
•	Goals:
-	Identify environmental conditions and health
outcomes of greatest relevance for children
-	Identify best available data
-	Develop most informative measures
-	Identify limitations, data needs, future
directions

-------
Topics Addressed
•	Environmental Contaminants
•	Body Burdens
•	Childhood Illnesses

-------
Environmental Contaminants
•	Outdoor Air Pollutants
•	Indoor Air Pollutants
•	Drinking Water Contaminants
•	Pesticide Residues
•	Land Contaminants

-------
Criteria Air Pollutants - Exceedance of Standards
Measure E1
Percentage of children living in counties in which
air quality standards were exceeded

•		 Ozon\ /
eight-hour standard /
Ozone
one-hour standard

PM-2.5
Carbon
monoxide «


IrVl |
ArM-1 U
f 1- '	
SOURCE: U.S. Environmental Protection Agency, Office of Air and Radiation, Aerometric Information
Retrieval System
33

-------
Criteria Air Pollutants - Daily Air Quality Index
Measure E2
Percentage of children s days with good, moderate, or
unhealthy air quality
Good
No Monitoring Data
Moderate
Unhealthy
SOURCE: U.S. Environmental Protection Agency, Office of Air and Radiation, Aerometric
Information Retrieval System
34

-------
Criteria Air Pollutants - Long Term
Measure E3a
Long-term trends in annual average concentrations
of criteria pollutants
70%
PM-10, percent
of annual standard
Nitrogen dioxide,
percent of
annual standard
Sulfur dioxide,
percent of a
annual standard
SOURCE: U .S. Environmental Protection Agency, Office of Air and Radiation, Aerometric Information
Retrieval System
35

-------
Drinking Water
Measure E6
Percentage of children living in areas served by public water systems
that exceeded a drinking water standard or violated treatment requirements
Any health-based violations
Microbial "^-Treatment and filtration
contamin
Chemical and radiati
Lead and copper
Nitrate/nitri
SOURCE: U.S. Environmental Protection Agency, Office of Water, Safe Drinking Water Information System
(Percentages are estimated)
36

-------
Body Burdens
•	Concentrations of Lead in Blood
-	in children age 5 and under
•	Concentrations of Mercury in Blood
-	women of childbearing age
•	Concentrations of Cotinine in Blood
-	marker for exposure to Environmental
Tobacco Smoke
-	in children under age 18

-------
90th percentile
Median
1976-1980
1988-1991 1992-1994
SOURCE: Centers for Disease Control and Prevention, National Center for Health Statistics,
National Health and Nutrition Examination Survey
1999-2000
38

-------
Lead Body Burdens
Measure B2
Median concentrations of lead in blood of children ages 1-5, by race/ethnicity
and family income, 1999-2000
Hispanic
Black
non-
Hispanic
White
non-
Hispanic
All
Races/
Ethnicities
0	1	2	3	4	5
Median concentrations of lead in blood (ng/dL)
SOURCE: Centers for Disease Control and Prevention, National Center for Health Statistics,
National Health and Nutrition Examination Survey
> 200% of Poverty Lev
100-200% of Poverty L«
< Poverty Level
All Incomes
>200% of Poverty Leve
100-200% of Poverty L
< Poverty Level
All Incomes
> 200% of Poverty Level
100-200% Poverty Le*
< Poverty Level
All Incomes
> 200% of Poverty L^
100-200% of Poverty
< Poverty Level
All Incomes
Ivel
level
39

-------
Mercury Body Burdens
Measure B4
Distribution of concentrations of mercury in blood of women of
childbearing age, 1999-2000
Concentration of mercury in blood (parts per billion)
SOURCE: Centers for Disease Control and Prevention, National Center for Health Statistics, National Health
and Nutrition Examination Survey
Note: EPA's reference dose (RfD) for methylmercury is 0.1 micrograms per kilogram body weight per day.
This is approximately equivalent to a concentration of 5.8 parts per billion mercury in blood.
40

-------
Cotinine Body Burdens - marker for ETS exposure
Measure B5
Concentrations of cotinine in blood of children
90th percentile
SOURCE: Centers for Disease Control and Prevention, National Center for Health Statistics
National Health and Nutrition Examination Survey
41

-------
Childhood Illnesses
Scope:
Important childhood diseases and disorders that
may be influenced by exposure to
environmental contaminants
•Respiratory Diseases
•Childhood Cancer
•Neurodevelopmental Disorders
•Birth Defects (CA data only)

-------
Asthma Prevalence
	Measure D1
Percentage of children with asthma
14%
Children ever
diagnosed
with asthma
Children with asthma
in the past 12 months
Children ever
diagnosed
with asthma
and having an
asthma attack
in the past
43
SOURCE: Centers for Disease Control and Prevention, National Center for Health Statistics, National
Health Interview Survey
Note: The survey questions for asthma changed in 1997; data before 1997 cannot be directly compared to data in 1997 and later.

-------
Asthma Prevalence
Measure D2
Percentage of children having an asthma attack in the previous 12 months,
by race/ethnicity and family income, 1997-2000
> 200% of Poverty Levi si
100-200% of Poverty L ivel
< Poverty Level
All Incomes
> 200% of Poverty Levi
ll


100-200% of Poverty L
ivel


< Poverty Level
All Incomes



> 200% of Poverty Levi
si

100-200% of Poverty L
ivel

< Poverty Level


All Incomes


> 200% of Poverty Levi si
100-200% of Poverty L svel
< Poverty Level
All Incomes
SOURCE: Centers for Disease Control and Prevention, National Center for Health Statistics,
National Health Interview Survey
44

-------
ER Visits for Respiratory Effects
	Measure D3
Children's emergency room visits for asthma and other
respiratory causes
500
All asthma and
other respiratory causes
Acute upper respiratory
infections
1992	1993	1994	1995	1996	1997
SOURCE: Centers for Disease Control and Prevention, National Center for Health Statistics
National Hospital Ambulatory Medical Care Survey
45

-------
All asthma and other respiratory causes
Acute bronchitL
Acute upper respiratory infections
46

-------
Incidence
Mortality
Cancer incidence and mortality for children under 20
SOURCE: Incidence data from National Cancer Institute, Surveillance, Epidemiology and End Results Program;
mortality data from Centers for Disease Control and Prevention, National Center for Health Statistics,
National Vital Statistics System
Childhood Cancer Incidence and Mortality
Measure D5
47

-------
Mental Retardation
	Measure D7
Children reported to have mental retardation, by race/ethnicity
and family income, 1997-2000
14
White Black
non- non-
Hispanic Hispanic
< Poverty 100-200% >200%
Level of Poverty of Poverty
Level Level
SOURCE: Centers for Disease Control and Prevention, National Center for Health Statistics,
National Health Interview Survey

-------
Birth Defects in California
Measure S5
Number of birth defects in California per 1,000 live births and
fetal deaths
Heart Defects Oral Cleft Defects Down Syndrome Intestinal Defects Limb Defects
SOURCE: California Birth Defects Monitoring Program
49

-------
Conclusions
•	Areas of improvement, including:
-	reduced blood lead levels and exposure to
secondhand smoke
-	modest decreases in exposure to air
pollutants and drinking water contaminants
•	Areas of concern, including:
-	prenatal mercury exposure
-	rising prevalence of asthma
•	Much remains to be learned about how
pollutants affect children's health

-------
Acknowledgments
•	Authors:
-	Tracey J. Woodruff, US EPA
-	Daniel A. Axelrad, US EPA
-	Amy D. Kyle, UC Berkeley
-	Onyemaechi Nweke, US EPA
-	Gregory G. Miller, US EPA
•	Thank you!!! to:
-	EPA Office of Children's Health Protection
-	CDC National Center for Health Statistics

-------
AMERICA'S CHILDREN
and THE ENVIRONMENT
Measures of Contaminants, Body Burdens, and Illnesses
'My* A
•-.•v ,v ;
http://www.epa.gov/envirohealth/children

-------
THE NATIONAL
CHILDREN'S
STUDY
Peter Scheidt
Program Office and Interagency Coordinating Committee
U.S. Department of Health and Human Services
NICHD, CDC, NIEHS
U.S. Environmental Protection Agency
53

-------
Children have increased
vulnerability to environmental exposures
-> Critical windows of vulnerability during
development
-> Immature mechanisms for detoxification and
protection
-> Differences in metabolism and behavior that
may yield higher exposure in the same
environments
c

-------
55

-------
v^. Rationale for the
^J\ National Children's Study
From The President's Task Force on Health Risks and
Safety Risks to Children, 2000
-> Compared to adults, children are especially vulnerable to
environmental exposures - metabolism, behavior
-> Exposures to some agents demonstrate potential for serious
developmental effects - lead, prenatal alcohol
-> Current known exposures of high frequency - pesticides, violence,
media
-> Numerous high burden conditions with suspected environmental
contribution - learning disabilities, autism, diabetes, asthma, birth
defects, premature birth
-> Existing research too limited in size & scope to answer the
questions
-> Life-course (longitudinal) design needed to correctly link with
multiple exposures and multiple outcomes
Why Now?
Since the 1950s, many environmental factors have been
introduced (chemicals in air, food, water, and soil) to
increase the effects of the environment and its interaction
with the genetic constitution of the developing fetus and
the child. Others (DDT) have been decreased, at least in
the U.S.
Since the 1950s, many technological advances have been
made (identifying biomarkers, mapping the human
genome, computerization, etc.) that would contribute to
the ability to identify environmental risks
56

-------
ijp PL 106-310
Children's Health Act of 2000
(a)	PURPOSE- ... to authorize NICHD to conduct a national longitudinal
study of environmental influences (including physical, chemical,
biological, and psychosocial) on children's health and development.
(b)	IN GENERAL- The Director of NICHD shall establish a consortium of
representatives from appropriate Federal agencies (including the CDC
and EPA) to-
(1)	plan, develop, and implement a prospective cohort study, from birth to
adulthood, to evaluate the effects of both chronic and intermittent
exposures on child health and human development; and
(2)	investigate basic mechanisms of developmental disorders and environmental
factors, both risk and protective, that influence health and developmental
processes.
(e) AUTHORIZATION OF APPROPRIATIONS- There are authorized to be
appropriated to carry out this section $18,000,000 for fiscal year 2001,
and such sums as may be necessary for each the fiscal years 2002
through 2005.
57

-------
•f[\ Study Concepts
-> Longitudinal study of children, their families and
their environment
-> National in scope
-> Environment defined broadly (chemical, physical,
behavioral, social, cultural)
-> Study common range of "environmental"
exposures and less common outcomes
(n~ 100,000)
-> Environment & genetic expression

-------
•f[\ Study Concepts (con't)
-> State-of-the-art technology -
tracking, measurement, data
management
-^Consortium of multiple agencies
-> Extensive public-private
partnerships
-> National resource for future studies

-------
Study Population - Issues
->Generalizability to U.S. population
-> Additional study populations, e.g.
^Specific high-risk populations
^Agricultural
^Industrial
-> Economically disadvantaged
->Women of child-bearing age - possible
effects on fertility & pregnancy

-------
Criteria for Core Hypotheses
-> No single hypothesis
-> Hypothesis required for costly elements
-> Important for child health & development
(prevalence, severity, morbidity, mortality, disability,
cost, public health significance)
-> Reasonable scientific rationale
-> Require the large sample size (~100,000)
-> Measurable with study of this size
-> Requires longitudinal follow-up

-------
Jdr" Priority Outcome Areas
"fC (and example hypotheses)
-> Undesirable outcomes of pregnancy (infection and
mediators of Inflammation during pregnancy are major factors
associated with pre-term birth)
-> Neurobehavioral development (proposed -
environmentally induced biochemical and physiological
conditions of birth and infancy, including maternal
hypothyroidism, neonatal hyperbilirubinemia, and others, are
associated with learning and cogitative disabilities,.
Injury (Repeated head trauma w/o anatomic damage -
cumulative adverse effects on neurocognitive development)

-------
Jdr" Priority Outcome Areas
"fC (and example hypotheses)
-> Asthma (maternal stress during pregnancy is
associated with the prevalence and severity of asthma
in offspring)
-> Obesity and physical development
(Obesity and insulin resistance is associated with
impaired glucose metabolism in pregnancy and
interacting factors in the physical and social
environment)

-------
Associations and Interactions in
the National Children's Study
Asthma
Chemical
Expos.
Infection
Gene
expression
Social
Environ
Physical
Environ
Health
Care
Medicine &
Pharm
Birth
Defects
Development
& Behavior
Growth
Fertility &
Pregnancy
64

-------
jjp Measures Anticipated -
Exposures
-> Environmental Samples: air, water, dust
-> Bio-markers for chemicals: blood, breast milk,
hair, tissue, etc.
-> Interview and history
-> Serology and medical data
-> Housing & living characteristics
-> Family and social experiences
-> Neighborhood and community characteristics

-------
Anticipated -
-> Fetal growth and outcome of pregnancy
-> Birth defects and newborn exam
-> Growth, nutrition and physical development
-> Medical condition and history: illness (e.g.
asthma), conditions, & injuries
-> Cognitive and emotional development
-> Mental, developmental and behavioral
conditions
t<\ Outcomes
66

-------
€ Projected Time Line
2000-2004	Pilot study/methods development work
2001-2002	Form advisory committee and working groups
Periodically: Meetings, peer reviews, consultations
Mid 2003 Finalize specific hypotheses, develop study design
Mid 2005 Select initial centers or alternatives and pilot test core
protocol
Late 2005 Begin full study with vanguard centers
2005-2007 Enroll additional centers
2008-2009 First preliminary results available from pregnancy
2007-2030 Analyze data as collection continues, publish results
throughout:
67

-------
^ Potential Benefits of the NCS
for Prevention of Diseases

Condition
Potential
Reduction
Potential
Annual Benefit
Pre-term Birth
10%
$0.6 Billion
Asthma
25 %
$3.2 Billion
Obesity & Diabetes
12.5 %
$14.5 Billion
Juvenile Diabetes
15 %
$15 Billion
Injuries
10%
$39 Billion
Schizophrenia
33 %
$3.1 Billion
Learning problems
One cause comparable to lead
8%
$100 Billion

-------
c NCS - Funding Status
-> FY '02 - $6 M
FY '03 - $10 M Proposed
-> FY '04 - $26 M Estimated need
-> Funding for FY '05 & beyond to
NIH/HHS-EPA/OMB
-> Congressional appropriation uncertain

-------
c The NCS will provide
-> The answer to concerns about known exposures
during childhood to potential toxicants
-> The power to determine absence of effects or benefit
of exposures to various products important for our economy
-> Causal factors for a number of diseases and conditions
of children with suspected environmental causes
-> How multiple causes interact to result in multiple
outcomes
-> Large sample size required to apply knowledge of the
human genome to understand multifactoral genetic
conditions
-> Identification of early life factors that contribute to
many adult conditions
-> A national resource to answer future questions by
using stored biological and environmental samples and the
extensive data for decades to come

-------
Contact information
Check the Web site:
httD://NationalChildrensStudv.aov
Join the listserv for news and
communication
Contact us at ncs@mail.nih.gov

-------
It
THE NATIONAL^
CHILDREN'S
STUDY
The End
72

-------
Environmental Health Valuation


for Children:


Research in Europe

Pascale Scapecchi & Nick Johnstone


National Policies Division


OECD


OECD (( 1) OCDE

-------
Context
Much fewer studies in Europe than in the United
States
However, recent growth in interest in European
countries
OECD project on the valuation of environmental
health risks to children
OECD «# o<

-------

European studies

Very few economic European studies
valuation of children's health
on the
•
Greater emphasis on epidemiological work

•
Most European valuation studies estimate
related quality of life (HRQOL) measures
Health-
•
Only one study which estimates WTP to
children's health
protect

OECD ({(3) OCDE

-------
Valuation of Air Pollution in Europe
Navrud (2001) estimates parental WTP to avoid ill-
health episodes for their children.
Health impacts considered: asthma attacks and
coughing.
Sample: parents of children under 17 years of age.
Possibility of comparing parents' WTP for their own
health with parents' WTP for their children's health.
Mortality risk reduction is valued higher than
morbidity risk reduction.
Results show that WTP to prevent the child from
illness is higher than WTP to prevent an adult from
illness.
OECD (( 4) OCDE
76

-------
16 D Questionnaire
•	Apajasalo et al. (1996a) —- health-related quality of life (HRQOL)
measure of adolescent aged 12-15.
•	16 multiple choice questions representing one health-related
dimension (e.g. mobility, vision, hearing, breathing, etc)
•	Sample: "normal" and affected children (children waiting for organ
transplantation, children with genetic skeletal dysplasias, and
children with epilepsy).
•	New approach: adolescents fill in the questionnaire by themselves,
& questionnaire sent to their parents for comparison.
•	Main Results:
-	The profiles differ significantly according to the diagnosis.
-	The measures obtained from the children and the parents differ
-	Differences between boys and girls.
•	Conclusion: Reliable HRQOL measures of adolescents' health
should be based on data collected from the adolescents
themselves.
OECD «# OCDE

-------
17 D Questionnaire
Apajasalo et al. (1996b) -- HRQOL measure for
children aged 8-11 years.
Based on the 16D questionnaire, they construct a
measure consisting of 17 dimensions.
Sample: affected and non-affected children.
The children completed the questionnaire with the
help of an interviewer.
Similar results to the 16D study: the profiles vary
according to the diagnosis.
Reliable estimates of the HRQOL of children can be
obtained when children fill in the questionnaire by
themselves.
However, recognition that limited cognitive capacities
of young children.	oecd^Hocde
78

-------

Children's QoL Assessment
•
Manificat and Dazord (2002) assess quality of life

(QoL) among 4 to 12 year old children.
• The questionnaire covers 27 items covering the main

paediatric QoL domains, e.g. family life, social life,

children's activities and health.
•
Sample: ill and non-ill children.
•
Children have to fill in the questionnaire by

themselves.
•
Results show differences across ages, health status

and living condtions.

OECD 7>OCI)K
79

-------
Ongoing Work Programmes
The Pan-European Programme assesses the evaluation of
transport-related health impacts, with a particular emphasis on
children. Countries involved: Austria, Switzerland, France,
Malta, The Netherlands, and Sweden + WHO.
•	Children's Environment and Health Action Plan for Europe
(CEHAPE) undertaken by WHO, to tackle the environmental
risk factors that most affect European children's health and
providing concrete tools to address them. Countries involved:
Member countries.
•	The RANCH Project addresses the effects of noise on
children's cognition and health. Countries involved: the
Netherlands, Spain, Sweden and the UK.
•	Work in Norway : projects referenced in the Norwegian
Research Database (http://dbh.nsd.uib.no/nfi/english/)
OECD «# OCDE

-------
OECD Workshop
Objectives:
•	Review the state of knowledge;
•	Assess the different valuation approaches; and,
•	Highlight the needs for further research and political
action.
Structure: 4 sessions:
•	general overview of differences;
•	conceptual and methodological issues;
•	comparison of methodologies; and,
•	policy perspectives.
Key issues:
•	Unique challenges;
•	Availability of data;
•	Valuation methodology; and,
•	Benefit transfer
OECD «# 0(

-------
Findings (1)
Overview of the differences between adults and
children
-	Risk differences
•	Children are not little adults
•	Heterogeneity between children
•	Great number of uncertainties
-	Valuation differences
•	4 potential sources of valuation differences: age, risk
preferences, context of valuation, and perspective.
•	Affect estimates
•	Estimated VSL of a child > Estimated VSL of an adult
OECD (( 1(| (X

-------

Findings (2)
Conceptual and methodological issues
-
Formulation of children's preferences:

• Parental perspective most appropriate

• Application of intra-household allocation model (unitary vs. pluralistic)

• Est'd WTP to reduce risk for children > Est'd WTP to reduce risk for adults.
-
Transfer of adults' values

• Risk of under-estimation

• Adjust adults' values with relevant marginal rate of substitution between

adults' and children's health values.

Discountinq children's health

• Scarcity of relevant examples

• Time-varying discount rate may be appropriate

• Long term benefits accounted with care

Economic uncertainties

• Key sources of uncertainty: risk context, time, irreversibility, formulation of

children's preferences, valuation context and altruism.

• Children's health value included in parent's health value.

• Methodological concerns of greater importance. OECD (( 11 OCDE
83

-------
Findings (3)
Comparison of methodologies
-	Stated vs. Revealed preferences techniques
•	Stated-preferences techniques more appropriate.
•	Revealed-preferences more demanding and difficult to implement, in
particular in that context of valuation.
-	WTPvs. QALYs
•	WTP impose less restrictions on the structure of individual preferences
but are more sensitive to the respondent thinking.
•	Any standard chosen is arbitrary
•	The choice will depend upon the setting
-	Health outcome measures
•	Studies conclude that perspective of children is preferred
•	Multi-attribute utility instruments provide reliable results
•	But what is the validity of the value obtained?
OECD 0(

-------
Findings (4)
Policy perspectives
-	Children are highly vulnerable to environmental
degradation
-	Children are not little adults
-	Morbidity and mortality risks reduction greatly differ
-	Research on the valuation of children's health should
be encouraged and supported
-	More comparative studies in different countries
-	Better risk and economic assessments are required
OECD ««3 OCDE

-------
Conclusions
Misallocation of resources devoted toward children (and
between children and adults)
•	Linked to allocation (misallocation) of resources between
morbidity and mortality
•	Leads to wrong priorities being set across different
impacts and wrong standards within individual impacts
•	Much more information and research data are necessary
to provide reliable policy advice
OECD(( 14 ocde
86

-------
Summary of Q&A Discussion Following Session I
Matt Clark (EPA/NCER) asked Dr. Scheidt whether there were any economists doing
willingness-to-pay studies in association with the National Children's Study. When Dr.
Scheidt responded "No," Dr. Clark suggested that a "wealth of data that would be very
useful for policy matters" could be acquired for very little incremental cost, and he stated
that similar joint efforts have been successful in the past. He strongly urged Dr. Scheidt
to have economists participate and develop some survey materials that could augment the
patient study.
Dr. Scheidt responded that the Study group was "very open to that kind of thing" and, in
fact, has an economist/sociologist (Bob Michaels from the University of Chicago)
involved on the advisory committee, although they've "not engaged in that level of
economic study." He invited any interested economists to contact the Study group
directly.
Glenn Harrison (University of Central Florida) asked two questions of Dr. Scheidt. He
first stated that health economists can easily find studies that relate to the number of
children who have hospital stays and diagnosis codes, etc., but they often can't find links
to cost measures. Acknowledging that the costs are difficult to measure, he asked
whether the Study was going to link to existing surveys that can actually give information
on the costs of delivering healthcare. His second question concerned the issue that
"surveys of children's health . . . very often start too late"—that is, they don't provide
information on fetal deaths and infant deaths. The resultant "sample selection bias" can
make a dramatic difference to the inferences one draws.
Dr. Scheidt responded that they definitely anticipate merging and linking to appropriate
existing data sets (e.g., from the Census Bureau), and he stated that there is a white paper
and workshop in the planning phase designed to examine all identifiable data sets
relevant to the study which are candidates for linking. He said that this search is wide
open, ranging "from social data to atmospheric air pollution" data being measured
through satellite technology at NASA.
Dr. Scheidt acknowledged the concern with the question on fetal deaths and particularly
with health risk issues from the critical period of early in the first trimester onward.
Citing the logistical and economic difficulties of following the entire sample of those of
child-bearing age from pre-pregnancy on, he stated that they at least hoped to track
women whose children are enrolled in the study and who subsequently become pregnant.
Kerry Smith (North Carolina State University) commented on the difficulty, partly due to
confidentiality reasons, in linking spatial data (i.e., latitude and longitude) to the
households involved in the surveys, and he asked Dr. Scheidt to pay attention to getting
economists access to that information, if possible. When, Dr. Scheidt asked for clues
about how that might be accomplished, Dr. Smith clarified that what is needed is "a
87

-------
convenient way in which researchers can ask for the matching to take place by a third
party." He acknowledged that certain for-profit entities allow linking to their databases,
but this introduces the issue of cost.
Dr. Smith then asked whether in doing the design work there is "a provision to actually
talk to household members about how they think about these choices" (i.e., the
precautions parents take with their children). He suggested focus groups, cognitive
interviews, etc. as means of ensuring that the right questions are being asked "as opposed
to just itemizing what we think we'd like to know without talking to them."
Dr. Scheidt responded, "Yes, unequivocally," and mentioned that an extensive set of
focus group discussions had begun and would be expanded as the protocol becomes
clearer.
Dr. Smith asked whether the results of those discussions would be reported before the
survey goes out, and Dr. Scheidt responded that information is posted on the website.
Rachel Nugent (NIH Fogarty International Center) called attention to the fact that both
people and environmental contaminants move across borders and that "there are a lot of
particularly vulnerable populations of children in the U.S. who may have foreign
backgrounds or otherwise be influenced by other country backgrounds." She asked Dr.
Scheidt to speak to the issue of coordinating the study with other countries.
Dr. Scheidt cited the Tri-National Commission's efforts in addressing the environmental
impact of NAFTA and in urging Canada and Mexico to carry out "coordinated an parallel
studies that would be quite an advantage to us to provide ranges of exposures that we
would not otherwise have and potential sample size reductions." Dr. Scheidt
acknowledged the potential problem of including participants from other countries who
may migrate. In the study, they clearly anticipate including Spanish-speaking subjects
and not excluding systematically any potential migrants, and they plan to provide long-
term follow-up of anyone who does migrate. He stated that although the details of that
had not yet been determined, they would do as much as is feasible.
Bryan Hubbell (EPA/OAQPS) brought up the issue of the limited network of monitors
for many environmental contaminants. He wondered how the study was ensuring that
"certain populations that don't happen to live where a monitor is" are not excluded and
asked whether they planned to do additional monitoring.
Dr. Scheidt reiterated that the data from numerous sources (e.g., NASA) were being
merged. He also stated that the study anticipated supplementing available data with "an
extensive degree of monitoring" itself and was exploring the range of technologies
available for doing this economically and efficiently. The study working groups and pilot
studies are considering these issues.
88

-------
J.R. DeShazo (UCLA School of Public Policy) stated that the household's totality of
response to a perceived risk is important to factor into an evaluation of how households
are economically responding to risk. He emphasized that remedial actions taken once a
health outcome is expressed, and not just defensive mitigating actions, involve dedicating
time and money resources by household members. He asked whether that consideration
"was coming front and center in the survey instrument."
Dr. Scheidt asked for a clarification and Dr. DeShazo replied that "the fundamental issue
is how parents evaluate the portfolio of health risks their children could face—the level of
risk they perceive and what they do to defend and mitigate the risk of exposure to the
child, and then once the child shows symptoms what kind of remedial behavior they
undertake to reduce their level of risk . . " in other words, the intra-household process of
identifying and dealing with risks to children.
Dr. Scheidt responded that it was an "interesting question" and, saying that they had not
focused on the decision-making dynamic in families, he wondered how they might go
about doing that. He acknowledged that study participants, as a consequence of
participating in the study, would learn a lot about health risks that their children face, or
don't face, and that they are a bit concerned about the potential impact this will have on
the long-term outcomes of the study. He closed by restating that they had not studied
how parents process that information.
Ed Chu, the session moderator, closed the session by urging everyone to look at the study
website, and he commented that one of the primary reasons for having Dr. Scheidt speak
at the workshop was to stimulate economists' interest and involvement in the study.
89

-------
Valuing Environmental Health Risk Reductions to Children
PROCEEDINGS OF
SESSION II: HOUSEHOLD DECISION MAKING
A WORKSHOP SPONSORED BY THE U.S. ENVIRONMENTAL PROTECTION AGENCY'S
NATIONAL CENTER FOR ENVIRONMENTAL ECONOMICS (NCEE), NATIONAL CENTER FOR
ENVIRONMENTAL RESEARCH (NCER), AND OFFICE OF CHILDREN'S HEALTH PROTECTION;
AND THE UNIVERSITY OF CENTRAL FLORIDA
October 20-21, 2003
Washington Plaza Hotel
Washington, DC
Prepared by Alpha-Gamma Technologies, Inc.
4700 Falls of Neuse Road, Suite 350, Raleigh, NC 27609

-------
ACKNOWLEDGEMENTS
This report has been prepared by Alpha-Gamma Technologies, Inc. with funding from the National Center
for Environmental Economics (NCEE). Alpha-Gamma wishes to thank NCEE's Kelly Maguire and
Project Officer Nicole Owens for their guidance and assistance throughout this project.
DISCLAIMER
These proceedings are being distributed in the interest of increasing public understanding and knowledge of
the issues discussed at the workshop and have been prepared independently of the workshop. Although the
proceedings have been funded in part by the United States Environmental Protection Agency under
Contract No. 68-W-01-055 to Alpha-Gamma Technologies, Inc., the contents of this document may not
necessarily reflect the views of the Agency and no official endorsement should be inferred.
11

-------
TABLE OF CONTENTS
Session II: Household Decision Making
Commissioned Paper: Benefit Cost Analysis and the Entanglements of Love
Ted Bergstrom, University of California, Santa Barbara	1
Weak Substitution, Environmental Vulnerability, and Choice
Mary Evans, University of Tennessee 	25
Abstract of Preliminary Research on Family Decision Making and the Value of
Preventing Childhood Development Impairment
Sandra Hoffman, Resources for the Future	55
Discussant
Robin Jenkins, U.S. EPA, National Center for Environmental Economics 	56
Discussant
Mark Agee, Pennsylvania State University 	62
Summary of Q&A Discussion Following Session II	69
ill

-------
Benefit Cost Analysis and
the Entanglements of Love
Theodore C. Bergstrom *
November 1, 2003
1 Introduction
Suppose that after careful thought, a parent reports that she is willing to pay
up to $100 to save her child from one day of cold symptoms.1 How can we use
her answer and those of other parents to analyze the benefit of public projects
that affect child health? Would it make sense to calculate the expected number
of child days of cold symptoms that a project would prevent and to multiply
this number by the average willingness to pay of a sample of parents?
But if a child has two parents, maybe we should value the child's health at
the sum of the two parents' willingness to pay. Or, if the two parents' answers
differ, perhaps we should use the maximum or perhaps the minimum of their
two answers. And what of the child's own valuation on improved health? Why
shouldn't we count that as well? To answer questions like these, we need to
* Aaron and Cherie Raznick Professor of Economics, University of California, Santa Bar-
bara. This paper was prepared for a conference on Valuing Environmental Health Risk Reduc-
tions to Children, sponsored by the US EPA's National Center for Environmental Economics,
the National Center for Environmental Research, and the University of Central Florida. I am
grateful for helpful suggestions from Robin Jenkins of the EPA and Mark Agee of Penn State
University.
1A series of carefully conducted studies have asked questions similar to this. Viscusi, Magat
and Huber [25] interviewed individual parents who were asked their willingness to pay for
hypothetical "safer" insecticides and toilet cleaners that would reduce health hazards for their
children by specific amounts. Liu et al [16] found that a sample of Taiwanese mothers were
willing to pay an average of about US $57 to avoid a cold for one of their children and about
$37 to avoid a cold for themselves. (An earlier study by Alberini et al [1] asking Taiwanese
adults about their willingness to pay to avoid colds for themselves found very similar values.)
Dickie and Ulery [11] asked parents about their willingness to pay for the reduction of cold
symptoms for a single child and for themselves. Dickie and Gerking [10] surveyed individual
parents, asking their willingness to pay for a hypothetical sunscreen that reduced risk of skin
cancer by a specified amount.
1

-------
step back and examine the logic of benefit-cost analysis as applied to families
in which some individuals care about the well-being of others.
2 What Can Benefit-Cost Analysis Do?
Without explicit instructions about how to compare one person's benefits with
those of another, we can not expect benefit-cost analysis to tell us whether a
public project should or should not be adopted. The best we can hope for
from benefit-cost analysis is to learn whether a project is potentially Pareto
improving. To see why this is so, it is useful to revisit a device found in Paul
Samuelson's beautiful 1950 paper, "Evaluation of the Real National Income [23].
Samuelson introduces what he calls "the crucially important utility possibility
function" to help us understand the logic of benefit cost analysis.
Consider a community with two people. A possible project would produce a
public good valued by both persons. In order to produce it, community members
will have to reduce their total consumption of private goods. If the project
is not implemented, then alternative divisions of the total quantity of private
goods will determine different utility distributions. These possible distributions
generate a "utility possibility frontier" as described by the curve UP in Figure
1.
Figure 1: Utility Possibilities and Benefit Cost
UP-
UP'-
If the project is implemented there will be less private goods to be distributed
between the two people, but each will enjoy a higher level of public goods. This
will generate a new utility possibility frontier UP'. Because the curves UP'
and UP are not "nested" there is not an obvious way to determine which is the
better situation. Some utility distributions are attainable only if the project is
not implemented and others are attainable only if the project is implemented.
2

-------
Suppose that we know that we know that if the project is not implemented,
then the utility allocation will be the point A in the graph. We see from the
figure that the curve UP' contains points above and to the right of A. This
implies that it is possible to produce the public good and to divide its costs in
such a way that both individuals are better off than they were at A. When this
is the case, we say that the project is potentially Pareto improving. Stating this
formally:
Definition 1 A public project is potentially Pareto improving if it is possible
to implement the project and to distribute its costs in such a way that in the
resulting allocation some community member(s) are made better off and none
are made worse off.
In a suitably simple economy, benefit-cost analysis can determine unambigu-
ously whether a project is potentially Pareto improving. One such "suitably
simple economy" is the following: A possible public project produces a public
good that benefits several individuals and harms none.2 The economy has a
single pure private good.3 Implementing the project requires the input of a
known amount of private goods.
If individuals are selfish, the logic of benefit-cost analysis in this economy is
very simple. An individual's "willingness-to-pay" for the project is defined to
be the maximum amount of private good that she would be willing to sacrifice
in order to enjoy the benefits of the public goods provided by the project. The
benefit-cost test compares the sum of all individuals' willingnesses-to-pay for
the project to its cost. It is quite easy to see that for this simple economy,
the sum of individual willingnesses-to-pay exceeds total cost if and only if the
project is potentially Pareto improving.4
The relation between benefit cost analysis and potential Pareto improvement
becomes more complex when some of the beneficiaries are children who are loved
and supported by parents, and even more complex if the parents care about
each others' happiness. In this paper, we try to untangle the logic of these
attachments a few strings at at time.
2	It is not hard to extend these principles to an economy where some individuals benefit
and others are harmed. But this simpler model suffices to illustrate the principles.
3	The single-good assumption is appropriate in a multi-good economy if adopting and paying
for the project does not result in changes in the relative prices of the private goods.
4If the sum of willingnesses to pay exceeds total cost, then it is possible to pay for the
project while assessing no individual a share of the cost smaller than her willingness to pay.
Implementing the project and paying for it with any such assessment constitutes a Pareto
improvement. Conversely, if the project is potentially Pareto improving, there is a way to
assess the costs of the project so that nobody is worse off after the project is implemented.
Each individual's assessment would, by definition be smaller than his willingness-to-pay. Since
these assessments add to the cost of the project it follows that the project would pass the
benefit-cost test.
3

-------
In order to extend the principles of benefit-cost analysis to a family, we need
to specify and apply a theory of household decision-making for that family. Of
course the world is filled with a great variety of family structures, which differ
in membership and in the distribution of decision-making authority. As we will
see, the prescriptions for benefit-cost analysis can vary significantly with the
family type.
As we extend benefit-cost analysis to multi-person households, we face the
question of how to reinterpret the criterion of potential Pareto improvement.
This paper takes the position that the government is not able to intervene in
the distribution of private goods within the households. Thus we consider a
project to be potentially Pareto improving if and only if there is a way to assign
the costs to families so that given the household decision structure in each family,
no individual is made worse off and at least one is made better off.
3 A Single-Parent Household
Suppose that an economy is made up of households consisting of a single parent
and a dependant child. Assume that each parent cares about her own consump-
tion and her child's health and consumption and is selfish with respect to those
outside her household. Assume also that children care only about their own con-
sumption and health. Consider a government project that uses private goods as
inputs and produces a public good which increases the health of children. How
can we determine whether or not the project is potentially Pareto improving?
Let each parent's utility function take the form:
U (x, v(k, h))	(1)
where x and k are the amounts of private goods consumed by the parent and
the child respectively and h is a measure of the child's health. The child has no
income and receives consumption goods only from its parent. The parent has
an after-tax income, to which she allocates between her own consumption and
that of her child.5 Then, contingent on the child's health being h, the highest
utility that a parent can achieve with after-tax income to is
U*(m,h)= max U(x,v(k,h)).	(2)
x-\-k
-------
Definition 2 If the sum of all parents' willingness-to-pay for a project exceeds
the cost of the project, it is said to pass the benefit-cost test for parents.
We can show that if a project fails the benefit-cost test for parents, then it
is not potentially Pareto improving. To say this in another way:
Remark 1 In an economy with single-parent households, if a project is poten-
tially Pareto improving, then it must pass the benefit-cost test for parents.
The proof is simple, but instructive. If the project is potentially Pareto
improving, it must be possible to implement the project and to assign its costs
it in some way so that no parent is worse off and at least one is better off than
if the project is not implemented. Suppose that a project fails the benefit-cost
test. Then in order for the project to be funded, some parent must be assessed
a cost greater than her willingness to pay. Since the parents have complete
control of their family incomes, this means that the parent who is forced to pay
more than her willingness to pay for the project will be worse off if the project
is implemented. Therefore the project cannot be potentially Pareto improving.
From Remark (1), we see that even though the Pareto criterion accounts for
the well-being of children, it would be misguided to measure benefits by adding
the value that children place on their own health to the values that their parents
place on it. Adding children's valuations to those of their parents would lead
to acceptance of projects that do not pass the benefit-cost test for parents and
which are therefore not potentially Pareto improving.
The converse of Remark (1) is not in general true. That is, there may be
projects that pass the benefit-cost criterion for parental preferences, but which
do not allow a Pareto-improving reallocation in which the well-being of children
as well as parents is (weakly) improved. This is not very surprising, especially
since it has not been assumed that parents agree with their children about what
is good for them. But, more remarkably, the converse fails to be true, even if
parents and children share the same preferences over alternative combinations
of child consumption and child health.
Parent's preferences are said to be benevolent if the parent's preferences over
the child's consumptions coincide with child's.6 Stated more formally, we say
that
Definition 3 A parent's preferences are benevolent if the parent's utility func-
tion is Ui {xi,Vi(ki,hi)) where Vi represents the child's preferences.
Even if parents have benevolent preferences, it is possible that some projects
that pass the benefit-cost test parental preferences will lead to outcomes that
''Though this usage is common in welfare economics, those who have been parents and those
who have been children will recognize that parents who are "benevolent" by this definition
would not always act in the best long-run interests of their children.
5

-------
are worse for some children. This can happen because the child's health may
be such a strong substitute for the child's income that with a healthier, the
parent will reduce the amount of consumption goods transferred to the child by
so much that the child is actually worse off after the transfer than before.
Figure 2: Parent's Consumption and Child's Utility
x
UP'
UP
v
This effect is illustrated in Figure 3. The curves labeled UP and UP' shows
combinations of parents' consumption and child's utility that are possible be-
fore and after a project is implemented. The point A on UP represents the
preferred point of the parent if the project is not implemented and the point B
on UP' represents the point preferred if the project is implemented. With the
indifference curves that we have drawn, the parent will prefer B to A despite
the fact that the child has a lower utility at B than at A. Notice that in Figure
3, the indifference curves drawn are consistent with parental benevolence, since
the parents always prefer higher v to lower. One could imagine, for example,
that for an impoverished family, an improvement in the child's health would
induce the parents to require the child to earn its own food. Under these cir-
cumstances, despite their benevolence toward the child, the parents might favor
an improvement in child health that is accompanied by such a large reduction
in income transfers to the child that the child is worse off. Section 8.1 of the
appendix, presents an algebraic example of preferences for which this is the case.
4 Lovebirds without Kids
Archie and Bess are a couple. They care about their own consumption of private
goods and their own health. They also care about each other's happiness. Archie
and Bess spend a good deal of time together and so each has become a good
judge of the other's happiness, though this information arrives with a brief lag.
6

-------
Let XA(t) and Xfi(t) be expenditures on consumption, and let h,A(t) and hB(t)
be measures of health for Archie and Bess respectively at time t.
Archie's happiness at time t is determined by the function
UA(t) = vA (xA(t),hA(t)) + aUB(t - 1)	(4)
and Bess's happiness is determined by the function
UB(t) = vB	+ bUA(t - 1).	(5)
Elsewhere [4] and [6], I show that this dynamical system is stable under
plausible dynamics if and only if ab < 1. As these earlier papers explain,
this stability condition limits the mutual intensity of their care for each other's
happiness.
The government is considering a women's health project that will improve
Bess's health by a known amount A. Archie and Bess are separately interviewed
about their willingness to pay for this improvement. Bess realizes that if her
health improves, this will have a direct effect on her happiness, which Archie
will observe and enjoy, which in turn will make Bess herself happier, and so
on ad infinitum. Can either Bess or Archie extract a reasonable answer to
the interviewer's question from the blur of reflected happiness in this hall of
mirrors?7
Equations (4) and (5) can be "inverted" to determine more conventional
utility functions that depend only on the consumption and health of Archie and
Bess. If consumptions and health levels are constant over time, equations (4)
and (5) determine a time path of utilities for Archie and Bess that converges to
equilibrium values. To find these equilibrium values, we set U^(t) = UA(t — 1),
UB(t) = UB(t — 1) in Equations (4) and (5) and solve. Then U^(xa, xB,h,A,hB)
and UB(xA,xB,hA,hB) are the equilibrium utilities that result from the con-
stant outcome (xA,xB,hA,hB). These utility functions are as follows:
U*A{xA,xB,hA,hB) = 1 (vA(xA,hA) + avB(xB,hB))	(6)
1 — ab
and
UB(xA,xB,hA,hB) = 			 (vB(xB,hB) +bvA(xA,hA))	(7)
1 — ab
Let us call va(-) and vB(-) the private utility functions of Archie and Bess
respectively and let us call U\(-) and U\j(-) their social utility functions. We
assume that Archie and Bess are able to untangle their affections so as to find
private and social utility functions as in Equations (6) and (7). There remain
some tricky questions to answer. If know that their social utility functions
7Miles Kimball [15] suggested the hall-of-mirrors metaphor.
7

-------
take this form, how can we use Archie's and Bess's reported valuations for im-
provements in Bess's health in a benefit-cost analysis? To answer this question,
we need some assumptions about the way that Archie and Bess make family
decisions.
4.1 Archie the Dictator
Gary Becker's famous "Rotten Kid Theorem" [2] operates on the assumption
that family decisions are determined according to the preferences of a single
benevolent household member.8 Although this assumption is politically incor-
rect and, I believe, descriptively inaccurate for modern Western households, it
remains worth studying for at least two reasons. First, the simplicity of this
model makes it an instructive place to develop intuition and understanding that
can be extended to other environments. Indeed since almost all economists in-
terested in the economics of the family have cut their teeth on Gary Becker's
patriarchal family [2], this model has become a benchmark against which alter-
native theories need to be tested. More importantly, not all of the households
that interest us are modern and Western. Some of the most important appli-
cations of benefit cost analysis to public health programs are likely to concern
traditional societies and highly patriarchal societies.
Assume that Archie has no direct control over his own health or that of
Bess, but he is able to determine the amount of consumption expenditure that
each receives, subject to a budget constraint. Then he chooses xA and xB to
maximize his social utility function
UA{xA,XB,hA, hB) = vA(xA,hA) + avB(xB,hB)	(8)
subject to the budget constraint xA + xB = M, where M is the family income.
For given health levels and family income, the possible distributions of the
private utilities, vA and vB are points lying on or below the curved line in
Figure 4.1. Archie's indifference curves over these possible distributions will
be straight lines with a slope of —1/a and his preferred distribution of private
utilities will be at the point marked A*
Suppose that benefit-cost analysts interview Bess and ask her the following
questions.
B.l What is the largest amount of your personal consumption that you would
be willing to give up in order to improve your health by A units?
8In Becker's treatment, although Archie is benevolent, Bess is selfish. I think he chose to
do this not out of misogyny, but because he wanted to avoid the hall-of-mirrors complications
of mutual affection.
8

-------
Figure 3: Utility Possibilities
B.2 Given the way that Archie allocates income in your family what is the
largest amount of family income that you would want to give up in order
to improve your health by A units?
Let us also suppose that they interview Archie and ask him:
A.l What is the largest amount of family income that you would be willing to
give up in order to improve Bess's health by A units?
When will the answers to these questions be different and when will they be
the same? And how should we use the answers in benefit-cost analysis?
A Harmonious Case
Let us assume that Archie and Bess both have positive but diminishing marginal
utility9 for their own consumption of private goods and that Bess's private utility
function is of the special form,
i'B (xb ,hs) = fb (xb + khB)	(9)
9We have assumed that utility functions are additively separable between Archie's and
Bess's private utilities. In the additive form, the v functions are unique up to positive afRne
transformations. The property of diminishing marginal utility for private consumption there-
fore has operational content in that it is preserved under all positive afRne transformations.
9

-------
where fc is a constant and where the function fs{ ) displays positive but dimin-
ishing marginal utility. In this case Archie and Bess will be in perfect agreement
about the value of an improvement in Bess's health. Bess's answer to question
B.l be will be the same as her answer to B.2 and also be the same as Archie's
answer to question A.l.10 In order to determine whether the health project is
Pareto improving, a benefit-cost analyst could use this answer to represent the
whole family's willingness to pay.
Remark 2 If Archie and Bess both have diminishing marginal utility for pri-
vate goods, if Bess's utility function is of the form	h) = fsi^B + khg)
and, Archie chooses the distribution of family income between private goods for
himself and for Bess, then all three questions, B.l, B.2, and A.l have the same
answer, which is kA. If the health project is implemented and Archie and Bess
pay any amount less than kA, both will be better off if the plan is implemented.
If they must pay more than kA,then if it is implemented,, at least one of them
will be worse off.
It is easy to see that Bess's answer to question B.l must be fcA. If she gives
up p < fcA of her own consumption and her health increases by A, the net effect
is to increase her private utility and if she gives up exactly p = kA her private
utility remains unchanged.
If the family spends p to improve Bess's health by A, Archie will allocate
private consumption between himself and Bess by choosing xa and xg to maxi-
mize his utility, va(xa, hA) + afB(xB + khB + kA), subject to the constraint that
xa+xb = M—p. If we define ys = xs + hs + kA, then we can restate this max-
imization problem as the equivalent problem: Maximize va(xa, hjC) + afs(yB)
subject to xb + Vb = M + kh + (kA — p). If the project is implemented and
the family pays p, the net effect is seen to be the same as that of increasing
Archie's budget by fcA — p. Thus Archie would be willing to pay any amount
p up to fcA for the project. Therefore Archie's answer to Question A.l is fcA,
which is the same as Bess's answer to B.l. Since we have assumed that both
spouses have diminishing marginal utility of private consumption, it also true
that an increase (decrease) in Archie's budget results in an increase (decrease)
in both xa and ys and hence in Bess's private utility. Therefore Bess's answer
to Question B.2 is also fcA.
Generous Archie?
With the utility function described in the previous section, Archie will agree
to spend family funds on Bess's health if and only if she would be willing to
pay for the increase entirely out of her own consumption. But if Bess's private
10This will be true for any positive a, that is, even if Archie is very selfish, so long as he
cares a little about Bess's well-being.
10

-------
utility function takes the additively separable form, vb(xb, hg) = fB(xB) + khs
where Archie and Bess have diminishing marginal utility of private consumption,
Archie will be willing to "share the cost" of an improvement in Bess's health.
In this case, Archie's answer to Question A.l is greater than Bess's answer to
Question B.l and equal to Bess's answer to B.2.
More generally consider the case where Archie considers Bess's health and
her consumption to be complements, where complementarity is defined as fol-
lows.
Definition 4 Person A regards the health and consumption of Person B as
complements if for any distribution of consumption between A and B, an in-
crease in Person B's health does not reduce A's marginal rate of substitution
between B's consumption and A's consumption.
A proof of the following result can be found in the Appendix.
Remark 3 If Archie and Bess both have diminishing marginal utility for private
goods and if Archie regards Bess's health and consumption as complements, then
Archie's willingness to pay for an improvement in Bess's health exceeds the
amount that she would be willing to pay for this improvement out of her own
consumption.
In the example discussed in Remark 2, Bess's health and consumption are
not complements but substitutes. In this case, if Bess's health improves, Archie
reduces the amount of consumption goods that she receives so as to leave her
private utility unchanged. It is possible however, even where Archie is benev-
olent, that an improvement in Bess's health would induce him to reduce her
allocation of consumption goods by so much that she is left worse off than he
would choose her to be if she were less healthy. This is a case where he regards
her health and her consumption as very strong substitutes. The example that
we discussed in Section 3, where an improvement in child health led the benevo-
lent parent to choose an outcome that is worse for the child can be reinterpreted
to illustrate this case, with Archie cast in the role of parent and Bess in the role
of child.
5 Non-dictatorial Outcomes
In modern Western societies, we do not expect either adult member of an ordi-
nary household to have dictatorial power over the allocation of private goods.
We consider two alternate theories of household allocation. One of these is
a bargaining model in which the Nash cooperative bargaining solution to the
household environment. The other is a model in which Archie and Bess reach
unanimous agreement on outcomes based on a shared notion of fairness.
11

-------
5.1 Fairness Norms and a Household Welfare Function
Suppose that in the course of their relationship, Archie and Bess build a mu-
tually agreed notion of "household fairness" which governs their decisions. As
Samuelson [24] suggests, this consensus might be expressed by means of a house-
hold "social welfare function which takes into account the deservingness or eth-
ical worths of the consumption levels of each of its members." We could reason-
ably assume that this social welfare function would be an increasing function
of each member's social utility as defined in Equations (6) and (7). Thus the
household welfare function would be of the form
An appealing case can also be made that the household welfare function should
be linear in its two arguments, so that we have
for some a between 0 and 1.
A social welfare function with linear weights on individual social prefer-
ences would be implied if the couple formed their ethical preferences by "veil-
of-ignorance" reasoning of the sort: "How would you want to divide household
resources if there were an equal chance that I were you and you were me?"
Although this kind of ethical reasoning can establish the existence of a social
welfare function of the form of Equation (11), it is not sufficient to determine
the parameter a that weights Archie's UA relative to Bess's UB. For example,
our argument so far gives one no special reason to think that a = 1/2 is more
"fair" than any other a between 0 and 1. The scaling of the functions v a and
vB is arbitrary and if these function are rescaled, then in order to preserve the
same household preferences, the weight a would have to be altered.11 As they
cooperate with each other, Archie and Bess would need to develop a household
consensus that determines the weight a that both regard as "fair". As Bin-
more [8] suggests, in the long run we might expect these weights to be close to
the weights that would sustain a Nash cooperative bargaining solution for the
household, since if there were a significant and persistent difference, the partner
who could do much better with bargaining and threatening might defect from
the social consensus expressed by the household social welfare function.
Recall from Equations (6) and (7) that the social utility functions are them-
selves linear combinations of the private utility functions, va(xa, ^a) and vb(xb, hB).
In particular, we have
1:lAbram Bergson [3] introduced the notion of a social welfare function for an entire society.
Harsanyi [14] proposed ethical foundations for such a function. Binmore [8] suggested that
small, tightly-woven groups like families, may evolve a concept of group fairness that is well
described by a household social welfare function.
W(UX(xA,xB,hA, hB),UB(xA, XB, hA, hB)).
(10)
W(U*A,U*B)=aU*A + (l-a)U*B
(11)
12

-------
W(U*a,U*b) =	+«^(-)) +Y^b(M-) + bvA(-)) (12)
Since preferences determine utility functions only up to an increasing trans-
formation, and since by assumption ab < 1, we have an equivalent household
welfare function if we multiply the expression (12) by 1 — ab. Doing this and
combining terms, we have
W = (a + 6(1 — a)) va(xa, hA) + (1 — a + aa)vB(xB, fig). (13)
If Archie and Bess share a household social welfare function, then the in-
terpretation of their answers to benefit-cost questions is really easy. If money
is taken from household income to help pay for a public project that improves
Bess's health, they both agree about how the cost will be shared in terms of
their private consumption. Thus each of them would give the same answer to
the question "How much would you be willing to pay for an increase of A in
Bess's health?" The appropriate number for benefit-cost analysts to use is the
number given by either of them. It would be a mistake to use the sum of their
answers, since they both would agree that the sum as twice as much as they
would want to pay.
5.2 Bargaining Equilibrium
The pioneering work on models of household bargaining was done by Marilyn
Manser and Murray Brown [19] and Marjorie McElroy and Mary Horney [20],
who proposed to model household decision making with the Nash cooperative
bargaining model. In these papers, a marriage is modelled as a static bilateral
monopoly. A married couple can either remain married or they can divorce
and live singly. There is a convex utility possibility set S containing all utility
distributions (C/i, C/2) that could possibly be achieved if they remain married.
The utility of person i if he or she divorces and lives singly is given by V^. It
is assumed that there are potential gains to marriage, which means that the
there are utility distributions (C/i, C/2) in S that strictly dominate the utility
distribution (Vi,!^)- These papers propose that the outcome in a marriage
will be the symmetric Nash bargaining solution where the "threat point" is
dissolution of the marriage with both persons choosing to live singly. According
to the Nash bargaining theory, the outcome in this household will be the utility
distribution (U^U^) that maximizes (Ui — Vi)(U2 — V2) on the utility possibility
set S*.12 In this theory the outcome in a marriage is completely determined by
12This expression is sometimes known as the Nash product. John Nash [21] proposed a set
of axioms for resolution of static two-person bargaining games such that the only outcomes
that satisfy the axioms maximize the Nash product on the utility possibility set.
13

-------
the utility possibility set and by the position of the threat point, (Vi, V2). This
theory has the interesting prediction that social changes that affect the utility
of being single will affect the distribution of utility within the household and
hence may change household spending patterns, even if they have no effect
on the budget of the household, while changes in the apparent distribution of
earned income within the household will have no effect on the distribution of
utility in the household if they do not change the threat point from being single.
Shelly Lundberg and Robert Pollak [18], [17] propose an alternative Nash
bargaining model. They suggest that for many marriages the relevant threat
point for the Nash bargaining solution should be not divorce, but an "uncoop-
erative marriage" in which spouses would revert a "division of labor based on
socially recognized and sanctioned gender roles." The Lundberg-Pollak model
makes predictions that differ significantly from the divorce-threat model. For
example, in the Lundberg-Pollak model, if government child-allowances are paid
to mothers rather than to fathers, the threat point shifts in favor of the moth-
ers. Accordingly, the outcomes of cooperative bargaining within households
are likely to be more favorable to women. By contrast, in the divorce-threat
model, changing the nominal recipient of welfare payments when the couple is
together will have no effect on the household outcomes if there is no change in
the beneficiary in the event of a divorce.
To many married persons it seems unlikely that couples would resolve dis-
agreements about ordinary household matters by negotiating under the threat of
divorce. If one spouse rejects the other's proposed resolution to a household dis-
pute, the expected outcome is not a divorce. More likely, there would be harsh
words and burnt toast until another offer or counteroffer is made. It might even
be that if the couple were to persist forever in inflicting small punishments upon
each other, it would be worse both of them than a divorce. Nevertheless, the
divorce threat may not be credible because divorce imposes large irrevocable
costs on both parties, while a bargaining impasse need last only as long as the
time between a rejected offer and acceptance of a counteroffer.
Nash's axiomatic approach to cooperative bargaining gives us no direct guid-
ance about the appropriate threat points for bargaining in a marriage. But use-
ful insight can be gained from Ariel Rubinstein's [22] model of noncooperative
bargaining with alternating offers. Ken Binmore [7] extended the Rubinstein
model to the case where each bargaining agent has access to an "outside op-
tion". Binmore's model is like the Rubinstein model, except that each person
has the option of breaking off negotiations at any time and receiving a payoff of
the value of an outside option.
The Rubinstein-Binmore model, as applied to marriage lends formal support
to the Lundberg-Pollak notion of bargaining. The non-cooperative bargaining
model predicts that household outcome will either be the Nash bargaining so-
14

-------
lution in which the threat point is delayed agreement and burnt toast so long
as this Nash solution is better for both than divorce. Thus the divorce threat
will influence the outcome only if the bargaining solution reached under the
threat of burnt toast is worse than divorce for one of the partners. In case the
divorce threat is relevant, one partner enjoys all of the surplus and the other is
maintained at a point of indifference between being divorced and being single.
In our example with Archie and Bess, suppose that health levels are deter-
mined by public policy while the allocation of private goods is determined by
bargaining. Suppose that the utility that each would obtain in the absence of
agreement depends on total household income and on the health of each. Where
x is total household income, let us represent these "threat point utilities" by the
functions Ta(xJia) and Tb(xJib) The Nash bargaining theory predicts that
the outcome of household bargaining will be an allocation of private goods that
maximizes the Nash product
subject to the constraint that xa + %b = x
For fixed levels of health, the Nash product in Expression (14) works like a
utility function a household utility function in the sense that private goods are
allocated in such a way as to maximize this function. If a public health project
is implemented and their household is taxed, the utility possibility frontier for
Archie and Bess will be altered and so will their threat points. The household
bargaining theory predicts a new allocation of utility. Neither Archie nor Bess
cares directly about the value of the Nash product, but only about his or her
own utility in the solution to the bargaining problem. Therefore they will in
general not agree on their willingnesses to pay for a specific change in health.
(UX-Ta)(U*b-Tb).
(14)
Figure 4: Health and Bargaining
UP
UP
15

-------
In a bargaining environment, not only is it possible that Archie and Bess
have differing willingness to pay for a public project. It may even be impossible
to get them to agree to fund a project that strictly increases the set of possible
utility allocations.13 This can happen because the project may shift bargaining
power and/or the shape of the utility possibility frontier in such a way that the
bargaining outcome is worse for one of them, even though the project shifts the
utility possibility frontier strictly outward. This effect is is illustrated fairly in
Figure 5.2. The initial utility possibility frontier is shown by the curve UP and
the initial threat point is located at T. The point that maximizes the Nash
product (14) is labelled E. Now suppose that a public health project is intro-
duced and a fixed amount of tax is collected from the Archie-Bess household.
One result of the project and the tax is to shift the utility possibility frontier
outward from UP to UP'. A second result is to move the threat point from T
to T'. Then the Nash bargaining theory predicts that the equilibrium outcome
for the family moves from E to E'.
We see that a curious thing has happened. The health project and asso-
ciated tax seems to be an unambiguous boon for Archie and Bess, since their
utility possibility frontier moves everywhere outward. But if the project is im-
plemented, the actual outcome in the household must be the corresponding
bargaining solution E' and as we see in the diagram, E' assigns a smaller utility
to Archie than his utility in the initial equilibrium E. Therefore Archie would
oppose this change.
Thus if we continue to use the benefit-cost test to ask whether a public health
project is potentially Pareto improving, we would have to use the minimum, of
Archie's and Bess's valuations to evaluate the benefits to this household.
6 Couples with Kids
After all this foreplay, it is time to bring a child into the Archie-Bess household
and contemplate how to apply benefit-cost analysis to projects that improve
children's health. A natural starting point for this theory is to think of the
child's health as a "public good" that is valued by both Archie and Bess. Let
us assume that Archie and Bess care about each others' happiness, as well as
the health and well-being of their child and about their own consumptions of
private goods.
Suppose that they have social utility functions of the form
U*A(xA,xB,xc,hc) = vA(xA,vc(xc,hc)) + avB(xB ,vc{xc ,hc)) (15)
13Lundberg and Pollak [17] apply similar reasoning to suggest that in the absence of binding
contracts, married couples may not be able to make efficient household decisions about matters
that affect relative bargaining power.
16

-------
UB(xA,xB,xc,hc) = vB(xB,vc(xc,hc)) + bvA(xA,vc(xchc)) (16)
where xc is the child's consumption, he is the child's health and where vc (xc, he)
measures the child's well-being.
6.1 Father Archie Calls the Shots
Let us begin by considering the Beckerian assumption that Archie is dictator
of the allocation of private goods. Then given the level of child health he and
family income x, Charlie chooses the consumption levels xA, xB, and xc so as to
maximize Expression 15 subject to the family budget constraint, xA+xB+xc =
x. Archie's willingness to pay for a small improvement A in the health of the
child is approximately A times his marginal rate of substitution between child
health and his own consumption. Assuming that he chooses to spend a positive
amount of the household income on the consumption goods for Beth and for
the child, Archie's marginal utility of consumption for the child is equal to
his marginal utility of consumption for himself and also equal to his marginal
utility of consumption for Bess. Therefore, Archie's marginal rate of substitution
between child health and his own consumption is equal to his marginal rate of
substitution between the child's health and the child's own consumption. But
this marginal rate of substitution is seen to be simply the ratio of the two partial
derivatives of the function vc(xc,hc) that measures the child's health. Thus
Archie's willingness to pay for a small increase A in the health of the child is
approximately
Thus Expression (17) is Archie's answer to the question "How much money
would you be willing to pay for an increase of A in the health of your child?"
Suppose that the child has the same perception of its welfare as do its par-
ents and its preferences are represented by the same function vc(xc,hc) that
appears as an argument in the parents' utilities. If the child is questioned about
the amount of consumption that it would be willing to forego for an increase
of A in its health, its answer would be A times its marginal rate of substitu-
tion between health and consumption. We see from Equation (17) that this is
precisely the same as Archie's marginal rate of substitution between the child's
health and family income. It follows that the child's answer would be the same
as Archie's.
We seem to have a kind of "Rotten Kid Theorem" here, but does it extend
to Bess? Given that Archie dictates the household allocation of consumption
goods, how will Bess answer the question "How much of the household income
would you be willing to surrender to pay for an increase of A in the health of your
child?" Bess realizes that if family funds are paid to support child health, the
dhc
(17)
17

-------
resulting changes in private consumption by family members will be determined
according to Archie's preferences. Given that Bess realizes this, the amount she
would be willing to have the family spend to pay for an improvement of A in
the child's health can be shown to be
w°=H'< (S+S~B) ¦	
-------
6.3	Allocation with Divorced Couples
Suppose that after producing a child, Archie and Bess get a divorce. After the
divorce, they have little affection for each other, but both care about the child's
well-being. Bess has custody of the child and they set up separate household
accounts, with each of them having a separate income. The only way that
Archie can contribute consumption goods to the child is by giving money to
Bess, who can allocate it as she chooses between herself and the child. Suppose
that Archie makes no voluntary payments to Bess because only a fraction of the
money that he gives her would be spent on the child's consumption.
In this situation, the appropriate measure for benefit cost purposes would
be the sum of their two answers. If Archie is willing to pay an amount Wa
and Beth an amount Wb for an improvement of A in the child's health, then
both would benefit from implementing the project if amounts Pa < Wa and
Pb < Wb are collected from the two parents. Thus their total willingness to
pay is Pa + Pb- In this case, a benefit cost study that measured benefits of a
child health project as the sum of the willingnesses to pay of a single parent for
each child would underestimate the value of the project.
6.4	What About Grandparents and Aunts and Uncles?
A child's parents are not the only people who care strongly about its health
and well being. Across almost all known societies, other close relatives besides
parents are intensely concerned about the well-being of a child.
The theory of evolutionary biology gives us strong reason to expect that this
must be true. The chances that our ancestors survived to reproduce successfully
depended not only on the behavior of their own parents, but on the amount of
help they got from their grandparents, uncles and aunts. If behavior toward
one's near relatives is inherited, then the fact that we are products of successful
evolution implies that humans can be expected to display significant willing-
ness to care for their near relatives. In 1964, the great evolutionary biologist,
William D. Hamilton [13] developed a theory that offers a systematic quantita-
tive prediction of the extent to which individuals, on average, will care about
their relatives. This prediction has come to be known as Hamilton's Rule.
Biologists define the "coefficient of relatedness" between two animals of the
same species to be the probability that a rare "gene" found in one of these
animals will also appear in the other. For sexual diploids if mating is between
unrelated individuals, the coefficient of relatedness between two full siblings is
1/2, that between half-siblings is 1/4, that between an individual and a full
sibling's child is 1/4, that between full cousins is 1/8, that between parent and
offspring is 1/2, that between grandparent and grandchild is 1/4, and so on.
Hamilton proposes that natural selection would produce individuals who try to
19

-------
maximize inclusive fitness where inclusive fitness is defined to be a weighted
sum of one's own reproductive success and that of one's siblings, half-siblings,
and cousins of various types, where the weights are coefficients of relatedness.14
(Of course some care has to be taken to avoid double-counting of say, one's
children's fitness and that of one's grandchildren.)
Hamilton stated his rule as follow:
The social behaviour of a species evolves in such a way that in
each behaviour-evoking situation the individual will seem to value
his neighbor's fitness against his own according to the coefficient of
relatedness appropriate to the situation.
Hamilton's rule implies that when faced with the option of sacrificing c units
of its own reproductive success in order to increase the success of a relative whose
coefficient of relatedness is k, by b units, the decision maker should make the
sacrifice if it passes the benefit-cost test kb > c.
Should we add the willingness to pay of a child's grandparents and other
relatives to that of its parents? Or does this amount to double-counting as
we found to be the case if we added the willingness to pay of its two parents
in an intact family? To answer this question in a really satisfactory way, we
would need a theory of decisions for extended families. In extended families
where transfers occur or are expected to occur between the grandparents and
the parents of a child, it might be argued that the family operates under a single
budget and that a parent's answers to questions about her willingness to pay for
improvements in the child's health incorporate her understanding of the psychic
benefits this improvement will confer on the grandchildren and her beliefs about
how the costs of this improvement would be shared under family transfers.
On the other hand, it seems likely that in most Western families, adult sib-
lings have independent family budgets with no significant sidepayments between
families. In this case it is appropriate for benefit cost analysis to add the will-
ingness to pay of a child's other relatives to that of its parents. As we argued
in the case of a divorced couple, if changes in a child's health and in the tax
burdens of its relatives do not otherwise affect the pattern of transfers between
relatives, then a public project that improves a child's health and costs its rel-
atives less than the sum of their willingnesses to pay allows a potential Pareto
improvement.
14More recent theoretical work [9] [12] [5] has shown that Hamilton's rule is strictly correct
only where the benefits and costs from interaction between relatives take a linear form.
20

-------
7 Conclusion
We have tried to untangle some of the strings of familial affection in order to
give practical guidance for benefit cost analysis. Some of these strings untie
relatively easily. For example, the members of a single-parent household will
not enjoy a Pareto improvement if the household is assessed a cost greater than
the parent's willingness to pay. Thus adding willingness to pay of children to
that of their parents would lead a benefit cost analysis to accept projects that
are not potentially Pareto improving.
We showed that with reasonable restrictions, the reflected happiness between
loving couples can be resolved into a well-defined household welfare function
over allocations. If the household acts to maximize such a function, then the
willingness to pay stated by either parent is an appropriate measure of the
benefits to the entire household. If there are relatives outside of the household
who also have significant willingness to pay, then a proper accounting of benefits
from child health should include the valuations of these relatives.
For households with bargained outcomes or for divorced households, inter-
pretation of parents' responses to questions about their valuation of a child's
health can, in principle be more complex. We have shown that married couples
who allocate private goods by bargaining may differ in the amount of household
funds that they would be willing to spend on the child's health. In these cases,
strict adherence to the principle of identifying potentially Pareto improving
projects suggests using the smaller of the answers proposed by the two parents.
The recommendation is quite different for divorced couples so estranged that the
noncustodial parent makes no voluntary contributions to the custodial parent's
household. In this case, benefit cost analysis should add the willingness to pay
of the noncustodial parent to that of the custodial parent.
8 Appendix
8.1 An Example
Each parent i has an income m > 1 and a utility function of the form f/j =
Xi + 2vi(ki, hi)1/2, where the function Vi(ki, hi) is a utility function representing
child i's preferences. There are two possible values for hi. These are hi = 0
and hi = 1. where initially hi = 0, and the public project is adopted, hi = 1,
and where Vi(k, 0) = k and Vi(k, 1) = k/2 + 4/9. The total cost of the project
is N/6, where N is the number of parents. Each parent allocates her income
Xi and ki so as to maximize her utility. When hi = 0, this occurs where k = 1
and Xi = rtii — 1. Thus if the project is not adopted, each parent i has utility
TOj + 2 — 1 = TOj + 1 and each child has utility 1. If the project is adopted,
then the parent the parent optimizes by choosing ki = 0 and = m;. In
21

-------
this case the utility of child i is Vi = A = A/9 and the utility of the parent is
rrii + 2 A1'2 = rrii +4/3. Each parent has a willingness to pay of 1/3 for the
project to be implemented and so the sum of willingnesses to pay is N/3. Since
the cost of the project is N/6, it passes the parental benefit-cost test. But, as
we have seen, if the project is implemented, each child's utility will be reduced
from 1 to 4/9.
Moreover, if the project is implemented, in order for children to be as well
off as before the project, each child must receive at least k = 10/9. Any transfer
scheme in which each child receives at least 10/9 and where parents pay a total
of N/6 for the project must be worse than the outcome without the project for
at least some of the parents.
8.2 Proof of Remark 3
Let xa and xg be the consumptions that Archie would choose for himself and
Bess if the the health project is not implemented. Let pt, be the maximum
amount that Bess would be willing to sacrifice out of her own consumption
in order to improve her health by A. Then it must be that Bess is indifferent
between her initial situation and the allocation in which her health is hg + A and
her consumption is reduced to xg — Pb- If the family budget is reduced by ps
and Bess's health is improved by A, then since Beth has diminishing marginal
utility of private consumption and since Archie views Beth's consumption and
her health are complements, it must be that Archie's preferred allocation of
private goods out of the new budget is one in which Bess's consumption is
reduced by less than ps ¦
22

-------
References
[1]	A. Alberini, M. Cropper, T. Fu, A. Krupnick, D. Shaw, and W. Harrington.
Valuing health effects of air pollution in developing countries: The case of
Taiwan. Journal of Environmental Economics and, Management, 34:107-
126, 1997.
[2]	Gary Becker. A Treatise on the Family. Harvard University Press, Cam-
bridge, Ma, 1981.
[3]	Abram Bergson. A reformulation of certain aspects of welfare economics.
Quarterly Journal of Economics, 51:310-334, 1938.
[4]	Theodore Bergstrom. Love and spaghetti, the opportunity cost of virtue.
Journal of Economic Perspectives, 3(2):165-173, Spring 1989.
[5]	Theodore Bergstrom. On the evolution of altruistic ethical rules for siblings.
American Economic Review, 85(1):58—81, March 1995.
[6]	Theodore Bergstrom. Systems of benevolent utility functions. Journal of
Public Economic Theory, 1(2):71-100, 1999.
[7]	Ken Binmore. Bargaining and coalitions. In Alvin Roth, editor, Game The-
oretic Models of Bargaining, pages 259-304. Cambridge University Press,
Cambridge, 1985.
[8]	Ken Binmore. Just Playing. MIT Press, Cambridge, MA, 1998.
[9]	L. Cavalli-Sforza, Luigi and Marcus W. Feldman. Darwinian selection and
"altruism". Theoretical Population Biology, 14:268-280, 1978.
[10]	Mark Dickie and Shelby Gerking. Valuing children's health: parental per-
spectives. Technical report, University of Central Florida, Dept of Eco-
nomics, UCF, Orlando F1 32826, 2003.
[11]	Mark Dickie and Victoria Ulery. Parental altruism and the value of avoid-
ing acute illness: Are kids worth more than parents. Technical report,
University of Central Florida, Dept of Economics, UCF, Orlando F1 32826,
2002.
[12]	Alan Grafen. The hawk-dove game played between relatives. Animal Be-
haviour, 27(3):905-907, 1979.
[13]	William D. Hamilton. The genetical evolution of social behavior, i and ii.
Journal of Theoretical Biology, 7(1):1—52, July 1964.
[14]	John Harsanyi. Cardinal welfare, individualistic ethics, and the interper-
sonal comparison of utility. Journal of Political Economy, 63:309-321, 1955.
23

-------
[15]	Miles Kimball. Making sense of two-sided altruism. Journal of Monetary
Economics, 20:301-326, 1987.
[16]	Jin-Tan Liu, James M. Hammitt, Jung-Der Wang, and Jin-Long Liu.
Mother's willingness to pay for her own and her child's health: A con-
tingent valuation study in Taiwan. Health Economics, 9:319-326, 2000.
[17]	Shelley Lundberg and Robert Pollak. Bargaining in families. Technical
Report 8642, NBER, 2001.
[18]	Shelly Lundberg and Robert Pollak. Separate spheres bargaining and the
marriage market. Journal of Political Economy, 101(6):998-1011, Decem-
ber 1993.
[19]	Marilyn Manser and Murray Brown. Marriage and household decision
theory-a bargaining analysis. International Economic Review, 21(1):21—
34, February 1980.
[20]	Marjorie McElroy and Mary Horney. Nash-bargained decisions: Towarda
generalization of the theory of demand. International Economic Review,
22:333-349, 1981.
[21]	John Nash. The bargaining problem. Econometrica, 18(1):155-162, Jan-
uary 1950.
[22]	Ariel Rubinstein. Perfect equilibrium in a bargaining model. Econometrica,
50(1):97-109, January 1982.
[23]	Paul Samuelson. Evaluation of real national income. Oxford Economic
Papers, 2(1) :1—29, January 1950.
[24]	Paul Samuelson. Social indifference curves. Quarterly Journal of Eco-
nomics, 70(l):l-22, February 1956.
[25]	W. Kip Viscusi, Wesley A. Magat, and Joel Huber. An investigation of the
rationality of consumer valuations of multiple health risks. RAND Journal
of Economics, 18(4):465-479, Winter 1987.
24

-------
Preliminary research: Please do not cite
Weak Substitution, Environmental Vulnerability, and Choice
10/6/03
V. Kerry Smith, Mary F. Evans, H. Spencer Banzhaf, and Christine Poulos*
I. Introduction
Feenberg and Mills' [1980] concept of weak substitution provides the basis for
expanding the role of revealed preference methods in measuring individual willingness to
pay to reduce pollution-related health effects. To support this argument we outline a
graphical analysis of how weak substitution can enhance our use of models based on
averting and mitigating behaviors. Moreover, the proposed structure offers an economic
framework for describing why different groups might be considered more or less
vulnerable to the health effects of pollution. Our discussion begins by summarizing
Smith and Banzhaf s [2003] proposed approach to describe how weak complementarity
and the Willig condition contribute to the estimation of the economic value for reducing
pollution's impact on environmental amenities.
Discussions of the preference restrictions used to recover information about
consumers' values for non-market goods largely ignore weak substitution. For example,
after reviewing the concept in some detail in his 1992 book, Freeman [2003] reflected the
apparent professional consensus and excluded weak substitution from his revised edition.
He maintains that an assumption of less than perfect substitution is not especially
* Paper prepared for presentation at EPA/UCF Workshop: Valuing Environmental Health Risk Reductions
to Children, October 20-21, 2003. University Distinguished Professor, CEnREP, North Carolina State
University and University Fellow Resources for the Future, Assistant Professor, University of Tennessee,
Knoxville, Fellow, Resources for the Future, and Assistant Professor, University of Missouri, Columbia,
respectively. Smith's research was partially supported by U.S. Environmental Protection Agency (Grant
#R-82950801). Thanks are due to Dan Phaneuf for comments on aspects of this research and to Susan
Hinton for help in preparing this paper.
25

-------
informative without including a more detailed preference specification. Following Bartik
[1988], Freeman argues that, at best, substitution offers upper and lower bounds for
Hicksian welfare measures.1 By contrast, our analysis suggests weak substitution may be
comparable to weak complementarity in its ability to enhance the insights derived from
revealed preference analysis of individual choice.
This paper provides a short primer on the logic underlying our arguments. We
rely on graphical analysis and some simple analytical arguments but stop short of
attempting to develop an empirical example. Section two outlines the first building block
for understanding the link between weak substitution and choice. Our focus parallels
Willig's [1978] use of weak complementarity. That is, we treat changes in environmental
services as analogous to quality changes in private goods. This strategy redirects attention
to selecting the private good whose price offers the most informative index of how a
quality change affects consumer choice. Section three defines weak substitution and
illustrates how a graphical analysis informs the structuring of economic models to
recover preference information for the non-market resource with this restriction. By
considering the price index capturing the effects of changes in environmental services,
we isolate the private demands most likely to isolate an individual's non-market/market
tradeoffs. Section four introduces a role for separability and with it a framework that
demonstrates how an individual or household's economic circumstances, together with
their physical (or health) conditions, contribute to a definition of their vulnerability to
pollution. The last section describes how our logic relates to two past applications and
1 Neill [1988] also discusses the prospects for substitution and complementarity relationships for groups of
private commodities serving as bounds for willingness to pay measures.
26

-------
how the framework might be used in new revealed preference (RP) or combined RP and
stated preference research.
II. Analysis of Consumer Surplus with Indifference Curves
Following conventional discussions of Hicksian consumer surplus, we begin with
the analysis of price changes. In analytical terms using indirect utility (V(-)) or the
expenditure function (
-------
functions- a conditional expenditure function that acknowledges the level of q can
influence expenditures onX, labeled e (•), and an expenditure function which includes
expenditures on q, labeled 
-------
price for the good used to represent the environmental service. However, the choice
process is described in a format that prevents her from actually choosing the amount
desired at the available price.
As a rule, prices do not exist for environmental services. Thus, a comparison of
Ae and Ae* diverts attention from an issue that seems more relevant in models destined
for empirical analysis. This issue concerns the selection of a private good (and hence a
price) that provides the greatest insight into the importance of changes in q for an
individual's observable choices. To develop this argument we need to consider a
different way of describing the WTP for a change in q. This alternative entails selecting
a single commodity and defining the price change that is equivalent to the change in q.
We define this equivalence by specifying the changes in q and the selected good's price
that require the same compensation (or payment) to restore the original utility level.
Thirty-five years ago, Fisher and Shell [1968] proposed a similar strategy in
considering the issues associated with adjusting prices for quality changes. They
demonstrate two results that seem underappreciated in the literature on non-market
valuation.4 The first of their results on price indexes relates to a quality change in a
private good equivalent to augmenting the quantity of that good. This formulation of
how quality influences preferences is the only one that allows the definition of a quality
adjusted price (for the good experiencing the quality change) independent of the amounts
of all other commodities being purchased. The second of their key theorems concerns
another aspect of preference restrictions and focuses on conditions, other than simple
repackaging (or equivalently quantity augmentation), that allow quality adjustment to the
29

-------
price of one or more goods other than the one experiencing the quality change.5 Their
examples for this type of preference restriction implicitly recognize a role for weak
complementarity6:
"... suppose there is a quality change in refrigerators. If this
change simply makes one new refrigerator deliver the services of
some larger number of older ones, then the simplest price
adjustment in the cost-of-living index is indeed an adjustment in
the price of refrigerators. On the other hand, if that quality change
also increases the enjoyment obtained from a quart of ice cream,
then an adjustment in refrigerator price will not suffice; an
adjustment in the price of ice cream is also called for. Indeed, if
the only effect of a refrigerator quality change is to augment
the enjoyment attained from ice cream, then the simplest
adjustment is one made only in the price of ice cream, even
though the quality change takes place in refrigerators. In this
case, an adjustment in the price of refrigerators can be made to
suffice; the magnitude of that adjustment, however, will depend on
the quantities demanded of all goods. An adjustment in the price
of ice cream will also suffice; the magnitude of that adjustment,
however, will only depend on the quantity of ice cream and the
quantity of refrigerators" (p. 123, bold highlights added).
The conditions that assure the quality change can be reflected in a subset of
private goods' prices parallel the conditions required to recover estimates of the role of
non-market services (quality) from consumers' choices of private goods. This link is
consistent with our proposal to consider the equivalence relation between quality changes
and price changes as a guide to revealed preference modeling.7
To develop this logic we reconsider the measurement of consumer surplus for a
price change, first in Marshallian terms. Consider one of the private goods, X, and a
4	Banzhaf [2001] makes this point in discussing the adjustment of cost of living indexes for non-market
environmental services.
5	Hanemann [1984] addresses cases similar to this general idea in his evaluation of cross product
repackaging for quality.
6	This concept had not been defined at the time they developed their reasoning.
7	As we noted, this argument is similar to Neill's [1988] use of substitution and complementarity
restrictions for sets of private goods in relation to a non-marketed good to establish bounds for WTP.
30

-------
change in the price of Xfrom /, to P0 (with Pl > P0 ). I'{)TSRI\ represents the change
Marshallian consumer surplus (MCS) for an ordinary demand curve in panel a of Figure
3. Our argument is outlined for the linear case, but holds as an approximation for other
cases. Equation (4) provides an algebraic description of P0TSRPl.
MCS^-P^X.+^-PjX^-X,)	(4)
Rearranging terms, we have equation (5):
MCS^fc-P^X i+jr0)	(5)
Panel b of Figure 3 provides an alternative representation of this relationship. Assume
the interior budget constraint corresponds to a price of Xat P} and the exterior to P0. The
tangency at D corresponds to X1 and at B to X0 . Marshallian consumer surplus for this
linear case is exactly equal to the average of CD+BA. This is established directly once
we recognize that, with numeraire good priced at unity, the following relations hold:
m0L = PlXl
m0N = P0X i
m0M = P0X0
(6)
o
m0J = PlX0
As a result, CD =m0L-m0N and AB=m0J-mjVL . Substituting from equation (6) yields
the relationship given in equation (7).
-Pa )Xt +(P, )x, ]
MCS = \{Pl -P0 )(X, +X0 )=\{CD+AB)
To use this logic to illustrate the connection between changes in q and equivalent
(in welfare terms) price changes, we assume weak complementarity. Let Xand q be
weak complements. The indifference curves for a given utility level with different levels
31

-------
of q will intersect at R on the vertical axis (i.e. at zero consumption level for X) as in
Figure 4, panel a. This feature follows from weak complementarity. That is, when there
is no consumption of X the non-market good has no value to the consumer. We assume
that qi>q0 . Quality improvements imply an inward fanning of indifference curves
because the levels of the weak complement (X) and the numeraire (z) required to maintain
the same level of utility decrease as q increases.
Now consider the definition of price change for X equivalent to the quality change
in q. For the income level defined by the vertical intercept T in the figure, the budget
constraints tangent to indifference curves labeled V(q0)and V(q1) at B and C,
respectively, provide the equivalent price change. Moreover, the average of CD and AB
in this case corresponds to the Hicksian willingness to pay for this price reduction (e.g.
from the budget constraint tangent to V(q1) to the one tangent to V(q0)) or equivalently
the WTP for the increase in environmental services from q0 to ql. This conclusion
follows because the utility level associated with these indifference curves is the same by
construction (i.e. V(q0)=V(q])). As in our earlier discussion of price changes, the
argument holds exactly for the case of a linear Hicksian demand function and
approximately with nonlinear demands.
Panel b of Figure 4 introduces a third indifference curve, l/(q]), corresponding to
a quality level of ql but a different utility level. Comparisons of V(ql) and V(cj])
isolate the individual's choices holding income constant. We examine the case where
prices correspond to the outer budget constraint. Therefore, j(CD+EF) corresponds to
32

-------
the Marshallian consumer surplus for the Hicksian equivalent price change and thus for
the change in environmental services as well.
Smith and Banzhaf [2003] use this relationship (i.e. between AB+CD) and
¦y(CD+EF)) to illustrate how weak complementarity and the Willig [1978] condition can
be combined to recover Hicksian consumer surplus measures for the amenity values
provided by enhanced quality. Here we argue that Willig's focus on the relationship
between the Marshallian consumer surplus for a quality change relative to the level of
consumption of a private good (that is a weak complement to quality) and the price
adjustment for that good with respect to quality is another way of suggesting that the
conditions required for quality adjusted price indexes parallel those needed to define
welfare measures for changes in quality.
III. Weak Substitution
Feenberg and Mills [1980] define weak substitution as another form of demand
interdependency that arises for private goods and environmental services when the
relative prices of those interdependent private goods are "high". In the case of weak
complementarity, the choke price of the private good is crucial to isolating the marginal
value of an increase in the related non-market environmental service (see Smith and
Banzhaf [2003] for details). In the case of weak substitution, the level of consumption of
a private good is defined, but the interaction between the private good and quality (or
non-market services) implied by this definition is different. That is, we assume there
exists a level of consumption for the private good (the weak substitute) above which
improvements in the non-market environmental service have no value. For price levels
33

-------
of the weak substitute above the level corresponding to this threshold, consumption of the
weak substitute is below the threshold and quality improvements are valuable. Formally,
if Pa defines the price that induces the threshold consumption level, Xa, as in equation (8),
then the condition for weak substitution is given in equation (9).8
Vp_
Vm
= Xa	(8)
dV_
dq
, =0 \/P
-------
AB = 4r(Pz(^)-Pz(Vo)}zo	(10a)
Pr
CD=y{Pz{^)~PzM)-^	(10b)
a
UaB+CD)=H-i-W (9l )-P; (?„ )>(z, +z„)	(10c)
2
P
y1 a J
When Pa is normalized to unity, this relationship reduces to one that is comparable to our
earlier description of the measurement of the Hicksian willingness to pay for a quality
change. In this case however, we use the price of the numeraire and not the price for the
private good serving as the weak substitute.
As in the case of our discussion of weak complementarity, introducing a third
indifference curve, here at the level corresponding to a quality level of qi (labeled V (<;/,)
in Figure 6b), also yields the Marshallian measure of the quality change (or price change
in z) as ~Pa "CAB+FG) in the figure.
In some respects, our simple interpretation in Figure 6b may be misleading. The
relative prices of X and z determine consumption levels of both goods. As we noted,
Figure 6b assures that when z=0, consumption of X equals Xa, the threshold beyond
which q no longer has value. The pivoting of the budget constraints at this point are
intended to illustrate increases in the relative price of z in comparison to X, with the price
for X remaining at Pa9 To some degree this representation is artificial because the
diagram illustrates relative prices for a given income. Thus, Pa could be represented for a
9 The actual slope of the budget constraint is _f /'-y/ ']. Increases in the price of z relative to Pa forXpivot
the budget constraint inward.
35

-------
different income level without pivoting atXa. Figure 7 recasts the diagram starting the
budget constraint at a position where P 
-------
not important) or weak complementarity is associated with larger amounts of
consumption that might be associated with specialty desserts (e.g. ice cream cakes that
must be refrigerated for some period time).
These examples have been quite simple. As a result, we relegated all other goods
to the background without really explaining how this outcome was accomplished. In
short, z was implicitly a composite of all other goods. Our graphical strategy sought to
focus on the properties of each of the two goods' prices as candidates for the quality
adjustment. In the process we implicitly assumed that one of them was an index for
everything else.10 One way to take account of other goods' prices in developing
restrictions that use weak substitution imposes separability. This next step is discussed
below.
IV. Separability and Environmental Vulnerability
To limit our attention to the relationships between z, X, and q and allow a more
explicit treatment of the remaining private goods, we introduce a new argument in the
preference function, r, which designates a composite of all other goods. Pr represents its
price (or price index).11 Separability of z, X, and q from r implies we can write the
indirect utility function as in equation (11).
V = V(v(P,Pz,q,mv), |i(Pr,m-mv))	(11)
Separability also assures that consumption choices can be described as if they were
undertaken as part of a process that decomposes the budget into components for each set
10	This was not what Fisher and Shell intended in their discussion of the problem.
11	r can be a vector of goods and Pr a vector of prices. We do not need to be more specific here because our
objective is to illustrate how separability allows the role of Pr to be limited to its influence on income
effects.
37

-------
of separable goods. Thus, we can specify mv as the expenditures on Xand z, given levels
of q. mv will be a function of all goods' prices (P ,PZ, and Pr) as well as q and m.n This
strategy "focuses" the patterns of influence. That is, separability allows us to distinguish
the roles of the prices of goods in the sub-function, P and Pz, from all other goods'
prices, Pr. All prices continue to affect the conditional demand for X and z. However,
those outside the sub-function enter through reallocation of income about separable sets
of goods. With this specification, the influence of both Pr and m are observed through
the income effect. Perhaps more importantly the effects of q on the demands for other
goods are observed exclusively through the reallocation of income (e.g. m-mv) and their
income effects. There are no separate substitution effects attributable to q with other
private goods.
Consider now the definition of conditional weak substitution given in equations
(12a) and (12b). Equation (12a) defines the conditional demand for X, given the budget
allocation between X and z and all other goods implied by mv.
vP
dv
dq
= Xa	(12a)
p=pn
= 0 V P
-------
Hanemann and Morey [1992]). {Pz (ql )-Pz (q0 ))z0 evaluates initial consumption of z at
the old and the new prices with Pz(lo)' zo representing initial expenditures and Pz(
-------
economic and demographic (or other) factors. Because the two considerations influence
observed responses, efforts to isolate sensitive groups and measure their responses must
resolve a difficult identification problem. The analysis must take account of the
economic consequences of the threshold defining vulnerability.
Interpreting the separability restriction as a means of introducing household
models illustrates an alternative use of the framework. For example, Chiappori's [1988]
collective household model imposes budget decomposition on the allocation of income in
the household through his structuring of the household decision process. Combining this
preference assumption with a further restriction that different members of the household
consume different goods, we extend the use of weak separability to develop some
specific insights into the use of averting or mitigating behavior models within a
household setting. To illustrate this point, consider a two-person household where
changes in q only affect individual one. Each household member consumes an exclusive
private good, represented by X1 and X2 respectively. In addition, assume that, for
individual one, the exclusive consumption good, X1, serves as a weak substitute for q.
Chiappori's efficient household model yields budget decomposition similar to what we
described in equation (12). However, the interpretation of the decomposition in the
household setting is different.14 In Chiappori's model, the expenditure groups represent
consumption by different people in the household, rather than simply different groups of
goods consumed by the same person as in our equation (12). Equation (13) clarifies the
distinction. Let f1 (•) designate the first individual's indirect utility function viewed as
environmental quality across individuals and use these physical or technical distinctions along with
differences due to economic circumstances (since X=0 for all cases).
14 See Smith and Van Houtven [2003] for a discussion of some of the welfare implications of the model.
40

-------
the solution to Chiappori's efficient household. ^1(-) is a function of all goods' prices,
income, and q. The left-hand side of equation (13) simply indicates that the indirect
utility realized by individual one depends upon the prices for all the goods consumed by
the household, q, and income. The right-hand side recognizes the exclusive goods
consumed by each of the household members. As given here, individual one consumes
the first good but not the second.
t(Pl,P2,q,m) = Vl(Pl,q,s(Pl,P2,q,m))	(13)
s(-) represents the income available to individual one. The remainder, m-s(•), is the
portion of household income available to individual two.
Equation (13) suggests two possibilities for defining the threshold level of
commodity one implied by weak substitution, either in terms of V1^) or «'(•) The
central issue is whether the definition for Xla implies that the income allocation is held
constant. Equation (14a) uses Roy's identity for the Marshallian demand to define the
price function for P1 (e.g. labeled here as Pla) that yields Xla in terms of V1^).
K
= XIa	(14a)
1 a
h=ha
vls
Equation (14b) defines weak substitution holding individual one's available income (,s(-))
constant.
i';
= 0 \/ Pt
-------
t=Vl+Vlsq	(15)
Substituting from (14b) for Px < Pla we have equation (16) for prices that satisfy this
inequality.
£lq=® + Vs 'Sq	(16)
In other words, the model suggests that the tradeoffs we observe associated with
individual one's choices in response to a change in q reveal any reallocation of income
due to the change in q. This result holds provided we assume his (or her) income is held
constant in defining Pla.
In contrast, if we define weak substitution in terms of /' (•), the model suggests
that, for Pla defined in this way, the incremental value of changes in q is counterbalanced
by budget reallocation within the household. Equations (17a) or (17b) illustrate the
result.
l\=0=Vg1+Vs1sq	(17a)
V1

-------
choices, and the responses of other household members to changes in some element of
environmental quality or services. In the next section we discuss how the framework
presented above relates to a few recent papers that sought to use household responses and
specific sensitive populations to estimate individual willingness to pay for improvements
in q.
V. Implications for Empirical Models
Weak complementarity has been the dominant restriction used in revealed
preference approaches to measure the economic values for changes in non-market
environmental services. More generally, complementarities between goods and services
are important to the processes through which new goods (or services) generate
improvements in individual well being. Bresnahan and Gordon [1997] developed this
important insight in the introduction to their volume on incorporating new goods in cost
of living measures. They used the example of artificial light to introduce their argument,
noting that over the past century:
"A series of new goods, such as whale oil for lamps, gaslight, and
then the electric light bulb, rapidly lowered the costs of using artificial
light, a commodity which is complementary to a wide variety of
household and workplace activities. Thus, as artificial light grew
cheaper, activities which had been economic only for short parts of the
day spread to evening, activities confined to summer became year-
round, and jobs became easier to perform" (p.3)
These types of complementarities have cascading effects - with cheap artificial
light transforming the allocation of individual time throughout the day and creating a new
mix of demands for goods and services - both new and old.
43

-------
An important dimension of these effects is a set of discontinuities which was not
emphasized by Bresnahan and Gordon and is central to Smith and Banzhaf s [2003]
generalization ot weak complementarities. Relationships between goods and services
may change substantially at different levels of consumption. Recognizing and using this
information should be regarded as a preference restriction. Weak complementarity at a
point and weak substitution are restrictions that provide a conceptual basis for developing
these restrictions in ways that can be used in understanding individual choice.15 That is,
weak substitution's definition requires that we identify a point of discontinuity in the role
for quality (or environmental services). As a result, it is adding more information than
alternative specifications of substitution patterns (as might be associated with classes of
preference functions, such as a constant elasticity specification).
To our knowledge, none of the empirical analyses of the costs of mitigating or
averting behavior has imposed weak substitution. Moreover, while several papers have
offered recommendations for future empirical analyses that follow the implications drawn
from our combined models with separability and weak substitution, these studies do not
suggest how the additional data would be used in estimation.16 By specifying a formal
structure, we believe the empirical implementation may be clearer. To illustrate this
point we selected two applications that consider environmental impacts on family
members - Agee and Crocker [1994] and Mansfield et al. [2002],
(a) Agee and Crocker [1994. 1996]
15	We owe this observation to Dan Phaneuf who commented on the Smith-Banzhaf argument for weak
complementarity by noting it created discontinuities and with them more information for revealed
preference models.
16	Shogren [2001] listed as his top two recommendations in a paper on valuing effects of environmental
hazards on children's health: (a) "pay more attention to how decisions of intrahousehold resource allocation
and distribution are made by caregivers; and (b) begin efforts to construct a systematic framework to help
44

-------
Both Agee and Crocker papers investigate parental willingness to pay for risk
information and therapy for children with differing confirmed levels of body lead. The
focus of their analysis is on the decision to undertake chelation treatment given
information about health risks, parental background (including education and labor
market status), body lead levels, and household income. The authors are careful to
include time costs in their estimates for the full costs of treatment (e.g. blood chelation).
They consider average estimates for parental time commitment but do not have specific
information on time allocations to the chelation activity or other activities for each
family. The income, employment status, wage rates, family composition, and education
levels are specific to each group.
Agee and Crocker find the chelation decision was related to parental education,
family status (e.g. father present), lead levels, and measures of the full price. The results
suggest more educated mothers and traditional families are less likely to choose the
therapy. The authors suggest this outcome could imply a substitution of caregiving time
for the therapy. Our framework would suggest that this behavioral response is exactly
where one should expect to recover information about the economic value of reducing
lead in the environment based on its impact on children. However, the information
available to these authors provides no specific details on time allocation choices of
parents, children's health status (that might serve as the threshold for a weak substitution
effect), or reallocation of resources within the household. The authors correctly
conjecture these issues are likely to be important but are unable to estimate a structural
model without these details.
organize how we think about the interaction of environmental threats and the behavioral choices of society,
caregiver and children..." (highlights of author's summary, pp.7-8).
45

-------
(b) Mansfield et al. T20021
Mansfield et al. [2002] propose an ambitious diary study of families with
asthmatic children to investigate parental responses to high ozone warnings. The authors
hypothesize that changing the mix of activities undertaken by children offers a behavioral
response to changes in air pollution. They use a national sample including asthmatics
and non-asthmatics during times when high ozone might be expected so it seems
reasonable to anticipate observed responses. The authors' preliminary results focus on
total hours spent outdoors and the fraction of the day spent outside. Specifically, for each
activity in which the child participated parents were asked to whether the activity was
undertaken "totally indoors", "mostly indoors", "half indoors and half outdoors", "mostly
outdoors", or "totally outdoors". Based on the time diaries, the authors calculated the
total number of hours spent in each of these categories. The results do not adhere to
one's a priori expectations. On average, parents of asthmatic children indicated that their
children spent more time mostly outdoors more often than parents of non-asthmatics. Of
parents who reported their child's time was spent "totally outdoors", the average hours
were higher among parents of non-asthmatic children. However, as in the "mostly
outdoors" category, the differences across the two groups were not large and did not
appear to be significantly different. Multivariate analysis confirms the simple analysis -
ozone warnings did not have a differential effect for the two groups.
The weak substitution framework suggests a potential explanation for this finding:
the need to separate the sample with children distinguished by a health threshold. This
partition might reflect parental judgments about when high levels of ozone are likely to
be problematic in ways that are comparable to our weak substitution relationship. Our
46

-------
model also suggests that the household's capacity to reallocate resources among its
members should affect the recoverability of the air quality effect. Our comparison of
weak substitution defined as £l =0 versus V]q =0 illustrated the importance of resource
allocation. While our model focused on reallocation in terms of income, a reformulation
to reflect time reallocation is a straightforward extension. The lesson for this analysis is,
as Shogren suggested, greater attention to caregivers' time and monetary reallocations.
Overall, these two examples suggest that behaviors consistent with the weak
substitution framework seem quite likely. What appears to be missing are data that
include not only responses concerning the weak substitute, but other goods or services (or
in the case of the household, the person) that a weak substitution model suggests is the
best source for choice information.
47

-------
z
A
B
X
C	D
Figure 1: Conventional Description of Willingness to Pay - Price Reduction
m = income
WTP = willingness to pay for a
change in the rationed good
from qotoqi
Aq = q}- q0
r = price of rationed good
A XoB = Ae*
B to C = r • Aq
m to R = r ¦ q0
Legend for Figure 2
m-WTP
T
Figure 2: Freeman's Description of Lankford Model - Change in Rationed Goods
48

-------
Price
Quantity (X)
Panel a
Figure 3: Marshallian Consumer Surplus for a Price Change
m
o
N
M
L
J
X
X
Panel b
49

-------
R
T
Panel a
Figure 4: Illustrating Quality Change
z
V(q )
R
T
X
Panel b
50

-------
z
X
Xa
Figure 5: Weak Substitution
X
Panel a	Panel b
Figure 6: Weak Substitution and Hicksian Demand
51

-------
Figure 7: Weak Substitution with Alternative Relative Prices

-------
References
Agee, Mark D. and Thomas D. Crocker, 1996, "Parental Altruism and Child Lead
Exposure: Inferences from the Demand for Chelation Therapy," The Journal of Human
Resources 31 (3): 677-691.
Agee, MarkD. and Thomas D. Crocker, 1994, "Parental and Social Valuations of Child
Health Information," Journal of Public Economics 55 (September): 89-106.
Banzhaf, Spencer H., 2001, "Quality-Adjusted Prices: Theory, History, and Application
to Air Quality," Ph.D. Dissertation, Department of Economics, Duke University.
Bartik, Timothy J, 1988, "Evaluating the Benefits of Non-marginal Reductions in
Pollution Using Information on Defensive Expenditures," Journal of Environmental
Economics and Management 15: 111-127.
Blackorby, Charles, Daniel Primont, and R. Robert Russell, 1978, Duality, Separability
and Functional Structure: Theory and Economic Applications (New York: New
Holland).
Blundell, Richard and Jean-Marc Robin, 2000, "Latent Separability: Grouping Goods
without Weak Separability," Econometrica 68: 53-84.
Bockstael, Nancy E. and Kenneth E. McConnell, 1993, "Public Goods as Characteristics
of Non-Market Commodities." Economic Journal 103 (September): 1244-1257.
Bradford, David F. and Gregory G. Hildebrandt, 1977, "Observable Preferences for
Public Goods," Journal of Public Economics 8 (October): 111-132.
Bresnahan, Timothy J. and Robert J. Gordon, 1997, "Introduction," The Economics of
New Goods, edited by T.J. Bresnahan and R.J. Gordon (Chicago: University of Chicago
Press for NBER).
Chiappori, Pierre-Andre, 1988, "Rational Household Labor Supply," Econometrica 56
(January): 63-89.
Feenberg, Daniel and Edwin S. Mills, 1980, Measuring the Benefits of Water Pollution
Abatement (New York: Academic Press).
Fisher, F.M., and K. Shell, 1968, "Taste and Quality Change in the Pure Theory of the
True Cost-of-Living Index," in J.N. Wolfe, editor, Value, Capital and Growth: Papers in
Honor of Sir John Hicks (Chicago: Aldine Publishing Co.): 97-140.
Freeman, A. Myrick III, 2003, The Measurement of Environmental and Resource Values:
Theory and Methods, Second Edition (Washington, DC: Resources for the Future).
53

-------
Hanemann, W. Michael, 1984, "Discrete/Continuous Models of Consumer Demand,"
Econometrica 52 (May): 541-562.
Hanemann, W. Michael and Edward Morey, 1992, "Separability, Partial Demand
Systems, and Consumer's Surplus Measures," Journal of Environmental Economics and
Management 22: 241-258.
Maler, Karl G., 1974, Environmental Economics: A Theoretical Inquiry (Baltimore: John
Hopkins University Press for Resources for the Future).
Mansfield, Carol, George Van Houtven, F. Reed Johnson, Zachary Pekar, Jui Chen Yang,
Timothy Gibson, and Douglas Crawford Brown, 2002, "Behavioral Reactions to Ozone
Alerts: Study Design for Assessing Parents' Values for Protecting Their Children's
Health," Presented at the Southern Economic Association Meetings, November 24-26,
2002.
Neill, Jon R., 1988, "Another Theorem on Using Market Demands to Determine
Willingness to Pay for Non-traded Goods," Journal of Environmental Economics and
Management 15 (2): 224-232.
Shogren, Jason F., 2001, "Children and the Environment: Valuing Indirect Effects on a
Child's Life Chances," Contemporary Economic Policy 19 (October): 382-396.
Smith, V. Kerry and H. Spencer Banzhaf, 2003, "A Diagrammatic Exposition of Weak
Complementarity and the Willig Condition," American Journal of Agricultural
Economics (forthcoming).
Smith, V. Kerry and George Van Houtven, 2003, "Recovering Hicksian Consumer
Surplus within a Collective Model: Hausman's Method for the Household,"
Environmental and Resource Economics (forthcoming).
Willig, Robert D., 1978, "Incremental Consumer's Surplus and Hedonic Price
Adjustment," Journal of Economic Theory 17 (February): 227-53.
54

-------
ABSTRACT
Family Decision Making and the Value of Preventing Childhood Developmental Impairment
Alan Krupnick1, Wiktor Adamowicz,2 Ann Bostrom3 and Sandra Hoffmann1
Resources for the Future, 2U. of Alberta, 3Georgia Institute of Technology
Objectives. This multi-disciplinary study has three objectives: (1) to provide more comprehensive
valuation of reducing risk of childhood developmental impairment neurotoxins by estimating parental
willingness to pay (WTP) to reduce lead-paint hazards; (2) to develop more accurate methods of
eliciting parental WTP to protect children's health by testing the impacts of using intra-household
resource allocation models rather than unitary household models; and (3) to develop more systematic
approaches to designing non-market survey instruments by using mental models research to better
understand the underlying decision processes.
Experimental Approach. Current research on parental WTP to protect children's health from
environmental hazards models household choice as an action by a single decision-maker with a unitary
utility function and a pooled budget. A large body of literature in economics and sociology of the
family and in cognitive psychology suggests that these assumptions are flawed, and may lead to
incorrect measures of parental WTP to protect children's health from environmental hazards. The study
proposed here tests these assumptions.
Environmental neurotoxins can have permanent effects on children's intelligence, motor development,
and attention. New research suggests that environmental exposure may be a larger contributor to
developmental impairment than previously thought. Lead paint exposure will be used as the representa-
tive neurotoxin hazard. Lead paint abatement options provide an excellent vehicle for testing unitary
versus bargained household models in estimating parental WTP to protect children's health. EPA cur-
rently relies on more limited cost-of-illness and human capital estimates of the benefits of protecting
children from lead and other neurotoxin hazards.
This study has two phases. The first phase will elicit "mental maps" of parents' risk perceptions and
decision making about reducing their children's health risks from lead paint. Thirty couples will be
interviewed individually and as a couple about their risk perceptions, definitions of the decision problem
and choice set, and roles in family decision making. Results from phase one will guide development in
phase two of a attributed-based/conjoint contingent behavior survey of 250 couples to assess the
influence of individual and shared parental risk perceptions and preferences on household choice of
lead-paint abatement to protect children.
Expected Results. This research will produce estimates of individual and household willingness to pay
for reduced risk of developmental impairment in children. It will also generate measures of the degree to
which estimates from bargained and unitary household models differ. Insights into household decision
making on environmental health risk issues from the valuation and mental model results will help guide
future valuation efforts as well as neurotoxin risk communication programs.
55

-------
Robin Jenkins's Policy Discussion of
"Benefit Cost Analysis and the Entanglements of Love"
Theodore C. Bergstrom, In progress, October, 2003 and
"Weak Substitution, Environmental Vulnerability, and Choice"
V. Kerry Smith, Mary F. Evans, H. Spencer Banzhaf, and Christine Poulos, October 6, 2003
I will discuss Prof. Bergstrom's paper, the Smith et al paper, and end with connections
between the two.
Prof. Bergstrom's paper has two broad objectives. The first is to try to figure out how
introducing an important aspect of real life into economic models will affect our interpretation of
benefit-cost analysis (BCA). The important aspect is family relationships. How will accounting
for the interdependencies of families affect how we conduct and interpret economic analysis?
Prof. Bergstrom's second objective is to simultaneously figure out whose willingness to
pay (WTP) is valid when conducting BCA for families. (Should one examine the mother's, the
father's or the child's WTP? Some combination thereof?)
Both of these objectives are important to policy-makers. EO 13045, issued by the
Clinton administration in the late 1990s, directed policy makers to consider health risks to
children. This introduced a new need for policy analysts to try to look separately at the impact
of government policies on children. It turns out that this task is virtually impossible if one relies
on the traditional tools of economics in which individuals maximize utility for themselves
subject to a budget constraint. Children are not rational beings, they don't have access to the
family budget and they don't operate as individuals. Children are inherently connected to family.
Thus, a new question arose - how do we manipulate the tools of economics to find out whether
we've improved the welfare of children? Prof. Bergstrom's paper is taking a stab at the answer.
He's really asking how we use the traditional tools of economics (utility theory and BCA) in
new, perhaps somewhat unconventional ways, in order to get at valid measures of the effect of
policy on child and family welfare.
Prof. Bergstrom starts by reminding us what exactly we get from BCA, anyway. BCA
tells us whether a project leads to a potential Pareto improvement (PPI), a situation where the
winners could potentially compensate the losers. In the simplest model of an economy, to check
for a potential Pareto improvement, one would just examine whether the sum of individuals'
WTP exceeds the cost of the project.
Prof. Bergstrom suggests, however, that the relationship between BCA and potential
Pareto improvements gets more complicated when you move away from the individual and try to
model family relationships in which people care about one another's well-being. To understand
the effect of family relationships he shows that we need a theory of household decision-making.
He proceeds to examine households of different compositions and with different decision
structures:
56

-------
Household Compositions, Decision-Making and WTP
Household Composition
Decision-Making Structure
Whose WTP for BCA?
Single parent
Dictator
Parental WTP
Child-free Couple
One is dictator
Dictator's WTP

Unanimity
WTPm=WTPf

Bargaining solution
Minimum WTP
Married couple with child
One is dictator
Dictator's WTP

Unanimity
WTPm=WTPf

Bargaining solution
Minimum WTP
Divorced couple with child
(independent budget)
Separate dictators
WTPm + WTPf
Extended family
(independent budget)
Separate dictators
Follow rule for nuclear
family + WTP ext fam
Prof. Bergstrom offers a new criterion for passing BCA when family decisions are being
accounted for: The sum of parents' WTP must exceed project costs to pass the BC test for
parents. As he has worded it, the criterion clearly reveals whose perspective counts: parents.
We don't add a child's WTP to parents or we'd end up accepting projects that don't meet this
new criterion.
For each household construct, what he considers to get the third column, is a hypothetical
government project that improves the health of children for the families with kids, or that
improves the health of the female member for the families without kids. As the third column
shows, to assess this criterion, depending on the composition and decision-making structure of
the family, the policy analyst would collect WTP from different people. The following rule
seems to fall out:
Rule for appropriate WTP:
"Interview all families who care about the child and operate with independent budgets. For each
of those families,
a)	if there is not unanimity between the adults, then count the WTP of the head (dictator) or, if
there is not a head, count the WTP that is the lower of the two adults' WTP;
b)	if there is unanimity, count the WTP from either individual."
This suggestion has immediate practical implications. For analysts relying on parental data, one
must now acknowledge that parental WTP might overestimate if the un-interviewed spouse has a
lower WTP, or for divorced couples, it might underestimate since really we should be adding in
57

-------
the WTP of the ex-husband/wife too.
In addition, if there are segments of the population that are more or less likely to be
characterized by one or another of the family compositions or the decision-structures then those
sub-populations might be systematically mis-represented. One can see how this is likely to be a
real problem since culture is an important determinant of the prevailing power structure within
the family. For example, the head-of-household model is probably more common among
Hispanic families than non-Hispanic. In addition, certain family compositions are more likely
among certain sub-populations - single parent families among inner-city African Americans.
Thus the mis-representation of certain sub-cultures might follow from neglecting to model one or
another of the possibilities in the table.
Prof. Bergstrom points out that in most of these models, a project could pass the BC test
for parents, and children could experience a decline in utility. This is because of substitutability
between the child's health and consumption. The parent might reduce the child's consumption
by enough to offset an improvement in the child's health, so that the child's utility actually
declines. Prof. Bergstrom offers a practical example of an impoverished family that requires a
child to go to work once his health is restored. This is a disturbing possibility — that by relying
on parental WTP, policy makers might choose projects that ultimately lead to reductions in the
direct utility and consumption of children.
This is a disturbing possibility and one to be aware of but, as Prof. Bergstrom himself
points out, at least for western cultures, it seems like a remote one. In response to the child's
improved health, the parent would have to reduce the child's consumption by an amount big
enough to offset the improved health. I would assert that in Western families, health might be
more realistically modeled as complimentary to consumption, since healthy kids probably eat
more, are more concerned about social pressures regarding dress and play, and probably recreate
and travel more too. In other words, improving children's health is not likely to decrease their
direct utility for families in the U.S. at least not as a consequence of parents considering child
health to be a substitute for child consumption of goods.
An interesting question is that as this paper develops, will there be more examples in
which direct child utility declines even though a project passes the parental BC test — examples
more applicable to western cultures. Are there certain cases or circumstances where we should
be concerned about this outcome? Clearly there are outlier parents - drug addicts, for example —
for whom we should be reluctant to rely on parental WTP to represent the well-being of children.
But are there also broader cases or situations for which we should exercise caution when relying
on parental WTP?
A final comment about the importance of Prof. Bergstrom's work. It goes beyond
implications for the interaction of policy and child health. In fact, just as children are inherently
family members, so too are most adults. In the final section of his paper, Prof. Bergstrom
suggests adding together the WTP of divorced couples and adding to the WTP of a nuclear
family, the WTP of extended family members with independent budgets. These suggestions
58

-------
imply that for adults we should also collect WTP for adult health from family members operating
in households with independent budgets. For cultures like ours and most Western ones, where
household budgets are fairly independent but there is a great deal of concern for extended family
member's health, Prof. Bergstrom's work suggests that individual WTP probably understates
society's valuation of health.
Turning to the Smith, Evans, Banzhaf and Poulos paper . . .
The major thrust of this paper is to propose a new framework for revealed preference
research. As a policy analyst, after reading the first few paragraphs of this paper, I was excited
about the significance of what they suggest. Clearly, analysts would find very useful yet another
set of conditions under which we could tease out values of non-market goods or more
specifically, for the EPA, environmental goods. The paper sets out to show that when certain
conditions hold regarding the interrelationship between a private market good and a substitute
environmental good, economists can study the demand for the private good and tease out
valuation of the environmental one.
We are accustomed to thinking this way about complimentary goods - for years
economists have been learning about the value of natural areas by examining recreation demand;
e.g., the demand for fishing or for beach house rentals. As the authors of this paper highlight,
Rick Freeman's much-cited text on Measuring Environmental and Resource Values covers the
conditions surrounding weak complementarity. His latest version of the text, however, has all
but eliminated discussion of weak substitutability. The authors intend, with this paper, to correct
this oversight and to encourage economists to study, or at least be more aware of, this neglected
category of market products - that is, substitutes for difficult-to-value non-market goods.
For weak complementarity, two conditions must be met. Together they suggest that
when the price of the complementary market good is at or above its choke price (so that
consumption is zero), changes in the environmental good have no welfare significance. Weak
substitutability also requires that two conditions hold. Together they suggest that there exists a
level of consumption for the private good (the weak substitute) above which improvements in
the non-market environmental service have no value. In other words, if the price of the weak
substitute is high enough, consumption of the market good is low and increases in the
environmental good are valuable.
One can easily see how positing the conditions for weak substitutability might offend
environmentalists. The hypothetical is that there is a market good that is a substitute for some
set of environmental services. If the price of that market good drops low enough, then people no
longer have a need for the non-market environmental services. Years ago when I taught an
undergraduate environmental economics course, following Tom Teitenburg's textbook, I would
start the semester by exposing students to ideas of the "optimists" and the "pessimists." The
citations to these schools of thought go back at least to the 1960s. The pessimists believed the
world's population was growing at a rate that would outstrip resources and lead to a collapse
59

-------
with starvation and misery. The environmentalists in the classroom were sympathetic to these
ideas. The optimists believed that advances in technology would outpace resource depletion and
eventually replace the need for the natural world. Generally speaking, the optimists viewpoint
was disliked by the environmentalists.
Drawing a connection to the paper, in general, studying goods that are complementary to
nature, suggests that we have a continuing need for the natural world. These products
complement or enhance our experience of the natural world. However, studying goods that
substitute for nature, the very notion that goods can completely replace the demand for
environmental goods, is in concert with the ideas of the optimists. The weak substitutability
condition that there are prices of the market good below which changes in the quality of the
environment are no longer valued might be more offensive to some people than the choke price
assumption behind weak complementarity.
This leads to a practical question: Is the weak substitutability condition more restrictive
or less representative of practical experience as well? I think not as long as one carefully
characterizes the case under study. The private market good substitutes for a specific subset of
services derived from an environmental good, and generally not for the entire set of services.
The paper would do well to emphasize this caveat.
This point is illustrated by the examples identified by Smith et al in which weak
substitutability might lend insight. Both are drawn from the existing literature and in both, the
substitute goods are medical care and certain aspects of a cleaner environment. In other words,
the consumer is substituting an averting or mitigating behavior for a cleaner environment, in
both examples for their children. Agee and Crocker look at chelation therapy as a substitute for
removing lead from a child's environment; Mansfield et al study removing children from the
out-of-doors as a substitute for cleaning the air of pollutants that exacerbate asthma.
Certainly, if analysis of weak substitution can be as illuminating for environmental
valuation as weak complementarity has, as the authors suggest, then the examination of averting
and mitigating behaviors takes on a new importance. A simple application that comes to mind
might be to examine purchases of bottled water as the route to valuing cleaner water.
I wanted to draw attention to an important connection between the two papers. In the
final section of the Smith et al. paper, the authors highlight that in order to impose weak
substitution in analyses of the costs of mitigating or averting behavior, analysts need more
information about decision-making within the household . Clearly, Bergstrom's paper is a step
in the right direction to fulfilling that need.
To close, both papers advance the state of knowledge regarding non-market valuation
which ultimately is useful to policy-makers as an input into BCA; for analysts at the EPA for
BCA of environmental policy. The Bergstrom paper gives insight into family decision-making
and into the appropriate willingness-to-pay measures for children's health. The Smith, et al
paper suggests a new approach to valuing environmental services, and in the examples they
60

-------
offer, the value stems from protecting human health. These papers make clear contributions to
advancing policy-makers understanding of the benefits of human health protection.
61

-------
Mark Agee's Comments on: "Weak Substitution, Environmental Vulnerability, and Choice,"
by V. Kerry Smith, Mary F. Evans, H. Spencer Banzhaf, and Christine Poulos.
This paper examines usefulness of identifying a threshold demarcating
consumption and non-consumption of substitutes linked to an environmental amenity to
assess Hicksian welfare changes associated with amenity improvements.
The paper provides an interesting and seeming applicable framework for use of
revealed preference data to valuing environmental amenities. A highly desirable aspect
of this framework is its focus on observable goods demands (i.e., does not necessitate
specification/estimation of the consumers' underlying home production technologies) and
the consumers' observed tradeoffs between these demands.
The first part of the paper lays out a detailed graphical portrayal of the concept of
welfare measurement using weak substitution as another form of demand
interdependency that arises for private goods and environmental services. I found the
approach very interesting, and most of my comments/questions focus on my thoughts as
to how the approach might be put in to practice.
Application-related questions/comments:
On page 14, z is referred to (from this point on) as a "mitigating good." Given
this characterization, are we saying (or can we say) in effect that the separability notion is
one of partitioning the classes of goods (averting and/or mitigating) that identify as
substitutes in the individual's choice set—of which are then linked to the individual's
utility and environmental amenity through perceived health, or risk to health, etc.? If so,
then it seems that a binary choice model might be useful in identifying and estimating
key thresholds of interest (you refer to these thresholds as points of discontinuity in the
role for q) as well as identify the conditional demand for z?
•	e.g., suppose Xa represents a substitute (averting) good such that, at a threshold, z
(a mitigating good) is no longer demanded. A binary choice model similar to
Agee and Crocker (1996) and Dickie and Gerking (1991) could be used to
identify either positive or zero consumption levels of z by estimating:
•	v(P, Pz,Pr, q, y, m) + error,
or
•	y[Pz, P, q, r, ™v (P, P„Pr, q, y, m)\ + error,
where v(-) is an econometric specification of a conditional indirect utility function for z,
and y accounts for personal characteristics.
Estimation of the probability of "positive" consumption would also provide
62

-------
•	an estimate of P*,
the price of Xa that reduces z to zero (or the individual's estimated choice index below
the threshold), and
•	an estimate of (the Marshallian) z as a function of prices, income and personal
characteristics: z(Pz,P,Pr,q,m,y) = - dvO/dPz ^ase(j on anaiySis 0f the
dv(-)/dm
threshold of no consumption.
The paper also mentions the importance of demographic and physical traits that
contribute to characterizing individuals who are differentially vulnerable to the
environmental hazard in question. If the model can be estimated as a threshold of zero
consumption, it seems that the level of q is also important in identifying key aspects of
the price P* since the relationship between goods linked to q can change as q changes:
•	e.g., with reference to child lead exposure—there are burdens of body lead such
that mitigation is not even considered; burdens such that mitigation together with
exposure reduction are absolutely necessary (complementary goods); and burdens
(likely close to the threshold) such that exposure reduction can suffice for
chelation therapy (substitute goods).
•	This of course would change the sign of the coefficient for P* (i.e., only used,
used with chelation, used as a substitute to chelation) in the estimated v(-)
expression.
In these cases, it seems that a set of interactions between P* and q (and between P* and
some of the personal characteristics) would be necessary to identify any critical
thresholds for potential sign-changes of the P* coefficient, as well as to correctly identify
(calibrate) measures of z0, zi, and P* for estimation of WTP.
Another estimation issue is encountered if X is multidimensional. If X is a vector, is
it in general necessary to identify the entire vector of X in order to correctly specify z?
•	e.g., z might denote (one-dimensional) medical care consumption that mitigates
air pollution respiratory ailments; and X denotes exposure reducing activities that,
at a point, may render mitigation either very improbable or unnecessary..
Can the problem of multidimensionality of P in z be dealt with adequately by
estimating a specification of v[-] (e.g., like the second v[-] specification mentioned
above) that includes each individual's observed fraction, mv, of their budget allocated
towards z and a single selected element of P (such as the price of air purifiers)? If so,
since mv is a function of all prices, q, income, and personal characteristics, it seems that
63

-------
one might want to account for possible correlation between mv and the error term to
avoid potential bias of the P and other coefficients in the binary choice regression (see
e.g., Rivers and Vuong (1988) or Wooldridge (2002), p. 472 for cross section data; or
Jones and Landwehr (1988) for panel data).
—or—
Might it be possible to construct a one-dimensional averting activities index (like
intensity of use from a limited set of "most used" averting activities) with an
accompanying averting cost index that condenses P to a single measure for estimation?
Sources:
Agee, Mark, and Thomas Crocker. 1996. "Parental Altruism and Child Lead Exposure:
Inferences from the Demand for Chelation Therapy," Journal of Human
Resources 31: 677-691.
Dickie, Mark, and Shelby Gerking. 1991. "Willingness to Pay for Ozone Control:
Inferences from the Demand for Medical Care," Journal of Environmental
Economics and Management 21:1-16.
Jones, J. Morgan and Jane T. Landwehr. 1988. "Removing Heterogeneity Bias from
Logit Model Estimation," Marketing Science 7(1): 41-59.
Rivers, D., and Q.H. Vuong. 1988. "Limited Information Estimators and Exogeneity Tests
For Simultaneous Probit Models." Journal of Econometrics 39: 347-66.
Wooldridge, Jeffrey M. 2002. Econometric Analysis of Cross Section and Panel Data
Cambridge, Mass.: MIT Press.
64

-------
Suppose X9 represents a substitute [averting] good such
that—at a threshold—z [a mitigating good] is no longer
demanded. A binary choice framework can identify
either positive or zero consumption levels of z using:
•	+ error
--or--
. v[Pz,P,q,r,mv(P,Pz,Pr,q,y,m)] + error
where
K~) = specification of a conditional indirect utility
function for z
Y = personal characteristics
Estimation of the probability of "positive" consumption
provides
• an estimate of
P
*
a
•	the price of X3 that reduces z to zero (the
individual's estimated choice index below the
threshold)
•	an estimate of (the Marshallian) z :
dv(.)/dPz
z(Pz,P,Pr,q,m,y)
dv(-)/dm
65

-------
Mark Agee's Comments on: "Benefit Cost Analysis and Entanglements of Love,"
by Theodore C. Bergstrom
This paper develops a creative approach to addressing some important questions
about aggregation of willingness to pay (WTP) when WTP has altruistic connections
between individuals within a family. Four specific questions are addressed:
•	When considering a public project that increases children's health, would it make
sense to calculate the aggregate value of that project by summing the average
parent's WTP by the total number of parents; or
•	if parents (within the same household) reveal WTP differences for the project,
should the maximum or minimum of these values be used (exclusively) to
represent the entire household's value; and
•	should children's own values be accounted for and included; and
•	should values of family members outside the immediate family be counted.
These questions are addressed within the frame of a specific criterion: is the project
Pareto improving? That is:
•	is it possible to implement the project and assign project costs to families in such
a way that, given the household decision structure, no family member is made
worse off and at least one member is made better off?
The initial part of the paper rearticulates this criterion by demonstrating that, if
parents have complete control of their family incomes, if one parent is forced to pay more
than his/her WTP for a project (implying project costs exceed the sum of parental WTP),
then the project is not Pareto improving—and thus the benefit-cost test for parents
provides a clear-cut gauge of satisfaction of the Pareto criterion.
The benefit-cost test is then applied to a variety of family structures involving one or
two parents with and without a single child, and a variety of family preference and
distribution structures common to the household decisionmaking literature. The paper
acknowledges that the preference and distribution assumptions of some of the models do
not accurately portray modern U.S. households. However, a few of these models had
structural assumptions that came (somewhat) close to the frameworks found in the
limited number of literature studies examining parental valuations of specific child health
attributes. Specifically:
•	A parent decisionmaker who allocates after-tax income between own utility-
enhancing consumption and child utility-enhancing consumption;
•	A parental utility function that derives utility from parental perceptions of child
utility; and
•	An exogenous index of child health that enhances parent utility through child
utility.
Although the basic structure of these examples (within the context of the above
assumptions) preclude the possibility of endogeneity of parental decisions regarding child
health and child numbers, the paper demonstrates a result similar to prior studies:
•	that parental WTP for a child's health improvement equals the health
improvement times that adult's MRS between own consumption and child health,
66

-------
but it also finds that
•	the parent not charged with income allocation may reveal a different WTP for the
same child's health improvement—if this parent's marginal utility of child
consumption exceeds her marginal utility her own income allocation,
in which case it is appropriate to
•	use only the lowest of the two parents' WTPs to represent the family's WTP for
the child's health improvement.
If a general household welfare function is used to link all family members' utilities, then:
•	only one parent's WTP (which equals the other parent's and child's WTP) should
represent the entire household's maximum WTP for the child health
improvement.
However, if family utilities are disconnected (e.g., because of divorce or independent
budget arrangements) then
•	the sum of adults' WTP for the child health improvement satisfies the benefit-cost
test.
These results suggest that aggregate WTP estimates of children's health
improvements may inaccurately reflect true WTP if calculated simply as the "average" of
parent's WTP multiplied by the number of U.S. parents. The paper highlights the
importance of how specification of family allocation processes and the structure of family
preferences can potentially impact aggregate WTP estimates. The question of whether to
aggregate by U.S. family numbers, number of U.S. parents, or by U.S. parents
categorized by their degree of "control" over household resources provides a good start to
better understanding how aggregate WTP for children's health improvements can most
justifiably be approximated. Listed below are some questions/comments that came to my
mind upon several readings of the paper:
First, I'm not entirely convinced of the plausibility of the argument on page 4
regarding strong substitutability between parental income and child health and the
potential for children being made worse off by their parents. The argument in the
household utility function that seems to be missing here is parental own health, which has
an intergenerational link (heritability) to child health. Would healthier parents (who
likely have healthier children) necessarily behave in this fashion? The question closely
parallels the discussion of parent and child endowments and parental investments in child
human capital found in Becker and Tomes (J. of Labor Economics., 1986, 4:S1-S39).
Although children with better endowments are much more productive/efficient utilizers
of human capital investments, better endowed parents still discount investments in their
(better endowed) children at lower rates and invest more resources in them.
Second, does the current preference structure impose that parents treat their children
as a "collective child" with an expressed, single willingness to pay for their improved
health? An important question in my mind is—if we randomly select one child in a
family would the parent's willingness to pay for that child's health improvement,
multiplied by child numbers be the correct sum? For example, suppose expressions (10)
and (11) in the paper incorporate a summation over n (predetermined) family members
thus creating a household welfare function that varies also by n. With this (seemingly)
67

-------
minor change, would the appropriate number for benefit-cost analysts likewise be a
single number representative of either parent or a single child—or would summing either
parent's response across child numbers overstate aggregate WTP by a factor of n-21
As an additional thought, the current preference structure has child utility nested in
parental utility reflecting the concern each parent has for their child, i.e., the parental
marginal utility of child utility. Perhaps child numbers could enter as in Becker, Murphy,
and Tamura (J. of Political Economy, 1990, p.S12-S37) who assume that, with diminishing
marginal utility of children, parental altruism is negatively related to child numbers (they
also refer to parental altruism as an intergenerational discount rate applied by parents to the
per capita consumption of their children). With this modification, how might diminishing
marginal utility of children relate to the parents' determination of WTP for child health
improvements, and does it provide any insights as to whether parental WTP is for 1 In or
some other fraction of child numbers?
Finally, another question might be posed with reference to the role of child health
in the determination of family utility. Certainly, the current exogeneity of child health in
child utility enables ease of mathematical tractability; however, if child utility is modified
such that parental allocations of child consumption goods also influence child health,
would endogeneity of child health bring about any changes in your results?
68

-------
Summary of Q&A Discussion Following Session II
Ronnie Leven (EPA, Region 1) stated that in addition to complications in the theory,
there are complications in the cases being used, specifically the lead abatement studies.
She went on to assert that "chelation therapy is not all good"and actually leads to a spike
in blood lead levels and a correlating spike in brain lead levels. She also noted the spike
in exposure after certain types of abatement that release lead paint dust into the air to be
inhaled, and she listed encapsulation, replacement, delay, improved maintenance, and
temporary removal as alternatives to immediate abatement. She ended by saying that the
studies on purchasing bicycle helmets also had some complicating health issues.
Scott Grosse (Centers for Disease Control and Prevention) suggested this extension for
Professor Bergstrom: Consider willingness to pay for health as a public good and not just
as a private good. Dr. Grosse stated that "if we treat health as a private good, then you
can't include the willingness to pay for the extended family members who also have
children of their own."
Professor Bergstrom responded by saying that he was thinking about how a particular
questionnaire related to a specific project should be framed ("it might be a local
community project, it might be a national whole airshed, or it might be a national
project"). To him it seems reasonable to ask someone, "How would you feel about an
improvement in the health of your child?" or, in fact, "How would you feel about an
improvement in the health of each of your seven children?" and then on to, "How would
you feel about an improvement in the health of your nephew?" Professor Bergstrom said
that he would propose evaluating that project then by building up from these micro-
answers.
Grosse: "But, it may be that people are not willing to spend "n" times as much to help
"n" children."
Professor Bergstrom reiterated that you would ask a sequence of questions-regarding the
valuation of the health of one's own children, and then on to a relative's children, and
even on to a stranger's children-as the building blocks to constructing a policy.
Ellen Post (Abt Associates, Inc.) told Professor Bergstrom that she was bothered by the
fact that assigning the willingness to pay value entirely to the dictator parent in a
dictatorial household model (and thereby ignoring the other parent because that person,
though possibly willing, has nothing to pay with) basically incorporates ////ra-household
politics into the policy assessment. She further stated that the belief "if one person
controls the budget then only that person's willingness to pay should be counted" would
be true for all similar situations of valuing non-market goods.
Professor Bergstrom replied, "Yes, we'll be a little careful about the "should," of course,
but essentially if you believe that your policy will not change the structure of household
69

-------
decisions, then it seems to me a realistic assessment of policy is the consequences of the
policy given the household structure." He also provided the reminder that, at least in
theory, this is "a benevolent dictator who buys the flavor of ice cream that his children
want, and so on," and clarified that "whether that theory is an accurate theory is yet
another question."
Post: "I guess I was just saying that the idea of saying that there are different kinds of
families with different intra-family politics which determine people's budgets-that has a
lot wider implications than, say, just children's health risks.
Bergstrom: "Indeed, I agree."
Laurie Chestnut (Stratus Consulting, Inc.): stated that she was "alarmed" at the
suggestion that we should be seeking to determine various relatives' willingness to pay
for a particular child's health. Citing a paper concerning altruism from a few years ago
by Jones Lee, she said it was her understanding that "if what people care about is other
people's utility, then the optimal allocation is just adding up everyone's willingness to
pay for their own utility change, and that takes care of everybody." She further stated that
it's only in the case where one cares about another's consumption of a specific thing that
the willingness to pay issue gets more complicated. She closed by saying, "what we care
about is each other's happiness, which I think is the primary model of families caring
about each other."
Professor Bergstrom responded that it's a bit tricky with a divorced family, where the
money transferred from the non-custodial parent to the custodial parent does not go
directly to the child-but, a health benefit does. He stated that if a divorced parent
voluntarily transfers money to the custodial parent, then one can presume that they had
come to an agreement about where marginal money should go.
Chestnut: "So, health might be a special case in some circumstances . . ."
Professor Bergstrom replied that he had put a lot of thought into the relatives issue and
concluded that if you're not willing to give your brother some money and say "go buy
your child some medicine," maybe that suggests that you find no extra valuation in that.
"On the other hand, it may be that institutions aren't well set up."
Don Kenkel (Cornell University) asked this of Robin Jenkins, referring to her discussant
presentation of Ted Bergstrom's paper: "Is the potential Pareto improvement criterion
really consistent with the Executive Order?-sort of plucking out children and saying,
"This deserves special attention."" Is this suggesting that even if the social welfare
function doesn't pass the sufficiency test, we should still worry about children's
welfare?"
Robin Jenkins: "In other words, do we want transfers to children?
70

-------
Kenkel: "Even if the parents don't want them."
Spencer Banzhaf (Resources for the Future) followed with a comment that he said is
actually very similar, and he pointed out that toward the end of Ted Bergstrom's
presentation, wasn't it ironic that there was clearly a potential for Pareto improvement,
but "the husband was made worse off because of the reallocation in the family?" He
concluded that "that irony raises this doubt about the usefulness of potential Pareto
criteria when the agency in charge of the policy does not actually have the power to
reallocate wealth . .
Kerry Smith (North Carolina State University) said that he had a different take: "If we
take the way Ted characterized the household model and different views of it and look at
what he's doing ... In environmental economics we have sort of two ways we've
approached looking at environmental policy: One way has been to present people with
programs, and we've said, "Here's a program to do this-or do something else, on a
national level." So the commodity that was actually in preferences was however they
conceived of the program, and there were all sorts of questions raised with respect to
"Well how should people have preferences about programs?" He's actually proposing to
commoditize programs, and so he's basically saying, "We're going to introduce a
program that has these kinds of consequences and let's be very, very specific about what
those consequences are-it would improve the health of child #1 this much, child #2, the
wife, and so forth. . . . But the challenge then becomes converting programs into what
commodities would change, so that now we have, if we get very, very specific, with
individuals-my nephew's health is going to improve; my niece is going to go down the
rathole or something-I then have to think about exactly how is that going to be
accomplished with the policy or with the program. So, we don't get around the challenge
of connecting policy to commodity outcomes-we simply do a better job of identifying the
features and preferences that we can recover from behavior and what we can't recover
from behavior, whether it's stated or revealed."
Professor Bergstrom responded that if people are asked how they feel about a policy that
they don't understand very well (which is typical), "their answer will be of some interest
to politicians, of course, but to welfare economists of less interest I suppose. The point
is, it always seems to me a useful thing to map policies to consequences as well as
possible."
Glenn Harrison (University of Central Florida) directed his comment at Professor
Bergstrom but said it was motivated by Robin's "paternalistic"comment that kids are not
rational. He said he was disappointed that Professor Bergstrom didn't address the issue
of how kids form preferences, and he lamented that all of the discussion so far in the
workshop totally ignored the kids preferences and went straight to the parents. "Now, I
understand the simplicity of doing that-I understand the logistic ease of doing that, but
it's a pretty sloppy way of thinking about the social welfare function. Just because kids
71

-------
are vertically challenged, we disenfranchise them." He summarized Robin's viewpoint
as being that kids don't have well-formed preferences so we can't rely on them, and then
he posed the question: "Shouldn't we be thinking about kids' preferences as state-
dependent, broadly defined, where the state is the information they have and at a certain
age and with a certain information load, they act more consistently over time? And the
question, then, is what implications does that have when you start thinking about social
willingness to pay as distinct from aggregating up from an individual willingness to pay?
And indeed, arguably we're interested in social willingness to pay."
Professor Bergstrom replied that it was "a lovely question" that he was reluctant to tackle
at the time due to the upcoming lunch break. He offered to speak to Dr. Harrison on the
subject later.
J.R. DeShazo (UCLA) commented, "Ted's presentation, I think, illustrates that when we
elicit values from parents what we're really eliciting are household preferences for
investments in children's health and the benefits that accrue to children as perceived by
the households, so that households can express the schedule of values for the children as
a function of the number of children they have, as a function of the governance structure
within the household. I simply want to point out that this approach misses the
fundamental theoretical construct that we're trying to recover, which are the net benefits
to the child of the policy. I think we should just recognize the disconnect between what
we can measure and theoretically what we optimally would like to know in order to set
the optimal policy."
Professor Bergstrom replied, "That is a technical issue that in a sense I did address. The
question is to what extent do the parents' evaluations of the child help represent the
child's interests. . . If there's complementarity between the child's health and the child's
conception, then definitely the parent will always be acting in the child's interest, and if
there's not, there are cases where indeed the parent will not, despite the fact that the
parent cares about the child's interests. I can't give you magical answers, you know."
Sandra Hoffman (Resources for the Future) said, "It seems to me that there is a logical
inconsistency in the discussion we're having here, because on the one hand we're saying
that children do not have well-formed preferences-they haven't developed the judgment
that's able really to express preferences-and yet when we look at parents' preferences,
we're looking at the child's utility entering the parent's utility function. So there's a
transformation going on there. We're not directly measuring the child's benefits, and
maybe we don't want to. So it seems to me that J.R.'s point still stands-we're at best
getting at a transformation, and perhaps . . ."
Professor Bergstrom clarified that this is not such a difficult issue-the parent's and the
child's notions of what's good for the child "needn't be identical-it's as simple as that."
72

-------
Glenn Harrison said, "But no one here has assumed that the child does not have well-
formed preferences."
Professor Bergstrom responded, "I've never made such an assumption. I think it's a very
interesting issue, but again, I don't want to spend our lunchtime here."
73

-------
Valuing Environmental Health Risk Reductions to Children
PROCEEDINGS OF
SESSION III: VALUING FETAL AND INFANT HEALTH EFFECTS
A WORKSHOP SPONSORED BY THE U.S. ENVIRONMENTAL PROTECTION AGENCY'S
NATIONAL CENTER FOR ENVIRONMENTAL ECONOMICS (NCEE), NATIONAL CENTER FOR
ENVIRONMENTAL RESEARCH (NCER), AND OFFICE OF CHILDREN'S HEALTH PROTECTION;
AND THE UNIVERSITY OF CENTRAL FLORIDA
October 20-21, 2003
Washington Plaza Hotel
Washington, DC
Prepared by Alpha-Gamma Technologies, Inc.
4700 Falls of Neuse Road, Suite 350, Raleigh, NC 27609

-------
ACKNOWLEDGEMENTS
This report has been prepared by Alpha-Gamma Technologies, Inc. with funding from the National Center
for Environmental Economics (NCEE). Alpha-Gamma wishes to thank NCEE's Kelly Maguire and
Project Officer Nicole Owens for their guidance and assistance throughout this project.
DISCLAIMER
These proceedings are being distributed in the interest of increasing public understanding and knowledge of
the issues discussed at the workshop and have been prepared independently of the workshop. Although the
proceedings have been funded in part by the United States Environmental Protection Agency under
Contract No. 68-W-01-055 to Alpha-Gamma Technologies, Inc., the contents of this document may not
necessarily reflect the views of the Agency and no official endorsement should be inferred.
11

-------
TABLE OF CONTENTS
Session III: Valuing Fetal and Infant Health Effects
Preliminary Research: Combining Psychological Economic Methods To Improve
Understanding of Factors Determining Adults' Valuation of Children's Health
Cheryl Asmus, Colorado State University 	1
Pregnant Mother's Valuation of Own and of Child Health
Thomas Crocker, University of Wyoming	16
Commissioned Paper: Valuing Fetal and Infant Health: What Can Be Learned
From Empirical Health Economics Literature?
Don Kenkel, Cornell University	56
Discussant
Trish Hall, U.S. EPA, Office of Ground Water and Drinking Water	95
NOTE: Dr. V. Kerry Smith of North Carolina State University also presented discussant
comments, but his remarks are not included in these proceedings.
Summary of Q&A Discussion Following Session III	108
ill

-------
Of- -" A ¦.>'	'"<>. OF'	>-*'	^ ' *•.% ¦.>'	.*• ' *;\ ..>'
Combining Psychological and Economic
Methods to Improve Understanding of
Factors Determining Adults' Valuation
of Children's Health
. ->¦ . ->¦ . >¦	m
Cheryl Asmus, Paul Bell, John Loom is, and
Helen Cooney
Colorado State University
/
:fc&'3m;

-------
.'W^fe'
4*	uF- " ' '*;,	uf-	JF- ' ' •*;.,	'"" '''i; <*• •" "•*... •.-¦•'
Research Question
i f "f V f s ''^TV. ? ~ '$
• What are the factors that influence adults'
willingness-to-pay to avert risks to children's
health?
•	Stressor -> Nitrate (N03) contaminated drinking
water
•	Model -> Theory of Planned Behavior
•	Method -> Survey and choice task
2

-------
Attitudes
Subjective
Norms
Behavioral
Intentions
Knowledge
Beliefs
Perceived
Control
Actual
Behavior
3

-------
gLv	¦W>^'''vi
•jf '^.	wf •;• .;.•'	-jf ^S. ¦ >'	-jf \:;	_-
Participants
5 "'^^. / "¦ ,; '"¦'$ ^ '-' '1§ f '-¦ " J? f f %.
•	520 participants
•	90 from Fort Collins (control group)
•	215 from the Eastern Plains of Colorado (experimental)
•	115 in consequential choice treatment
•	215 from the San Luis Valley of Colorado (experimental)
•	115 in consequential choice treatment
•	The survey/methodology will be piloted on a
sample of 30 participants (10 from each of the
geographical areas listed above)

-------
Procedure
yf \\	*5? V '	V	']{ \\	"3? Y'
Survey will assess various components of TPB
with respect to the stressor and the various
behavior options
Respondents will be presented with a
contingent valuation task
• '3? v	-S V	'5 V ¦' - ¦$ v
5

-------
Contingent Valuation Task
Do Nothing
_ ± " V'JC ¦
Purchase
Bottled Water
Risk of Severe
Health Effects
Risk of Moderate
Health Effects
6

-------
Procedure (cont.)
3^ *, ,•
M-
¦3 iL
Half of the respondents in the two
experimental conditions will be instructed
that one of the decisions they make in the
contingent valuation task will be binding
v V	'•? V	'yf v	¦ v	'''S V -
7

-------
Current Status
Vfti
A '->?¦ " - <#" ' -'A
Expert Advisory Groups
i, >	i, *,	t3s'
• Survey development
contingent valuation task

¦M,.J' *
1 V .
f •' .	• fifc
5g; V	'3? V '}f V '"3? V '"3? V
• •**».%£/. • .-r: v£ • **	• r	£T wsi^p-	~~ wff- <*'¦ ¦
mSk »: - rj$. "!fi/%
8

-------
yf-	if'- ',S^S. yf. yf- ' '	uT '	uf	>-*'
Nitrate Advisory Group
.•V ' '"9? V ¦	^ V	V	'-y( V	V V
•	Survey should assess knowledge of
different potential sources of nitrate
contamination
•	Survey should assess knowledge of how
responsibilities differ for public and private
water supplies
•	Items regarding source reduction strategies
should be broad
9

-------
uT '^:, >••'	uF' ' ' $., >-*'	uF' ' -N, >-*'	«#'• " '•V.i ->'	ur V' 'S
Nitrate Advisory Group
V ¦ !? V-	j/ V	)/ V	*$ V
•	Survey wording should avoid "test-like"
quality
•	Providing some participants with a reverse
osmosis filter would be problematic
.•V' '"5 V	'® V	''•)/ V	'*® V	: V'
10

-------
••if'''?1'
~ '-r'i'. ef- -,>'.vS, >?• '	:•	. «#'• ',"!'A >:	df'- '"".'A v? • '	'' ;% ¦',/
Health Advisory Group
' '"S; )t' '¦'• '5 V	*3? V	'"5? V	*$ V
•	Should determine if the communities under
investigation have had cases of blue baby
syndrome
•	Determine if each community has problems
with contaminants other than nitrate
•	Survey should include items pertaining to
pre-natal exposure
11

-------
Health Advisory Group
V. V	V	'V- V	yf V
respondents' own infants and infants in the
community
12

-------
5k,"'jS;,;','V V:	>	V, •».;	JL •».;
Methodology Advisory Group
•	Choice task should be simplified
•	The different behavior options should be
more compatible
•	A practice round would be helpful in the
consequential choice treatment
•	Survey should provide information
acknowledging that the options may be
beneficial for other reasons
13

-------
Methodology Advisory Group
v;	"s. v ¦ - 5 v	j v	j v ¦ -
• Costs for the different options should be
constructed to be credible and to ensure that
some participants will actually purchase the
option
' j? V	S? V "' S? V	' )? V	¦$ V
14

-------
Next Steps
'"i V ¦} V;'. f-, '- s " ••• V;'. '' s
Complete survey development
Translate survey into Spanish
Collect pilot data
'' 'jS.	uF 'jS »*	^	'	itf"
/.J "' >*>.	"'	>.
?V '. '? V	'? V	'? V	'•? i;
*¦- • ' -r: v£. ^(fr-%j£?*
- 'Y;	' •
15

-------
September 2003
Pregnant Mother's Valuation of Own and of Child Health*
by
Stefanos A. Nastis
and
Thomas D. Crocker
Department of Economics and Finance
University of Wyoming
Laramie, WY 82071-3985
JEL Classification: Dl; Q2
Keywords: Benefit transfers; birthweight productivity.
*A paper prepared for the USEPA Workshop on Valuing Environmental Health Risks to
Children, Washington, DC, October 20-21, 2003.
Although the U.S. Environmental Protection Agency provided financial support through Grant
#R2871601, the research has not been subjected to the Agency's required peer and policy review
and therefore does not necessarily reflect the views of the Agency.
16

-------
Table of Contents
Abstract	19
I.	Introduction	20
Figure 1. A Causal Chain	22
II.	The Endogeneity of Maternal Health	25
III.	Model and Econometric Procedures	27
A.	Model	27
B.	Econometric Procedures	30
IV.	Data Description	33
IV. Results	35
VI. Summary and Conclusions	41
References	43
Appendix	52
17

-------
List of Tables
Table 1. Summary Statistics	46
Table 2. Demand Estimates	48
Table 3. Mother's Period 2 Health (Weight Gain)	49
Table 4. Child's Period 2 Health (Birthweight)	50
Table 5. Mother's Value of Own Relative to Child Health	51
18

-------
ABSTRACT
The value an adult attaches to own health relative to child health is estimated when adult
health inputs are choice variables and adult health is an input to child health. Mothers' weight
gains during pregnancy and children's birthweights respectively measure adult and child health.
Estimates suggest mothers value child health about six times more than own health, and that this
relative value declines with number of siblings, increases with family income, and varies with
maternal consumption patterns.
19

-------
I. INTRODUCTION
This paper uses a pregnant woman's own - consumption in its various commodity -
specific forms to estimate the value she attaches to own - health relative to the health of the fetus
she carries. The current U.S. federal agency practice of transferring widely available adult health
benefit measures unadjusted to children's health gives the issue policy relevance.1 Also, except
insofar as it reduces household resources available to a child, the "child development" influence
of the commodity - specific particulars of a parent's own - consumption have been little studied
in economics. Yet parents engage in many activities which directly or indirectly give them
utility while simultaneously producing consequences for their children. Thus, for example, a
parent may drink alcohol excessively and subsequently abuse or neglect her child, or a pregnant
woman may indulge a diet which adversely affects the health of her fetus.
Because adults do not resemble children either biologically or economically, the current
U.S. federal agency practice of using unadjusted adult health economic benefit measures to
assess the benefits of improving children's health is suspect. Differences between the biological
responses of adults and children to many identical environmental stressors are widely
acknowledged. And children live with adults whose internal household allocation and
investment behaviors can amplify or temper these biological responses. The degree of
amplification or tempering many differ between adult and child because of differences in the
choices adults make for themselves and for their children. For example, the scope of the
activities adults choose for themselves is commonly less restricted than those they choose for
their child. Also, adult investments in children's health can be riskier and thus the return to
human capital investments less than equivalent own - health investments, given that children
1 See, for example, the health benefit transfer procedures propounded in Kuchler and Golan (1999), and in U.S.
Environmental Protection Agency (2000).
20

-------
have no performance records indicative of potential investment payoffs. Markets to insure
against this risk are more incomplete for children than for adults. But children have longer
expected life spans than do adults, which allows them to accumulate more human capital than an
adult whose capital stock is already largely built. Given the concavity of health investments
(Grossman, 1972) in producing human capital, the marginal productivity of investments in
children will exceed that of genetically similar adults. In general, the value to adults of own
relative to children's health improvements is an empirical question influenced by the relative
prices and the properties of the not always identical health hazard risk - reduction technologies
applied to adults and to children. Thus a similar health hazard exposure may induce quite
different marginal benefits and marginal costs for adult and for child health — physical,
intellectual, and emotional.
To estimate the relative value adults attach to own health relative to child health we focus
on the intrauterine environment a pregnant woman provides her fetus. The impact of the
intrauterine environment upon child health and development and ultimately upon that child's
adult well-being is a recent concern in economics that has a very large literature in other
disciplines. This noneconomic literature suggests the health of a fetus and its adult well-being
are connected through the causal chain depicted in Figure 1 involving both adult caretaker
behaviors and biological processes. The starting point is maternal health endowments and
behaviors. Maternal endowments and behaviors are linked to the intrauterine environment and
fetal growth, and to contemporaneous maternal health. The intrauterine environment and the
mother's contemporanceous health positively affect birthweight, which is positively linked to the
child's post-natal health (Institute of Medicine, 1990). Post-natal health is a significant positive
21

-------
Figure 1. A Causal Chain
Birthweight
Intrauterine
environment
Mother's
health
Post-natal health
Maternal endowments and behaviors
I
Adult production and consumption
determinant of the child's ultimate adult production and consumption and of its societal
contribution (Becker and Murphy, 1992).
For two reasons, our attention here is limited to the first three levels of Figure 1. First, as
Figure 1 suggests, there is evidence that the effects of lower birthweight are long-term, even
intergenerational (e.g. Hack et al., 1995; Barker, 1998; Currie and Hyson, 1999; Henriksen,
1999; Agee and Crocker, 2002). Lower birthweight children are less healthy than their peers,
and they do less well in school. Increasing birthweight increases adult earnings and schooling
(Behrman and Rosenzweig, 2001). Given that birthweight drives post-natal child health, it is
plausible that an adults' relative valuation of own to post-natal child health reflects the in utero
22

-------
investment the pregnant mother made. If so, an estimate of the own/fetus health valuation will
be predictive of the evolution of her own/child health valuation.
Second, maternal health has positive and negative impacts on post-natal child health. In
the post-natal setting, increased parental consumption or investments in own health impact the
household budget constraint, implying that child health and parent health are substitutes. Better
parent health or more consumption then implies lesser child health. But better parent health
frequently means the parent can provide the child a better quality of care, resulting in enhanced
child physical, intellectual, and emotional health. For the young child the net effect of an
increase in parental consumption or health investment depends on the sign of the sum of these
two factors. This sign is an empirical question dependent upon the mix of phenotypes of
individual household members and upon the determinants of intrahousehold resource allocations
to these members. Grasping the complexities of the mix and the determinants can be a daunting
analytical and empirical task, especially if adult and child behaviors are jointly determined or if
household public goods are present. In contrast, the sign of the connection between maternal
health and fetal child health is unambiguous: it is positive and unidirectional from mother to
2	3
fetus. This positive and unidirectional linkage is widely recognized in the medical literature.
A mother's morbidity and poor health habits result in growth retardation in utero, and,
consequently, a
2	See, however, The Economist (2003) which reviews literature suggesting that the fetus, when stressed, allocates a
greater share of available resources to brain development. No evidence exists that this reallocation fully
compensates for poor maternal health or health practices in all dimensions of post-natal child health.
3	ACC/SCN (2000) provides a thorough review. Other reviews are to be found in Battaglia and Simmons (1979)
and in Kramer (1987). This same literature presents evidence consistent with the marginal products of many post-
natal inputs for lower birthweight children being less than those for normal birthweight children. Thus the
disadvantage of lower birthweight may become progressively greater with age.
23

-------
reduced birthweight for her child.4 The health of the fetus defined in terms of its realized
birthweight for a given gestation period is the result only of its genotype and the
contemporaneous health behaviors and health state of its mother. No other intervening or
mediating influences enter.
Both the biomedical health and the health economics literatures report the results of
extensive research on the determinants of birthweight.5 The economics literature can be
distinguished from the biomedical literature by the emphasis of the former on the endogeneity of
many health inputs, unobserved heterogeneity, and selectivity of women who become pregnant
and who produce live births. This paper extends the previous economics literature in two ways.
First, while continuing to account for input endogeneity and selectivity, it treats the pregnant
mother's health as endogenous. Second, this treatment of the health of the pregnant mother as
endogenous permits derivation of the value this mother attaches to own health relative to the
health of her fetus. We find the contemporaneous, endogenous health of the pregnant mother to
be a significant determinant of the health of her fetus, where fetus health is defined as its live
birthweight. Our representative mother values the health of her fetal child about six times more
than she values her own health.
The next section discusses the implications of the endogeneity of contemporaneous
maternal health for estimates of birthweight production functions. A third section develops a
model of birthweight production which provides restrictions for an econometric specification.
The data used to estimate the birthweight production function are described in a fourth section
4	The medical literature defines low birthweight (LBW) as a weight at birth less than 2500 grams, about 5.5 pounds.
LBW results from premature birth and intrauterine growth retardation. This purportedly universal threshold fails to
consider variations in genetically determined normal birthweights.
5	Economic interest in the issue appears to have first appeared with Rosenzweig and Schultz (1982). Representative
subsequent treatments in the economics literature include Grossman and Joyce (1990), Rosenzweig and Wolpin
(1991), Levinson and Ullman (1998), Warner (1998), Evans and Ringel (1999), Joyce (1999), and Abevaya (2001).
24

-------
and estimation results take up the fifth section. A summary of and caveats about results
conclude.
II. THF FNDOGENEITY OF MATKRYAI HI AI TII
Epidemiological research (e.g., Kirchengast and Hartman, 1998; Shapiro et al., 2000)
unequivocally concludes that a mother's weight gain during her pregnancy has a strong positive
influence on her child's birthweight. This weight gain is a function of her preconception health
endowment and her nutritional and morbidity state while pregnant (Institute of Medicine, 1990).
Her nutritional and morbidity state while pregnant is influenced by her contemporaneous health
input behavior (Osami and Sen, 2003). That is, the pregnant mother's health as measured by her
contemporaneous weight gain is an endogenous input to her child's birthweight. Physicians
recognize this as they recommend behaviors for individual mothers which they think will result
in a weight gain for her conforming to guidelines recommended by the American College of
Obstetrics and Gynecology (1998).
Although development economists (e.g., Strauss, 1986; Devlalikar, 1988) frequently treat
weight as an endogenous variable in studies of labor productivity, none of the economics
literature dealing with birthweight takes account of the possible endogeneity of the pregnant
mother's health as measured by her pregnancy weight gain.6 To see the consequences of this
neglect for acquiring accurate insights into the determinants of birthweight, let the pregnant
mother's health (her pregnancy weight gain), hm, be determined by
hm=hm(z,y,vm)	(1)
6 Nor does it even account for the mother's anthropometry. Warner (1998) is an exception but he treats the mother's
weight gain as exogenous.
25

-------
where vm is the mother's phenotype (her genetic and social inheritances), y represents the health
infrastructure (predetermined or exogenous social, environmental, and economic factors such as
her marital status, employment, income, education, and access to health services), and z is a
vector of endogenous inputs such as prenatal care.
The health technology of the child (its birthweight) h° is described by
hc=hc(hm,z,y,vc),	(2)
where vc is the child's genotype. The relationship between the mother's and the child's health is
made explicit in (2) by including the mother's health, hm, as an argument in the child's health
n
technology. This same relation between parent and child health also holds for a young child,
but it is most vivid for a fetus.
The effect of a marginal improvement of the exogenous health infrastructure, y, and of
the endogenous health inputs, z, on the child's health is:
d^_djf_ dhc dhm
dy dy dhm dy	^
dhc dhc dhc dhm
dz " dz + dhm dz	(4)
The differences between reduced forms that ignore the endogeneity of maternal health in the
child's health technology and a structural system which accounts for endogeneity are the second
terms in expressions (3) and (4). That is, the marginal products of changes in y and in z depend
upon their direct biological effects, the Sh7dy and the dh7dz, and the indirect effects, the
(<9h7<9hm)( <9hmA9y) and the (<9h7<9hm)( <9hm/<9z), representing the mediating influence of the
mother's health. To neglect these indirect effects is to presume that parents ignore the effect of
7 For example, an ill parent can engage in fewer activities with her child. She is able to do less with and for her
child.
26

-------
own-health on child health. If the presumption is incorrect, then, for example, the negative effect
of a decline in exogenous health infrastructure or in positive endogenous health inputs on child
health will be understated. The decline directly reduces maternal health as well child health and
the decline indirectly reduces child health via its effect on maternal health. Similarly, the
presumption will understate the effectiveness of an infrastructure or chosen input increase since
an improvement in the mother's health improves the child's health.
III. MODEL AND ECONOMETRIC PROCEDURES
A. Model
Let a cooperative equilibrium exist between parents such that household preferences can
be described by a single preference function. Consider a two-period model, j = 1,2, where in the
first period the resolution (abort, carry) of the pregnancy is determined. In the second period the
fetus is carried to birth. The mother chooses the quantity of health inputs, Zj, to allocate to own
and to child health in each period. She also chooses own consumption of a composite good, Xj.
Her maximal expected two-period utility is then:
V= maxU(%(h1m(-\z1,yyi\x1) +
8{e{K)v\zi )+ (i- eiKW\4)\
(5)
where
V1 = max U(h£ {h% (z\,y, v™ ),z\,y,vc2), x\)
z2 ,x2
(6)
V2 = maxU(0,xl),
(7)
and
WM o,i]
(8)
27

-------
7 /'• -v • /',-••
(9)
Ij is income in period j, 8 is the mother's fixed discount rate, and 6(hf) is the probability that the
child is born alive. It is assumed that the mother does not die with the birth of the child. The
superscripts on z and x are 1 for a live birth of the child and 2 otherwise. 9 (hf) is a
monotonically increasing continuous function of the child's first-period health. The mother's
health is a pure investment commodity in that she values own health only as an input into her
child's health (Grossman, 1972). While pregnant, the mother makes first-period allocations of
health inputs based on her expectations of the child's survival, the health endowments, vcand vc,
health input prices, pz, social, environmental, and economic factors, y, and income, I, in both
periods. In the second period, the child is born or not, uncertainty is resolved, and the allocation
problem is static. The maximal expected utility for the live birth state is V1{z\ , x\ ), and
V2{xl ) otherwise. If the fetus is not born, its health is normalized to zero in the second period
and the optimum is = 1 . First-order conditions are derived in the Appendix.
Given a live birth, the mother's valuation of her child's health can be derived from the
dual of her second-period allocation problem. Presuming that the expenditure function
associated with this dual is continuous, strictly increasing, and unbounded above in U, as well as
nondecreasing, homogenous of degree one, concave, and differentiable in prices, this problem
can be written as
min pjx\\pjz\	(10)
z\ .x\ X2	22
subject to:
(11)
28

-------
where U\ is the mothers maximal utility in the second period, given a live birth. Efficiency
requires
u\-u\ = 0
p j + X U1l = 0
4. 3du*
p71 + ^
r)K r)K dh™
r)K \ dz\ dtt? dz\
= o.
(12)
(13)
(14)
where X is the Lagrange multiplier.
With a live birth, solution of the problem in (10) and (11) yields parental demand
functions for consumption, x\, and the health inputs, which, when substituted into the
budget constraint, yield the expenditure function for the second period

(15)
By the envelope theorem, the mother's valuation of a change in the health infrastructure is:
de2(-) ,dU\ (•)
&
A-
A
dU\ c)K dU\ C)K C)K

(16)
dh^ dy dh^ dh!£ dy
Substituting for X from expression (14):
de20
dy
r)ti2 c)h'l dh
dy dh% dy
~d% dhfdh?'
1 1 ^ "|j ffl 1 m
dz2 dn-, dz2
(17)
which says that, given a live birth, a mother's marginal valuation of an exogenous improvement
in health infrastructure is her monetized marginal rate of substitution between y and z.
29

-------
A mother's marginal valuation of her child's health is:
3e,(;) dU\ 3%
~^r=x
m dvc2
-p,
r)l% r)ti2 ()h"2
y dz\ dh% dz\ j
dK
(18)
\dv2j
that is, a mother's marginal valuation of an improvement in her child's health is defined as the
tradeoff between family income and the marginal improvement. Similarly, a mother's valuation
of own health is:
de2(-) xdU\dK
dvZ
dv"2
-p,
f)l% dh2 dh2
dz\ dh% dz\
dhc2 dh™
(19)
dtt? dv.
2 J
A mother's value of own health relative to her value of child health is then
den
dK (dh?/dvZ)
d%~dh" (d%/dvZ)'
dvc
(20)
which is the marginal improvement of own health relative to the marginal improvement in child
health.
B. Econometric Procedures
In contrast to the great bulk of the biomedical literature, the economic literature on the
household production of health emphasizes that technical processes together with prices and
income condition a person's or a family's health input choices. Thus simple correlations
between inputs and health outcomes cannot be used to determine causality. Specifically,
30

-------
unbiased estimates of technical family health relationships such as those derived above must be
obtained from a behavioral framework in which health inputs are endogenous. To account for
heterogeneity in the production of mother's and of child's (fetus') health, we propose the
following four equation system: 1) the child's health production to determine survival selection
through the first period; 2) the mother's health in the second period for children who survive the
first period; 3) the surviving child's health in the second period; and 4) the mother's demands for
health inputs in both periods.
Given the mother's utility maximizing quantities of health inputs, a linear representation
of the child's period one health production is
K = (y,Pi,P2)'A)'a2 + mc«3 +	(21)
where jJ + e{ - v,. jJ is the child's observable endowment, and ecx is that facit of the
endowment known to the mother but unobservable to anyone else. The (y,pi, P2)' and (Ii, I2)'
vectors determine the mother's utility-maximizing equilibrium quantities of the z\. Second
period prices and income appear in (21) because the child's first period survival depends on the
mother's expected second period behaviors. For hcx < 0, the mother expects the child will not
survive or a spontaneous abortion occurs. With hcx > 0 the mother carries the child to birth. The
child's first period health is therefore an indicator variable taking a zero if the child is not born
and one if it is born. Failure to account for selection in the resolution of pregnancies will bias
estimates of the consequences of the mother's second period decisions (Grossman and Joyce,
1990). Unacknowledged adverse selection, where women who make relatively small
investments in health care are more likely to give birth, will bias downward the estimated
productivity of health investments. Favorable selection, which refers to women who are more
31

-------
likely to give birth when they make large investments, will, when unacknowledged, impart an
upward bias.
The mother's second period health is
h~ = z2pi + yp2 + iim P3 + eZ,	(22)
and the surviving child's second period health is
/22c = z21^4 + ^5 + 4^6 + M% + <.	(23)
Given that the child survives the first period, the estimating equation of the mother's second
period decision rule for the z2 is
A =	+ Guc„0'72 + m + 02,	(24)
where 2 = «,<).
Our empirical strategy proceeds by obtaining first-stage estimates of the z. and then
applying the fitted values of these quantities to estimate the hc. and the h™. First period health
for the mother is considered to be predetermined. This is reasonable given that this first period
corresponds to the three months immediately after conception. The four equation system is
expressions (21) through (24) applies to our entire sample of pregnant women but expressions
(22) and (23) are observed only for women for whom > 0. Moreover, some health inputs
such as prenatal care visits will be influenced by whether or not h° > 0. These truncations imply
that the error terms among expressions (21), (23), and (24) will be correlated since some of the
32

-------
same health input factors that influence first period child survival also influence second period
child health. We correct for this problem by assuming that the joint distributions of (e[,ec2) and
of (e[,§2) have bivariate normal densities which allows application of Heckman's (1979) two-
stop selection correction procedure. Following Grossman and Joyce (1990), we implement the
procedure by estimating expression (21) as a bivariate probit function, compute the inverse of the
Mills ratio, and then insert this inverse as a regressor into expressions (22), (23), and (24).
IV. DATA DESCRIPTION
Our data come from the 1988 National Maternal and Infant Health Survey (NMIHS), a
data set specifically designed to acquire information on pregnancy outcomes for American
women. After eliminating 13,479 observations with incomplete data, and data referring to
adolescent mothers, mothers more than 35 years old, gestations less than 20 and more than 45
weeks, and birthweights less than 400 and more than 6,000 grams, our full sample of 12,876
o
mother/child observations remained with 10,644 live singleton births. The NMIHS data was
augmented with physician visit costs and with cigarette price per pack for each of 48 states
(Montana and South Dakota refused to participate in the NMIHS).9 About 25 percent of the
sample mothers are homemakers exclusively. Their reservation wages were calculated using the
1983 estimated reservation wage equation of Hofler and Murphy (1994) inflated to 1988 by the
U.S. Consumer Price Index. The wages variable thus represents observed wages for working
mothers and calculated reservation wages for homemakers.
8	Grossman and Joyce (1990) and Werner (1998) employ similar elimination criteria.
9	Physician visit costs are calculated from the 2000 Medicare Physician Fee Schedule as the Nonfacility Fee Amount
deflated to 1988 by the U.S. Bureau of Labor Statistics' Consumer Price Index. Cigarette prices include all
applicable state taxes for 1988, as cited in the Tobacco Institute (1997).
33

-------
Table 1 provides summary statistics and descriptions of the data we employ. A Durbin-
Wu-Hausman test (Greene, 2000) suggested endogeneity for the mother's number of prenatal
care visits while pregnant and the number of weeks she delayed her initial visit after her last
menstrual period. If no care was sought, a delay of 45 weeks was assumed. This delay variable
is thus a negative correlate of the quantity and quality of care and should thus have a negative
coefficient in the probability of survival and birhtweight production function estimates. In
addition to the mother's health, other variables the test proposed as potentially endogenous in
one or more estimated expressions are the order of birth (parity) and smoking during the
pregnancy.
A long list of variables finally treated as exogenous appears in one or more estimated
expressions. Included among them are distance in minutes to a prenatal care facility and the
days not engaged in paid work while pregnant as measures of the time sacrifices the mother
made. The days the mother took off from work are thus treated as a medical necessity. Price
measures include the mother's wages and the cost of a pack of cigarettes. Her anthropometric
and sociodemographic features are represented by the mother's race, age, marital status, number
of household children, number of household smokers, prior smoking habits, number of prior
induced and spontaneous abortions, and prepregnancy body mass index (weight in kilograms
divided by height squared in meters), and the child's gender. Attributes of the mother's
pregnancy include the number of nights she was hospitalized while pregnant, gestation, and
dichotomous variables to indicate whether or not general pregnancy problems/complications
existed and whether efforts had to be made to prevent a premature delivery. The mother's
education, household income, WIC support, Medicaid or insurance coverage, drug use, residence
in a metropolitan area, mental health (CES Total Scale) while pregnant, and whether or not the
34

-------
pregnancy was wanted are included to reflect the mother's health knowledge, stress and
attitudes, and her propensity to seek medical care.
Our objective is to estimate expression (20), the marginal contribution of mother's health
to her child's health. Expression (20) says that this is simply the utility-maximizing marginal
product of a one unit change in mother's health upon child health. Thus, a meaningful
comparison of a change in mother's health (weight gain) relative to an improvement in child
health (birthweight) induced by a common source requires a common measure. This is
accomplished via a linear monotonic transformation of the distribution of mother's weight gain
and child's birthweight such that these distributions have identical means and variances. The
two transformations are:
birthweight in grams	^
! standard deviation of birthweight	^ ^
weight gain in grams
1 s tan dard deviation of weight gain
f	(26)
/—	weight gain in grams	\
+ {h - mean(		—				)).
standard deviation of weight gain
The i subscript refers to a sample child or mother and hc is the sample arithmetic mean for
expression (25).
V. RESULTS
The potential selectivity bias in a straightforward estimate of the birthweight expression
in (23) is quite strong since almost 18 percent of the pregnancies in our sample were aborted. If
the mother's second period utility is positively correlated with the child's birthweight, then
35

-------
abortions, which cause birthweight to be observed only for live births, push the observed
distribution of the mother's utility to the right. Following Heckman (1979), we correct this bias
by estimating expression (21) in linear form for the full sample, using two-stage probit (Lee et
al., 1980) while assuming a bivariate normal form. We then computed a correction factor, the
inverse Mills ratio, for each of the sample women who gave birth. This ratio, which had an
arithmetic mean of 0.3079, and a standard deviation of 0.0963, was then inserted as a regressor
in expression (22), (23), and (24). As a regressor, this selection correction factor can be
interpreted as proportional to the inverse of the probability that a pregnancy is terminated.
Because epidemiological evidence suggests that almost none of the mother's weight gain
occurs in the first trimester (Kramer et al., 1992), mother's first period health is treated as
exogenous in the birth probability equation and is measured by her anthropometric and
sociodemographic endowments. A Durbin-Wu-Hausman test (Greene, 2000) suggested
endogeneity of smoking, parity, the number of prenatal care visits, and delay of the first such
visit in the birth probability equation. Reduced form demand functions for these variables were
estimated by OLS for the full sample of 12,876 mother/child observations. All of the previously
mentioned exogenous variables were treated as regressors except for all of the variables
describing the mother's pregnancy attributes, prior abortions, drug use, and mother's body-mass-
index (BMI) immediately before her current pregnancy. The fitted values for these four
endogenous variables were then entered as regressors in the birth probability equation along with
mother's age, mother's wantedness attitude, and the immediately aforementioned exogenous
variables that were excepted from the demand estimates. Fitted versions of the number of
prenatal care visits, delay of the first such visit, and parity were statistically significant positive
correlates of birth probability and smoking was a statistically significant negative correlate. Of
36

-------
the exogenous variables entered in the birth probability equation, only the coefficients for
mother's age and for her BMI were significant at less than 5 percent. Both exhibited negative
signs.
Table 2 presents OLS estimates for the endogenous regressors in period 2. Since birth
selection for the current pregnancy cannot affect parity and is presumed not to affect period 2
smoking behavior, the demand expressions for these two variables do not include the Mills
correction and thus apply to both periods 1 and 2. Among the more notable of the results for the
smoking expression is the positive impact of prior smoking or smoking while pregnant and the
statistical insignificance of the impact of cigarette price upon smoking while pregnant. These
results are consistent with an addiction to smoking.10 The elasticity of the mother's smoking
while pregnant with respect to prior smoking is 0.967. Her education has an elasticity of -.379,
implying that she chooses to consume about 8 fewer daily cigarettes while she is pregnant when
she has an additional year of education.
Consistent with Rosenzweig and Schultz (1983), more educated women who earn higher
wages experience fewer pregnancies and have them later in life. However, the Table 2 finding
that women living in urban environments have higher fertility is contrary to Rosenzweig and
Schultz (1983). Black mothers and mothers who are depressed get pregnant less frequently, all
else equal.
10 Another source of addiction is alcohol intake. Rosenzweig and Wolpin (1991) and Warner (1995) find that
alcohol use is not a significant influence upon birth weight. Our preliminary birthweight regressions confirmed this
result, perhaps because more than 90 percent of the pregnant mothers in our full sample drank less than one
alcoholic beverage per month. Some 83 percent of the full sample were nondrinkers while pregnant and only 1.1
percent drank more than one drink daily. Our NMIHS full sample is likely not rich enough to capture any
birthweight effect from alcohol.
37

-------
The estimated demand equations for the number of prenatal care visits (xvisit) and the
delay in the initial visit (visit) exhibit favorable selection, contrary to Joyce (2001). Most other
results are consistent with those obtained by Grossman and Joyce (1990). However, the Table 2
result that married women who are pregnant have lesser delays for their initial prenatal care visit
contradicts Grossman and Joyce (1990). The Table 2 finding that an unwantedness attitude and a
depressed state of mind reduce and delay prenatal care suggest that convincing pregnant women
to seek medical help is more than a matter of simply manipulating economic and easily observed
sociodemographic factors.
Table 3 presents 2SLS estimates for the mother's period 2 health (mother's weight gain,
transformed). Only visit-hat is endogenous. It thus represents fitted values from the visits
equation in Table 2. The Mills correction for selection is statistically insignificant. Most Table 3
explanatory variables, including visit-hat, are also insignificant but weight gain is positively
responsive to black mothers and to women who were big before becoming pregnant, and
negative with respect to mother's age. The positive and statistically significant result for wages
is consistent with numerous biomedical and economic results (e.g., Grossman, 1972) indicating a
positive association between health and wealth. The positive signs attached to the statistically
significant coefficients for depression (ces-total) and drug use defy ready explanation.
Table 4 presents birthweight equations estimated by TSLS. Each column of Table 4
represents a combination of the endogeneity or the exogeneity of weight gain for mothers who
gave live births, h%, and a correction or lack of such for selection via the termination of a
pregnancy. When a selection correction is made as in Columns (1) and (2), because the inverse
Mills ratio is a non-linear function of the variables included in the first-stage probit model, the
38

-------
second-stage model is identified even if the regressors in the first and second stage models are
identical. Nevertheless, as an extra identification precaution, two exclusion restrictions were
imposed. First, there is at least one covariate in the first-stage not in the second-stage; second,
there is at least one variable in the instruments for each endogenous variable that does not appear
in the second-stage. Basically, enabling variables such as income, insurance, etc., were assumed
to affect birthweight only indirectly through the mother's weight gain.
A version of Sargan's (1976) test, known as the Pagan-Hall (1983) test, for
misspecification in models with instrumental variables failed to reject at the 1 percent level the
hypothesis of no misspecification for our complete set of instruments.11 However, a Breusch-
Pagan (1979) test revealed heteroskedasticity between the error terms in the mother's weight
gain and the child's birthweight equations. Consequently, three-stage least squares estimates
were obtained. These 3SLS estimates caused the Mills selection correction variable to become
insignificant in the birthweight equation and reduced the magnitude of the coefficient for the
number of prenatal care visits without altering its statistical significance. Coefficient estimates
and levels of statistical significance for the other covariates in Table 4, including especially the
endogenous and the exogenous versions of the variables for mother's health, were essentially
12
unchanged. Given the focus of the paper upon the mother's value of own relative to child
health, the following discussion centers upon Table 4 and its value implications.
All columns of Table 4 indicate that mother's pregnancy weight-gain is a positive and
statistically significant influence upon the child's health. More importantly, by treating the
11	See Godfrey (1988, pp. 174-176) for a succinct exposition of these tests.
12	For example, the 2SLS estimate for - hat with selection is .160 with a standard error of .014; for the 3SLS
estimate it is . 170 with a standard error of .019.
39

-------
mother's weight gain as endogenous, one increases this positive influence by a factor of three.
Failure to take into account the indirect effect of the mother's health upon the child's health
greatly underestimates the importance of the mother's health behaviors to the child's health.
Apart from mother's health, there are only very minor differences across columns in the
coefficients for the Table 4 regressors. However, there are substantial differences for the
selection and no selection results for the case of mother's health endogeneity as well as that for
exogeneity. The effect of the number of prenatal care visits is two times higher with than
without selection, as is the effect of parity. An accounting of selection has little effect upon the
birthweight influence of drugs, smoking, gestation, or gender.
Expression (20) implies that the coefficient on h% - hat in Table 4 measures the mother's
value of own relative to her child's health. For the case of no selection, - hat has a value of
.170, implying that the representative mother values her child's health about six times more than
her own health. Thus four conclusions emerge from Table 4: 1) pregnant mothers value child
health more than own health; 2) mother's health and child health are complements; 3) the
indirect effect of maternal behaviors increases the estimated contribution mother's health makes
to child health; and 4) selection due to pregnancy termination does not affect the estimated
contribution of maternal health to child health.
The contribution of maternal health to child health and thus the mother's value of own to
child health was also estimated for subsamples of the NMIHS women who gave live births.
Table 5 gives the results. The most striking difference emerges for nulliparous women (this
pregnancy is their first child) and women who already have at least one child. If mother's health
40

-------
is endogenous, nulliparous women value their child's health relative to their own health more
than twice as much as do other women who have had children. This result is consistent with the
tradeoff between the quantity and the "quality" of children emphasized by Becker and Lewis
(1973). Nonsmoking mothers appear to value their children more highly than do smokers. More
income, as reflected in the income, medicaid/no medicaid, and married/not married subsamples
seems to increase the relative value of child health. Education also increases this relative value,
perhaps because of the opportunity costs of the time a mother expects she will subsequently have
to devote to a born child.
VI. SUMMARY AND CONCLUSIONS
We have presented a framework suitable for estimating the value an adult attaches to own
health relative to child health when health inputs are endogenous and adult health is an input to
child health. Heretofore, no research has specifically examined the impact of adult behaviors on
own health and thence upon child health. Though our focus is upon pregnant woman and the
child they carry, the framework could, likely at considerable cost in analytical and empirical
complexity, be extended to the care adults provide post-natal children. A parent's discretionary
behaviors affect her contemporaneous health and this health impacts what she can do with and
for her child. What she does with and for her child influences its health. By aiding her own
health, the mother helps her child's health.
When the maternal health input to child health is treated as endogenous our empirical
results indicate that, on average, pregnant mothers value the prospective health of their as yet
unborn children about six times more than they value own health. Treatment of the maternal
health input as exogenous will reduce the estimated impact of this input upon child health
41

-------
relative to the impact when it is treated as endogenous. Consequently, for a given observed child
health improvement, part of the contribution of maternal health to this improvement will be
attributed to other health inputs, thus reducing the estimated value of maternal health relative to
child health.
Our empirical results also suggest that the mother's relative valuation of own and of child
health is sensitive to her personal and family characteristics and behaviors such as number of
siblings for the child, family income, and maternal consumption patterns (e.g., smoking).
The result that pregnant mothers value own health considerably less than they value child
health promotes skepticism about the one-to-one transfer of adult health benefits measures to
children. Whatever the average relative valuation employed, they also promote caution about
use of a one-size-fits-all constant for these transfers.
42

-------
References
Abrevaya, J. (2001), "The Effects of Demographics and Maternal Behavior on the Distribution
of Birth Outcomes," Empirical Economics 26:247-257.
ACC/SCN (Administrative Committee on Coordination/Sub-Committee on Nutrition) (2000), J.
Podja and L. Kelley, eds., Low Birth weight. Nutrition Paper #18, Geneva, Switzerland:
World Health Organization.
Agee, M.D., and T.D. Crocker (2002), "Parents' Discount Rate and the Intergenerational
Transmission of Cognitive Skills," Economica 69:143-154.
American College of Obstetrics and Gynecology (1998), Guidelines for Perinatal Care.
Washington, DC: American Academy of Pediatrics.
Barker, D.J.P. (1998), Mothers. Babies and Diseases in Later Life. London, U.K.: Churchill
Livingston.
Battaglia, F. C., and M.A. Simmons (1979), "The Low-Birth-Weight Infant," in F. Falkner and
J.M. Tanner, eds. Human Growth. Vol. 1, New York, NY: Plemum Press.
Becker, G.S., and H.G. Lewis (1973), "On the Interaction between the Quantity and Quality of
Children," Journal of Political Economy 8:S279-S288.
Becker, G.S., and K.M. Murphy (1992), "The Division of Labor, Coordination Costs, and
Knowledge." Quarterly Journal of Economics 107:1137-1160.
Behrman, J.R., and M.R. Rosenzweig (2001), The Returns to Increasing Body Weight. Working
paper, Department of Economics, University of Pennsylvania.
Breusch, T.S., and A.R. Pagan (1979), "A Simple Test for Heterosckedasticity and Random
Coefficient Variation," Econometrica 47:1287-1294.
Currie, J., and R. Hyson (1999), "Is the Impact of Health Shocks Cushioned by Socioeconomic
Status?" The American Economic Review 89:245-250.
Deolalikar, A.B. (1988), "Nutrition and Labor Productivity in Agriculture, "The Review of
Economics and Statistics 70:406-413.
Evans, W.N., and J.S. Ringel (1999), "Can Higher Cigarette Taxes Improve Birth Outcomes?"
Journal of Public Economics 72:135-154.
Godfrey, L.G. (1988), Misspecification Tests in Econometrics: The Lagrange Multiplier
Principal and Other Approaches. Cambridge, U.K.: Cambridge University Press.
Greene, W.H. (2000) Econometric Analysis. 4th ed., Princeton, NJ: Prentice Hall.
43

-------
Grossman, M. (1972), "On the Concept of Health Capital and the Demand for Health," Journal
of Political Economy 98:983-1007.
Grossman, M., and T.J. Joyce (1990), "Unobservables, Pregnancy Resolutions, and Birth Weight
Production Functions in New York City," Journal of Political Economy 98:983-1007.
Hack, M., N.K. Klein, and G. Taylor (1995), "Long-term Developmental Outcomes of Low
Birth Weight Infants," The Future of Children 5:19-34.
Heckman, J.J. (1979), "Sample Selection Bias as a Specification Error," Econometrica 47:153-
161.
Henriksen, T. (1999), "Fetal Nutrition, Fetal Growth Restriction and Health Later in Life," Acta
Paediatrica 429s :4-8.
Hofler, R.A., and K.J. Murphy (1994), "Estimating Reservation Wages of Employed Workers
Using a Stochastic Frontier," Southern Economic Journal 60:961-976.
Institute of Medicine (1990), Subcommittee on Nutritional Status and Weight Gain During
Pregnancy, in Nutrition During Pregnancy. Washington, DC: National Academy of
Sciences.
Joyce, T. (1999), "Impact of Augmented Prenatal Care on Birth Outcomes of Medicaid
Recipients in New York City," Journal of Health Economics 18:31-67.
Joyce, T. (2001), "Self-Selection, Prenatal Care, and Birthweight among Black, Whites, and
Hispanics in New York City," Journal of Human Resources 29:762-794.
Kirchengast, S., and B. Hartman (1998), "Maternal Prepregnancy Weight Status and Pregnancy
Weight Gain as Major Determinants for Newborn Weight and Size," Annals of Human
Biology 25:17-28.
Kramer, M. (1987), "Determinants of Low Birth Weight: Methodological Assessment and Meta-
Analysis," Bulletin of the World Health Organization 65:663-737.
Kramer, M., F. McLean, E. Eason, and R. Usher (1992), "Maternal Nutrition and Spontaneous
Preterm Birth," American Journal of Epidemiology 136:574-583.
Kuchler, F., and E. Golan (1999), Assigning Values to Life: Comparing Methods for Valuing
Health. Agriculture Economics Report No. 784, Food and Rural Economics Division,
Economic Research Service, U.S. Department of Agriculture, Washington, DC.
Lee, L., G.S. Maddala, and P.R. Trost (1980), "Asymptotic Covariance Matrices of Two-Stage
Probit and Two-Stage Tobit Methods for Simultaneous Equations with Selectivity,"
Econometrica 48:491-503.
Levinson, A., and F. Ullman (1998), "Medicaid Managed Care and Infant Health," Journal of
Health Economics 17:351-368.
44

-------
Osami, S., and A. Sen (2003), "The Hidden Penalties of Gender Inequality: Fetal Origins of Ill-
Health," Economics and Human Biology 1:105-121.
Pagan, A.R., and A.D. Hall (1983), "Diagnostic Tests as Residual Analysis," Econometric
Reviews 2:159-218.
Rosenzweig, M.R., and T.P. Schultz (1982), "The Behavior of Mothers as Inputs to Child
Health: The Determinants of Birth Weights, Gestation, and Rate of Fetal Growth," in
V.R. Fuchs, ed., in Economic Aspects of Health. Chicago, IL: University of Chicago
Press.
Rosenzweig, M.R., and T.P. Schultz (1983), "Estimating a Household Production Function:
Heterogeneity, the Demand for Health Inputs, and Their Effects on Birth Weight,"
Journal of Political Economy 9:723-746.
Rosenzweig, M.R., and K.I. Wolpin (1991), "Inequality at Birth: The Scope for Policy
Intervention," Journal of Econometrics 50:205-228.
Sargan, J.D. (1976), Testing for Misspecification after Estimating Using Instrumental Variables.
Unpublished paper, London, U.K.: London School of Economics.
Shapiro, C., V.G. Sutija, and J. Bush (2000), "Effect of Maternal Weight Gain on Infant Birth
Weight," Journal of Perinatal Medicine 28:428-431.
Strauss, J. (1986), "Does Better Nutrition Raise Farm Productivity?" Journal of Political
Economy 94:297-320.
The Economist (June 14, 2003), "The Child is Father to the Patient," :78-79.
Tobacco Institute (1997), "Historical Compilation," The Tax Burden on Tobacco, vol. 32.
U.S. Environmental Protection Agency (2000), Guidelines for Preparing Economic Analyses.
EPA 240-R-00-003, Washington, DC.
Warner, G.L. (1995), "Prenatal Care Demand and Birthweight Production of Black Mothers,"
The American Economic Review 85:132-137.
Warner, G.L. (1998), "Birthweight Productivity of Prenatal Care," Southern Economic Journal
65:42-63.
45

-------
Table 1. Summary statistics, n=12,876
Variable
Mean
Std. Dev.
Description of variables
abortions
.4344517
.9231301
Number of previous abortions, induced and spontaneous
alive
.8265219
.3786747
Dichotomous: 1 if child was born alive
attitude
.4558356
.498065
Dichotomous: 1 if pregnancy was not wanted
bweight
2579.276
1111.054
Baby's birth weight in grams
ces_total
13.99845
12.10674
CES Depression scale for pregnant mother
children
1.180147
1.384186
Number of children in the household
cigprice
130.9913
9.75674
Price of one packet of cigarettes, including all taxes in cents (1988 US
dollars)
distance
20.40993
16.35411
Distance in minutes to prenatal care provider
drugs
.0755332
.2642599
Dichotomous: 1 if mother used drugs in the 12 months before delivery
gender
.4740597
.499346
Dichotomous: Baby's gender 1 if female
gestation
35.97053
5.837431
Length of gestation in weeks
health_child
2.321469
1
Child's health index, transformation of child's birth weight
health_mother
2.321469
1
Mother's health index, transformation of mother's weight gain
income
25672.04
21067.1
Total annual household income, whole dollars (1988 US dollars)
insurance
.6350523
.4814341
Dichotomous: 1 if mother had health insurance at delivery time
mage
26.57325
4.242554
Mother's age in years
marital
.6201629
.485365
Dichotomous: 1 if married
mbmibefore
28.04287
5.512001
Mother's Body Mass Index before pregnancy
medicaid
.3010469
.4587308
Dichotomous: 1 if covered by Medicaid
meduc
12.59558
2.318349
Mother's education in years
metro
.777027
.4162565
Dichotomous: 1 if family lives in metropolitan area
nights
7.8185
13.80952
Number of nights hospitalized during pregnancy (excluding delivery)
parity
.8675456
1.428883
Number of previous pregnancies
premature
.3535259
.4780827
Dichotomous: 1 if action was taken to prevent premature delivery
prenatalcost
361.44
27.406
Cost of prenatal care visit, whole dollars (1988 US dollars)
46

-------
Table 1. Summary statistics (cont.)
priorsmoke
5.077975
9.224811
Number of cigarettes smoked by the mother prior to pregnancy, per day
problems
.8952314
.3062669
Dichotomous: 1 if complications with pregnancy
race
.528425
.499921
Dichotomous: 1 if mother is black
smokers
.480817
.8589116
Number of smokers in the household
smoking
3.065995
6.889637
Number of cigarettes smoked by the mother during pregnancy, per day
totaldays
66.40775
110.6519
Total days the mother did not work
visit
10.41171
8.45774
Weeks since pregnancy started before first prenatal care visit
wages
37043.31
23570.97
Mother's wages annually, whole dollars (1988 US dollars)
wgain
9586.068
9881.782
Mother's weight gain during pregnancy, in grams, after birth
wic
.3422257
.4744731
Dichotomous: 1 if WIC food aid provided during pregnancy
xvisits
11.23893
6.130369
Number of prenatal care visits
Note: The mother's and the child's health index transformation is explained in the text. The WAGES variable also includes
reservation wages for homemakers, as explained in the text.
47

-------
Table 2. Demand Estimates
visit	xvisit	smoking	parity

Coefficient
t
Coefficient
t
Coefficient
t
Coefficient
t
distance
-.085
-18.12
.027
7.64
-.005
-2.14
-.003
-3.98
attitude
3.202
14.27
-1.381
-8.09
.189
2.37
.054
2.27
children
.535
8.38
-.273
-5.64
.231
8.00
.368
42.46
wic
-2.185
-9.95
1.440
8.63
-.121
-1.30
-.054
-1.94
insurance
-1.482
-7.01
.653
4.06
-.190
-2.09
-.318
-11.63
marital
-1.386
-6.92
.872
5.73
-.309
-3.23
-.010
-0.35
mage
-.123
-5.78
.046
2.84
.034
3.75
.034
11.71
race
-2.978
-9.20
2.463
10.00
-.068
-0.78
-.288
-11.14
meduc
-.227
-5.87
.208
7.07
-.092
-5.01
-.073
-13.19
wages
-,103e-04
-2.70
,688e-07
2.37
,476e-07
2.83
-,336e-07
-6.65
income
,266e-04
-5.36
,128e-04
3.39
-,930e-07
-3.96
,299e-08
0.42
medicaid
.929
4.01
-.460
-2.61
.084
0.81
.120
3.84
total days
,225e-04
0.03
.002
2.96
-.001
-1.98
,162e-07
0.02
metro
-.485
-2.46
.247
1.65
.063
0.68
.142
5.12
cigprice
.011
1.28
-.014
-2.17
-.002
-0.49
-,047e-03
-0.04
cestotal
-.073
-4.70
.060
5.06
.006
1.97
-.005
-5.09
prenatal cost
-.004
-1.33
.005
1.95
.001
0.50
.002
4.36
priorsmoke
.045
5.23
-.007
-1.13
.584
142.20
.012
10.12
mills
22.689
8.37
-17.234
-8.36
—
—
—
—
Constant
15.350
9.80
9.028
7.58
.349
0.50
.153
0.73
Observations	10,644	10,644	12,876	12,876
F-statistic	102.42	48.30	1293.63	247.29
R*	.1548	.0795	.6443	.2572
Note: All estimates were obtained by ordinary-least-squares.
48

-------
Table 3. Mother's Period 2 Health (Weight Gain)
K
Coefficient
t
distance
-.001
-0.51
attitude
-.003
-0.08
children
.008
0.97
wic
-.074
-1.50
insurance
-.058
-1.50
marital
.002
0.05
mage
-.014
-5.58
race
.010
4.29
meduc
.005
0.84
wages
,126e-07
3.14
income
,123e-07
1.78
medicaid
-.003
-0.11
total days
,146e-03
-1.69
metro
-.032
-1.32
cigprice
-,458e-03
-0.48
cestotal
.004
4.35
prenatal cost
.001
1.67
priorsmoke
-,848e-04
-0.08
mbmibefore
.074
45.11
gestation
.003
2.08
drugs
.111
3.17
visit-hat
-.008
-0.56
mills
.161
0.76
Constant
.208
0.63
Observations
10,644

F-statistic
99.45

R2
.1708

Note: Estimated by two-stage-least-squares. Variables denoted with a "-hat" are endogenous.
49

-------
Table 4. Child's Period 2 Health (Birthweight)
(1) Endogenous/ (2) Exogenous/	(3) Endogenous/	(4) Exogenous/No
Selection	Selection	No Selection	Selection
K
Coefficient
t
Coefficient
t
Coefficient
t
Coefficient
t
visit-hat
.018
1.49
.019
1.74
.008
1.70
.007
1.51
xvisits-hat
.051
5.07
.055
5.47
.029
3.20
.028
3.09
parity-hat
.039
3.96
.040
4.05
.020
2.18
.016
1.77
smoking-hat
-.012
-9.98
-.012
-10.29
-.010
-8.95
-.010
-8.95
drugs
-.071
-3.28
-.066
-3.03
-.068
-3.13
-.062
-2.83
h™ ~hat
.160
11.51
—
—
.170
12.32
—
—
K
—
—
.055
9.76
—
—
.057
10.11
mills
.346
4.88
.423
6.00
—
—
—
—
gestation
.134
144.49
.136
147.20
.134
144.20
.136
147.00
gender
-.104
-9.45
-.104
-9.45
-.106
-9.60
-.106
-9.63
Constant
-3.639
-20.34
-3.530
-19.75
-3.185
-20.82
-2.956
-19.55
Observations
10,644

10,644

10,644

10,644

F-statistic
2546.65

2533.73

2855.86

2836.60

R2
.6831

.6820

.6824

.6809

Notes: All estimates were obtained by two-stage-least-squares. Variables denoted with a "-hat" are endogenous. The columns
denote whether or not h™ was treated as endogenous, and whether or not a selection correction was made.
50

-------
Table 5. Mother's Value of Own Relative to Child Health.
With endogeneity	Without endogeneity
Subsample
Observations
Coefficient
z
R2
Coefficient
z
R2
Whites
5234
.1546
5.19
.5189
.0552
6.25
.5558
Blacks
5410
.1789
6.38
.7000
.0527
7.14
.7165
No other children
3967
.1005
3.08
.6622
.0524
5.72
.6777
One or more children
6677
.2254
8.52
.6261
.0601
8.16
.6524
Pregnancy wanted
5628
.1621
5.42
.6562
.0531
6.78
.6688
Pregnancy not wanted
5016
.1810
6.45
.6211
.0604
6.82
.6319
Not insured
3922
.1717
5.32
.6125
.0658
5.97
.6200
Insured
6722
.1700
6.60
.6759
.0489
7.17
.6936
Unmarried
4158
.1902
6.40
.5650
.0712
6.88
.5925
Married
6486
.1561
5.05
.6842
.0458
6.52
.6971
Not on Medicaid
7344
.1589
5.95
.6863
.0535
8.05
.6940
On Medicaid
3300
.2065
6.01
.4565
.0663
5.83
.4897
Live outside
metropolitan area
2312
.1955
3.29
.6234
.0813
6.30
.6279
Live inside
metropolitan area
8332
.1673
7.83
.6038
.0509
7.84
.6134
Nonsmoker
7808
.1440
5.99
.6703
.0475
7.20
.6823
Smoker
2836
.2381
5.26
.4572
.0811
6.80
.4632
Education<=12 years
6458
.1864
6.85
.6240
.0614
7.62
.6388
Education >12 years
4186
.1391
4.58
.7044
.0472
5.37
.7132
Homemaker
4014
.1833
5.97
.5411
.0591
5.84
.5688
Employed
6630
.1718
6.41
.6939
.0563
8.13
.7060
Income< 10000
3250
.1960
4.61
.4428
.0724
5.19
.4801
Income <=50000
6925
.1640
6.68
.6610
.0563
8.07
.6729
Income >50000
1059
.1497
3.74
.6747
.0196
1.21
.6852
The results are based on two-stage least squares without selection.
51

-------
Appendix
52

-------
Appendix
The problem of the parent is to maximize her expected "lifetime" utility. Her maximal
expected "lifetime" utility in the first period is:
V= maxE{U(%[h?(.\z1,yyilx1) +	(A.l)
Zl,*l
si8wv\4,4)H i-
V'= ma*u(h;(h?(4,yX),4,y,vl),xl)	(A.2)
zl ,x2
V2 = maxt/(0,jc22)	(A.3)
x22
WM0,1]	(A.4)
I j = PxXj + PZjPj	(A.5)
where Xj is period j parental consumption, Ij is period j income, 8 is the parent's discount rate and
9 is the probability of a "normal" pregnancy. We assume that each period's income is
predetermined (labor supply decisions for the mother are exogenously determined), the parent's
discount rate 8 is fixed and the probability of the child born alive 9 is a monotonically increasing
continuous function of the child's health in the first period. Superscripts on x and z in equation
(A.5) are omitted for notational simplicity. The mother chooses X21 and z2' at the beginning of
period one.
Substituting the budget constraint (A.5) into equations (A.l), (A.2) and (A.3), the system
becomes:
V = maxE{U(h;(h?(Zl,y,vm),zj,vf \Iy- pxzx) +
(A. 6)
)+ (i-emK^y,<)^yy,\W\zl)]}
V = max U(hc2(h™(z\,y,v™),z\yy2),I2 - p2z\)	(A.7)
z2
53

-------
V2 = max£7(0,12 - p2z22)	(A.8)
z2
The optimal 7.2 is the solution to equation (A.6). Efficiency requires that:
rl3Udh£ dU dhc2 dh2 dU
V ' <)K dz\ + <)K dh™ dz\ ~ Pl dcx	(A'9)
The first term in equation (A.9) is direct effect on utility of a change in z on the
child's health, as would be predicted if only the "reduced-form" effect was considered,
and the second period, given that the child does not survive, the mother does not consume
any health inputs for the child, therefore:
V2:x2 = —	(A. 10)
o
Equation (A.8) states the familiar result of consumer theory that in order to
maximize utility, the parent must allocate resources so as to make the ratios of marginal
utilities equal to the ratios of prices.
Efficiency for equation (A.6) requires that:
dEU r)h; dEU r)h; dh" dEU dCl e r)d . r)d 2	,
()h'\ dzl + r)h; dhT dzl ~ dc, dz,Pl+ [dz,V ~ dZlV ]~°	(A11)
where
dd dd dh: dtt Sh™
(A'12)
54

-------
Equation (A. 11) is the marginal change in the probability of survival in the second period
from a change in z. The solution of the problem comes from solving the first-order
condition (A.8), and then (A. 10).
55

-------
Valuing Fetal and Infant Health:
What Can Be Learned from Empirical Health Economics Research?
Don Kenkel, PhD
Department of Policy Analysis & Management
Cornell University
Currently, Academic Visitor
Department of Economics & Related Studies
University of York
Heslington York
United Kingdom
dsklO@cornell.edu
dkl6@york.ac.uk
October 2003
Preliminary draft prepared for presentation at "Valuing Environmental Health Risk Reductions to
Children," October 20 - 21, 2003, Washington DC. Comments welcome.
56

-------
I. Introduction
Despite many encouraging trends in environmental quality, serious environmental health
threats to fetal, infant, and child health remain. For example, some research suggests that
particulate matter air pollutants may be associated with higher infant mortality.1 In 2001
approximately 25 percent of children lived in counties that exceeded the annual standard for
particulate matter (US EPA 2003). This suggests a similarly large fraction of all pregnant
women may be exposed to unhealthy levels of particulates The importance of fetal and infant
health is underscored by the inclusion of data on birth defects in California as a Special Feature
in the most recent EPA (2003) report on America's Children and the Environment. As the EPA
notes, "birth defects are leading cause of infant death in the first year of life, accounting for about
20 percent of infant deaths in 1999." Although some birth defects are inherited, environmental
and public health policies may be able to reduce nongenetic risk factors for birth defects, and
improve fetal and infant health more generally.
Benefit-cost analysis of policies to improve fetal, infant and child health requires
valuation of those health improvements. A number of studies extend market and non-market
approaches to estimate willingness to pay for child health. Because children are not in the labor
market and do not make independent consumption decisions, the studies focus on parents'
decisions that affect the health and safety of their children. Analysis of parents' child safety seat
use, automobile purchases, and bicycle helmet purchases provides estimates of willingness to pay
1 In addition to the references in US EPA (2003), Chay and Greenstone (1999, 2001)
analyze data on infant mortality, birthweight, and air quality improvements in the early
1970s and the early 1980s. Their findings suggest a strong relationship between air
quality as measured by total suspended particulates and infant mortality rates, and a
somewhat weaker relationship between air quality and birthweight.
57

-------
for child mortality risks , summarized as the value of a statistical life for a child (Carlin and
Sandy 1991; Mount etal. 2000; and Jenkins, Owens and Wiggens 2001). Other child health
effects valued include: child lead exposure (Agee and Crocker 1996); colds (Liu etal. 2000);
risks of non-melanoma skin cancer (Dickie and Gerking 2001); lifetime cancer risks (Maguire,
Owens and Simon 2001); and child health effects related to secondhand smoke exposure (Agee
and Crocker 2001). However, there seem to be few if any existing estimates specific to the
value of fetal and infant health.
The goal of this paper is to examine the implications of empirical health economics
research for the valuation of fetal and infant health. Section II sets the stage by reviewing
illustrative empirical evidence on the various ways parents invest in prenatal health, including
market purchases such as medical care, and lifestyle changes such as smoking cessation. Section
III presents a simple version of the standard household health production model, to serve as the
theoretical framework for the valuation expressions and the empirical research to be reviewed.
Section IV reviews health economics research that estimates infant health production functions.
Combining the estimates of the marginal product of prenatal care with estimates of the full price
paid yields estimates of parental marginal willingness to pay for infant health. Section V reviews
studies of maternal demand for cigarettes and alcohol during the pre-natal period, and discuss the
implications for fetal and infant health valuation. Section VI discusses health economics studies
2
In the recent review by Neumann and Greenword (2002), all of the studies of effects
associated with prenatal exposure use the cost-of-illness approach. The review includes
estimates of willingness to pay to reduce infants' mortality risks, based on Dickie and
Nestor's (1999) analysis of the results of Joyce, Grossman and Goldman (1989).
58

-------
that estimate the impacts of public policies on fetal and infant health, and explores whether these
can be used to shed light on the health valuation question.
At the outset, limitations of the scope of this paper should be noted. The emphasis of the
paper is on lessons from health economics, so the environmental economics research literature is
not reviewed in depth. The valuation approach is to infer parents' willingness to pay for fetal
and infant health based on their preferences as revealed in the markets for medical care,
cigarettes, and so on. The paper does not review studies from three other approaches that shed
light on health valuation: the cost-of-illness approach; the contingent valuation or stated
preference approach; or the quality-adjusted life year approach used in cost-effectiveness
analysis. To date, most evaluation studies relevant to fetal and infant health follow the cost-of-
illness approach; for summaries of these studies see Neumann and Greenwood (2002). Cost-of-
illness estimates provide a lower bound to willingness to pay (Berger et al. 1987, Kenkel 1994),
so these estimates are a way to check the plausibility of willingness to pay estimates from other
approaches. Two contingent valuation studies estimate willingness to pay related to infertility
risks (Neumann and Johanneson 1994, Smith and Van Houten 1998), but the implications for the
value of fetal and infant health are not clear. Finally, in principle it should be possible to follow
the common approach in cost-effectiveness analysis and estimate the number of quality-adjusted
life years lost from fetal and infant poor health and death. For example, the Harvard Catalogue
of Preference Scores includes weights to calculate the quality-adjusted life years for children with
a range of neurologic disabilities. However, in all of the cases the preference scores were
measured based on author or clinical judgement, and so may not reflect either parental or societal
preferences over these health states.
59

-------
II. Parental Investments in Prenatal Health: An Overview
Table 1 provides an overview of maternal investments in prenatal health. The data are
from the National Maternal and Infant Health Survey (NIMHS) 1988, conducted by the National
Center for Health Statistics. The 1988 NMIHS consists of three independent national files of live
births, fetal deaths and infant deaths. The full sample consists of 18,594 mothers who had a live
birth, fetal death or infant death in 1988. Of these 18,594 mothers, 9,953 women had live births,
"3
3,309 had late fetal deaths and 5,332 had infant deaths. Table 1 presents the patterns of prenatal
investments for the full sample and for each of the sub-samples.
As can be seen in Table 1, during the prenatal period women invest both money and time
to improve fetal health. Virtually all (98 percent) of pregnant women in the U.S. receive prenatal
medical care, and on average make almost 13 prenatal visits. Additional data from the NMIHS
(not reported in Table 1) indicate that about a third of the women report paying for the prenatal
care out of their own income. In addition, some of those whose care was covered by private
insurance or Medicaid still incurred out-of-pocked costs due to copayments or coinsurance,
although data on this was not collected in the NMIHS. Women also incurred time costs to travel
to and receive prenatal care; the average travel time to prenatal care for NMIHS respondents was
about 21 minutes.
3 In 1988, there were 3,898,922 live births to women between 15 and 49 years of age, 15,259
fetal deaths of 28 weeks or more gestation, and 38,917 infant deaths to United States residents.
The overall probability of the 1988 NMIHS selection was about 1 of every 354 live births, 1 of
every 4 fetal deaths and 1 of every 6 infant deaths. The overall response rate for the national file
of 18,594 mothers is 71%; it is 74% for live birth mothers, 69% for fetal death mothers, and 65%
for infant death mothers.
60

-------
In the full sample, 38 percent of pregnant women also attend prenatal childbirth class, but
fewer women in the fetal death and infant death samples attend such classes. About 80 percent
of pregnant women take multivitamins and/or minerals at least three days a week after they found
out they were pregnant, up from about 25 percent of women who took vitamins before they found
out they were pregnant.
Women also commonly make lifestyle changes after they find out they are pregnant. In
the 1988 NIMHS, pregnancy is associated with a drop in the prevalence of smoking from 30
percent to 22 percent. Even those women who continue to smoke while pregnant still on average
report that they decreased the amount, from about 16 cigarettes per day to 12 cigarettes per day.
Pregnancy is also associated with a drop in the prevalence of drinking alcohol, from 45 percent to
21 percent. And those women who continue to drink while pregnant on average report that they
decreased the amount, from 9 drinks per month to about 3 and a half drinks per month. The
only exception to these patterns is that is somewhat more common for women to quit exercising
after they discover that they are pregnant than it is for women to start exercising.
The data in Table 1 are presented to make the broad point that during the prenatal period
women make substantial investments in fetal health. Table 1 neglects some investments, such as
changes in maternal diet and illicit drug use, as well as all paternal investments in fetal health.
On the other hand, because the data are self-reported, the changes in maternal behavior may be
over-stated. It also should be noted that while women invest in fetal health, in many cases their
choices are not optimal from the public health viewpoint, i.e. their choices do not maximize fetal
health. For example, public health goals call for increasing the proportion of women who
receive early and adequate prenatal care from its 1998 level of 74 percent to a 2010 target level
61

-------
of 90 percent (USDHSS 2000). Nevertheless, it is clear that many women are willing to
sacrifice money, time, and cigarette and alcohol consumption to improve the health of their
unborn children. The next section outlines the standard economic approach to modeling this
behavior.
III. Conceptual Framework
This section considers a highly simplified one-period version of Grossman's (1972)
household production model of the demand for health and health-related goods. Assume the
mother receives utility from consuming a numeraire good X, her infant's health IH, and from
smoking cigarettes S: U = U(X, IH,S). The mother may purchase in the market prenatal medical
care (M), which does not provide utility directly, but is used to produce the commodity infant
health according to a household production functions. Infant health is also assumed to depend on
maternal smoking, and exogenous influences such as environmental quality, E: IH = IH (M, S,
E).
The mother chooses X, S, and M to maximize her utility subject to a standard budget
constraint and the household production function. The first order conditions for this
maximization problem implicitly define goods demand functions for X, S, and M as functions of
market prices, income, and the parameters that describe preferences (U (.)) and the technology of
household production (IH (.) ). Formally, the model also includes a commodity demand function
for infant health, which is conceptually distinct from the infant health production function.
Before discussing empirical applications, some brief comments on this theoretical model
are in order. Grossman's (1972) seminal model contains two key features: first, that health is a
commodity produced in the household; and second, that health is a form of human capital. The
62

-------
focus here is on behavior during the prenatal period and the production of fetal and infant health,
so the model is simplified to one period and abstracts from the dynamics of health capital over
the life cycle. Grossman (2000) provides a comprehensive review of theoretical and empirical
work based on his human capital model of health, while Currie (2000) contains an intertemporal
model of parents' investments in child health. By focusing on the mother's utility function, the
model also abstracts from the more complex problem of decision-making within the family. This
extension is discussed in a series of recent health economics papers (Jacobson 2000, Case and
Paxson 2001, Bolin, Jacobson and Lindgren 2001, 2002), as well as by Bergstrom (2003).
The general structure of the household production model of health provides the
conceptual framework for a great deal of empirical research in health economics. One approach
is to estimate a structural health production function as a function of endogenous health inputs
and exogenous factors. Section IV of this paper reviews research on the household production
of infant health in the U.S. , but the approach has also been commonly used in the context of
low-income countries (e.g., Barrera 1990, the Cebu Study Team 1992).
The Grossman model also provides the explicit or implicit framework for empirical
studies of the demand for various health-related goods. Section V of this paper reviews some
recent research on the demand for cigarettes and alcohol by pregnant women. These papers are
extensions of an extensive empirical literature reviewed in several chapters of the Handbook of
Health Economics: Chaloupka and Warner (2000) review empirical studies of the demand for
cigarettes; Cook and Moore (2000) review empirical studies of the demand for alcohol; and
Kenkel (2000) reviews empirical work on the demand for prevention broadly defined. As
Kenkel (2000, p. 1685) points out, while some empirical studies have tight links between the
63

-------
structure of the theoretical model and the empirical specification, more commonly the theoretical
model only provides general guidance for the empirical investigations, for example in terms of
the explanatory variables to be included in a demand model.
A number of recent studies take one step further away from structural models, and focus
on reduced-form estimates of the impacts of public policies on health, including the impact of so-
called "natural policy experiments." This approach can be used to study the impact of policy
changes on health outcomes and on the use of health inputs. For example, Currie and Gruber
(1996) examine the impact of Medicaid expansions on infant mortality; Currie and Grogger
(2002) examine the combined impact of Medicaid expansions and welfare reform on both the use
of prenatal care and fetal deaths. However, it is not in general appropriate to interpret the
estimated equations as either structural production functions or demand functions. As
Rosenzweig and Schultz (1983) demonstrate, the estimated coefficients from such hybrid
equations will generally be mixtures of preference and technology parameters. Section VI
discusses some examples from this body of research.
IV. Health Production Function Estimates and the Value of Infant Health
Willingness to Pay Expression
The health production function approach is a well-established method in environmental
economics research on the valuation of health as a non-market commodity (Freeman 1993, pp.
344-360). It is a revealed preference approach to valuation, where consumers' demand for a
health input reveals the value they place on the health output. The model in section III can be
used to derive the standard expression for marginal willingness to pay (MWTPe) for a health-
improving change in environmental quality (E). To complete the model sketched above, assume
64

-------
a simple goods budget constraint: Y = X + pm M + ps S; where Y is income and pm and ps are
the money prices of medical care and cigarettes. (Recall that X is the numeraire good so its price
is normalized to one.) To find the MWTPe, set the total derivative of the utility function equal to
zero and substitute in the first order conditions. After some manipulations, the change in income
necessary to hold utility constant after a change in environmental quality can be expressed as a
ratio of the technological parameters of the production function, which can be interpreted as the
marginal rate of technical substitution between E and M in producing infant health:
MWTPe = d Y/ d E = (IHE/ Mm ) Pm
As Freeman (1993, p. 349) stresses, one of the advantage of this expression is that on the
right hand side "all of the measures are functions of observable variables that can be calculated
given knowledge of the production function." Strictly speaking, the valuation expression
involves the individual's perceptions of the parameters of the health production function. It is
therefore typically assumed that, at least on average, individual perceptions are correct, so
econometric knowledge of the production function translates into knowledge of consumers'
perceptions of the production function. Unless extra data are collected on individual perceptions,
this type of assumption is common in the revealed preference approach. For example, many
studies of the value of a statistical life make comparable assumptions about the risks associated
with labor market and consumption decisions (e.g. Viscusi 1992a, Jenkins, Owens, and Wiggins
2001).
The household health production approach has been used to estimate marginal
willingness to pay for air quality-related health improvements for adults (Gerking and Stanley
1986) and infants (Joyce, Grossman and Goldman 1987). Dickie and Gerking (1991) extend the
65

-------
analysis to consider multiple symptoms, i.e. multiple health outputs. If the number of health
inputs exceeds the number of symptoms to be valued, it is still possible to express the marginal
willingness to pay for each symptom as a ratio of the technological parameters of the household
health (symptom) production function. Agee and Crocker (2001) provide a recent example of
the approach with multiple health outcomes, namely child and adult health. They use cross-
sectional data on parents who are smokers from a 1991 follow-up of the 1988 National Maternal
and Infant Health Survey (NMIHS). Each household in the sample has a three-year-old child.
Agee and Crocker use these data to estimate a structural household production model of parents'
smoking behavior, adult health, child secondhand smoke exposure, and child health.
The expression for MWTPe relies on the assumption that prenatal care is only demanded
as an input into infant health production. In many cases, important health inputs either provide
utility directly, such as cigarette smoking in the model sketched above, or enter some other
household production function.4 When an input like S is jointly demanded for several reasons,
the MWTPe can not be expressed as the marginal rate of technical substitution between E and S.
Instead, unobservable utility terms remain in the expression. Section V below discusses an
approach to health valuation in this situation.
The approach in previous environmental economics studies is to estimate directly the
necessary parameters of the health production function, including the marginal product of
environmental quality on health (IHe). As is noted elsewhere (Freeman 1993, p. 349, Dickie
1999), implementing this approach is thus very demanding of the data. Of particular relevance to
4More accurately, cigarette smoking jointly enters the infant health production function,
enters the mother's utility function directly, and enters the health production function of
the mother. This extension is sketched below in section V.
66

-------
the current literature review, health economics data sets often lack the necessary measures of
environmental quality. However, marginal willingness to pay for an improvement in fetal and
infant health (MWTPm), i.e. the marginal rate of substitution in consumption between X and IH,
is not as demanding. It can be shown that:
MWTPih = Um / Ux
= (d Y/ d E ) / IHe
= pm / Mm
The MWTPih thus requires only an estimate of IHm from health economics research on
infant health production functions, and a corresponding measure of the price of prenatal care.
The MWTPe can be thought of as the value of a standardized improvement in environmental
quality or any other exogenous change that yields a marginal change in infant health. It can be
used to value any public policy change that improves infant health at the margin, assuming, of
course, that the policy analyst has an outside estimate of the infant health improvement (i.e., a
term analogous to IHe).
Empirical Estimates of Infant Health Production Functions
Table 2 lists eleven studies that estimate the marginal product of prenatal care in
improving infant health. Seven of the studies use microdata and measure infant health by
birthweight in grams. Three studies use county- or state-level aggregate data and measure infant
health by the percentage of infants born at a low birthweight (below 2500 grams); one study uses
county-level data on neonatal mortality rates. The use of prenatal care is usually measured in
terms of whether it was initiated in the first trimester of pregnancy or delayed. In addition to
67

-------
measures of prenatal care, all of the studies include endogenous health inputs such as maternal
smoking and other variables such as maternal age and schooling.
The research on infant functions addresses a number of specification issues. The
functions are generally specified to be linear, although Rosenzweig and Schultz (1983, 1988)
also estimate Cobb-Douglas specifications, and Warner (1988) also estimates linear with
interaction terms, quadratic, and square root specifications.5 As might be expected,
specifications have evolved over time to address new research questions. Several of the studies
suggest that the parameters of the infant health production function vary significantly by race. In
another extension, Warner (1998) emphasizes the importance of maternal anthropometric
measures such as height and weight. Warner (1995, 1998) also explores whether subsequent
more frequent prenatal visits substitute for delaying pre-natal care after the first trimester.
The research on infant health production also addresses a number of econometric issues.
Following Rosenzweig and Schultz (1983), a central concern has been the endogeneity of the
health inputs. Rosenzweig and Schultz show that with individual heterogeneity that is known to
the mother but unobservable to the econometrician, ordinary least squares (OLS) yields biased
estimates of the parameters of the health production function. Their empirical results suggest
that women with health problems may seek prenatal care earlier to compensate, causing OLS to
underestimate the productivity of prenatal care. Rosenzweig and Schultz and most subsequent
studies use two stage least squares or a related instrumental variables technique to treat prenatal
care and other health inputs as endogenous. This approach generates a research debate across the
5Rosenzweig and Schultz (1983) also consider the translog specification, but it is
rejected in favor of the Cobb-Douglas.
68

-------
studies about the validity of the identifying exclusion restrictions and the explanatory power of
the instrumental variables as predictors of input demand.
Grossman and Joyce (1990) argue that in addition to treating health inputs as endogenous,
it is important to control for self-selection in the resolution of pregnancies as live births or
induced abortions. The selectivity bias could be in either direction. They find strong selection
effects for blacks but not whites, with the results suggesting that among blacks the unobserved
factors that increase the probability of a live birth are correlated with unobserved factors that
increase use of prenatal care and increase birthweight. Subsequent studies that use vital statistics
data also control for selectivity bias (Joyce 1994, Liu 1998). However, other recent studies such
as Warner (1998) that use data from surveys of mothers can not, because the data necessary to
estimate the selection equation are lacking.
Because of the variety of specifications, econometric methods, and data sets used, it is
difficult or impossible to determine a single 'best' estimate of the marginal product of prenatal
care in improving health. There is a strong consensus in the research that prenatal care is
productive, but the precise magnitude varies. A few examples illustrate typical results. After
controlling for the endogenous choice of health inputs, Rosenzweig and Schultz (1983) results
imply that prenatal care delay decreases birthweight by approximately 50 grams. Warner (1995)
estimates a monthly delay productivity of between 25 and 30 grams for black mothers. After
controlling for both endogeneity and selectivity bias, Liu (1998) estimates that each month of
prenatal care delay decreases birthweight by 76 grams. To consider a different health outcome
measure, Corman, Joyce and Grossman (1987) estimate that prenatal care reduces black mortality
69

-------
by 1.82 deaths per 1000 live births, but reduces white mortality by only 0.30 deaths per 1000 live
births.
Illustrative Calculations of Willingness to Pay for Infant Health
Marginal willingness to pay for infant health can be calculated by combining an estimate
of the marginal productivity of prenatal care in improving infant health with an estimate of the
price of prenatal care. Ideally, the price should be specific to the sample used to estimate the
infant health production function, in terms of both geographic area and time period. In addition,
although the simple model presented above abstracted from these complications, price should be
measured as the out-of-pocket cost paid by the mother after insurance, plus additional travel and
time costs incurred to receive the care. In practice, developing such a price measure is
challenging, so the following calculations should be viewed as illustrative.
Suppose the full price (out-of-pocked monetary costs plus travel and time costs) of
reducing one month of prenatal care delay is $300. From the studies reviewed above, this
reduction in delay might increase birthweight by 25 to 76 grams. Assuming the increase is 50
grams, the implication is that maternal marginal willingness to pay is about $6 per extra gram of
birthweight.
As another illustrative calculation, combining the cost of prenatal care with the estimate
from Joyce, Grossman and Goldman (1989) of the marginal product of prenatal care in reducing
neonatal mortality yields the willingness to pay for a small reduction in neonatal mortality risks.
As is conventional, this can be conveniently summarized as the value of a statistical life. Dickie
70

-------
and Nestor (1998) conduct the needed calculations to derive the per birth value of $43,000 to
$750,000 for whites and $59,00 to $1,450,000 for blacks.
V. Demand Function Estimates and the Value of Infant Health
Willingness to Pay Expression
The model sketched in section III can be extended to focus on mothers' consumption
choices that affect both their own health and fetal health. Suppose now that the parent receives
utility from consuming S, X, and her own health H, and additional utility from her child's health
IH. Assume the parent's utility is a separable function of consumption utility (U) and utility (W)
from child health: utility = U (X, S, H) + W (IH). Parent health and child health are produced
according to household production functions: H = H(S) and IH = IH (S). (This abstracts from the
use of prenatal care, to simplify the presentation). A smoking parent chooses S and X to
maximize her utility subject to a standard budget constraint and the household production
functions; call these optimizing choices S* and X *, with the corresponding parental and child
health consequences H* and IH*. For the parent who finds it optimal to quit smoking, call her
optimizing choices of consumption S** = 0 and X**, with the corresponding parental and child
health consequences H** and IH**. The net benefits of quitting smoking are therefore given by:
(1) NB = {U [ 0, X **, H**)] + W [IH**]} - {U [S*, X *, H*] + W [IH*]}
Equation (1) can be seen as the motivation for the empirical research on smoking during
pregnancy discussed below. If NB > 0, the individual is observed to quit smoking; otherwise the
individual remains a smoker. Equation (1) provides the basis for comparative static predictions
71

-------
about the determinants of smoking cessation. Pregnancy increases the net benefits of smoking
cessation through the W[ ] terms in equation (1). Standard arguments suggest that cigarette
prices, income, and various demographic characteristics and life cycle events also enter as
possible determinants of NB.
To derive the implications of maternal smoking decisions for valuing infant health,
consider a smoker who quits because of pregnancy or a new child. Before children her optimal
choice was to smoke, so the parent's direct utility from consumption is lower when she quits to
improve child health:
(2)	? U = U ( 0, X **, H**) - U (S*, X *, H*) < 0
Measured in utility units, ? U is the net consumption utility foregone in order to invest in
infant health. But for the smoker who finds it optimal to quit, NB > 0, which implies:
(3)	?U
-------
psO , pxO and income Y°, define the indirect sub-utility from consumption before child health
concerns with choices S*, X* and H* as v* = v(psO, pxO, Y°). Given the same prices and
income, but rationing the consumer to S** = 0, let her indirect sub-utility be given by v** =
v(ps0, pxO, Y°; s** = 0). A dollar-valued measure of the utility from goods consumption
foregone to invest in health is the compensating variation (CV) in income implicitly defined by:
(4)	v(ps0, pxO, Y°) = v(ps0, pxO, Y° + CV; S** = 0)
This compensating variation is the amount the consumer would have to be paid after she
has quit smoking to give her just as much consumption utility as she received when she was a
smoker. Because the parent quit smoking due to infant health concerns, CV will be the operable
definition of the parent's willingness to pay for infant health.
The CV can be approximated using standard methods from applied welfare economics
(Deaton and Muellbauer 1980, Varian 1978). In that approach, the CV implicitly defined by
equation (4) can be related to an area of consumer's surplus measured to the left of a
compensated (utility-held-constant or Hicksian) demand curve for cigarettes. The appropriate
area is measured with reference to a "virtual price" of cigarettes, psl that would convince the
consumer to quit smoking even before child health concerns (see Neary and Roberts 1980, and a
similar application by Kenkel 2002). The empirical estimates reviewed below provide measures
of the effect of pregnancy, a new child, and cigarette prices on the decision to quit smoking. The
estimated effects can be used to calculate the virtual price increase that has the same effect as a
pregnancy or new child. Consumers' surplus can then be calculated using estimates of the price
elasticity of smoking. Although the price elasticity estimates will correspond to an ordinary
73

-------
demand curve, the area CV measured with an ordinary demand curve approximates the exact
measure of compensating variation (Willig 1976).
The simple model used to derive parents' willingness to pay for infant health abstracts
from addiction, a potentially important aspect of decisions about cigarette and alcohol
consumption. Consumers' surplus needs to be carefully interpreted in the context of an addictive
good. Most smokers report a desire to quit, but this does not invalidate economic models of
smoking (Viscusi 1992b). The fact that they continue to smoke despite a stated desire to quit
means that quitting is costly; addiction may mean that this cost is better interpreted as the pain of
quitting rather than the foregone pleasure of smoking. Regardless how this cost is interpreted,
estimates of the costs smokers incur to quit smoking when pregnant reveal their willingness to
pay for child health.
Another concern is that estimates of smokers' or drinkers' willingness to pay for infant
health underestimate the average parents' willingness to pay. Research suggests that smokers
have different risk preferences from nonsmokers (Hersch and Pickton 1995, Viscusi and Hersch
2001). Assuming their willingness to pay for child health shows the same patterns, the estimates
of average smokers' willingness to pay will be a lower bound to average parents' willingness to
pay for child health. A related issue is that smokers appear to process risk information
differently (Viscusi, Magat, and Huber 1999). Smokers may have placed a low weight on
reports linking secondhand smoke to child health. In this case, smokers may be relatively
unwilling to change their behavior partly because they are using low risk assessments. These
limitations should be kept in mind.
Empirical Estimates of Maternal Demand for Cigarettes and Alcohol During Pregnancy
74

-------
Table 3 lists six empirical studies of the demand for cigarettes and alcohol during
pregnancy. Four of the studies explore the determinants of pregnant women's smoking
participation, i.e. whether or not they are regular smokers. One study explores the determinants
of both smoking participation and the daily consumption of cigarettes conditional on being a
current smoker; another study explores the determinants of drinking participation and the
monthly consumption of alcohol conditional on being a drinker. Three of the studies of smoking
participation use data from the national natality files, which starting in 1989 include an indicator
of whether the mother smoked during pregnancy. The first such study by Evans and Ringel
(1999) uses a sample of over 10 million births between 1989 and 1992, and the subsequent
studies by Ringel and Evans (2001) and Gruber and Koszegi (2001) increase the sample size
further by extending the sample period. Colman, Grossman and Joyce (2003) use an alternative
data set overseen by the CDC, and by pooling together data from 10 states over the years 1993 -
1999 obtain a sample of 115,000 women. The remaining two studies - Bradford's (2002) study
of smoking and Kenkel and Lin's (2003) study of drinking - use a sample of about 6,000 women
from the 1988 NMIHS, and its 1991 Followup. Although it is a smaller sample over a limited
time period, the NMIHS provides much more detailed information about smoking and drinking
behavior and the women's circumstances.
The main focus of research on the demand for cigarettes and alcohol during pregnancy is
to estimate the price-elasticity of demand, to explore whether higher taxes might be effective to
change these prenatal behaviors and thus improve health. For example, Ringel and Evans (2001)
estimate a price elasticity of -0.7, which suggests that cigarette taxes may be a powerful tool to
reduce smoking during pregnancy. However, as Corman, Grossman and Joyce (2003) point out,
75

-------
their estimate may be too optimistic: real cigarette prices have risen 60 percent since 1997, but
smoking during pregnancy dropped by only 7.6 percent, not the 42 percent drop implied by a
price elasticity of -0.7.
Bradford (2002) uses data from the 1988 NMIHS and its 1991 Followup to conduct a
more in-depth study of smoking behavior during pregnancy. Kenkel and Lin (2003) also use the
NMIHS data to conduct a similar study of drinking behavior during pregnancy. The NMIHS is a
sample of women who were pregnant in 1988, and some but not all of these women were again
pregnant when they were re-surveyed in the 1991 Followup. As a result, these studies are able
to estimate the impact of both pregnancy and prices on maternal behaviors. For example,
Bradford finds that during pregnancy light smokers reduce consumption by 1.6 cigarettes per day,
moderate smokers reduce their consumption by 3.4 cigarettes per day, and heavy smokers reduce
their consumption by 5.7 cigarettes per day. Analogously, Kenkel and Lin estimate that during
pregnancy, a drinking mother reduces her alcohol consumption by 4.5 drinks per month, and
again there are differences in the response of light and heavy drinkers. Because they also
estimate the price elasticity of cigarette and alcohol demand by pregnant women, the studies by
Bradford (2002) and Kenkel and Lin (2003) can be used to implement the valuation approach
based on consumers' surplus calculations.
Illustrative Calculations of Willingness to Pay for Infant Health
To illustrate the approach to infant health valuation, this section presents back-of-the-
envelope calculations of the values that are revealed by smoking and alcohol consumption
decisions. Bradford (2002) estimates that light smokers voluntarily forego from $610 to $800 in
consumers' surplus in response to pregnancy, while heavy smokers having their first child forego
76

-------
over $2,800 in surplus during pregnancy. According to information from the USDHSS (2001),
smoking during pregnancy increases the rate of perinatal mortality (still births and neonatal
deaths) from about 8 per 1,000 births to about 10 per 1,000 births. A heavy smoker who quits
while pregnant therefore gives up $2,800 to reduce perinatal mortality risks by 0.002. Together
these numbers imply that the value of a statistical life for an infant is $1.4 million. While many
caveats obviously apply, this back-of-the-envelope estimate compares reasonably well to other
estimates of the value of life of adults and older children.
Analogously, Kenkel and Lin (2003) calculate the consumers' surplus drinking mothers
give up during pregnancy. On average, during the entire pregnancy, the forgone consumer
surplus for an average drinking mother is about $37.8.6 However, this average obscures
important differences between three groups of drinking mothers: light drinkers, moderate
drinkers, and heavy drinkers. It is useful to explore the surrendered consumer surplus for sub-
populations since studies have shown that heavy drinking mothers impose higher risks on their
n
unborn children than moderate and light drinking mothers do. A drinking mother with less than
31 drinks monthly is defined as a light drinker; one with 31 to 59 drinks monthly is defined as a
moderate drinker, one with at least 60 drinks monthly is defined as a heavy drinker. This
definition is the same as that in most alcohol studies. On average, a heavy drinking mother
surrenders $451.3 in consumer surplus; a moderate drinking mother surrenders $ 247.9 and a
6	For those drinking mothers who choose to quit during pregnancy, the surplus foregone can only
be viewed as a lower bound of their perceived value to invest in their unborn child's health.
7	In Quelette et al. (1977), babies born to heavy drinkers had twice the risk of abnormality over
those born to abstainers or moderate drinkers. They find that 32% of infants born to heavy
drinkers demonstrated congenital anomalies; as compared to 14% in the moderate group and 9%
in the abstinent group.
77

-------
light drinking mother surrenders $24.2. Heavy drinkers, compared to moderate and light ones,
give up much more in consumer surplus since they perceive a larger benefit from reducing
alcohol consumption during pregnancy. A conservative incidence rate is that among heavier
o
drinking women the incidence of Fetal Alcohol Syndrome (FAS) is 1 in every 1000 live births.
Because an average heavy drinking mother reveals her willingness to pay to reduce probability of
FAS by 1/1000 is $451.3, this implies the value of a statistical case of FAS is $451, 300. By way
of comparison, the cost of illness estimates of Harwood and Napolitano (1985) value a case of
FAS at about $347,000 (in 1990$).
VI. Impact of Public Policies on Infant Health and Implications for Valuation
Willingness to Pay Expression
There are at least two challenges to estimating willingness to pay from empirical studies
of the impact of public policies on infant health. First, it may not be possible to recover the
necessary structural parameters from the reduced form equations estimated. Second, in many
cases a change in public policy represents a non-marginal change. The first point is similar to
Rosenzweig and Schultz's (1983) criticism of what they term "hybrid" health equations. In a so-
called hybrid equation, one input, for example prenatal care, and variables like income and prices
that are the determinants of the other inputs, are regressed against a measure of health. The
results are often interpreted as the causal effect or marginal product of prenatal care. However,
Rosenzweig and Schultz (1983) argue that this interpretation is invalid: they show that the
8 Estimates of FAS prevalence vary from 0.5 to 3 per 1000 live births in most populations.
However, the prevalence rate in some American Indian communities is as high as 9 per 1000
births.
78

-------
estimated effect of prenatal care on health from such an equation embodies both technology
parameters of the health production function and preference parameters of the utility function.
Even compared to a hybrid health equation, the approach of many empirical health
economics studies is further away from structural estimation of the household health production
function. For example, Currie and Gruber (1996) estimate the impact of Medicaid expansions on
infant mortality. In essence, Medicaid as a determinant of the use of prenatal care has been
substituted into the hybrid equation. The resulting reduced-form equation may be a desirable
approach to estimate the impact of the specific policy change. But the estimated effect of
Medicaid now combines the impact of Medicaid on the use of prenatal care (a demand effect)
and the impact of the use of prenatal care on infant health (a production function effect). It might
be possible to use additional information or assumptions about some of the structural demand
parameters to disentangle the effects. If the policy change can be treated as a marginal change, it
would be then be possible to derive estimates of the marginal product of prenatal care in
producing infant health. Such an estimate could be used in the expression derived above for the
marginal willingness to pay for infant health.
However, as has been already noted, in many cases discrete policy changes represent non-
marginal changes. Bockstael and McConnell (1983) suggest that it may be possible to value
such changes with reference to the areas behind appropriate marginal value and marginal cost
curves. Bockstael and McConnell provide a general discussion of welfare measurement in the
household production framework, emphasizing the distinction between a commodity such as
infant health and the market goods that are used as inputs to produce the commodity. They show
how the welfare effect of a change in the level of a public good can be measured either in the
79

-------
hypothetical 'market' for the commodity (output) or in an actual market for a good (input). The
welfare measure in the goods market is empirically implementable, and corresponds to the
change in the area behind a compensated demand curve for an input that is caused by a change in
the level of a public good. Dickie and Gerking (1991) use this approach to infer willingness to
pay for ozone control from the demand for medical care.
Some empirical studies estimate the impact of a policy on both the commodity infant
health and its impact on the use of a health input like prenatal care. This raises the hope that the
results are informative about the change in the area behind the demand curve for prenatal that
results from the policy change. As in the similar example of Dickie and Gerking (1991), it is
probably reasonable to assume that prenatal care satisfies the assumptions needed for the
Bockstael and McConnell (1983) approach to be valid. And although privately and publicly
insured consumers may pay little or no out-of-pocket monetary costs for prenatal care, a demand
curve can be still derived with respect to travel and time costs incurred. With estimates of the
demand curve and how the policy changes the demand curve, in principle it should be possible to
implement the welfare measurement derived by Bockstael and McConnell (1983). However, the
problem of recovering structural parameters from reduced-form equations re-appears. In this
case, the problem is to recover a demand function from a reduced-form equation showing the
impact of a policy change on input usage. For example, it may not be clear if the estimated effect
of the policy is on the demand side through changes in consumers incentives or the supply side
through changes in providers' incentives. It may again require extra information or assumptions
to identify the parameters of the demand function needed to implement the Bockstael and
McConnell (1983) welfare measure.
80

-------
Empirical Estimates of the Impact of Public Policies on Infant Health
Table 4 lists 10 studies of the impact of public policies on infant health. In all but one
study, the focus is on public policies such as Medicaid that are targeted at low-income and
disadvantaged populations. All of the studies examine the impact of a policy on some measure
of infant birthweight, with the focus often being on low birthweight and very low birthweight as
the most serious adverse outcomes. At least four of the studies also estimate the impact of the
policy under study on the use of prenatal care. At least some of the remaining studies measure
prenatal care, but it may not be used as an outcome variable. For example, Currie and Cole
(1993) include prenatal care use as an explanatory variable to estimate the impact of AFDC
participation on infant birthweight, controlling for differences in prenatal care use. Currie and
Cole (1993) do not provide a structural interpretation of the estimated impact of AFDC
participation, but note that it may combine an income effect with an additional effect due to
improved access to a range of other services from the welfare system.
Illustrative Calculations of Willingness to Pay for Infant Health
Existing research on the impact of natural policy experiments on infant health inputs and
outcomes does not support calculations of willingness to pay for infant health. As discussed
above, such calculations require additional information or assumptions to: (a) recover structural
parameters from the estimation results; and (b) implement the appropriate welfare measure for a
marginal or non-marginal change. Alternatively, an avenue for future work might be to re-
analyze these data sets to estimate the value of infant health.
VII. Discussion
81

-------
The review of empirical health economics research suggests a potential vein to be mined
for information on the value of fetal and infant health. As discussed in section III, it is probably
most straight-forward to derive estimates of maternal marginal willingness to pay for infant
birthweight. Because birthweight is associated with infant mortality and a range of subsequent
outcomes, it is a useful summary of infant health. Similarly, estimates of the value of
birthweight are potentially useful for the benefit-cost analysis of a variety of environmental and
public health policies. For example, food safety regulations to prevent exposure to Toxoplasma
gondii reduce risks for infants (Roberts and Frenkel 1990). Previous analyses that value reduced
infant mortality risks based on the discounted present value of lifetime earnings may substantially
underestimate willingness to pay.
82

-------
References
Agee, Mark and Thomas D. Crocker (1996). "Parental Altruism and Child Lead Exposure:
Inferences from the Demand for Chelation Therapy." Journal of Human Resources 31: 677-691.
Agee, Mark and Thomas D. Crocker (2001). "Smoking Parents' Valuations of Own and
Children's Health." Working Paper, Department of Economics, Pennsylvania State University -
Altoona.
Barrera, Albino (1990). "The Role of Maternal Schooling and Its Interactions with Public Health
Programs in Child Health Production." Journal of Development Economics 32: 69-91.
Berger, Mark, Glenn Blomquist, Don Kenkel and George Tolley (1987). "Valuing Changes in
Health Risks: A Comparison of Alternative Approaches," Southern Economic Journal 4: .
Bergstrom, Ted. (2003). "Benefit Cost Analysis and the Entanglements of Love." Presentation
at "Valuing Environmental Health Risk Reductions to Children," October 20 - 21, 2003,
Washington DC.
Bockstael, Nancy and R. McConnell (1983). "Welfare Measurement in the Household
Production Framework." American Economic Review 73: 806-814.
Bolin, Kristian, Lena Jacobson, and Bjorn Lindgren (2001). "The Family as the Health Producer
-	When Spouses are Nash-bargainers." Journal of Health Economics 20: 349-362.
Bolin, Kristian, Lena Jacobson, and Bjorn Lindgren (2001). "The Family as the Health Producer
-	When Spouses Act Strategically." Journal of Health Economics 21: 475 - 495..
Bradford, W. David (2001). "Pregnancy and the Demand for Cigarettes." Working Paper,
Department of Health Administration and Policy, Medical University of South Carolina.
Bresnahan, Brian W. and Mark Dickie (1995). "Averting Behavior and Policy Evaluation."
Journal of Environmental Economics and Management 29: 378-392.
Carlin, P. And R. Sandy (1991). "Estimating the Implicit Value of a Young Child's Life,"
Southern Economic Journal 58 (1): 186-202.
Case, Anne and Christina Paxson (2001). "Mothers and Others: Who Invests in Children's
Health?" Journal of Health Economics 20: 301-328.
Cebu Study Team (1992). "A Child Health Production Estimated from Longitudinal Data."
Journal of Development Economics 38: 323-351.
83

-------
Chaloupka, Frank J. and Kenneth Warner (2000). "The Economics of Smoking." Handbook of
Health Economics, Joseph Newhouse and Anthony Culyer, Editors (North-Holland).
Chay, Kenneth Y. and Michael Greenstone (1999). "The Impact of Air Pollution on Infant
Mortality: Evidence from Geographic Variation in Pollution Shocks Induced by a Recession."
NBER Working Paper No. 7442.
Chay, Kenneth Y. and Michael Greenstone (2001). "Air Quality, Infant Mortality, and the Clean
Air Act of 1970." Working Paper, Department of Economics, University of Chicago.
Colman, Greg, Michael Grossman and Ted Joyce (2003). "The Effect of Cigarette Excise Taxes
on Smoking Before, During and After Pregnancy," Journal of Health Economics forthcoming.
Corman, Hope, Theodore Joyce, and Michael Grossman (1987). "Birth Outcome Production
Function in the United States." Journal of Human Resources 22 (3): 339 - 360.
Currie, Janet (2000). "Child Health in Developed Countries." Handbook of Health Economics,
Joseph Newhouse and Anthony Culyer, Editors (North-Holland). 1053 - 1090.
Currie, Janet and Nancy Cole (1993). "Welfare and Child Health: The Link Between AFDC
Participation and Birth Weight." American Economic Review 83 (4): 971-985.
Currie, Janet and Jonathan Gruber (1996). "Saving Babies: The Efficacy and Cost of Recent
Expansions of Medicaid." Journal of Political Economy.
Currie, Janet and Jeffrey Grogger (2002). "Medicaid Expansions and Welfare Contractions:
Offsetting Effects on Prenatal Care and Infant Health?" Journal of Health Economics 21: 313-
353.
Currie, Janet, L. Nixon, and N. Cole (////) "Restrictions on Medicaid Funding of Abortion -
Effects on Birthweight and Pregnancy Resolutions." Journal of Human Resources 31 (1): 159-
188.
Deaton, Angus and John Muellbauer (1980). Economics and Consumer Behavior. Cambridge:
Cambridge University Press.
Devaney, Barbara, Linda Bilheimer, and Jennifer Schore (1992). "Medicaid Costs and Birth
Outcomes: The Effects of Prenatal WIC Participation and the Use of Prenatal Care." Journal of
Policy Analysis and Management 11 (4): 573-92.
Dickie, Mark (1999). "Willingness to Pay for Children's Health: A Household Production
Approach." In Valuing Health for Environmental Policy With Special Emphasis on Children's
Health Protection. Proceedings of the Second Workshop in the Environmental Policy and
84

-------
Economics Workshop Series, March 24 - 25, 1999. Edited by Shi-Ling Hsu, Environmental Law
Institute, Washington DC.
Dickie, Mark and Shelby Gerking (1991)."Valuing Reduced Morbidity: A Household Production
Approach," Southern Economic Journal 53: 967-983.
Dickie, Mark and Shelby Gerking (1991). "Willingness to Pay for Ozone Control: Inferences
from the Demand for Medical Care." Journal of Environmental Economics and Management 21:
1 - 16.
Dickie, Mark D. and Shelby Gerking (2001). "Parents' Valuation of Latent Health Risks to the
Children." " In Economic Valuation of Mortality Risk Reduction: Assessing the State of the Art
for Policy Applications, Proceedings, November 7, 2001. Edited by Abt Associates, Inc.
Dickie, Mark and Deborah V. Nestor (1999). "Valuation of Environmental Health Effects in
Children: A Survey." Working Paper.
Dubay, Lisa, Robert Kaestner, and Timothy Waidman (2001). "Medical Malpractice Liability
and Its Effect on Prenatal Care Utilization and Infant Health." Journal of Health Economics 20:
591-611.
Evans, William N., and J.S. Ringel (1999). "Can Higher Cigarette Taxes Improve Birth
Outcomes?" Journal of Public Economics 72: 135-154.
Frank, Richard G., Donna M. Strobino, David S. Salkever, and Catherine A. Jackson (1992).
"Updated Estimates of the Impact of Prenatal Care on Birthweight Outcomes by Race." Journal
of Human Resources :629-642.
Freeman, A. Myrick (1993). The Measurement of Environmental and Resource Values: Theory
and Methods. (Washington DC: Resources for the Future)
Gray, Bradley (2001). "Do Mediciad Physician Fees for Prenatal Services Affect Birth
Outcomes?" Journal of Health Economics 20: 571-590.
Grossman, Michael (1972). "On the Concept of Health Capital and the Demand for Health."
Journal of Political Economy 80 ( 2). March/April.
Grossman, Michael (2000). "The Human Capital Model." Handbook of Health Economics,
Joseph Newhouse and Anthony Culyer, Editors (North-Holland).
Grossman, Michael and Theodore J. Joyce (1990). "Unobservables, Pregnancy Resolutions, and
Birth Weight Production Functions in New York City." Journal of Political Economy 98 (5, part
1): 983 - 1007.
85

-------
Gruber, Jonathan and B. Koszegi (2001). "Is Addiction 'Rational'? Theory and Evidence,"
Quarterly Journal of Economics 116 (4): 1261-1305.
Hersch, Joni and Todd S. Pickton (1995). "Risk-Taking Activities and Heterogeneity of Job-
Risk Tradeoffs." Journal of Risk and Uncertainty 11 (3): 205-17.
Ippolito, Pauline M. and Richard A. Ippolito (1984). "Measuring the Value of Life Saving from
Consumer Reactions to New Information," Journal of Public Economics 25: 53-81.
Jacobson, Lena (2000). "The Family as Producer of Health - An Extended Grossman Model."
Journal of Health Economics 19: 611-637.
Jenkins, Robin, Nicole Owens, and Lanelle Wiggins (2001). "Valuing Reduced Risks to
Children: The Case of Bicycle Safety Helmets," Contemporary Economic Policy, 19: 397-408.
Johansson, Per-Olov (1993). Cost-Benefit Analysis of Environmental Change (Cambridge
University Press).
Jones, Andrew (1990). "An Econometric Investigation of Low Birth Weight in the United
States" Journal of Health Economics 10: 81-99.
Joyce, Theodore (1994). "Self-Selection, Prenatal Care, and Birthweight among Blacks, Whites,
and Hispanics in New York City." Journal of Human Resources 29(3): 762-94.
Joyce, Theodore (1999). "Impact of Augmented Prenatal Care on Birth Outcomes of Medicaid
Recipients in New York City." Journal of Health Economics 18: 31-67.
Joyce, Theodore J., Michael Grossman, and Fred Goldman (1989). "An Assessment of the
Benefits of Air Pollution Control: The Case of Infant Health." Journal of Urban Economics 25:
32-51.
Kenkel, Donald (1994). "The Cost of Illness Approach," in George Tolley, Donald Kenkel and
Robert Fabian, editors, Valuing Health for Policy: An Economic Approach (Chicago: University
of Chicago Press).
Kenkel, Donald (2000). "Prevention," in the Handbook of Health Economics, A.J. Cuyler and
J.P. Newhouse, editors. North-Holland, 2000, pp. 1675-1720.
Kenkel, Donald (2001). "Using Estimates of the Value of a Statistical Life in Evaluating
Consumer Policy Regulations." Manuscript prepared for a conference on Valuing the Health
Benefits of Food Safety, sponsored by the FDA, USD A, CDC, EPA, and others, September 13 -
15, 2000, College Park Maryland (invited speaker).
86

-------
Kenkel, Donald (2002a). "Approaches to Valuing Health for Policy: Common Ground and
Disputed Territor." Manuscript prepared for the "Health Valuation Workshop," University of
Central Florida, Orlando, March 18 - 19, 2002.
Kenkel, Donald (2002b). "Investments in Prevention: Health Economics and Healthy People. "
In Lindgren, B (ed) Individual Decisions for Health. London: Routledge. Forthcoming.
Kenkel, Donald and Tsui-Fang Lin (2003). "The Impact of Pregnancy and Taxes on Women's
Alcohol Use." Prepared for presentation at the Workshop on the Social Cost of Alcohol,
Neuchatel Switzerland, October 24-25 2003.
Levinson, Arik and Frank Ullman (1998). "Medicaid Managed Care and Infant Health." Journal
of Health Economics 17: 351-368.
Liu, Gordon G. (1998). "Birth Outcomes and the Effectiveness of Prenatal Care," Health
Services Research 32 (6): 805 - 823.
Liu, Jin-Tan, James K. Hammitt, Jung-Der Wang, and Jin-Long Liu (2000). "Mother's
Willingness to Pay for her own and her Child's Health: A Contingent Valuation Study in
Taiwan." Health Economics 9 (4): 319-326.
Maguire, Kelly B., Nicole Owens and Nathalie Simon (2001). "What Do Organic Baby Food
Purchases Tell Us About Parental Value for Reductions in Risks to Children's Health." In
Economic Valuation of Mortality Risk Reduction: Assessing the State of the Art for Policy
Applications, Proceedings, November 7, 2001. Edited by Abt Associates, Inc.
Mansfield, Carol (2001). "Discussion of Section V: Comments on 'Smoking Parents' Valuations
of Own and Children's Health' by Agee and Crocker." " In Economic Valuation of Mortality
Risk Reduction: Assessing the State of the Art for Policy Applications, Proceedings, November 7,
2001. Edited by Abt Associates, Inc.
Mount, Timothy, W Weng, and William Schulze (2000). "Automobile Safety and the Value of
Statistical Life in the Family: Valuing Reduced Risk for Children, Adults, and the Elderly."
Working Paper, Department of Applied Economics and Management, Cornell University.
Neary, J.P. and K.W.S. Roberts (1980). "The Theory of Household Behavior Under Rationing,"
European Economic Review 13: 25-42.
Neumann, J. and H. Greenwood (2002). "Existing Literature and Recommended Strategies for
Valuation of Children's Health Effects." National Center for Environmental Economics
Working Paper.
Neumann, P. and M. Johannesson (1994). "Willingness to Pay for In Vitro Fertilization: A Pilot
Test Using Contingent Valuation," Medical Care 32 (7): 686-699.
87

-------
Reichman, Nancy and Maryanne Florio (1996). "The Effects of Enriched Prenatal Care Services
on Medicaid Birth Outcomes in New Jersey." Journal of Health Economics 15 (4): 455-476.
Ringel, J. and William Evans (2001). "Cigarette Taxes and Smoking During Pregnancy,"
American Journal of Public Health 91: 1851-1856.
Roberts, Tanya and J. K. Frenkel (1990). "Estimating Income Losses and Other Preventable
Costs Caused by Congenital Toxoplasmosis in People in the United States." JAVMA 196 (2):
249 - 256.
Rosenzweig, Mark R., and T. Paul Schultz (1983). "Estimating a Household Production
Function: Heterogeneity, the Demand for Health Inputs, and Their Effects on Birth Weight."
Journal of Political Economy 91 (5): 723-746.
Rosenzweig, Mark R., and T. Paul Schultz (1988). "The Stability of Household Production: A
Replication." Journal of Human Resources 23 (4): 535-549.
Rosenzweig, Mark R., and T. Paul Schultz (1991). "Who Receives Medical Care - Income,
Implicit Prices, and the Distribution of Medical Services Among Pregnant Women in the United
States." Journal of Human Resources 26 (3): 473-508.
Tolley, George, Donald Kenkel and Robert Fabian (1994). Valuing Health for Policy: An
Economic Approach (Chicago: University of Chicago Press).
U.S. Department of Health and Human Services [USDHHS] (2000). Healthy People 2010:
Objectives for Improving Health.
U.S. Environmental Protection Agency [EPA] (2003). America's Children and the Environment:
Measures of Contaminants, Body Burdens, and Illnesses.
Varian, Hal R. (1978). Microeconomic Analysis. New York: W. W. Norton & Co. Inc.
Viscusi, W. Kip (1992a). Fatal Tradeoffs: Public and Private Responsibilities for Risk (New
York: Oxford University Press).
Viscusi, W. Kip (1992b). Smoking: Making the Risky Decision (New York: Oxford University
Press).
Viscusi, W. Kip. (1996). Economic foundations of the current regulatory reform efforts. Journal
of Economic Perspectives 10: 119-134.
Viscusi, W. Kip and Joni Hirsch (2001). "Cigarette Smokers as Job Risk Takers," Review of
Economics and Statistics 83: 269-280.
88

-------
Viscusi, W. Kip, Wesley A. Magat, and Joel Huber (1999). "Smoking Status and Public
Responses to Ambiguous Scientific Risk Evidence." Southern Economic Journal 66 (2): 250-
270.
Warner, Geoffrey (1995). "Prenatal Care Demand and Birthweight Production of Black
Mothers," American Economic Review 85: 132-137.
Warner, Geoffrey (1998). "Birthweight Productivity of Prenatal Care," Southern Economic
Journal 65 (1): 42-63.
Willig, R. (1976). Consumer Surplus without Apology. American Economic Review 66: 589-
597.
89

-------

Full Sample
Live Birth Sample
Fetal Death Sample
Infant Death Sample

N=18,594
N=9,953
N=3,309
N=5,332
exercise 3+ times a week before found out pregnancy
47%
47%
42%
43%
exercise 3+ times a week after found out pregnancy
42%
42%
36%
34%
start to do exercise 3+ times after found out pregnancy
7%
8%
6%
6%
quit doing exercise after found out pregnancy
13%
13%
13%
15%
months of doing exercise during pregnancy
2.76
2.77
2.07
1.87
Prenatal Investment-Prenatal Care

Full Sample
Live Birth Sample
Fetal Death Sample
Infant Death Sample

N=18,594
N=9,953
N=3,309
N=5,332
prenatal childbirth class
38%
38%
27%
19%
prenatal care
98%
98%
97%
94%
number of prenatal visits
12.87
12.90
11.32
10.27
Prenatal Investment-Vitamins Intake (at least 3 days a week during the three 3 months before found out pregnancy)

Full Sample
Live Birth Sample
Fetal Death Sample
Infant Death Sample

N=18,594
N=9,953
N=3,309
N=5,332
multivitamins and/or minerals
26%
26%
25%
25%
Vitamin A
2%
2%
2%
2%
Vitamin C
4%
4%
5%
4%
Folic Acid
1%
1%
1%
1%
Calcium
4%
4%
4%
4%
Iron
9%
9%
10%
11%
Zinc
1%
1%
1%
1%
Prenatal Investment-Vitamins Intake (at least 3 days a week during the three 3 months after found out pregnancy)

Full Sample
Live Birth Sample
Fetal Death Sample
Infant Death Sample

N=18,594
N=9,953
N=3,309
N=5,332
multivitamins and/or minerals
81%
81%
79%
76%
Vitamin A
3%
3%
3%
3%
Vitamin C
4%
4%
4%
4%
Folic Acid
3%
3%
3%
2%
Calcium
8%
8%
8%
8%
Iron
33%
33%
33%
35%
Zinc
2%
2%
1%
2%
Prenatal Investment-start to take vitamins at least 3 days a week after found out pregnancy

Full Sample
Live Birth Sample
Fetal Death Sample
Infant Death Sample

N=18,594
N=9,953
N=3,309
N=5,332
multivitamins and/or minerals
57%
57%
56%
52%
Vitamin A
2%
2%
2%
2%
Vitamin C
2%
2%
2%
2%
Folic Acid
2%
2%
2%
2%
Calcium
6%
6%
6%
6%
Iron
27%
27%
26%
27%
Zinc
1%
1%
1%
1%
90

-------
Prenatal Investment-Cigarette Consumption

Full Sample
Live Birth Sample
Fetal Death Sample
Infant Death Sample

N= 18,594
N=9,953
N=3,309
N=5,332
smoking participation before the 12 months of delivery
30%
30%
33%
37%
smoking participation after found out pregnancy
22%
22%
25%
30%
number of cigarettes per day before found out pregnancy
4.84
4.83
5.28
5.84
number of cigarettes per day after found out pregnancy
2.69
2.68
3.01
3.43
quit smoking after found out pregnancy
8%
8%
8%
7%
Prenatal Investment-Conditional Cigarette Consumption

Full Sample
Live Birth Sample
Fetal Death Sample
Infant Death Sample

16.15
16.15
16.24
15.92

Fi
.11 (Safh&TS
L
ve B(frfe238ifi)ple
;etal HJ&athC®aftiple
InfanttEte&tiOS^mple
lumber of cigarettes per day after found out pregnancy




N=^&>
N±SJ&2
drinking participation before the 12 months of delivery






drinking participation after found out pregnancy
21%
21%
16%
18%
number of drinks monthly before found out pregnancy
4.07
4.06
3.92
4.39
number of drinks monthly after found out pregnancy
0.72
0.72
0.78
1.09
quit drinking after found out pregnancy
25%
25%
26%
25%
Prenatal Investment-Conditional Alcohol Consumption

Full Sample
Live Birth Sample
Fetal Death Sample
Infant Death Sample
number of drinks monthly before found out pregnancy
9.03
(N=7,185)
9.02
(N=3,781)
9.37
(N=1,318)
10.24
(N=2,086)
number of drinks monthly after found out pregnancy
3.50
(N=3,145)
3.47
(N=1,738)
5.03
(N=522)
6.17
(N=885)
91

-------
Table 2: Infant Health Production Function Estimates
Study
Health output
Data
Rosenzweig & Schultz
(1983)
Birthweight in grams
1967-69 U.S. National
Natality Followback Surveys
Corman, Joyce and
Grossman (1987)
Neonatal mortality;
Low birthweight (below 2500
grams)
county-level data
Rosenzweig and Schultz
(1988)
Birthweight (grams)
1980 National Natality
Survey
Joyce, Grossman and
Goldman (1989)
Neonatal mortality
1977 county-level data
Grossman and Joyce (1990)
Birthweight (grams)
1984 New York City Vital
Statistics
Jones(1990)
Low birth weight (below 2500
grams)
1984 state-level data
Frank, et al. (1992)
Low birthweight (below 2500
grams)
1975 - 1984 county-level data
from natality files
Joyce (1994)
Birthweight (grams)
1984 New York City Vital
Statistics
Warner (1995)
Birthweight (grams)
1980- 1990 New York City
Vital Statistics
Warner (1998)
Birthweight (grams)
1988 NMIHS
Liu (1998)
Birthweight (grams)
1984 Virginia birth and
abortion certificates
92

-------
Table 3: Maternal Cigarette and Alcohol Demand during Pregnancy
Study
Demand measure
Data
Evans and Ringel (1989)
Smoking participation
1989 - 1992 national natality
files
Ringel and Evans (2001)
Smoking participation
1989 - 1995 national natality
files
Gruber and Kosegi (2001)
Smoking participation
1989 - 1996 national natality
files
Bradford (2002)
Smoking participation;
conditional quantity demanded
1988 National Maternal and
Infant Health Survey, and 1991
Followup
Colman, Grossman and
Joyce (2003)
Smoking participation
1993 - 1999 Pregnancy Risk
Assessment Monitoring
System
Kenkel and Lin (2003)
Drinking participation;.
Conditional quantity demanded
1988 National Maternal and
Infant Health Survey, and 1991
Followup
93

-------
Table 4: Empirical Estimates of Public Policies on Infant Health
Study
Policy
Outcome
Devaney, Bilheimer and Schore
(1992)
WIC participation;
prenatal care
birthweight;
Medicaid costs
Currie and Cole (1993)
AFDC participation
birthweight
Reichman and Florio (1996)
New Jersey Health Start
Program
birthweight;
hospital costs
Currie, Nixon and Cole (1996)
restrictions on Medicaid
funding of abortion
birthweight;
pregnancy outcomes
Currie and Gruber (1996)
Medicaid expansions
birthweight;
infant mortality
Levinson and Ulman (1998)
Medicaid managed care
prenatal care;
birthweight
Joyce (1999)
New York State's Prenatal
Care Assistance Program
birthweight
Dubay, Kaestner and Waidmann
(2001)
malpractice insurance
reform
prenatal care;
birthweight
Gray (2001)
Medicaid physician fees
prenatal care;
birthweight
Currie and Grogger (2002)
Medicaid expansions;
Welfare reform
Use of prenatal care;
fetal deaths
94

-------
Valuing Fetal and Infant Heath
Effects
Trish Hall
US EPA
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	

-------
Overview
•	Introduction
•	Why valuation is important
•	Discussion of the papers from a policy
perspective
•	Summary
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	

-------
Why is it important to value
fetal and infant health effects?
•	The ability to monetize benefits is
critical to the regulatory development
process
-	Benefit transfer using adult values is controversial
•	Improved information regarding fetal
and infant health effects
-	Birth defects
-	Fetal loss
-	Endocrine disrupters
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	

-------
Why is it important to value
fetal and infant health effects?
• Magnitude of the problem: Fetal Loss
Example
-Approximately one million fetal losses per
year in the US
• small change in risk = large reduction in cases
= large benefits
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	
98

-------
Comparison in brief
•	Mother's decisions regarding her own
health reveals how she values the
health of her unborn child
•	Specific values of unborn child or infant
are not considered
•	Same data set: 1988 National Maternal
and Infant Health Survey
•	A good first step
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	

-------
Policy Implications:
Nastis & Crocker
•	Conclusion: A mother values the health of
her fetal child about six times more than she
values her own health
•	Interpretation?
- Fetal child can be valued at six times the
adult value (either VSL or WTP)?
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	
100

-------
Points to Consider
Nastis & Crocker
•	Impact of elimination criteria
-	did not include gestations less than 20 weeks
-	only considered singleton births to mothers 35 or
younger excluding adolescents
•	Specific findings may not have an impact on
national-level analysis
-	A mother's first-borne child is valued more than
her subsequent children
-	Nonsmoking mother value their children more
highly than smokers do
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	
101

-------
Policy Implications
Kenkel
•	Conclusion: Varies depending on
which approach is used
-	prenatal care and birthweight
-	cessation of smoking or drinking
•	Interpretation:
-	Designed to explore existing health data
but illustrative calculations could be
beneficial to policy analysis
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	
102

-------
Policy Implications
Kenkel
•	Conclusion from Prenatal care and
Birthweight:
-	Marginal WTP $6 per extra gram of birth
weight
•	Interpretation?
-	Could be useful if we can determine a
relationship between exposure and
birthweight
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	
103

-------
Policy Implications
Kenkel
•	Conclusion: VSL for an infant can be
calculated using the two methodologies
-	Prenatal care = $43,000 to $1.5 million
-	Smoking cessation = $1.4 million
•	Interpretation-
-	Provides estimate of magnitude of loss
from mother's perspective
-Application may be limited
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	
104

-------
Points to Consider
Kenkel
Voluntary vs. Involuntary Risks
- smoking/drinking vs. exposure to
environmental contaminants
Consider using elimination criteria outlined in
Nastis & Cocker
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	
105

-------
Note about Terminology
• Both papers use terms such as child, fetal
child, neonate, and infant interchangeably
-	consistency needed
-	clarify what specific valuation refers too
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	

-------
Summary
Fetal and infant valuation is an extremely
complex issue but also extremely important to
public policy
Quantitative applications limited
- but improves are ability to discuss the
magnitude of impacts
V aluing Environmental Health Risk
Reductions to Children- Oct. 20,
	onm	
107

-------
Summary of Q&A Discussion Following Session III
Glenn Harrison (University of Central Florida) opened by saying, "I guess only Kerry
Smith could with a straight face say that he did a study of fertility amongst couples and
concluded that there is an important interaction between the male and female," to which
Dr. Smith retorted, "This came as a big surprise to me!"
Directing his next comment to Tom Crocker and Don Kenkel, Harrison continued by
stating what he felt was a very important issue for the purpose of this workshop: the fact
that "the extent to which the mother or the parent cares about the kid's health relative to
her own, or we try to draw the similar source of conclusions about the ratio of willingness
to pay" may depend on other motives in addition to the commonly assumed motive of
health concerns. He noted that Dr. Smith and Dr. Crocker were careful to talk about
contemporaneous sets of choices, and he suggested, "But let's have a minimal-minimal-
two-period contemporal choice by the mother, where the mother-forgive me if there are
any pregnant women in the house, because they'll kill me-where the mother only cares
about the consumption value of the child, from her own perspective, in the future period.
In other words, if the kid is born unhealthy, it's a pain for the mother-it reduces her
consumption in the following period, and that at least deserves some weight-we'll let the
data put what actual weight is on it." Harrison went on to explore the situation in which
the mother cares about the child's health exactly to the extent of her own, claiming that
"it could be contemporaneous because it could cause pregnancy complications for the
mother herself if she doesn't look after the kid, so that they're highly correlated, but very
physically." He closed by reiterating that "it could be that everything you two [i.e.,
Crocker and Kenkel] label as the ratio of caring about the infant's health to the mother's
health is simply the mother caring about her future consumption, and it's got nothing to
do with the children." This is a fundamental idea, he said, that "everyone at this
workshop has to address somehow rather than just impose their politically correct view
on the observed behavior."
Tom Crocker responded that he absolutely agreed that "introducing an additional period,
or a sequence of periods, after the birth into the mother's expected utility" makes sense.
He stated that it was mentioned but not made explicit in the model "in that the mother has
to worry about how much effort she will have to put forth and the extent to which she can
care for the child." He acknowledged that this was a good point, though a very complex
issue.
Don Kenkel said that he agreed also and explained that in the interest of constructing a
simple model, he had assumed that the only reason women were interested in good health
was through the preference function. For clarification, he asked Dr. Harrison whether he
was saying that health decisions also enter the budget constraint with regard to future
consumption. When Harrison confirmed this, Kenkel responded that he had "assumed
that away" and acknowledged Dr. Smith's point that these assumptions are important to
the willingness to pay expression and interpretations.
108

-------
Harrison then followed up with a related observation regarding the smoking data that had
been cited. He emphasized that the data revealed dramatic reductions in expectant
mothers' smoking during the pregnancy (and he acknowledged that the timing—after the
first trimester or before—is an important issue) followed by a resumption of smoking or
of higher levels of smoking after the pregnancy is over. To make his point, he further
stated that if the data show that the mothers tend to resume the same level of smoking
that they engaged in before the pregnancy, then there would be some basis for assuming
that the smoking modification was motivated by concerns for the fetuses' health.
Reiterating Dr. Smith's point, he closed by saying, "You've gotta have some more
handles in order to draw that and tease those motives."
Don Kenkel responded by saying, "Empirically, you're exactly right-a lot of women quit
smoking, but just during pregnancy, and there's this incredible recidivism effect where
after the pregnancy is over they start back up smoking."
Dr. Crocker added, "From a structural perspective, what you're saying also implies that
to explain the consumption of drinking or smoking post-natal requires that one go back
and look at the mother's decisions while she was pregnant, simply because her decisions
while she was pregnant may very well affect her demand for inputs-smoking, drinking-
after the child is born."
Dr. Smith commented that one of the reasons why he and his colleagues were interested
in looking at the restrictions of the negative preferences anew is that "weak
complementarity and weak substitution are actually examples of discontinuities in
preferences, where there is a change-and that's really what's important about them-and
we lose track of that when we focus exclusively on the zero consumption level." He
went on to agree with Glenn Harrison that there are lots of other points of discontinuity
where there are abrupt changes in behavior, and he stated that these situations provide
really important information for answering some of these questions. He concluded by
adding that "oftentimes, in the health data sets and these kinds of other behaviors there
are real opportunities to get at those discontinuities, and we're just failing to use them."
Dr. Crocker commented that he doesn't understand the appeal to weak complementarity
when dealing with health considerations. He stated that it makes sense when dealing
with recreation—for example, if your pond is polluted and you don't fish, then you'll
have no demand for fishing rods. He concluded by saying, "But if you're in poor health,
why it is that you have no demand for health inputs is a bit beyond me. I would think
you'd have more of a demand for health inputs, for cleaner air, if in fact you're in poor
health."
Glenn Harrison replied that there are certain segments of the population for whom the
non-pecuniary costs of availing themselves of health inputs are massive. He cited the
"huge differences in black/white fetal death rate and infant death rate" that most studies
attribute largely to the real costs of getting off work, traveling to a healthcare setting,
arranging baby care, and so forth. In closing he said that "there are some stories there"
for anyone who takes the time to tease the racial differences apart.
109

-------
Partly in response to Dr. Crocker's comment, Dr. Smith offered this clarification: "If a
person is not sick chronically with asthma or something else, you don't have a demand
for care-giving activities. That would be an example of weak complementarity. If on the
other hand, you live in an area where there is a high level of ozone or something and you
have a child ... in a highly polluted area with ozone or something, you might take some
mitigating behavior, which would be more like weak substitution-you would not allow
the child to play outside, let's say, on an ozone-alert day or something like that. So, the
point is the discontinuity arises as a consequence of the child's condition and what state
the child is in in relationship to the environmental conditions, and it could be either weak
complementarity or it could be weak substitution."
Scott Grosse (Centers for Disease Control and Prevention) responded to Dr. Crocker's
paper by saying that he thought the meaning of "health" needs to be made clearer. He
stated that weight gain during pregnancy is only one dimension of an expectant mother's
health and that there are many other dimensions of the mother's health that possibly
relate differently to the infant's health. He also suggested that it would be useful to have
a comparable measure, such as healthy days-healthy days of the child, healthy days of
the mother-by which one could actually make a comparison. Dr. Grosse went on to
point out that gestational diabetes actually leads to higher birth weight and that anemia
might present a different relationship for every dimension of maternal health. Factors
such as these make it very difficult to generalize about the relative value of weight gain
during pregnancy.
In agreement, Don Kenkel replied, "And to make Kerry's point: We have multiple
attribute health production-health outputs, as they say. Just think of the extra structure
we have to impose to estimate all the different marginal products."
110

-------
Valuing Environmental Health Risk Reductions to Children
PROCEEDINGS OF
SESSION IV-AM: AGE-SPECIFIC VALUE OF STATISTICAL LIFE ESTIMATES
A WORKSHOP SPONSORED BY THE U.S. ENVIRONMENTAL PROTECTION AGENCY'S
NATIONAL CENTER FOR ENVIRONMENTAL ECONOMICS (NCEE), NATIONAL CENTER FOR
ENVIRONMENTAL RESEARCH (NCER), AND OFFICE OF CHILDREN'S HEALTH PROTECTION;
AND THE UNIVERSITY OF CENTRAL FLORIDA
October 20-21, 2003
Washington Plaza Hotel
Washington, DC
Prepared by Alpha-Gamma Technologies, Inc.
4700 Falls of Neuse Road, Suite 350, Raleigh, NC 27609
l

-------
ACKNOWLEDGEMENTS
This report has been prepared by Alpha-Gamma Technologies, Inc. with funding from the National Center
for Environmental Economics (NCEE). Alpha-Gamma wishes to thank NCEE's Kelly Maguire and
Project Officer Nicole Owens for their guidance and assistance throughout this project.
DISCLAIMER
These proceedings are being distributed in the interest of increasing public understanding and knowledge of
the issues discussed at the workshop and have been prepared independently of the workshop. Although the
proceedings have been funded in part by the United States Environmental Protection Agency under
Contract No. 68-W-01-055 to Alpha-Gamma Technologies, Inc., the contents of this document may not
necessarily reflect the views of the Agency and no official endorsement should be inferred.
11

-------
TABLE OF CONTENTS
Session IV-AM: Age-Specific Value of Statistical Life Estimates
Preliminary Research: Valuation of Cancer Risks in Children and Adults in Minnesota
Chris Dockins, U.S. EPA, National Center for Environmental Economics (NCEE)	1
The Effects of Age and Family Status on the Value of a Statistical Life: Evidence
From the Automobile Market
William Schulze, Cornell University 	4
Willingness To Pay for Reduced Risk: Inferences From the Demand for Bicycle Helmets
Nicole Owens and Lanelle Wiggins, U.S. EPA, NCEE	18
Discussant
Kelly Maguire, U.S. EPA, NCEE 	38
Summary of Q&A Discussion Following Session IV-AM	43
ill

-------
Valuation of Cancer Risk in Children and Adults in Minnesota
Nathalie Simon and Chris Dockins, US EPA1
presented by Chris Dockins
In Minnesota, there is some expectation among health policy professionals that
"negligible risk" may soon be defined more stringently for cancer risks that affect children,
resulting in standards for cancer risk setting of 1 in a million rather than the current level of 1 in
100,000. How exactly such standards could be effected in practice is a difficult question, and it
is possible that the de facto outcome of this type of policy change would be a 1 in a million
negligible risk standard for virtually all contaminants in an effort to protect children.
Faced with the possible need to assess the merits of this approach the Minnesota
Department of Health (MDH) asked EPA for information on recent or ongoing work that could
answer questions about the extent of risk avoidance, especially cancer risk, that the public
believes is appropriate for adults and children. The economics literature is quite sparse in the
areas of children's health valuation and cancer risk valuation generally and as such currently
contains little guidance on this question. To fully address this issue requires assessment of at
least two largely unaddressed sources of heterogeneity in risk valuation: age (or, more
specifically, adult/child status) and cancer.
Surprisingly, given the current crises faced by state governments across the country, the
MDH secured state funding to investigate these questions with an eye toward developing a
survey of state residents. After learning that EPA's National Center for Environmental
Economics (NCEE) and Office of Children's Health Protection (OCHP) had an ongoing interest
in promoting work in this field, MDH approached EPA for technical consultation regarding
survey development on these issues. After a series of meeting in which we each presented our
existing work on valuation and children's health and identified mutual research interests, we
resolved to develop a survey instrument that would address the following questions:
How do public preferences compare with private preferences for risk reduction
How do Minnesota residents perceive and value lifetime or long-term cancer risks
What generally are the public and private preferences in Minnesota for reduced risks of
dying from cancer as an adult from (1) child exposure and (2) adult exposure?
How does the length of the latency period affect valuation estimates for cancer risk
reductions? Does this vary between adult exposures and child exposures?
1 The opinions expressed in this paper are those of the authors and do not necessarily represent the US
EPA. Also, this work is being developed in consultation with a consortium of government analysts and
academicians in Minnesota, including: Pamela Shubat, Chuck Stroebel, and Amy Lockheart at the Minnesota
Department of Health (MDH); Pat Welle at Bemidji State University; Andy Klemer at the University of Minnesota-
Duluth; and Becky Judge, St. Olaf College.
1

-------
We began by holding a series of focus groups in order to understand some fundamental
perceptions and concerns about environmental cancer risks in Minnesota. These focus groups
included parents and non-parents, and sought to represent a general cross-section of the adult
population in the state. Because about half of the state population live in the greater
Minneapolis-St. Paul metropolitan area, we held four of the eight focus groups we conducted in
that area with the remaining four groups held in the cities of Mankato, Rochester, Duluth, and
Bemidji. The locations outside of the twin-cities area were chosen to capture regional variation
in risk attitudes and perceptions. In particular, Mankato was chosen because of the heavy
agriculture industry in the area, while Duluth was chosen because of its size and role as a
shipping port and industrial center. Bemidji was chosen as representative of the northern portion
of the state which is dominated by lakes and recreational opportunities. The area around Bemidji
also has a relatively large Native American population. Finally, Rochester is one of the three
largest cities in Minnesota and was chosen to be representative of the state to the southeast of the
twin cities.
Distinct materials were developed to guide the discussion in each focus group and these
materials evolved over time.2 In general, we developed scenarios to address both public and
private "goods" that would reduce carcinogenic exposure to adults and children. These
exposures would cause cancer with a latency period. We began by testing scenarios for risk
reductions that were based on specific contaminants (e.g., benzene) and specific types of cancer
(e.g, brain cancer and kidney cancer). Further, in an attempt to separate risk reduction
possibilities for adults and children we initially focused on scenarios with separate exposures and
policies for adults and children. For example, some hypothetical situations considered policies
targeted narrowly at reducing exposure to carcinogens in schools, which would primarily, but
not exclusively, affect children.
Because MDH and NCEE wanted to focus on long-term cancer risks, we initially
presented focus groups with a presentation of lifetime cancer risks including detailed information
on the when risks were reduced. In their most complex form this information was presented as
distributions of risk over a lifetime based on data from the National Cancer Institutes SEER
database. We also presented separate displays of magnitude of lifetime risk reduction
accompanied by stylized displays of the distribution of the reduction over time.
What did the focus groups tell us? First, any scenario that involved public risk reduction
paid for through a tax mechanism was rejected. The size and distribution of state taxes is simply
too sensitive a topic to be included in the hypothetical scenario. Interestingly, however, when
public interventions are portrayed as a rise in prices for associated commodities, food prices, for
example, there was little rejection of the policies based on payment mechanism.
Local issues played a key role in the perceptions of environmental policies to reduce
cancer risk. In Duluth, for example, residents were keenly aware of surface water quality and
2In keeping with Paper Work Reduction Act requirements, no more than 9 people participated in each of
our focus group discussions. Topics and materials developed to guide discussions varied across focus groups.
2

-------
could even recall specific state recommendations on fish consumption. The presence of a
Superfund site near Bemidji seemed to increase the respondents sensitivity to environmental
cancer risks in that area, while Mankato's concern for programs that might target pesticides
reflected the region's economic reliance on agriculture.
We also found that respondents demanded more information on background mortality
risks before responding to choice questions about cancer risk reductions. In fact, we modified
the draft questions to explicitly account for non-cancer mortality risk to satisfy these concerns.
Respondents also had difficulty linking specific exposure scenarios with specific cancers and
other illnesses. Further, when the proposed risk reduction scenario required modification to
existing systems in their home (e.g., installation of air filters), respondents required additional
information on current risk levels before proceeding.
Finally, very initial responses suggest that there is little sensitivity to the timing of risk
reductions associated with children's exposures to environmental carcinogens. This is perhaps
not surprising considering there is a minimum of 30 years before childhood exposures become
manifest in cancer outcomes. If choices for children's risk reduction reflect standard discounting
assumptions then the difference in a risk reduction 40 years hence from one 50 years hence is
small in present value terms. On the other hand, insensitivity to timing could simply reflect that
parents are considering only whether the child will receive a risk reduction at any time,
regardless of the time or magnitude. Additional interviews are necessary to determine how
adults are considering long term risks to children.
So, where are we now? We have found that respondents accept our framing the scenario
in terms of persistent environmental carcinogens in food as the source of environmental cancer
risk and risk reduction. Also, respondents seem to accept distinguishing public and private
programs by whether a testing program is optional (labeled foods at a premium price) or
mandatory (all foods at an increased price). Risk reductions to adults vs. children are
distinguished by whether the testing programs focus on contaminants primarily associated with
(1) long-term cancer risks from child exposure, or (2) cancer risks from adult exposure. We
have loosely correlated these with cancer initiators and cancer promoters, respectively.
Our risk communication devices are based on the grids recently developed and used by
others in the literature (Alberini, et al.; Krupnick, et al.; Corso, et al.; Cameron and DeShazo).
These grids include cancer and non-cancer mortality risks typically over a 20-year time period in
order to respond to MDH's interest in views of lifetime cancer risks. We plan to experiment
with animation to convey the timing and magnitude of risk reductions in additional cognitive
interviews.
We anticipate continuing with survey development in the coming months. If all
continues to go well, MDH will have the option of implementing their survey as early as summer
2004.
3

-------
I lll EFFECTS OF AGE AND FAMILY STATUS ON THE VALUE OF STATISTICAL
LIFE:
EVIDENCE FROM THE AUTOMOBILE MARKET AND A NATIONAL SURVEY OF
AUTOMOBILE USE
by
Timothy Mount, William Schulze
Weifeng Weng, Ning Zhang,
Department of Applied Economics and Management
Cornell University
Laurie Chestnut
Stratus Consulting
This research was supported by United States Environmental Protection Agency Cooperative
Agreement Number CR824393-01-1. Data analysis was supported by the Robinson endowment,
Cornell University. We would like to thank Margaret French for her assistance in preparing the
manuscript. All conclusions and remaining errors are the sole responsibility of the authors.
Research Strategy
We develop a theoretical model in which automobile safety is shown to be a family public good
where the marginal cost of purchasing and operating a safer automobile is set equal to the usage-
weighted sum of the values of statistical life (VSL) of family members.
Using this theoretical result we can estimate the VSL for different family members (children,
adults and seniors) by collecting primary data on automobile usage by family members that is
combined with secondary data from both the automobile market and the FARS data set on
automobile accidents.
An important issue that has clouded the potential reliability of the VSL obtained from estimated
hedonic price functions for automobiles (that include risk of death) is that prior studies have
shown what appears to be a positive correlation between fuel consumption and the price of
automobiles rather than the expected negative correlation since people should be willing to pay
less for cars with poor fuel economy (See Atkinson and Halvorsen, 1990, and Dreyfus and
Viscusi, 1995). Our theoretical work provides a possible explanation that also suggests a revised
estimation procedure.
4

-------
Theory: Fuel Consumption
To begin, we address the problem of fuel consumption by considering the case of a single
individual (buyer) with no family who may, or may not, survive for a single period. We then
consider the choice of fuel consumption by makers.
The Buyer's Decision:
Let
c = consumption,
w = wage income,
r = risk of a fatal automobile accident per mile driven,
II = probability of survival without automobile fatality risk,
Il-r = probability of survival with automobile fatality risk,
m = total miles driven
a = level of some other automobile attribute
P(r,a) = automobile price per mile driven (decreasing in r)
F*(r,a)) = fuel consumption per mile (increasing in r and a)
G = price of fuel
U(c,a,m) = strictly concave utility function.
Note: subscripts or primes denote derivatives where appropriate.
Note that we propose that the individual realizes that the fuel consumption of the car is itself a
function of the attributes of the automobile. We will justify this proposal when we consider the
manufacturer's decision below.
To abstract from the life cycle issues of owning and financing an automobile, we analyze the
problem in terms of the annualized price per mile of owning the vehicle, P, without loss.
The buyer is assumed to maximize expected utility,
where it is assumed that the death state provides no utility because the individual has no family,
subject to the budget constraint,
(IT-rm)U(c, a, m),
(1)
(II-rm)(w-c) - P(r,a)m - GF*(r,a)m = 0.
(2)
The optimal choice for r, risk per mile, is determined by
VSL = -(Pr + GFr*),
(3)
where
VSL = (U/Uc) + w - c.
(4)
5

-------
The optimal choice of the attribute, a, is determined by
Ua/Uc = m(Pa + GFa*)
(5)
The total miles driven, m, is determined by
Um/Uc - rVSL - GF* = P
(6)
The Maker's Decision
Competitive automobile manufactures will be forced to minimize the cost per mile of driving
their automobiles including both the capital and fuel cost per mile of automobile life given the
choice of other characteristics (r and a).
Consider the design problem of a particular manufacturer with a cost of production per mile of
life for the cars that they offer of C(r,a,F). Given a particular choice of r and a by buyers, the
maker is forced by competitive pressure to minimize the total cost per mile to buyers,
The condition for optimal fuel consumption in the engineering design of the vehicle is then
This implies that there is an optimum fuel consumption F*(r,a) for any choice by consumers of r
and a and the cost function relevant for the hedonic price solution for profit maximization over r
and a by the maker is C*(r,a,F*(r,a)).
The maker faces a hedonic price function only defined in r and a, P(r,a), not fuel consumption
which is optimized in the engineering design of the vehicle, and maximizes profits
C(r,a,F) + GF.
(7)
-C F = G.
(8)
P(r,a) - C*(r,a,F*(r,a))
with respect to a, implying
(9)
and with respect to r, implying
Pr=Cr*.
(10)
6

-------
In summary, given G, the price of fuel, the choice of F will be made by the automobile maker
since fuel usage will be optimized by makers for any combination of attributes chosen by
consumers. Consumers and makers are faced with a hedonic price function P(r,a) which is the
envelope curve of the cost tradeoffs for makers and value tradeoffs for consumers between
attributes. Buyers face a pre-optimized choice of fuel consumption, F*(r,a), for each level of
attributes that they choose in their purchase decision.
If these arguments are correct, the appropriate procedure is to estimate F*(r,a) and P(r,a) and use
(3) above to estimate the VSL for the individual from these relationships and the price of
gasoline, G.
The VSL for Family Members
The model developed above can be extended to a family setting by using the Nash cooperative
bargaining between parents approach employed by McElroy and Horney (1981).
Let
i = 1, 2,...,,n denotes individual family members,
i = 1 denotes the mother,
i = 2 denotes the father,
i = k = 3,	,n denotes children,
c; = consumption of the ith family member,
w; = wage of family member i,
r= automobile fatality risk per family member per mile,
11; = probability of survival, excluding automobile fatality risk, of i,
m = total vehicle miles driven
mj = total miles of driving for family member i
P(r,a) = automobile price per mile driven,
F*(r, a) = fuel consumption per mile driven,
Uk (ck,a,mk) = child's utility function,
U'( c;;...., mi,a,(IIk-r)Uk(ck,mk),....) = parent's utility function, and
E'= individual expected utility in separation (i = 1, 2).
In the Nash cooperative bargaining solution,
[(ITi-rmOU1 - E1] [(n2-rm2)U2 - E2],
(11)
is maximized with respect to C;, r, a, m, and m;,
subject to the budget constraint,
n
^ (n; -rm;) (w; - Ci) - (P - GF*)m = 0,
(12)
z = l
7

-------
and constraints on the use of the car such as,
m - m; > 0 i = 1,	, n
so that no individual family member can ride more miles than the car itself travels, and
mi + m2 - mi2 - mk > 0 k = 3,...., n
so that no child can ride more miles than the parents can collectively drive the child. Note that, to
avoid pointless complication of the model, mi2 is taken to be a constant.
The resulting conditions for choosing the level of automobile risk and miles driven imply that the
individual VSLs of family members all take the form:
VSL; = UYU'c + w; - c; i = 1,	,n.	(13)
The choice of automobile risk, r, is determined by
2 kiVSLi = -(Pr + GFr*)	(14)
2 = 1
where usage weights for the vehicle for each family member are defined as k, = m,/m.
Estimation Strategy
Since available measures of vehicle safety are affected by selection bias (more dangerous drivers
are attracted to Corvettes and safer drivers to minivans) the FARS data set was used to estimate
vehicle risk for vehicles in the sample for a standard driver. This corrected risk is used in
estimating the hedonic vehicle price function.
Hedonic vehicle price and separate fuel consumption equations were estimated to calculate the
per mile cost of reducing risk for vehicles purely as a function of vehicle (not driver) attributes.
This allows estimation of the rhs of equation (14).
Since data were not available on m, and m, we conducted a national survey to obtain the
necessary information to allow estimation of the VSL for children, adults, and seniors using
equation (14).
Because m, and m are endogenous variables in a system of simultaneous first order conditions, a
two-stage procedure is required to obtain consistent estimates using equation (14). In the first
stage, reduced-form equations for m, and m are estimated using appropriate exogenous variables.
The predicted m, and m that are uncorrected with the residuals in equation (14) are then used as
instrumental variables for m, and m to obtain consistent estimates of the VSL for children, adults
and seniors.
8

-------
Survey Design and Implementation
The survey consisted of two parts, a telephone screening survey used to develop an appropriate
sample and collect information on usage, followed by a mail survey used to collect subjective
probability measures.
Both the telephone and mail surveys were extensively pre-tested and revised prior to
implementing a pilot aimed at 80 households to formally test the telephone/mail survey
methodology.
The purpose of the telephone survey was to identify appropriate households and to obtain data on
automobile usage that was judged too difficult for respondents to fill out themselves in a mail
survey.
Both the telephone and the mail survey were developed following Donald Dillman's Tailored
Design Method (1999).
The telephone survey:
•	Determined if a household met the requirements for the sampling.
•	Asked for detailed information on automobiles owned or leased by the household and
elicits information on the residents' ages and relationships.
•	Elicited the total mileage driven and percentage of miles that each member of the
household rides in each of the three most driven cars. Needless to say, these are difficult
questions and necessitated a personal telephone interview with trained interviewers.
•	Employed a random digit-dialing sample of 8519 telephone numbers from Sample
Survey Inc. (Note that random digit dialing produces a large number of non-household,
disconnected, or ineligible numbers for household surveys.)
•	Was implemented between July 1 and August 5, 2001 and employed a minimum of 13
attempts to reach each telephone number.
The overall response rate was 40%. This produced 1,235 completed interviews. Of these, 926 or
75% agreed to participate in the mail survey.
The follow-up mail survey:
•	Was titled "WHAT ARE YOUR VIEWS ON AUTO SAFETY," and shows a picture of a
family next to a Ford Windstar (thanks to Ford for granting permission to use the photo).
•	Thanks the respondent and repeats the information on the most, second most and third
most driven automobiles.
9

-------
•	Asks about insurance and repair costs and features of each of the vehicles.
•	Collects subjective risk information from respondents that requires use of risk ladders and
asks for a subjective risk assessment of having a fatal accident (compared to the average
driver in the same type of automobile) for the respondent, for a child's risk of dying
relative to an adult's risk in a serious automobile accident and for their perceived risk of
the safety of the vehicles that they drive.
The mail survey was sent in waves from July 6, 2001 to August 6, 2001. The survey packet
included a letter, a $5 cash incentive, the 12-page survey booklet, and a post-paid return
envelope. The overall response rate after multiple contacts including reminder phone calls for
the mail survey was 74% with 625 completed surveys, exceeding the initial target of 600.
Estimating the Components of the VSL Model
Vehicle Risk (Step 1):
Data from the Fatal Accident Reporting Service (FARS) and National Personal Transportation
Survey (NPTS) were used to estimate the driver independent risk of vehicles. This analysis has
been presented in full in a report to the EPA (Environmental Protection Agency) and a research
paper.
This risk was determined by the probabilities of having different types of accidents (one-vehicle,
two-vehicle and multi-vehicle), and the probabilities that the occupants will survive in these
accidents. All of these probabilities are functions of the vehicle's characteristics and the
characteristics of the driver and the occupants.
The safety rating of each type of vehicle is computed using the same set of characteristics for the
driver and the occupants.
The safety rating used in the hedonic models for each type of vehicle was computed under the
assumption that there are two adults in each vehicle who drive 14,000 miles in a year. The effect
of making this assumption (as shown in the standardized risk scales) is that some vehicles, which
have high-observed rates of fatalities, such as pickup trucks, have lower predicted rates of
fatalities. The reason is that the specified occupants are more safety conscious (e.g. by wearing
seat belts) than the typical behavior of the actual occupants in the fatality data.
10

-------
Unadjusted Scales for the Risk of Mortality
Low Fatality
1
2
3
4
5
6
7
8
9
10
11
1 n
0
L0 20 30 4
tO 50 60 70 80 90 100+
small sedan &


1



wagons
0
30.8


101.4
middle sedan &





wagons
2.4
22*5
76.6


large sedan & wagons
8.2 17*7 44.9
luxury sedan &
wagons





0
9.5 48



small & mid.





specialties
1.7
33'8

83.6

luxury sports

0
2&2


99.7
small suv

38.9 53'4
69.8


large suv

0
21A


110.1
van (minivan)





0
24.'6

91.5

small pickup
12.1 25.1 53.7
large pickup
10 17.6 23.7
Note: The scale is based on the observed total fatalities in year 1996-1997 per 100,000 vehicles (1995 model year) on road
per 10,000 miles driven (average annual miles driven is 13989 miles).
11

-------
Standardized Scales for the Risk of Mortality
Low Fatality
1
2
3
4
5
6
7
8
9
10
11
3	6 9 12 15 18 21 24 27 30 33+
small sedan &
wagons
7.1 9.2 14.0
middle sedan &
wagons
4.4 6.b 9.3
large sedan & wagons
4.3 6.5 8.5
luxury sedan &
wagons
3.5 7.2 15.3
small & mid.
specialties
7.1 9.$ 16.6
luxury sports
13.4 2^3 47./^
small suv
15.5 17.1118.1
large suv
6.7 9.4 15.5
van (minivan)
4.0 5.5 7.0
small pickup
11.0 15.4 14.7
large pickup
7.3 8.fe 11.8
Note: The scale is based on predicted total fatalities per 100,000 vehicles (1995 model year) per 10,000 miles driven with 2 occupants.
12

-------
Hedonic Models (Step 2):
Parameter Estimates for the Hedonic Equations

Model A

Model B

Variable
Estimated
t ratio
Estimated
t ratio

Coefficient

Coefficient

Dependent
Fe_city

P auto

Constant
2.5689
14.13
7.7174
25.45
Value Retained
0.0549
3.35
0.4594
11.10
Mortality Rate
0.0258
1.99
-0.0690
-3.53
Injury Rate
0.0330
4.01
-0.0161
-1.31
Reliability Rating
0.0170
5.05
0.0617
5.23
Acceleration
-0.2290
-8.04
0.6014
13.99
Traditional Styling
-0.2786
-5.21
0.6035
7.56
Class2
-0.1873
-16.56
0.2426
14.34
Class3
-0.2751
-14.69
0.3734
13.28
Class4
-0.2852
-19.29
0.6752
29.76
Class5
-0.6397
-37.47
0.8127
31.94
Class6
-0.4846
-24.84
0.6558
22.67
Class7
-0.4352
-27.49
0.3398
14.31
Year91


0.1137
6.31
Year92


0.2100
10.53
Year93


0.2977
13.16
Year94


0.3880
15.30
Year95


0.4474
16.14
Ford
0.0347
1.90
-0.0972
-3.58
GM
0.0334
1.94
-0.0879
-3.44
Chrysler
0.0196
1.12
-0.1148
-4.43
Germany
-0.0562
-2.84
0.1489
5.05
Japan
0.0470
2.73
-0.0430
-1.71
MB
-0.0078
-0.33
0.5237
14.89
R2
0.7626

0.8996


-------
Estimating VSL by Age Group and Household Type (Step 3):
Note that the simple one car model can be easily extended to multiple vehicles if the last car
purchased is the most-driven, newest vehicle and is chosen subject to the constraint of prior
vehicle purchases.
In the case of multiple vehicles, the allocation of driving miles across family members and
vehicles is endogenous and reduced form equations must be used to predict this allocation and
miles ridden and driven.
Thus, our estimates of the VSL for family members uses the optimizing condition for the risk
level choice for the most-driven vehicle since other, now non-optimal, vehicles may be retained
in a family fleet because of transactions costs.
Three typical family groups own most of the total 783 vehicles used in the analysis:
1)	PA: pure adults family (424 vehicles);
2)	AK: family with both kids and adults (267 vehicles);
3)	PS: pure senior family (57 vehicles).
To address possible income effects on the VSL, we divide families into three types:
1)	Low income family: Per Capita Income<=$ 15000;
2)	Middle income family: $15000$37500.
Three no intercept OLS regressions were run, one for each of three family groups. (If we run
regressions with intercepts, the intercepts are insignificant).
The average ages for adults, seniors and kids in our data set are 39.8, 74.2 and 7.8
respectively, so the value for each group can be interpreted as the average VSL for that age.
The estimated results are inconsistent with the simple discounted present value of life-year
model.
The estimated results without intercepts are shown in the following table:
14

-------
Estimated VSL for Families
Family
Type
Income
Type
Sample
Size
Person Type*
VSL
(million)
t value
PA
Low
67
Adult low
6.81
9.37

Middle
188
Adult middle
6.07
13.63

High
169
Adult high
7.27
14.88
AK
Low
133
Adult low
3.36
8.36



Kid low
2.54
3.64

Middle
120
Adult middle
3.79
8.96



Kid middle
5.12
6.46

High
14
Adult high
-
-



Kid high
-
-
PS
Low
9
Senior low
7.67
4.60

Middle
31
Senior middle
8.42
6.85

High
17
Senior high
8.25
3.35
Note:
1. *Person Type is Defined as:
Adult low: adults from low-income families;
Adult middle: adults from middle-income families;
Adult high: adults from high-income families;
Kid low: kids from low-income families;
Kid middle: kids from middle-income families;
Kid high: kids from high-income families;
Senior low: seniors from low-income families;
Senior middle: seniors from middle-income families;
Senior high: seniors from high-income families.
2. -means insufficient sample size to obtain reliable estimates.
Fragility
It should be noted that the analysis so far has omitted an important effect that has not
previously been considered, fragility. Seniors are, on average, more fragile than adults and
kids are, on average, less fragile than adults. From the mail survey data, people's perception
of the likelihood of a 70-year-old person dying compared to an average adult when involved in
a serious accident is about 39% higher. For children, the survey data shows that the
perception of the likelihood of a 8-year-old child dying compared to an average adult when
involved in a serious accident is about 12% lower.
15

-------
Pooling the data for the different income groups and adjusting for fragility does imply that, with
the exception of parents facing the financial stress of raising children, that the VSL for kids,
adults without kids, and seniors follows the humped shaped pattern predicted by theory.
Fragility Adjusted VSL ($million) by Family Group
Age Group
Fragility
Unadjusted VSL
Fragility Adjusted
VSL
Kids(AK)
3.63
4.13
Adults(AK)
3.72
3.72
Adults(PA)
6.62
6.62
Seniors(PS)
8.44
6.07
Income Elasticity Measurements
Income elasticities can be obtained by assuming that
VSL=a+b(Y-averageY), and estimating:
MCrisk =(ak,ds + bjdds (Y-averageY))(Mkid/TVM)
"'"(^adults baduits (Y-averageY))(Maduit/TVM
(^senior bsenior
(Y-averageY))(Msemor/(TVM)
Note that the estimated coefficients for the constant term, a, are estimates of the VSL for average
per-capita income by person by family type and the income elasticity at this point is:
b(averageY)/a.
The very low income elasticities (0-.33) obtained in this study suggest that utility may depend on
many things other than money income. Recent research on the psychology of happiness suggests
that income plays a relatively minor role compared to family, friends, and work satisfaction.
Income Elasticity Estimates
Family
Type
Sample
Size
Per Capita
Income
Person
a
(million)
t
value
b
t
value
elasticity
PA
424
40776
Adult
6.67
22.28
18.19
2.05
0.111
AK
267
18709
Adult
3.59
12.25
0.62
-0.02
-0.003
AK
267
18709
Kid
3.64
6.80
65.08
1.14
0.335
PS
57
26462
Senior
8.18
8.97
7.97
0.14
0.026
16

-------
Income elasticities are obtained by assuming that VSL=a+b(Y-averageY), and estimating:
MCrisk =(ak,ds + bjdds (Y-averageY))(Mkld/TVM)
"'"(^adults baduits (Y-averageY))(Mat|uit/TVM
(^senior bsenior
(Y-averageY))(Msemor/(TVM)
Note that the estimated coefficients for the constant term, a, are estimates of the VSL for average
per-capita income by person by family type and the income elasticity at this point is:
b(averageY)/a.
17

-------
Draft - Do Not Quote or Cite
Willingness to Pay for Reduced Accident Risk for Children:
Inferences from the Demand for Bicycle Safety Helmets
Robin R. Jenkins
Nicole Owens
Lanelle Bembenek Wiggins
November 6, 2003
Abstract: This paper develops a household production model in which parents produce
bicycling safety for their children. Using data from the National Survey on Recreation and the
Environment, via a random utility model, we estimate conditional indirect utility as a function of
bike safety and infer WTP for reduced risk of fatal and non-fatal head injury. We estimate
parental values for reducing biking risks faced by their children. We obtain estimates of parental
values for children that include a VSL of $9.5 million and a VSI of $7.0 million.
* Any opinions, findings, and conclusions or recommendations expressed in this publication are
those of the authors and do not necessarily reflect the views of the Environmental Protection
Agency. The authors are grateful to Mark Agee, Amy Ando, Nishkam Agarwal, Charles
Griffiths, Julie Hewitt, Carol Mansfield, Salvador Martinez and Shannon Sullivan for their help
and comments. Any errors are our own.
Robin R. Jenkins: Economist, United States United States Environmental Protection Agency,
Office of Policy, Economics, and Innovation, National Center for Environmental Economics
phone: 202-566-2292
fax: 202-566-2338
email: ienkins.robin@epa.gov
Nicole Owens: Economist, United States United States Environmental Protection Agency,
Office of Policy, Economics, and Innovation, National Center for Environmental Economics
phone: 202-566-2297
fax: 202-566-2338
email: owens.nicole@epa.gov
Lanelle Bembenek Wiggins: Economist, United States United States Environmental Protection
Agency, Office of Policy, Economics, and Innovation, National Center for Environmental
Economics
phone: 202-566-2372
fax: 202-566-2338
wiggins.lanelle@epa.gov
18

-------
1. Introduction
Improving children's health is a relatively new federal priority. The Clinton
Administration's Executive Order (E.O.) 13045 directs policy makers to examine and reduce
health and safety risks to children. This directive has led to a new need for more accurate
measures of the benefits of policies that improve children's health; more specifically, for better
estimates of the economic value of reducing childhood risks. To date, the economics literature
contains only a handful (albeit a growing handful) of such estimates, whereas it contains a
multitude of estimated values of reducing risks to adults. Thus for policy applications, at
present, analysts must choose between two practical but conceptually lacking alternatives for
child health values. The first is to rely on estimates of the medical costs associated with an
illness. The second is to transfer estimates of willingness-to-pay (WTP) for risk reductions
estimated for adults to child populations.
The first approach has been labeled "cost-of-illness" and usually involves estimates of
direct medical expenditures and of the more indirect cost due to lost work time (or for children,
future lost work time). Conceptually, the cost-of-illness approach simply measures ex post costs
and does not attempt to measure the loss in utility due to pain and suffering or the costs of any
averting behaviors that individuals have taken to prevent an illness. Some consider a cost-of-
illness estimate to be a lower bound estimate of WTP because it fails to account for many effects
of disease, such as lost leisure time or pain and suffering (Harrington and Portney 1987; Berger
et al. 1987). Others suggest that cost-of-illness might exaggerate risk values since in some cases
the cost of averting behaviors that prevent a medical condition can be far less than the ex post
costs of treatment (insert cite). Economists widely recognize that the preferred measure to assess
the benefits of federal policy is to estimate WTP for ex ante risk reduction rather than using cost-
of-illness estimates (U.S. EPA 2000).
Due to a lack of WTP estimates for children, at present analysts routinely transfer WTP
estimates for adult risk reductions to child populations. The appropriateness of these transfers is
questionable (Dockins et al. 2002; Agee and Crocker 2003). Researchers are currently asking
whether a risk reduction of the same character and size should be valued differently when
experienced by children as compared to adults. This paper is a first step in shedding light on this
issue by estimating parental values for reducing the risk of a bicycle injury to their children. In a
future paper, the authors will estimate adult values for reducing the same risk to themselves and
compare the values.
Many of the estimates of adult health and safety values have been derived via hedonic
wage analyses. For obvious reasons, this methodology is not viable for analysts focused on
children or for analysts seeking insight regarding the differences between adults and children. A
valuation alternative that does hold promise, however, is analysis of safety product markets.
19

-------
Of particular promise is the bicycle helmet market. A bike helmet is a personal safety
product whose ownership is generally assigned to a single individual, not to a family or some
other group which would render it impossible to assign the benefits of the safety product to one
person. In addition, bike helmets are owned by young and old alike leaving open the possibility
of discerning a relationship between age and willingness-to-pay for safety. This paper and future
work will take advantage of these desirable attributes of the bicycle helmet market by examining
households' purchase decisions regarding helmets for adults and children. We develop a
household production model in which adults produce bicycling safety for themselves or parents
produce it for their children. Via a random utility model, we estimate conditional indirect utility
as a function of bike safety and infer WTP for reduced risk of fatal and non-fatal head injury.
We estimate parental values for reducing biking risks faced by their children and, in the future,
we will estimate adult values for reducing their own risks.
Data were obtained from a telephone survey that was part of the most recent National
Survey on Recreation and the Environment. Respondents can be separated into two groups:
parents who report having a child age 5 to 14 who had bicycled within the previous 12 months,
and adults age 20 to 59 who report having bicycled themselves within that same time frame.
In addition to socioeconomic information, we have information on the amount of bike-riding, the
perception of helmet laws, the importance of helmet features and the price paid for the helmet.
Data on the risk of bicycling is obtained from the Centers for Disease Control (CDC) and varies
according to age, gender and race.
Previously in the economics literature, analyses of safety product markets have estimated
the value of risk reduction by assuming that the marginal benefit of risk reducing activities
equates with their marginal cost. These papers have lacked price information and have based
estimates of the value of risk reduction solely on estimates of implicit values (and amounts) of
time and/or on estimates of monetized dis-utility (Blomquist 1979 and 1991; Blomquist, Miller
and Levy 1996; Carlin and Sandy 1991). Other product market analyses estimate lower bound
values of risk reduction based directly on highly aggregated product prices (Dardis 1980;
Garbacz 1989; Jenkins, Owens and Wiggins 2001). The current paper adopts a different
approach, one developed outside the safety product literature. Dickie and Gerking (1991) and
Agee and Crocker (1996) develop household production models in which utility depends upon
health or risk of family members. The household makes many unobserved choices regarding the
production of health or risk but there is one observable discrete choice, such as whether or not to
purchase medical care. The probability of the discrete choice is estimated via a random utility
model with which welfare effects can be computed (Small and Rosen 1981). For the current
paper, the discrete choice is whether or not to purchase a helmet.
To estimate how consumer WTP for risk reduction varies with the age of the beneficiary
requires an ability to discern the age of the beneficiary. Previous analyses have examined
spending on safety products that benefit an entire household — smoke detectors (Dardis 1980,
Garbacz 1989) and automobile size (Mount et al. 2000) — or that benefit only children or only
20

-------
adults — car safety seats and motorcycle helmets (Carlin and Sandy 1991; Blomquist 1991;
Blomquist, Miller and Levy 1996). Our analysis is unique in that bicycle safety helmets are used
by all age groups but are purchased for specific individuals. This allows us to estimate
separately the WTP for bicycle safety for children and adults.
The choice of whose preferences to rely upon to determine the value of childhood risk
reductions is an important one. Dockins et al (2002) suggests that the parental perspective is
advantageous for multiple reasons.1 The current paper examines parental decisions regarding
bicycle safety, specifically regarding the purchase of a child bicycle helmet. Also reported by
parents are other variables in the demand function for helmets, such as the perception of helmet
laws and the amount of time a child spends riding. Thus, for children's safety, we estimate a
parent-determined WTP.
The following sections of the paper develop a model of household production and then
translate propositions from the model into empirically testable form. We describe the data that
we analyze and the tentative results that we obtain via a logit model of the purchase decision.
2. Household Production Model
This section presents a household production model of utility from which we derive the
compensated demand for bicycle safety helmets. The household perspective is chosen in order
to represent helmet purchase decisions made by parents for their children. However, the model
can be easily adjusted to represent adults making decisions only for themselves, without explicit
consideration of children. The model will illuminate the important underlying variables in the
discrete choice decision of whether or not to purchase a helmet. The model also provides
structure for the estimation of risk valuation. We derive from the model an equation to represent
adult willingness to pay for own risk reduction as well as parental willingness to pay for child
risk reduction. This section draws heavily from household production and random utility models
developed by Dickie and Gerking (1991), Agee and Crocker (1996) and Agee, Crocker and
Shogren (2001), which in turn drew from Small and Rosen (1981).
Parents derive utility, Up, from consumption of commodities and activities, Z/(, produced
for themselves, and from the risk they perceive themselves as facing by riding a bicycle, Rp.
Parents also derive utility from commodities and activities, Zc, produced for each of their i = 1,
..., n children and from the risk they perceive their children face, Rc, from bicycling. If we
represent a single-period, two-generation family in which children's utility is additive to parents'
utility but separable, then:
'Four possible perspectives for valuing children's risk reductions are suggested by
Dockins et al. (2002): that of society, the child, adult-as-child, and parent.
21

-------
n
11P = UP (ZP> RP ) + Z apr (y >cr (Zc, ,RC, \	(!)
7=1
where U'z >0 and U'R <0. Up(.) is quasi-concave and increasing in at least some of the Z's, and is
finite whenever some of the Z's are zero (Small and Rosen 1981). Parents combine their time,
effort and market good purchases to produce child-related commodities, Zc. The up and uc
functions differ because children and their parents experience their environments differently.
The multiplier api{) converts child /' s utility into his parents' utility and depends on y which
represents family characteristics that affect the conversion of childhood utility into parental
utility.
Parents also combine their time, effort and market purchases to affect the level of risk, R,
faced by themselves and their children from riding bicycles:
where D represents the amount of time spent riding a bicycle, H represents the use of a bicycle
safety helmet, b is the level of risk per unit of riding time, assuming no helmet-wearing, that is
expected for the rider and varies according to the risk taking behavior of the individual, and y
represents any family characteristics, such as parents' educations, which may influence risk
perception. As mentioned already, helmets are unique in their ability to reduce by significant
proportions the risks of injury and death from bicycling. Spending on helmets produces nothing
of value other than reductions in R.
Let q denote a vector of market prices for commodities and activities; t denote a vector of
parental time inputs, and ts denote time spent away from paid work. Then the parental income
constraint can be written as a sum of expenditures on own and children's consumption,
where = (7/; + w/,);/ p, c, hp, hc\ and w = Y/(T-ts) so that parents' income, Yp, determines
their opportunity cost of time, w. Parents choose lr Rp, Zp, Rc and Zc to maximize total utility in
(1) subject to (2) and (3).
Assume that parental WTP is the largest income that parents must forego after a
reduction in expected risk, to maintain ex ante expected utility. Parental WTP estimates the
value to the household of a reduction in the level of risk, b, that the rider is expected to face. Let
Vp(.) denote the parents' indirect utility function from the above utility maximization problem.
R^H^r)
(2)
yp = rPZP + rhPHP+ X (rc,zc, + n,c,Hc)
(3)
22

-------
Given the properties of expression (1), Vp(.) is continuous and strictly increasing in household
income, Yp, and thus can be inverted to find the expenditure function, e[.], satisfying
Differentiating (4) with respect to b yields adults' or parents' marginal willingness to pay
for a reduction in the expected riskiness in either their own or their child's bike riding activity.
where X =dVJdYp is the marginal utility of income.
Expression (5) portrays parents' marginal disutility of the expected risk of bicycling
converted to monetary units via the marginal utility of income. In general, this measure is
empirically intractable because actual utility levels are not observed. However, an empirical
representation is available via a discrete choice model of the decision to purchase a bicycle
helmet.
The household production model portrayed in (1) through (5) can be adjusted to represent
adults making decisions for themselves without explicit consideration of children by assuming
Zci = 0 and Rci = 0 for all i.
3. Empirical Model
A parent's decision to purchase a helmet or not is a discrete choice based upon
information about the risk reduction provided by helmets. Let vH denote the maximum
attainable expected utility if a helmet is purchased and let vQ denote the maximum attainable
expected utility if a helmet is not purchased. For households characterized by b and y, the choice
to purchase a helmet or not is made by comparing these two expected utility levels, given
income, and a wage-price vector, r = (w, q)\
H - 1 if vM - vn > 0
(6)
H - 0 otherwise.
The utility difference is specified econometrically as
UP = Vp(r,D,b,r-,e[r,D,b,r^-
(4)
MWTPb = & / cb = -(1 / X)fflp / cb,
(5)
vh(-)-vo(-)= x'fi+ £
(7)
23

-------
where Xis a vector whose first element is unity and whose remaining elements measure
arguments of the conditional utility function in (4), is a parameter vector and e is a random
error component. The probability of purchasing a safety helmet, conditional on e, is
Pr(//= 1) = F(X'J3+ s\	(8)
where H= 1 if a helmet is purchased and 0 otherwise, and F(.) is the symmetric distribution of V
conditional on s.
Let X' P be the inner product of explanatory variables and estimated coefficients, with
each explanatory variable except risk set equal to its sample mean. Assume that e is distributed
standard logistic and note that bicycle expenditures are a small part of the family budget. Then,
following Small and Rosen (1981) if the compensated demand for a bicycle helmet is
approximated by its Marshallian counterpart, parents' MWTPh in expression (5) is approximated
by
MWTPb = -{Pb/X)F{Xp\	(9)
where is the estimated coefficient for risk, /. is the estimated marginal utility of income,
F(.) is the standard logistic cumulative distribution function and /*( X' ft ) is the estimated
probability of purchasing a helmet.
Helmets seem essential to reducing the risk of head injury from bicycle riding thus the
concern shown by Bockstael and McConnell (1983) that (9) will yield an incomplete measure of
the true MWTPb in (5) is lessened.
4. Data Description
The primary data source for this work is the 2000 National Survey on Recreation and the
Environment (NSRE), conducted by the U.S. Department of Agriculture's Forest Service. Other
data we relied upon includes income and population data from the U.S. Census Bureau, weather
data from the National Climatic Data Center, and pedal cycle injury and death statistics from the
Centers for Disease Control and Prevention (CDC).
The NSRE is a random-digit-dialed phone survey of U.S. residents over age 16. The
survey collected information from the American public on demographics, participation in a
multitude of outdoor activities, and opinions concerning environmental and natural resource
24

-------
issues. Between July 2000 and July 2001, the NSRE asked respondents questions related to
bicycling, especially regarding bicycle helmet purchases and use. Respondents were either
asked a series of questions related to their own bicycle helmet purchasing decisions (adult
module) or, if the respondent had a bike riding child between the ages of 5 and 14, questions
related to purchasing decisions for that child's bicycle helmet (child module). Respondents were
asked about the amount of bike riding they (or their child) did, their beliefs regarding the
existence of helmet laws, the price they or another family member paid for their (or their child's)
helmet, the factors influencing their choice of helmet, their (or their child's) expected helmet use
patterns at the time of purchase and a question to determine if the respondent would have
changed their helmet purchase decision after being given accurate information on the risk
reduction provided by helmets.
In order to maximize the number of responses to the bicycle helmet modules subject to a
constraint on the length of each interview and because of the anticipated difficulty contacting
respondents with bike riding children of an appropriate age, most of the respondents were asked
the child module first. The first question in the child module asked if the respondent has a bike
riding child between the ages of 5 and 14. An affirmative answer to that question led to the
remaining questions in the children's bike helmet module. If the respondent did not have any
bike riding children of an appropriate age, the questions in the adult bike helmet module were
asked. A concern that we were not getting responses to the adult questions from any parents
who had bike riding children led to approximately 100 interviews in which respondents with
bike riding children were also asked the adult questions.
The initial data set included 15,010 observations.2 After eliminating observations for
respondents who did not ride a bicycle in the past year or have a bike riding child between the
ages of 5 and 143, the samples contained 2,463 respondents with a bike riding child between the
ages of 5 and 14, and 1,493 adult respondents who rode bikes themselves. Observations with
missing data values for variables included in our regression analysis were eliminated, as were
observations where the respondent (or her child) had a helmet, but the helmet was not purchased
by herself or an immediate family member.4 In order not to lose those observations where
2The data set comes from versions 5, 7, and 9 of the NSRE which contain the adult and
child bicycle helmet modules.
Observations were eliminated from the data set if income was greater than $4 million
(this eliminated the few income observations that were greater than 3 standard deviations from
the mean); if the respondent answered that his or her helmet would last over 50 years; if the
respondent was less than 19 years old; and for the child questions, if the parent age minus the
child's age was less than 15.
4In this situation, respondents were not asked further helmet questions because it is
unlikely they would have known the helmet purchase price or other factors that went into the
25

-------
household income was the only relevant variable that was missing, we used data from the 2000
Census5 to create a proxy income variable. Proxy income is equal to the median family income
by race for the zip code in which the respondent lives. If the respondent lived alone or in a
house with roommates, proxy income is equal to the mean individual income by race for his or
her zip code. The final data set used for our analysis includes 1,984 child observations and 941
adult observations. Means and standard deviations of the data for children are summarized in
Table 1.
Of the 1,984 child observations, 89.5% were helmet purchasers. These numbers are
similar to a 1999 U.S. Consumer Product Safety Commission (U.S. CPSC 1999) survey that
found 84% of bike riding children under 16 own a helmet.
About 12% of the U.S. population is covered by state or local helmet laws (BHSI2001).
All 20 of the state laws are specific only to children and 30 of the 83 local laws apply to all ages,
with the others being specific only to children. Interestingly, 52% of the respondents in our
child sample said that there was a law in their community or state requiring children to wear
bicycle helmets and 17% said they did not know whether there was a law applicable to children.
People may believe that a helmet law exists in their community when they are exposed to a
helmet education campaign. For example, McDonald's Corporation ran a national campaign
encouraging helmet use for children and adults. Whether the respondents are correct in their
knowledge of helmet laws in their community or not, it is their perception of the law that will
drive their helmet purchasing decisions.
The federal safety standard for bicycle helmets (U.S. CPSC 1998) ensures that all bicycle
helmets manufactured after March 10, 1999 must meet a minimum level of safety. It is unlikely
that manufacturers would create helmets that go too far beyond this standard. To make a helmet
safer than the federal standard would require additional cost to the manufacturer, but also more
weight and size to the helmet making it less likely to be bought or worn (U.S. CPSC 1998).
Even though helmets themselves do not differ significantly in their levels of protection, different
levels of risk-taking behavior or exposure to risk during riding will cause individuals to face
different risk reductions provided by their helmets. The CDC reports annual pedal cycle deaths
and injuries by age, race, and gender. We combine this data with information on population and
the percent of population that rides a bicycle in order to assign a fatal and non-fatal risk measure
to each individual in our sample that is equal to the average for that individual's age-gender-race
purchase decision.
5Data was obtained from Census 2000 Summary File 1 and Census 2000 Summary File 3
(http://www.census.gov/main/www/cen200Q.html - accessed May - June 2003).
26

-------
group.6
5. Empirical Implementation
We estimate a purchase equations representing parents' purchases of helmets for
children. The indirect utility function in (4) suggests that the purchase decision depends on a
variety of variables including the wages of the family and the price of the helmet. We combine
these two variables and include in the equation one variable measuring household income less
the helmet price. To measure the amount of time spent riding, D, we include a variable
indicating the number of days ridden by the bicycler during the previous 30 days. Since the
prevailing weather during the month in which the survey was administered would influence the
number of days a bicyclist might ride, we also include the average temperature during that month
and a term that interacts avidity with temperature. We include separately both the rate of death
and the rate of injury to measure the risk of bicycling. To represent the attributes of families, y,
that either affect the conversion of childhood utility into adult utility or affect risk perception we
include a variety of socioeconomic information: age, gender, race and education of the bicycler
and/or his parents. (For the child equation we substitute parent's education for rider's education
and we include an indicator variable for whether the parent respondent rode. For age, we include
both the parent's age and the child's.) Finally, we include two indicator variables that indicate
whether the respondent believed that there was a helmet law requiring use or whether the
respondent was unsure.
Table 2 presents the results of four logit models of the decision to purchase a helmet for
children and adults. The first column gives the primary results. The sign of the coefficient on
the child's age is positive, in agreement with the burgeoning consensus among studies targeted at
children that parent's valuation of risk reduction varies inversely with child age (Agee and
Crocker 2003). The suggestion is that as a child ages through middle childhood, the probability
that a bike helmet will be purchased for him declines. Similarly, parents of male children are
less likely to purchase a helmet. This could indicate a greater parental acceptance of risky
behaviors undertaken by boys compared to girls or undertaken by older children compared to
younger ones. Alternatively, parents of boys and older children might have lower expectations
regarding their children's compliance with parental wishes for the child to wear a helmet.
Parents would naturally be reluctant to purchase a helmet that they believe it will not be worn.
The probability that a helmet will be purchased for a child increases if the respondent
believed there was a law requiring helmet usage or if the respondent was unsure about the
presence of a helmet law. This finding bodes well for the recent dramatic increase in popularity
of helmet laws for youngsters. Parents really are responding to such laws. However, parents
6For the child observations, we assume that the race of the child is the same as the race of
the respondent.
27

-------
also respond positively to uncertainty about the existence of a law.
As expected, greater household income positively affects the probability that a parent
will purchase a helmet for her child as does greater education level. The indicator variables for
black and white race are significant and negative. Relative to households of other races (Asian,
Pacific Islander, American Indian and others) these households are less likely to purchase a
helmet for their child. The indicator variable for whether or not the parent rides a bike is
significant and positive suggesting a greater awareness of bike safety issues by parents who
themselves bike.
A parent's age is not correlated with the probability of purchasing a helmet. The
indicator variable for whether or not the parent rides a bike is significant and positive suggesting
a greater awareness of bike safety issues by parents who themselves bike. As expected, the rate
of death and injury faced by the child as a consequence of bicycling is positively and
significantly associated with the probability of purchasing a child's helmet.
In addition to the primary model described above, we estimate three additional logit
models. To check the sensitivity of the results to the rather rough measure of avidity represented
by the number of days ridden and the temperature variables, we re-run the logit and omit those
three variables in specification (2), and omit just the two temperature variables in specification
(3). The results are robust. Finally, we wished to include measures of the importance to the
respondent of the helmet's appearance and comfort level. We include two variables indicating
whether the respondent believed these features of the helmet were important in specification (4)
and find that neither is significant, nor does their inclusion alter substantially the coefficients
estimated for the remaining variables.7
Our principal purpose is to approximate parental WTP for child risk reduction. To do
this we estimate for each respondent the percentage change in income necessary to keep utility
constant when bicycling risk is reduced by one percent. This is achieved by evaluating equation
(9) and converting to percentage terms. For specification (1) the resulting values of statistical
life and injury for children are $9.5 million and $7 million, respectively. These estimates vary
between $8.9 and $9.9 million for VSL and $7 and 8.4 million for VSI among the four
specifications.
6. Discussion
These estimates of VSL for children are higher than most VSL estimates reported in the
7The appearance and comfort variables are constructed with responses to questions in the
survey regarding the importance of comfort and appearance and with the days ridden variable
thus we omit direct measures of days ridden and temperature from specification (4).
28

-------
literature for adults. A good summary estimate of a set of high quality, policy relevant adult
VSL studies is provided by the EPA. To analyse proposed regulations, EPA relies on a VSL
estimate of approximately $6 million (in 2000 dollars). This estimate was derived by fitting a
Weibull distribution to 26 adult VSL studies (21 that use the hedonic wage method and five that
examine stated preferences). The suggestion is that parents value reductions in risk to their
children by more than adults value reductions in risk to themselves. However, this suggestion is
made with caution since there are important differences between the nature of the risk being
examined and the valuation estimation methodology in the current study compared to the adult
studies. In the future, we plan to estimate adult willingness to pay for reductions in bicycle risk
and will more confidently make direct comparisons to our estimates for children.
Our VSL estimates for children are quite high relative to the two values for children
found in the published literature: between $0.75 million (Carlin and Sandy 1991) and $4.0
million (Jenkins, Owens and Wiggins 2001)8 However, these two studies were examinations of
direct time and/or money expenditures on safety products. So, again the methodology is
different enough to suggest caution in making comparisons.
To get an idea of the relative magnitude of the VSI estimates, we gathered information
about the medical costs of non-fatal bicycle injuries in the U.S. A review of hospital discharge
data in Washington state (1989-1991) found that treatment for nonfatal bicycle injuries among
children ages 14 and under costs an average of $218,000 per injured child (Bicycle Helmet
Safety Institute 2003). A cost of illness (COI) estimate would add to these direct costs, such
indirect costs as the value of the parent's lost work time from caring for the sick child and the
value of future lost wages due to any brain injury or long term debilitating injury to the child.
Even after accounting for these indirect costs, the COI of non-fatal bicycle injury would be far
less than our VSI estimates. Dickie and Gerking (1991) also obtain estimates of WTP that are
higher than corresponding medical costs. Using a similar model and empirical method as in the
current paper, the estimates of WTP for ozone control turn out to be about double the medical
expenses associated with treating respiratory illness that is associated with ozone pollution.
While the magnitude of the difference is much smaller, these findings and ours give examples
when estimates of WTP are substantially higher than comparable COI estimates. The
unmonetized costs such as pain-and-suffering might be the explanation.
On the other hand, the opposite is suggested by the WTP estimates inferred from demand
functions for chelation therapy to reduce child lead burdens. Agee and Crocker (1996) estimate
parental WTP for a 1 percent reduction in child lead burden as falling between $11 and $104.
Lutter (1994) converts these WTP estimates into estimates of the value of a lost IQ point and
obtains values that range from $1,100 to $1,900. Lutter compares these parental WTP estimates
8These estimates are in 1997 dollars. The estimate for Jenkins, Owens and Wiggins
(2001) is the upper limit of a range beginning at $1.1 million.
29

-------
to government COI estimates of lost income due to lowered IQ and finds the latter to be much
higher, approximately $8,800 per lost IQ point.
Dockins et al. (2002) attribute this difference to the fact that the WTP estimates represent
the parental viewpoint while the COI estimates represent a lower bound of what a child should
be WTP him or herself (if lending constraints were relaxed). Our own paper suggests that
parental WTP for children is actually much higher than COI. The nature of the risks being valued
by the two studies is quite different. Children with high body burdens of lead exhibit long-term
cognitive and adaptive behavior deficits. In the short term, effects include hyperactivity, poor
attention and learning problems. Risks of bicycling include catastrophic brain injury, concussion
and contusion. A second important difference between the two studies is the education and
income levels of parents in the study sample. Parents in the Agee and Crocker (1996) study had
an average education level of 11 years and an average income of only $17,000 (1985 dollars).
Almost 70 percent of our sample went to college and they earn an average of $60,000 (2000
dollars). Thus the Agee and Crocker paper examined a risk that imposes intangible effects, the
most devastating of which are in the distant future, and estimated the WTP to reduce this risk
among relatively low income parents. The current paper examines a risk that poses a dramatic
immediate physical threat and estimates WTP among relatively high income parents. In light of
these difference, the larger gap between WTP and COI for non-fatal bicycle injury is easier to
understand.
In a future version of this paper, we will estimate a logit equation representing adult's
helmet purchase decision for self. This will enable us to compare values of risk of the same
nature and similar magnitude for children and adults.
30

-------
Table 1

Means of Variables for Child Bicyclists
(Standard deviations are in parentheses)

children

(n=1984)
Own
0.895

(0.307)
Age
9.513

(2.871)
Parent's Age
38.855

(7.650)
Male
0.534

(0.499)
Helmet Law - Yes
0.519

(0.500)
Helmet Law - Don't Know
0.171

(0.377)
Household Income Minus Price($1000)
58.247

(39.635)
Black
0.088

(0.284)
White
0.887

(0.316)
Parent Rides
0.583

(0.493)
Highschool
0.284

(0.451)
College
0.671

(0.470)
Days Ridden
11.008

(10.937)
31

-------
Fatal Head Injury Risk
5.300 E-6

(3.669 E-6)
Non-Fatal Head Injury Risk
3.083 E-3

(1.488 E-3)
Monthly Mean High Temperature (degrees)
62.413

(20.974)
32

-------
Table 2
Econometrics Results for Logit Model of Purchase Decision for Child
Variable
(1)
(2)
(3)
(4)
Constant
1.225
(1.030)
0.573
(0.913)
0.898
(0.958)
0.338
(0.924)
Age
-0.122**
(0.048)
-0.093**
(0.045)
-0.120**
(0.046)
-0.082*
(0.046)
Parent's Age
0.014
(0.012)
0.021*
(0.011)
0.020*
(0.011)
0.019*
(0.011)
Male
-1.848**
(0.735)
-1.858***
(0.694)
-1 942***
(0.715)
-1.900***
(0.699)
Helmet Law - Yes
1.905***
(0.195)
1 877***
(0.183)
1.866***
(0.188)
1.887***
(0.184)
Helmet Law - Don't Know
1.012***
(0.224)
0.946***
(0.208)
0.896***
(0.212)
0.956***
(0.209)
Household Income Minus
Price ($1000)
0.01***
(0.003)
0.01***
(0.003)
0.01***
(0.003)
0.01***
(0.003)
Black
-2.578**
(1.035)
-2.882***
(0.976)
-3.00***
(1.002)
-2.802***
(0.972)
White
-1.528*
(0.882)
-1.903**
(0.828)
-1.912**
(0.853)
-1.823**
(0.828)
Parent Rides
0.631***
(0.164)
0.551***
(0.154)
0.570***
(0.158)
0.540***
(0.155)
Highschool
0.764**
(0.319)
0.673**
(0.297)
0.815***
(0.304)
0.712**
(0.302)
College
1.303***
(0.323)
1 247***
(0.298)
1.298***
(0.305)
1.301***
(0.304)
Fatal Head Injury Risk
199886**
(82924)
195097**
(78693)
210878***
(80635)
189992**
(79207)
Non-Fatal Head Injury Risk
254.2**
(129.4)
286**
(122.4)
276.3**
(125.2)
305.9**
(123.1)
33

-------
Table 2
Econometrics Results for Logit Model of Purchase Decision for Child
Variable
(1)
(2)
(3)
(4)
Days Ridden
-0.016
(0.027)

-0.001
(0.007)

Temp
-0.009*
(0.005)



Days Ridden * Temp
0.0003
(0.0004)



Appearance Factor



9.810
(863.3)
Comfort Factor



13.398
(825.1)
Number of observations
1984
2159
2103
2114
Likelihood Ratio
232.724
238.397
233.163
247.622
34

-------
References
Agee, Mark D. and Thomas D. Crocker. 1996. "Parental Altruism and Child Lead Exposure:
Inferences from the Demand for Chelation Therapy," The Journal of Human Resources.
Agee, Mark D. and Thomas D. Crocker. 2003. "Transferring Measures of Adult Health Benefits
to Children: Some Issues and Results," presented at the Western Economic Association
International Annual conference, Denver, Colorado, July 13, 2003.
Agee, Mark D., Thomas D. Crocker and Jason F. Shogren. October 2001. "A Hicksian
Approach to Assessing Welfare Effects of Child Physical Abuse using Discrete Choice Models,"
Working Paper.
Berger, M.C., G.C. Blomquist, D. Kenkel, and G.S. Tolley. 1987. Valuing Changes in Health
Risks: A Comparison of Alternative Measures. The Southern Economic Journal 53: 977-984.
Bicycle Helmet Safety Institute, "Mandatory Helmet Laws", last revised October 16, 2001.
(http://www.bhsi.org/mandator.htm) accessed on 10/23/01.
Bicycle Helmet Safety Institute (http://www.hgsi.org/stats.htm) 2003.
Blomquist, Glenn. 1979. "Value of Life Saving: Implications of Consumption Activity,"
Journal of Political Economy, 87:540-558.
Blomquist, Glenn. 1991. "Motorist Use of Safety Equipment: Expected Benefits or Risk
Incompetence?" Journal of Risk and Uncertainty, 4:135-152.
Blomquist, Glenn C., Ted R. Miller, and David T. Levy. 1996. "Values of Risk Reduction
Implied by Motorist Use of Protection Equipment: New Evidence from Different Populations."
Journal of Transport Economics and Policy, January 1996.
Bockstael, N. and K. McConnell. 1983. "Welfare Measurement in the Household Production
Framework." American Economic Review , 73:806-14.
Carlin, P. and R. Sandy. 1991. "Estimating the Implicit Value of a Young Child's Life,"
Southern Economic Journal, 58:186-202.
Consumer Reports Online, "Head Cases", June 1999.
(http://www.consumerreports.org/main/detail.jsp?CONTENT%3C%3Ecnt_id=34975&FOLDER
%3C%3Efolder_id=34843&bmUID= 1004378401378) accessed on 9/19/01.
Dardis, R. 1980. "The Value of a Life: New Evidence from the Marketplace," American
35

-------
Economic Review, 70:1077-1082.
Dickie, Mark and Shelby Gerking. 1991. "Willingness to Pay for Ozone Control: Inferences
from the Demand for Medical Care," Journal of Environmental Economics and Management.
Dockins, Chris, Robin R. Jenkins, Nicole Owens, Nathalie B. Simon and Lanelle Bembenek
Wiggins. 2002. "Valuation of Childhood Risk Reduction: The Importance of Age, Risk
Preferences and Perspective," Risk Analysis, 22 (2): 335-46.
Garbacz, C. 1989. "Smoke Detector Effectiveness and the Value of Saving a Life," Economics
Letters, 31:281-286.
Harrington, W. and P.R. Portney. 1987. Valuing the Benefits of Health and Safety Regulation.
Journal of Urban Economics 22: 101-112.
Jenkins, Robin R., Nicole Owens and Lanelle Bembenek Wiggins. 2001. "Valuing Reduced
Risks to Children: The Case of Bicycle Safety Helmets," Contemporary Economic Policy,
19(4):397-408.
Lutter, Randall, and John R. Morrall III. 1994. "Health-Health Analysis: A New Way to
Evaluate Health and Safety Regulation." Journal of Risk and Uncertainty, vol. 8, pp. 43-66.
Mount, T. W. Schulze, W. Weng, H. Kim and L. Chestnut. 2000. "Valuing Reduced Risk for
Households with Children or the Retired," presented at the American Economics Association
Annual Convention, Boston MA.
Small, Kenneth A. and Harvey S. Rosen. 1981. "Applied Welfare Economics with Discrete
Choice Models." Econometrica 49: 105-30.
U.S. Consumer Product Safety Commission. 1998. "16 CFRPart 1203: Safety Standard for
Bicycle Helmets; Final Rule." Federal Register, vol. 63, no. 46, pp. 11711-11747 (March 10,
1998).
U.S. Consumer Product Safety Commission. 1999. "National Bike Helmet Use Survey."
(http://www.cpsc.gov/library/helmet.html) accessed on 9/18/01.
U.S. Census Bureau. 2001a. "U.S. Population Estimates by Age, Sex, Race, and Hispanic
Origin: 1980-1999." (http://vvvvvv.census.uov/population/estimates/nation/natdoc rl.txt)
accessed on 8/1/01.
U.S. Census Bureau. 2001b. "Profile of Selected Economic Characteristics: 2000."
(http://factfinder.census.gov/servlet/QTTable?ds_name=ACS_C2SS_EST_G00_&geo_id=01000
US&qr_name=ACS_C2SS_EST_G00_QT03) accessed on 10/24/01.
36

-------
U.S. Centers for Disease Control and Prevention. 2001. "Injury Mortality Reports-Pedal
Cyclist Traffic-Related Deaths and Rates Per 100,000: 1994-1997."
(www.cdc.gov/ncipc/data/us9794/Mvpctr.htm) accessed on 8/1/01.
U.S. Environmental Protection Agency. 2000. Guidelines for Preparing Economic Analyses.
EPA 240-R-00-003, pp 81-83.

-------
Valuing Environmental Health Risk Reductions to Children Workshop
Session IV Discussant Comments: Age-Specific Value of Statistical Life Estimates
Kelly Maguire
US EPA, National Center for Environmental Economics
October 21, 2003
The two papers presented in this session provide empirical evidence using revealed
preference methods to estimate to values associated with risk reductions to children. Thus far,
we have heard presentations regarding theory and conceptual models that may be used to
determine how children's health risk reductions can be valued and whose preferences should be
used to elicit such values. Today, we are turning our attention to empirical applications.
In terms of policy, while EPA does not currently use age adjustments in our formal
benefit-cost analyses, the research in this area can help inform policy decisions by providing
information on how children's risks are valued relative to risks to adults. Such information can
be used to prioritize decisions and highlight areas where new decisions might be needed
regarding risk reductions.
Both the automobile and bicycle helmet studies presented in this session rely on revealed
preference methods in which the analyst examines trade-offs between price and risk, or risk
reduction, for a market good to infer how people might value the risk reduction. As we know,
our traditional tools for valuation are often difficult to apply to children who do not control a
budget and may or may not behave in a rational manner. In addition, there may be issues
associated with asking a parent their willingness to pay for their own child in a stated preference
study because of the emotions associated with that question. Product market studies, such as
those presented in this session, hold promise because we can explore what parents spend on
safety for their children, thus using real market transactions by the decision-maker to infer
values.
In Bill Schulze's paper the "good" is an automobile, or more specifically, automobile
38

-------
safety. The idea is that children ride in cars that have various safety ratings. By looking at the
choices parents make with regards to car purchases and time spent in the car the authors infer
values of a statistical life for a child, as well as other family members.
They use a Nash cooperative bargaining model to motivate the decision-making, where
investments in safety are a function of parent and child utility. They then conduct a
telephone/mail survey where they ask people about the kinds of cars they drive and how many
miles each member of the household spends in each car. This information is combined with
safety ratings for the various cars to infer VSLs for adults, children, and various types of
households.
The good in this study is well-defined and there is very accurate and detailed information
regarding the safety of automobiles. Unfortunately, people likely value their cars for more than
just safety. When deciding what car to purchase safety is likely to be a factor, along with
comfort, fuel economy, and type. If these features of the car purchase decision are not carefully
controlled then the authors may be over-estimating the VSL by attributing all of the spending to
safety.
In the bike helmet study, the "good" is a bicycle helmet, which is used to protect against
fatal risks and non-fatal injuries. The authors use a household production model to motivate the
decision-making and a telephone survey where people are asked how much money they spent on
a helmet and other information regarding its use. They combine this information with death and
injury data to estimate a value of a statistical life and injury to a child.
The good in this study is also well-defined. In fact, the primary purpose of the good is
safety for a specific member of the household. For these reasons it is not necessary to tease out
the usage by various family members, as in the automobile study or with other goods that are
jointly consumed. In addition, because the primary purpose of the good is safety it is plausible
to attribute most, if not all, of the spending to risk reduction. While some of the spending may
be for comfort and style, it is probably difficult, if not impossible to tease out these individual
39

-------
effects.
Turning now to the results. In the automobile study the authors estimate a variety of
VSLs. I focus on the adult-child comparisons because these are most relevant for policy and
comparable to the bicycle helmet study. The authors find comparable results for both the adult
and child VSLs when they aggregate across income groups - both estimates are around $5
million. In terms of policy analysis, the results at the aggregate level, which is what we would
be most likely to use in a benefit-cost analysis, may be somewhat comforting in that applying
one value to all individuals comports with their results.
In the bicycle helmet study the authors estimate a VSL and a VSI - or value of a
statistical injury - for children. They plan to add similar results for adults in the future. Their
VSL for a child in about $9.5 million, or double the results found in the automobile study. This
creates a problem for the policy analyst who must decide how to use this information. Here we
have two results, both for fatal, immediate risks, both involving decisions made from the parental
perspective, and one result is double the other. While we do not know if these differences are
statistically significant, there may be reasons why we would expect them to differ in magnitude.
In the automobile study the parent may feel a certain amount of control over the risk
reduction. That is, the parent probably drives more safely when the child is in the car - or
believes that he or she is a better driver than others on the road, in general. The parent may
avoid particularly dangerous traveling situations, such as avoiding the roads when there is heavy
traffic or rain. By taking these precautionary measures the parent may feel a certain amount of
control over the risk and is not using the car, exclusively, to mitigate the risk associated with
driving. These alternative behaviors are not reflected in the analysis and therefore may result in
a lower VSL estimate than if they were included.
On the other hand, the parent may feel little or no control over the child on his or her
bicycle. This is likely to vary somewhat with age. For smaller children the parent may
accompany the child on the bike, maintaining control over the types of trails used and how fast
40

-------
or reckless the child rides. However, as the child gets older and becomes more independent with
bike-riding, the parent loses some control - or transfers some of it to the child - and the bicycle
helmet is the only safety feature that remains from the parent's perspective. Hence, the bicycle
helmet might represent all of the mitigating behavior that is used, in which case the VSL
estimated might be higher than in situations where the parent has other options.
Turning to the value of a statistical injury estimated in the bicycle helmet study. The
authors estimate a value of approximately $7.0 million, which is almost as high as the VSL
estimated and much higher than expected. It is likely the case that the parent is buying
protection from serious injuries associated with a cycling accident, as opposed to cuts and
scrapes - injuries that could potentially be very debilitating and painful. These serious injuries
could be devastating and therefore may in fact carry a value that nears that associated with death.
In terms of contribution to the literature, both of theses studies provide much needed
estimates of risk reductions to children. Currently, only a few estimates exist and it is useful to
add to this literature as we build our understanding of these values. In the automobile study, it
would be useful to have estimates for morbidity, which can be calculated given the data used in
the paper.
In terms of applications to policy, analysts are very often interested in how results can be
applied in a benefit transfer context. OMB has stated that estimates should only be transferred
when the context in the study parallels the policy question. Many of the environmental policies
at EPA deal with risks that have a long latency period, which may or may not ultimately end in
death. In these cases it may not be appropriate to use estimates for fatal, immediate deaths in
policies where there is a long latency period. The authors should address how their results may
be used in a policy context given that the nature of the risks in these studies may differ from
those used in environmental policy.
In summary, both studies provide much needed estimates for how people value risks to
41

-------
children. Even if the context does not mimic that found in many environmental policies, these
studies are a step in furthering our understanding of how we might value risks to children, in
general.
42

-------
Summary of Q&A Discussion Following Session IV-AM
Anna Alberini (University of Maryland) opened by asking for "Just a clarification from
Nicole Owens and Lanelle Wiggins-How did you calculate the baseline risks that you used
in your regressions?"
Nicole Owens (EPA NCEE) responded that "the CDC has data on the number of people
that were either killed or injured as a result of biking. We adjust that to take into account
the fact that people are wearing helmets, so for kids and adults the percentage is different,
but eighty percent of kids out there are wearing helmets and we know how many of them
died or were injured taking into account that helmet use. So we back out either the
number that would be injured or would have died in the absence of helmet use and call
that baseline risk."
Albernini followed up with, "But how does it vary across the respondents?-which is my
original question."
Owens clarified by stating that the data is tabulated by age, race, and gender and,
therefore, is not really a personal measure of risk.
Albernini closed by asking, "So, potentially there is correlation with age, which is also
one of the regressors?" and Dr. Owens responded, "Yes."
Don Kenkel (Cornell University), addressing the researchers of the bicycle helmet study,
observed that "your risk variable is just some kind of non-linear combination of other
variables on your right hand side, so in some sense, you could question are you picking
up the effect of risk versus the effect of quadratic interaction terms of those other
variables . . ."
Nicole Owens (EPA NCEE) responded that they have "tried really hard to get a more
individual measure of risk and so far it's been kind of unsuccessful." She said they had
thought of implementing people's zip codes to help make some assumptions, but even
that wouldn't provide information about "where they ride, whether it's urban or rural," so
they still are left with the problem that the number they get from the CDC is categorized.
Lanelle Wiggins added that they also tried to include some notion of exposure by using
the "days ridden" variable within the risk variable, but that had created some troubles
also.
A1 McGartland (EPA) said that the automobile market and bicycle helmet study presentations
brought his thoughts back to yesterday's lunchtime talk and the issue of how kids deal
with low-probability events or high-probability events. He asked if either of the research
groups had "thought at all about perceived risk versus your measured objective risk and
whether that might influence or put your results in perspective."
43

-------
William Schulze (Cornell University) said that they "actually collected in the mail survey
perceived risk information, and I used that for the fragility, so we've got at least the
engineering risk, is what I call it, or the non-person-specific risk that we calculated from
the FARS data set-we also have subjective risk from our survey that we haven't had a
chance yet to utilize and compare the results both ways."
Lanelle Wiggins said they also had thought about that and in their survey had included a
question designed to get at risk perception, something to the effect of, "If you knew that
the helmet you purchased provided this much risk reduction, would you still have
purchased it?" She concluded by saying that they are still trying to figure out an effective
way to get at and use that information.
Robin Jenkins added, "I think Lanelle mentioned the NSRE was done over the phone, so
we struggled a lot with the original questions we had for trying to ask people about their
perceived risks, and you just can't explain that over the phone. It wasn't going very well,
and then we also had no control over when the survey was implemented, so we came to
give up that endeavor." She said she believes the NSRE is scheduled to be redone in
2005, which will present a new opportunity to try to gather that kind of information.
Barbara Kanninen asked Dr. Schulze whether he had explicitly accounted for family size
in the automobile study model. In explaining why this would matter, she stated that
current laws regarding air bags and child safety seats coupled with space issues eliminate
the option of purchasing a safe Volvo stationwagon once you have three kids-they
simply won't fit into what might be your vehicle of choice. She personally knows several
families who, as soon as they had three kids, traded in their Volvo wagons for Ford
minivans. She continued, "So, I was thinking, especially when you get to the multinomial
choice models, that might be a place where you can really account for that, because
basically, choice hasn't changed. They, of course, I think are very much thinking of
safety, but they also don't have some of the safe cars available to them anymore."
Picking up on a related issue, Dr. Kanninen said, "I'm not a family economist, so maybe
I'm out on a limb here, but-depending on the size of your family, if you have one kid
versus five kids, at some point when you've got a lot of kids you have to sort of mentally
think of them as a pack. They're sort of not individuals any more-it's like mentally,
financially, emotionally, everything-you're dealing with the kids. And so I was kind of
wondering if when it comes to decision-making, especially when it comes to the
pocketbook, do families with single children actually value that one child a lot more than
if you divided that five-kid family into five? ... So, to use an inappropriate term, there
might be sort of like almost "diminishing marginal returns" to a family who has more
kids, and I wondered if that might come out of this model as well."
Dr. Schulze replied that although he fully agrees with Dr. Kanninen's first comment, he
is reluctant to say much about what's going on with the kids and the parents. He added
that the VSL switching that goes on between kids and adults in the lower income and
44

-------
middle income groups (with kids worth less than adults in the lower income group, and
adults worth less than kids in the middle income group) is something he doesn't fully
understand. He also commented regarding "the fact that parents' values are so low
relative to families which you don't have children in, so something strange is going on
here, and I don't want to claim that I understand it." He agreed that a multinomial model
would probably be a productive way to move forward "along with adding some
additional explanatory variables that we have collected ... we know there are broken
homes and things like that." In closing, he reiterated that "there's a lot more work that
needs to be done with the data that we collected, and anything I would say would be very
speculative. I already speculated a little, but / don't understand it at this point."
Ted Bergstrom had this to say: "I don't do econometrics very often, but I'm used to
seeing numbers with confidence intervals around them. Kelly Maguire remarked that she
thought the differences between the bicycle helmet study and the car study values of child life
were big. I didn't think they seemed so big, and I especially thought that neither of them
seemed terribly reliable. I would guess that a responsible policy analysis would suggest
that you should provide some confidence intervals."
After a long pause, Dr. Schulze drew laughter by responding simply "Yes." He was quick
to add that one shouldn't be misled by a "t" of 14 on an estimate of VSL because an
enormous amount of statistical machinery is behind that calculation, and he conceded,
"You need someone smarter in statistics than I to construct a confidence interval." He
went on to say, "You know, there are many questions about the functional forms we used
to predict the allocation of miles, so I fully agree with that. What we know about the
VSL is that it falls in a certain range, and that range is not unreasonable compared to the
physical science estimates we get in the environment, where an order of magnitude is
great. Well, we certainly know the order of magnitude, and there's not a lot of evidence
that old people are worth a lot less than adults, and there's not any reliable evidence that
we should value kids less, and EPA uses one number for everybody, and I don't see any
support for changing that until we know a lot more. So, I think you're exactly right
because of that confidence interval."
Ted Bergstrom then directed a similar question to the bicycle helmet researchers: "How
about the bike study-have you got any notion about how confident you are in your
numbers? Would you be surprised if you were off by a factor of 10 or even 5?"
Nicole Owens answered that early in the study they were actually getting estimates that
were much, much higher and since they haven't calculated confidence intervals, they
would not be surprised at all if they off by a factor of that much.
Robin Jenkins quickly added, "But we can. We can estimate them, so we will.
45

-------
Charles Griffiths (U.S. EPANCEE) directed his comments to Dr. Schultze, first stating
that he would view thirteen telephone calls to a home with no response as a refusal. He
cited the fact that many people use their caller ID function to screen calls and simply
won't pick up if they don't recognize the caller.
Dr. Griffiths then made his main point by stating, "I was curious what the theoretical
justification was for miles in the utility function. It seemed like an intermediate product-
you would sort of go somewhere in the miles required to get something that's useful for
the utility function. I noticed it was in the kid's utility function, too, and there I'd have
trouble citing it-I mean you'd think it would be negative, but, you know, I gave my son a
Gameboy and now he loves to ride in the car . . ."
He closed with this final question regarding econometrics: "I noticed in your model the
omitted category was the high-income kids and child VSL. I assume that was done for
econometric purposes, but my question is just: Does that force then the equation to
implicitly assume the same VSL for the high-income because . . .?"
Dr. Schulze responded, "No, there were actually different coefficients-there wasn't a
subscript working on them, so no-they could be different for each group."
He addressed the phone call comment by saying, "The thirteen calls-sure, who knows?
It's just that you call a number thirteen times and it never gets a pick-up-you're right, it
could be caller ID, but then they'd have to have been there ... I don't know-who knows?
My recommendation is forget telephone surveys, forget mail surveys, go to the web-
based and you need to get some approval on a better approach. I certainly would not do
this again-it's a very expensive, very inefficient way of collecting data. You know, it
was the only thing available to us at the time. So, that's my position on that."
Dr. Schulze continued by asking, Dr. Griffiths: "In terms of your point about miles in the
utility function, how many miles did you drive last year?" to which Dr. Griffiths answered,
"10,000." Dr. Schulze concluded, "You must like driving-you spend a lot of money
doing it. That's my answer. I mean, people do it-they like doing it. . . . We spend a
major part of our life doing it, and I just didn't want to make the model more
complicated." He acknowledged the point that "it actually is an intermediate good" but
said that for simplicity's sake he "just dumped it in there." He closed by saying he didn't
give it more attention because he didn't think that was where the theoretically interesting
stuff was.
Ronnie Levin (U.S. EPA) opened by commenting that in her experience the utility of
telephone surveys depends on what you're doing. She said, "We do a lot of targeted
randoms and not random digit dialing, so when we're surveying sectors, we do a lot of
surveys using the telephone, and it works very well."
46

-------
Regarding the bicycle helmet study, Ms. Levin then asked, "In the CDC injury data, do
they record whether the injuree was wearing a hemet?"
Nicole Owens answered that although that information was included in some "smaller
surveys that observed hospital emergency room visits," the CDC doesn't collect those
data.
Levin continued, saying that "injuries in most sports don't occur equally distributed
across the population of participants, and so that may be something to further investigate.
Now, the purchasing is happening by the parent, who is not the person who's actually
exhibiting the risky behavior. But, again, going back to A1 McGartland's comment that
perceptions of what is risky behavior vary by age and sex-and so that's something else for us
on the finessing of loose ends. But, I think it doesn't negate in any way the fact that I find
the numbers remarkably similar, also."
Mary Evans (University of Tennessee) stated her interest in the issue of how decisions
about multiple mortality risks are made. Addressing Dr. Schulze, she said, "You have
this kind of inherent in your structure, where you have a baseline survival probability and
you assume sort of an additive framework, so that risks just add on top of each other. I
wonder if you could comment on how an alternative framework in which you have
individuals confronting risks in a sequence-in other words, I have to survive some
baseline risk in order to then be confronted with and ultimately deal with fatality risk-
how that might change the way that you think about estimating VSL."
Dr. Schulze responded that he would need a blackboard to work through that explanation,
and he borrowed a quote from his doctoral microeconomics professor, who claimed not
to be "smart enough to do economic theory except using mathematics as a crutch." He
said the question required "real thought" and a lot of figuring.
Evans commented that she and Kerry Smith "have done a little bit of work looking at
that" and offered to speak with Dr. Schulze afterwards, to which he responded, "I would
love that-it's even better if / don't have to do it."
Bryan Hubbell (U.S. EPA) addressed Lanelle Wiggins and Nicole Owens regarding his
concern "about this issue of jointness in production: You're using the cost of the bicycle
helmet somewhere in there-I couldn't figure exactly whether they have individual prices or
not, but if you do have individual prices, there is this issue that you're producing this utility
for your child by buying him a bicycle helmet that looks ugly. And they're also less
likely to wear it. So, there are two things they're buying when they pay additional price
for a helmet beyond what the minimum price might be to get an effective helmet. And if
there's a systematic willingness to pay either to improve the child's utility because of the
ugliness factor or to get them to increase their compliance behavior, that could affect
your VSL in an upward fashion. Now, the compliance behavior-you may want that to be
part of it, but you certainly don't want to include the child's willingness to pay or the
47

-------
parent's willingness to pay to reduce the child's disutility for appearance's sake into that
VSL." Dr. Hubbell suggested finding some way to adjust the price of the bicycle helmet
"to get rid of, or net out, this co-production of this other utility." As a related example,
he cited the purchase of bottled water, where you're not just buying the safety-you're
buying the taste and other things. He added that figuring out a way to pull that factor out
might make the numbers more in line with some of the adult values.
Nicole Owens responded that the idea made sense but it was not something they had
thought of.
Cristina McLaughlin (U.S. FDA) commented that she thought the use of temperature data
in the bicycle helmet study was very creative and she asked whether they had also used
that data to examine if temperature actually played a role in the decision to use the
helmet.
Nicole Owens responded that the original intention was to predict days ridden as a
function of temperature and precipitation. She said that they found that temperature was
a significant factor: i.e., when it was warmer, kids rode more. The data also indicated
that precipitation had no effect, but she stated that it was difficult to draw any really
strong conclusions regarding this because the associated equation "was dismal."
T.J. Wyatt (U.S. EPA/OPP/Division of Biological and Economic Analysis) asked the
presenters to comment on how they think the absolute cost of the risk mitigating decision
plays into the estimate of VSL. He pointed out that "a helmet that costs 25 or 30 bucks
seems like a fairly small expenditure to avoid risk, whereas an auto purchase would be a
major commitment of resources, particularly for low-income families." He questioned to
what extent they thought that might have something to do with the difference in
magnitude of their estimates.
Lanelle Wiggins responded that they had thought about getting at that issue by trying to
figure out how to measure the time it takes to use a helmet and somehow incorporate that
into the price. She added that because of the way the model is currently structured, with
the helmet price simply subtracted from income to reduce the amount available to spend
on all other goods, it probably wouldn't change the results that much. She
acknowledged, however, that it is an important point.
Dr. Schulze affirmed that "a single mom, newly divorced, and with a kid, might very well
have a hard time getting auto financing, and that could be affecting our values, so that's a
very good point."
Nicole Owens added that helmets, especially for kids, are very cheap and effective.
People were asked how long they thought the helmets would last and their answers along
with the prices of the helmets yielded an annualized price of the helmet somewhere
around $6. She continued to say that combining this information with the risk data, it
48

-------
brings back A1 McGartland's point about what is the risk that parents perceive. She closed by
stating that it also might be that for $6 "you're almost buying yourself out of the uncertainty
associated with what the specific to your kid might be" or you might feel better about the
cost per use of a rarely used helmet.
Ted Bergstrom asked, "What is the reduction in the probability of the child dying as a
function of having that [helmet]?"
Nicole Owens responded with the figure "2 million,"-the annualized price divided by the risk
reduction for kids, yielding the estimated VSL.
Bergstrom continued, "So we're saying that all the parents who do not buy helmets, by
your methodology, think their kids are worth less than $2 million. . . . How do you get to
the $10 million statistic?" He asked whether that comes from all the extra paid for fancy
helmets.
Robin Jenkins said that the paper they had written previously in fact did make it that
simple-just looking at the price divided by the risk reduction. She clarified that the
current study involves a household production model, in which the household is
producing safety and the decision to purchase a helmet is really representing the indirect
utility function of the household. So, the valuation is on risk-the model is designed to
represent something beyond what you would get from such a simple calculation. She
closed by saying, "So, I guess it's all embedded in the modeling-the fact that we've got
this random utility model that's representing the conditional utility function."
Bergstrom said, "But somehow that seems to me it's producing something from nothing."
He added that the data shows that there are a lot of people willing to pay at least $2
million for a statistical child's life, and he reiterated, "That's all you've got. And so now
you've got a complicated model that produces $9 million. How can this be? I love
magic, but. . . where's it coming from?"
Jenkins responded that "It's not really magic-it's just a representation of the household's
decision that is trying to build into the equation other considerations about the valuation
of safety . . ." When one of the participants clarified, "You're dividing by the marginal
utility of money-you're not dividing by the price," she added, "Exactly-marginal utility
of income."
Someone commented, "And if it's true that 90 percent of them are buying helmets, then
the lower bound is $2 million but the mean is going to be up there . . ."
Jenkins added that the 2 million figure was just "off the top of their heads" and may not
be right. She acknowledged that the "other paper" dealt with a higher value, closer to 4
million.
Owens clarified that the other figure was "the adult valuation . . ."
49

-------
Scott Grosse (Centers for Disease Control and Prevention) asked whether any of the
researchers had tried to calculate VSLs by parents' education levels and added that
"There is a large literature suggesting that parental education leads to different valuations
of children's health and investments in children's health, so it would be plausible that the
VSL would differ by the education level."
William Schulze responded that their study represented "sort of a first look" at these data,
and he affirmed that they did have information on that. Furthermore, he said that the
parent education level is "exactly one of the variables that I hope to use to try to figure
out what's going on with kids' and adults' relative values switching between income
classes." He conjectured that the hypothesis that "lower-income families have lower
education" levels might help explain the situation.
Nicole Owens explained that parent education was not factored into their equation,
although it was significant, so the relationship remains to be explored.
Glenn Harrison (University of Central Florida) commented that listening to these study
reports every couple of years always brings to mind the fact that "there are other sources
of data that are extraordinarily rich that might be worth accessing, and it's the sort of
thing an agency could do if they approach the right people." As one such source, he
mentioned HMOs, "particularly Health Partners, based in Minnesota" as groups that
engage in the collection of a lot of health-related data about cars, bike helmets, smoking,
alcohol, etc. He continued by saying that "basically, they're collecting information from
their target population about all sorts of risk factors" because they're interested in
educating the people on the risks of obesity and all sorts of things and the interactions
between them. The bottom line is they want to lower their own costs. Dr. Harrison
explained that although these organizations are generally extremely reluctant to let others
access their data, agencies can talk to them "and get cooperative agreements with some of
these places." As an example, he said the CDC has negotiated such arrangements with
some HMOs. He closed by testifying to the "incredible" quality and value of the data
available from these groups because "they track everything else about these people as
well."
50

-------
Valuing Environmental Health Risk Reductions to Children
PROCEEDINGS OF
SESSION IV-PM: AGE-SPECIFIC VALUE OF STATISTICAL LIFE ESTIMATES (CONT'D)
A WORKSHOP SPONSORED BY THE U.S. ENVIRONMENTAL PROTECTION AGENCY'S
NATIONAL CENTER FOR ENVIRONMENTAL ECONOMICS (NCEE), NATIONAL CENTER FOR
ENVIRONMENTAL RESEARCH (NCER), AND OFFICE OF CHILDREN'S HEALTH PROTECTION;
AND THE UNIVERSITY OF CENTRAL FLORIDA
October 20-21, 2003
Washington Plaza Hotel
Washington, DC
Prepared by Alpha-Gamma Technologies, Inc.
4700 Falls of Neuse Road, Suite 350, Raleigh, NC 27609

-------
ACKNOWLEDGEMENTS
This report has been prepared by Alpha-Gamma Technologies, Inc. with funding from the National Center
for Environmental Economics (NCEE). Alpha-Gamma wishes to thank NCEE's Kelly Maguire and
Project Officer Nicole Owens for their guidance and assistance throughout this project.
DISCLAIMER
These proceedings are being distributed in the interest of increasing public understanding and knowledge of
the issues discussed at the workshop and have been prepared independently of the workshop. Although the
proceedings have been funded in part by the United States Environmental Protection Agency under
Contract No. 68-W-01-055 to Alpha-Gamma Technologies, Inc., the contents of this document may not
necessarily reflect the views of the Agency and no official endorsement should be inferred.
11

-------
TABLE OF CONTENTS
Session IV-PM: Age-Specific Value of Statistical Life Estimates (Cont'd)
Not All Deaths Are Created Equally: Understanding Individual Preferences for
Reductions in Morbidity-Mortality Events
Trudy Cameron, University of Oregon, and J.R. DeShazo, UCLA	1
Valuation of Environmental Risks to Children's Health
Mark Dickie and Shelby Gerking, University of Central Florida 	62
Discussant
Glenn Harrison, University of Central Florida	100
Summary of Q&A Discussion Following Session IV-PM	105
ill

-------
DRAFT-Preliminary and Incomplete
Please do not cite or quote without permission from the authors
An Empirical Life-cycle Model of Demand
for Mortality and Morbidity Risk Reduction
J.R. DeShazo and Trudy Ann Cameron1,2
Abstract
This paper explores empirically the way that demand for health-enhancing and life-extending
programs varies over the life-cycle for individuals. We test the hypothesis that, at any given
current age, an individual's schedule of marginal utility for future risk reductions rises on average
with the age at which the future adverse health status would be experienced. However, as
individuals age, we also hypothesize that there is a systematic downward shift in these schedules
of marginal utility for risk reductions at future ages. Using data from a representative national
sample of US households, we estimate the net effect of these two offsetting age effects for various
risk-reducing policies. We identify the systematic age-varying determinants that explain why
demand for some programs varies significantly with age, while demand for other programs does
not.
1	Senior authorship is not assigned. Nominal lead authorship will rotate though the series of papers associated with this
project.
2	Department of Policy Studies, UCLA, and Department of Economics, University of Oregon, respectively. We thank Vic
Adamowicz, Richard Carson, Maureen Cropper, Baruch Fischhoff, Jim Hammitt, Alan Krupnick, and V. Kerry Smith for helpful
reviews of our survey instrument and/or comments on our early results. Rick Li implemented our survey very capably. Ryan
Bosworth and Graham Crawford have provided able assistance. This research has been supported by the US Environmental
Protection Agency (R829485). It was was also encouraged by Paul De Civita of the Economic Analysis and Evaluation Division
of the Healthy Environments and Consumer Safety Branch (HECS) of Health Canada and has been supported in part by Health
Canada (Contract H5431-010041 /001 /SS). This work has not yet been formally reviewed by either agency. Any remaining errors
are our own.
1

-------
1 Introduction
Empirically, scholars know little about how demand varies, over individuals' life-cycles, for programs that
reduce the risk of morbidity and mortality. These programs include publicly provided environmental, safety
and health programs as well as privately available preventative care and medical therapies. Understanding
how demands for these programs vary with age has become increasingly important to several fields in
economics.
Environmental and regulatory economists measure the social benefits of publicly mandated environmen-
tal, health and safety regulations. At the prompting of policymakers, they are now seeking to determine
whether the sizes of the social benefits from these programs vary by age group (Smith and Evans, 2003;
Alberini, et al., 2003).
Health economists have a longstanding focus on how individuals' investments in their health vary through-
out their lifecycle. While this literature has made considerable theoretical advances in understanding the
life-cycle determinants of demand for health (Grossman, 1972, Chuma and Erhlich, 1990, Johannsson, 1997;
Erhlich, 2002) there has been a shortage of empirical analyses that test the hypotheses implied by these
theories.
Finally, we are in the midst of a number of major demographic shifts, including a general aging of
the population. Public economists have become interested in how changes in health and longevity affect
individuals' consumption of government programs such as Social Security, Medicare, Medicaid (Hamermesh,
1995; Hurd et al., 1995, 1995, Gan et al., 2003).
The central contribution of this paper is an empirical exploration of the way individual demand for health-
enhancing and life-extending programs varies with age. We test two hypotheses about how individuals will
value risk-reductions over their life cycle. We motivate these hypotheses though a [stochastic dynamic
optimization] model in which the individual chooses a quantity of a risk-reducing program in each period
throughout her life cycle (Ehrlich, 2000). Our first hypothesis is that, at any age, individuals will derive
2

-------
increasing marginal utility from reducing risks that come to bear later in life. This comports with the
intuition that marginal value of health investments rises with age and concurrently that life-saving programs
will grow more valuable with age.
Our second hypothesis is that as individuals age, there is systematic downward shift in their schedule of
marginal utility for risk reductions at future ages. Our second hypothesis is based in the assumption that
there are strong complementarities between health and other commodities. Individuals only learn about the
extent of these complementarities as they age. With greater age, the declining quality of health begins to
have appreciable affect on the marginal utility of consumption. Such age-induced learning causes individuals
to decrease their expected value of future consumption. In response, individuals intertemporally adjust by
shifting their consumption forward in time. As the value of future consumption declines, so does the shadow
value of investment in life-extension. The effect of this process is to diminish the value of investment in
current and future life-saving programs.
While this model illustrates these two countervailing dynamics, the net effect on age-varying demand
for risk reductions is ultimately an empirical question. To evaluate these two hypotheses and to assess this
empirical question, we develop an estimating specification that enables individuals to express their demands
for risk-reduction programs that will alter the time-pattern of risk faced during their remaining life cycle.
This empirical model makes several contributions to the existing empirical literature (Krupnick et al., 2002;
Alberini et al., 2003; Evans and Smith, 2003).
First, we cast the demand for risk reducing programs in an option price framework (Graham, 1982). This
approach is appropriate for the vast majority of public and private life-saving programs, since they involve a
stream of certain costs and uncertain future benefits. Second, this model recovers the individual's marginal
utility of avoiding a year spent in a morbid condition, the marginal utility of a year spent in a recovered
state, if the condition is not fatal, and the marginal value of avoiding a lost life-year. Our specification allows
these marginal values to shift with both the individual's current age and the future age at which the risk of
3

-------
each particular health state is reduced. For each age cohort, we are able to estimate the schedule of marginal
values associated with reducing risks over all future ages.
We estimate this model using data from an innovative national survey of over 1,300 U.S. citizens. In the
survey, individuals were asked to choose between programs that reduced the probability of experiencing a
future time profile of undesirable health states. The time profiles for these health states were described in
the context of the individual's current age and nominal life expectancy. Each profile described the future age
of onset of an illness, the level and duration of pain and disability that could be expected to follow, including
surgery and hospitalization, and the number of life-years lost relative to nominal life expectancy. The risk-
reducing programs consisted of an ongoing annual diagnostic test for a specific illness. If the individual is
found to be at risk for the illness, they would be given drug therapies, and prescribed lifestyle changes, that
would reduce their risk of experiencing the illness profile. These data enable us to evaluate how individual
demand for avoiding a future year of morbidity and premature mortality varies with each individual's current
age and with their age during each future period of reduced health (or they age that they would have been,
had they not experienced premature mortality).
Controlling for the individual's current age, we find that the marginal utility of avoiding a future lost life-
year rises with age. Controlling for the age at which the undesirable future health states would potentially be
experienced, we find that as individuals grow older, their marginal value of current and future risk reductions
declines. Together, these effects produce distinct schedules of marginal utility for each age cohort. To
evaluate the present value of these future risk reductions, these marginal values must be discounted, and the
risk reduction normalized to 1.00, to obtain the present value benefits of avoiding a "statistical" case of a
particular morbidity/mortality health profile.
We conclude our analysis by exploring how a number of selected types of risk-reducing programs will
be valued by different age groups. Most previous empirical studies (notably wage-risk studies) have focused
on the risk of sudden death, so we emphasize these health profiles here. Through simulations, we evaluate
4

-------
the social benefits of a program that reduces the risk of sudden death for five age groups: 25-, 35-, 45-,
55-, and 65-year-olds. Because the individuals enjoy the risk reduction immediately, any differences that are
observed are determined exclusively by the age-specific declines in the marginal value of risk reductions. We
find in models with no explicit age effects, and in models with linear age effects, that our inferred values of
statistical lives (VSLs) decline by age cohort. In more general models with quadratic age effects, the pattern
is non-monotonic. First one effect dominates, then the other.
Our second policy simulation, a latency period of five years is hypothesized before the risk of sudden death
would materialize so that benefits would begin to accrue. In this context, discounting becomes important
when calculating the VSL, but the discounted latency period (five years) is constant across age groups.
As should be expected, we find differences in age-specific VSLs that are roughly comparable to the first
simulation.
Our third policy simulation considers how all age cohorts would value a reduction in the risk of death at
the common age of 70. A model with no explicit age effects suggest strongly that the VSL increases as the
respondent's age gets closer to 70. In models with age effects, however, the pattern of VSLs is non-monotonic
due to the competing influences of the two types of age variables in our models. For models with age effects,
we find no clear differences in the VSL for this policy which is consistent with the findings of Krupnick et
al., (2002) and Alberini et al., (2003). Our results suggest that the reason for no apparent age effect may be
the existence of offsetting effects of discounting and downward shifts in the marginal utility of risk reduction
with age. Younger cohorts have a higher marginal utility for the risk reduction but this value is discounted
over a longer time period. Older cohorts express a lower marginal utility for the risk reduction but discount
it over a shorter period.
In our final policy simulation, we hold the age group constant (focusing only on 25 year olds) in order
to evaluate the implied VSLs for policies with latency periods that vary by 5, 15, 25, 35 and 45 years. In
this simulation, we utilize only the upward sloping schedule of marginal utility for future risk reduction
5

-------
associated with 25 year olds. Therefore, any difference in VSLs results from differences in (f.) the slope of
the marginal utility schedule for future risk reduction, (2.) the discount rate, and (3.) the latency period
over which discounting occurs. In a specification that ignores age effects, there in an apparent strong decline
in the VSL as the latency period for the mortality risk increases. In a model with linear age effects, this
pattern persists but is somewhat attenuated. In a model with quadratic age effects, however, the pattern
becomes again non-monotonic.
2 Theoretical Model of Life-Cycle Demand
This model follows Ehrlich (2002) who develops a life-cycle model of demand for risk reduction programs.3
To maximize lifetime utility, individuals choose quantities of a risk-reducing program, I(t), and a composite
consumption activitiy, Z(t), in each period, t.4 Demand for risk reduction arises because individuals face a
conditional per-period arrival frequency, f(t), of life-threatening events (such as major illnesses) that lead
to mortality.
Individuals control the flow of f(t) though the purchase of risk reducing programs in the following way:
f(t) = j(t)-I(t)	(I)
where I(t) = I(m(t), M(t)\ e(t), t),
and where j(t) > 0 is the exogenous conditional probability of a major illness, which is determined by heredity
in conjunction with biological and environmental risks. I(t) defines the difference between the exogenous risk
and the individual's actual risk, j(t) — f(t).5 These risk-reducing programs are produced using inputs of time,
3This model generalizes similar life-cycle models by Conley (1976), Shepard and Zeckhauser (1984), Rosen (1988), and
Johansson (2001).
4For simplicity, we assume that individuals habor no bequest motives. Nor do individuals participate in insurance markets
that are designed to protect against "living too long" (by purchasing guaranteed annuuities) or "living too short" (by purchasing
life insurance).
5 This formulation simplifies the derivation of the time path for I* (t).However, it abstracts from the possibility that current
period expenditures on risk reduction could affect the the conditional risks in future periods.
6

-------
m(t), and market goods M(t). Production of these programs is also determined by efficiency parameters,
e(t) (reflecting the individual's human capital or education level), and their current age, t. In keeping with
theories of aging (Kirkwood, 1977; Kirkwood and Rose, 1997; Sozou and Seymour, 2003), health risks are
assumed to rise at an increasing rate throughout the remaining lifespan (j(t)) = dj(t)/dt > 0.
The cost function for I(t) is given by
where w is the wage rate per unit of human capital. The wage rate also represents the opportunity cost
of time, where w = w(e)/e. All prices, P(t), and efficiency parameters, e(t), are held constant across the
life cycle. For simplicity, assume that a = 2 so that the cost function for the risk-reducing program is
C(I(t)) = cl(t)2. The production function for risk reductions is subject to diminishing returns to scale
because of the fixed scale of the human body.
In each period the individual will consume a flow of health-state denominated time, h(t), and a flow from
a composite consumption activity, Z(t). We treat the risks of morbidity and mortality as independent risks
in our empirical analysis. However, we assume here, for simplicity, that they are monotonically related to
one another.6 Health-state denominated time is assumed to be a decreasing and concave function of f(t)
to capture the positive correlation between the risk of a life-threatening illness and associated morbidity.
Health denominated time, h( ), may range from perfect health to acute morbidity as function of :
The argument /3 represent shifts in medical technologies that reduce the levels of morbidity associated with
6 As a practical matter this assumption limits us only in that we cannot theoretically explore the determinants of individuals'
marginal rate of substitution between morbidity and mortality as health states. Such tradeoffs are not the focus of this paper;
see Cameron and DeShazo (2004) for an exploration of these issues.
C(I (t)) = c(t) I (t)a where a > 1 and c (t) = c(w (t) ,P(t),e (t) , t),
(2)
h (t) = (h f (t) , f3) with h'(-) < 0 and h"{¦) < 0.
(3)
7

-------
h(f(t)). We assume that this measure of health-state denominated time perfectly exhausts each individual's
time constraint.
The consumption activity Z(t) is produced by combining purchased market goods, M(t), at constant
unit prices (P) and the individual's time, m(t). The individual purchases these market goods subject to an
instantaneous wealth constraint:
where A(t) = dA(t)/dt is the rate of change in savings in period t.7 Parameters r and w denote the market
interest rate and the wage rate, h (t) represents healthy labor time, and the full price of consumption,
Pz = 1, is the numeraire. The individual knows her terminal condition (age of death) only stochastically,
which represents an innovation on Ehrlich and Chuma (1990). In the following equation, E represents
the expectation operator which applies to the stochastic length of life, D, while p denotes the individual's
subjective discount rate.
Individuals choose optimal time paths for Z and I to maximize8:
The individual maximizes (5) subject to equations (1) and (3), A(t) > 0, as well as a vector of exogenous
parameters: a = w,e,P,p,j. The terminal conditions, A(D) > 0 and J(A(D),D;a) = 0 must hold. The
optimal time paths for {Z * (t), I * (t)} are found by applying the stochastic dynamic programming approach
7 To avoid any discontinuity, which occurs whenever A(t) assumes its boundary value, the individual optimizes subject to
A(t) > 0. Furthermore, without an insurance market, it is impossible for the individual to die with negative wealth.
8The instantaneous utility function (.) is to be concave and possess other standard properties (Judd, 1998).
A(t) = r A(t) +w h(f (t)) — cl2 (t) — Z (t),
(4)
8

-------
(Judd, 1998) as determined by the Hamilton-Bellman-Jacobi condition:
Jt = ~{P + f*)J + U{Z*,h (/*)) + J a [rA + wh (/*) - cV2 - Z*\
(6)
where Jt = dJ(A(t),t\a)/dt and where Z* and I* satisfy the optimality conditions:
Uz(Z*, h (/*)) = J a
(7)
2d* = J/JA + [w + (l/JA)Uh(Z\h(f*))][-h'{!*)}= v*0
(8)
Equation (8) describes the conditions that shape the optimal time path of investment in risk reduc-
tions over individuals' life cycles. On the left hand side is the marginal cost of the risk-reducing pro-
gram. On the right-hand side is the complete value of the risk reduction which consists of two terms.
The first term, (J/J a), describes the value of the individual's remaining life span. The second term,
[w + {\IJa)TJh(Z*, h (/*))][—h! (/*)], characterizes the change in utility derived from this remaining life
span as a result of reducing morbidity.
2.1 Marginal Value of Future Risk Reduction
This model predicts that the time path of investment in risk reduction will rise with the conditional prob-
ability of risk. To see this explicitly, assume (for the sake of expositional ease only) that the individual's
utility function is separable in healthy time and consumption. From equation (8) we can show the path of
risk reduction investment depends upon two countervailing influences:
\
![d(J/JA)/dt + (Uh/JA)(-h')(r-p-f(t*))\
P(t) =
- [(u> + (Uh/JA)) h" + (Uhh/JA) (h02] j{t)
>
(9)
= v (t),
9

-------
where A = 2c — [w + (Uh/JA)h" — (Uhh/JA){h')2 > 0 and X = dX/dt. Examining the first term, the time
path of investment in risk reduction depends upon the rate of increase in exogenous risks, j(t), associated
with aging. Concurrently, the marginal value of improving health-denominated time rises with j(t) and t.
The aging process raises the marginal benefits of investment in risk reductions. Concurrently, the marginal
value of improving health-denominated time rises with j(t) and t. Therefore, we hypothesize that individuals
will express a higher marginal value for risk reductions that occur at later ages. This should be true even
though there are diminishing returns to increasing investments in risk reduction. The first two terms inside
the braces (9) illustrate how the value of protective investments rises with the value of reducing the risk of
mortality, d(J / Jj^/dt, plus morbidity, (Uh/J a) (~h') (r — p — f(t*)). We discuss the time path of these two
terms in more detail below.
2.2 Health and Consumption Complementarities
Traditional theoretical expositions of this class of models leave open the question of whether utility from
the consumption activity Z(t) and health h(t) are separable, i.e., whether Uzh(t) = 0 (see Grossman, 1972;
Chuma and Ehrlich, 1990; Ehrlich, 2000). While the modeling exercise is less complicated if the separability
assumption is invoked, such an a priori assumption seems unwarranted. To begin with, the production
function for Z(t) is assumed to require the individual's non-market time, m(t) (Ehlrich, p. 345, 2000). The
health quality of this input should affect the level of utility that the individual derives from the consumption
activity. It is much more likely that the individual's time, m(t), and market goods, M(t), are complements
in consumption, rather than perfect substitutes. As the level of morbidity h(f(t)) rises, the quality of the
individual's time input, m(t), should fall. So should the utility derived from the consumption activity, Z(t).
These theoretical relationships are supported by a large body of literature on the physiological and cognitive
effects of aging (Kenney, 1989; Gfellner, 1989; Posner, 1995). As individuals age, their ability to derive
utility from market goods declines. With increasing age, individuals begin to have trouble driving a car, for
10

-------
example, or enjoying the same recreational activities as they did in their youth or middle age. With age,
the level of utility they derive from basic market goods declines. They may eventually experience difficulty
in feeding themselves, dressing, and moving about freely.
To see the theoretical importance of this separability assumption, consider the individual's optimal con-
sumption path:
Z(t) = -[Uz(t)/Uzz](r-p-r(tj)	(10)
-l{Uzh{t)/Uzz{t)]ti{f*{t))f\t)
If Uzh{t) = 0, the second term in (10) drops out. Examining the first term, we get the well-known result
due to Yaari (1965) that lifetime consumption rises only if the market discount rate exceeds the subjective
discount rate and the conditional rate of mortality. Both theoretically (Sozou and Seymour, 2003) and
empirically (DeShazo and Cameron, 2003), scholars have shown that subjective discount rates rise with age.
Therefore, even with the assumption of separability, the quantity of consumption is likely to fall with age.
However, once the assumption of complementarities between health-denominated time and commodities is
made (i.e. that Uzh(t) > 0), the rate of decline with age will be even greater. As shown by equation (8) this
decline in the value of future consumption will, in turn, lower the marginal value of current and future risk
reduction.
2.3 Learning with age and shifts in the marginal value of future risk reductions
This model assumes that individuals have perfect information on all parameters over the course of their life
cycle. But what would be the implications if, instead, individuals learned about the aging process as they
aged? Current-period expectations about the future values of parameters are likely to be biased towards
their current-period value. Through learning, however, individuals might update their future expectations by
assessing trends in key parameters over their recent life histories. Candidate parameters for updating might
11

-------
include the individual's conditional risk of a life-threatening illness, j(t), the individual's subjective discount
rate, her future wealth constraint or the extent of complementarities between health and consumption (i.e.
if Uzh{t) > 0). While the effect of learning about any of these parameters is likely to cause individuals
to revise their future time path of consumption downward, we argue that the possibility of learning about
complementarities between health and consumption is the most plausible, since such knowledge is most likely
to be acquired though the personal experience of aging.9
If individuals do progressively learn, as they age, that health and consumption commodities are strong
complements, the exogenous rise in j(t) with age will cause the value of consumption in future periods
to fall. Intertemporally, individuals will respond to this knowledge by reallocating consumption to earlier
time periods where it will yield more utility. This action, in turn, reduces the value of investment in risk
reduction in future periods; from equation (8) the remaining value of reducing mortality risk (J/Ja) and
morbidity, [w + (1/JA)Uh{Z*, h(f *))][—h'(f*)] will decline. Based on our conjecture of age-driven learning,
we hypothesize that as individuals age, their schedule of marginal utility for future risk reduction will decline
with their current age. A related (and empirically testable) consequence of this conjecture is that individuals'
projected schedules of future marginal utility of consumption should vary systematically with their current
age. Specifically these schedules of future marginal utilities of consumption should be steeper and turn down
later in life for younger age groups relative to those of older age groups.
3 Data and Survey Methods
Our data were collected in a national random survey of U.S. adults in 2002. The innovative feature of our
survey consisted of a conjoint choice exercise wherein individuals could purchase a program that reduced
their risk of experiencing specific illnesses over future periods of their life. These programs were described
9Ehrlich (2003) provides comparative static analysis for all of these parameters except for changes in the complementarity
between health and consumption.
12

-------
as involving annual diagnostic testing and, if needed, associated drug therapies and recommended life-style
changes. Each program required a constant annual payment in return for reducing the risk of an illness
profile. Each illness profile is a description of a time sequence of health states associated with a major
illness that the individual is described as facing with some probability over the course of his or her lifetime.
We briefly describe the development, design and administration of this survey instrument below. A fuller
description is available in Appendix A.
3.1 Survey Development and Design
In order to effectively describe the illness profiles, the associated risk, and the programs that reduced these
risks, we conducted extensive one-on-one interviews (i.e., cognitive interviews) and pre-testing. We conducted
36 cognitive interviews over the nine-month development period. During this period the survey went through
four significant revisions. We pretested the last three versions. These three pretests involved a total of 1,500
respondents over a three-month period. We also benefited greatly from a peer review panel that evaluated
the second of the four versions of the instrument.
The final conjoint survey is structured around four modules: 1) the introductory module, 2) a tutorial for
the illness profile and the risk-reducing program, 3) the presentation of the choice sets, and 4) a debriefing
and follow-up module. For the sake of brevity, we focus below on only the risk-reducing program and the
design of the illness profiles in the context of the conjoint choice set.
For the risk reduction programs in our survey, we specified combinations of diagnostic testing and drug
therapies because respondents viewed these as technically feasible and potentially effective. Respondents
were familiar with comparable and pre-existing diagnostic tests such as mammograms, pap smears and
prostrate exams, or the new C-reactive protein tests for heart disease. Important from our perspective was
the fact that this class of interventions could plausibly be applied to all of the illnesses upon which we
focused. The effectiveness of these programs was described using a risk grid (Krupnick, et al., 2002).
13

-------
The payment vehicle for each program was presented as a co-payment that would have to be paid by the
respondent for as long as the diagnostic testing and medication was needed. For the sake of concreteness
we asked the respondents to assume the payments would be needed for the remainder of his or her lifespan
unless they actually experienced that illness. Costs were expressed in both monthly and annual terms. To
ensure that respondents carefully considered their budget constraint, we included a "cheap talk" reminder
as well as language to discourage overstating their willingness to pay.
3.2 Illness Profiles in Choice Sets
Each conjoint choice set presents the respondent with the attributes of two illness profiles: the illness name,
the age of onset, medical treatments, duration and level of pain and disability and a description of the
outcome of the illness. This is followed by a description of the cost and effectiveness of the risk-reducing
program. Subject to several plausibility constraints, we randomly varied these attributes across each illness
profile. Both the age of onset and the final stage of each illness are determined by the respondent's current
age. Gender specific illnesses (e.g., breast and prostate cancer) are chosen to comport with the respondent's
gender.
We summarize the results of the choice set design process in Table 2. The first row in this table presents
the frequency with which each of the twelve illnesses appeared in the choice sets. The remainder of the
table presents the mean levels of each of the risk, morbidity, and mortality attributes associated with that
illness. While the mean levels of the costs, baseline risk, and risk change are very comparable across all
of the illnesses, the average levels of the other attributes vary greatly across illnesses. For example, heart
attacks are associated with much shorter periods of pain, hospitalization, and death than is lung cancer.
14

-------
3.3 Sample and Survey Administration
Our conjoint choice survey and a separate health-profile survey were administered by Knowledge Networks
to approximately 1,800 panelists. Each survey required about 30 minutes to complete. Respondents were
paid an incentive for completing the conjoint choice survey. Respondents' ages ranged from 25 to over 90
years of age. Our response rate for those panelists contacted was 79 percent. Attrition response bias may
be present between the point when Knowledge Networks made their initial contact to join their panel and
the point when we initially contacted each panelist. To address potential sample selection bias, we are
preparing to implement sample selection correction procedures using the Knowledge Networks database of
initial telephone contacts and other attrition data.
4 An Empirical Option Price Model of Life Cycle Risk Reductions
We now turn to develop an empirical model in which individuals can express their option price for a program
that intertemporally redistributes their investment in risk reductions over their remaining life span. (See
Cameron and DeShazo (2003) for a more general discussion of this model.)
4.1 Indirect utility from health states
We develop a simple model of the individual's future undiscounted indirect utility as a function of their
health state in that future period. We expand upon most earlier empirical treatments by considering four
distinct health states: 1) a pre-illness healthy state, 2) illness state, 3) a post-illness recovered state and 4)
a dead state.10 We define each of these states as a time segment. Within each segment, the individual's
health status is assumed (for now) to be relatively homogeneous.
To capture an illness profile, we use sets of dummy variables that collectively exhaust the period of time
10Within our empirical model, the illness states are further differentiated into one of twelve specific illnesses, each of which
can exhibit a wide variety of different symptom-treatment profiles that may last from zero to six years. In appropriate cases,
the illness may also be chronic, lasting for more than six years.
15

-------
between the individual's present age and the end of his nominal life expectancy. In Figures 1 and 2, we
depict examples of these four discrete health states. Let i index individuals and let t index time periods11.
The dummy variable Pre-illness	year jt take a value of 1 in years when the individual enjoys a healthy state.
When the health state ends, the value of Pre-illness	yearit changes to 0 and remains there for the rest
of the individual's expected lifespan. At the end of the healthy period the individual may die suddenly or
become sick. Let the dummy variable Illness	yearit take on a value of 1 at this point and remain equal to
1 for the years during which the individual is ill. When he is not sick, it takes a value of zero. The dummy
variable labeled Recover	yearn takes on value of 1 in the years between the conclusion of the illness and
the individual's expected time of death. Finally, we define Lifeyear	lostit to distinguish the extent to which
death is premature (that is, the time between death and what would otherwise have been the individual's
nominal life expectancy).
Next we define the future undiscounted indirect utilities per unit of time in each health state. Let
these marginal utilities be denoted as Ss for an episode of type s, where s in our model can be illness,
recovered status, or a life-year lost to premature death. Let the undiscounted utility from each future year
in a particular health state be defined relative to no new illness. In other words, we normalize utility on
the level of utility being experienced by the individual in their current health state. We abbreviate Pre-
illness_year,, to pre,,. Mlness_year,, to illRecover_year(, to rev,, and Lifeyear_lost(, to lyl,, to allow
more-compact notation.
Vit = f3f(Yit) + 50preit + Siillit + S2rcvit + S3lylit + t]it	(11)
Let the undiscounted marginal utility of some function of current income, f{Yu), be the parameter /3. Let the
undiscounted (dis)utility from each future year of illness be defined as (5i, from each year of the post-illness
recovered state be 82, and from each year of being prematurely dead be (53.
11 Time may be measured in years, months, or even a smaller units of time, depending on the degree of resolution needed.
16

-------
Our basic specification assumes that the undiscounted (dis)utility of a year of illness or injury is a constant
(in the homogeneous specification). Let agejo denote the current age of respondent i. This is distinct from
the age of respondent i in future period t, which we will denote ageu¦ The individual's current age is just
another personal characteristic that we can allow to shift the marginal (dis) utility of a sick-year and the
marginal (dis)utility of a lost life-year.
We allow the indirect utility in each future period to depend upon the age of the individual while they
are experiencing the health state corresponding to that period. Age in period t may shift the marginal utility
of transformed income and of each health status:
Vit = [Pv+^ageu+^agel+^Y^fiYu)	(12)
+ [<5io + 8iiageit + S12ageft] illit
+ [620 + 52iageit + S22ageft] rcvit
+ [630 + 631 ageit + S32age2t] lykt + Vit-
Or,
Vit = Pof(Yit) + Piageitf(Yit) + (32age2ltf(Ylt) + (3 3Yltf(Ylt)
+S will it + Snageuillit + S12age%illit
+520rcvit + S21ageitrcvit + 522age2itrcvit
+&3alykt + 631 ageulykt + S32age2tlylit + i]it
The disutility of each of these states will be interpreted as being the same as the utility associated with
avoiding them. The dummy variables, illit , rcVit, and lylit adjust the limits of the summations used for the
present value of future continued good health, future intervals of illness, recovered time, and life-years lost.
In this paper we assume that the individual uses the same discount rate, r, to discount both future money
17

-------
costs and health states. 12
With this set-up, we can develop a structural model of the ex ante option price that an individual will
be willing to pay for a program that reduces his/her risk of a morbidity/mortality profile over the future.
Define the present discounted value of indirect utility V?k for the ith individual when j = A if the program
is chosen and j = N if the program is not chosen. The superscript k will be S if the individual suffers the
illness (or injury) and H if the individual does not suffer the illness.
The pattern of income and program costs under the four different health states will be relevant to the
individual's indirect utility in each state. We define ryl as the fraction of the individual's income that will be
earned while the individual is sick, should he suffer the illness in question. With adequate disability insurance
or sick leave, this fraction might be assumed to be 1.00. Let 72 be the fraction of income received if the
individual is no longer living, but would have been, had they not suffered the illness. This parameter will
be assumed to be zero in our empirical models, but a non-zero value could be invoked to activate a bequest
motive. The parameter 73 is the fraction of the cost of the program that must be paid while the individual
is suffering from the illness in question. Logically, the program would be unnecessary in this health state, so
we will assume that 73 is typically zero. Likewise, the individual would not participate in the program if
dead, so we will be assuming that 74 = 0.
4.2 Present Discounted Values of Indirect Utility
The present value of indirect utility if the individual does choose the program and does suffer the illness
takes the following form. All summations below will run from 0 to Tj, the remaining number of years in the
12 Empirically estimated discount rates for future money as opposed to future health states are suspected to differ to some
extent. Discount rates also differ across individuals and across choice contexts, time horizons and sizes and types of outcomes
at stake. No comprehensive empirical work has been undertaken that conclusively demonstrates the relationships between
money and health discount rates.
If we were to choose hyperbolic discounting for our specification, all of the discount factors in the expressions for present
discounted value, below, would need to be changed from 1/(1 + r)£ to 1/(1 + i)\ Other than this, the formulas will be the
same.
18

-------
individual's nominal life expectancy:
pdv(i?°) = ft E /(I,;" T + ft E	7,	("J
(l+r)	(l + r)
, 0 V- «s4/C^ - 4*) 0 y- O^t* " ~ 4*)
P2 2^ 777 7 h Ps	777 7
(l + r)	(l + r)
^ illft ^ ageitillft ^ age2tillft
+dl° 2^ + dn n , + dl2 2^
(1 + r)	^ (1 + r)	^ (1 + r)
, r	rcvit , r	ageitrcvft ^ age%rcvft
+4» E (T^« +E^ E "(T^
^ lyl£	^ ageitlylft ^ age2itlylft As
+,s» L (7777+^ L TTTTjr+^ L-jiTTf+
where Yit* = Y; (pre^ + 7liZZ$ + rc^ + 72lylft) and c£* = cf (pre^ + j3illft + rc^ + 74lylft) . Y*t and
cft* are sufficiently general to allow for a number of different assumptions about how individuals view their
potential income and how they view their cost obligations under each program in different health states.
What individuals assume about their future income and program costs, if they choose the program, has
implications for the formulas we develop in later sections. For their future income, our default assumption will
be that individuals expect constant real annual income Y{ in each future year until the expected time of death
if the individual gets the illness. When 71 = 1 and 72 = 0, the term pre^+71iZZ^+rey^+72Zj/Z|J = (l — lyl^j
in equation (13) will be nonzero in those periods when the individual is still alive. While earned income is
likely to suffer if the individual gets the illness, we assume that their annual income can be sustained through
insurance coverage. For program costs, we assume that the annual costs of the risk-management program in
question are incurred in the years leading up to the onset of the illness or injury, but are not paid while the
individual is sick or injured.13 If the individual recovers from the illness or injury, rather than dying from
it, they will again participate in the risk-management program until their death. When 73 = 74 = 0, the
term preft + 73illfi: + rev^ + 74Zj/Z^ = pre^ + rev^ in equation (13) will be non-zero only prior to the onset
13While the individual is sick, the health testing program would provide no further information, and we assume that the
major traffic accident is likely to result in the vehicle being "totaled" so that a new vehicle, with its safety features, would not
be acquired until the individual has recovered from his or her injuries.
19

-------
of the illness or during the recovered state.
The present value indirect utility, if the individual does choose the program but does not suffer the illness,
involves no illness, recovery, or reduced lifespan. Thus, the expression for indirect utility takes the following
form:
PDV(VtAH) =	(14)
(1 + r)
(1 + r)
+/^3(Xi ~ ct)I(Xi - ct) (i , \t + e^H
(1 + r)
In this case, both income and the annual costs of program will continue until the end of the individual's
nominal life expectancy. However, there are no benefits in the form of illness-years or lost life-years avoided.
Present value indirect utility, if the individual does not choose the program but does suffer the illness, is
given by:
povivD =	(15>
(1 + r)	^ (1 + r)
2Z^ (1 + r)	3Z^ (1 + r)
^ illft ^ ageitillft ^ age^tillft
+4'»^(TT77+	+
, r	rcvft , r	a9eitrcvft ^ age2itrcvft
+4» ^ (TT77 + 21 ^ "OTTT " ^ "0T7T
+^0 E +fci E	E +e"s
^ (1 + rf	^ (1 + rf	^ (1 +rf
The individual's lifespan is potentially reduced, so future income continues only until the time of death, and
the disutility of the illness, any recovery period, and any life-years lost will be relevant.
Present value indirect utility, if the individual does not choose the program and does not suffer the illness,
20

-------
is:
pdv (v%nh) = (30f(Yi)j27-^+Pif(yi)J27vEr^	as)
(1 + r)	^ (1 + r)
W(K)£-f^
(1 + r)
+/?3 (^)/(^)E TTTTt+z?11
(1 + r)
Recall, the individual assumes that his current income level will be sustained until the end of his lifespan in
the absence of premature mortality.
4.3 Expected indirect utility
In deriving the individual's option price for the program, given the ex ante uncertainty about future health
states, we need to calculate expected Utilities. In this case, the expectation is taken across the binary uncertain
outcome of getting sick, S, or remaining healthy, H. The probability of illness or injury differs according to
whether the respondent participates in the risk-reducing intervention program. Let the baseline probability
of illness be II^V'S if the individual opts out of the program, and let the reduced probability be II^'S if the
individual opts in. The risk change due to program participation, AII^'S, is presumed to be negative.
21

-------
Expected utility if the individual buys program A is:
e [v%a]sh = x pdv{v]AS) + (i - nfs) x pdv(vah)
j30 ^2	+I3t^2 a9euf{Y"^c"')
= n
AS
(l+r)'
,«	g4j(Y:t-c?t*) 3 \- (Y'-cinfiY^-cin
+<°2	(l+r)®	+ Pi 2^	(1+r-)®
+<5l° E IJ+rf + 511 E
i c \ "
	 012
a9eitM^it
(l+r-)*
(1+r)®
+^20
(1+r)'

+^0 y.
ageitlylit
(i+ry
ImW + 53i 2_^ \i+rf + 032 ~(i+7)
^ ^ ageitlylit j ^r-A-S
+ (1 - Itfs)
A>/(^ - cf) E (TW + M(Yi - E
ageit
(l+r)®
+@2f (Yi - cf) ^2 {l+rf + £'
AH
[17)
Expected utility if the program is not purchased (i.e. "no program", N), with the expectation taken over
22

-------
uncertainty about whether the individual will suffer the illness, is:
e [v%N] StH = nf s x pdv{v%ns) + (i - nfs) x pdv{v%nh)
a \ " f(Y£.) I q \ " ageufiY*)
Po 2_^ (l+r)' + PiZj (l+r)'
= n;
NS
2 2^ (l+r-)'
{1+ry
+<5ioE
Hit,.
(l+r-)*
-^11 VT"!"" +«?12
(l+r)'
(l+r)'
+^oE(^+^iE£m^+^2E£2£M
+^0 y.
lyl?t _|_ J	ageulyljt . c		
(l+r)	Z-/ (l+r) ^"32^ (1+r)
(l+r)'
ageltlylft _|_ £AT,S
+ (i - nf5)
ageit
+/3S
_iVff
v!8)
Details concerning the simplification of the expected utility difference £7 [V^4] ^ H~E [Vf] s ffare provided
in an Appendix. Concerning the time paths of future income and program costs, we will maintain the
hypothesis that (7-^ 72, 73, 74) = (1, 0,0,0). In words, usual income is sustained through illness by insurance,
but not after death (there are no bequests), and program costs are only paid while alive and healthy.
We make use of a number of notational abbreviations in getting to the expected utility difference formula.
First, let An^4,5 = (il^4,5 —	. Then, there are many distinct present discounted value terms. We
23

-------
abbreviate each of these as follows:
7 A
rpdvci
= E
pdvef
= E
pdvif
= E
pdvrf
= E
pdvlf
= E
(1 + r)
Preft
(1 + r)t
Hlft
(1 + rf
rcvft
(1 + r)t
lyift
agepdvcf =
agepdvef =
agepdvif =
agepdvrf =
age-it
(1 + r)t
ageitpreft
(1 + rf
ag&itilljt
(1 +r)t
ageitrcvft
(1 + rf
age2pdvcf =
age2pdvef =

a9eit
(1 +r)t
a9eitPreu
(1 +r)t
age2pdvif = ^ a^tdkt
(1 + r)
a9eitrcvit
(1 +r)t
age2pdvlf = ^
age2pdvrf =
(1 + r)	1 ^ (1+rf
Notice that the following two relationships hold, since the indicator variables for each health status are
mutually exclusive and exhaustive:
pdvef = pdvef + pdvif + pdvrf + pdvlf
agepdvcf = agepdvef + agepdvif + agepdvrf + agepdvif
age2pdvcf = age2pdvef + age2pdvif + age2pdvrf + age2pdvlf
To accommodate the different time profiles of income and program costs over the individual's remaining
lifespan, we must also define two additional terms
24

-------
pdvyf = ^ PrCit +	+ rC^it + l2lylit^ = pdvef + Jipdvif + pdvrf + 72pdvlf
(l+r)
pdvpf = Y ^ +	+ + ^lyl® = pdvef + - ¦,/»/;¦/;' + ^ + 7^
(l+r)
^ (pre^ + 71^^ + ^4 + 72^)2 , , 2 , ¦ , , , 2 , ,
pdvyyi = y 		j	— = pdvei + 7 jpum j + pdvri + 7 2pdvli
(l + r)
(preit + Jsittft + rcwit + J^ylit)2 ,	o t ¦ ,	2,,
pdvppi = > 		j	— = pdvei + jipdvii + pdvri + 74pdvk
(l + r)
(pref + 7^llf + rcvft + 72lylft) (pref + 73iZZ^ + rc^ + 74ZyZ^)
pdWi = ^^	
= pdvei + 7i73 pdvii + pdvri + 7274pdvli
The Appendix shows that the expected utility difference driving the individual's choice between Program
A and the Neither Program aternative can then be written as follows (there will be an analogous utility-
25

-------
difference for the B program versus the Neither Program alternative).
E [Vf] - E [Vf] = [cf ] (-1) /30 [(1 - n^) pdvci + nfspdvpi]	(19)
+ [cf] (—1) /3i [(l —	agepdvci + Ilfsagepdvpij
+ \ct\ (~ 1) P2 [(l — n/15') age2pdvci + Ilfs age2pdvpi]
+ [ct\ (-1) MX [(l - nf's) pdvci + Ilfspdvypi]
+ icf]2P 3 [(! - nf5) pdvc{ + II fspdvppi]
+/30yiAnf's (pdvyi - pdvci)
+/3iyiAnf's (agepdvyi — agepdvci)
+/321;AII^'S' (agelpdvyi — age1pdvc{)
+(35YiMlfLS (pdvyyi - pdvci)
+<5ioAII fspdvii + 5uAIlfsagepdvii + 8\2lSXlfs age2pdvii
+52oAIlfspdvri + 52iAIlfsagepdvri + 522AIlfsage2pdvri
+Sso^ifSpdvli + SsiAIlfsagepdvli +	age2pdvli + Si
4.4 Ex ante option prices
The respondent's implied ex ante option price for program A can be determined by setting the expected
utility difference equal to zero and solving for the vale of cf that makes the equality hold. First however,
the unknown utility parameters must be estimated. For parameter estimation, all terms involving the
same /3 parameter must be combined. These constructed variables, listed according to their corresponding
parameters, are:
26

-------
/30 :	[cf ] (-1) [(1 - nf5) pdvci + nfspdvpi]
+ YiAIlfs (pdvyi - pdvci)
j31 :	[cf] (—1) [(l — nf5) agepdvci + Ilfsagepdvpi\
+ liAIlf'5 (agepdvyi — agepdvci)
f32 '¦	[cf ] (—1) [(l — nf5) age2pdvci + Ilfsage2pdvpij
+ liAIlf'5 (agelpdvyi — age1pdvc{)
/3S :	[cf] (-1) 21i [(l - nf5) pcfcc; + Tlfspdvypi]
+ lcf]2 [(l _ nf5) pdvci + Ilfspdvppi]
+ Y?Anfs (pdvyyi - pdvci)
4.5 Solving for option prices from estimated models
Once the parameters have been estimated, we can solve for the payment cf that would make the utility-
difference exactly zero. This yields a quadratic form of the type 0 = Ax2 + Bx + C, where x = cf. The
squared term in cf will be activated only if /33 7^ 0 , and will bear the coefficient:
A = /3S [(l — nfs) pdvci + Ilfspdvppij
27

-------
The linear coefficient on cf will be.
B =
-B =
I30 (-1) [(l - nf's) pdvci + Ilfspdvpi]
+j31 (—1) [(l — agepdvci + Ilfsagepdvpi]
+/32 (—1) [(l — agelpdvci + Ilfs age2pdvpij
+/3S (-1) 2Yt [(1 - nf5) pdva + nfspdvyPi]
(30 [(l — pdvci + Ilfspdvpi]
+/31 [(l — agepdvci + Hfsagepdvpij
+/32 [(l — age2pdvci + Hfs age2pdvpij
+/33 2Yi [(l - nf's) pdvci + Tlfspdvypi\
Finally, the terms not involving cf can be collected as:
C = /30 YiATlfs (pdvyi - pdvci)
+/31 Yi/SXlfs (agepdvyi — agepdvci)
+/32 YiMVfs (age2pdvyi — age2pdvc{)
+^3 Yi2AIltS (Pdvyyi ~ Pdv^)
+<5io AIlfspdvii + <5n AIlfsagepdvii + <512 ATlfsage2pdvii
+(?20 Allfspdvri + S21 AIlfsagepdvri + S22 AIlfsage2pdvri
+^30 AIlfspdvli + (S31 AIlfsagepdvk + $32 AIlfsage2pdvli + £j
28

-------
where AI^4,5 = — Ilfr's is a negative number for each of our risk reduction scenarios. If the error term
can be considered to be zero, the systematic portion of the difference in expected utilities can be solved to
yield point estimates of the option price.
Many practitioners currently use samples drawn from the joint distribution of the maximum likelihood
parameter estimates to generated simulated 90% confidence intervals for the option price predictions. In
this exercise, it is possible either to ignore the error term, or to replace it with a random draw from a unit
logistic distribution before computing the value of the C term for each replication. As usual for a quadratic
formula, fitted values of option price for each simulation will be given by:
A -B ± VB2 - AAC
C% ~	2A
If B2 — 4AC > 0, the equation has two distinct real roots. In cases where only one of these roots is positive,
the correct solution will be obvious. In models where j33 = 0, the formula for cf is simply linear, rather
than a quadratic form. The A term is zero, and the B term loses its component in j33 so that cf = —C/B
4.6 Fully quadratic marginal utilities
The discrete choice among program alternatives can thus be modeled as depending upon the marginal utility
of income and the marginal (dis)utilities of time in each health state. The marginal utility of income may
involve up to four parameters, f30, f31, f32, and /33, depending upon whether it is allowed to depend on both
the linear and squared values of the respondent's age in the future period when the income is to be enjoyed,
and on the level of income itself. Further generality will also be explored in this paper. In particular, the
age of the respondent at the time he or she is being asked to make these tradeoffs will be allowed to influence
the indirect utility function. The baseline marginal utility parameters /30, <5io, $20; and ^30 will be allowed
to shift with ageio and with age20, making indirect utility potentially fully quadratic in the respondent's
current age. Also, (31, (5n, 821, and £31 can be allowed to shift with ageio, which will allow for an interaction
29

-------
between current age and the age at which income or a particular health status is to be experienced. While
we do not expect, a priori, that each undiscounted marginal utility in our model will be fully quadratic
in both age now and age-at-event, we wish to allow the data to reveal nonlinearities, including maxima or
minima over the range of current ages or ages when income or health status is to be experienced.
From the simple undiscounted indirect utility function in equation ( ), it is necessary to go through several
steps to achieve the estimating form that can be used to explain respondent's choices among risk-reduction
programs. We see from equation ( ) that the difference in expected present value indirect utilities associated
with choosing a risk-reduction program is a function of the illness profile as captured by the pdvif, pdvrf,and
pdvlf terms, as well as a function of the individual discount rate assumed for each respondent. In this
analysis, we assume = r, the same for each respondent, and we conduct sensitivity analyses with respect
to the magnitude of this discount rate.
In our empirical application, equation (19) is the basis for estimation of the random utility choice model
that explains individuals' choices among the three alternatives presented in each choice scenario: Program
A, Program B, or Neither Program. There is an analogous difference in expected utilities between Program
B and the Neither Program choice. All choices posed to respondents were three-way choices, so the models
will be estimated using McFadden's conditional logit estimator (or appropriate modifications of this model).
4.7 From maximum annual payment to PDV of payment stream
The option price for the program that accomplishes this decrease in illness probabilities is the common certain
payment, regardless of which way the uncertainty about contracting the illness is resolved, that makes the
individual just indifferent between paying for the program and enjoying the risk reduction, or not paying for
the program and not enjoying the risk reduction. This payment, cf*, will make E [V^4] — -©[V^] = 0. The
amount of money cf* is the maximum constant annual payment that the individual will be willing to make,
regardless of whether he suffers the illness, in order to purchase the program that reduces his probability of
30

-------
suffering the illness from II^V'S to II^'S.
While the payment cf is the maximum annual payment the individual is willing to make, these payments
are necessary for the rest of the individual's life, so the present value of these payments must be calculated.
In this context, however, there is some uncertainty over just what will constitute "the rest of the individual's
life," since this may differ according to whether the individual suffers the illness or not. We will use the
expected present value of this time profile of costs, with the expectation taken over whether or not the
individual suffers the illness when they are participating in the program.
E
PV(cf) j	(20)
(i - n^) (^t)pdvct + (n^) (^)pdvpf
(5*) [(i - n^) pdvcf + (Pdvp?)\
In the case where the marginal utility of income is constant, so that = f32 = (3S = 0, the denominator of
the option price formula is just (30 [(l — II^'S) pdvcf + (pdvpf)] , so that capitalizing this payment over
the rest of the individual's life allows the terms in square brackets to cancel. The formula for the present
value of the streams of annual maximum payments willingly made to avoid a specified health profile reduces
31

-------
to:
^oYiAnf5, (pdvyi - pdvci)
+5ioAHfspdvii + 5nAHfs agepdvii + 5i2AHfsage2pdvii
+52o
-------
dummy variable, say Df, that indicates whether the health state occurs in alternative A. This new parameter
would describe the marginal utility of the generic health outcome to be avoided. This health outcome is
"sudden death this year" in many existing empirical studies. In this case, we would have:
= AILfsl3 ^[SDf]
= (<5//30) An/1'5 to avoid death (Df = 1)
= 0 for "no program," where (Df = 0)
When the marginal utility of income is heterogeneous across individuals, these simplifications are not possible.
The process of calculating the expected present value of program costs does not produce a term that cancels
with everything but /30. The expected present value can still be calculated, but the formulas will remain
functions of both (l —	and II^'S and the other arguments of the B term above.
4.9 Value of a statistical illness (VSI)
The expected present discounted value in equation (20) pertains to the maximum annual willingness to pay
for a small risk reduction, An^15. There is a tradition in the mortality valuation literature of ignoring the
size of the risk difference involved, AII^'S, and scaling each expected present value option price to the amount
that would correspond to a 100% risk difference. To convert our expected present value option price to
something that might be termed the "value of a statistical illness" (VSI), we could divide by the absolute size
of the risk reduction. In our study, all probability changes AII^'S are negative, while the absolute magnitude
of these changes will be positive. Multiplication by Anf-S / lAn^l will amount to multiplying by -1, which
will change the effective sign on each of the terms involving this ratio. Using the same abbreviations B
and C for the detailed expressions defined above, if the researcher desires measures of a quantity that is
comparable to traditional VSL estimates, the effective formula for the value of a statistical illness, in the
E
PV(cf)
33

-------
case where (53 = 0, will be:
E
VSI =
PV(C?)
Air451
C [(l — Ufs) pdvcf + (pdvpf^]
b I Air451
In the special case where the marginal utility of income is simply a constant, this formula simplifies to:
E
PV(cf)
AIL^I
— Po
-l
l30Yipdvli
—Siopdvii — Snagepdvii — S\2age2pdvii
—S2opdvri — S2\agepdvri — S22age2pdvri
—Ssopdvli — Ssiagepdvli — 5s2age2pdvli
An/
(21)
where we take advantage of the fact that pdvyi + pdvli = pdvci so that (pdvyi — pdvci) = —pdvli.
Across the distribution of the logistic error term, Si, the expectation is zero, so the expected value of a
statistical illness depends only on the systematic portion of equation (21). The VSI in this case will depend
upon the different marginal utilities of avoided periods of illness, recovered status, and premature death and
on the way these marginal utilities vary with age at the time each health status is experienced. It will also
depend upon the time profiles for each of these states as embedded in the terms pdvif, pdvrf, and pdvlf,
as well as agepdvif, agepdvrf, agepdvlf and potentially age2pdvif, age2pdvrf, age2pdvlf, and (implicit
in this model) upon the individual's own discount rate.14
In this simple model with a constant marginal utility of income, increases in income Yi will increase the
predicted point estimate of the VSI. The effect of income on VSlf is given by dVSlf /dYi = pdvlf which
is non-negative. Thus the effect of an increase in income on the predicted VSI will be larger (i.) as more
14 Subsequent work will preserve individual discount rates as systematically varying parameters, to be estimated with reference
to the individual's responses to a hypothetical "how to take your lottery winnings" question. Here, discount rates are presumed
to be exogenous and constant across individuals. Our empirical work explores the consequences of using different discount rate
assumptions.
34

-------
life-years are lost, (ii.) as the individual is older, so that life-years lost come sooner in time. The effect of
income on VSI can be estimated more generally if the marginal utility of income is not constant.15
The error term e in equation (??) is assumed to be identically distributed across observations in a manner
appropriate for conditional logit estimation. Given the transformation needed to solve for the VSI, however,
the error term in the VSI formula will be heteroscedastic, with smaller error variances corresponding to cases
with larger absolute risk reductions, |AII^'5| .
In expectation, the fitted value of a statistical illness can potentially vary systematically across types
of illnesses according to the labels assigned to the illnesses, the symptoms and treatment associated with
them, the individual's characteristics besides just age now and age-at-event, perceptions of risks associated
with the type of illness, and prior experience with that illness. This heterogeneity can be accommodated by
making the indirect utility parameters <5i, 82, and (53 depend upon other individual characteristics. In future
empirical models, the addition of illness labels and a symptom-treatment profile (within the illness state) will
convey to the respondent some information about what health consequences might ensue from each illness
we describe. These illness characteristics can be expected to shift the value of <5i, the marginal (dis)utility
of a sick-year. The marginal utility of each period of recovered health status, 82, could be allowed also to
vary by type of illness as well, since the illness labels may connote the degree of "health" that nominal
recovery from that illness actually implies. Finally, the marginal utility of a lost life-year may depend
upon the health state prior to death. In the meantime, readers should keep in mind that the essentially
randomized design of the illness profiles, conditional only on the individual's age and gender (and excluding
nonsensical combinations), ensures that omitted variables bias concerning attributes of each illness profile
15 Nothing in this specification precludes negative point estimates of the VSI. A positive VSI estimate will result if the
estimated value of the marginal utility of income, f3, is positive and there are negative values for the marginal utilities of
illness-years, recovered-years, and lost life-years (the (5s).
The key undiscounted marginal utility parameters are not presently constrained to be strictly positive (for income) and strictly
negative (for episodes of undesirable health profiles). This is especially a concern when these marginal utilities are permitted
to vary systematically with of the attributes of the illness profile and/or the characteristics of the individual in question. The
marginal utility of income, the scalar parameter /3 in our simplest models, bears a point estimate that is robustly positive, but
positive values for one or both of the systematically varying parameters capturing the marginal utility of an illness-year ((5-|) or
a lost life-year (£3) can push an individual fitted value of the VSI for a particular morbidity/mortality profile into the negative
range.
35

-------
will be minimized in this analysis.
4.10 VSIs versus Conventional VSLs
The existing literature, especially the hedonic wage-risk literature, focuses on society's willingness to pay for
incremental reductions in the chance of a sudden accidental death in the current period. In general, there
are no age effects, and agejo is the same thing as "age-at-event" (agen). In the framework of our illness
profiles, such an event would be captured by zero years of morbidity and death in the current year, with the
remainder of the individual's nominal life expectancy experienced as lost life-years. Since the terms in pdvif
and pdvrf will be zero, our analog to the conventional VSL formula will be simply:
E
E[VSL\ = —
where pdvlf = ^
PV(c
Anfs|
(1 +rf
-<*30 , v \
Yi pdvlf	(22)
The summation in the formula for pdvlf is from the present until the individual's nominal life expectancy.
This interval depends upon the individual's current age, so even in a model with homogeneous preferences,
the VSI will vary with age. The VSI also depends upon the individual's income, and of course, the individual's
discount rate will also matter. See Cameron and DeShazo (2003) for discussion of calculating policy-relevant
VSLs with this model.
5 Results and Discussion
For this paper, we examine the model in equation (19 ) and a number of its special cases. Our estimating
sample consists of stated preferences for 5 sets of three-way program choices provided by roughly 1320
respondents from an originally representative sample of roughly 2000 from the US population. 16
16 For this analysis we have dropped the choices of individuals who appear to have spent too little total time on the five
choice tasks to have allowed fully-considered selections. An Appendix details the consequences for parameter estimates in our
36

-------
Table 1 compiles estimation results for three different specifications estimated for three different assump-
tions about individual discount rates: 3%, 5%, and 7%. These rates were chosen based on the official
range of values recommended for benefit-cost analysis by the Science Advisory Board of the US EPA. For
each discount rate, we calculate the various present discounted value terms (capturing the time profiles of
morbidity and mortality) employed in the construction of variables for use in the estimating specification.17
5.1 Estimating Specifications
Our baseline model allows for the level of income to affect the marginal utility of additional income, but
excludes any age effects on the marginal (dis)utilities of health states. Our "No Age Effects" specification
E[Vf\-E[Vf] = /30
[cf] (-1) [(l - nf5) pdvci + Ilfspdvpi]
+Yi Allf's (pdvyi - pdvci)
icf] (_1) 2Yi [(l ~ nf5) pdv°i + Tlfspdvypi]
+[ct\2 [(l ~~ nf's) pdvci + Tlfspdvppi]
+Y?	{pdvyyi — pdvci)
+5i0AHfspdvii + <52oAIlf'spdvri + <530 Allf'spdvli + £
+/^3 *
(23)
Our "Linear Age Effects" model allows the marginal utility of income to be shifted by the respondent's
current age (ageio), and allows the marginal (dis)utility of a sick-year, a recovered-year and a lost life-year to
shift with both the respondent's current age (ageio) and the respondent's age at the time that health state
preferred specification as our criteria for rejecting observations are successively weakened, leaving more and more respondents
in the estimating sample. We focus on a subset of people we will characterize as "careful choosers who do not explicitly reject
the choice scenarios." Selectivity correction exercises are pending.
17In current models, we lean heavily on linearities that allow us to estimate our parameters using packaged software algorithms
for McFadden's conditional logit models.
37

-------
is being experienced (i.e. the "age-at-event," ageu):
E[V*]-E[V^] = (/300 + Poiageio)
+/^3
[cf] (-1) [(l - nf's) pdvci + Ilfspdvpi]
+Yi AIV^S (pdvyi - pdvci)
[cf] (-1) 2Yi [(l - nf5) pdvci + Ilfspdvypi]
+ [c^]2 [(l — n^) pdvci + Ilfspdvppij
+Y?AIlfs [pdvyyi — pdvci)
+(<5ioo + SWiagei0)AIlfspdvii + <5n AIlfs agepdvii
+(^200 + S2oiageio)AIlfspdvri + 52iATlfsagepdvri
+(^300 + S30iagei0)ATlfspdvli + <53i ATlfs agepdvk + Si
(24)
The most general model described in Table 1 is our "Quadratic Age Effects" model. This model retains
the same formulation for the marginal utility of income, but allows for each of the (dis)utilities of the three
38

-------
different health states to be fully quadratic in the respondent's age now (ageio) and age-at-event (ageu).
E[VtA] -E[VtN] = (/300 + floia9eio)
+/®3 *
[cf ] (-1) [(l - nf5') pdvci + Ilfspdvpi]
+liA^f's, (piiwyi - pdvci)
[cf] (-1) 2Y; [(l - nfs') pcfcc; + Ilfspdvypi\
+[cf ]2 [(l — nf5') pdvci + IIfspdvppi\
+Y?Allfs (pdvyyi — pdvci)
+(<5ioo + SWiagei0 + 8W2age%)Mlf's pdvii
+5iioATlfsagepdvii + <5m ATlfs agepdviiageio + Si2AIlfsage2pdvii
+(^200 + 52oiageio + 82o2age%)Mlf's pdvr{
+S210 AIlfs agepdvri + S211 ATlfs agepdvriageio + 522AIlfsage2pdvri
+(^300 + Ssoiageio + 8zo2age%)A~Nfs pdyli
+SsioAIlfsagepdvli + (JsiiAn^agepcfoZiagfeio + feAIlf'5age1pdvl{ + Si
(25)
The marginal utility of income should be positive, but is not constrained to be so. Our competing specifica-
tions also involve several parameters that describe the marginal (dis)utility of a sick year, a recovered year,
and a lost life-year. Intuitively, the marginal utility of a sick-year should be negative, but we do not enforce
this restriction. The estimated marginal utility of a lost life-year may also depend on several parameters,
and these parameters are also estimated freely from the observed choices, without sign restrictions. In
general, one would expect that the marginal utility of a lost-life-year would be negative.18 For our two
models with age effects, we will provide figures that show the systematic variation in these two marginal
utilities as a function of age-at-event, for each of a 25, 35, 45, 55, and 65-year-olds.
18 A positive marginal utility associated with a lost life-year might be expected only when the illness is question constitutes
a "fate worse than death." For certain illnesses, such as severe Alzheimers' disease, we might expect that death would "come
as a blessing." In any situation where the pre-death state was less onerous, however, we would expect death to be unwelcome,
and hence that the marginal utility of a lost life-year would be negative.
39

-------
5.2 Parameter Estimates
In Table 4 which present our parameter estimates, we will emphasize the middle set of three models, for
the 5% discount rate assumption. Our "No Age Effects" specification shows robust significance and the
expected signs on all five core parameters. The marginal utility of income is positive, but declines with the
level of income. The marginal utilities of sick-years, recovered years, and lost life-years are all negative,
and (somewhat surprisingly) each has a similar point value. The surprising result is that recovered years
are not interpreted by respondents to be equivalent to health pre-illness years. Despite our having intended
respondents to view these years as equivalent to health years, they do not. They seem to be imputing reduced
health or reduced function to these recovered years. The similarity in the magnitude of the marginal utility
of a sick-year and a recovered-year, however, may be due to the fact that the illnesses are described as major
life-threatening illnesses, including cancers, respiratory disease, and stroke, for example.
The "Linear Age Effects" model makes the main empirical point in this paper. In this model, the
respondent's current age is permitted to shift his or her marginal utility of income (see Figure 3A), and the
marginal utility of each health status is allowed to depend on the respondent's current age and on the age at
which they would experience each year of each health status. The marginal utility of income declines with
the current age of the respondent.19 The marginal utilities of sick-years and lost life-years are less negative,
the greater the current age of the respondent, but more negative with the age at which these health states
would be experienced, controlling for current age. These findings are fully consistent with the two main
hypotheses discussed in the theoretical section of this paper. For recovered years, the results are somewhat
less precise. Age-at-event makes the marginal utility of a recovered year significantly more negative, but
the respondent's age now has no statistically discernible effect upon the marginal utility of a recovered year.
The anticipated marginal utility of a recovered-year appears to be independent of the current age of the
19We have explored the consequences of allowing the marginal utility of income to depend upon age-at-event. However, a
noticeable proportion of fitted MU(Y) estimates are then negative. Negative MU(Y) produces nonsensical results for the implied
WTP for an avoided sick-year, recovered-year, or lost life-year, since the marginal utility of income acts as the denominator of
the WTP formula.
40

-------
respondent in these data.
One troubling feature of the Linear Age Effects models is the persistence of positive values for the marginal
utilities of all three health states for some future ages. These positive marginal utilities lead to negative
WTP estimates in those early future years and will tend to bias downward the present value employed as an
estimate of the Value of a Statistical Illness (VSI). Figures 3B, 3C, and 3D show that, for example, WTP
to avoid a statistical sick-year, recovered-year, and lost life-year for a currently 25-year-old respondent (the
line tagged with "25") appear to be negative for the first few years into the future. We suspect that many
respondents, feeling currently rather healthy, doubt that the health risk we describe will actually affect them
in the next 5-10 years, although the possibility of becoming ill in the years beyond that is more credible.
It is not clear whether this should be interpreted as a form of scenario rejection in response to our stated
preference choice scenarios, or whether this is a legitimate property of people's preferences.
Recall that there is no opportunity for any respondent to express a negative willingness to pay explicitly.
At a minimum, respondents can imply that the value they place on a program is zero (i.e. no greater than
the cost of the Neither Program alternative, available at zero net cost). To determine whether these negative
fitted WTP estimates in the linear models are merely an artifact of a too-restrictive functional form, we
estimate a specification that allows the marginal utilities associated with all three health states to be fully
quadratic in both age now and age-at-event.
It would be desirable, in our quadratic model, also to allow the marginal utility of income to be a fully
quadratic function of both age now and age-at-event. However, as Figure 4A reveals, generalizing the
marginal utility of income in this way leads to occasional negative fitted values for the marginal utility of
income. Since this marginal utility serves as the denominator in WTP calculations, negative and zero
values are particularly problematic. Pending further exploration of models that restrict the marginal utility
of income to be strictly positive, we revert to the simpler specification where the marginal utility of income
depends only upon current age.
41

-------
For the 5% discount rate, the "Quadratic Age Effects" model reveals individually statistically significant
point estimates on the quadratic and interaction terms in age-at-event for sick-years. It also reveals sta-
tistically significant point estimates on the age-now term and the interaction term for lost life-years. None
of the additional parameters for recovered-years is individually statistically significant, but the maximized
log-likelihood increases by almost seven.
Figures 4B, 4C, and 4D reveal the consequences of allowing a more general functional form. For each
current age, the only relevant portions of these curves lie to the right of that current age. These diagrams
strongly suggest that most respondents place zero value on avoiding a sick-year that will occur prior to their
50s. They may tend to believe, on average, that they will remain healthy until their 50s. Respondents who
are currently younger place higher value on avoiding future sick-years at specified ages than do currently
older respondents (for those same specified ages). Similar patterns, to a greater or lesser degree, are apparent
for recovered-years and lost life-years.
5.3 Potential Extension
The Quadratic Age Effects specification creates a strong impression that it will be desirable to break away
from linear-in-parameters models, in spite of their extremely attractive properties for ease of estimation.
In particular, our next task is to specify a non-linear model wherein we estimate the logarithms of the
marginal utilities of income and years in each health state, rather than their absolute levels. The logarithmic
transformation will prevent the fitted marginal utility of income from going negative and will prevent the
marginal utility of a sick-year, a recovered-year, and a lost life-year from being positive. In the simple case
with no age effects, it seems appropriate to specify a model of undiscounted utility of the form:
Vit = exp[/30 +/33yit] f{Ylt)	(26)
- exp [<5io] ilia - exp [<5i0] rcvit - exp [<530] lylit + Vu-
42

-------
This form constrains the marginal utility of income to be positive, equal to exp [/30 + P3Yit] ¦ It also constrains
to be negative the marginal utility from each health state [illitlrcvit,lylit\ in each year. In the more
general case where all marginal utilities are fully quadratic in the respondent's current age, age^, and the
respondent's future age-at-event, ageu,the undiscounted utility will be of the form:
Vit = exp [/300 + f301agei0 + l302age2M + l3wageit + Pnageitagei0 + (32age% + (33Yit\ f(Yit) (27)
-	exp [
-------
assumption ($2.6-$2.8 million). For the 7% discount rate model, it is lower ($1.6-$1.65 million).
How do the WTP results from our model compare to those of earlier VSL results? Many hedonic wage
estimates of "the" VSL estimate wage-risk tradeoffs for middle-aged white males in blue collar jobs. For
comparison with earlier results, we should consider just the VSI for an illness profile consisting of sudden
death at age 45 for a 45-year-old. However, in order to highlight the generality of our WTP models,
compared to earlier VSL models, we will consider four classes of simulations:
Simulation 1. How would a 25-, 35-, 45-, 55-, and 65-year-old value a reduction in the chance of sudden
death starting now?
Simulation 2. How would a 25-, 35-, 45-, 55-, and 65-year-old value a reduction in the chance of sudden
death starting 5 years from now?
Simulation 3: How would a 25-, 35-, 45-, 55-, and 65-year-old value a reduction in the chance of sudden
death starting at age 70?
Simulation 4. How would a 25-year-old value a reduction in the chance of sudden death starting 5, 15,
25, 35 and 45 years from now?
Table 6 summarizes the results of these four classes of simulations for the No Age Effects model, the
Linear Age Effects model and the Quadratic Age Effects model. For each simulation, we make 1000 random
draws from the joint distribution of the maximum likelihood conditional logit parameters. For each set of
parameter values, we calculate the desired VSL We report the median of this distribution, as well as the
5th and 95th percentiles.
The No Age Effects model in Table 6 is our model that conforms as closely as possible to most previous
studies. This model does not differentiate the marginal utility of a lost life-year according to the age of the
respondent now or the age the respondent would have been during each life-year lost. The median VSI can
be expected to differ with the respondents current age, however, because our model emphasizes life-years
and involves discounting. Remarkably, despite these differences from previous models, our median VSI for
44

-------
sudden death for a 45-year-old is $6.82 million, with simulated 90% confidence bounds of ($5.34 million to
$8.83 million). This range of estimates compares very closely to the $6.1 million estimate used routinely by
the US EPA in their major benefit-cost analysis. Evidence for any sort of a "senior death discount" is sparse
in simulations 1 and 2. The medians decline monotonically with the current age of the respondent, but the
differences are small. In Simulations 3 and 4, there are larger effects. In Simulation 3, we see substantial
increases in the VSI for sudden death at age 70 as the respondent is closer to 70 in age. In Simulation 4,
where 25-year-olds are asked to consider risks of sudden death at increasingly distant future times, the VSI
falls substantially and significantly.
However, our data emphatically reject the No Age Effects model in favor of a model that acknowledges
the systematic variation of WTP for risk reductions with respect to the respondent's age now and the age at
which they would experience future lost life-years. The second column of VSI results in Table 6 reveals, for
the Linear Age Effects model, a considerably lower median VSI of $2.08 million for sudden death this year
for a 45-year-old. The bootstrapped confidence interval is wide, and admits for values as low as $50,000 and
as high as $4.42 million. However, one must keep in mind that fitted WTP for avoided adverse health states
is negative during the "early future" for the inflexible Linear Age Effects models. These spurious negative
values will tend to bias downward our estimates of VSI for each age group.20
In Table 6, for simulation 1 under the Linear Age Effects model, the decline in WTP with the respondent's
current age is evidenced in the lesser VSI associated with increasing current age. These calculations suggest
the presence of a "senior discount" in WTP to avoid sudden death.21 This same decline with current age is
exhibited in simulation 2 (sudden death in 5 years). In simulation 4, because of discounting, WTP to avoid
sudden death in more remote future years also falls. The progression in WTP is non-monotonic, however,
for simulation 3 which pertains to sudden death at age 70 for people of different ages now.
20	Less biased estimates await constrained estimation of a model featuring a positive marginal utility of income and negative
marginal utilities of adverse health states.
21	Perhaps, however, sudden death is viewed as less likely for older respondents. It is possible that respondents substitute
lower risks than the survey instrument suggests, interpreting their own risk to be lower than the "average" that they assume is
being quoted in the survey.
45

-------
For the Quadratic Age Effects model in Table 6, however, the situation is rather different. First of all, the
bootstrapped confidence intervals are even wider because of the greater number of statistically insignificant
parameter estimates in this specification. The quadratic models also allow WTP for avoided sick-years,
recovered-years, and lost life-years to increase much more quickly with age-at-event than they do in the
Linear Age Effects models. This can lead the positive effect of age-at-event to dominate the negative effect
of age-now on VSIs over some parts of the range of simulations. However, one must keep in mind that the
estimated VSIs may be biased (ambiguously) because the quadratic forms can fit slighted negative or slightly
positive values for the undiscounted WTP for avoided sick-years, recovered-years, and lost life-years when
the true value probably ought to be positive but very close to zero.
In Table 6, none of the simulated VSI progressions based on the Quadratic Age Effects model are
monotonic. This is a consequence of the countervailing positive effect of age-at-event, and the negative
effect of age now, on undiscounted WTP for future years in each health state. These processes are further
confounded by the discounting process.
6 Conclusions
Policy analysis with respect to risk-management programs requires detailed information about consumer
demand for these programs. We begin with a concise theoretical model, adapted from Ehrlich (2001) that
produces two key insights. First, individuals will derive increasing marginal utility from reducing risks that
they will face later in life, which implies that individuals will be willing to pay more to reduce risks that will
afflict them when they are older (and correspondingly less to reduce risks that will afflict them when they
are younger). The second insight is that health and other consumption goods are likely to be complements.
As individuals age, they learn more about the extent of complementarity between health and other goods-in
particular, they learn that future consumption will provide less utility because of declining physical well-
being. Hence they are inclined to shift more consumption forward in time and their willingness to pay for
46

-------
health risk reductions will fall as they are older.
Which of these two countervailing effects will dominate is an empirical question, so we have set out to build
a formal utility-theoretic model that captures the relevant considerations in private ex ante consumer choices
about incurring ongoing expenditures to reduce risks to life and health. Most past studies have focused on
current-period costs and current-period benefits. In contrast, our model recognizes the future time profiles of
illnesses and injuries for which individuals may choose to act to reduce their risks. Intertemporal consumer
optimization requires explicit treatment of the interaction between disease latencies and individual discount
rates. Our model permits us to derive option prices for programs that reduce well-defined types of risks.
Option prices are the appropriate theoretical construct for decision-making under uncertainty, where the
uncertainty in this case concerns whether the individual will actually suffer the illness or injury that the
proposed risk reduction measure addresses.
While we believe that it is important to preserve information about the nature of the risk reduction
involved (its size, and perhaps the baseline risk), we show that our option price WTP formulas lead naturally
to what we have labeled as the "value of a statistical illness" (VSI). The VSI is the present discounted value
of the stream of maximum annual payments that the individual would be willing to pay for the specified
(typically small) risk reduction, scaled up proportionately to correspond to a risk reduction of 100%. This
construct is analogous to the more familiar, but more-limited, concept of the value of a statistical life (VSL).
A VSL is typically constructed by looking simply at the static single-period willingness to pay for a specified
risk reduction, and scaling this willingness to pay up to a 100% risk reduction. However, static VSL estimates
do not typically vary with important morbidity/mortality attributes such as latency, time profiles of illness,
symptoms and treatments, outcomes, or life-years lost.
In the empirical analysis presented in this paper, we first consider a model wherein preferences are
considered to be homogenous across all types of individuals and where the marginal (dis)utility of a sick-year
or a lost life-year is independent of the respondent's age now and his or her age at the time he or she would
47

-------
be experiencing that health state (or the age that they would have been, had they not died prematurely).
Even these very simple models can be used to display the sensitivity of option prices to the timing of events
in an illness profile. The pattern of future health states in question matters for willingness to pay to avoid
different types of risks to life and health..
Our empirical analysis also demonstrates conclusively that the current age of the respondent, as well as
the prospective age at which they will experience illness or premature death, will have a systematic effect
on willingness to pay for programs that reduce health risks. These findings are relevant to the current
debate about whether there should be a "senior death discount" in assessing the health benefits of costly
risk reductions. The choices made by the individuals in our sample strongly suggest that, ceteris paribus,
the older an individual is when asked to begin paying for a particular health risk reduction, the less he or she
will be willing to pay. However, this tendency can be confounded by the fact that for individuals of a given
age, willingness to pay for health risk reductions increases with the age at which these health risks would
be experienced. Any given individual, looking forward, may feel that they would be willing to pay more to
reduce risks to their health that materialize when they are older. This tendency may feed the intuition that
the benefits of risk reductions should be, if anything, higher for older persons. However, across individuals
of different ages, individuals who are older seem willing to pay less to reduce risks to their health.
48

-------
7 References
Alberini, A., M. Cropper, A. Krupnick, N. Simon (2002) "Does the Value of Statistical Life Vary with Age
and Heath Status?" Resources for the Future Discussion Paper 02-19, Washington, DC. April.
Arthur, W. B. (1981). "The economics of risks to life." American Economic Review 71: 54-64.
Cameron, T.A. and J.R. DeShazo (2003) "Toward a General Model of Demand for Healthy, Morbidity
and Mortality States" Mimieograph.
Conley, B.C. "Value of Human Life in Demand for Safety" American Economic Review 66 (1): 45-55
1976
DeShazo, J.R. and T.A. Cameron (2003) "A Biological Model of Discounting" Mimieograph.
Dreze, J. (1962) ""L'Utilite Sociale d'une Vie Humaine"." Revue Francaise de Recherche Operationalle
6: 93 - 118.
Ehrlich, I. and H. Chuma (1990). "A model of the demand for longevity and the value of life extension."
Journal of Political Economy 98: 761-782.
Ehrlich, I. and Y. Yin (1999). Endogenous Longevity: Assessing the Role of Life Protection as a Deter-
minant of Life Expectancy and the Related Inter-Temporal Choices, mimeo.
Ehrlich I (2000) "Uncertain lifetime, life protection, and the value of life saving" Journal of Health
Economics 19, 341-367.
Ehrlich I (2001) "Erratum to "Uncertain lifetime, life protection, and the value of life saving" [Journal
of Health Economics 19 (2000) 341-367]" Journal of Health Economics 20 459-460
Can, L., M. Hurd, et al. (2003). "Individual Subjective Survival Curves". Mimeograph.
Gfellner, B. (1989). "Perceptions of Health, Abilities, and Life Satisfaction Among Very Old Adults."
Perceptual and Motor Skills 68: 203-209.
Chez, G. R. and G. S. Becker (1975). The Allocation of Time and Goods over the Life Cycle. New York,
Columbia University Press.
49

-------
Graham, D.A. (1981) "Cost-benefit Analysis Under Uncertainty." American Economic Review 71 (4):
715-725 1981
Grossman, M. (1972). The Demand for Health: A Theoretical and Empirical Investigation. New York,
Columbia University Press.
Jones-Lee, M. W. (1982). The Value of Life and Safety. Amsterdam, North Holland.
Judd, K. (1998). Numerical Methods in Economics. Boston, MIT Press.
Ried, W. (1998). "Comparative Dynamic Analysis of the Full Grossman Model." Journal of Health
Economics 17: 383-425.
Rosen, S. (1988). "The value of changes in life expectancy." Journal of Risk and Uncertainty 1: 187-203.
Hamermesh, D. S. (1985). "Expectations, Life Expectancy, and Economic Behavior." Quarterly Journal
of Economics 100 (2): 389-408
Liu, J.-T. and J.K. Hammitt (2003) "Effects of Disease Type and Latency on the Value of Mortality
Risk," NBER Working Paper No. wl0012.
Johansson, P. O. (2002). "On the definition and age-dependency of the value of a statistical life." Journal
of Risk and Uncertainty 25(3): 251-263.
Jones-Lee, M. (1974) "The Value of Changes in the Probability of Death or Injury." Journal of Political
Economy 82(4): 835 - 849.
Jones-Lee, M. W., M. Hammerton, et al. (1985). "The Value of Safety: Results of a National Sample
Survey." Economic Journal 95: 49-72.
Kenney, R. A. (1989). Physiology of Aging: A Synopsis, 2nd ed. Chicago, Year Book Medical Publishers.
Kirkwood, T. B. L. (1977). "Evolution of Ageing." Nature 270: 301-304.
Kirkwood, T. B. L. and M. R. Rose (1997). "Evolution of senescence: late survival sacrificed for re-
production." Philosophical Transactions of the Royal Society of London Series B-Biological Sciences 332
(1262): 15-24.
50

-------
Krupnick, A., A. Alberini, et al. (2002). "Age, Health and the Willingness to Pay for Mortality Risk
Reductions: A Contingent Valuation Study of Ontario Residents." Journal of Risk and Uncertainty 24:
161-186.
Mrozek, J. R. and L. O. Taylor (2002) "What Determines the Value of Life? A Meta-Analysis." Journal
of Policy Analysis and Management 21(2): 253 - 270.
Posner, R. A. (1995). Aging and Old Age. Chicago, University of Chicago Press.
Schelling, T. C. (1968). "The life you save may be your own," in. Problems in Public Expenditure
Analysis. S. B. Chase. Washington, DC, Brookings Institute.
Sheppard, D. S. and R. Zeckhauser (1984). "Survival versus consumption." Management Science 30:
424-439.
Skrzycki, C. (2003). Under Fire, EPA Drops the 'Senior Death Discount'. Washington Post, Tuesday,
May 13, 2003, E01..
Smith, V. K., M. F. Evans, et al. Do the "Near" Elderly Value Mortality Risks Differently? Mimeograph.
Sozou, P.D. and R.M. Seymour (2003) "Augmented discounting: interaction between ageing and time-
preference behaviour" Proceedings of the Royal Society of London, Series B-Biological Sciences, 270 (1519):
1047-1053.
U.S. Environmental Protection Agency (1999). The Benefits and Costs of the Clean Air Act 1990-2010,
Report to the U.S. Congress.
Viscusi, W. K. (1993) "The Value of Risks to Life and Health." Journal of Economic Literature 31: 1912
- 1946.
Yaari, M. E. (1965). "Uncertain Lifetime, Life Insurance and the Theory of the Consumer." Review of
Economic Studies 32(2): 137-150.
51

-------
Appendices A and B are available from the authors upon request.
52

-------
Table 1
Descriptive statistics for Risk Reduction Programs
Variable
Mean
Std. Dev.
Min
Max
Risk Reduction Programs
Present discounted sick-years
Present discounted recovered-years
Present discounted lost life-years
Monthly cost
Risk change
2.236
0.4746
2.596
$31.03
.0034124
2.525
1.356
2.938
29.46
.0016695
0
0
0
2
-0.006
16.277
14.589
17.803
140
-0.001
Respondents
income
age
$ 50,606
51.60
33,533
15.11
5,000
25
150,000
93
53

-------
Table 2
Distribution of Program Characteristics within Illness Types
Breast Prostate Lung Colon Skin Heart Heart	Respiratory	Alzheimer's
Variable	Cancer Cancer Cancer Cancer Cancer Attack Disease Stroke Disease Diabetes Disease
N=
599
548
1111
1107
1122
1144
1150
1154
1108
1097
1103
Cost
31.649
29.095
31.611
30.866
31.162
30.677
30.148
30.724
30.888
30.290
30.480
baseline_risk
0.0167
0.0155
0.0166
0.0159
0.0169
0.0166
0.0167
0.0165
0.0162
0.0156
0.0161
risk_change
-0.0033
-0.0035
-0.0034
-0.0035
-0.0035
-0.0035
-0.0034
-0.0034
-0.0034
-0.0033
-0.0034
mod_pain_duration
41.68
38.79
64.55
69.78
75.35
29.62
84.81
31.61
56.76
54.76
55.53
sev_pain_duration
18.32
18.84
34.15
30.35
13.89
10.70
37.19
10.76
31.73
23.59
25.09
hospital_duration
2.190
2.287
2.184
2.167
2.054
1.012
2.129
0.839
1.950
2.157
27.177
hosp_open_end
0.000
0.000
0.026
0.034
0.031
0.014
0.047
0.095
0.037
0.013
0.073
minor_surgery
0.329
0.336
0.320
0.476
0.543
0.441
0.344
0.432
0.337
0.000
0.000
major_surgery
0.331
0.314
0.357
0.524
0.457
0.159
0.312
0.167
0.292
0.000
0.000
latency
16.66
18.13
18.69
18.19
17.33
20.15
18.97
21.25
20.95
17.83
21.96
life-years lost
11.072
11.535
10.175
8.388
9.725
13.191
7.070
11.920
7.492
13.235
8.840
die_suddenly
0.000
0.000
0.000
0.000
0.000
0.523
0.000
0.510
0.000
0.000
0.000
die_sick
0.404
0.339
0.354
0.224
0.291
0.073
0.106
0.063
0.204
0.861
0.842
die_after_chronic
0.000
0.000
0.395
0.380
0.298
0.210
0.624
0.231
0.410
0.139
0.158
recover
0.596
0.661
0.251
0.396
0.412
0.194
0.270
0.196
0.386
0.000
0.000
54

-------
Table 4
Sensitivity of Parameter Estimates to Alternative Discount Rate Assumptions
3% discount rate	5% discount rate	7% discount rate
Parameter and description
No
Linear
Quadratic
No
Linear
Quadratic
No
Linear
Quadratic
of variable(s)
Age
Age
Age
Age
Age
Age
Age
Age
Age

Effects
Effects
Effects
Effects
Effects
Effects
Effects
Effects
Effects
/?00 (linear net income term)
3.83E-05
6.11E-05
0.0000726
4.62E-05
8.29E-05
0.0001009
5.336E-05
9.658E-05
0.0001293

(8.60)***
(4.20)***
(4.54)***
(8.31)***
(4.37)***
(4.81)***
(7.97)***
(4.14)***
(4.94)***
Pm (agei0 interaction)

-3.65E-07
-5.44E-07

-5.61 E-07
-8.34E-07

-6.296E-07
-1.119E-06


(-1.64)
(2.15)**

(1.98)**
(2.58)***

(1.84)*
(2.83)***
/?3 *E-9 (DMU(Y) term)
-0.1350
-0.1514
-0.1468
-0.2130
-0.195
-0.1917
-0.2670
-0.2398
-0.2378

(4.40)***
(4.08)***
(3.95)***
(4.62)***
(4.17)***
(4.09)***
(4.78)***
(4.22)***
(4.17)***
Sick years









^100 An fpdvi,
-7.4602
0.0323
-69.0238
-9.6248
2.0959
-85.1591
-11.582
3.6529
-105.5609

(6.01)***
(0.00)
(-1.40)
(5.41)***
(-0.17)
(-1.35)
(4.79)***
(-0.23)
(-1.33)
SmAUfpdvitxaget0

0.6526
-2.1009

1.3143
-3.6357

2.2562
-6.0954


(4.22)***
(-1.52)

(5.04)***
(1.68)*

(5.43)***
(1.86)*
^102 &nf pdviixage?0


0.0018


-0.0243


-0.0931



(-0.13)


(-1.01)


(2.22)**
8X j o Allf5agepdvii

-0.5568
3.5999

-1.1362
5.3522

-1.9604
8.0433


(2.76)***
(1.72)*

(3.55)***
(1.77)*

(4.04)***
(1.88)*
8luMlf agepdvii xagei0


0.0352


0.1025


0.2536



(-1.13)


(1.86)*


(2.66)***
5nmfage2 pdvii


-0.0428


-0.0842


-0.167



(1.861*


12.23)**


(2 731***
55

-------
Recovered years
^200 ^nfpdvrt
S2inAnf pdvrt Xagetll
S2ll2M\ f pdvr x ageiti
S2WAnfagepdvrt
S2UATlfs agepdv^ *agej0
S22 Milage! pdvrj
Lost life-years
^300An fpdvl,
^301An fpdvli xagel0
^302Anfpdvl, xagei0
S3 j 0 kl\f agepdvi
S3llAUfagepdvlt x age^
S32Anfage2 pdvi
Alternatives
Log L
-6.4233 47.7764 -41.111
(2.86)*** (2.59)*** (-0.47)
0.2011 -3.5696
(-0.67) (-1.16)
-0.0048
(-0.16)
-0.9283 4.6351
(2.31)** (-1.12)
0.0592
(-0.85)
-0.062
(-1.28)
-6.9292 11.0571 16.3833
(7.70)*** (-1.29) (-0.34)
0.6312 -1.7962
(5.13)*** (-1.25)
-0.0177
(-1.49)
1.0102
(-0.47)
0.0563
(1.87)*
-0.0315
(-1.35)
19788 19788 19788
-7175.195 -7146.39 -7141.535
-0.6921
(3.88)***
56
-9.3288 65.9143 -82.4354
(2.70)*** (2.49)** (-0.69)
0.5672 -7.3201
(-1.11) (-1.47)
-0.0238
(-0.45)
-1.5232 9.1315
(2.34)** (-1.43)
0.1453
(-1.17)
-0.1318
(-1.59)
-12.691 86.569 -143.7686
(2.51)** (2.37)** -0.92
1.1393 -13.751
(-1.36) (1.76)*
-0.0807
(-0.87)
-2.3448 16.5341
(2.32)** (1.74)*
0.3319
(-1.55)
-0.2643
(1.92)*
-9.4543
(6.70)***
17.6609
(-1.45)
1.2888
(5.80)***
-1.3365
(4.56)***
44.1991
(-0.68)
-2.3208
(-1.02)
-0.0525
(2.41)**
0.7263
(-0.22)
0.1225
(2.31)**
-0.0579
(-1.51)
-11.9302
(5.75)***
24.7046
(-1.5)
2.2547
(5.96)***
-2.2651
(4.89)***
81.9856
(-0.97)
-2.6087
(-0.75)
-0.1224
(3.13)***
0.0092
0
0.2461
(2.68)***
-0.1064
(1.71)*
19788 19788
7186.375 -7149.029
19788
-7142.45
19788 19788
-7196.076 -7154.093
19788
-7143.988

-------
Table 5
Sensitivity of Fitted VSIs in Estimating Sample to Alternative Discount Rate Assumptions


3% discount rate

5% discount rate

7% discount rate

No
Linear
Quadratic
No
Linear
Quadratic
No
Linear
Quadratic
Descriptive Statistic
Age
Age
Age
Age
Age
Age
Age
Age
Age

Effects
Effects
Effects
Effects
Effects
Effects
Effects
Effects
Effects
Sample mean VSI ($ million)
4.17
4.09
4.65
2.2
3.65
8.92
1.96
2.69
2.11
Sample 5th %
0.13
0.03
0
0
0
0
0
0
0
Sample 25th %
1.21
1.26
1.45
0.61
0.96
1.09
0.36
0.71
0.82
Sample 50th %
2.4
2.62
2.78
1.54
2.01
2.11
1.07
1.59
1.65
Sample 75th %
4.16
4.28
4.13
2.88
3.29
3.11
2.29
2.68
2.46
Sample 95th %
11.74
8.51
8.25
6.76
6.65
6.16
6.4
5.46
4.72
In the choice scenarios presented to respondents, there was no opportunity for any individual to express a negative willingness to pay for
a program. At most, they could choose the other alternative, or "Neither Program." As a consequence, for these descriptive statistics, we
interpret negative fitted point values of the VSI for a particular program as zero values, both in computing the marginal mean and in
describing the percentiles of the marginal distribution.
57

-------
Table 6
VSI for Four Classes of Sudden Death Scenarios (US $ million)
No Age Effects	Linear Age Effects Quadratic Age Effects

Age
Now
Age at
Death
Latency
50% ( 5%,95%)
50% ( 5%,95%)
50% ( 5%,95%)
1. Simulation:
25
25
0
7.40
5.79,9.58)
2.94
0.08,6.34)
1.32
-4.03,7.55)
Sudden death
35
35
0
7.19
5.62,9.31)
2.74
0.35,5.53)
2.90
-0.87,7.20)
this year
45
45
0
6.82
5.34,8.83)
2.08
0.05,4.42)
3.59
0.69,6.89)

55
55
0
6.36
4.97,8.24)
1.35
-0.58,3.34)
3.98
1.19,7.33)

65
65
0
5.68
4.44,7.36)
0.15
-2.24,2.42)
3.70
0.12,7.50)
2. Simulation:
25
30
5
5.71
4.47,7.40)
4.00
2.21,6.57)
2.11
-0.39,5.42)
Sudden death
35
40
5
5.50
4.31,7.13)
3.86
2.41,5.86)
3.07
1.63,5.03)
in 5 years
45
50
5
5.13
4.02,6.65)
3.34
2.25,4.77)
3.27
2.17,4.65)

55
60
5
5.67
3.66,6.06)
2.72
1.70,3.86)
3.19
1.99,4.65)

65
70
5
4.00
3.12,5.18)
1.75
0.41,3.04)
2.49
0.96,4.10)
3. Simulation:
25
70
45
0.49
0.38,0.63)
1.36
1.00,1.96)
2.08
1.45,3.01)
Sudden death
35
70
35
0.82
0.64,1.06)
1.99
1.53,2.64)
2.51
1.90,3.36)
@ fixed age (70)
45
70
25
1.34
1.04,1.73)
2.53
2.02,3.22)
2.57
2.05,3.19)

55
70
15
2.32
1.81,3.01)
2.98
2.46,3.66)
2.54
1.84,3.28)

65
70
5
4.00
3.12,5.18)
1.75
0.41,3.04)
2.49
0.96,4.10)
4. Simulation:
25
30
5
5.71
4.47,7.40)
4.00
2.21,6.57)
2.11
-0.39,5.41)
Sudden death
25
40
15
3.36
2.63,4.36)
4.32
3.22,6.07)
3.32
2.09,5.19)
varying latency
25
50
25
1.92
1.50,2.48)
3.52
2.65,4.90)
3.63
2.61,5.26)

25
60
35
1.03
0.80,1.33)
2.41
1.81,3.34)
3.11
2.25,4.46)

25
70
45
0.49
0.38,0.63)
1.36
1.00,1.92)
2.08
1.45,3.01)
NOTE: Based on 1000 random draws from joint distribution of estimated parameters. Models do not restrict the signs of
parameters and do not restrict the sign of fitted VSI to be non-negative; no negative or zero values drawn for marginal
utility of income
58

-------
Examples of Illness Profiles
Figure 1: A nonfatal illness (with recovery) that reduces life expectancy
preit
Pre-illness_yearjt
1111111111
00000000000
00000000000000000
0000000
sickjt
lllness_yearjt
0000000000
11111111111
00000000000000000
0000000
rcvn
Recover_year,t
0000000000
00000000000
11111111111111111
0000000
ly'it
Life-yea r_lostjt
0000000000
00000000000
00000000000000000
1111111

Health status -*




Time from	0	onset	recovery	death nominal life
now	to	tr	td expectancy, te
Figure 2: A fatal illness
pre/t
Pre-iiinessjyearjt
1111111111111111
00000
0000000000000000000000000
sickjt
lllnessjyear,t
0000000000000000
11111
0000000000000000000000000
rcvjt
Recover_year,t
0000000000000000
00000
0000000000000000000000000
ly'it
Life-yea r_lostjt
0000000000000000
00000
1111111111111111111111111

Health status -»


Time from	0	onset death	nominal life
now	to	td	expectancy, te
59

-------
b0_sim25
bO sim45
b0_sim35
bO sim55
.00006
a .00005
•s .00004
25 25 25 25 25 25 25 25 25 25 25 25 25 25 25 25 25
35 35 35 35 35 35 _ 35 35 35 35 35 _ 35 35 35 35 35 35
45 _45_ 45_ _45_ 45_ _45 _ 45_ _45_ 45_ _45 _ 45_ _45 _45_ 45_ _45_ 45
55 55 55 55 55 55 _ 55 55 55 55 55 _ 55 55 55 55 55 55
.00003
65 65 65 65 65 65 _ 65 65 65 65 65 _ 65 65 65 65 65 65
H	i	i	i	i	i	i	i
25 35 45 55 65 75 85 95
agecount
Marginal Utility of Income
Figure 3 A
o
if
2.5
2
1.5
1
.5
0
-.5
-1
-1.5
-2
-	wtpr_sim25
-	wtpr_sim45
25-	-55- '65-
35 t H ^ 65
45--==-- 65

-	wtpr_sim35
-	wtpr_sim55
25-§g:^r 45-55.
45 -55- ^ -
55'
65'
§5-'
65
65'
25
35
45
85
55 65 75
agecount
WTP to Avoid a Statistical Recovered-year
95
Figure 3C
wtpi_sim25
wtpi_sim45
wtpi_sim35
wtpi_sim55
2.5
2
1.5
1
.5
0
-.5
-1
-1.5
-2
25--It "It45
25
35-
45-
55-
65-
H
25
25
45-11-55
45-'55,-5^" £5
oc 55--ST 35-^r 4S--- ^5-' .65-
25-— "35—^ 45-- 55— 65-
25-f^ 35 45 45 ^ 55 §5- '
55 65
65-"
35-
-45-
55-
65-
-35
-45
55-
35
Jt5
55
65-'
65-
-45-
55-
65-
3^
55-
55
65-'
65
55-'
65
65-'
65-
65
35 45 55 65 75 85
agecount
WTP to Avoid a Statistical Sick-year
95
Figure 3B
-	wtpl_sim25
-	wtpl_sim45
-	wtpl_sim35
-	wtpl_sim55
2.5
o
if
1.5
-1
-1.5
25-
g l
25 11 35 -5 45
35-45-
45-"55-
55 -
-25
25-
-25--35-35--^5,-^5 55-- 65
- -55-
55-65-
65-
65-'
--BJ5-
35
-45-
55
25-2?-"35-55" "45-	§5
-S-SS-35- ^--45-„ 55-55 ^
45
55-
65-
45-
55-
-45-
55-'
55
§5-
65-
65'
65-'
65-
65-
65-
65-'
65
25
35
45
55
75
85
65
agecount
WTP to Avoid a Statistical Lost Life-year
95
Figure 3D

-------
b0_sim25
bO sim45
b0_sim35
bO sim55
.0015
.001
.0005
-.0005
25


65
55
25'


3,5
35-'
55-'
65
35'
"-65
55
-55
45 ~45_ .45
35-3S-J5-
-65
55
-65
-65
45-'
55^
45-
45
25"
25"" J35"
2s-" 35-	45
25 35 35 45 45
«	45,3b-
"65 65_-65_65_-65
55-
55
25 35 45 55 65 75
agecount
Marginal Utility of Income
85
95
Figure 4A
-	wtpr_sim25
-	wtpr_sim45
-	wtpr_sim35
-	wtpr_sim55
55 65 75
agecount
WTP to Avoid a Statistical Recovered-year
Figure 4C
-	wtpi_sim25
-	wtpi_sim45
6x5
-	wtpi_sim35
-	wtpi_sim55
"65
55
65
55
'5§
v65
^45
--35
¦45
•55
-45
-251'
Ji:

25
^5
25 ^45
x-35^-55
M/fA 5
25.-^5- 4> ^
Jg>45" ® /
"55""
25
35
45
55
75
65
agecount
WTP to Avoid a Statistical Sick-year
85
95
Figure 4B
o
if
-	wtpl_sim25
-	wtpl_sim45
65
6?
65
2.5
2
1.5
1
.5
0
-.5
-1
-1.5
-2
55
X5
"5§
65
4§
35
25
""5§
-45
"-5§
-65
-¦55
1 S§J i§.3& ^
-	wtpl_sim35
-	wtpl_sim55
25
/
2^ 3,5
25^ 35x 45
25" 55-"45-''55
2§" ?5-V"
2S"55-V" ^65
25" 35-",. - 55 65x"
25—45- 55/ —
25 £ 35 45 « bt> 65
£? 35 45 55 65
II 4165 65
25
35
45
55
75
85
65
agecount
WTP to Avoid a Statistical Lost Life-year
95
Figure 4D

-------
Valuation of Environmental Risks to
Children's Health*
Mark Dickie
and
Shelby Gerking
Department of Economics
University of Central Florida
Orlando, FL 32826
October 8, 2003
*The US Environmental Protection Agency partially funded the research described here under R-
82871701-0. The research has not been subjected to Agency review and therefore does not
necessarily reflect the views of the agency, and no official endorsement should be inferred.
Gerking acknowledges the hospitality of CentER, Tilburg University, where this research was
begun, as well as support from Visiting Grant B46-386 from the Netherlands Organization for
Scientific Research (NWO). For numerous constructive comments on earlier drafts, we thank
Anna Alberini, participants in the workshop on Valuation of Children's Health organized by the
National Policies Division, OECD Environment Directorate, and seminar participants at Tilburg
University and University of Durham.
62

-------
1. Introduction
Do parents value improvements in their children's health more than improvements in
their own health? This question bears directly on central issues in research on family behavior
including resource allocation between family members and the extent of parental altruism toward
their children. It also has important implications for public policy in light of the growing
worldwide emphasis on protecting children's health from environmental hazards (Scapecchi
2003). Nevertheless, little is known about how parents allocate health-related resources between
themselves and their children despite the fact that public policy measures for protecting children
operate at least partly through parents or other adult caregivers. Also, the few studies that do
examine how parents value their own health relative to their children's health focus more heavily
on morbidity (Liu et al. 2000 and Dickie and Messman 2003), base estimates on crude health
measures (Agee and Crocker 2001), and reach widely differing conclusions. For example,
Jenkins, Owens, and Wiggins (2001) find that the value of a statistical life (VSL) of a child
(about $3 million year 2000 dollars) is about two-thirds of that for a parent, while Mount et al.
(2001) find that the VSL for parent and child are about equal ($7.3 million in year 2000 dollars).
Additionally, Liu et al. (2000), Dickie and Messman (2003), and Agee and Crocker (2001) find
that parents are willing to pay about twice as much to reduce morbidity risk for their children
than for themselves.
This paper uses unique field data on skin cancer to estimate parents' marginal rates of
substitution between morbidity and mortality risks to themselves and to their children. Skin
cancer risk, previously considered in related context by Dickie and Gerking (1996, 1997), is a
common affliction that can but usually does not result in death. Also, solar radiation exposure
during childhood is an important determinant of lifetime skin cancer risk (e.g., Reynolds, et al.
63

-------
1996, Robinson, Rigel and Amonette 1997, American Academy of Dermatology 1997 and
Creech and Mayer 1998) and people accumulate as much as 80% of lifetime exposure before the
age of 18.
From a conceptual standpoint, a key advantage of this study is that morbidity and
mortality risks are treated together in a consistent theoretical framework. Prior studies of health
risks treat either morbidity or mortality, but not both, yet these two health outcomes are
obviously related (i.e., death is a possible outcome of illness). Also, the expected utility model
developed shows how to make econometric estimates of the desired marginal rates of
substitution as risk-risk tradeoffs from an indifference map. Whereas Viscusi, Magat and Huber
(1991) used risk-risk tradeoffs to see how people evaluate different sources of risk, this study
looks at how parents make interpersonal risk trade offs between themselves and their children, as
well as how they make trade offs between morbidity and mortality risks from the same disease.
An important methodological advantage of the study is that data are collected using an
experimental design that randomizes health risk changes presented to parents. This feature
sidesteps a number of econometric problems because risk changes are exogenous treatments that
are orthogonal to individual characteristics. Additionally, although marginal rates of substitution
are obtained from parents' stated preference bids, the desired estimates are ratios of bids. Thus,
the problem identified by Diamond and Hausman (1994) and Cummings et al. (1997) that stated
preference bids overestimate what people actually would pay may be at least partially
ameliorated. Additionally, use of stated preference bids may in any case be more appropriate
than revealed preference value estimates because tastes do not have to be disentangled from a
household production technology (Hanemann 2003).
64

-------
The paper is divided into four additional sections. Section 2 develops an expected utility
model with compound probabilities for a parent-child "family" in which either person might get
skin cancer and then might die from this disease. Section 3 describes field data on perceived risk
of skin cancer and willingness to pay to avoid the disease collected from 610 parents in
Hattiesburg, MS during the summer of 2002. Section 4 presents results indicating that for the
full sample, parents' estimated marginal rate of substitution between health risk reductions for
their children and health risk reductions for themselves is about 2. This estimate, however,
exhibits considerable variation across sub-samples of parents. It is larger for white parents than
for black parents, larger for sons than daughters, and larger for younger children than older
children. Section 5 concludes.
2. Model
This section presents a one-period expected utility model with state dependent utility
functions to guide the experimental design, data collection and empirical analysis. The model
consistently treats both morbidity and mortality risk from skin cancer in a "family" composed of
one parent and one child. This approach abstracts from several issues considered elsewhere to
make application tractable in the field study. For example, the model does not consider
divergent interests between family members (see Mount et al. 1991 and Smith and van Houtven
2002) because expenditures to reduce risks of skin cancer represent a small fraction of family
budgets. The child is assigned no role in household decision-making; in consequence, the parent
is assumed to allocate family resources to maximize his or her own expected utility. Only one
child is included in the model to focus on how parents make tradeoffs between their own health
and the health of their children, rather than on how parents allocate resources among different
children. A one-period model is presented so as to emphasize parent-child tradeoffs and
65

-------
consistent treatment of morbidity and mortality while abstracting from latency periods and time
preferences. Extensions of the model to introduce latency periods and two or more children are
briefly described at the end of this section.
The parent's expected utility is a probability-weighted sum of utilities in 3 =9 possible
states of the world that depend on whether the parent and child are healthy, sick, or dead. Four
probabilities determine which of the nine states of the world actually emerges: (1) the probability
that the parent will get skin cancer (S ), (2) the conditional probability that the parent will die
from skin cancer given that the disease is contracted {D ), (3) the probability that the child will
get skin cancer ( Sc), and (4) the conditional probability that the child will die from skin cancer
given that the disease is contracted (Dc). This approach has at least broad similarities to models
previously applied in the literature on environmental risks to health. In their model of health
consequences of exposure to hazardous wastes, Smith and Desvousges (1986, 1987) split the
unconditional risk of death from exposure into the probability of exposure and the conditional
probability of premature death given exposure. Eeckhoudt and Hammitt (2001) examine how a
specific risk to an individual's health should be valued when the individual faces independent
life-threatening background risks. Both of these models, however, envision only two health
states (alive and dead) and thus do not explicitly treat morbidity, and neither model considers the
allocation of health resources in a family.
In the model applied in this paper, the four probabilities are determined as shown in
equation (1).
S^S^Z^X,) D,=D,(Z,j = p,c,	(1)
66

-------
In this equation, probabilities of getting skin cancer and of dying from the disease if it is
contracted are influenced by predetermined factors (Q., / = p, c ) such as genetic characteristics
like complexion and sensitivity of skin to sunlight. Still, the probabilities are endogenously
determined because parents may purchase goods (e.g., hats, sun lotions, medical care) for
themselves and their children (Z., j = p,c) to reduce the chances of getting skin cancer and to
reduce conditional death risk if the disease is contracted. Because the experimental design
applied in the field study manipulates the four probabilities, S, and Dj also are specified as
functions of treatments X . and 8 ,.
As described in Section 3, the treatments are hypothetical sun lotions that resemble
currently marketed products but offer greater skin cancer protection. If purchased, the
hypothetical sun lotion would replace any currently used sunscreens, resulting in a savings in
expenditure on existing products but no attenuation of the risk reduction offered by the
hypothetical sun lotion. Any changes in other protective actions Z. (e.g., seeking less
evaluation of skin damage during medical checkups) are assumed to be negligible. (See Dickie
and Gerking 1996 for a model incorporating adjustments in protective behavior.) Also, for ease
of exposition, treatment parameters have the property cS . / ok. = cl), / c8 . = -1.
Perceived skin cancer risks are incorporated into the expected utility model as shown in
equation (2).
£(£/) = (l-^)(l-Sc)£/0(7)
+ (l-Sc)Sp[(l-Dp)Up(Y) + DpVp(Y)-\ + (l-Sp)Sc[(l-Dc)Uc(Y) + DcVc(Y)-\ (2)
+ SpSc[(l-Dp)(l-Dc)Upc(Y) + (l-Dc)DpWp(Y) + (l-Dp)DcWc(Y) + DpDcWpc(Y)l
where U0 denotes utility in the state where both parent and child are healthy, if , denotes utility
in a state in which either the parent or child (j = p,c) contracts skin cancer but the other does
67

-------
not and neither dies, Vj denotes utility in a state in which either the parent or child ( j = p,c )
dies from skin cancer but the other does not get it, U denotes utility in the state where both
parent and child get skin cancer but neither dies, W. denotes utility in the state in which both
parent and child contract skin cancer and one of the two dies (j = p, c ) but the other does not,
and W denotes utility in the state in which both parent and child die from skin cancer. In
states in which the parent and/or child die, parental utility is not restricted to zero; for example, if
the child dies, the parent's life may still go on and if the parent dies utility may be obtained from
a bequest. Also, Y denotes the parent's wealth net of: (1) expenditures for self- and child-
protection goods (Z.) and (2) bids for treatments presented in the experimental design (X. and
5j). The parent's gross wealth is denoted as y and for simplicity here is assumed to be the
same in all health states. (See Shogren and Crocker (1991) for a model incorporating differences
in wealth between health states.)
The model can be manipulated to obtain parents' willingness to pay for reduced
morbidity and mortality risks to themselves and their children. Ratios of marginal willingness to
pay values provide measures of parents' marginal rates of substitution between: (1) morbidity
risk to themselves and to their children, (2) mortality risk to themselves and to their children, (3)
morbidity and mortality risk themselves, and (4) morbidity and mortality risk to their children.
Assume that the parent already has chosen expected utility maximizing values of self- and child-
protection expenditures in each health state, and A . and 8 , are initially zero. Then, willingness
to pay for reduced risk of skin cancer to the child is obtained by setting dE(U) = 0
= dkp =d8p =ddc and computing
68

-------
-dytax, ={(1-S„)[(C„-UJ + DC(UC -FJ]
+ 5,(1 -Dr)[(Ur-U^) + Dc(U^-Wc)] + SrDrWr-Wr) + De(W,-W^]}l A.
In equation (3), A denotes the expected marginal utility of wealth and is positive if the marginal
utility of wealth is positive in each state. Also, the numerator of the right hand side of equation
(3) is positive if the utility difference in each term of the sum is positive (i.e., healthy is preferred
to sick, sick is preferred to dead, one person sick is preferred to two people sick, etc.). Then,
dy I dXc < 0 and gross wealth must fall to hold expected utility constant if the child's morbidity
risk is reduced.
Similarly, willingness to pay for a small reduction in perceived conditional death risk
faced by the child, holding all other perceived health risks constant, is
-dylBS, = S,{{l-St%U, +	-WJ + Dp(WpA. (4)
ThusSy /dSc < 0 if c)y/ dXc <0. Because perceived unconditional risk of death from skin cancer
is Rc = SJ)c, equations (3) and (4) can be combined to obtain the parent's willingness to pay to
reduce the child's unconditional death risk:
r 1 -S. ^

f 1 -D ^
-dy/dRc = f {~dyldXc)+	{\!Sc)(Sy!d8c).	(5)

Thus<9y /dRc < 0 if cy / cXc <0. The parent's marginal rate of substitution, or risk-risk tradeoff,
between the child's unconditional risk getting skin cancer and unconditional risk of dying from
the disease equals (dy/dRc)/(dy/dSc). This ratio measures the parent's relative valuation of
reducing mortality and morbidity risks for the child. However, if skin cancer is an event that
may occur in the future, the absolute magnitudes of dy/c/^and dy/dSc cannot be used to
estimate the value of a statistical life or of a statistical case of skin cancer (i.e., the willingness to
pay today to save a life or to avoid a case today).
69

-------
Key comparative static properties of willingness to pay expressions in equations (3) - (5)
are similar those found in the more familiar setting of one individual facing mortality risk only
(Jones-Lee 1974). For example, parental willingness to pay to reduce the child's morbidity or
mortality risk increases with gross wealth and with the initial levels of risk faced by the child, if
the expected marginal utility of wealth is decreasing in wealth and in initial risk levels. Also, for
marginal reductions in small risks of morbidity or mortality, willingness to pay is approximately
proportional to the size of the risk change.
Similar properties apply to parents' willingness to pay for reduced risks to themselves.
These values, which can be obtained by parallel calculations corresponding to equations (3) -
(5), are useful in their own right and as benchmarks for assessing the magnitudes of parents'
valuations of their children's risks. It will be of interest to test whether parents' marginal rates
of substitution between unconditional risks to their children and unconditional risks to
themselves equal unity, i.e., whether (dy/cXc)j(vy IcXp) = 1 and whether
(dyldRc)l(dyldRp) = 1.
The model may be extended to a temporal setting incorporating a latency period before
the possible onset of skin cancer and including an arbitrary number of children in the family.
The specific extension envisioned features identical children who face a longer latency period
than do their parents. In this broader model, willingness to pay for reduced risk for the parent or
a child falls as the number of children rises, if the marginal utility of aggregate family
consumption is higher when more children are present. Willingness-to-pay values and parent-
child marginal rates of substitution depend on weighted sums of utility differences similar to
those appearing in the one-period setting, as well as on discount factors determined by latency
periods and parents' subjective discount rates. Like the individual utility differences appearing
70

-------
in equations (3)-(5), the discount factors are components of parents' valuations that need not be
separately identified to estimate willingness to pay or marginal rates of substitution. While
measures of parents' discount factors for latent health risks would be of interest, these measures
might be better estimated in a study that focused on latency of one risk to one person, rather than
on morbidity and mortality risk for two people.
3. Data Collection
Data on risk beliefs about skin cancer and willingness to pay to avoid this disease were
collected during summer of 2002 using a self-paced, interactive, computerized instrument. All
respondents were residents of the Hattiesburg, MS metropolitan statistical area. Hattiesburg is
located in the southern part of Mississippi, has a mean annual high temperature reading of 77.5
degrees Fahrenheit, a subtropical climate, and a large number of sunshine days each year. Thus,
residents have experience with consequences of exposure to ultraviolet radiation from sunlight.
The sample was drawn by random digit dialing after removing business, government, and
cellular telephone numbers. When the calls reached adults, interviewers described the general
purpose of the survey (federally funded research on health risks to parents and their children),
asked whether they had at least one biological child between the ages of 3-12 living at home, and
asked whether they were willing come to the University of Southern Mississippi to participate in
the survey. Biological children were singled out for inclusion in the study because skin cancer
risk is partly determined by genetic characteristics inherited from parents (e.g., fairness of skin
and sensitivity of skin to sunlight). The age range was chosen to have children old enough to
regularly spend time outdoors, but young enough for parents to exert substantial control over
their activities. Respondents were paid $25 for completing the 30-minute questionnaire.
71

-------
The final sample consisted of 610 parents; children did not participate in the survey. The
survey obtained information about the parent/respondent and one sample child (chosen at
random from among biological children living at home if more than one in the 3-12 year age
range was present. Information was obtained about the number of children in the household, but
other questions about children pertained only to the sample child in order to limit the length of
the interview, to avoid repetitive questioning, and because the model presented in Section 3
assumes that parents treat each child equally. Of sample parents, 75.4% were white, 20.0% were
African-American, 4.6% were members of other races, 23.4% were male, 16.9% were under the
age of 40, mean household income was $53,000 per year, 75.9% were married, and 59.0%
worked full time. Because of random selection, about half (50.5%) of the sample children were
male. The average age of sample children was 7.07 years. Also, parents were generally familiar
with skin cancer: (1) 95.4% had heard of skin cancer, (2) 83.8% knew of someone (public
figures, friends, or relatives) who had been diagnosed with this disease, (3) 22.1% knew of
someone who had died from skin cancer, (4) 80.3% had thought about the possibility of getting
skin cancer, (5) 3.4% had been diagnosed with this disease themselves, and (6) 71.1% had
considered the possibility that one of their children might get skin cancer.
Chances of getting skin cancer were assessed using an interactive risk scale that closely
resembled the grid squares used by Krupnick et al. (2002). This approach was used because risk
information appears to be better understood using this type of visual aid (Corso, Hammitt, and
Graham 2001). As shown in Figure 1, the scale depicted a large square divided into 20 rows and
20 columns showing 400 equal-sized smaller squares. Initially, all 400 of these squares were
green. Parents changed green squares to red ones to represent the amounts of risk. By pressing a
button at the bottom of each column of squares, they could recolor a column of 20 squares from
72

-------
green to red (or from red back to green) and the color of any individual square could be changed
by clicking on it with a mouse. A box beneath the scale showed the percentage of red squares
out of 400. This calculation was updated each time one or more squares was re-colored. Before
using the scale to estimate skin cancer risk, parents practiced using the risk scale for an unrelated
event (a possible auto accident) and were told about the meaning of "chances in 400". Also,
they were told to consider only the chances of getting this disease (or of getting it again if they
had already had it), rather than how serious the case might be. Parents then used the risk scale
to estimate lifetime chances of getting skin cancer, first for themselves and then for their sample
children. In making these estimates, they could take as much time as they desired and could
make as many changes in the risk scale as desired. Table 1, discussed momentarily, presents the
frequency distribution of these risk estimates.
After providing lifetime skin cancer risk estimates for themselves and their children,
parents were: (1) provided with information about skin cancer, (2) asked a series of questions
about skin cancer risk factors, and (3) given an opportunity to revise these estimates. The idea
behind asking respondents to estimate lifetime skin cancer risk a second time was to help them
pin down their estimate as well as they could before moving on to the remaining portions of the
survey. In particular, they were told that according to the National Cancer Institute, the average
person in the United States has a lifetime risk of getting skin cancer of 18% and were questioned
about skin color and sensitivity to sunlight, family history of skin cancer, time spent outdoors in
direct sunlight, past sunburns, and use of sun protection products and protective clothing. Brief
narratives provided information about how these aspects have been related to skin cancer risks in
epidemiological studies. To elicit the revised lifetime skin cancer risk perception estimates,
73

-------
parents again were shown the previously described risk scales for themselves and their sample
child as they originally were marked, and were given an opportunity to make changes.
After this task was completed, parents were asked about their perceived severity of skin
cancer: "Suppose that a doctor tells you that you have skin cancer and you begin treatment.
What do you think is the chance that you would die within five years of this diagnosis?" Parents
answered for themselves and their sample child using a risk scale like the one shown in Figure 1.
Responses are interpreted as estimates of the conditional risk of death from skin cancer given
that the disease is contracted.
Table 1 presents frequency distributions of parents' perceived lifetime risk of skin cancer
and conditional risk of death from skin cancer both for themselves and for their children. For
perceived lifetime risk, the frequency distribution shown pertains to the initial risk estimates. As
it turned out, parents made only small revisions in their initial lifetime risk estimates for
themselves (the two estimates of mean risk are virtually the same, 23.9%), but revised risk
estimates for children were on average about 1.5 percentage points lower than initial risk
estimates (19.0 vs. 20.5), a significant difference at the 1% level. Table 1 indicates considerable
variation in perceptions about lifetime skin cancer risk, with some parents believing that skin
cancer is highly unlikely and a smaller number of other parents believing that skin cancer is
virtually inevitable. Regarding the possibility of death from skin cancer, about two-thirds of
parents believed that their conditional risk of death given a diagnosis of skin cancer is 10% or
less and about three-fourths of parents believed that if similarly diagnosed, their sample child's
conditional risk of death is 10% or less. This outcome suggests that parents were aware that skin
cancer is seldom fatal.
74

-------
Table 2 shows estimates of mean lifetime of getting skin cancer and mean conditional
risk of dying from this disease for various sub-samples of parents. These sub-samples are further
analyzed in Section 4. As shown, white parents estimated that their own lifetime risk of getting
skin cancer exceeded that of their sample child (27.6% vs. 22.8%, a statistically significant
difference at the 1% level), whereas among blacks, the corresponding difference was not
significant at conventional levels (11.8% vs. 12.9%). Parents in both racial groups appear to
have overestimated this risk. Ries et al. (1999) found that whites have a lifetime chance of 21%
of getting either melanoma or non-melanoma skin cancer and African-Americans have a
corresponding risk of less than a 1%. The fact that the survey introduced the possibility of
getting skin cancer again if the parent had already had it does not appear to be an important
complicating factor in this regard. Sample members are relatively young and few reported
having been previously diagnosed with this disease.
Table 2 also shows that parents reported higher mean conditional death risk estimates for
themselves (12.2%) than for their sample children (9.4%), a statistically significant difference at
1%. Differences in these estimates between white and black parents are quite small. Thus, it
appears that parents generally believe that skin cancer risks for their children are lower than their
own. This outcome may reflect parents' beliefs that they take greater precautions to protect their
children from skin cancer risk with their own children than their parents did in an earlier period
when less was known about the hazards of solar radiation exposure. Also, it may reflect a belief
that skin cancer will take longer to develop in children than in parents together with the idea that
delayed risks are perceived as smaller. Finally, Table 2 indicates that among whites, who
comprise 67% of the sample: (1) mothers believed that their own risks of skin cancer exceeded
75

-------
those for fathers, (2) parents thought that their sons' and daughters' risk was about the same, (3)
parents believed that risks faced by younger children exceeded those for older children.
The final section of the survey assessed willingness to pay for a hypothetical sun
protection product that would reduce skin cancer risk for both the parent and the child when used
as directed. The approach of using a single product to get willingness to pay means that parents
do not make separate bids to protect themselves and their children as in Liu et al. (2000) and
Dickie and Messman (2003). This procedure is aimed at reducing the potential problem that
parents might feel that they "should" bid more for child protection than for protection for
themselves. Parents became familiar with this product by reading a label that was designed to
look like those used on bottles of over-the-counter sun lotions (see Figure 2). The label indicated
that the hypothetical sunscreen would be similar in most respects to currently marketed products
(available in a variety of SPFs, offer protection against premature aging of skin, non-
comedogenic, oil-free, and unscented), but that it would offer greater levels of skin cancer
protection.
Eight labels were used in the study. Except for differences in the amount of skin cancer
reduction offered, labels were identical in every respect to control for other possible motivations
driving the purchase decision such as to prevent or get a suntan and guard against aging or
wrinkling of skin (see Dickie and Gerking 1996 who more fully discuss these possibilities). Four
labels varied reductions in risk of getting skin cancer, while four other labels varied reductions in
conditional death risk of this disease. Table 3 shows the reductions in risk stated on each of
label. Labels A, D, E, and H offered equal percentage reductions in skin cancer risk (either 10%
or 50%) for both adults and children. Labels B and F offered relatively greater skin cancer
protection for children, while Labels C and G offered protection for adults. Each respondent was
76

-------
shown two randomly assigned labels. One of these offered reduced risk of getting skin cancer
and the other offered reduced conditional death risk from skin cancer. The order in which these
labels were presented was randomized.
After respondents were given time to read the label as if buying a product for the first
time, the risk scale was used to show the amount by which the hypothetical sunscreen would
reduce skin cancer risks for themselves and their children. Then, parents were asked, "Now
please think about whether you would buy the new sun protection lotion for yourself or your
child. Please do not consider buying it for anyone else. Suppose that buying enough of the
lotion to last you and your child for one year would cost $X. Of course, if you did buy it, you
would have less money for all of the other things that your family needs. Would you be willing
to pay $X for enough of the sunscreen to last you and your child for one year?" The value of X
was varied between $20 and $125. When responses were affirmative, parents were asked if they
would pay a higher price; when responses were negative, they were asked whether a lower price
would be paid. This procedure was repeated for the second label assigned to the parent.
4. Empirical Estimates
Data described in Section 3 are used to obtain estimates of the marginal rates of
substitution described in Section 2. Marginal rates of substitution are inferred from estimates of
an equation describing parents' willingness to pay for the hypothetical sunscreen. This equation
was obtained from the model presented in Section 2 by totally differentiating equation (2),
setting dE(U) = 0, and interpreting the bid for the sunscreen as the change in wealth, dy. The
equation estimated is
\n(wu) = d'fi+x'j + iL +vit,	(6)
77

-------
where i indexes parents and t = 1,2 indexes the two experimental treatments (labels) assigned to
parent i. In equation (6), wjt denotes willingness to pay for one year's supply of the sun lotion,
dit denotes a vector of attributes of the sun lotion including the risk changes for the parent and
child as described on the label, and /3 represents the corresponding vector of coefficients. The
P coefficients measure effects of risk changes on (the log of) willingness-to-pay and must be
estimated to infer marginal rates of substitution. Also, xt represents a vector of measured
characteristics of the parent, child or family, /represents the corresponding vector of
coefficients, and ui and vit are uncorrelated mean zero normal random variables with variances
au and <7V, respectively. Thus, vit reflects uncontrolled factors varying over parents and over
treatments, while ui captures the impact of uncontrolled factors specific to the parent (or her
child or family) and constant over treatments. Among the many factors that might be reflected in
the individual-specific error component are unobserved genetic endowments, current spending
on sunscreen lotion, concern for skin cancer risks to herself and her child, and propensity to
misstate willingness to pay in response to hypothetical questions. Willingness-to-pay is assumed
log-normally distributed in view of its non-negativity and the positive skewness typically
characterizing its distribution.
Random assignment of labels to parents implies that risk changes are exogenous
experimental treatments that are independent of all measured and unmeasured individual and
family characteristics. As a consequence, randomization avoids two potential problems that
would otherwise complicate estimation of willingness to pay for reduced risks and marginal rates
of substitution. First, variables measuring risk change are orthogonal to characteristics such as
initial perceived risks, income, number of children in the household, and race and gender of
78

-------
parent and child, so that the dit is orthogonal to x . Thus, the specification of the variables in r
has no effect on the estimate of p.
Second, random assignment implies that dit is uncorrelated with //,, so that /3 may be
estimated consistently in a random-effects framework. Without random assignment (e.g., with
non-experimental data), the risk changes to be valued are likely to be correlated with unobserved
individual characteristics. Previous research indicates that inferences about intra-family
allocations may be seriously misleading when heterogeneity of this sort is uncontrolled (Pitt and
Rosenzweig 1990, Pitt, Rosenzweig, and Hassan 1990). Fixed-effects methods would remove
family-specific heterogeneity but are less efficient that random-effects when heterogeneity is
absent, as it is under randomization of experimental treatments. Instrumental-variable methods
represent an alternative approach to the heterogeneity problem that are frequently used when
repeated observations on individuals are not available. But randomization allows consistent and
efficient estimation of /3 without resorting to use of instrumental variables.
Estimates of equation (6) are obtained by maximum likelihood. Respondents did not
directly report their bids for the sunscreen, but the interval in which willingness-to-pay lies may
be inferred from responses to the initial and follow-up questions asked about each sun lotion
(Hanemann, Loomis and Kanninen 1991). Let w"t and w\t respectively denote the natural
logarithms of the upper and lower bounds of willingness-to-pay for parent label t. Thus w"t
equals the log of the lowest price at which the respondent declined to purchase the sunscreen (or
+oo if she responded "yes" to both initial and follow-up questions), while wlit equals the log of
the highest price at which the respondent agreed to purchase the sunscreen (or -oo if she
responded "no" to both initial and follow-up questions). Then the probability that the natural
79

-------
logarithm of willingness-to-pay lies between the upper and lower bounds, conditional on
un equals
where O denotes the standard normal cumulative distribution function. The sample log-
likelihood function is
where N equals the number of parents in the sample and / denotes the normal density function.
The automated routine included in the econometric package LIMDEP and used to maximize the
log-likelihood function computes the integral in equation (8) using Monte Carlo simulation.
Estimates of equation (6) are presented in Table 4. Covariate definitions are in column 1,
their sample means are presented in column 2, and results from two regressions using the full
sample of 610 parents are in columns 3 and 4. Five covariates are dummy variables that reflect
the reductions in skin cancer risk shown on the eight labels (see Table 3). GET shows whether
the label presented a reduction in the chance of getting skin cancer or a reduction in the
conditional risk of dying from it. Thus, GET=1 for Labels A-D and GET=0 for Labels E-H.
Also, PARENTCHG=1 if the label offered parents a 50% reduction in risk for themselves and
KIDCHG=1 if the label offered a 50% risk reduction for their children. Interactions of GET and
(1-GET) with PARENTCHG and KIDCHG show whether the risk reduction pertained to getting
skin cancer or the conditional risk of dying from it. Label E, offering a 10% reduction in the
conditional risk of dying from skin cancer for both parents and children, is represented by setting
all five dummies equal to zero.
(7)
N +c0 2
i> jn Luf(u)du >
(8)
i—1	-co 1
80

-------
The column 3 regression uses only the five label dummies as covariates and column 4
shows the outcome when covariates measuring household income and number of children in the
family are added. In both of these regressions, likelihood ratio tests at the 1% level reject the
null hypotheses that: (1) the variance of the parent-specific error is zero and (2) all slope
parameters are jointly zero. Asymptotic t-statistics, presented in Table 4, show that each
coefficient estimated differs significantly from zero at the 5% level or lower under a two-tail test.
As expected, coefficients of the label dummies change little when controls for family
characteristics are added.
In columns 3 and 4, the positive coefficients of GET*PARENTCHG, GET*KIDCHG, (1-
GET) *PARENTCHG, and (1-GET) *KIDCHG indicate that parents are willing to pay more for
larger risk reductions than for smaller risk reductions. Although this outcome is broadly
consistent with the conceptual model presented in Section 2, larger risk reductions bring about
less than proportional increases in willingness to pay (see Hammitt and Graham 1999 for further
discussion of this issue). For example, as shown by the coefficient of GET*KIDCHG, a five-fold
reduction in risk to children of getting skin cancer (from 10% to 50%) increases willingness to
pay by a little more than 40%. Also, likelihood ratio tests at the 1% level reject the null
hypothesis that coefficients of GET*PARENTCHG and GET*KIDCHG are equal as well as the
null hypothesis that coefficients of (1-GET)*PARENTCHG, and (1-GET)*KIDCHG are equal.
In fact, the numerically larger coefficients of the risk change treatments for children suggest that
parents are willing to pay more for skin cancer risk reduction for their children than they are for
risk reduction for themselves. This point is developed more fully below in the context of
estimating parents' marginal rates of substitution between skin cancer risk to their children and
skin cancer risk to themselves.
81

-------
In column 4, the positive coefficient of household income indicates that, all else constant,
an increase in income by $10,000 increases willingness to pay for the hypothetical sunscreen by
3%. At sample mean household income of $53,000, the estimated income elasticity of
willingness to pay for the hypothetical sunscreen is about 0.16. Also, the negative coefficient of
the number of children in the household suggests that an additional child (of any age) in the
household reduces willingness to pay for the hypothetical sunscreen by about 8%. This outcome
is consistent with the discussion in Section 2 that fewer resources are invested in risk reduction
per child when more children are present.
Estimates of marginal rates of substitution are computed as ratios of marginal willingness
to pay from the column 4 regression. For example, the marginal rate of substitution between
unconditional morbidity risk for the parent and child is estimated as the ratio of the coefficient of
('GET*KIDCHG) to the coefficient of (GET*PARENTCHG), multiplied by the ratio of the
sample mean change in the level of unconditional morbidity risk for parents to the sample mean
change in the level of unconditional morbidity risk for children. A parallel procedure is used to
estimate the marginal rate of substitution between conditional death risks for the parent and
child. The marginal rate of substitution between unconditional death risks for the parent and
child then is estimated by combining the marginal valuations of morbidity and conditional
mortality risk using equation (5), and taking the ratio of the resulting child valuation to the parent
valuation.
The outcomes of these calculations, based on the column 3 in Table 4, are shown in
column 2 of Table 5. These results indicate parents are willing to pay about twice as much to
reduce the risk of getting skin cancer for their children as they are to reduce it for themselves.
Similarly, the child vs. parent unconditional mortality marginal rate of substitution estimate is
82

-------
2.33 (again see column 2 of Table 5). Standard errors of these estimates, reported in Table 5,
indicate rejection of the null hypotheses that these marginal rates of substitution are equal to
unity. That parents are willing to pay more to reduce risks to their children's health than they are
willing to pay to reduce risks to their own health is of particular interest because age at onset of
skin cancer is in the more distant future for children than for parents. Based on the discussion of
latency in Section 2, if the time to onset of illness were the same both for parents and children,
the marginal rate of substitution values may well be larger.
Column 2 of Table 5 also reports calculations of parents' marginal rates of substitution
between the unconditional risk of dying from skin cancer and the unconditional risk of getting
skin cancer for themselves and for their children. Whereas the marginal rates of substitution
discussed above reflect tradeoffs between the same risk faced by different people, these
calculations reflect tradeoffs between different types of risk faced by the same person. As shown
in Table 5, parents' marginal rate of substitution between unconditional death risk and
unconditional morbidity risk for themselves is 19.16 and the corresponding value for their
children is 21.78. These estimates indicate that parents are willing to pay approximately 20
times more to reduce unconditional death risk by one unit than to reduce unconditional morbidity
by one unit. Although, this outcome supports the idea that public policies aimed at reducing
death risk are much more important to people than policies aimed at reducing morbidity, it may
not generalize to related situations. Skin cancer is frequently not life threatening and while
treatment may be disfiguring, patients generally expect to resume normal activities. Other
illnesses and injuries may exact a greater toll on health if death does not occur and in these cases
the marginal rate of substitution between mortality and morbidity may well be lower.
83

-------
In addition to obtaining point estimates of marginal rates of substitution for a
representative parent, it is of interest to examine how health risk tradeoffs may vary with the
characteristics of parents or children. To obtain this information, the Table 4, column 4
regression was re-estimated for sub-samples defined by (exogenous) genetic characteristics that
may be associated with differences in perceived risks and other initial endowments. A useful
starting point in this regard is to compare marginal rates of substitution for whites and blacks.
As discussed in Section 3, average perceived risks of skin cancer by white parents are roughly
twice as large as those for black parents. Estimates shown in Table 5 indicate that the four
marginal rates of substitution for whites are roughly similar to those obtained for the full sample
(notice that the 460 white parents represent 67% of 610 parents in the full sample). These
estimates, however, differ substantially from those for blacks; in fact, a likelihood ratio test at
1% rejects the null hypothesis that marginal rates of substitution for the two groups are equal.
For blacks, two of the marginal rates of substitution could not be computed because the
coefficient of GET*PARENTCHG was negative and did not differ significantly from zero at
conventional levels. Also, the marginal rate of substitution for child vs. parent unconditional
mortality is significantly less than unity at the 1% level, suggesting that black parents may be
less altruistic toward their children than are white parents. This interpretation, however, should
be treated quite cautiously because of the relatively small number of black parents in the sample.
The significant racial differences in valuation estimates suggest that pooling sub-samples
of black and white parents to estimate marginal rates of substitution is inappropriate. Thus, in
light of the relatively small sample size for blacks, outcomes from additional demographic
breakdowns shown in Table 5 are computed only for parents in the white sub-sample. The first
of these compares marginal rate of substitution estimates for 351 white mothers and 109 white
84

-------
fathers. Whereas both mothers and fathers similarly evaluate the child vs. parent unconditional
mortality tradeoff, the child vs. parent unconditional morbidity tradeoff for fathers is about unity
and about half the magnitude of that found for mothers. Thus, in comparison to mothers, fathers
appear to be relatively less concerned with morbidity than mortality. This outcome leads fathers'
marginal rate of substitution between mortality and morbidity for their child to be larger than that
for mothers (50.05 vs. 19.06).
Also, parents appear to place significantly greater weight on reducing both morbidity and
mortality risk for sons than for daughters. Estimates of child vs. parent marginal rate of
substitution for unconditional morbidity is 2.60 for sons and 1.14 (not significantly different
from unity) for daughters. Corresponding estimates of the marginal rate of substitution for
unconditional mortality are 5.40 for sons and 2.01 for daughters. The null hypothesis that
marginal rates of substitution for sons and daughters are equal is rejected at 1% level. Thus,
relative to their own health, parents appear to be willing to invest more in health risk protection
for sons than daughters.
Finally, parents are more protective of younger children than older children. The child
vs. parent marginal rate of substitution estimates for unconditional morbidity are 1.42 for
children aged 3-7 years and 2.22 for children aged 8-12 years; however, these estimates do not
differ significantly from zero at the 1% level. On the other hand, corresponding estimates of the
marginal rate of substitution for unconditional mortality are significantly larger for young
children than for older children (4.38 for children aged 3-7 years vs. 1.73 for children aged 8-12
years). This finding is consistent with recent evidence that health risk protection for young
children is valued more highly than that for older children. Nastis and Crocker (2003), find that
85

-------
mothers-to-be value the expected postnatal health of their unborn child as much as six times
more than the expected post-partum state of their own health.
5. Conclusions
This paper has presented new empirical estimates aimed at valuing environmental risks
affecting parents and children. The application focused on skin cancer, the most common form
of cancer in the U.S. Links between environmental exposure to ultraviolet radiation and skin
cancer are well established, and chances of getting skin cancer, for a given amount of exposure
to solar radiation, depend partly on observable genetic characteristics such as skin type and
complexion. The theoretical model is developed from the viewpoint of parents and supports
empirical valuation of morbidity and mortality risks faced by both parents and children in a
consistent framework. Risk is treated as endogenous and is measured as the risk perceived by
survey respondents. The method for estimating willingness to pay rests on directly estimating an
indifference relation showing utility-constant trade-offs between morbidity risks, mortality risks,
and consumption goods.
The model provides a basis for computing parents' marginal rates of substitution between
risk of death from skin cancer faced by both themselves and their children. This calculation
shows how parents value children's health relative to their own and may be useful benefits
transfer in situations where willingness to pay for reduced risk to adults have been established
but corresponding values for children are not available. The model is estimated using data
collected by an interactive computerized questionnaire administered on the University of
Southern Mississippi campus during summer of 2002. Key aspects of the experimental design
were to: (1) determine parents' perceptions of skin cancer risk to themselves and their children,
and (2) obtain willingness to pay for skin cancer risk reductions. Risk reductions were presented
86

-------
to parents using randomly assigned labels of a hypothetical sun lotion that offered different
amounts of protection to adults and children. Random assignments of risk reductions facilitate
estimation of marginal rates of substitution between parent's health and children's health. For
example, parents' marginal rate of substitution between their children's unconditional lifetime
risk of dying from skin cancer and the corresponding risk for themselves is 2.33. Thus, parents
view their children's health as more than twice as valuable than their own. Also, parents see the
reductions in mortality risk to be about 20 times more valuable than reductions in morbidity risk
both for themselves and their children. This outcome suggests that the morbidity component of
benefits for environmental risk reduction may be quite small.
87

-------
References
Agee, Mark D. and Thomas D. Crocker. 2001. "Smoking Parents' Valuations Of Own And
Children's Health." Presented at Association of Environmental and Resource Economists
conference, Assessing and Managing Environmental and Public Health Risks, Bar Harbor,
Maine, June 13-15, 2001. http://www.aere.org/.
American Academy of Dermatology. 1997. Melanoma Risk Factors Fact Sheet.
American Cancer Society. 1998. Non-Melanoma Skin Cancer Resources. Bethesda, MD.
Corso, Phaedra.S., James K Hammit and John D. Graham. 2001. "Valuing Mortality-Risk
Reduction: Using Visual Aids To Improve The Validity Of Contingent Valuation," Journal
of Risk and Uncertainty 23, 165-84.
Creech, L.L. and J. A. Mayer. 1998. "Ultraviolet Radiation Exposure in Children: A Review of
Measurement Strategies " Annals of Behavioral Medicine, 19, 399-407.
Cummings, R.D., S. Elliott, G. Harrison and J. Murphy, 1997. Are hypothetical referenda
incentive compatible? Journal of Political Economy 105: 609-621.
Diamond, Peter A. and Jerry A. Hausman. 1994. "Contingent Valuation: Is Some Number
Better than No Number?" Journal of Economic Perspectives 8: 45-64.
Dickie, M. and S. Gerking. 1996. "Formation of risk beliefs, joint production and willingness to
pay to avoid skin cancer." The Review of Economics and Statistics, 78: 451-464.
—, 1997. "Genetic Risk Factors and Offsetting Behavior: The Case of Skin Cancer," Journal of
Risk and Uncertainty, 15:81 -97.
Dickie, Mark and Victoria L. Messman. 2003. "Valuing Acute Illness In The Household: Are
Kids Worth More Than Parents?" Presented at the Association of Environmental and
Resource Economists workshop, Assessing and Managing Environmental and Public Health
88

-------
Risks, Bar Harbor, Maine, June 13-15. http://www.aere.org/.
Eeckhoudt, Louis R. and James. K. Hammitt. 2001. "Background Risks and The Value of A
Statistical Life," Journal of Risk and Uncertainty 23, 261-279.
Hammitt, James K. and John D. Graham. 1999. "Willingness to Pay for Health Protection:
Inadequate Sensitivity to Probability?" Journal of Risk and Uncertainty, 18, 33-62.
Hanemann, W. Michael. 2003. "Revealed and Stated Preference Approaches to Valuing
Children's Environmental Health," presented at Organization of Economic Cooperation and
Development "Workshop on the Valuation of Children's Health," Paris, Sep. 11-12 2003.
Hanemann, W. Michael, John Loomis and Barbara Kanninen. 1991. "Statistical Efficiency of
Double-Bounded Dichotomous Choice Contingent Valuation ," American Journal of
Agricultural Economics 73, 1255-63.
Jenkins, Robin R., Nicole Owens, and Lanelle B. Wiggins. 2001. "Valuing Reduced Risks To
Children: The Case Of Bicycle Safety Helmets," Contemporary Economic Policy, 19, 397-
408.
Jones-Lee, Michael. 1974 "The Value of Changes in the Probability of Death or Injury," Journal
of Political Economy 82, 835-49.
Krupnick, Alan, Anna Alberini, Maureen Cropper, Nathalie Simon, Bernie O'Brien, Ron
Goeree, and Martin Heintzelman. 2002. Age, Health, and the Willingness to Pay for
Mortality Risk Reductions: A Contingent Valuation Study of Ontario Residents. Journal of
Risk and Uncertainty 24: 161-86.
Liu, Jin-Tan, James K. Hammitt, Jung-Der Wang and Jin-Long Liu. 2000. "Mother's
Willingness to Pay for Own and Her Child's Health: A Contingent Valuation Study in
Taiwan," Health Economics 9, 319-26.
89

-------
Mount, Timothy, Weifun Weng, William Schulze, and Laurie Chestnut. "Automobile Safety and
the Value of Statistical Life in the Family: Valuing Reduced Risk for Children, Adults and
the Elderly," presented at the Association of Environmental and Resource Economists
Workshop on "Assessing and Managing Environmental and Public Health Risks," Bar
Harbor, Maine, June 13-15, 2001. htt p ://www.aere.0rq/.
Nastis, Stefanos and Thomas D. Crocker. 2003. "Mothers Valuation of Own and Unborn Child
Health," working paper, Dept. of Economics and Finance, University of Wyoming.
Pitt, MarkM., MarkR. Rosenzweig, and Md. N., Hassan. 1990. "Productivity, Health, and
Inequality in the Intrahousehold Distribution of Food in Low-Income Countries," American
Economic Review, 80, 1139-58.
Pitt, Mark M. and Mark R. Rosenzweig. 1990. "Estimating the Intrahousehold Incidence of
Illness: Child Health and Gender-Inequality in the Allocation of Time," International
Economic Review 31, 969-89.
Reynolds, K.D., J.M. Blaum, P.M. Jester, H. Weiss, S.J. Soong, R.J. Diclemente, "Predictors of
Sun Exposure in Adolescents in a Southeastern U.S. Population," Journal of Adolescent
Health, 19 (1996), 409-15.
Ries, L A G., M P. Eisner, C.L. Kosary, B.F. Hankey, B.A. Miller, L. Clegg, A. Mariotto, M P.
Fay, E.J. Feuer, B.K. Edwards (eds). SEER Cancer Statistics Review, 1975-2000, National
Cancer Institute. Bethesda, MD, http://seer.cancer.gov/csr/1975_2000 (2003).
Robinson, J.K., D.S. Rigel, and R.A. Amonette. 1997. "Trends in Sun Exposure, Attitudes, and
Behaviors: 1986 to 1996Journal of the American Academy of Dermatology, 37, 179-86.
Scapecchi, Pascale. 2003. "Valuation Differences between Adults and Children," presented at
Organization of Economic Cooperation and Development "Workshop on the Valuation of
90

-------
Children's Health," Paris, Sep. 11-12 2003.
Shogren, Jason F. and Thomas D. Crocker. 1991. "Risk, Self-Protection, and Ex Ante
Economic Value," Journal of Environmental Economics and Management 20:1-15.
Smith, V. Kerry and William H. Desvousges. 1986. "Asymmetries in the Valuation of Risk and
the Siting of Hazardous Waste Disposal Facilities," American Economic Review 76, 291-294.
Smith, V. Kerry and William. H. Desvousges. 1987. An Empirical Analysis of the Economic
Value of Risk Changes," Journal of Political Economy 95, 89-114.
Smith, V. Kerry and George Van Houtven, "Recovering Hicksian Consumer Surplus within a
Collective Model: Hausman's Model for the Household," working paper, North Carolina
State University (2002).
Viscusi, W. Kip, Wesley Magat, and Joel Huber, "Pricing Environmental Health Risks: Survey
Assessments of Risk-Risk and Risk-Dollar Trade-offs for Chronic Bronchitis," Journal of
Environmental Economics and Management 21, 32-51 (1991).
91

-------
Table 1. Frequency Distribution of Parents' Perceived Risks.
N=610.

Risk of Getting
Skin Cancer3
Conditional Risk of
Dying from Skin Cancer
Risk Range (%)
Parents
Children
Parents
Children
0-4.75
85
75
103
142
5-9.75
57
79
163
194
10 - 14.75
70
94
122
111
15 - 19.75
65
69
67
44
20 - 24.75
65
74
42
31
25 -29.75
66
73
26
23
30 -34.75
45
35
13
8
35 - 39.75
23
19
8
8
40 - 44.75
36
25
7
7
45 -49.75
6
5
5
2
50 - 54.75
53
32
19
11
55 - 59.75
4
2
2
1
60 - 64.75
5
7
3
0
65 - 69.75
0
1
0
0
70 - 74.75
5
2
2
0
75 - 79.75
6
5
0
0
80 - 84.75
2
3
0
0
85 - 89.75
3
2
0
0
90 - 94.75
8
5
0
0
95-100
6
3
0
0
aInitial risk assessment.
92

-------
Table 2. Parents' Mean Risk Perceptions (%).

Risk of Getting
Conditional Risk of
Sample
Sample
Skin Cancer"
Dying from Skin Cancer
Size
All Parents
23.90
12.24
610
All Children
20.54
9.44
610
Black Parents
11.79
12.98
122
Black Children
12.88
9.77
122
Whites:



All Parents
27.61
12.15
460
All Children
22.76
9.44
460
Mothers
29.79
12.54
351
Fathers
20.59
10.90
109
Daughters
22.76
9.39
230
Sons
22.76
9.49
230
Children aged 3 to 7 years
24.47
10.35
258
Children aged 8 to 12 years
20.57
8.28
202
aInitial risk assessment.
93

-------
Table 3
Hypothetical Sun Protection Product Labels
Percent Change in
Morbidity Risk
Label	Parent Child
A
10
10
B
10
50
C
50
10
D
50
50
E
0
0
F
0
0
G
0
0
H
0
0
Percent Change in
Mortality Risk
Parent Child
0
0
0
0
0
0
0
0
10
10
10
50
50
10
50
50
94

-------
Table 4
Willingness to Pay for Reduced Risk of
Skin Cancer

Full Samp
e
Variable
Mean
Estimate
(t-ratio)
Estimate
(t-ratio)
Constant
—
4.028
(130.32)
4.023
(86.63)
GET=1 if label changes risk of getting skin
cancer;
=0 if label changes conditional risk of dying
from skin cancer
0.500
-0.089
(-1.992)
-0.093
(-2.079)
PARENTCHG=1 if parent risk change = 50%;
=0 if risk change = 10%
0.498
a
a
K1DCHG=1 if child risk change = 50%;
=0 if parent risk change = 10%.
0.496
a
a
GET*PARENTCHG
0.249
0.251
(6.82)
0.252
(6.86)
GET*K1DCHG
0.251
0.436
(11.84)
0.437
(11.85)
(1-GET) *PARENTCHG
0.248
0.309
(8.38)
0.306
(8.30)
(1 -GET) *KIDCHG
0.245
0.340
(9.23)
0.339
(9.22)
FAMILY INCOME
($10,000 per year)
5.325
—
0.031
(7.66)
NUMBER OF CHILDREN
INHOUSEHOLD
2.075
—
-0.076
(-5.23)

—
1.029
(53.79)
1.023
(53.65)

-------
Table 5
Estimated Marginal Rates of Substitution
(Asymptotic Standard Errors in Parentheses)

White Parents
Marginal Rate of
Substitution
Full
Sample
Black
Parents
All
Mothers
Fathers
Child is
Daughter
Child
is Son
Child
Age 3-7
yrs
Child
Age 8-12
yrs
Child vs. Parent
Unconditional Morbidity
2.05
(0.35)
a
1.61
(0.27)
1.83
(0.35)
0.96
(0.38)
1.14
(0.24)
2.60
(0.88)
1.42
(0.29)
2.22
(0.74)
Child vs. Parent
Unconditional Mortality
2.33
(0.32)
0.52
(0.20)
3.28
(0.51)
3.24
(0.58)
3.35
(1.02)
2.01
(0.40)
5.40
(1.46)
4.38
(0.90)
1.73
(0.46)
Unconditional Mortality vs.
Unconditional Morbidity
(Parent)
19.16
(3.15)
a
11.35
(1.83)
10.76
(2.08)
14.36
(4.09)
10.42
(1.78)
14.17
(4.93)
7.86
(1.55)
24.60
(7.73)
Unconditional Mortality vs.
Unconditional Morbidity
(Child)
21.78
(2.59)
22.48
(8.98)
23.19
(2.84)
19.06
(2.57)
50.05
(15.74)
18.42
(3.55)
29.38
(4.84)
24.25
(3.58)
19.18
(4.71)
Number of Parents
610
122
460
351
109
230
230
258
202
a Estimate is negative but not significantly different from zero at the ten percent level.
96

-------
Figure 1. Risk Scale.
Next Question
What is your risk of getting skin cancer sometime
during the rest of your life?
When you're ready to move oil click the "Next
Question" button.
97

-------
Figure 2. One of Eight Sun Lotion Labels.
Developed with dermatologists to protect
skin from harmful effects of sun exposure.
Skin Cancer	Ultra
Protection	Waterproof
SPF		Parsol«> 1789
"Making the outdoois safer
foi you and youi family."
98

-------
New Skin Saver® sun protection lotion.
Skin Cancer Protection
v Used as directed in clinical trials. SkiiiSaver
reduced risk of skin cancer by:
v Used as directed in clinical trials. SkinSavei; had
no effect on the risk of dying if skin cancer
occurred.
More Skin Protection
Moie Ai oof fonniil.i - One .i|>|>lk.itkni Lists all thiy *
*	Noii-come<|o.illfrnienic * PABA-fiee * Unsc-enteil *	
DIRECTIONS: Apply fjeneionsly 
-------
Comments on "Not All Deaths Are Created Equally"
Glenn W. Harrison
Department of Economics
College of Business Administration
University of Central Florida
November 2003
Cameron and DeShazo undertake an ambitious survey to elicit valuations of health
episodes that respect the timing of the episode. They incorporate latency of onset, as well as
latency of the experience, for a wide range of possible health episodes. Mortality and morbidity
are treated in the same conceptual framework, which is a major attraction. In short, their attempt
to undertake valuation of the "health life cycle" is novel and important. The strengths of the
study are well presented by the authors.
They claim that one novelty of their work is that they calculate the value of a statistical
illness (VSI) rather than the value of a statistical life (VSL). Since many of the hedonic wage-
risk regressions also included non-fatal risks, this would seem to be a trivial innovation.
Important to do, but nothing to claim as novel.
Similarly, they claim to show that individual differences matter for the VSI and VSL.
Again, this is worth saying as loudly as possible, but isn't this already in older data, even if has
tended to be ignored in analysis? That is, many of the earlier studies included a rich array of
individual characteristics that could have been used to predict VSL estimates that would have
varied over those characteristics. So I am not sure what is conceptually novel here.
With respect to heterogeneity of VSI or VSL estimates, I worry a lot about the treatment
of negative predicted values. These appear to have been set to $0, but that causes obvious biases
in the aggregate estimates. One of the grubby secrets of VSL analysis, particularly in some
recent meta-analyses, is that negative or statistically insignificant estimates are dropped or set to
zero. One understands the desire of the Environmental Protection Agency to "see big VSL
numbers," but such mis-handling of the data is not acceptable. I am not sure that such things are
going on here, but the results appear to be very sensitive to how certain observations are
dropped, and this deserves more careful discussion.
Related to this, the practice of deleting "outliers" should be completely reconsidered. If
my data set includes a Bill Gates, who is an outlier in terms of income and wealth, and he
responds in a numerically extreme manner, then in what sense is that an outlier? There are
answers to this question that make sense, but they should not be enshrined in mechanical rules
for dropping subjects. The present analysis has some questionable bases for dropping
observations, and that needs further exploration.
100

-------
The massive cognitive burden on respondents is an obvious concern, but we need to start
somewhere if we are to examine health life-cycles. My concern here is tempered by the desire to
see someone spell out a complete framework for valuation, such as one has here. But one cannot
take the responses too seriously for policy work, given the uncontrolled context in which
subjects were responding and the unfamiliarity of the task.
Related to this concern, I believe it was a major error to start such a complex survey with
elaborate field survey procedures. While the Knowledge Networks technology is fascinating, it
makes much more sense to pilot surveys of this kind in a less constrained and more controlled
setting. My understanding is that such data points cost roughly $50 per subject, which seems a
lot for such a pilot study. I appreciate that the authors undertook a large number of focus groups,
and consulted some smart people in the field about design issues, but that is no substitute for
controlled comparisons of different ways of presenting tasks and evaluation of cognitive burden.
The authors implicitly take the view that there is only one way to generate a social VSL -
to estimate the individual VSL for different segments of the population and then take some
appropriately weighted average. Of course, there are other ways to arrive at the same concept.
One could elicit a household VSL rather than an individual VSL, and then weight those. Or one
could directly elicit a social VSL from individuals or households. There is no a priori reason for
the directly elicited social VSL to equal the weighted averages of individual or household VSLs.
Indeed, it would be interesting to see how they are related. In the same vein, estimates of
individual VSLs could be used in various social welfare functions to arrive at a social VSL. So
there are many paths to the social VSL, not just the one implicit here.
The statistical analysis is heavy on math that could be relegated to appendices, and light
on some of the nuts and bolts that likely drive the results. I should add that some of the "present
value math" is really very interesting, and notationally delicate, so I would not want to see that
lost; but it detracts from the general comprehension since it is better read off-line. My concern is
more with the way the data is handled. I have already discussed the handling of negative
valuations and outliers. But I missed the use of control for characteristics other than income,
own-age and age-of-onset of the disease. Since this is, after all, a conference on children's health
valuation, one is entitled to a "where is the beef?" question: why no controls for whether the
respondent has children, or how many? I suspect that there may be some dark computational
reason why more covariates are not thrown in, akin to why one sees so few covariates in the
"stated choice" literature. But this needs a simple explanation and evidence, rather than
assertions that it would not change results.
There are some hidden assumptions about risk aversion and individual discount rates that
need to be made explicit. These are not minor matters. Once we recognize individual
heterogeneity with respect to the valuation of uncertain future heath states, we have to confront
the fact that these estimated values will necessarily confound the certainty-equivalent valuation
of the health state, individual risk attitudes, individual discount rates, and possibly preferences
individuals might have over the temporal resolution of uncertainty. Is it possible that the first
component is constant with respect to the things it is claimed to vary over, but that the others
101

-------
vary? We simply do not know, and teasing these apart is a formidable enterprise. We do now
have relatively good estimates of risk attitudes and discount rates for individuals from controlled
laboratory experiments, in some cases conducted in the field with samples representative of
larger populations than college students, and one would hope that one could marry such
estimates to the valuations of health life-cycles to see what is really driving the VSI estimates.
The evidence so far suggests considerable heterogeneity of risk attitudes and individual discount
rates with respect to the standard observables, so we cannot ignore the issue by assuming
homogeneity (as is done here).
The authors loudly trumpet the claim that a "senior VSL discount exists!" This claim is
far too premature. The issue is an important one, but I am sensitive to a rush to judgment on such
an important issue from such a pilot study. My enthusiasm for the scope of this pilot study would
be nearly unbounded if I were not concerned that such claims would be ripped out of their
academic context. In terms of the old Latin motto, festine lente (hasten slowly).
102

-------
Comments on "Valuation of Environmental Risks to Children's Health"
Glenn W. Harrison
Department of Economics
College of Business Administration
University of Central Florida
November 2003
Dickie and Gerking get at risks to children through parental decisions. This is a sensible
way to get at children's valuation, and deserves to be explored further. Their elicitation scope is
also modest, which makes for a well-focused study. The study looks at morbidity and mortality
in the same framework, which is attractive. Related to this, one again sees the nice idea of a
"health life-cycle" as the conceptual setting for valuation.
Asking parents for valuation of risks to the health of children makes obvious sense, but
raises some questions. Which parent? The one that makes the decisions, or the one that was
picked at random? How would this differ from the situation in which both parents made the
decision, allowing them to endogenously resolve their internal household decision-making as
they do naturally?
One major concern, however, was whether subjects would or could keep morbidity and
mortality separate. It seems incoherent a priori that some product could reduce the risk of
contracting skin cancer but not reduce the risk of dying from it. At the very least, one would
surmise that they are highly, positively correlated. Given this, how do we know that the
responses that subjects make to the morbidity question are in fact just that, and do not include
concerns with mortality?
The data for blacks should e discarded. Given the low propensity of blacks to get skin
cancer, and the wide understanding of this by blacks, why should they ever rationally invest in
knowledge about the risks? At the very least, these data should be analyzed separately.
Related to this concern, why compare an individual's own perception of risk to the
population risk? It is quite possible that individuals know more about their own circumstances
than they do about the population as a whole. In particular, occupation may influence the amount
of time spent outside, which could be an important factor influencing individual risk.
Why rely solely on hypothetical responses? This is a setting in which it would have been
easy to use real incentives for risk elicitation, and possibly even elicit a real willingness to pay
(WTP) for sunscreen. Hypothetical bias is not obviously avoided by taking ratios for a marginal
rate of substitution, although that is an intriguing speculation worth investigation. We simply
have too much data on the unreliability and higher variance of hypothetical responses to ignore
when we are able to elicit responses for which subjects have real consequences.
103

-------
What effect might the existence of field substitutes for sunscreen have on responses?
Such field substitutes might be expected to play an important role, by censoring the WTP for a
new product. Simple statistical methods exist for handling this.
The elicitation format was double-bounded dichotomous-choice. That method is not
incentive compatible when responses are real, since the subjects have an incentive to reduce their
payment for the good by misrepresenting. Hence the assumption under which it generates more
information, that the underlying population of valuations is invariant to the repeated sampling
sequence, is invalid. One could conjure up heavily-parametric ways to correct for this, but that
seems like a costly thing to do when one could avoid the problem by design. In a hypothetical
setting, subjects have no incentives for any response, but one hardly wants to rely on that
premiss to defend the double-bounded procedure! In the future this procedure should be dropped.
For now, at the very least the analysis should just use the first response, and then see if there are
large differences when the second response is included.
The authors note that the WTP for risk reduction of a child exceeds the WTP for risk
reduction for the parent. This need not be a puzzle, since these are not the same good. If 80% of
exposure causing skin cancer occurs up to age of 18, which is apparently the case, then one is
simply buying more health benefit for children. There should be a simple way to normalize these
estimates to account for this.
104

-------
Summary of Q&A Discussion Following Session IV-PM
Bryan Hubbell (U.S. EPA) addressed J.R. DeShazo and Trudy Cameron saying that he,
too, finds it interesting that they "rushed to the conclusion that we have this big age
difference," and adding that he "tried to look quickly at the confidence intervals and . . .
as far as I could tell, all of them overlap, so there's no statistical difference between any
of those numbers." He also suggested that it might be worthwhile to look at the
possibility of controlling the variance with respect to age, because "it certainly looked
like there might have been higher variance in the responses from the older individuals."
Trudy Cameron responded that "the first and last confidence intervals, for the youngest
group and the oldest group, don't overlap. . . . Linking together, all the intervening ones
do have some overlap, but, fortunately, the first and last ones don't.
Addressing the other comment, Dr. Cameron stated, "We have been estimating models
that employ systematic differences in the errors, and one thing that does show up, not
surprisingly, is education level. If we include, specifically, a dummy for less than high
school education—those folks are way noisier in the information they're giving us—but
as I recall, there wasn't a lot of other action on the dimension."
Laurie Chestnut (Stratus Consulting, Inc.) addressed what she termed a "primarily
empirical question" to Trudy Cameron and J.R. DeShazo. She said that in the survey she
and her colleagues conducted, they asked about physical health, which declined with age,
although a simple question regarding enjoyment of life showed no decline with current
age. On the other hand, she said, peoples' expectations regarding quality of life 10 to 20
years in the future did show a decline. In considering the list of all the terrible things that
happen as we get older, she urges "some qualification of, maybe while some things
deteriorate, some heart-felt appreciation for what we have left might be moving in the
other direction." She clarified that this possibility interests her partly because she is
facing a "big birthday" soon.
She went on to ask another question regarding what appears to her to be some "counter-
intuitive results" (admittedly preliminary) from the study. This concern regards the
finding that the value of a current risk reduction was found to be less for someone who is
65 than for someone who is 70, going from half a million to two million over this 5-year
span. Reiterating that this seems counter-intuitive, she questioned whether this might be
an artifact of the way the researchers "chopped the things up."
Trudy Cameron responded, "There's this one little anomaly in those sloping graphs of the
portion that hangs down below zero: People, in considering risk profiles that involved
something dreadful happening to them in the near term, the next five years, weren't
interested in that program, quite typically. So, there's this sort of bias against near-term
risks. So, the near-term negative willingness to pay is there for all the age groups."
105

-------
Glenn Harrison interjected, . . you didn't want to tell people that "your life expectancy
is one year," -you've jacked it up by 8 years."
J.R. DeShazo answered that peoples' "nominal assessment of their life expectancy and
what we told them that the doctor would assess their life expectancy at were both inflated
compared to what their actual life expectancy was." He then explained a couple of issues
relevant to Laurie Chestnut's question, saying, "One is that people's information set
contains a focus on their immediate perceived risk, and then people seem to have
confidence intervals around that as they look further into the future—they're more
willing to accept that 5 or 10 years from now they may face a threat of a heart attack or a
stroke, whereas they really feel healthy today and so anything in the next 2 or 3 years
they disbelieve."
Speaking to Ms. Chestnut's initial point, Dr. DeShazo clarified that nothing in their
analysis suggests that older people value a year of their life less. However, he said, the
evidence does suggest that people begin to look at the gains from avoiding illness
differently as they get older. He cited the fact that after the age of sixty-five 50 to 60
percent of people have some chronic condition—"they're in some state of morbidity, and
their assessment of avoiding other kinds of morbidity in the future changes. Their
information set upon which they base their willingness to avoid a worsening of the health
state changes." He clarified that what he and his colleagues have found is that "how
much you're willing to pay to avoid a loss in the future changes because that loss looks
smaller the more morbid your current health state becomes."
Alan Krupnick (Resources for the Future) said that he also was interested in the question
about the latency result. He said that he and his colleagues had found a strong latency
effect in their study, which looked at the whole population aged 40 to 60 and their
willingness to pay for a reduction in the risk of death beginning at age 70 and going up to
80. They found a "strong lower willingness to pay for that contemporaneous risk
reduction." He asked the researchers to consider the question: "If you looked at it that
way, would you find that same effect?"
He then addressed two questions to Shelby Gerking: Stating that he and his colleagues
found that blacks are willing to pay more than whites for an equivalent risk reduction, he
asked whether Dr. Gerking and his colleagues had found that to be true also. In addition,
he commented that a lot of researchers have struggled with trying to get people to
understand conditional probability, and this seems so implausible to him. He asked for
their views on, for example, a person saying "When I apply this sunscreen, it doesn't
reduce my risk of getting cancer, but it reduces my risk of dying from cancer," which he
related to their latter cases.
In regard to Dr. Krupnick's first question, Trudy Cameron replied that she didn't know
off the top of her head but they could surely run a simulation that captures the same type
of information he had gathered and find an answer.
106

-------
Shelby Gerking conjectured that blacks aren't willing to pay a lot for sunscreen probably
because their risk of getting skin cancer is much lower than it is for whites "so the result
here is specific to the context in which it's estimated." He continued by saying,
"Regarding the plausibility of dying from skin cancer," it's an issue that he and Mark
Dickie "worried about quite a lot and then went ahead with on the basis of the results
they got initially." He closed by saying that it didn't seem to be as big a problem as he
initially thought it might have been and he was pleasantly surprised.
J.R. DeShazo stated, "We found that when you look at individuals' subjective assessment
of their risks for specific illnesses and then their ability to mitigate and defend against or
control those risks that there was a lot of variability in socio-economic and ethnic
characteristics."
Don Kenkel (Cornell University) said he was struck by the fact that "there is an active
market to get skin cancer—in the tanning booths—where people spend time and money
to do this." He commented that he wondered about the implications of the fact that there
is "a very real sense that skin cancer prevention is jointly produced with being pale." Are
there whites who aren't going to pay for sunscreen because of this? Furthermore, Dr.
Kenkel posited that some people's attitude that "I'm willing to make my kids have
disutility to keep them safe, but/want to look good" might explain some of the
differences observed.
Shelby Gerking responded that these questions and the ones brought by Alan Krupnick
are really important. He said that he was involved in a prior skin cancer study that looked
at the issue of the joint products associated with tanning beds and being out in the sun—
"some people want some tanning; others don't want premature aging and wrinkling—and
we tried to sort all of that out in an earlier paper and discovered that the joint production
effects . . . really weren't that important to consider. So, even though we do consider
them a little bit in this study, it's not to the same extent as in the earlier study, and I didn't
talk about that at all."
107

-------
Valuing Environmental Health Risk Reductions to Children
PROCEEDINGS OF
SESSION V: AIR POLLUTION AND ASTHMA IN CHILDREN
A WORKSHOP SPONSORED BY THE U.S. ENVIRONMENTAL PROTECTION
AGENCY'S NATIONAL CENTER FOR ENVIRONMENTAL ECONOMICS (NCEE),
NATIONAL CENTER FOR ENVIRONMENTAL RESEARCH (NCER), AND OFFICE
OF CHILDREN'S HEALTH PROTECTION; AND THE UNIVERSITY OF CENTRAL
FLORIDA
October 20-21, 2003
Washington Plaza Hotel
Washington, DC
Prepared by Alpha-Gamma Technologies, Inc.
4700 Falls of Neuse Road, Suite 350, Raleigh, NC 27609

-------
ACKNOWLEDGEMENTS
This report has been prepared by Alpha-Gamma Technologies, Inc. with funding from
the National Center for Environmental Economics (NCEE). Alpha-Gamma wishes to
thank NCEE's Kelly Maguire and Project Officer Nicole Owens for their guidance and
assistance throughout this project.
DISCLAIMER
These proceedings are being distributed in the interest of increasing public understanding
and knowledge of the issues discussed at the workshop and have been prepared
independently of the workshop. Although the proceedings have been funded in part by
the United States Environmental Protection Agency under Contract No. 68-W-01-055 to
Alpha-Gamma Technologies, Inc., the contents of this document may not necessarily
reflect the views of the Agency and no official endorsement should be inferred.
11

-------
TABLE OF CONTENTS
Session V: Air Pollution and Asthma in Children
Valuing Reduced Asthma Morbidity in Children
W. Michael Hanemann, University of California, Berkeley, and Sylvia
Brandt, University of Massachusetts, Amherst	1
Behavioral Reactions to Ozone Alerts: What Do They Tell Us About
Willingness To Pay for Children's Health?
F. Reed Johnson, Research Triangle Institute	22
A Cross-Sectional Analysis of Asthma and Air Pollution
Charles Griffiths, Nathalie Simon, and Tracey Woodruff, U.S. EPA,
National Center for Environmental Economics. Presented by: Charles
Griffiths	47
Discussant
Bryan Hubbell, U.S. EPA, Office of Air Quality Planning and Standards	70
Discussant
Laurie Chestnut, Stratus Consulting	87
Summary of Q&A Discussion Following Session V	91
Closing Remarks
Mark Dickie, University of Central Florida	95
in

-------
Valuing Reduced Asthma Morbidity in Children*
Presented at
EPA Workshop of Valuation of Children's Health
October 20-21, 2003
Sylvia Brandt
Assistant Professor (corresponding author)
Department of Resource Economics
University of Massachusetts, Amherst
Brandt@resecon.umass.edu
Michael Hanemann
Chancellor's Distinguished Professor
Department of Agricultural Economics
University of California, Berkeley
Hanemann@are. B erkel ey.edu
Abstract
This economic study models household willingness-to-pay to minimize a specific health
endpoint: morbidity effects on children with asthma (defined as asthma symptoms including
coughing, wheezing and/or shortness of breath). The project addresses three main questions: 1)
what determines households' perceptions of risks to an asthmatic child, 2) what averting and/or
mitigating actions do households take, and 3) what are households' stated willingness-to-pay for
a reduction in their children's asthma morbidity.
Acknowledgements
The research is funded by the U.S. EPA - Science to Achieve Results (STAR) program grant R-
8296650. Mihail Samnaliev provided excellent research assistance for this project. Valuable
input on survey design was provided by members of the Fresno Asthmatic Children's
Environment Study [FACES] including Dr. Kathleen Mortimer, Dr. Jennifer Mann, and Beth
McDonald. We thank Dr. Matthew Sadof, Associate Director of Ambulatory Pediatrics at
Baystate Children's Hospital for his helpful suggestions and gracious assistance. The authors take
full responsibility for any errors or omissions.
This is a report on research in progress. Please do not quote without authors' permission. Please contact the
authors for the most recent report.
1

-------
DRAFT
I. Introduction
While both our medical understanding of the mechanisms involved in asthmatic episodes and the
resources devoted to its treatment have increased, the rise in asthma is a well-documented
international phenomenon. The CDC estimates that 14 million Americans have asthma,
including 5 million children. Despite dramatic improvements in the understandings of the
mechanisms of asthma and asthma therapies, from 1980-1994, the prevalence of the disease has
increased 75% and the mortality rate for children under 19 has increased 79%. Asthma is the 2nd
leading cause for pediatric emergency room visits (behind accidents) and is the most common
reason for school absenteeism.
The economic burden of asthma and asthma therapy in the United States is large and growing.
The majority of economic analyses of asthma use a cost of illness method (for reviews see
Jonsson (2000) and Weiss and Sullivan (2001)). These studies categorize costs into direct costs
(cost of medical treatment) and indirect costs (loss of production). The total direct and indirect
cost of asthma in the U.S. was estimated to be $6.2 billion in 1990 (Weiss, Gergen and Hodgen,
1992) and $12.7 billion in 1998 (Weiss and Sullivan, 2001)1. Lozano et al. (1999) estimate that
children, ages 1 to 17 years, with asthma incurred an average cost of $1129 per child per year in
total health care expenditures compared to $468 for children without asthma2. The intangible
cost of asthma, the loss of utility due to the disease, is omitted from this body of literature. A
second component missing from cost of illness studies is the cost of risk avoiding or risk
mitigating behavior. Cost of illness studies therefore should be taken as a lower bound of the
true cost of asthma.
While these direct costs of asthma are large enough to justify substantial policy interest, asthma
is also of great interest because it is a disease whose burdens have significant distributional
ramifications. The health burdens associated with asthma fall disproportionately on the young
and the poor in the United States. The increase in asthma has been largest in children (under age
18), and the rate of hospitalization for the disease is greatest for those from poor neighborhoods.
(See Koren, 1995, and Claudio et al., 1999.) A comparison of asthma hospitalization rates in
New York neighborhoods found that while children in lower Manhattan and Queens
neighborhoods with average household incomes greater than $57,000 had zero hospitalizations
from asthma, children in East Harlem, where the average household income is $19,000, had
hospitalization rates of 222 per 10,000 youths. (Claudio et al. 1999)
The valuation of reduction in asthma morbidity is of significant relevance for public policy
decisions targeted at children and susceptible populations. Asthmatics have physiologic
differences, such as more narrow airways, and, therefore may be more susceptible to the health
effects of air pollution. Relative to adults, children also may be more susceptible because they
are more physically active, spend more time outdoors and therefore breathe more pollutant per
1	The direct costs include cost of medical treatments: inpatient hospitalization, inpatient physician services,
emergency room care, outpatient care, outpatients physician services, medications). The indirect costs include: lost
workdays of caregiver, lost workdays of asthmatics, loss of lifetime earnings from asthma mortality.
2	Total expenditures included prescriptions, ambulatory provider visits, emergency room visits, and hospitalizations.
2

-------
DRAFT
pound of body weight than do adults (American Academy of Pediatrics, 1993). Therefore,
asthmatic children represent a susceptible population of particular policy interest. This project
consists of a first survey to analyze households' risk perception, their risk reduction behavior and
the costs of averting and mitigating behavior and a second survey on stated willingness to pay for
reduced asthma morbidity in children.
II. Theoretical Models
A. Modeling Willingness to Pay: A Household Production Approach
Households' behavior will be modeled using a health production approach as introduced by
Grossman (1972). Unlike Cropper (1981), this study will follow the approach of Gerking and
Stanley (1986) in which ambient air quality will enter as a factor in the production of health.
This study models household behavior to minimize a specific health endpoint: morbidity effects
of pollution on children with asthma (defined as asthma symptoms including coughing,
wheezing and/or shortness of breath). We are interested in the incidence of asthma symptoms
among children clinically diagnosed with asthma, not with the prevention of the disease. The
surveys will ask what choices the household makes to minimize the risk of asthma exacerbation
in that survey period; therefore, the household model will not be dynamic.
Following the standard household model (Freeman, 1993), the health outcome is a function of
pollution exposure and the mitigating and averting behavior of the household. The standard
approach assumes that individuals know their health production function, choose their level of
output optimally and choose inputs to minimize costs. An important contribution of this study is
that our surveys on households will provide information on households' risk perceptions, their
averting and mitigating behavior, the costs of such behavior, and the households' evaluation of
effectiveness of their actions. Our estimation therefore does not rely on proxies for perceived
risks and does not assume households perfectly predict risk.
The health outcome, S, is a measure of asthma morbidity (e.g. cough, wheezing, or shortness of
breath). This outcome will depend on pollution exposure, D; mitigating behavior, B; and other
socio-demographic variables, Z. Mitigating behavior includes preventative medication and other
investments that reduce the effect of pollution exposure. For example, control medications, an
entire class of drugs for mitigation, are prescribed to reduce the hyper-responsiveness and
inflammation of asthmatics' airways3. In addition to mitigating behavior, the household can also
engage in averting behavior, A, which includes actions to minimize the exposure to pollution; an
example of averting behavior is the purchase of home air filters. As a result, pollution exposure,
D, is a function of both pollution level, C, and averting behavior, A.
To summarize, the measure of asthma morbidity is written as a function of exposure, mitigation,
and other covariates that affect health outcome:
3 Control medications for severe asthmatics include inhaled corticosteroids. For moderate asthmatics, cromolyn
sodium can be prescribed to reduce airway hyper-responsiveness.
3

-------
DRAFT
(1)	S = S(D, B, Z)
where the exposure to pollution is a function of the levels of pollutants and the household
averting behavior:
(2)	D= D(C, A)
where
S = measure of asthma morbidity
D = realized exposure to environmental pollution
C = levels of pollutants
B = mitigating behavior of household
A= averting behavior of household
Z = covariates that affect health outcome
The utility of the household is a function of consumption goods (X), leisure (L), and morbidity of
the asthmatic child (S). As in Dickie (1999), the household maximizes a single utility function
where children are "passive" in that they comply with parents'/guardians' decisions.
(3)	U= U(X, L, S).
The implication of this utility function is that pollutants affect household well-being only
through their impact on health and they have no other associated disutility.
The household has a budget constraint that total income equals total expenditures on
consumption goods and on averting and mitigating behavior. The household loses days at work
when the severity of the asthmatic symptoms warrants the child's absence from school. The
budget constraint is written:
(4)	I + W(T - L- aS ) = X + Pa A + Pb B
where
I = non-wage income
W = wage rate
T = total available time to work
X = consumption goods
L = leisure time
Pa = price of averting behavior
Pb = price of mitigating behavior
aS = lost days of work due to attending to child with asthma symptoms
Px = 1, the price of bundle of consumption goods is normalized to one
The household maximizes its utility function (3) subject to its budget constraint (4) with respect
to the choice variables, X, L, A and B. Using (1) and (2) in the utility function, the household's
maximization problem is:
4

-------
DRAFT
(5) Max	U(X, L, S (D(C,A), B, Z))
X,L,A,B
subject to X + Pa A + Pb B = I + W(T-L)-Wa5( D(C, A), B, Z )
The resulting first order conditions for an interior solution are:
(6a) cU/dX = X
(6b) dU/dL = A.W
(6c) (au/as) (cS/cD) (oD/oA) = X [Pa + Wa (8S/SD) (cD/dA)]
(6d) (au/as) (dS/dB) = X [Pb + Wa (dS/dB)]
where X is the Lagrangean multiplier. Some manipulation yields:
(7a) raj/as^) =Wa + 	P;l	
(du/ax)	(5S/SD) (8D/8A)
(7b) rau/as^ =wa +	ph
(au/ax)	(as/aB)
The solution to the first order conditions is a set of household demand functions for leisure, for
consumption goods, for averting behavior, and for mitigating behavior:
(8)	X = X(Pa, Pb, W, I + WT, C, Z)
(9)	L = L(Pa, Pb, W, I + WT, C, Z)
(10)	A = A(Pa, Pb, W, I + WT, C, Z)
(11)	B = B(Pa, Pb, W, I + WT,C, Z)
Our collaboration with Fresno Asthmatic Children's Environment Study [FACES] makes it
possible for us to use parametric methods to estimate the functions for realized exposure to
environmental pollution, D = D(C,A), and for asthma morbidity given exposure to pollutants, S
= S(D, B, Z). The data from our economic surveys of risk mitigating and averting behavior will
enable us to estimate the demand functions for A and B in (10) and (11). Combining these pieces
of information and choosing appropriate functional forms for these expressions will make it
possible for us to identify the underlying household utility function, U(X,L,S) (see Hanemann,
1991 and Hanemann & Kanninen, 1999).
This information can be utilized to estimate the household's willingness to pay for either a
marginal or non-marginal reduction in pollution levels. If the demand functions (8) - (11) are
substituted into the original utility function, one obtains the indirect utility function
(12)	U = V(Pa,Pb,W,I + WT,C,Z).
5

-------
DRAFT
Given a change in pollution levels from C° to C1, the household's utility changes from U° =
V(Pa, Pb, W, I + WT,C°, Z) to U1 = V(Pa, Pb, W, I + WTjC1, Z). Suppose this change is an
improvement. The household's willingness to pay for the change is given by the quantity WTPC
where:
(13)	V(Pa, Pb, W, I + WT - WTPc,C\ Z) = V(Pa, Pb, W, I + WT,C°, Z)
The household's marginal willingness to pay for a small increment in pollution, AWTPC / AC,
can be shown to be measured in terms of mitigating behavior by
(14)	AWTPC/ AC = -Pb [(5S/5C) / (dS/dB)].
and in terms of averting behavior by
(15)	AWTPC/ AC = -Pa[(0S/0C)/(0S/0A)].
The implication of this household health production model is that the marginal WTP for a
reduction in pollution can be estimated using observable costs of household behavior, and the
non-marginal WTP can be estimated using the utility function that is recovered when one
combines the observed demand functions for mitigating and averting behavior together with the
health production functions D(C,A) and S(D, B, Z).
B. Stated Willingness to Pay: Contingent Valuation
Our model assumes that a child's well being is a part of a household utility function which
determines parent's behavior. We propose a utility maximization model which follows previous
models in that area (Rosenweig and Shultz, 1983; Gerking and Stanley, 1996; and Dickie and
Gerking, 1986). Household's utility is a function of a vector of market goods not related to
health, X, a vector of health related goods, Z, income, I and health, H. The utility function is a
random utility model linear in income and covariates, and has the general form:
(1)	U0 = U0 ( X, Z, H, I ) + 80
A simple model of health production defines health as a function of health capital (K) and
averting/mitigating behavior (A) which is determined by a set of health beliefs (B). This set of
beliefs, includes risk perceptions, self-efficacy regarding desired outcomes, etc.
(2)	H = H (A, K,)
The theoretical marginal willingness to pay (WTP) is the maximum amount that households are
willing to pay to mitigate their asthma by forgoing some of the market goods and hold the utility
at a constant level. Individuals are asked to pay a dollar amount, W for reduction in asthma
morbidity, and with some positive probability they agree to this amount. Then their utility
function is:
(3)	Ui = Ui(X, Z, H, I-W) + 8!
6

-------
DRAFT
The probability that they will say yes to this amount is
(4)	Pr[Yes] = Pr [Ui(X, Z, Hls I-W) + si > U0(X, Z, H0,1) + 80
which can be rewritten as
(5)	Pr [80 - 81] < Ui(X, Z, Hb I-W) - U0(X, Z, H0,1) = AU = WTP
i.e. at the point of indifference, where
(6)	Ui(X, Z,Hh I-W) - U0(X, Z,H0,1) = 0
the marginal value of reduction in morbidity equals to the marginal disutility of paying for this
reduction. The utility function is assumed to have the following functional form
u = u (a + p *1) and the difference in utility, AU is assumed to have a logistic cumulative
density function.
(7)	AU = (1+e"AU)"1
Then,
(8)	AU = (oci - oco ) - p *W
The median WTP is calculated by
(9)	Pr [Ui(X, Hi, I-W) > U0(X, H0,1) ] = 0.5
Modeling directly the WTP function (which is assumed to be a linear random function), an
approximation of compensating surplus, using the formula derived by Hanemann (1984) has the
form:
(10)	Pr[Yes] = (l+e"a"pw)^
where a is the grand intercept evaluated at the mean values of the covariates and (3 is the
estimated coefficient for W.
Median WTP is calculated by solving the above expression for Pr[Yes] = 0.5 which yields
(11)	Median WTP = e ~(a/p)
Median WTP is calculated for the positive part of the probability function, by integrating within
the interval
T
(12)	Mean WTP = J[1 - Gwtp]dW ,
7

-------
DRAFT
where Gwtp is the distribution function of the true willingness to pay. T is infinite for the true
willingness to pay and is truncated at some value for the purpose of estimation.
C.	Socio-economic Indicators and Risk Reducing Behavior
Significant research has documented the disparities across ethnicities in hospitalization rates for
asthma; however, the empirical quandary is disentangling which of the correlated social
economic status indicators are the factors that create the disparity in morbidity. Recent research
indicates that minority children were more likely to underuse preventative medications that could
reduce asthma severity (Fiscella et al., 2000; Halterman et al., 2000; Eggleston et al., 1998). This
underuse of preventative medication in minority populations is consistent even when there are
not disparities in financial access and insurance coverage (Lieu, et al, 2002). Thus we are
complementing standard economic instruments with elements used in the public health literature
and psychological literature, specifically the Health Belief Model and Theory of Planned
Behavior.
The Health Belief Model [HBM] predicts health behavior as a function of four groups of
determinants, each of which leads to specific beliefs and incentives that are then motivators for
preventative action. Commonly used to predict preventative behavior, HBM is particularly
appropriate to our study of households' actions to minimize asthma triggers and comply with
asthma control medication regime. The four major components of the HBM are: perceived
susceptibility/vulnerability, perceived severity of the disease, perceived benefits from taking
action, perceived barriers from undertaking action.
The Theory of Planned Behavior [TPB] explains behaviors as functions of behavioral intentions,
which are explained by the individual's attitude and subjective norms toward performing the
specific behavior (Fishbein and Ajzen, 1975). Attitudes are based on beliefs about the likelihood
of an event and evaluation of the consequences of a particular action (Smith and Stasson, 2000).
Social norms are determined by what is socially acceptable and by personal motivation to
comply with family expectations. An additional element of interest is that of self-efficacy, the
individual's perceived ability to perform specific actions under specific conditions (Bandura,
1977). We believe that the elements of these models will contribute to understanding of risk
reducing behavior, particularly with respect to compliance with asthma management protocol.
D.	Prior expectations
Self-efficacy will be quantified through a standard five-point psychometric scale measuring self-
reported efficacy in managing asthma. Applied to asthma mitigating behavior, parents with high
level of self-efficacy would be expected to be more effective in their interventions in their child's
asthma. In the context of the major domains affecting asthma care, self efficacy affects averting
behavior on three levels: (1) the amount of attention that the child receives from the medical care
providers4; (2) school acceptance and attention on the part of teachers and nurses, and (3)
compliance to medications, in cases where the long term beneficial effect of asthma medications
is not known to parents.
4 Due to subtlety in asthma symptoms, some parents could not get admission by the emergency room registration
unless they were very assertive, and others reported to have avoided emergency rooms because they couldn't
persuade the registration that their child needed to be examined
8

-------
DRAFT
Perceptions about risk are expected to have a positive effect on WTP, however this effect would
be uneven. Risk factors which have a 'salient' effect, (i.e. perceived to be riskier to asthma
outcomes as compared to what the scientific risk is) is expected to inflate WTP. Factors that are
perceived less risky than they should be will have a deflating effect on WTP.
E. Statistical Analysis of Survey Data
Three types of statistical analysis will be performed, dealing with household choice of averting
and mitigating behavior in the context of a health outcomes production function, the
determinants of household risk perceptions, and estimation of responses to stated preference
questions.
Household Choice of Averting and Mitigating Behavior, and Health Production Function
This involves estimating the behavioral equations for averting and mitigating behavior (10) and
(11), together with the reduced form health production function S = S(D(C,A),B,Z). Both of
these involve some issues arising from how the variables are measured.
Because both mitigating and averting behavior consist of discrete actions, an index of behavior
will be constructed. For mitigating behavior that is repeated daily, the components of the index
will be the frequency of each type of behavior over the previous three months. Likewise, for
averting behavior that is repeated, the index will be a function of the frequency of each type of
averting behavior. In the case of averting behavior, however, there is a class of actions that are
essentially one-time investments. Therefore, there will be a second component of the averting
index for fixed averting investments. Because the behaviors are discrete and the indices are
inherently ordered, the demand system will be estimated using ordered probit. Maximum
likelihood estimation will be used.
The dependent variable in the health production function is asthma morbidity. Because
households are observed over multiple periods, we can improve upon existing valuations of
asthma by disaggregating morbidity into presence of asthma symptoms and the severity of
symptoms if present. The presence of symptoms is an indicator variable.
S = 1, if symptoms are present
S = 0, otherwise.
If symptoms are present, then the severity of symptoms (M) is rated on a scale from one to ten.
M = 1 ,..., 10 where 1 indicates mild symptoms and 10 indicates extreme symptoms.
Therefore, a two-stage estimation will be used. The first stage is a binomial logit where the
outcome is the presence of symptoms (S=1,0). If symptoms are present, then in the second stage
the severity of symptoms (M) is estimated as a Poisson process.
Risk Perception
A goal of the study is to analyze the determinants of household's perceptions of the risk that
different risk factors pose to their child and to investigate how their risk perceptions compare
9

-------
DRAFT
with objective assessments by medical and scientific experts. By asking households to evaluate
the impact of typical asthma triggers on their child's asthma symptoms, we can create discrete
dependent variable that is an index of the household's risk perception. An example is to ask, "If
your child is exposed to tree pollen are his/her asthma symptoms: greatly affected, slightly
affected, not affected at all?" The epidemiological data provides an index of the degree of that
child's asthma response to fluctuations in pollen. Using these data we can construct a
contingency table of the households' subject indexes and the objective risk indexes.
The risk perceptions variable takes the form of a ranking by the respondent of the seriousness of
each risk factor for that household. Because of the form of this dependent variable, we will use a
model for an ordered categorical response variable, such as ordinal probit or logit, when
analyzing the rankings to investigate what are the significant socio-demographic factors that
influence the household's perceptions of risk and whether factors such as the age of the child or
recent onset of symptoms affect risk perceptions. The other major issue is the correlation
between subjective household perceptions of risk factors and objective assessments of these
factors by scientific experts. To test this relationship, we can use a limited dependent variable
model where the dependent variable is the subjective risk index, and independent variables
include the objective risk index, household characteristics and relevant interaction terms
III. Empirical Study
A.	Collaborative Economic and Epidemiological Study
One criticism of studies of households' behavior is estimation bias due to omitted variables (see
Atkinson and Crocker, 1992 and Harrington and Portney, 1987). By collaborating with an
extensive epidemiological study of the effects of air pollution on asthmatic children [Fresno
Asthmatic Children's Environment Study, FACES] we minimize the potential for omitted
variable bias. The FACES study includes a large sample, follows households over multiple years
and will incorporate the most detailed socio-demographic, indoor air quality and pollution
monitoring data collection effort to date (California Air Resources Board). This project
complements the work of Rowe and Chestnut (1986) and O'Conor and Blomquist (1997) by
focusing on children's health and generating detailed data on children's clinical health status and
household behavior.
The FACES cohort includes children with clinically diagnosed asthma, residing in a section of
Fresno County, California5. Children are 6-10 years of age at intake and will be followed for
approximately 4 years. The study population will include children who have a physician's
diagnosis of asthma and at least one of the following: 1) reported utilization of or valid
prescription for asthma medication in the previous 12 months; or 2) symptoms consistent with
asthma in the past 12 months; or 3) an emergent asthma visit or hospitalization in the past 12
months. The requirements for asthma medication use, symptoms, or health care utilization are to
minimize the chance of enrolling subjects whose asthma is quiescent (remission). Children who
meet these criteria may be enrolled regardless of the severity of asthma.
B.	The Study Area
5 FACES has been recruiting households for the survey since 2000.
10

-------
DRAFT
Located in the Central Valley of California, Fresno County has a population of 815,734 which
has increased by 19.8% since 1990. Forty-four percent of the population is of Hispanic or Latin
origin, followed by forty percent of white origin, eight percent Asian and five percent African-
American. The Fresno population has lower medium income, less education, poorer living
conditions and a greater percent of residents below the poverty line as compared to the rest of
CA. For example, median household income for 2001 was $34,725 as compared to $47,493 for
California. The proportion of residents with a high school degree was 67.5% as compared to
76.8%) for the rest of the state, and the proportion of residents below the poverty line was 22.9 %
while that in CA was 14.2% (US Census data, 2000). The asthma hospitalization rate in Fresno
is among the highest in California at 28.8 per 10,000 (California Facts, 2003).
A study of pediatric asthma-related hospital discharges in California shows that the very young
children (0-4 years of age), African-American children and males were over represented in the
discharge population (see Table 1).
Table 1: Pediatric Asthma-Related Discharges in California
Variable	Frequency	Percentage of
discharges
Age
0-4	64,260	57
5-11	33,485	29
12-17	16,229	14
Race
White	46,696	57
Latino/a	30,986	27
African-American 28,802	25
Asian	7,490	7
Gender
Male	71,935	63
Female	42,039	37
Source: Calmes, Leake and Carlisle, "Adverse asthma outcomes among children hospitalized with asthma in CA",
Pediatrics, 1998; 101(5), 845-50. This study includes 114,000 records from hospital discharge records.
C. The FACES Cohort
The FACES study has complete screening interviews for 473 households, baseline interviews for
241 households, and currently has 205 participating households. The major reasons households
who inquired about the study were ineligible to participate include: other chronic disease, lived
in house for less than three months, sleep at home less than five nights/week, and planned to
move within two years (Mann, 2003).
11

-------
DRAFT
The ethnicity of the children in the FACES study is representative of the Fresno general
population. Forty-three percent of the sampled parents were Hispanic, followed by 16.7% black
and 37.5% white. The unemployment among the FACES cohort is more similar to that of the
population hospitalized for asthma, than the general Fresno population.
Table 2: Number of Asthma-Related Hospitalizations of FACES Cohort by Race
Race
% of FACES Sample
Zero
Hospitalizations
One or more
Hospitalizations
Hispanic
43.0%
19 (61%)
12 (39%)
Black
16.9%
5 (42%)
7 (58%)
White
33.8%
19 (76%)
6 (24%)
Source: Authors' analysis of FACES survey data.
The majority of the interviewed households were covered by health insurance (90.3%). Almost
70%) households had at least one parent who was affected by asthma. Table 3 presents a general
description of the households participating in FACES.
Table 3. Characteristics of Households Participating in FACES
Household
Characteristics
Selected Variables
Relative Frequencies
Employment
Mother employed
61.1% Yes
38.9% No

Father employed
69.4% Yes
27.8% No
Health Insurance
Is child currently covered by
health insurance?
90.3% Yes
9.7% No




Health History
Mother diagnosed with asthma?
48.6% Yes
51.4% No

Father diagnosed with asthma*?
31.9% Yes
61.3% No
Source: Authors' analysis of FACES baseline survey data.
* 2.8% missing
FACES data on asthma hospitalization, ER visits and intensive care unit visits showed that
34.7%) of the children had been hospitalized at least once in their life, 36.1%> had received
unscheduled asthma care (such as emergency room) and 12.5% had been placed in intensive
care units because of asthma. As expected, number of hospitalizations was lower among
Hispanic and white (Table 2), which is consistent with state level hospital discharge data. For the
state of California, African-Americans were hospitalized 3 times more for asthma than any other
ethnic group. In our sample we get consistent results: the percentage of blacks enrolled in the
FACES program (16.7%>) is much greater than the percentage of blacks for the Fresno population
(5.3%>). The average age of children in the FACES cohort is between eight and nine years.
Table 4. Characteristics of Children Participating in FACES
Child Characteristics
Selected Variables

Ages of children
Mean age (standard deviation)
Median age
8.6 years (1.8)
9 years

Frequency by current grade in
school
5.6% in kindergarten
1st grade = 20.9%
2nd grade=13.4%
12

-------
DRAFT


3rd grade= 16.4%
4th grade= 17.9%
5th grade = 23.9%
6th grade = 7.5%
Health History
age of mother when child was born
Mean = 26.7(5.5)
Median = 27.5 years

Gestation length
26.4% Early
29.2%) Late
44.4% On time

Child seen by doctor or other
health care provider for a chest
illness before the age of 2 years
44.4% Yes
54.2% No

Was child ever hospitalized
because of asthma?
34.7% Yes
65.3% No

Presence of hayfever or allergic
rhinitis
29.1% Yes
65.3% No
5.6%) missing
Source: Authors' analysis of FACES baseline survey data.
D. Initial Findings of EPA-STAR Project
Survey One
We have conducted five focus groups in Fresno, California and nine personal interviews in
Springfield, Massachusetts. The focus groups and interviews were conducted over an eleven
month period, from July 2002 to May 2003. In the summer of 2003 the survey instrument was
reviewed by asthma specialists including Drs. Kathleen Mortimer, University of California-
Berkeley School of Public Health, and Matthew Sadof, Associate Director of Ambulatory
Pediatrics at Baystate Children's Hospital. During the fall of 2003, the team wrote the protocol
for contacting families and tracking all surveys and correspondence. By late October 2003, the
survey will be mailed to all households participating in the Fresno Asthmatic Children's
Environment Study and households with an asthmatic child who were either ineligible or
declined to participate in longitudinal environment study. We extended the sample group to
include families outside of FACES because recruiting for the epidemiological study was lower
than predicted.
Risk Reducing Behavior
Through these focus groups and interviews we identified issues central to the survey. In common
to all respondents was the increase in the monitoring of the child's health, and in some cases
caregivers changed or terminated careers to increase supervision. The goal of the monitoring was
to "catch the asthma before it was too late", that is to employ rescue medication while they were
still effective in increasing lung function. The need for constant monitoring entails both reduced
earnings and psychosocial costs due to the strain on family and social relationships.
There was a wide range in responses to questions on risk reducing behavior employed by
households. A surprising result of the focus groups and interviews was that when initially asked
if the household had changed anything due to the asthmatic child's health, respondents tended to
significantly underestimate their change in behavior. Then when directed through a series of
13

-------
DRAFT
specific changes or activities pertaining to reducing triggers, households revealed a range of
changes from small to extensive. Our conclusion is that it is often very difficult for households to
identify "what they do for asthma" because either the child had been experiencing respiratory
distress for such a long time that there is no basis for comparison or the changes have become
such a routine that it is difficult to compare their behavior over time.
One disturbing finding in the focus groups was the length of time between onset of symptoms
and correct diagnosis of asthma. Despite national guidelines on diagnosing and managing
asthma, the median time until diagnosis was over 1 year. In multiple cases, children were
repeatedly hospitalized over multiple years before being correctly diagnosed with asthma. This
delay reflects both a need for more training of healthcare providers (Halterman et al, 2000;
Cloutier et al, 2002) as well as lack of continuity of care.
Past experience with healthcare providers was correlated with a sense of self-efficacy in
controlling asthma symptoms. Those households that experienced a long delay between
symptoms and diagnosis were less likely to feel that they were able to control asthma symptoms.
In contrast households that were provided with asthma management plans had a sense of
improved self-efficacy. Self-efficacy has been shown in previous studies to be positively
correlated with risk reducing behavior. Thus in modeling compliance with medication, and
mitigating and averting behavior, then length of time between symptoms and diagnosis may be
an important factor.
An early hypothesis was that income, transportation and lack of health insurance were dominant
barriers to general healthcare. We found that in our study group the most significant barriers to
care were lack of access to asthma specialists due to insurance protocols and insufficient supply
of urgent care facilities. In addition, "gatekeepers," either receptionists who schedule
appointments within the medical practice or triage nurses in emergency rooms, were commonly
cited as impediments to reaching physicians during asthma episodes.
Several respondents voiced concern over balancing all the actions that could reduce asthma
morbidity versus instilling a sense of confidence or creating a sense of being "normal" for the
child. This points out that the clinical guidelines for optimal household behavior may deviate
from household behavior when the psychosocial costs of the risk reducing behavior are
incorporated.
Risk Perception
Respondents were able to list common asthma triggers and to rate which they felt were most
significant to their child (see Table 5). When the allergy testing is completed we will be able to
compare stated risk of allergens to clinically measured objective risk.
During the focus groups we observed inconsistencies between subjective and objective risk from
air pollution. Respondents felt strongly that air pollution was a significant trigger and was
significantly worse during the summer months. However, in the Fresno-Clovis area the
concentrations of particulate matter are higher during the winter months, posing a real threat to
asthmatics. The discrepancy could be due to the public awareness of high ozone alert days in the
summer and the lack of such campaign for particulate matter.
14

-------
DRAFT
Table 5: Parents' Perception of Asthma Triggers
ininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininHinininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininininini&inBnininininininininininin

Environmental factors that made child

Rank
wheezing worse
% of Yes responses
1
Weather (multiple options allowed)
44.8
2
Physical activity
44.8
3
Cold or flu
39.7
4
Cold air
37.9
5
Air pollution
36.2
6
Pollen, grasses
32.8
7
Windy conditions
29.3
8
House Dust
22.4
9
Outdoor smoke or fires
15.5
10
Molds
13.8
11
Perfume or Odor
12.1
12
Wood smoke
12.1
13
Cigarette smoke
12.1
14
When crops are being sprayed
10.3
15
Pets
10.3
16
When fields are being plowed
6.6
17
Others
5.2
Source: Authors' analysis of FACES baseline survey data.
Medical Intervention
A critical component of risk reducing behavior is compliance with prescribed asthma medication
and monitoring of respiratory function using a peak flow meter. While respondents were able to
list most of the medications their child took for asthma, it was apparent that there were wide
discrepancies in understanding of the role of each medication. There was significant concern
over the side-effects of inhaled steroids despite the clinical evidence that their benefits greatly
outweigh their risks. In addition personal disposition was evident in both the manner in which
the child's guardian interacted with the healthcare provider and with compliance. For example,
while a written asthma management plan and peak flow meter are standard and critical tools for
asthma management, less than half of the FACES cohort used either.
Table 6: Asthma Management
Has a physician or other health provider given 48.6% Yes
a written plan for managing asthma?	50.0% No
1.4% missing
Does child use a peak flow meter?	40.3% Yes
59.7% No
Source: Authors analysis of FACES baseline survey data.
15

-------
DRAFT
Counter to our expectations, there was not an ethnic disparity in the use of a written management
plan (Table 7).
Table 7: Use of a Written Management Plan bj Race
Race	Yes	No
""Hispanic	15(48%)	15(52%)	
Black	7 (58%) 5 (42%)
White	11(44%) 14(56%)
Total % with	48.6	50.0
management plan
(1,4%=missing)
Source: Authors' analysis of FACES baseline survey data.
Initial analysis indicates an association between parents' behavior and personal experience with
asthma. For example more than half of the children were not seen by a medical care provider for
a chest illness before the age of 2, which was associated with whether parents had history of
asthma themselves as shown in Table Eight. Among 62.5% of families where both parent were
diagnosed with asthma child was seen by a health care provider for chest illness before the age of
two, as opposed to 27.3% in families where none of the parents had asthma. Child was taken to a
HCP for chest illness more often in families where the mother had asthma as compared to
families where the father had asthma. It should be noted that due to the small number of
observations, whether the differences are significant is not determinable. Additionally we are not
asserting a causal link. At the same time race did not play a role in whether the child was seen by
a doctor for chest illness (Table Nine).
Table 8: Parental Asthma and Respiratory Illness of Children Before Age Two
mmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmnBnmmmmmmmmmmmmmmmmmmmmmmrlmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmmm
Parental Asthma
% of
Child was seen before
age of two

total
Yes
No
Neither
31.4%
6 (27.3)
16(72.7%)
Both mother and
11.4%
5 (62.5)
3 (37.5%)
father



Mother but not father
35.7%
14 (56%)
11 (44%)
Father but not mother
21.4%
7 (44%)
9 (56%)
Source: Authors' analysis of FACES baseline survey data.
16

-------
DRAFT
Table 9. Race and Respiratory Illness of Children Before Age Two
Race
% of total
Yes
Child was seen before age of two
No
Hispanic
43.0%
12 (38.7°/
'o) 19(61.3%)
Black
16.9%
6 (50%)
6 (50%)
White
33.8%
12 (50%
12(50%)
Other (or missing)
5.6%
2 (50%)
2(50%)
Source: Authors' analysis of FACES baseline survey data.
Survey Two: Contingent Valuation
The second component of the economic valuation of reduced morbidity is a contingent valuation
question. Critical to this instrument is that the scenario be relevant and realistic. From the
discussions in the focus groups we developed two types on contingent valuation questions. In
the first scenario we asked parents to trade work-hours for reduced number of bad asthma days.
The second scenario proposed a hypothetical insurance program that would provide additional
services that were predicted to reduce asthma symptoms. We will conduct additional focus
groups and interviews to refine these questions.
IV. Future Research
Currently the team is awaiting the data from the first survey on risk perception and household
behavior. We are in addition in the process of designing a contingent valuation instrument.
Similar to the development of the first survey, we will use extensive focus groups and interviews
to develop a valid instrument. Some aspects of previous CV instrument are discussed below.
Some of the studies employing WTP for a specific commodity include earlier studies by
Chestnut and Row (1986) and Dickie and Gerking (1996). In the first study asthmatics were
asked about their maximum WTP to implement a program that would abate pollution and will
reduce the number of asthma bad days by half. The payment vehicle in this study was WTP for
an increase in taxes per year. In the Dickie and Gerking (1996) study elicited maximum WTP to
relieve one symptom for 1 day and WTP to reduce daily one-hour maximum concentrations of
pollutants by 1/10"6 for 1 day.
Blumenschein et al. (2001) conducted a field experiment comparing hypothetical and actual
purchase decisions for an asthma management program. Subjects received either a dichotomous
choice contingent valuation question (where three bids were offered, $ 15, 40, and 80) or were
given the opportunity to actually enroll in the program. In an earlier study (Blumenschein and
Johannesson 1998), as well as in Blumenschein et al. (2002 ) the same authors used both a
dichotomous choice and a bidding game approach to elicit willingness to pay for asthma cure. In
another study by Barner J.C. et al. (1999) patients were presented with a hypothetical 8-week
asthma management program and patients were asked how much they would be willing to pay
for the program as well as how much time they would be willing to spend on the program.
17

-------
DRAFT
Risk-risk valuation and risk income tradeoffs were proposed by Viscusi, Magat and Huber
(1991) to value risk reduction for contracting a lung disease. Respondents were asked to choose
between two alternative cities which differed in the probability of getting a lung disease and the
probability of dying in an auto accident. Individuals were presented with different scenarios until
they were indifferent between the alternatives. The point of indifference was used to measure the
MWTP for decrease in the risk of lung disease as well as the ratio between the two risks.
Krupnick and Cropper (1992) used the same valuation setting to measure the effects of
familiarity with the disease on WTP and Sloan et al. (1998) used the same tradeoffs to estimate
the value of risk of multiple sclerosis. In a more recent study, Blomquist and O'Conor (1997)
emphasized the need to separate respondents into people familiar and people unfamiliar with the
disease. They proposed a hybrid form of WTP elicitation and found that it worked among people
familiar with asthma but was unreliable among respondents unfamiliar with the disease. In the
WTP question, respondents were asked to choose between two hypothetical drugs A or B, that
differed in their effectiveness and safety and then elicited WTP for a third, improved drug that
has greater effect (but was equally safe) than drug A and was safer (but had the same effect) than
B .
In summary, earlier contingent valuation studies have elicited WTP for programs aimed at
reduction of asthma symptoms, while later research has focused on risk reduction and risk-risk
tradeoffs. Elicitation of WTP needs to be conducted using a specific payment vehicle that makes
the payment scenario tangible to respondents, and in case of risk valuation, the benefits from a
proposed risk reduction need to be easily comprehensible by respondents.
V. Conclusion
Asthma presents social scientists with complex questions. This project seeks to integrate
elements of the Health Belief Model and Theory of Reasoned Action to model household risk
reducing behavior and risk perceptions. We can use these survey results to model a household
health production function. In addition using the epidemiological data we can compare subject
to objective risk assessments. The final stage of the project will be to administer a contingent
valuation instrument on reduced asthma morbidity.
18

-------
DRAFT
References
American Academy of Pediatrics, "Committee on Environmental Health", Pediatrics. 1993; 91,
1212.
Atkinson, S. and T., Crocker, "Econometric Health Production Functions: Relative Bias from
Omitted Variables and Measurement Error," Journal of Environmental Economics and
Management. 1992; 22, 12-24.
Bandura, A., Social Learning Theory. Prentice-Hall Inc: Englewood Cliffs, NJ. 1977.
Calmes, Leake and Carlisle, "Adverse asthma outcomes among children hospitalized with
asthma in CA", Pediatrics. 1998; 101(5), 845-50.
California Facts, 2003. Report available at:
http://www.ehib.org/cma/links/CaliforniaAsthmaFacts010503.pdf.
Cameron, T., "Concluding Remarks", EPA Workshop on Valuation, Session IV, March 24-25,
1999.
CDC Statistics on Asthma Prevalence, April 25,2001 available from www.cdc.gov.
Claudio, Luz, Leon Tulton, John Doucette, Philip J. Landrigan, "Socioeconomic Factors and
Asthma Hospitalization Rates in New York City," Journal of Asthma. 1999; 36(4), 343-350.
Cropper, M., "Measuring the Benefits from Reduced Morbidity," American Economic Review.
1981; 71(2), 235-40.
Dickie, M., "Willingness to Pay for Children's Health: a Household Production Approach", EPA
Workshop on Valuation, Session IV, March 24-25, 1999.
Dickie, M., Gerking, S., "Formation of Risk Beliefs, Joint Production and Willingness to Pay to
Avoid Skin Cancer", The Review of Economics and Statistics.1996: 451-62.
Eggleston, PA., FJ. Malveaux, AM Butz, et al, "Medications Used by Children with Asthma
Living in the Inner City," Pediatrics. 1998; 101, 349-354.
Fiscella K., P. Franks, MR Gold, CM Clancy, "Inequality in Quality: Addressing
Socioeconomic, Racial, and Ethnic Disparities in Health Care," Journal of American Medical
Association. 2000; 283, 2579-2584.
Fishbein and Ajzen, Understanding Attitudes and Predicting Social Behavior. Prentice-Hall Inc:
Englewood Cliffs, NJ. 1975.
Freeman, M., Measurement of Environmental and Resource Values. Resources for the Future:
Washington, D.C.. 1993. 344-350.
19

-------
DRAFT
Gerking, S., and S. Stanley, "An Economic Analysis of Air Pollution and Health: The Case of St.
Louis," Review of Economics and Statistics. 1986; 68 (1), 115-121.
Grossman, M., "On the Concept of Health Capital and the Demand for Health," Journal of
Political Economy. 1972; 80(2), 223-55.
Halterman, JS., A. Aligne, P. Auinger, JT. McBride, PG. Szilagyi, "Inadequate Therapy for
Asthma Among Children in the United States," Pediatrics. 2000; 105(suppl), 272-276.
Hanemann, W. Michael and Barbara Kanninen, "The Statistical Analysis of Discrete-Response,"
In Valuing the Environment Preferences: Theory and Practice of the Contingent Valuation
Method in the US. EC and Developing Countries, edited by Ian Bateman and Ken Willis,
Oxford: Oxford University Press, 1999.
Hanemann, M., "Willingness to Pay and Willingness to Accept: How Much Can They Differ?,"
The American Economic Review. 1991; 81 (3), 635-647.
Hanemann, M., "Welfare Evaluations in Contingent Valuation Experiments with Discrete
Responses." American Journal of Agricultural Economics. 1984; 66(3): 332-41.
Harrington, W. and P. Portney, "Valuing the Benefits of Health and Safety Regulation," Journal
of Urban Economics. 1987: 22(1), 101-12.
Jonsson, Bengt, "Measuring the Economic Burden in Asthma," In Asthma's Impact on Society.
editors Weiss, Kevin, Sonia Buist, and Sean Sullivan, Marcel Dekker: New York, NY. 2000.
251-268.
Koren, H. S., "Associations Between Criteria Air Pollutants and Asthma," Environmental Health
Perspectives. 1995: 103(6), 235-42.
Lieu, T. et al, "Racial/Ethnic Variation in Asthma Status and Management Practices Among
Children in Managed Medicaid", Pediatrics. 2002; 109(5), 857-865.
Lozano, Paula, Sean Sullivan, David Smith and Kevin Weiss, "The Economic Burden of Asthma
in U.S. Children: Estimates from the National Medical Expenditure Survey", The Journal of
Allergy and Clinical Immunology. 1999; 104(5), 957-63.
Mann, J. "Presentation on FACES" 2003.
Mortimer, Kathleen and Herman Mitchell, A Guide for Helping Children with Asthma. National
Cooperative Inner-City Asthma Study. National Institutes of Allergy and Infectious Diseases,
NIH.
O'Conor, Richard and Glenn Blomquist, "Measurement of Consumer-Patient Preferences Using
a Hybrid Contingent Valuation Method" Journal of Health Economics. 1997; 16 (6), 667-683.
20

-------
DRAFT
Rosenweig, M. and T.P. Schultz, "Estimating a Household Production Function:
Heterogeneity, the Demand for Health Inputs, and Their Effects on Birth
Weight," Journal of Political Economy. 1983; 91, 723-46.
Rowe, Robert and Lauraine Chestnut, "Oxidants and Asthmatics in Los Angeles: A Benefits
Analysis," Report to the Office of Policy Analysis, U.S. EPA, 1986.
Smith BN, Stasson MF, "A comparison of health behavior constructs: Social Psychological
Predictors of AIDS-Preventive Behavioral Intentions," Journal of Applied Social Psychology.
2000; 30 (3), 443-462.
Weiss, K., P. Gergen and T. Hodgen, "An Economic Evaluation of Asthma in the United
States," New England Journal of Medicine. 1992: 326, 862-866.
Weiss, K. and S. Sullivan, "The Health Economics of Asthma and Rhinitis. I. Assessing the
Economic Impact," The Journal of Allergy and Clinical Immunology. 2001; 107(1), 3-8.
US Census data, 2000.
21

-------
Behavioral Reactions to Ozone Alerts:
What Do They Tell Us About Willingness-to-Pay for Children's Health?
Carol Mansfield
F. Reed Johnson
George Van Houtven
Jui Chen Yang
RTI International
Zachary Pekar
U.S. Environmental Protection Agency
Douglas Crawford Brown
University of North Carolina
October 2003
Paper prepared for presentation at
EPA/UCF Workshop: Valuing Environmental Health Risk Reductions to Children,
October 20-21, 2003

-------
Twelve-year-old Justin Turnage's asthma flared up again this year, and his doctors say ozone is
the likely culprit, said Turnage's mother, Deborah Leonard of Raleigh. Now Leonard hopes that
board games and music lessons will keep her son indoors on ozone alert days.
"Summer is going to be very hard for him," Leonard said.
(James Eli Shifter "Triangle Skies Smoggier," News & Observer, May 1, 2001)
1. Introduction
Ozone does not directly cause asthma, but triggers symptoms in susceptible individuals,
including young children and asthmatics. The most direct averting action an individual can take
to avoid the health problems associated with ozone is to stay indoors. In addition to the medical
costs associated with treating and controlling asthma, high levels of ozone pollution limit the
outdoor activities in which susceptible individuals, such as a young, asthmatic child can
participate. In the language of economics, high ozone levels reduce an individual's or a family's
choice set, and as the quote at the top of the page implies, this imposes welfare costs on the
family beyond the expenses for medical treatment.
According to the latest report by the American Lung Association (AI_A), while ozone
levels have declined in some areas of the country ozone pollution is increasing in others (AI_A,
2001b). Table 1 lists the 15 counties with the highest ozone levels and the number of orange,
red, and purple ozone alerts between 1997-1999. According to the U.S. Environmental
Protection Agency (EPA), in 1998 approximately 21% of children lived in counties where ozone
standards were exceeded on at least one day (EPA, 2001). Asthma prevalence also increased
over the decade of the 1990's. Among children in the U.S., asthma is now the most common
chronic illness (EPA, 2001). An estimated 26.3 million people had been diagnosed asthma at
some point in their lives according to data collected in the 1998 National Health Interview
Survey presented by the ALA (ALA, 2001b). The 5-17 year old age group had the highest
prevalence of physician diagnosed asthma, which is estimated to have increased from 130.1
per 1,000 people in 1997 to 135.0 per 1,000 individuals in 1998. Several studies provide
evidence of the link between ozone and asthma. A recent study in the Journal of the American
Medical Association (Friedman et al., 2001) documented fewer admissions of children to the
emergency room for asthma attacks in Atlanta during the 1996 summer Olympics. Atlanta
residents were encouraged not to drive and ozone levels were lower during that period than
normal.
Several studies have looked at defensive behavior in response to high levels of ozone
pollution. Bresnahan, Dickie, and Gerking (1997) used data from a panel of adults in the Los
23

-------
Angeles area who were contacted between 2-5 times over a 12 month period and asked about
their activities in the previous 2 days and their medical expenses. Their results indicate that
individuals do change their behavior in response to poor air quality by reducing time spent
outside on a day-to-day basis.
A recent survey conducted by RTI International in the summer of 2000 provides
additional evidence supporting the results from Bresnahan, Dickie and Gerking (1997).
Approximately 6,100 respondents from over 1,000 counties were asked about their knowledge
of the ozone alert program. Forty-six percent of the counties represented in the survey
experienced at least one day of code orange (or worse) air quality in 2000, covering 75 percent
of the respondents. Thirty-seven percent of respondents in these counties were aware of the
ozone alert system, compared with 28 percent of respondents in counties that did not
experience a code orange (or worse) day.
Table 1. Number of High-Ozone Days in America's 15 Most Ozone-Polluted Counties
Number of High Ozone Days in the Unhealthy Ranges, 1997-
1999
County
State
Orange
Red
Purple
San Bernardino
California
160
74
52
Riverside
California
154
54
24
Kem
California
167
55
4
Fresno
California
178
44
5
Tulare
California
180
19
0
Harris
Texas
78
43
21
Fulton
Georgia
92
18
8
Los Angeles
California
72
28
10
Rockdale
Georgia
70
31
4
Anne Arundel
Maryland
85
23
2
Mecklenburg
North Carolina
89
18
0
Sevier
Tennessee
91
11
0
Blount
Tennessee
88
12
0
Ventura
California
89
8
2
Knox
Tennessee
81
13
0
Source: American Lung Association (ALA). 2001a. "State of the Air: 2001." New York: American Lung
Association.
24

-------
In counties that had experienced a code red (or worse) day during the summer of 2000,
41 percent of respondents were aware of the system, compared with 33 percent in counties that
had not experienced a code red (or worse) day. Of those who resided in counties that had
experienced a code red day and were aware of the ozone alert system, 58 percent correctly
reported that their county had experienced a code red day during that summer. On ozone alert
days, 38 percent of the respondents reported driving less and spending less time outdoors, 19
percent reported only spending less time outdoors, 7 percent reported only driving less and 36
percent reported no changes in their behavior. In addition, people who are not working at least
part-time, including homemakers, the unemployed, students and retirees were more likely to
report reducing the time they spent outdoors on high ozone days. Because these groups have
more opportunity to be outside and more control over their schedules, we might expect to see
greater responsiveness on their part. Furthermore, people who reported excellent or good
health were less likely to report reducing outside time on high ozone days compared to people
with fair or poor health.
A number of studies have valued the benefits of reducing ozone through averting
behavior or with a contingent value (CV) study. Dickie and Gerking (1991) examined the
decision to seek medical care. They found that willingness-to-pay (WTP) for ozone levels that
never exceeded 12 ppm was 2 to 4 times higher than medical cost savings associated with the
reduction in ozone. Rowe and Chestnut (1985, 1986) asked a WTP contingent value question
for a 50% reduction in "bad asthma days." WTP estimates based on 65 responses from adult
asthmatics and approximately 18 parents of children with asthma range from $11.81 to $53.80
to avoid one day of asthma symptoms ranging from no symptoms to moderate symptoms (in
1990 dollars). More recently, Yoo and Chae (2001) conducted a CV survey of WTP to reduce
ozone levels in Korea, and Farber and Rambaldi (1993) conducted a CV survey to determine
adult exercisers' WTP to improve air quality. Johnson, Banzhaf, and Desvousges (2000) report
WTP of CAN$158 for one day of asthma symptoms with significant activity restrictions and
lower amounts for less severe restrictions.
Importantly, however, none of these studies has specifically examined behaviors and
values related to protecting children from ozone exposure. There are many difficulties involved
with estimating benefits for children. Children do not make decisions for themselves and do not
have income, thus traditional WTP measures cannot be elicited from them. In the place of
values elicited from children, researchers typically measure the WTP of parents to protect their
25

-------
children from health risks, often inferring WTP from decisions to purchase market goods that
contribute to safety such as cars or bicycle helmets (Schulze et al., 2000; Jenkins et al., 2000).
This study was designed to fill this gap in children's health research. Its primary focus is
to investigate how parents of young children alter their behaviors in responses to high ozone
concentrations and how these behaviors are affected by the presence of high-risk (i.e.,
asthmatic) children in the household. In the process, it addresses a number of key research
questions including:
To what extent are children's risks from exposure to high ozone levels offset by
defensive/averting behaviors?
•	How much do parents value reductions in potentially harmful ozone exposures to their
children?
•	What costs (direct and indirect) are incurred by parents and children as a result of
behaviors to avert ozone exposures?
•	To what extent are people aware of and how much do they benefit from the presence of
ozone alert systems?
The primary data for this study was collected during the summer of 2002 through a
series of surveys with selected households across the US. This paper describes the conceptual
foundation for the study, the methods used for data collection and analysis, and the results of
some preliminary analysis.
2. Conceptual Framework
Bresnahan, Dickie and Gerking (1997) use a household production approach to develop
a model of decisions about seeking medical care and limiting time outdoors to avoid high ozone
levels. Following their model, we can specify the child's utility function as:
U= U(H, X, A, Z)
where H measures health status, X represents market goods, A measures an activity
such as outdoor leisure and Z measures exposure to ozone. In this very simple model, we
assume that parents have altruistic feelings for their child and maximize their child's utility. The
child's utility depends on his or her health and the activities he or she pursues during the day.
Under the assumptions of Bresnahan, Dickie and Gerking, health is produced using activity,
exposure to pollution, stock of preexisting health capital (K), and other human capital (S).
26

-------
H = H(A, Z, K, S)
Finally, the parent faces a full-income budget constraint:
I + wT = qxX + qAA + qMM(H) + wG(H)
Full income is composed of non-labor income (I) and the wage rate (w) multiplied by
total time available (T). The variables qx, qA.and qM represent time-inclusive prices for X, A and
M(H) (medical care) and equal the sum of the money price and time required to consume one
unit of the good (qj = Pj + wtj). Finally, G(H) is the time lost on market and non-market activities
as a function of current health status. The parent maximizes the child's utility subject to the
budget constraint. Under standard assumptions, the optimal level of A* can be derived from the
first order conditions for utility maximization.
(1) A* = A(qx, qA, qM, w, T, I, K, S, Z)
3. Survey Design
To inform the model, we conducted a series of eight surveys with a common set of
households across the country during the 2002 ozone season. The core of the data collection
effort is a series of six activity diaries (i.e., time and activity surveys). Time and activity surveys
are commonly used in transportation studies and in risk assessment and exposure analysis to
estimate actual exposure levels that individuals experience based on their activity patterns.
Each panel member completed an initial survey at the beginning of the summer to
collect some basic information and explain the activity diaries. After this, each member of the
panel was sent six activity diaries. A debriefing survey/stated preference survey was
administered in mid-December. The eight surveys adhere to the format described below:
•	Survey 1 (June 2002)
-	Screener—identifies households who qualify for the sample.
-	Baseline Questionnaire—collects information about the household, their dwelling,
neighborhood and health
•	Surveys 2-7 (July - September 2002)
-	6 Activity Diaries—record child's activities and health status for selected day
27

-------
• Survey 8 (December 2002)
-	Stated Preference Survey—presents hypothetical activity choice scenarios
-	Debriefing—collects information on awareness and perceptions about ozone levels
and alert system.
In the following sections, we describe the characteristics of our sample and provide more
detail on the design of the surveys, in particular the time and activity surveys
3.1 Panel Selection and Mode of Administration
We focused our data collection efforts on two samples—children with asthma and their
parents and children without asthma and their parents. Because of the acute effect of ozone on
asthmatics, parents of children with asthma may be more educated about ozone pollution and
the need to take defensive action (stay indoors) on high ozone days. Organizations such as the
American Lung Association publish guidelines that recommend limiting time outdoors on high
ozone days to avoid asthma and other respiratory problems. In addition, the ozone alerts
themselves provide information on which subpopulations should be limiting time outdoors for
each level of alert (see Table 2).
Table 2. Air Quality Index Color Code Guide
Air Quality
Health Effects
Good—AQI: 0-50
(Green)
Moderate—AQI: 51-100
(Yellow)
Unhealthy for Sensitive
Groups—AQI: 101-150
(Orange)
Unhealthy—AQI: 151-200
(Red)
Very Unhealthy—AQI: 201-300
(Purple)
No health effects are expected.
Unusually sensitive people should consider limiting prolonged
outdoor exertion.
Active children and adults, and people with respiratory disease, such
as asthma, should limit prolonged outdoor exertion.
Active children and adults, and people with respiratory disease such
as asthma, should avoid prolonged outdoor exertion; everyone else,
especially children, should limit prolonged outdoor exertion.
Active children and adults, and people with respiratory disease such
as asthma, should avoid all outdoor exertion; everyone else,
especially children, should avoid prolonged outdoor exertion.
Notes: AQI refers to the Air Quality Index. An AQI of 100 is equivalent to the National Ambient Air Quality Standard
(NAAQS). An AQI greater than 100 is considered to be above the national standard or NAAQS. An AQI
Calculation Table is available online to convert raw ozone concentrations to the Air Quality Index.
Source: U.S. Environmental Protection Agency (EPA). 2002. "Air Quality Guide for Ozone." .
28

-------
The respondents are all members of the Harris Interactive (HI) online market research
panel. The Harris panel consists of individuals who self-select onto the panel and have agreed
to participate in surveys over the internet. HI recruited the sample for this project and
administered the survey over the internet. The panel includes families in which, during the
summer of 2002, there was an asthmatic child or nonasthmatic child aged 2 to 12 years old and
at least one parent stayed home with the child during the day. An initial sample of 777
households was recruited in June and began taking surveys in July. An additional 200
households were recruited in July and began taking surveys in August. Approximately one-half
of the children in the panel are asthmatic. Response rates for the activity diary surveys were as
follows:
•	95% of those who qualified based on a brief screening survey took baseline survey to
form a panel of 977 individuals
•	977 people completed 2,940 diaries
-	80% completed at least 1 diary
-	12% completed 1 diary
-	11% completed 2 diaries
-	11% completed 3 diaries
-	14% completed 4 diaries
-	15% completed 5 diaries
-	17% completed 6 diaries
We chose this population because we believe this sample provides the most direct
measure of the efforts parents take to protect their children against the health risks of ozone.
Very little data exists on the averting behavior of both children and adults on high ozone days.
Children and especially children with asthma are a sensitive sub-population. We expect that in
general these groups (or their parents) will engage in a higher level of averting action than other
groups in the population. While the activities of this population may not generalize to other
groups, such as working parents with children in daycare, by focusing on the actions of children
who are home with their parents during the day, we expected to get the cleanest measure of the
direct actions parents take to protect their children's health. The survey was conducted over the
summer, when ozone is a problem and most school-age children are at home.
Respondents were drawn from the 35 metro areas in the US with the worst ozone
pollution (roughly corresponding to the counties with the worst ozone pollution in Table 1). The
29

-------
ranking is based on the number of code purple, red or orange days in 2001 (ALA, 2001a). See
Figure 1 for the locations of the 35 metro areas.
Figure 1. Metro Areas in the United States with the Worst Ozone Pollution.
Redd ins
Hamsburg({. ITOew York
* 	 Philadelphia
aster
Sacramento
Pitl
Modesto
* Merced
* Fresno
Visalik
* Bakersfrel(
Cincinnati
igton
LouisviJ
NafsKvilk—ftroXv
Memphis Chattel
Ral
Phoenix
Dallas
Biimingh:
>ato:
Houston
3.2 Survey 1: Screener and Baseline Questionnaire
The HI panel was screened at the beginning of the summer for families who met the
inclusion criteria. Those families who met the criteria completed the baseline survey. In this
survey, we collected information about the household's demographic characteristics, dwelling in
which the family lives, the child including the child's health and questions about the amount of
time the child usually spends on different activities. In addition, the parents of children with
asthma were asked a series of questions about the severity of the child's asthma, medications
the child takes, and changes to their house and lifestyle they have made to help control their
child's asthma.
30

-------
3.3 Surveys 2-7: Daytime Activity Diaries
The core of the research project is the activity diaries. These diaries were to be filled out
on-line by the parent within 48 hours of receiving the diary to minimize problems with recall.
Unlike a mail-in paper activity survey, we know the date and time the respondent completed the
survey. When a respondent missed a particular day or too much time elapsed, we asked the
respondent to provide information on their activities for another day rather than asking them to
remember what they had done several days ago. In total, each respondent was sent 6 diaries
to complete.
The diary takes the respondent through their child's day from the time the child woke up
until they went to sleep or 8:00pm (whichever came first). Respondents were instructed to
choose from a menu of activities and indicate the starting and stopping time of each activity.
The activities were drawn from the CHAD database, a database of activity diary studies
maintained by EPA. The CHAD database provides some information on the average level of
exertion (sufficient to calculate metabolic rates) associated with the activity, which will be useful
for the exposure assessment.
In addition to the start and stop time, respondents were asked to specify their
assessment of the level of physical exertion associated with the activity, whether the activity
took place indoors or outdoors, the location of the activity (at home or away from home with a
general description of how far from home in terms of driving time), whether there was a cost to
the activity and whether the activity was scheduled in advance. At the end of the diary,
respondents were asked about symptoms their child suffered during the day. Parents of
children with asthma were asked questions about their child's asthma and medication use
during that day and over the past week.
To avoid sensitizing the panel to ozone pollution through participation in the survey, we
did not inform the panel about the purpose of the survey beyond telling them that we were
looking for data on their activities. We have linked behavior to actual ozone levels on reporting
days.
The survey days were selected to include a variety of ozone conditions, where some of
the low ozone days were chosen with the same temperature as high ozone days. Ozone alerts
are predicted in the afternoon for the next day. Because the panel was connected by the
internet, HI was able to respond quickly and send out surveys based on these reports. The
31

-------
strategy for choosing the days of the interviews was based on balancing the need to collect
activity information under a variety of weather and ozone conditions with the cost of
administering the survey and burden on the panel.
Figure 2 presents the range of ozone and temperature conditions captured during the
study period. The larger circles correspond to cities with larger sample sizes. Ozone and
temperature are highly correlated, with worse ozone conditions associated with higher
temperatures.
Figure 2. Temperature-Ozone Distribution on Survey Days
115
Temperature
110
105
100
95
90
85
80
Green	Yellow	Orange	Red
Ozone Alert Level
Note: Larger bubbles indicate larger groups of respondents.
Los Angeles and San Diego respondents have been excluded from this figure.
3.4 Survey 8: Stated Preference Survey and Debriefing
We conducted the final debriefing and stated-preference survey in April 2003. The
purpose of this survey was to collect information about other variables that are important for
interpreting the time and activity data. Information collected includes the individuals' level of
knowledge about ozone and the health effects of ozone, their self-reported response to ozone
32

-------
(whether they consciously changed their schedule on high ozone days), and their subjective
assessment of the risks they and their children face from ozone pollution.
The activity diaries provide information about whether and how the child's schedule
changes in response to ozone conditions. The primary averting behavior to avoid ozone
exposure is to stay indoors. We expected that on high ozone days, some of the children on the
panel would stay indoors more than on low ozone days. But the activity diaries do not directly
collect information on the value the parents place on this lost outdoor time. To estimate that
value, the debriefing survey contains one of two series of stated-choice tasks based on either a
medicine commodity or city commodity. We discuss only the medicine version of the survey
here.
Like some actual antibiotics, the hypothetical medicine commodity requires limited
exposure to sunlight. Figure 3 contains the text that explains this feature of the medicine. Table
3 shows the attributes and levels used to construct the choice profiles. The experimental
design consisted of three randomly assigned blocks of five choice sets with two alternatives
each. We employed Zwerina, Huber, and Kuhfeld's (1996) algorithm to search for a near-
optimal design. Figure 4 shows an example choice task.
Figure 3. Definition of Outdoor Time Attribute
Assume that at the beginning of the summer, your family doctor tells you that [child's name]
needs to take a medicine during the summer as a preventive measure. In other words,
[child's name] is not sick, but [he/she] needs to take medicine to prevent an illness from
developing. ...
[Child's name] would have to limit the time spent outdoors on the days [he/she] takes
[his/her] medicine. Even on cloudy days or when [he/she] is wearing sunscreen, extended
exposure to the sun will make the medicine less effective.
33

-------
Table 3. Medicine Attributes and Levels
Attribute
Levels
Maximum number of minutes in the sun
allowed per day
•	10min
•	45 min
•	1 1/4 hours
Length of time child takes medicine
3 day
12 days
20 days
Total cost of medicine for the summer
•	$10
•	$40
•	$75
•	$150
Figure 4. Example Choice Task
Medicine A
Medicine B
Medicine Features
Number of days [name] would
have to take the medicine.
Maximum recommended outdoor
time on days when [name] takes
medicine.
Total cost of medicine to you.
(The cost not covered by
insurance).
3 days
during the summer
45 minutes
$150
for the summer
12 days
during the summer
10 minutes
$10
for the summer
Which medicine would you
purchase?
(Please check one box.)
Purchase A
~
Purchase B
~
34

-------
3.5 Supporting Data Collection Activities
In addition to the information collected from the panel we collected information on
predicted and actual AQI levels and weather-related data such as high temperatures on survey
days for each city. We also collected a copy of the newspaper in each city to document the
manner in which air pollution and ozone pollution information is presented.
4. Preliminary Results
4.1 Activity Survey
Our sample consists of 977 parents. As reported above, 780 households (80 percent) of
the sample completed at least one activity diary. Out of the 780 households, 486 (62 percent)
responded to the valuation and debriefing survey. Table 4 reports demographic characteristics
of the sample, including comparison between the households with asthmatic and non-asthmatic
children. On average, a household had 2 children with an annual household income less than
$75,000. One-third of the parents had college education or higher. Table 5 presents summary
statistics for the children and their activities by asthmatic and non-asthmatic. The median age
was 6 years old. Sixty-four percent of the asthmatic children and 50 percent of the non-
asthmatics were male. Thirty-three percent of the asthmatics and 25 percent of the non-
asthmatics participated in organized sports teams or lessons that practiced and played outdoors
during 2002 summer. Besides organized sports teams and other scheduled activities, both
asthmatic and non-asthmatic children spent 3 hours watching TV and 1 hour or more playing
video games each weekday.
Based on equation 1, the optimal level of averting behavior is a function of prices, health
capital and ozone exposure. We use two measures of averting behavior, the total hours that the
child spends indoors during the day and the proportion of the child's day spent inside. The
ozone forecast for the day represents the level of ozone exposure. We created a dummy
variable for days that were code orange or red where the excluded category is days that were
code green or yellow. Individuals who check the ozone forecast for the day may reduce their
child's outdoor time on code orange or red days. Individuals who do not check the ozone
forecast may observe their child suffering from symptoms and reduce the child's outdoor time.
Leaving aside the use of medicine, we expect that the optimal level of averting behavior will
increase in children with lower health capital, in our sample children with asthma. However,
35

-------
children with asthma may use either long-term daily medications or short-acting medications to
control their asthma that could affect the relationship between spending time outdoors and
health risks. Because the sample includes stay-at-home parents, the wage rate (or reservation
wage to join the workforce outside the home) is not included in the equation.
Table 4. Sample Characteristics

Non
Asthmatic
(n = 506)
Asthmatic
(n = 473)
All
Households
(N=979)
U.S.
Population
Children (median)
2
2
2
1
<$35,000
23%
16%
19%
42%
$35,000-75,000
40%
40%
40%
34%
% white
82%
88%
85%
75%
% high school grad
17%
19%
18%
32%
% coll grad or grad school
29%
37%
33%
25%
Table 5. Children: Characteristics and Activities


All Children
(N=979)
Asthmatic Non-asthmatic
Children Children
(n=473) (n=506)
Average age

6
7
5
% male

57%
64%
50%
% outdoor sports
(median hrs)

29%
(6)
33%
(6)
25%
(5)
% outdoor other
(median hrs)

15%
(7)
19%
(7)
11%
(6.5)
% indoor sports
(median hrs)

14%
(3)
16%
(4)
12%
(3)
% indoor other
(median hrs)

15%
(3)
15%
(3)
15%
(2)
Hours Spent on Watching TV (median)
3
3
3
Hours Spent on Playing Video Games
(median)
1
2
1
36

-------
We used the proportion of the child's day spent indoors (%INDOOR) to estimate the
level of averting behavior in response to high ozone levels and other factors that influence
parents' decisions about their children's daily activities. Besides temperature (TEMP), we also
monitored the ozone forecast for the selected days, which represented the level of ozone
exposure. CODE_RED indicates high-ozone days and AWARE indicates whether the parent
was aware of the color-coded ozone warning system. Three regional dummies (WESTCOAST,
NORTHEAST, and SOUTHEAST) capture climatic and other geographic differences. In
addition, we also include household and child characteristics in the analysis, including annual
household income (INCOME), whether the child is male (MALE), child's age (AGE), whether the
child prefers to play outdoors in the summer (OUTDOOR), and number of hours each weekday
the child spent on watching TV and/or playing video games (TVGAME). The mean value and
expected sign of each variable at diary level are reported in Table 6.
We excluded observations from Los Angeles and San Diego for the analysis. Both cities
have more than one weather monitoring station. We have not yet identified which sample
household lives near which station, so we have not included these observations in the
preliminary analysis. In addition, diaries covering less than 4 hours on a given day were
dropped from the sample.
Table 7 reports multivariate regression results, controlling for repeat observations from
the same household. Overall, both asthmatic and non-asthmatic children regressions are
significant at 1% level, but explanatory power is low. TEMP and CODE_RED are positive and
significant for both asthmatic and non-asthmatic regressions, indicating that children spent less
time indoors on cooler and non-code red days. Both effects are larger for children with asthma
than for children without asthma. These are the only coefficients that are significant for both
groups.
The interaction term (RED_AWARE) between CODE_RED and AWARE is negative in
both regressions, but not significant. Asthmatic children who live on the west coast spent less
time indoors while non-asthmatic children in the southeast spent more time indoors. The
positive asthmatic interaction term WESTCOAST_AWARE indicates that parents who were
aware of the ozone alert system and live on the west coast were more likely to have their
asthmatic children spend more time indoors. Oddly, parents who were aware of the ozone alert
system and live in the southeast were more likely to have their non-asthmatic children spend
less time indoors.
37

-------
38

-------
Table 6. Mean Value of Dependent and Independent Variables
and Hypothesized Coefficient Sign
Variable
Asthmatic
Non-
Asthmatic
Hypothesized
Sign
%INDOOR (%)
65.75
64.08

TEMP (°F)
94.38
94.82
+
CODE_RED
0.29
0.28
+
AWARE
0.55
0.53
+
RED_AWARE
0.14
0.15
+
WESTCOAST
0.09
0.09
?
NORTHEAST
0.41
0.29
?
SOUTHEAST
0.28
0.34
?
WC_AWARE
0.03
0.04
?
NE_AWARE
0.23
0.15
?
SE_AWARE
0.22
0.23
?
MALE
0.50
0.72
+
AGE (years)
6.15
6.93
-
PREFERS OUTDOORS
0.41
0.38
-
TV/GAMES (hrs)
6.09
6.23
+
INCOME ($10,000)
7.09
6.27
?
An interesting result is that children with asthma who play more video games and watch
TV also spend a greater proportion of their time indoors, while nonasthmatic children do not. It
seems tautological that more time spent in an indoor activity will increase the proportion of
indoor time. However, nonasthmatic children who play more video games apparently offset this
effect by also engaging in more outdoor activities.
Among children without asthma, male children, older children, and children in higher-
income households spend more time outdoors.
39

-------
Table 7. Regression: Dependent Variable Proportion of Time Indoors
Coefficient (Standard Error)
Asthmatic	Non Asthmatic
TEMPERATURE
0.83
***
(0.18)
0.51
***
(0.19)
CODE_RED
6.01
k
(3.21)
4.53
k
(2.65)
RED_AWARE
-5.58

(4.20)
-3.79

(3.52)
WESTCOAST
-14.29
kk
(6.06)
3.58

(5.48)
NORTHEAST
6.14

(4.27)
1.40

(3.36)
SOUTHEAST
-5.30

(4.97)
7.87
k
(4.13)
WESTCOAST_AWARE
17.30
¦k-k-k
(6.81)
1.10

(7.85)
NORTHEAST_AWARE
-2.32

(5.31)
1.89

(3.52)
SOUTHEAST_AWARE
4.40

(5.60)
-7.14
*
(4.33)
MALE
-3.05

(2.28)
-5.57
kk
(2.24)
AGE
-0.47

(0.32)
-0.99
***
(0.33)
PREFERS OUTDOORS
-7.87
kk"k
(2.47)
-3.79

(2.44)
TV/GAMES
0.72
***
(0.19)
-0.19

(0.12)
INCOME
-0.09

(0.26)
-1.04
kkk
(0.29)
CONSTANT
-15.79

(16.45)
27.91

(18.88)
Number of observations

669

853

Probability > F-statistic
0.0000

0.0001

R-squared
0.1204

0.0822

Number of clusters

149

174

3.5 Stated-Preference Survey
Table 8 reports results of random-effects probit analysis of the stated-preference data for
all respondents, children with asthma, and children without asthma. A likelihood-ratio test for
structural difference between the two subsamples is marginally insignificant (p=0.12). All
coefficients are divided by the negative of the cost coefficient to eliminate scale differences
among models. The number of summer days affected by the medication has a strong, negative,
and significant affect on indirect utility for all three samples. The direct effect of the restriction
on the number of outdoor minutes per day was insignificant and was dropped from the models.
However, the interaction between days and minutes is significant and has the correct positive
40

-------
sign in all cases. The coefficients for both DAYS and DAYS*TIME are significantly smaller for
the asthmatic sample than the non-asthmatic sample, indicating a smaller willingness of
households with asthmatic children to pay for outdoor play time.
Cost was interacted with four variables to permit heterogeneity in the marginal utility of
money and thus WTP. The income interaction has the correct positive sign, but is significant
only in the pooled model. The asthmatic sample is willing to pay significantly less if the child is
male and significantly more if the child prefers to play outside. In contrast, the non-asthmatic
sample is willing to pay more if they live in the northeast, but none of the other cost interactions
are significant.
Table 9 contains some illustrative WTP estimates for the pooled model using the worst
combination of DAYS and TIME (20 summer days with maximum outdoor time of 10 minutes
per day). Households are willing to pay an average of $73 to reduce the number of days from
20 to 12, holding the time restriction constant at 10 minutes. They are willing to pay an
additional $29 to increase outdoor time to 45 minutes per day, holding affected days constant at
12. Moving from the worst combination of DAYS and TIME to the best combination in the
experimental design (3 days and 90 minutes) is worth an average of $175. With the exception
of the difference between $150 and $175, all estimates are significantly different from each
other.
Table 8. Random Effects Probit: Dependent Variable Probability of Choice
Coefficients Scaled by -pcost (Standard Error)

Pooled

Asthmatic
Non-Asthmatic
Days
-8.740
AAA
(0.632)
-7.046
AAA
(0.705)
-11.952
*** (1.239)
Days*Time
0.070
AAA
(0.007)
0.064
AAA
(0.008)
0.086
*** (0.013)
Cost
-1.000
AAA
(0.143)
-1.000
AAA
(0.161)
-1.000
*** (0.286)
Cost*lncome
0.037
A
(0.018)
0.028

(0.022)
0.050
(0.036)
Cost*Male
-0.384
AA
0.150)
-0.175
A
(0.174)
-0.674
(0.289)
Cost*Northeast
0.488
A A
(0.154)
0.632

(0.184)
0.260
*** (0.291)
Cost*Prefers








Outside
0.477
A
(0.146)
0.339
A
(0.174)
0.612
(0.283)
Number of
observations
Log likelihood
1135
-648.08871
540
-292.73422
595
-349.6708
41

-------
Number of clusters
227
119
108
^significant at the a = 0.05 level; """significant at the a = 0.01 level; ""^significant at the a = 0.001 level
Table 9. Money-Equivalent Utility Differences
Relative to (Days=20, Time=10)
Full Sample
Total
Summer
Days
Outdoor
Minutes per
Day
WTP
90%
Confidence
Interval
3
90
$175
$146
$207
12
90
150
128
178
12
45
107
90
127
12
10
73
62
87
20
10
0
0
0
5. Future Directions
This paper outlines the research strategy and data collection efforts including a
preliminary analysis of the data. Ultimately, we will use the revealed preference (RP) data from
the activity diaries with the stated preference data from the conjoint survey to address the four
key research questions identified in the introductory section. Further analysis of the RP data in
particular will be used to measure how parents and children alter their indoor/outdoor and
related behaviors in response to ozone alerts. Second, by combining RP and SP data, we hope
to estimate parents' welfare losses from their child's exposure to high outdoor ozone levels.
Preliminary results suggest modest differences between revealed and stated preferences for
outdoor playtime between parents with a child who has asthma and parents with a child who
does not have asthma. Other household characteristics also affect outdoor play preferences.
Third, through the RP-SP analysis, we will measure the implicit costs to parents of
restricting their children's outdoor activities (independent of outdoor ozone levels). Because the
SP data allows estimating the marginal utility of money, we can estimate parents' demand for
childrens' outdoor time, at least in the context of the hypothetical commodity, and to estimate
how this demand varies systematically across households.
42

-------
A fourth component of the research project, which was not discussed in this paper,
involves a risk assessment and exposure model. The modeling effort will follow the children
through their day using a GIS-based system to capture actual exposure to ozone on the activity
diary data. The model will be used to predict the net impact of behavioral changes on health
risks from ozone exposures.
Finally, each of the components above will allow us to assess the informational benefits
associated with ozone alert system. Preliminary results suggests a relatively low level of
awareness of the system. The survey data will allow us to gauge both the awareness of and
reactions to different levels of ozone alerts and how they vary across households and urban
areas. The net benefits of the ozone alert system will include both the value of reductions in
health risks associated with these defensive activities and the lost value (opportunity cost)
associated with restricting outdoor and other activities.
43

-------
References
Adamowicz, W., J. Swait, P. Boxall, J. Louviere, and M. Williams. 1997. "Perceptions
versus Objective Measures of Environmental Quality in Combined Revealed and
Stated Preference Models of Environmental Valuation." Journal of Environmental
Economics and Management 32:65-84.
American Lung Association (ALA). 2001a. "State of the Air: 2001." New York:
American Lung Association.
American Lung Association. 2001b. "Trends in Asthma Morbidity and Mortality."
January, 
Bresnahan, B.W., M. Dickie, and S. Gerking. 1997. "Averting Behavior and Urban Air
Pollution." Land Economics 73(3):340-57.
Chiang, Jeongwen, and Lung-Fei Lee. 1992. "Discrete/Continuous Models of
Consumer Demand with Binding Nonnegativity Constraints." Journal of
Econometrics 54:79-93.
Crawford-Brown, Douglas. 1997. Theoretical and Mathematical Foundations of Human
Health Risk Analysis. Kluwer Academic Publishers.
Dickie, M., and S. Gerking. 1991. "Willingness to Pay for Ozone Control: Inferences
from the Demand for Medical Care." Journal of Environmental Economics and
Management 21 (1): 1 -16.
Farber, S., and A. Rambaldi. 1993. "Willingness to Pay for Air Quality: The Case of
Outdoor Exercise." Contemporary Policy Issues 11 (4): 19.
Friedman, M.S., K.E. Powell, L. Hutwagner, L.M. Graham, and W.G. Teague. 2001.
"Impact of Changes in Transportation and Commuting Behaviors During the 1996
Summer Olympic Games in Atlanta on Air Quality and Childhood Asthma."
Journal of the American Medical Association 285(7): 897-905.
Hanemann, W. Michael. 1884. "Discrete/Continuous Models of Consumer Demand."
Econometrica 52(3):541 -561.
Hensher, D.A., and M. Bradley. 1993. "Using Stated Response Choice Data to Enrich
Revealed Preference Discrete Choice Models." Marketing Letters 4(2): 139-151.
Jenkins, Robin, Nicole Owens, and Lanelle Bambenek Wiggins. 2000. "Valuing a
Statistical Child's Life: The Case of Bicycle Safety Helmets." Presented at the
AEA/ASSA Annual Meetings, Boston, MA, January 8, 2000.
Johnson, F.R., M.R. Banzhaf, and W.H. Desvousges. 2000. "Willingness to Pay for
Improved Respiratory and Cardiovascular Health: A Multiple-Format Stated-
Preference Approach." Health Economics 9:295-317.]
44

-------
Larson, Douglas M., and Daniel K. Lew. 2001. "Measuring Individual-Specific Shadow
Values of Leisure Activities." Paper presented at the 2001 W-133 Meetings,
Miami, February 27.
Larson, Douglas. 1993. "Joint Recreation Choices and Implied Values of Time." Land
Economics 69(3): 113-128.
Louviere, Jordan, J. 1996. "Combining Revealed and Stated Preference Data: The
Rescaling Revolution." Paper presented at the 1996 AERE Workshop, Lake
Tahoe, CA.
Morikawa, T. 1993. "Correcting State Dependence and Serial Correlation in the RP/SP
Combined Estimation Method." Transportation 21:153-165.
Nie, N., and J.P. Robinson. May 2001. "Time-diary Measurement on the Internet: An
National Experiment." Paper presented at the annual meeting of the American
Association for Public Opinion Research, Montreal.
Rowe, Robert D., and Lauraine G. Chestnut. 1985. Oxidants and Asthmatics in Los
Angeles: A Benefits Analysis. Final report prepared for U.S. EPA. Document No.
EPA/230/7-85/010.
Rowe, R.D., and L.M. Chestnut. 1986. Addendum to Oxidants and Asthmatics in Los
Angeles: A Benefits Analysis.
Schulze, William, Laurie Chestnut, Timothy Mount, Weifeng Weng, and Hong Kim.
2000. "Valuing Reduced Risk for Children." Presented at the AEA/ASSA Annual
Meetings, Boston, MA, January 8, 2000.
Shaw, W. Douglass, and J. Scott Shonkwiler. 2000. "Brand Choice and Purchase
Frequency Revisited: An Application to Recreation Behavior." American Journal
of Agricultural Economics 82(3):515-530.
Stieb, David M., Paul De Civita, F. Reed Johnson, Matthew M. Manary, and Aslam H.
Anis. 2000. "The Economic Value of Avoiding Acute Cardiorespiratory Morbidity
Associated with Air Pollution: An Integrated Approach to Benefits Transfer."
Working Paper, Healthy Environments and Consumer Safety Branch, Health
Canada. December 20.
U.S. Environmental Protection Agency. 1996. Air Quality Criteria for Ozone and
Related Photochemical Oxidants. Washington, DC.
U.S. Environmental Protection Agency. 2001. "America's Children and the
Environment: A First View of Available Measures.
.
U.S. Environmental Protection Agency (EPA). 2002. "Air Quality Guide for Ozone."
.
45

-------
von Haefen, Roger H. 2000. "'Bottom-Up' Discrete-Continuous Consumer Demand
Models with Binding Non-Negativity Constraints." Working Paper. Division of Price
and Index Number Research, U.S. Bureau of Labor Statistics. October 14.
von Haefen, Roger H., and Daniel J. Phaneuf. 2000. "A Comparison of Continuous
and Count Data Demand System Models with an Application to Seasonal
Recreation Demand." Working Paper. Department of Agricultural and Resource
Economics. North Carolina State University. December 27.
Weiss, Kevin B., Peter J. Gergen, and Thomas A. Hodgson. 1992. "An Economic
Evaluation of Asthma in the United States." New England Journal of Medicine
326:862-866.
Yoo, Seung-Hoon, and Kyung-Suk Chae. 2001. "Measuring the Economic Benefits of
the Ozone Pollution Control Policy in Seoul: Results of a Contingent Valuation
Survey." Urban Studies 38(1 ):49-60.
Zwerina, Klaus, Joel Huber, and Warren F. Kuhfeld. 1996. "A General Method for
Constructing Efficient Choice Designs." SAS Working Paper.
http://citeseer.ni.nec.eom/rd/89088587.376089.1.0.25.
	Download/http:qSqqSqftp.sas.comqSqtechsupqSqdownloadqSqtechnoteqSqts62
9.pdf.
46

-------
A Cross-Sectional Analysis of Asthma Medication Use and Air Pollution:
A Preliminary Analysis
Charles Griffiths, Nathalie B. Simon, Tracey Woodruff1
U.S. Environmental Protection Agency, National Center for Environmental Economics
Asthma is a chronic lung disease that is characterized by intermittent, recurring episodes of
wheezing, breathlessness, tightness of the chest, and coughing. These episodes are caused by
inflammation of the airways that carry air into and out of the lungs. Asthma is considered to be a
growing problem in the United States, especially among children. The prevalence of asthma
increased 46 percent between 1982 and 1993 in the United States. While increases in prevalence
have been documented in all age, race, and gender groups, the increase has been most significant
among children, individuals under the age of 18, in which prevalence has increased by a
staggering 80 percent since 1982.
While the exact causes of the illness remain unknown, asthma attacks can be triggered by
exposure to allergens (such as dust mites, pollen, mold, pet dander, and cockroach waste), strong
fumes, respiratory infections, exercise, dry or cold air, as well as air pollution (including ozone
and particulate matter). Despite recent efforts to reduce ambient levels of air pollution,
approximately 46 million people lived in counties that did not meet the air quality standards for
at least one of the six criteria pollutants in 1996. The combination of poor air quality with other
triggers is often most extreme in urban centers where a disproportionate number of minority and
low income households reside.
A relatively large number of studies exist that focus on the relationship between air pollution and
serious asthma attacks resulting in Emergency Room visits or Hospital Admissions; however,
very few studies exist that focus on mild to moderate asthma attacks. Those studies that do
examine mild forms of asthma symptoms generally are diary studies that follow a small group of
asthmatic individuals over time and focus on the effects of short-term increases in air pollution
exposure. This paper presents the preliminary results of a cross-sectional analysis of the effects
of chronic exposure to air pollution on the incidence of asthma attacks, as measured by the use
of short-term "quick relief medication.
Literature Review
The relationship between short-term increases in ambient levels of air pollution and asthma
outcomes has been documented in a number of venues using two types of studies: daily time
series studies and diary studies. Daily time-series studies are used to model the relationship
between daily levels of ambient air pollution and daily counts of a health outcome. By focusing
1 The views expressed in this paper are those of the authors and do not necessarily reflect the position of
the Environmental Protection Agency.
47

-------
on a particular city or area, these types of studies limit the amount of data required since the
population acts as its own control. For instance, there is no need to control for socio-
demographic characteristics or behavioral patterns as these are thought to remain relatively
constant in the population from one day to the next. Only weather and seasonal variation need to
be included in the model in addition to the daily air pollution levels. Diary studies on the other
hand, follow a group of individuals over time and ask that participants keep track of symptoms,
behaviors, and medication use over the study period. Studies of this type must control for
personal characteristics of the panel members, differences in behaviors, as well as weather and
pollution.
Daily time-series studies have been used to model the relationship between air pollution and a
number of health outcomes including daily mortality and other relatively severe respiratory
outcomes such as hospital admissions, emergency room visits and doctor visits - with a
relatively large segment of the studies focused on asthma. In a study by Walters et al. (1993),
for instance, daily levels of S02 and black smoke were found to have a positive association with
hospital admissions for asthma in Birmingham, UK. A similar result was found in Birmingham,
Alabama in a study focused on hospital admissions due to pneumonia and Chronic Obstructive
Pulmonary Disease (of which asthma is a component) among elderly inhabitants (Schwartz
1994). Also found was a positive association between air pollution levels and doctor visits for
asthma in London (Hajat et al. 1999). In Barcelona, Spain, a positive association between
emergency room visits for Chronic Obstructive Pulmonary Disease and air pollution levels was
found (Sunyer et al.). While these studies are indicative of the detrimental effects of short-term
increases in air pollution on rather severe asthma outcomes, they give no indication of the
chronic effects of air pollution exposure on respiratory health nor do they generally provide
evidence of detrimental effects that are milder in nature.2
Diary studies, using data collected from a panel of individuals, can provide some indication of
the effects of air pollution on less severe health outcomes. They model symptoms experienced
by panel members as a function of air pollution levels. A number of studies of this sort have
found positive and significant effects of air pollution exposure on exacerbation of asthma
symptoms. Neukirch et al. (1998) found measurable short-term effects of low-level air pollution
in Paris France on nonsmoking asthmatic adults diagnosed with mild or moderate asthma.
Similarly, Peters et al. (1996) found that asthmatic children in Erfurt and Weimar Germany and
Sokolov in the Czech Republic suffered more symptoms (cough, shortness of breath, wheezing)
and reduced pulmonary expiratory flow when exposed to higher levels of air pollution, although
same day effects were relatively weak compared to cumulative effects over 5 days. Ostro et al.
(1991) also found a strong association between daily air pollution levels (specifically airborne
acid aerosols, particulates, and sulfates) and increased asthma symptoms among a panel of
asthmatics in Denver, Colorado. Similar results have been reported in the Utah Valley (Pope et
al. 1991), Glendora California (Krupnick et al. 1990), and the Netherlands (Hiltermann et al.
2 Two exceptions to this statement are a study by Zeghnoun et al. (1999) conducted in Le Havre France
and an ongoing study by Simon et al. (2002) in San Francisco, California. Both studies look at air pollution and the
purchase of quick relief asthma medications.
48

-------
1998) among other places.
While diary studies are useful in isolating the effects of short-term increases in pollution on
milder outcomes, these studies face several difficulties. Among these difficulties, as noted by
Schwartz et al. (1991) is the fact that daily symptom rates are often highly correlated from one
day to the next and the heterogeneity among subjects causes dependencies in the data. Some
study results are also limited by the availability of particulate pollution measures - relying on
TSP data rather than PM10 data - while others are limited by panel size or length of study
period.3 Because these studies tend to have relatively short study periods (often less than 1
year), they do not generally provide any indication of the effects of chronic exposure to air
pollution on asthma symptoms.
In contrast to the studies described above, our study examines the effect of longer term or
chronic exposures to air pollution on asthma symptoms as measured by the purchase of quick
relief asthma medications across the state of California. We hypothesize that chronic exposure
to air pollution may make an individual more susceptible to asthma attacks, causing an increase
in the use of quick relief medications. Rather than consider the effects of daily increases in air
pollution levels, this study focuses on differences in average pollution levels across zip codes
and the effect of these observed differences on the purchase of quick relief asthma medications.
Methodology
This study looks at the effects of differences in long-term or chronic air pollution exposures on
the occurrence of asthma attacks, where asthma attacks are proxied by the number of
prescriptions for quick relief asthma medication. The total count of prescriptions for quick relief
asthma medication is explained using measures of asthma triggers and other cofactors. The
study utilizes a dataset of asthma drug prescriptions for a large percentage of the pharmacies in
the state of California and GIS layers of spatial factors.
In this study, our "health" outcome (filling asthma prescriptions) is not a "direct" effect of air
pollution exposure, but rather a secondary effect. That is, the true sequence of events goes as
follows: long-term exposure to air pollution makes an individual more susceptible to asthma
triggers leading to an exacerbation of asthma symptoms which in turn causes an increase in
asthma medication use. The increase in asthma medication use eventually (perhaps with a lag)
leads to the filling of a prescription. Because the urgency with which a prescription will need to
be filled will vary across individuals and their initial stock of asthma medication, making short
term effects difficult to observe, we focus on longer periods of time during which increased air
pollution should be correlated with increased prescriptions, over and above the amount necessary
for normal stock replacement.
3 TSP or Total Suspended Particulates includes all suspended particulates regardless of size. PM10, on the
other hand, is defined as those particulates measuring 10 microns in diameter or less. PM10 is considered a more
relevant measure of particulate pollution for epidemiological studies. These particulates are thought to be the most
detrimental since they can be inhaled deeply into the lung.
49

-------
Prescription data are provided for each five-digit zip code in the state and are segregated by
five-year age groups and the level of asthma severity of the patient. Asthma severity is classified
as mild intermittent, mild persistent, moderate persistent, and severe, based upon the number and
combination of prescriptions that the patient fills for both quick-relief and maintenance asthma
medicine over the 12 month calendar year (NIH, 1997). Generally, asthma medications fall into
one of two categories: (1) short-term treatments intended to provide quick relief in the event of
an asthma attack and (2) long-term maintenance therapies intended to prevent asthma attacks.
Mild asthmatics are those patients prescribed a quick-relief medication only. Patients with mild
persistent asthma not only are prescribed a quick-relief medication but are also prescribed a
single controller or maintenance therapy. Moderate asthmatics are prescribed two controllers
operating by different modes of action in addition to the quick relief medications, while severe
asthmatics are prescribed three controllers with different modes of action. Should an individual's
asthma severity level shift over the 12 month period, the individual is assigned to the most
severe of the categories for which he/she qualifies. A list of the quick acting and controlling
asthma medication is listed in Table 1.
Asthma triggers included in the study include air pollutants (e.g., particulate matter and ozone),
which are the primary factors of concern, as well as temperature. Additional cofactors included
are population demographics (e.g., median household income, percent urban population), and
seasonal or quarterly dummies. The inclusion of other spatial factors such as pollen and the road
network were considered, but not included in this model.
Data Description
The number of prescriptions for quick acting asthma medication was obtained from NDChealth
(hereafter, NDC), a Phoenix-based company that maintains prescription-related data for
marketing research. NDC maintains two datasets of use for this study, a "retail pharmacy"
database and a "patient" database. The pharmacy database contains dispensing records from
approximately 36,000 pharmacies nationwide, and captures approximately 70% of the volume of
traditional pharmacy-dispensed prescriptions. Hospital, military and mail order pharmacies and
prescriptions dispensed to institutionalized patients are not included in this database, which may
pose a problem in the future as mail order prescriptions grow, but is probably not important here.
The patient database is a subset of approximately 14,000 of the pharmacies in the pharmacy
database. The patient database is a more complete database, in many cases including the patients
age and gender, along with a unique patient identifier so that the history of a patient may be
followed. Not included in the database, and unknown to NDC, is any information that could
personally identify a patient (such as a name, address or phone number) and NDC has been very
careful not to release any individual patient data, even with the anonymous identifier.
For this study, the total counts of the number of prescriptions for quick-acting asthma medication
in a five digit zip code for each quarter from 1998 to 2001 were used. Data are given by
dispense quarter and the zip code of the dispensing pharmacy. These data are further
disaggregated by the age of the patient, with age groups defined as: ages 0 to 4, ages 5 to 9, ages
10-14, ages 15-17, ages 18-44, ages 45-64, ages 65 and up, and age unknown as well as asthma
50

-------
severity.
The prescription data used in this analysis are limited in the following way. They only include
counts of prescriptions for quick relief asthma medication from those pharmacies that
"consistently" report this information. "Consistent" reporting is defined by NDC as pharmacies
for which fewer than 11 days of data are missing in any 30 day period. While the number of
consistently reporting pharmacies remains relatively stabile in a zip code over time, the number
of pharmacies reporting across zip codes varies widely and may affect the number of
prescriptions dispensed for quick relief asthma prescriptions.
To control for the number of pharmacies reporting while maintaining the privacy of the
pharmacies themselves, NDC provided us with a proxy measure that would be strongly, if not
identically, correlated with number of pharmacies in each zip code reporting asthma
prescriptions. Specifically, they calculated the ratio of the number of pharmacies reporting
asthma prescriptions to the number of pharmacies reporting prescriptions for either antibiotics or
pain relief medications during the same time period. This ratio allows us to identify and control
for fluctuations in asthma prescriptions attributable to variations in the number of pharmacies
reporting rather than those that are attributable to changes in weather, air pollution or other
factors.
The air pollution data come from the California Air Resource Board and are made publicly
available. Daily observations on the levels of PM10, S02, NOx, and ozone are available for 361
monitors across California. PM10 (in micrograms per cubic meter) and the 8 hour maximum
value of ozone (in parts per million) were available with good spatial coverage across California.
The 24 hour average value of S02 (in parts per million) and the daily average concentration of
NOx (in parts per million) were available for a subset of the sites. The daily observations for all
of the pollution measures were averaged over the quarter for each monitor.
The weather data come from the National Climatic Data Center. Daily observations for the
average, minimum, and maximum temperature, precipitation, as well as the dew point
temperature, and the minimum and maximum relative humidity were obtained for 37 active
weather stations across California. The dew point temperature and relative humidity measures
were eventually dropped due to a lack of adequate spatial distribution. Since it is generally
believed that cold weather events are correlated with asthma attacks, the average minimum
temperature over the quarter was used in this analysis.
While the coverage of air pollution and weather data offer an acceptable representation of the
state, each zip code does not necessarily contain an air pollution or weather monitor. An
algorithm was needed to link the zip code with two disparate points: the zip code and the air
pollution monitor or the weather station. Kriging methods to spatially interpolate the data were
explored, but given the preliminary nature of this analysis, the simpler method of linking each of
the zip codes with the nearest monitor and station that contained data within 25 miles was used.
Zip codes were linked to monitor and stations for each of the four years individually, so a zip
code could potentially draw data from more than one location over the course of the study. To
51

-------
be considered in a year, a pollution monitor was required to have PM10 data and a weather
station was required to have precipitation data in the first quarter. Of potential concern is that
zip codes were linked to monitors without regard for airshed, elevation, or wind direction, and
spatial autocorrelation and spatial heterogeneity were not evaluated in this study. Future
analyses will try to incorporate these elements as well.
Demographic data for each zip code were obtained from the 2000 U.S. Census. Total population
counts by age and race, as well as other demographic data, were collected at the zip code level
from both the SF1 (100-percent, short form) and SF3 (sample, long form) datasets. Ultimately,
we decided to control for population by using prescriptions per capita as the left hand side
variable; however, we included various characteristics of the population as explanatory
variables, including the percent of the population in each race, population density, percent urban,
and median income.
The summary statistics for the data used in this analysis are listed in Table 2. The unit of
observation is the five-digit zip code. For the sixteen quarters from 1998 to 2001, data were
available at one point or another for 852 of the 1919 zip codes in California. Together, 7,735
observations of quarterly counts for quick relief medications were available. When linked with
the regressors, however, between 7639 and 7097 observations were available for primary
analysis, and 3284 observations were available if S02 and NOx were included.
Empirical Results
Since our prescription data were reported by five-digit zip codes of various sizes and population
density, we control for this variation by normalizing the prescription counts by the size of the
total population of the zip code using information from the 2000 Census. The effects of cold
temperature extremes are captured using the average minimum temperature for the zip code over
the quarter, and cyclical variation and seasonal allergies are controlled for using quarterly
dummies. Demographics included here are race and median household income. Since we are
explaining prescriptions per capita, the percentages of these demographic categories in each zip
code are used. In addition, we have included both population density and percent of population
in an urban area as explanatory variable, as well as an interaction between percent urban and
median income. Finally, we have included a trend variable to control for annual changes that are
not otherwise captured.
Since counts of quick relief asthma medications are relatively large for each quarter at the zip
code level, we examine the effects of pollution using simple, weighted OLS regressions, where
the weight is the ratio of the number of pharmacies reporting in each zip code during the quarter
to the number of pharmacies reporting in the alternative market.4 We built the model by first
incorporating time and weather variables. In this case, the weather variable of interest is
minimum temperature, as cold temperatures are thought to exacerbate asthma symptoms. We
then added population density and demographic characteristics at the zip code level. Finally, we
4The alternative market is defined as prescriptions for analgesics and/or antibiotics.
52

-------
added pollution variables to our models. PM10 and ozone measures were introduced separately
in the analysis before including them in the same regression. We then added S02 and NOx to
the final regression to see the effect. The results of the final regressions including the pollution
measures are reported in Table 3.
Focusing first on the demographic variables, household income is positive and statistically
significant in all of our models, indicating that households with higher incomes are more likely
able to afford the prescriptions. Race also seems to matter, with Hispanics showing a greater
likelihood of purchasing asthma prescriptions in California. On the other hand, being Asian or
black has a negative effect on asthma prescriptions for quick relief medications. We had no
predispositions as to the direction of the coefficient on the race variables, recognizing only that
race could be a significant determinant of exposure to triggers and susceptibility to them. It
could be that Hispanics live in areas with higher levels of pollution or are otherwise exposed
more often to other asthma triggers compared to other segments of the population. Or, perhaps
differences in the occupations held by individuals comprising the various race/ethnic groups
exist that lead to differential exposure to asthma triggers. While population density has a
consistently positive and statistically significant coefficient, the coefficient on percent of the
population living in an urban area tends to be negative. We initially expected that urbanization
would have a positive effect on asthma prescriptions since exposure to asthma triggers is often
thought to be higher in urban environments.
Turning to our variables of interest, we find mixed results. Interestingly, PM10 has a
consistently positive and statistically significant effect on asthma prescriptions for quick relief
medications, with the exception of model 4 in which S02 and NOx are included as well as
ozone. Generally this means that higher levels of PM10 in one location are associated with a
greater number of total prescriptions per capita for quick relief asthma medications, all else
equal. In model 4, however, the effects of S02 and NOx dampens the effect of PM10
considerably. S02 and PM10 tend to be correlated, however, with S02 a potential indicator of
the acidity of the particulate pollution. Some argue that the acidity of the particulates contributes
to the incidence of various health effects including asthma. Because of the relatively poor
coverage offered by monitors reporting S02 and NOx, it is important to note that the number of
observations declined considerably compared to the other models reported here.
The effects of ozone on quarterly prescription counts of quick relief medications is not nearly as
pronounced. In fact, we find no statistically significant effect of ozone measures in any of our
models. Model 4 again provides a weak exception, with a barely statistically significant (at the
90 % level) coefficient on the ozone measure included in the regression. The sign of the ozone
coefficient in Model 4 is puzzling, though, as it is negative.
Effects by Asthma Severity
Recognizing that maintenance therapies could be dampening the effects of air pollution on
quick-relief asthma medication use and prescriptions, we stratified our data according to asthma
severity. Using counts of prescriptions per capita for each severity level as the dependent
variable, we ran four separate regressions using model 3 from Table 3. These results are
53

-------
reported in Table 4.
As suspected we see differential responses to air pollution levels by asthma severity. As in
Table 3, PM10 has a consistently positive and statistically significant effect on asthma
prescriptions for quick relief medications regardless of severity level. The magnitude of the
effect does vary, however, with mild and severe asthmatics showing the largest response. This is
not entirely surprising since mild asthmatics by definition do not take controller medications but
rely only on the quick relief medications to ease their breathing. On the other extreme, severe
asthmatics, while taking several maintenance therapies, may be more susceptible to exposure to
asthma triggers including air pollution levels, requiring larger numbers of prescriptions for quick
relief medications.
The effect of ozone levels on asthma prescriptions remains puzzling. When this variable is
statistically significant, it has a negative sign- the opposite of what we were expecting. It may
be that the effects of ozone exposure on asthma are more acute requiring daily time series
models to capture. This is a subject of future investigation.
While the direction of the effects of our other explanatory variables remain relatively unchanged
from our core model reported in Table 3, some of the differences in magnitude by severity level
are quite interesting. For instance, median household income has a statistically significant and
positive effect on asthma prescriptions across the board, but the magnitude of the effect is larger
for mild and severe asthmatics. One explanation of this effect could be cost related in that
households with lower incomes may choose to forgo prescriptions for mild asthmatics. For
asthmatics with more serious, persistent symptoms, the income effect is less pronounced,
indicating perhaps a willingness to purchase the prescriptions to alleviate these more intense
symptoms in spite of their cost. For the severe asthmatics, the effect of median household
income is again quite pronounced - approximately twice the size of the effect for the mild
asthmatic. This may again reflect cost concerns in that the multiple medications prescribed to
the severe asthmatic may result in a substantial expense. Instead, households with lower
incomes may forgo the additional treatments, continuing instead to purchase the medication
combinations prescribed to the moderate asthmatic.
Age Specific Effects
Given the dramatic rise in asthma among children, it is important to determine whether or not the
effects described above are age-specific. Including age-specific cofactors (such as the
percentage of specific age groups in each zip code) in the regression above was considered, but
using the prescriptions by age group in separate regressions gives a much more complete picture.
The difficulty in this disaggregation, however, is that while few zip codes have zero
prescriptions in the aggregate, a zip code may report zero prescriptions for a given age group in a
particular quarter. As can be seen by the number of observations equal to zero reported in Table
2, zero counts are a concern, particularly for the severe asthma category. This makes the linear
model used in Table 4 inappropriate.
The standard model for count data with zero observations is a Poisson model. The results of this
54

-------
model for the children's age categories of 0-4, 5-9, 10-14, and 15-17 are reported in Tables 5,
6,7, and 8 respectively. Table 9 is the Poisson regression of the remaining prescriptions,
included for completeness. Note that Table 9 includes the prescriptions for the adult category of
age 18 and above, but also all of the prescriptions listed for those of "unknown" age. Since the
dependant variable in the Poisson model is simply counts of prescriptions, we include population
and land area in the model as explanatory variables and remove population density. Otherwise,
all of the other explanatory variables used previously are included in the Poisson model as well.
In general, the Poisson models yield significant relationships, of the same form as in Table 4,
between air pollution and the number of asthma prescriptions. Strikingly, this is true, with few
exceptions across all age groups, for both PM10 and ozone. Of particular interest is the result
found by comparing the magnitude of the first order term for air pollutants across age groups for
any given severity level. When statistically significant, coefficients are generally larger for
children than for adults. Exceptions to this result for ozone are the 0-4 age group with mild
asthma and the 15-17 age group with severe asthma. The exception for PM10 is the 10-14 age
group with severe asthma. Additionally, this result appears to hold across severity classes.
Although we initially stratified our sample by severity level due to concerns that maintenance
drugs could dampen the impact of air pollution on the use of quick relief medications, this does
not appear to be the case. The implication of these general results is that children are more
highly affected by air pollution than adults. To more thoroughly test this hypothesis, however,
we would need to formally test if the coefficients are statistically different from one another
across models.
A potential alternative explanation for higher coefficients for children is that child asthmatics are
more quick to be medicated than adults. Because parents are making, or at the very least
assisting in, the decision to go to the doctor and fill prescriptions, there is a concern that parental
altruism could lead to higher rates of medication for children than for adults. That is, parents
could be more concerned about providing their children relief from asthma symptoms than adults
are for their own symptoms. We do not believe that our results provide evidence of this
altruistic effect however since the coefficients on the 10-14 and 15-17 age groups are still higher
than those for the adult age group. Since these teenaged groups are less reliant upon their
parents to make their decisions, we would expect to see a drop off in magnitude for these groups
if parents were in fact over-providing for their children. The absence of this decline leads us to
believe that the alternative explanation is not the primary driver of our results.
As before, minimum temperature is not significant but holds the expected negative sign for most
models. Population and land area are unsurprisingly positive and significant, but of a relatively
small magnitude in effect. The sign of the coefficient for the race percentages are often reversed
from Table 4, which will require further analysis.
One interesting result from these models is the reversal in sign for percent urban, median
household income, and their interaction. Median income was positive in Table 4, which can be
explained as the ability of wealthier households to afford prescriptions; but this is not the only
story that can be told with income. Wealthier households also have the ability to pay to avert the
asthma triggers, which would suggest a negative coefficient for income. The fact that the
55

-------
number of prescriptions for children declines with income suggests that wealthier parents are
better able to avoid the asthma triggers for their children. Further study is clearly warranted.
Conclusion
With the growing concern about increasing asthma rates, studies that further our understanding
of the causes of asthma exacerbation are timely. If, as our study shows, chronic exposure to
higher levels of air pollution leads to increases in asthma symptoms and the use of asthma
medication, then reductions in these air pollutants will produce benefits that have previously
been difficult to quantify. The benefits of reducing serious asthma attacks can be analyzed by
examining emergency room visits and hospital admissions. The benefits associated with a
decline in the outcomes analyzed here, the reduced use of quick acting asthma medication, have
been somewhat more elusive as they are not as easily observable as ER visits. In contrast to
diary studies, which examine the effect of short-term exposure to air pollution, this study looks
at the effect of longer term or chronic exposures to air pollution on asthma symptoms by
examining prescription data at the zip code level for California.
The results of Tables 3 and 4 show a statistically significant positive association between total
prescriptions per capita for quick-acting asthma medication and air pollution. Including
measures for both ozone and PM10, and controlling for temperature and demographics, we find
that PM10 is a more important driver in explaining the increase in prescriptions per capita than
ozone.
In the Poisson model of prescription counts presented in Tables 5-9, however, both pollution
measures are significant. Disaggregation by age class suggests that children are affected more
by air pollution than adults, and this effect appears to be true across severity levels. Additional
tests of statistical significance will be required in the future to be more certain of this effect.
This preliminary analysis shows that there are real consequences to long term exposure to air
pollution. We would, however, like to refine our approach in a number of ways. First, the data
used here only include pharmacies that report their prescriptions consistently, with no less than
eleven days of missing data per month. Adding the additional "inconsistent" pharmacies will
increase the number of zip codes analyzed and may reveal important interactions, but must be
done carefully to control for the additional noise. Second, there are additional variables that we
would like to consider including, such as PM2.5 and the number of days in which a zip code was
above a chosen pollution threshold. Third, there are a number of spatial issues that we should
address. Kriging techniques will eliminate the need to link zip codes to specific pollution
monitors and weather stations. We should also consider spatial factors that are currently
excluded, such as the north to south mountain line in California and the spatial impact of
CSMAs. The model may suffer from both heteroskedasticity and spatial autocorrelation, and
further analysis must be done. Finally, this data could be combined with cost information to get
an estimate of the benefits. All of these factors are expected to improve the analysis and the
usefulness of the results.
56

-------
Table 1: Asthma Medication
Symptomatic Therapy (Quick Relief)
Albuterol
Bitolterol
Isoetharine
Metaproteronol
Pirbuterol
Terbutaline
Controller Therapy (Long-term preventative')
Inhaled Corticosteroids
Beclomethasone
Budesonide
Flunisolide
Fluticasone
Triamcinolone
Leukotriene Antagonists
Motelukast
Zafirlukast
Zileutin
Long Acting Beta Agonists
Salmeterol
Xanthine Derivatives
Aminophylline
Dyphylline
Oxtriphylline
Theophylline
57

-------
Table 2: Summary Statistics

All Ages
Ages 0 to 4
Variable
Total
Asthma Severity
Total
Asthma Severity

Prescriptions
Mild
Mild
Persistent
Moderate
Severe
Prescriptions
Mild
Mild
Persistent
Moderate
Severe
Vumber of
Observations
7735
7735
7735
7735
7735
7413
7413
7413
7413
7413
Num. of Obs. = 0
0
1
5
11
80
0
117
753
3083
5851
nean
654.6344
315.155
194.381
96.87964
48.21875
32.22474
20.36234
8.872791
2.404829
0.5847835
standard error
478.487
238.5405
141.9122
72.01196
40.0092
33.54975
22.52427
9.897485
3.666297
1.57180i
nedian
540
255
161
80
38
22
13
6
1
L
nin
1
0
0
0
0
1
0
0
0
L
nax
3477
1819
975
509
297
416
300
115
46
2t
skewness
1.280852
1.385644
1.242319
1.280196
1.498094
2.816224
3.161856
2.517794
3.026762
4.490084
'mrtosis
5.031391
5.450395
4.903892
5.000356
6.157983
17.43484
21.43579
13.43679
18.81439
34.22081
Table 2 (continued): Summary Statistics

Ages 5 to 9
Ages 10 to 1 4
Statistic
Total
Asthma Severity
Total
Asthma Severity

Prescriptions
Mild
Mild
Persistent
Moderate
Severe
Prescriptions
Mild
Mild
Persistent
Moderate
Severe
Number of
Observations
7500
7500
7500
7500
7500
7526
7526
7526
7526
7526
Num. of Obs. = 0
0
93
397
1647
4013
0
72
391
1420
315C
nean
46.70533
25.71613
14.0568
5.108667
1.823733
54.44592
29.49043
15.52897
6.3941
3.032421
standard error
42.98077
24.56539
14.15905
5.935202
3.128466
47.55257
25.87634
14.98555
7.142006
4.45215^
nedian
35
19
10
3

42
22
11

1
nin
1
0
0
0
0
1
0
0

L
nax
354
217
146
51
31
391
192
146
66
4;
skewness
1.929676
2.076474
2.198623
2.095364
2.842234
1.760224
1.734572
1.952528
2.047073
2.57068
'¦mrtosis
8.289729
9.382513
10.80206
9.473676
14.19708
7.588125
7.232487
9.045644
9.842268
13.16101
58

-------
Table 2 (continued): Summary Statistics
Statistic
Ages 15 to 17
All Other Ages (Not including Unknown Age)
Total
Prescriptions
Asthma Severity
Total
Prescriptions
Asthma Severity
Mild
Mild
Persistent
Moderate
Severe
Mild
Mild
Persistent
Moderate
Severe
Vumber of
Observations
7372
7372
7372
7372
7372
7203
7203
7203
7203
7201
Num. of Obs. = 0
0
150
940
2885
4968
0
0
0
5
51
nean
24.94886
14.64162
6.619506
2.587222
1.100515
525.0769
238.6608
157.6731
84.77398
43.96904
standard error
21.68575
12.91774
6.794598
3.439279
2.216614
360.2993
168.8212
109.4798
59.77791
34.86793
nedian
19
11
5
1

437
196
132
71
3t
nin
1
0
0
0
0
13
5
2
0
L
nax
181
105
74
37
18
2631
1344
847
426
259
skewness
1.757481
1.653192
2.08444
2.202667
2.853334
1.241226
1.331471
1.253321
1.225283
1.443805
'mrtosis
7.937582
6.829426
10.7228
11.39897
12.98792
4.827885
5.198832
4.973804
4.725747
6.05052;
Table 2 (continued): Summary Statistics

Air Pollution Measures (Averaged over quarter)
Minimum
Daily
Temperature
(Averaged
over
Quarter
Demographic Characteristics (Zip Code Level)
PM10, Daily
Average
Ozone
(8-hour
Maximum)
NOx, Daily
Average
SO2, Daily
Average
Median
Household
Income
Percent of
Population
Described as
Black
Percent of
Population
Described as
Asian
Percent of
Population
Described
as Hispanic
Percent of
Population
in Urban
Areas
Number of
Observations
7674
7686
5760
4094
7735
7735
7735
7735
7735
7735
nean
34.31513
0.0392582
0.0623078
0.0025247
71.45207
50861.48
0.0643355
0.1192618
0.7050654
0.9721459
standard error
13.34303
0.0153724
0.0435055
0.0015707
1090.728
20244.88
0.0984705
0.1125274
0.2311541
0.0957774
nedian
32.86667
0.0368043
0.0510809
0.00242
52.22826
47573
0.0301147
0.0796252
0.7743347
1
nin
6
0.00924
0.0024422
0.0000247
25.97826
8855
0.0014281
0.0011747
0.0277646
C
nax
117.3297
0.111
0.233938
0.0096099
72844.09
164479
0.7851492
0.5902053
0.9782452
1
skewness
1.022263
1.008934
1.195364
0.6086475
62.44114
1.296063
3.664492
1.79449
-1.014786
-6.577531
'¦mrtosis
4.668436
4.238362
4.035975
3.138443
3933.274
5.903643
19.6607
6.317696
3.160587
58.08569
59

-------
Table 2 (continued): Summary Statistics

Population in Each Age (Zip Code Level)
Geographic Characteristics
Total
Ages 0-4
Ages 5-9
Ages 10-14
Ages 15-17
Other Ages
arealand
popden
Vumber of Observations
7735
7735
7735
7735
7735
7735
7735
7688
nean
36925.78
2708.643
2921.787
2690.622
1528.824
27075.9
1.08e+08
0.002573
standard error
18392.4
1884.077
2033.976
1764.129
986.8849
12531.04
3.14e+08
0.0031668
nedian
33520
2280
2425
2297
1344
25492
1.80e+07
0.0020154
nin
294
9
9
7
20
179
0
1.33e-0t
nax
105275
11955
12546
10151
5505
70049
3.68e+09
0.0420212
skewness
0.7853757
1.315397
1.309443
1.125955
1.049795
0.6008423
6.519869
5.466631
'curtosis
3.69066
5.062044
5.117194
4.533889
4.25487
3.245719
57.26882
56.7533/

-------
Table 3: Weighted OLS Regressions of Total Prescriptions per Capita
Weight=Number of Pharmacies Reporting
Variable
Model 1
Model 2
Model 3
Model 4
Ozone (8 hour max)
0.0130
(0.0722)
--
-0.1075
(0.0727)
-0.1674*
(0.0924)
Ozone2 (8 hour max)
-0.2387
(0.6934)
—
0.0622
(0.6989)
-0.2853
(0.8221)
Particulate Matter (<10u)
—
0.0006**
(0.0001)
0.0007**
(0.0001)
0.0001
(0.0001)
Particulate Matter2 (<10u)
—
-6.22xl0"6**
(8.36xl0~7)
-6.05xl0"6**
(8.51xl0~7)
-1.76xl0"6
(1.36xl06)
SO2
—
—
—
0.8224**
(0.2113)
NOx
—
—
—
0.0534**
(0.0140)
Minimum Temperature
-2.98xl0~7
(2.67xl0~7)
-2.71xl0"7
(2.65xl0~7)
-2.46xl0"7
(2.65xl0~7)
-3.30xl0"7
(2.40xl07)
Population Density
0.2303**
(0.0943)
0.1574*
(0.0916)
0.0640
(0.0946)
-0.1040
(0.1901)
Percent Urban
-0.0546**
(0.0086)
-0.0584**
(0.0084)
-0.0584**
(0.0085)
-0.6496**
(0.0417)
Median Household Income
l.llxlO"6**
(1.78xl0~7)
l.lOxlO"6**
(1.75xl0~7)
1.12xl0"6**
(1.76xl0~7)
8.51xl0"6**
(8.17xl07)
%Urban*Median HH Income
-1.19xl0"6**
(1.82xl0~7)
-1.15xl0"6**
(1.80xl0~7)
-1.19xl0"6**
(1.81xl0~7)
-8.61xl0"6**
(8.29xl07)
Percent Black
-0.0066**
(0.0026)
-0.0020
(0.0026)
-0.0033
(0.0026)
0.0236**
(0.0035)
Percent Asian
-0.0052**
(0.0022)
-0.0066**
(0.0021)
-0.0081**
(0.0022)
0.0030*
(0.0027)
Percent Hispanic
0.0234**
(0.0014)
0.0256**
(0.0014)
0.0260**
(0.0014)
0.0155**
(0.0018)
Quarter 2 (Apr, May, June)
-0.0022**
(0.0008)
-0.0021**
(0.0006)
-0.0003
(0.0008)
0.0042**
(0.0013)
Quarter 3 (July, Aug, Sep)
-0.0046**
(0.0008)
-0.0051**
(0.0006)
-0.0034**
(0.0008)
0.0015
(0.0013)
Quarter 4 (Oct, Nov, Dec)
0.0003
(0.0007)
-0.0011
(0.0007)
-0.0017**
(0.0007)
-0.0017*
(0.0010)
Trend
0.0002**
(0.0001)
0.0002**
(0.0001)
0.0002**
(0.0001)
0.0003**
(0.0001)
Constant
0.0646**
CO 00851
0.0532**
CO 00841
0.0559**
CO 00841
0.6510**
CO 04141
Number of Observations
7639
7628
7579
3284
R2
0 7413
0 7519
0 7534
0 5149
61

-------
Table 4: Weighted OLS Regressions of Prescriptions per Capita by Asthma Severity Level
Weight=Number of Pharmacies Reporting
Variable
Mild
Mild
Persistent
Moderate
Persistent
Severe
Ozone (8 hour max)
0.0076
-0.0615**
-0.0352
-0.0183**

(0.0343)
(0.0230)
(0.0123)
(0.0063)
Ozone2 (8 hour max)
-0.4843
0.2733
0.1807
0.0925

(0.3301)
(0.2210)
(0.1186)
(0.0604)
Particulate Matter (<10u)
0.0004**
0.0002**
0.0001**
0.00005**

(0.00003)
(0.00002)
(0.00001)
(6.30xl0~6)
Particulate Matter2 (<10u)
-3.24xl0"6**
-1.53xl0"6**
-8.42xl0"7**
-4.37xl0"7**

(4.02xl0~7)
(2.69xl0~7)
(1.44xl0~7)
(7.36x10 s)
Minimum Temperature
-1.04xl0~7
-9.25xl0"8
-2.73xl0"8
-2.18x10 s

(1.25x10-7)
(8.38x10 s)
(4.50x10 s)
(2.29x10 s)
Population Density
0.0616
(0.0447)
0.0176
(0.0299)
0.0032
(0.0161)
-0.0185**
(0.0081)
Percent Urban
-0.0270**
-0.0221**
-0.0063**
-0.0030**

(0.0040)
(0.0027)
(0.0014)
(0.0007)
Median Household Income
4.84xl0~7**
2.65xl0"7**
2.85xl0"7**
8.92x10 s

(8.33x10 s)
(5.57xl0"s)
(2.99x10 s)
(1.52x10 s)
%Urban*Median HH Income
-5.08xl0"7**
-2.80xl0"7**
-3.01xl0"7**
-1.02xl0"7**

(8.53x10 s)
(5.71xl0~6)
(3.07xl0"s)
(1.56x10 s)
Percent Black
-5.32xl0"6
-0.0007
-0.0017**
-0.0009**

(0.0012)
(0.0008)
(0.0004)
(0.0002)
Percent Asian
-0.0043**
-0.0018**
0.0009**
-0.0010

(0.0010)
(0.0007)
(0.0004)
(0.0002)
Percent Hispanic
0.0110**
0.0075**
0.0049**
0.0025**

(0.0007)
(0.0004)
(0.0002)
(0.0001)
Quarter 2 (Apr, May, June)
-0.0013**
(0.0004)
0.0006**
(0.0003)
0.0003**
(0.0001)
0.0001*
(0.0001)
Quarter 3 (July, Aug, Sep)
-0.0032**
(0.0004)
-0.0001
(0.0002)
-0.0001
(0.0001)
-0.00002
(0.0001)
Quarter 4 (Oct, Nov, Dec)
-0.0010**
0.0001
-0.0004**
-0.0003**

(0.0003)
(0.0002)
(0.0001)
(0.0001)
Trend
0.0001**
-0.00001
0.00004**
0.00003**

(0.00002)
(0.00002)
(8.61xl0~6)
(4.44xl0~6)
Constant
0.0244**
0.0226**
0.0058**
0.0031**

CO 00401
CO 00771
CO 00141
CO 00071
Number of Observations
7579
7579
7579
7579
R2
0 7398
0 7357
0 7757
0 7061
62

-------
Table 5: Poisson Regressions of Prescriptions Counts by Asthma Severity, Age 0-4
Weight=Number of Pharmacies Reporting
Variable
Mild
Mild
Persistent
Moderate
Persistent
Severe
Ozone (8 hour max)
10.97834**
20.54198**
14.51552**
-5.556807

(0.7579566)
(1.095515)
(2.160324)
(4.617046)
Ozone2 (8 hour max)
-124.1338**
-140.9479**
-146.5154**
4.105722

(7.629936)
(10.66574)
(21.7495)
(46.94952)
Particulate Matter (<10u)
0.0254418**
0.0146184**
0.0164327**
0.0357992**

(0.0008049)
(0.0011707)
(0.0024019)
(0.0051865)
Particulate Matter2 (<10u)
-0.0001945**
-0.0001164**
-0.000122**
-0.0003103**

(0.0000092)
(0.0000133)
(0.0000275)
(0.0000605)
Minimum Temperature
-0.00000031
-0.000012
-0.007008**
-0.0120306**

(0.0000042)
(0.00000794)
(0.0021487)
(0.0044281)
Population
0.0000139**
0.0000146**
0.0000115**
0.00000283**

(0.000000143)
(0.000000221)
(0.000000425)
(0.000000902)
Land Area
5.60e-ll**
5.2 le-11 * *
2.20e-10**
2.21e-10**

(9.26e-12)
(1.35e-ll)
(2.35e-l 1)
(4.64e-l 1)
Percent Urban
0.3792007**
-0.5283571**
-0.0521455
1.496921**

(0.1462972)
(0.1878531)
(0.4021566)
(0.5826956)
Median Household Income
-0.0000191**
-0.0000244**
-0.0000339**
0.0000285**

(0.00000318)
(0.00000427)
(0.00000869)
(0.0000119)
%Urban*Median HH Income
0.0000233**
0.0000262**
0.0000431**
-0.0000283**

(0.00000322)
(0.00000433)
(0.00000879)
(0.0000122)
Percent Black
1.763746**
1.344907**
1.45492**
0.9173831**

(0.0231235)
(0.0377671)
(0.0712859)
(0.1493707)
Percent Asian
-0.0656788**
0.179164**
0.4977336**
0.6452012**

(0.022915)
(0.0338249)
(0.0618611)
(0.1255217)
Percent Hispanic
-0.442947**
0.0482735**
-0.1543095**
-0.1006465

(0.015432)
(0.0237485)
(0.0457842)
(0.0935472)
Quarter 2 (Apr, May, June)
-0.4872949**
-0.4346739**
-0.2642642**
-0.1460141**

(0.0084444)
(0.0127981)
(0.0282335)
(0.0594354)
Quarter 3 (July, Aug, Sep)
-0.7119725**
-0.6088005**
-0.4013929**
-0.3089985**

(0.008591)
(0.0129702)
(0.0367868)
(0.0765735)
Quarter 4 (Oct, Nov, Dec)
-0.2182028**
-0.0244281**
-0.2728652**
-0.5376679**

(0.0065429)
(0.0101834)
(0.0202581)
(0.0425356)
Trend
0.0348096**
0.0461616**
0.1054164**
0.1559188**

(0.0005428)
(0.0008227)
(0.0016332)
(0.003684)
Constant
1.441439**
1.004536**
-0.9154803**
-3.354994**

(0.1452816)
(0.1871769)
(0.4033416)
(0.5953013)
Number of Observations
7305
7305
7305
7305
Pseudo R2
0.27
0.1719
0.1429
0.1098
63

-------
Table 6: Poisson Regressions of Prescriptions Counts by Asthma Severity, Age 5-9
Weight=Number of Pharmacies Reporting
Variable
Mild
Mild
Persistent
Moderate
Persistent
Severe
Ozone (8 hour max)
25.04567**
29.17234**
26.42362**
23.88336**

(0.6661456)
(0.8986673)
(1.466198)
(2.410876)
Ozone2 (8 hour max)
-236.7028**
-233.8192**
-216.934**
-177.3323**

(6.636413)
(8.818661)
(14.37449)
(23.72769)
Particulate Matter (<10u)
0.0261766**
0.0193574**
0.0166144**
-0.0002486

(0.0007097)
(0.0009517)
(0.0015273)
(0.0024129)
Particulate Matter2 (<10u)
-0.0002214**
-0.0001766**
-0.0001489**
-0.0000073

(0.00000802)
(0.0000108)
(0.0000174)
(0.0000278)
Minimum Temperature
-0.0000103**
-0.0000119**
-0.00000757
0.0000017

(0.00000442)
(0.00000592)
(0.00000871)
(0.000011)
Population
0.0000161**
0.0000138**
0.0000151**
0.000015**

(0.000000126)
(0.000000172)
(0.000000287)
(0.000000483)
Land Area
1.01e-10**
2.33e-l 1**
1.30e-10**
1.75e-10**

(7.67e-12)
(1.07e-ll)
(1.61e-ll)
(2.60e-ll)
Percent Urban
0.503357**
-0.1344909
-0.6599364**
0.718834**

(0.1178582)
(0.1502643)
(0.2223536)
(0.3413284)
Median Household Income
-0.00000436*
-0.0000151**
-0.0000186**
0.0000104

(0.00000251)
(0.00000338)
(0.00000511)
(0.00000712)
%Urban* Median HH Income
0.00000853**
0.0000156**
0.0000191**
-0.00000989

(0.00000255)
(0.00000343)
(0.00000519)
(0.00000726)
Percent Black
1.680733**
1.8477**
1.266928**
1.224149**

(0.0210708)
(0.0276487)
(0.0496734)
(0.0838741)
Percent Asian
-0.0508744**
-0.0538819*
0.3131667**
0.7048495**

(0.0202658)
(0.0277586)
(0.043206)
(0.0682401)
Percent Hispanic
-0.2934698**
-0.1937725**
0.1950645**
0.7475832**

(0.0137165)
(0.0185691)
(0.0310077)
(0.0530349)
Quarter 2 (Apr, May, June)
-0.2438594**
-0.2571905**
-0.223105**
-0.2601726**

(0.0075468)
(0.0102463)
(0.0166626)
(0.0265451)
Quarter 3 (July, Aug, Sep)
-0.3288345**
-0.3087052**
-0.3110525**
-0.4230715**

(0.0075107)
(0.010158)
(0.0164871)
(0.0263215)
Quarter 4 (Oct, Nov, Dec)
0.147668**
0.1694812**
0.0465003**
-0.1586523**

(0.0060709)
(0.0083697)
(0.0137708)
(0.0226232)
Trend
0.0267533**
0.0317252**
0.0593349**
0.0939584**

(0.0004748)
(0.0006429)
(0.0010717)
(0.001806)
Constant
0.8511665**
1.038824**
0.1434398
-2.493021**

(0.1171189)
(0.1497897)
(0.2223432)
(0.3411868)
Number of Observations
7392
7392
7392
7392
Pseudo R2
0.267
0.2003
0.1302
0.1039
64

-------
Table 7: Poisson Regressions of Prescriptions Counts by Asthma Severity, Age 10-14
Weight=Number of Pharmacies Reporting
Variable
Mild
Mild
Persistent
Moderate
Persistent
Severe
Ozone (8 hour max)
28.03876**
25.78255**
24.9128**
34.19182**

(0.6175952)
(0.8377723)
(1.298766)
(1.88376)
Ozone2 (8 hour max)
-238.5753**
-203.1505**
-203.1219**
-234.6526**

(6.038243)
(8.048794)
(12.47718)
(18.20284)
Particulate Matter (<10u)
0.0236093**
0.0180628**
0.0199478**
0.0039002**

(0.0006542)
(0.0008845)
(0.0013377)
(0.0018807)
Particulate Matter2 (<10u)
-0.0002218**
-0.0001555**
-0.0001527**
-0.0000521**

(0.00000744)
(0.00000996)
(0.000015)
(0.0000216)
Minimum Temperature
-0.0000102**
-0.00000448
-0.00000303
0.00000565

(0.00000365)
(0.00000412)
(0.00000632)
(0.00000661)
Population
0.0000169**
0.0000167**
0.0000154**
0.0000143**

(0.000000119)
(0.000000164)
(0.000000258)
(0.000000368)
Land Area
1.26e-10**
8.84e-l 1**
1.85e-10**
1.17e-10* *

(6.75e-12)
(9.41e-12)
(1.35e-ll)
(1.93e-ll)
Percent Urban
-0.1884366**
-0.0807457
0.7162738**
-0.3309048

(0.0946442)
(0.1230779)
(0.1918656)
(0.2470555)
Median Household Income
-0.0000101**
-0.00000234
0.00000718*
-0.00000828

(0.00000208)
(0.0000027)
(0.00000412)
(0.00000564)
%Urban*Median HH Income
0.0000116**
0.00000279
-0.00000919**
0.00000626

(0.00000212)
(0.00000276)
(0.0000042)
(0.00000576)
Percent Black
1.161845**
1.189558**
0.9960942**
1.400747**

(0.0209779)
(0.0287297)
(0.0454172)
(0.0617281)
Percent Asian
-0.2380888**
-0.3749473**
-0.3028831**
-0.3933124**

(0.019031)
(0.026684)
(0.0402521)
(0.0595809)
Percent Hispanic
0.1120475**
0.0784604**
0.4877225**
0.3494201**

(0.0128876)
(0.0176528)
(0.0276418)
(0.0390927)
Quarter 2 (Apr, May, June)
-0.2254476**
-0.138979**
-0.1730995**
-0.3981783**

(0.0070148)
(0.0096739)
(0.0148544)
(0.0208331)
Quarter 3 (July, Aug, Sep)
-0.2536546**
-0.2035237**
-0.2518753**
-0.4719592**

(0.0069258)
(0.0096004)
(0.0146603)
(0.0206247)
Quarter 4 (Oct, Nov, Dec)
0.1051401**
0.1196589**
-0.0479534**
-0.1766381**

(0.0057953)
(0.0081355)
(0.012476)
(0.0177604)
Trend
0.0289785**
0.0307646**
0.0559504**
0.0722986**

(0.0004429)
(0.0006113)
(0.0009569)
(0.0013776)
Constant
1.50311**
0.92306**
-1.060152**
-0.5046592**

(0.0943713)
(0.1228649)
(0.191468)
(0.2478894)
Number of Observations
7419
7419
7419
7419
Pseudo R2
0.2237
0.1822
0.1301
0.1067
65

-------
Table 8: Poisson Regressions of Prescriptions Counts by Asthma Severity, Age 15-17
Weight=Number of Pharmacies Reporting
Variable
Mild
Mild
Persistent
Moderate
Persistent
Severe
Ozone (8 hour max)
29.71956**
27.33543**
14.47834**
8.698405**

(0.8649563)
(1.286507)
(2.03114)
(3.117208)
Ozone2 (8 hour max)
-236.204**
-186.2598**
-105.4592**
-78.27337**

(8.356989)
(12.29424)
(19.44391)
(30.2128)
Particulate Matter (<10u)
0.0154751**
0.0059427**
0.0012557
0.0264459**

(0.0009089)
(0.0013446)
(0.0020533)
(0.003321)
Particulate Matter2 (<10u)
-0.0001425**
-0.0000679**
0.00000691
-0.0002815**

(0.0000102)
(0.0000152)
(0.0000232)
(0.0000387)
Minimum Temperature
-0.0000114**
0.00000057
-0.0000108
-0.0000149

(0.00000494)
(0.00000488)
(0.0000117)
(0.0000222)
Population
0.0000174**
0.0000148**
0.0000135**
0.0000189**

(0.000000173)
(0.000000261)
(0.00000042)
(0.000000622)
Land Area
1.45e-10**
8.43e-l 1**
5.23e-l 1**
3.07e-10**

(9.39e-12)
(1.43e-ll)
(2.33e-l 1)
(2.96e-l 1)
Percent Urban
-0.4985458**
0.0284296
1.657045**
1.178507**

(0.1269028)
(0.1741472)
(0.2981351)
(0.4284837)
Median Household Income
-0.0000129**
0.00000297
0.000019**
0.0000214**

(0.00000286)
(0.00000387)
(0.00000622)
(0.00000908)
%Urban*Median HH Income
0.0000126**
-0.00000643
-0.0000274**
-0.0000272**

(0.00000291)
(0.00000395)
(0.00000636)
(0.0000093)
Percent Black
0.4924911**
0.3758033**
-0.2861978**
0.256651**

(0.0329554)
(0.0496094)
(0.0817554)
(0.1179288)
Percent Asian
0.0228642
0.241977**
-0.5079986**
-0.1860021**

(0.0263798)
(0.0392867)
(0.0639874)
(0.0926533)
Percent Hispanic
0.3924904**
0.3152129**
0.9438262**
1.100225**

(0.0186063)
(0.0277944)
(0.0446383)
(0.0677743)
Quarter 2 (Apr, May, June)
-0.2539285**
-0.2323478**
-0.1070033**
-0.1099388**

(0.0099014)
(0.0148334)
(0.023374)
(0.0350731)
Quarter 3 (July, Aug, Sep)
-0.320088**
-0.3221051**
-0.1877639**
-0.2353972**

(0.009909)
(0.014892)
(0.0230491)
(0.0345231)
Quarter 4 (Oct, Nov, Dec)
0.0941159**
0.0920717**
-0.018283
-0.1739332**

(0.0082749)
(0.0125823)
(0.01995)
(0.0296074)
Trend
0.0210245**
0.0294507**
0.0543165**
0.0716631**

(0.0006325)
(0.0009492)
(0.0015128)
(0.0023154)
Constant
1.20086**
0.3136288*
-2.058707**
-3.390688**

(0.1269083)
(0.1746926)
(0.2975482)
(0.4285481)
Number of Observations
7266
7266
7266
7266
Pseudo R2
0.1673
0.1009
0.0643
0.0688
66

-------
Table 9: Poisson Regressions of Prescriptions Counts by Asthma Severity, Adults and
Unknown Age. Weight=Number of Pharmacies Reporting
Variable
Mild
Mild
Persistent
Moderate
Persistent
Severe
Ozone (8 hour max)
15.26694**
(0.2143931)
10.04354**
(0.2594564)
10.09516**
(0.3530363)
11.07596**
(0.494678)
Ozone2 (8 hour max)
-137.5383**
(2.068485)
-89.2845**
(2.462988)
-90.0965**
(3.343758)
-93.8065**
(4.698299)
Particulate Matter (<10u)
0.0118307**
(0.0002222)
0.0042399**
(0.0002723)
0.0047997**
(0.0003668)
0.0100922**
(0.0005134)
Particulate Matter2 (<10u)
-0.0000975**
(0.00000252)
-0.0000398**
(0.00000309)
-0.0000399**
(0.00000416)
-0.0001046**
(0.00000589)
Minimum Temperature
-0.0000093**
(0.00000108)
-0.000016**
(0.00000154)
-0.00000472**
(0.00000145)
-0.0000154**
(0.00000289)
Population
0.0000154**
(0.000000044)
0.0000153**
(0.0000000544
)
0.0000154**
(0.0000000744
)
0.0000158**
(0.000000102)
Land Area
1.38e-10**
(2.40e-12)
1.38e-10**
(2.95e-12)
1.30e-10**
(4.00e-12)
1.56e-10**
(5.34e-12)
Percent Urban
-0.0999528**
(0.0384481)
-0.6880752**
(0.0451822)
-0.5357108**
(0.0595523)
0.0711288
(0.0833222)
Median Household Income
-0.0000332**
(0.000000868)
-0.000041**
(0.00000104)
-0.0000366**
(0.00000136)
-0.000027**
(0.00000189)
%Urban*Median HH Income
0.0000309**
(0.000000879)
0.0000396**
(0.00000105)
0.0000337**
(0.00000138)
0.0000219**
(0.00000192)
Percent Black
-0.5558639**
(0.0089839)
-0.484152**
(0.0110197)
-0.8177009**
(0.0155494)
-0.3448258**
(0.0200027)
Percent Asian
-0.3890154**
(0.0065659)
-0.1740856**
(0.0079606)
-0.3039745**
(0.0108593)
-0.7124759**
(0.0155441)
Percent Hispanic
0.8090284**
(0.0046989)
0.8202236**
(0.0058361)
1.015736**
(0.0079707)
1.1424**
(0.0109535)
Quarter 2 (Apr, May, June)
-0.2346234**
(0.0024129)
-0.0663938**
(0.0029677)
-0.0634297**
(0.0040307)
-0.0832976**
(0.0055787)
Quarter 3 (July, Aug, Sep)
-0.3617096**
(0.0024305)
-0.1141889**
(0.0029412)
-0.1068483**
(0.0039732)
-0.1276086**
(0.0054838)
Quarter 4 (Oct, Nov, Dec)
-0.0433887**
(0.0020208)
0.0684658**
(0.002569)
-0.0132208**
(0.0035097)
-0.0756691**
(0.004855)
Trend
0.0003705**
(0.0001565)
-0.0150399**
(0.0001917)
-0.0016361**
(0.0002603)
0.0097948**
(0.0003599)
Constant
4.334623**
(0.0382833)
4.737721**
(0.0450788)
3.821455**
(0.0594479)
2.392236**
(0.0831055)
Number of Observations
7097
7097
7097
7097
Pseudo R2
0.2405
0.2062
0.1817
0.1549
67

-------
References
Hajat, S., A. Haines, S. A. Goubet, R.W. Atkinson and H.R. Anderson (1998). "Association of
Air Pollution with Daily GP Consultations for Asthma and Other Lower Respiratory Conditions
in London." Thorax. 54: 597-605.
Hiltermann, T.J.N., J. Stolk, S.C. van derZee, B. Brunekreef, C.R. deBruijne, P.H. Fischer, C.B.
Ameling, P.J. Sterk, P.S. Hiemstra, and L. van Bree (1998). "Asthma Severity and Susceptibility
to Air Pollution." European Respiratory Journal. 11: 686-693.
Krupnick, Alan J., Winston Harrington, Bart Ostro (1990). "Ambient Ozone and Acute Health
Effects: Evidence from Daily Data." Journal of Environmental Economics and Management.
18:1-18.
Neukirch, Francoise, Claire Segala, Yvon Le Moullec, Myriam Korobaeff, and Michel Aubier
(1998). "Short-term effects of low-level winter pollution on respiratory health of asthmatic
adults." Archives of Environmental Health. 53(5): 320-9.
NIH (1997). Practical Guide of the Diagnosis and Management of Asthma. U.S. Department of
Health and Human Services Public Health Service National Institutes of Health National Heart,
Lung, and Blood Institiute. Publication No. 97-4053; October 1997.
http://www.nhlbi.nih.gov/guidelines/asthma/asthgdln.pdf
Ostro, Bart D., Michael Lipsett, Matthew B. Wiener, and John Seiner (1991). "Asthmatic
Responses to Airborne Acid Aerosols." American Journal of Public Health. 81: 694-702.
Peters, Annette, Inge F. Goldstein, Ulrich Beyer, Kathe Franke, Joachim Heinrich, Douglas
Dockery, John D. Spengler, and H. Erich Wichmann (1996). "Acute Health Effects of Exposure
to High Levels of Air Pollution in Eastern Europe." American Journal of Epidemiology. 144(6):
570-81.
Pope, C. Arden, Douglas W. Dockery, John D. Spengler, and Mark E. Raizenne (1991).
"Respiratory Health and PM10 Pollution." American Review of Respiratory Disease. 144: 668-
674.
Schwartz, Joel (1994). "Air Pollution and Hospital Admissions for the Elderly in Birmingham,
Alabama." American Journal of Epidemiology. 139 (6): 589-98.
Schwartz, Joel, David Wypij, Douglas Dockery, James Ware, Scott Zeger, John Spengler, and
Benjamin Ferris, Jr. (1991). "Daily Diaries of Respiratory Symptoms and Air Pollution:
Methodological Issues and Results " Environmental Health Perspectives, 90: 181-187.
Simon, Nathalie, B., Charles W. Griffiths, and Tracey Woodruff (2002). "Air Pollution and
Asthma in San Francisco: The Effects of Short-term Exposure on Asthma Medication Use,"
paper prepared for the 2nd World Congress of Environmental and Resource Economists,
68

-------
Monterey, CA June 27.
Sunyer, Jordi, Marc Saez, Carles Murillo, Jordi Castellsague, Francesc Martinez, and Josep M.
Anto (1993). "Air Pollution and Emergency Room Admissions for Chronic Obstructive
Pulmonary Disease: A 5-year Study." American Journal of Epidemiology. 137(7): 701-5.
Walters, Sarah ,R.K. Griffiths, J.G. Ayers (1994). "Temporal Association Between Hospital
Admissions for Asthma in Birmingham and Ambient Levels of Sulphur Dioxide and Smoke."
Thorax, 49:133-140.
Zeghnoun, Abdelkrim, Pascal Beaudeau, Fabrice Carrat, Veronique Delmas, Onealy
Boudhabhay, Francois Gayon, Dominque Guincetre, and Pierre Czernichow (1999). "Air
Pollution and Respiratory Drug Sales in the City of Le Havre, France, 1993-1996."
Environmental Research, Section A, 81: 224-230.
69

-------
Comments on Air Pollution and
Asthma in Children
r
«
ML
Bryan Hubbell
U.S. EPA
Office of Air and Radiation
Office of Air Quality Planning and Standards
Innovative Strategies and Economics Group
70

-------
Context
Since 1990, at least 18 studies have been
published examining the relationship between air
pollution and symptoms in asthmatics
Findings have been equivocal, although most
have found significant relationships between at
least one air pollutant and asthma symptoms
Symptom measures vary, with some focusing on
individual symptom indicators, like cough, and
some focusing 011 symptom scales or asthma
attacks/episodes

-------
Impacts of Nonroad Diesel Emission Reductions on Asthmatics
End point (Study population)
Study
Avoided
Incidence in 2030
Asthma Attack Indicators
Shortness of Breath (African American asthmatics, 8-13)
Ostro et al. (2001)
15,000
Cough (African American asthmatics, 8-13)
Ostro et al. (2001)
31,000
Wheeze (African American asthmatics, 8-13)
Ostro et al. (2001)
24,000
Asthma Exacerbation - one or more symptoms
(Asthmatics, 5-13)
Yu et al. (2000)
530,000
Cough (Asthmatics, 6-13)
Vedaletal. (1998)
240,000
Asthma Attacks (Asthmatics, all ages)
Whittemore and Korn (1980)
160,000
Other symptoms/illness endpoints
Upper Respiratory Symptoms (Asthmatics 9-11)
Pope etal. (1991
120,000
Moderate or Worse Asthma (Asthmatics, all ages)
Ostro et al.
120,000
Acute Bronchitis (Asthmatics, 9-15)
McConnelletal. (1999)
4,700
Chronic Phlegm (Asthmatics, 9-15)
McConnelletal. (1999)
12,000

-------
Context
We currently do not assign a separate monetized
value to "asthma attacks" or other indicators of
asthma exacerbations
Estimates are dated (Rowe and Chestnut 1986)
May not reflect current best practices
Often not a good match between what has been
valued, i.e. a "bad asthma day" and what has been
measured, i.e. a day with wheeze or an "asthma
attack"
Gives the impression that asthma impacts are
unimportant relative to other health impacts

-------



What do these 3 papers add?
2 valuation/epidemiological studies
Hanemann and Brandt stated preference will provide
a specific WTP for asthma related symptoms plus an
understanding of costs of averting and mitigating
behavior
Mansfield et al. revealed/stated preference provides a
lower bound check on WTP for ozone related asthma
symptoms, reflected the value of reductions in
averting behavior.
1 epidemiological/valuation study
Griffith et al. examination of link between air
pollution and asthma medication use (could also
provide a lower bound estimate of marginal cost of
higher pollution levels)
74

-------
Valuation
Current estimates of values for asthma symptoms are
based on:
Rowe and Chestnut (1986) WTP for a "bad asthma day" ($43)
Tolley et al. (1986) WTP for a day with coughing ($25)
Berger et al. (1987) WTP for avoided "coughing spell" ($80)
Dickie and Ulery (2002) WTP for a child's "symptom day"
($160)
Not clear whether any of these provide a direct match
with the WTP for specific symptoms or clusters of
symptoms associated with an air pollution related asthma
exacerbation

-------
Hanemann and Brandt
Provides a consistent framework for estimating health
outcomes and valuation, incorporating averting and
mitigating behavior
Provides information on both revealed and stated
preferences
Morbidity outcomes capture both incidence and severity
However:
Does not appear to integrate information on ozone or other
pollution predictions (alert days)
Full set of data won't be available for 4 years!
Not clear on the CV question format. Consideration of conjoint
approach to allow for variation in the type and severity of
symptoms to match the epi design.

-------

Mansfield et al.
¦	Excellent temporal sampling design, especially in
selecting survey days to control for ozone-temperature
interactions
¦	Large sample with detailed activity diaries on days with
high and low pollution levels
¦	Unique combination of revealed averting behavior data
with stated preference data on value of averting behavior
¦	However:
Difficulties with benefit transfer because of the limitation to
stay at home parents
Representativeness of the Harris Interactive panel is
questionable
Would be useful to include ozone/PM directly in model rather
than "code red" day dummy
77

-------
Bottom line: Will the 2 new valuation studies
provide us with acceptable estimates of WTP?
Hanemann RP will work IF we can get epi functions
that have treated exposure averting behaviors as
endogenous (the FACES study seems like it might
provide this). SP would benefit from a conjoint
design. Long time frame for study still leaves us in
limbo	
Mansfield et al. specification yields some relatively
modest results -> a code red day only reduces time
spent outdoors by around 43 minutes for asthmatic
children, and 32 minutes for non-asthmatic children.
Based on the SP results, this is valued at around $4
for asthmatic children and $3 for non-asthmatic
children. Fairly low relative to other estimates.

-------
Asthma Epidemiology
¦	Most asthma studies use a small sample
(<200) with a diary approach
¦	Examinati on of medi cine use has been
limited
¦	Medicine use may provide an endpoint for
which cost information is readily available

-------
Griffith et al.
¦	Creative use of available data
¦	Use of quarterly averages for pollution effects to capture longer term
"excess" prescriptions seems appropriate
¦	Suggestions
Prescriptions are actually a measure of demand, conditional on asthma
attacks triggered by high pollution levels
Derive the demand functions using a household production function so
that the econometric specification will have a grounding in economic
theory and can be more easily compared/combined
Prescriptions per capita is potentially not as relevant as prescriptions
per asthmatic, especially for younger age groups where asthma
prevalence is higher
Use of alternate ozone metrics, e.g. cumulative number of hours over
60 ppb, may show a stronger relationship.
PM2.5 may be a better indicator than PM10
Should include interactions between quarter and air pollution metrics to
control for potential exposure differences
Controls for numbers of prescriptions for maintenance medications
might be important effect modifiers, representing substitutes for
averting behaviors or expected rescue medication use

-------
Implications of Griffith et al. for impact
assessment
¦	Based on the Model 2 results in Table 3, a program like
the proposed Nonroad diesel rule which would reduce
annual mean PM2.5 (and PM10) levels by about 0.5 ug,
might result in over 250,000 fewer prescriptions per year,
(note that CA alone has over 2.1 million prescriptions for
"quick relief' medicines each year)
¦	The age and severity stratified Poisson results seem to
make more sense for ozone, which may indicate that the
pooled OLS model is not the correct specification

-------
What Can We Learn by Integrating?
82

-------
Everyone should use a household
production framework
¦	Makes life easier for those trying to
compare/contrast results
¦	Ensures that averting and mitigating
behaviors are considered

-------
The Role of Medication Use in Assessing the
Benefits of Air Pollution Programs is Important
¦	Hanemann and Brandt results show that use of
medicine to mitigate risks from exposure is
potentially important
¦	Griffith results show that the impacts of air
pollution 011 "quick relief' prescriptions is
significant
¦	Remaining issue is how use of prophylactic
medicine modifies the effects of air pollution 011
"quick relief' medicine use

-------
Averting Behavior Matters
¦	Hanemann and Brandt show that some parents are
aware of air pollution as a potential trigger for
asthma. They include exposure averting behavior
in conceptual model for WTP - however, need to
assure that measures of averting behavior include
those specific to certain triggers, i.e. air pollution
¦	Mansfield et al. show that time spent indoors by
asthmatic children is related to information on
ozone alerts, and that parents are able to provide
values for that lost time outdoors.
¦	Not clear how Griffith et al. results might be
impacted by averting behavior. Might be able to
use ozone alert days in a quarter as a measure of
likely averting behavior.

-------
Conclusions
I-Ianemann and Brandt are taking an important step by integrating the
epidemiology and economic valuation studies, however, the
estimating equations for the epi study should be derived from the
same behavioral framework, perhaps including prices and income as
exogenous variables
To measure the benefit of air pollution reductions, a full
understanding of how knowledge of future air pollution affects
averting behavior needs to be integrated into the epi and valuation
models. Johnson et al. are providing that type of understanding.
Complete benefits models should take into account the endogenous
nature of the full pathway including event aversion (preventative
medicine), exposure aversion (reducing time outdoors), response
mitigation (use of rescue medication), and response (loss of school
days, doctor visits).
All three of these papers use data from CA. A comparative analysis
bridging all three datasets will be very informative.

-------
Laurie Chestnut, Discussant Comments Session V, Air Pollution and Asthma in Children
October 21, 2003
Common to all three studies
~	All three of the studies consider one or more behavior choices that avert or mitigate
potential health effects of air pollution for people with asthma. These represent new
contributions to the literature on valuation of asthma cure, management and symptom
reduction. Considering behavior choices is important because:
° Benefits estimates based solely on epidemiology dose-response estimates miss
most averting and mitigating costs, because the studies capture the health effects
that occur after averting and mitigating has taken place.
n This behavioral context provides an opportunity for revealed preference analysis.
n Stated preference survey design is enhanced when a realistic context is given to
the valuation questions, which household behavior and choices provide.
n Surveys can be used to collect information useful for estimating household
production models. These questions may be easier to answer than direct valuation
questions.
~	These studies will extend and deepen the literature where there are a limited number of
empirical estimates available for:
° WTP values for reducing the frequency of asthma symptoms
° WTP values for asthma cure and asthma management
~	All the studies face many challenges in dealing with asthma, which is a complex
condition:
Many factors and	> Onset of asthma	> Aggravation of asthma
potentially pollution	A
A
pollution and other triggers
n Symptoms are intermittent
n Great deal of heterogeneity among asthmatics regarding:
—	sensitivities to various triggers
—	frequency/severity of symptoms
87

-------
Hanemann and Brandt - Specific Comments
~	Study is focusing on asthma symptoms and exacerbations in children with asthma, and on
household response and prevention efforts. Work is being conducted in association with
an ongoing asthma patient research panel.
~	Household production model
n Conceptually sound
n Good use of survey to obtain data on household behavior and perceptions
n Challenges:
—	joint benefits of averting/mitigating activities, e.g. air conditioning
—	heterogeneity of sensitivities to triggers
—	timing dimension challenging to capture, e.g. symptoms, environmental
conditions, activities, and household responses will all vary day-to-day
~	Contingent Valuation
n Good to use multiple valuation approaches with same subjects. Will have
behavior information as well as answers to direct valuation questions.
n Risk perception information should be helpful
n Challenges
—	defining change in asthma symptoms is complicated by wide variation in
frequency and severity of symptoms across subjects and over time.
—	linkages to epidemiology study results on asthma symptoms will be
helpful for policy analysts, but sometimes difficult to define in valuation
context.
88

-------
Mansfield et al. - Specific Comments
~	Good conceptual framework: How much do parents change children's time outdoors in
response to ozone warnings? Sample includes about equal numbers of children with and
without asthma.
~	Internet survey approach with pre-selected panel allows efficient sampling of days with a
range of ozone/weather conditions.
~	Regardless of the WTP results, the study should proof informative regarding the public's
response to the ozone warning system.
~	Challenges:
n Are about 3 days per subject enough for the planned analysis? This means the
study is essentially a cross-sectional analysis making it harder to statistically
identify the effect of ozone warnings.
n Sensitivities to ozone vary among those with asthma—the asthma/non-asthma
subject split may not be the best. Perceptions about sensitivity to air pollution are
probably more important.
n Odd preliminary results for the AWARE dummy variable indicating awareness of
the ozone warning system. Might those who are more concerned about air
pollution also be more aware of the system, making awareness more of an
endogenous variable?
° WTP to keep outdoor time is not the same thing as WTP to reduce asthma
symptoms. The authors will need to think about how this information is useful to
policy analysts. Should this WTP value be added to WTP to reduce pollution-
related symptoms? How is the WTP value applicable in assessing benefits of
pollution control?
89

-------
Griffiths et al. - Specific Comments
~	Addressing the question of whether purchases of quick relief asthma prescription
medicines vary by location in relation to air pollution concentrations.
n Quick relief asthma medicines are generally used in response to the onset of
specific symptoms, rather than as a regularly used preventative medication.
° Implications if the study finds an association is that asthma aggravations are more
frequent in locations with higher pollution.
n Use of quick relief medicine is a mitigating behavior expected to prevent
symptoms from getting as severe as they might otherwise have.
~	As with any cross-sectional study design, challenges are collinearity and unidentified
confounders:
n Quick relief prescriptions (counts or per capita rates) will capture higher
prevalence of asthma as well as more frequent symptoms, unless the number of
patients with asthma in each location is known.
n Relative mix of controller and quick relief medicine usage may vary across
locations because of differences in health care providers, socioeconomics, etc.
n Density, poverty, traffic, pollution and asthma prevalence and management
practices may be inter-related. This may have something to do with differences in
preliminary results between PM10 and ozone. PM10 tends to be correlated with
urban density, ozone less so because it tends to drift from original emission
sources.
° There may be differences across locations between average pollution
concentrations and frequencies of pollution spikes.
n Not normalizing the age group analysis by population size in an area could
introduce population-related distortions. Using counts works fine for time series
analysis where the population is stable day-to-day, but more problematic for a
cross-sectional analysis.
90

-------
Summary of Q&A Discussion Following Session V
Ronnie Levin (U.S. EPA, Region 1) commented that a study has been done in France on
GI medications and drinking water quality "that has done some of what you're doing with
time series and prescriptions—it's sort of a combination of time series and drug
dispensures over time."
Ms. Levin also commented on the heterogeneity issue, which Laurie Chestnut dealt with,
saying, "Yes, it's preferable for analytical purposes to use continuous vs. dichotomous
variables. On the other hand, what parents get is dichotomous. So, if you want to not
only test the system but really want to test the effect, it's the dichotomous variable that
matters." She suggested perhaps just coding it, because "you lose information by
reducing it all to continuous measures."
Shifting her comments to the asthma study, Ms. Levin stated that "17 percent of repeat
hospital visitations are not people who are not following the orders—those are people
who have severe asthma that is not controlled." She added that despite our desire and
best efforts to reduce our exposures, both indoors and outdoors, that's not always an
option, and she mentioned that available medications often don't provide real options
either (she particularly cited the behavioral side-effects that many children experience on
steroids). Furthermore, Ms. Levin noted that "high-performing athletes who have
asthma" are exceptions, no more representative of typical asthma sufferers than Lance
Armstrong, the most recent winner of the Tour de France, is representative of cancer
survivors. She also clarified that "respiratory infection does cause asthma and people
may use that as a reason to get through the gatekeeper, but these really are associated,
and they're associated for lots of physiological reasons."
Ms. Levin acknowledged that parents of asthmatic children face tough challenges (saying
"no, you can't go outside" or "you have to take this medicine"), and said it's important to
recognize options. "Parents can mitigate their kids' behaviour without keeping them
inside—they can reduce the activity level and still let them go outside. In all of this,
there are sort of heterogeneity issues." She concluded with these statements: "One thing
on the use of continuous vs. the ozone alerts—you're right, you want to test whether the
ozone alerts are working, and you have to keep it in those discreet categories. But, I
think you're missing an important dimension of the data. . . . Even if the parents don't
know about the ozone alerts, the ozone itself can give them an alert by giving their kids
symptoms, and that's what the Dickie and Bresnahan study shows: just using the marker
of ozone levels, people do show reactions, and that's very evident in the data."
Sylvia Brandt responded, "Right, but one's a predictor and one's post hoc, so it's a
different timing issue as well."
Glenn Harrison (University of Central Florida) offered what he considered "a number of
small comments. First, to pick up on Michael's point about doing a real-world dimension
contingent evaluation instead of stated preference. There are precursors, actually—this
91

-------
Health Partners HMO again in Minnesota, plus several web sites for asthmatics actually
do provide emailed alerts that are, to some extent, tailored." Dr. Harrison wondered why
Dr. Hanemann expressed concern "about the hypotheticality and abstract state of
preference but not about the contingent evaluation, since this is possible to do and
eventually deliver."
A second issue raised by Dr. Harrison was in regard to household production functions,
methodological factors common to a lot of the studies presented. Addressing Dr.
Hanemann, he commented, "I heard what you're saying but perhaps what you're talking
about is the difference between something being locally flexible as opposed to being
applied and perhaps globally irregular or globally ill-behaved. ... If you could just talk a
little bit more bout that issue, that would be good.
Dr. Harrison continued with these comments: "Related to that, actually, I still have a
sense, notwithstanding Bryan Hubbell's comments, that there's a lot of what I call
'toothbrush modeling' going on in this field, where everyone will say, 'I swear allegiance
to the household production function,' but, frankly, that just lets you do anything you
want to do . . . it's no constraint whatsoever, and that's the important thing that you were
getting at, that there needs to be perhaps some minimal constraint. ... So, in the spirit of
looking for minimal constraints couldn't one, in the context of the stated preference stuff,
use the counterpart of the Garp, the weak axiom of cost minimization, as a minimal test
for some sort of rationality in the choices that you're looking at? That might provide a
very useful metric for the quality of the responses that you're getting that is comparable
across virtually all data sets."
Dr. Harrison agreed with what he termed the "very good comment about the repeat
visits" and added, "You need to be extraordinarily sensitive looking at those data. If
people, for example, are funded under Medicaid, ... if you want to get repeat
prescriptions or there's a slight change, you have to have another visit, and that's actually
a major issue, and so the funding source interacts mightily there. Your point is well
taken, but I wouldn't overemphasize that 17 percent."
Directing his final comment to Charles Griffiths, Dr. Harrison cited "a wonderful data set
called the National Ambulatory Care Survey data set. . . which combines prescribing
details in DCs and the ICD9 codes for the same patient by a doctor, not by self reports or
anything like that." He added that these data cover "many, many years and you know the
location." He cited the case of a graduate student of his who was able to use the data to
help correlate respiratory illness with criteria air pollutant levels. He closed by adding
that there's a companion data set from a survey of numerous hospitals that provides
information on outpatient and ER prescriptions. He commented that although this data
set is "not quite as nationally representative as the ambulatory care one, it would be a
wonderful corollary source to look at."
Michael Hanemann responded, "I'll answer a subset of those comments. Let me just
amplify what I meant with the health production functions. You see people doing
something and then you see consequences—you see health effects. That's intentional
92

-------
behavior, and there's a connection, but it's a mistake to think necessarily that the person
knew that this behavior would lead to that consequence . . . and it shows the behavior in a
fine-tuned way." Dr. Hanemann added that he thinks it's more an issue for evaluation
because it involves interpretation after the event "because often the marginal evaluation is
conducted by measuring the marginal cost of an outcome, and that assumes . . . sort of
fine-tuned optimization, which might be out of place." He expounded on this by adding
that someone might say, "Yes, I spent $10, but the reason I didn't spend more than $10 is
because this is the only thing I knew to do" rather than "the $10 was for this outcome.
So," he said, "I think one wants to look at behavior and everyone wants to look at
production functions, but I think you also want to look at preferences, and I think you
want to be cautious about excessive evaluation of the margin based on an x cost estimate
in marginal costs."
Addressing another of Dr. Harrison's comments, Dr. Hanemann stated, "Your suggestion
about tests of rationality and consistency certainly makes sense." He continued by
responding to Dr. Harrison's comment regarding the hypotheticality of stated preference
in this fashion: "You know, what's hypothetical is in the mind. . . . The important thing, I
think, is addressing and engaging the respondent—looking him in the eye and saying,
'Here's a trade-off' And the trade-off could involve statements that are entirely at
variance with the facts, and yet people can respond to them as though they are
meaningful. So . . . the issue is not 'does this item really exist?'—the issue is 'does the
person think there's a commodity there which is within his grasp?' And if you can get
the person to wrap his mind around it, you're in reasonably good shape. The trick is to
make the thing match the person's circumstances."
Dr. Harrison replied, "On that, I agree that there's an added artificiality of the matrix,
given an SP. That's your point."
Don Kenkel (Cornell University) directed his question to both of the first two studies and
said he "was wondering if there are data on other types of averting behavior" besides just
staying indoors. He continued, "Smoking cessation is one that pops into my mind, but to
show you that I'm not focusing just on smoking, I was also struck by the list of important
causes of asthma and noticed that pets weren't even on there." He said he thought that
maybe that's because people who find out that their kids have asthma immediately get rid
of their pets, and he said this raises a couple of issues: "One is that this is another
revealed preference kind of argument—you're giving up something—you're sacrificing
something—the pet, in this case. You're giving up utility, and so that's another way of
getting at the value of asthma." Dr. Kenkel closed by adding, "The other issue,
especially for Reed Johnson's work, is really the relative indoor air quality vs. outdoor air
quality—maybe it's better being outdoors on a bad ozone day than it is being indoors
with the three cats and the mom smoking."
Sylvia Brandt responded by saying, "In our survey, we have about twenty different kinds
of averting behaviors as well as five different pages of fixed things you can invest in to
reduce exposure. The issue about the pet is actually a really good example of where
93

-------
families do make trade-offs. This came out in a lot of the focus groups, this balancing of
normality versus asthma episodes, and so there are households where, yes, the child is
allergic to dander—and that was one of the asthma triggers listed under allergies—but
it's worth using the control or rescue medication at times the child needs it to keep the
sense of a normal household. So these are some of the issues of trade-offs that we are
looking at.
Unidentified woman: "Related to this last question: As you said, asthmatics spend more
time indoors than non-asthmatics. What if that explains why they're asthmatic, because
they're spending way too much time indoors? Is that something to consider?
Michael Hanemann: "No."
Sylvia Brandt responded, "I think it's often compensating behavior. A lot of the children
we talked to basically became video game players because they couldn't go outside and
play with the other kids."
Woman: "So, it's not like diabetes."
Bryan Hubbell (U.S. EPA/OAQPS) directed his comments to Reed Johnson and said, "I
wondered why you actually got a lot of endogenous variables on your right-hand side—
you had the TV and the games, which could be very much a function of severity of the
asthma, as could the preference of being outdoors."
Reed Johnson: "I agree."
Hubbell: "There actually wasn't severity in any of your models, which was surprising."
Johnson: "Not yet."
Michael Hanemann added, "That's also why it's valuable to have something like asthma,
which has a clinical diagnosis that goes along with it, so even if it's a self report, no one's
going to declare that unless they've had a doctor tell them that."
94

-------
Closing Remarks
Mark Dickie, University of Central Florida
First of all, thanks, of course, to all the sponsors. All of this was done with EPA money,
so I want to thank all the offices of EPA that put this on and Ed Chu, Will Wheeler,
Nicole Owens, and Kelly Maguire for lassoing everybody in and keeping things going in
the right direction in organizing this. I'd also like to thank some folks from outside who
have made sure that everything ran on schedule—the people from the contractor in from
EPA, especially Tina Connelly and Denise DeShen, for all their effort in making this
come off logistically, as well as John and David and Annie from SCG. Of course, I'd
like to thank all of you for your participation and for the interesting and important
research that was presented and for all of the great questions and discussion and so on.
You know, if you asked anybody to do this, I think we'd all sort of have a different list of
what should be summarized as being important or interesting from the workshop. These
are my subjective perceptions of the highlights, offered in sort of chronological order.
I'd first like to echo Ed Chu's comment that he made the first thing the first morning: the
increase in the amount of research attention that children's health valuation has gotten
now. Back in 1999 there really were almost no studies, except a couple by Tom Crocker
and Mark Agee and then a few that sort of incidentally included kids or some kind of kid
component along the way of doing some other type of research, whereas now there was
all this stuff presented here, and there are things that weren't on the agenda that really
could have been presented here, and there's a lot of research that's just getting started
now.
A second thing is, of course, many of the presentations, especially on the first day,
highlighted the importance of really understanding the structure of the household
decision process. In Bill Schulze's paper that doesn't really matter because in his model
you get the same willingness to pay expression to estimate whether you use his Nash
bargaining model or a Becker type of approach, but I think that's not a general result. I
know in Ted Bergstrom's paper it really matters what the household decision process is
in terms of just what it is we should measure. That's a question that a lot of people have
been asking since 1997, since the Executive Order—what should we measure when we're
out to value kids' health? Whose willingness to pay counts? I think Ted Bergstrom's
paper really advances our understanding of who we need to be talking to and what we
need to count and do we add it up or take the minimum, and so on. I think that would
highlight, too, the importance of testing between different models and how well they
capture the household decision process, along the lines of what Sandy Hoffman was
proposing to do in her talk.
A very much related idea is the importance of understanding how people in the household
respond to environmental risks, and that was something that came up so many times in
the workshop, starting with Mary Evans' paper and then on and on and on with the
household production stuff and the asthma paper all about behavioural reactions and the
95

-------
revealed preference papers on the value of a statistical life. I think it's clear that we need
to understand better how households react to these risks and how we can use that
information for valuation, and Michael Hanemann pointed out some of the difficulties of
doing that with the household production framework, even though that was featured kind
of prominently here.
Understanding household behavior probably includes understanding the behavior of
children and teenagers themselves, which is something we tend to ignore, but especially
as kids get older and have more autonomy and they're doing things like driving one of
the family vehicles, or choosing whether or not to wear their bicycle helmet, or going as a
teenager to a tanning salon to get a suntan, or managing their own asthma medication.
You know, we typically think of the parent as sort of controlling the behavior in the
household, but that's clearly not true all the time, so I think maybe some more attention
to the stuff that Bill Harbaugh was talking about and what drives children's behavior
would really be relevant to the outcomes that children experience and maybe to how we
value them, as well.
The fourth thing on my "top five list" was what can we learn from other areas of research
such as family economics, health economics, psychology, and decision sciences? All
those appeared in one presentation or another over the past couple of days.
And then finally, I think in the VSL session in each case the idea of somehow
consistently treating morbidity and mortality risks came up as an important feature. In
the first two studies it was injuries or death in a car or on a bike, and then in the last two
it would be illness or death. Consistently treating those two is often going to be
necessary to adequately value either one because of the connection between them.
So, that is my stab at a summary of some key points from the research.
96

-------