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UNITED STATES ENVIRONMENTAL PROTECTION AGENCY
WASHINGTON, D.C. 20460

OFFICE OF CHEMICAL SAFETY
AND POLLUTION PREVENTION

MEMORANDUM

Date:	January 13, 2019

SUBJECT: Glyphosate: Response to Comments on the Proposed Interim Decision Regarding
the Human Health Risk Assessment

PC Code: 417300; 103601; 103603; 103604; 103605;

103607;103608;103613

Decision No.: 558081

Petition No.: NA

Risk Assessment Type: NA

TXR No.: NA

MRU) No.: NA

DP Barcode: D455459

Registration No.: NA

Regulatory Action: Registration Review

Case No.: *178

CAS No.: 1071-83-6; 38641-94-0; 70393-85-0;
114370-14-8; 40465-76-7; 69254-40-6; 34494-04-7;
70901-12-1
40 CFR: §180.364



FROM: Monique M. Perron, Sc.D., Toxicologist
Risk Assessment Branch 1 (RAB1)

Health Effects Division (HED; 7509P)

THROUGH: Christine L. Olinger, Branch Chief
RAB1/HED (7509P)

TO:	Steven Peterson, Chemical Review Manager

Dana Friedman, Branch Chief
Pesticide Re-evaluation Division (PRD; 7508P)

The Office of Pesticide Programs received hundreds of thousands of public comments on the
proposed interim decision (PID) for glyphosate as part of registration review. Comments
regarding the human health risk assessment came from a wide array of stakeholders. Topics
relating to human health included concerns with the agency's cancer assessment, toxicological
studies, protection of children, and detections of glyphosate. These comments regarding the
human health risk assessment for glyphosate have been previously addressed in the Glyphosate:
Response to Comments on the Raman Health Draft Risk Assessment (D448021; M. Perron; 23 -
APR-2018) and did not result in changes to the agency's risk assessment.

During the public comment period, 65 open literature studies were also identified for the
agency's consideration (Appendix A). Of these, 23 were previously identified and considered by

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the agency as part of two open literature searches conducted to support the draft human health
risk assessment for registration review. The agency reviewed the open literature as part of the
Glyphosate Systematic Review of Open Literature (D417703; TXR 0056885; M. Perron; 12-
DEC-2017) for hazard identification and characterization purposes in order to identify studies
that could potentially impact the human health risk assessment. A fit-for-purpose systematic
review was also executed to obtain relevant and appropriate open literature studies with the
potential to inform the human carcinogenic potential of glyphosate and detailed in the Revised
Glyphosate Issue Paper: Evaluation of Carcinogenic Potential (D444689; TXR 0057688; G.
Akerman; 12-DEC-2017).

The remaining 42 studies identified during the public comment period were primarily journal
articles published since these searches were conducted. The majority of the studies did not
warrant detailed evaluation for a variety of reasons, such as no effects seen from glyphosate
exposure, administration via a non-relevant route, effects seen at doses higher than the current
points of departure, only one dose was tested eliminating the potential for dose-response
evaluation, lack of glyphosate measurements, and exposure or biomonitoring studies that would
not impact risk estimates. Furthermore, several studies were conducted in vitro or evaluated
biochemical or molecular effects that are difficult to translate into in vivo effects. These studies
are typically considered as part of mode of action/adverse outcome pathway (MOA/AOP)
analyses. As a result, in vivo studies are given more weight at this time.

For the in vivo studies identified, the most common limitations/deficiencies were related to the
nature of the test substance(s) used for exposure. Many of the studies used commercial
formulations or dilutions; however, direct measurements of the active ingredient were not
conducted in order to determine actual dose concentrations and/or identification information was
not provided for the formulation used. There are numerous glyphosate formulations and
providing a general product name, such as Roundup, does not provide the agency with
information to ascertain the exact formulation used and determine all of its chemical
components. Additionally, several studies were conducted in other countries and utilized
formulations that are not registered in the United States. As a result, the active ingredients and
other components of the formulation are unknown, and any potential effects cannot be attributed
to glyphosate and/or defined glyphosate concentrations.

Several in vitro genotoxicity studies were identified for consideration. Although positive results
were observed in some of these studies, there would be no impact on the agency's weight of
evidence evaluation of the genotoxic potential of glyphosate since there is sufficient evidence in
the existing database (described in the Revised Glyphosate Issue Paper: Evaluation of
Carcinogenic Potential; D444689; TXR 0057688; G. Akerman; 12-DEC-2017) to conclude that
the in vitro effects claimed by the study authors do not lead to genotoxicity in vivo.

Two additional journal articles on the association between glyphosate exposure and non-
Hodgkin lymphoma (NHL) were identified for detailed review (D455531; D. Miller; 6-JAN-
2020;). The following are brief summaries of these journal articles and the agency's reviews:

• Zhang et al. (2019) is a review article summarizing epidemiological studies between
2001 and 2018 on the association between glyphosate exposure and NHL. All of the
data/information included in the article was previously considered by the agency as part

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of the Revised Glyphosate Issue Paper: Evaluation of Carcinogenic Potential (D444689;
TXR 0057688; G. Akerman; 12-DEC-2017); however, the authors conducted their own
meta-analysis to incorporate recently published risk estimates for the Agricultural Health
Study (AHS) cohort (Andreotti et al., 2018) based on their a priori hypothesis that the
highest exposures to glyphosate will lead to increased risk of NHL in humans1. In its
detailed review of this study, the agency identified concerns with the meta-analysis as
performed by the authors. A supplemental analysis by the agency indicated that a lower
non-statistically significant meta-risk ratio of 1.14 (95% CI: 0.87-1.50) would be
obtained if the Andreotti et al. (2018) study is properly incorporated into the meta-
analysis2. Therefore, the meta-analyses performed by Zhang et al. (2019) would not
impact the conclusions presented in the agency's revised issue paper.

• Leon et al. (2019) is a pooled analysis of NHL in agricultural cohorts from France,
Norway, and the United States (the AHS cohort). For overall NHL malignancies and
NHL subtypes, except diffuse large B-cell lymphoma (DLBCL), glyphosate risk
estimates were less than 1. For DLBCL a somewhat elevated, but non-statistically
significant risk estimate of 1.36 (95% CI: 1.00-1.85) was observed. While the analysis
benefited from a combined cohort of more than 300,000 farmers and farmworkers from
different countries, only the cohort from the United States used actual measurement
instruments (self-administered questionnaire) for glyphosate exposure where a smaller
risk estimate for the DLBCL subtype of 1.2 (95% CI: 0.72-1.98) was obtained.
Furthermore, the nature and characteristics of the three cohorts differed in substantive
ways and it is not clear that the statistical adjustments made were adequate to account for
these differences.

Therefore, none of the open literature studies identified for the agency's consideration were
found to have an impact on the glyphosate hazard characterization, cancer assessment, or human
health risk assessment. The agency will continue to monitor the open literature for studies that
use scientifically sound and appropriate methodology and relevant routes of exposure that have
the potential to impact the risk evaluation of glyphosate.

1 Here, higher exposures correspond to higher levels, longer durations, and/or greater lags/longer latencies.
2We note, however, that the meta-estimate in our Revised Glyphosate Issue paper was 1.27 (95% CI: 1.01. 1.59)
while our updated estimate after incorporating the Andreotti et al. (2018) paper is 1.14 (95% CI: 0.87, 1.50). See D.
Miller; D455531; 6-JAN-2020 for details and information.

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Appendix A. Studies identified for the agency's consideration during the public comment period for the Proposed Interim
Decision (PID).

Author

Year

Title

Part of previous
agency open
literature
reviews?

Comments

Aiassa, D

2019

Evaluation of genetic damage in pesticide
applicators from the province of Cordoba,
Argentina

No

ecological epidemiological study; no
glyphosate-specific estimates; would be
assigned low quality ranking according to
process detailed in revised issue paper on
evaluation of carcinogenic potential

Alvarez-Moya, C

2013

Comparison of the in vivo and in vitro genotoxicity
of glyphosate isopropylamine salt in three different
organisms

Yes

--

Astiz, M

2009

Antioxidant defense system in rats simultaneously
intoxicated with agrochemicals

Yes

--

Baier, Carlos J

2017

Behavioral impairments following repeated
intranasal glyphosate-based herbicide
administration in mice

No

Argentina formulation; micropipette
administration into nostrils (not a relevant
route); one dose tested; no glyphosate
measurements

Benbrook, CM

2019

How did US EPA and IARC reach diametrically
opposed conclusions on the genotoxicity of
glyphosate-based herbicides?

No

review paper; examined list of studies
published since agency's evaluation;
supplemental tables comparing EPA and
IARC yielded one study to reexamine
(Gasnier et al. 2009 included in table
below)

Bolognesi, C

2009

Biomonitoring of genotoxic risk in agricultural
workers from five Colombian regions: association
to occupational exposure to glyphosate

Yes

--

Bolognesi, C

1997

Genotoxic Activity of Glyphosate and Its Technical
Formulation Roundup

Yes

--

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Author

Year

Title

Part of previous
agency open
literature
reviews?

Comments

Bolognesi, C

2011

Micronuclei and pesticide exposure

No

Review article for biomonitoring studies
for several pesticides; already considered
the one study on glyphosate (Bolognesi et
al. 2009) in previous systematic review

Brendler-Schwaab, S

2005

The in vivo comet assay: use and status in
genotoxicity testing

No

No glyphosate information

Caballero, M

2018

Estimated Residential exposure of agricultural
chemicals and premature mortality by Parkinson's
disease in Washington state

No

Glyphosate exposure assessed indirectly
using geospatial information on
residential address and crop data from
WA State from 2011-2015; residential
address was based only on address listed
in the death registry for 2011-2015 so the
analysis was cross-sectional in nature and
was unable to assess potential lifetime
exposure based on either changes in WA
State agriculture or changes in residential
address; statistical analysis only
considered the demographic variables sex,
race, marital status, and education
indicating limited ability to assess
confounding or control for co-exposure to
pesticides and other environmental
factors; given these limitations, the
modest risk estimate of 1.3 (95% CI: 1.0-
1.62) may reflect residual confounding
and is based on a low quality exposure
assessment that did not directly assess
individual exposure or allow the
investigators to assess lifetime exposure

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Author

Year

Title

Part of previous
agency open
literature
reviews?

Comments

Cavalli, VL

2013

Roundup disrupts male reproductive functions by
triggering calcium-mediated cell death in rat testis
and Sertoli cells

Yes

--

Dimitroy, BD

2006

Comparative genotoxicity of the herbicides
Roundup, Stomp and Reglone in plant and
mammalian test systems

Yes

--

Gasnier, C

2009

Glyphosate-based herbicides are toxic and
endocrine disruptors in human cell lines.

Yes

In previous open literature review
considered non-relevant; reexamination
indicated it is relevant; in vitro study
evaluating glyphosate alone and
formulations for cytotoxicity, estrogenic
activity, anti-androgenic activity and
aromatase disruption; only effect reported
for glyphosate alone was a statistically
significant, non-concentration dependent
increase in anti-androgenic activity

Gehin, A

2005

Vitamins C and E reverse effect of herbicide-
induced toxicity on human epidermal cells HaCat:
a biochemometric approach

Yes

--

Ghisi, N

2016

Does exposure to glyphosate lead to an increase in
the micronuclei frequency? A systematic and meta-
analytic review

Yes

--

Gillezeau, C

2019

The evidence of human exposure to glyphosate: a
review

No

Exposure study; review article; no impact
on risk estimates

Grisolia, C

2002

A comparison between mouse and fish
micronucleus test using cyclophosphamide,
mitomycin C and various pesticides

Yes

--

Heu, C

2012

Glyphosate-induced stiffening of HaCaT
keratinocytes, a Peak Force Tapping study on
living cells

Yes

--

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Author

Year

Title

Part of previous
agency open
literature
reviews?

Comments

Jiang, Xiao

2018

A commercial Roundup formulation induced male
germ cell apoptosis by promoting the expression of
XAF1 in adult mice.

No

Formulation by gavage at 60, 180 or 540
mg/kg/day and in vitro exposures; no
glyphosate measurements; potential
effects only seen at dose higher than
current POD

Kasuba, V

2017

Effects of low doses of glyphosate on DNA
damage, cell proliferation and oxidative stress in
the HepG2 cell line

No

In vitro study evaluating DNA damage,
cell proliferation and oxidative stress in
HepG2 cells tested at 3 concentrations of
glyphosate; non-statistically significant
increase in proliferative response; primary
DNA damage (comet assay) decreased
relative to negative control; increase in
MN and effects on lipid peroxidation
were not concentration dependent; no
convincing glyphosate-related effects

Kojima, H

2010

Endocrine-disrupting Potential of Pesticides via
Receptors and Aryl Hydrocarbon Receptor

No

Mini-review; only mention of glyphosate
is in one table and it was negative for all
receptors

Kongtip, P

2017

Glyphosate, and paraquat in maternal and fetal
serum in Thai Women

No

Exposure study; no impact on risk
estimates

Kubsad, D

2019

Assessment of Glyphosate Induced Epigenetic
Transgenerational Inheritance of Pathologies and
Sperm Epimutations: Generational Toxicology

No

Intraperitoneal injection of glyphosate at
25 mg/kg/day in pregnant females; not a
relevant route

Kwiatkowska, M

2016

DNA damage and methylation induced by
glyphosate in human peripheral blood mononuclear
cells (in vitro study)

No

Report primary DNA damage at high
concentrations of glyphosate (0.5 mM and
higher) and increase in DNA methylation
of p53 and pl6 promoters; methylation
was statistically significantly increased at
both concentrations for the p53 promoter,
but not pi6; not enough concentrations
tested to make a conclusion on impact of
glyphosate on p53 methylation.

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Author

Year

Title

Part of previous
agency open
literature
reviews?

Comments

Leon, M

2019

Pesticide use and risk of non-Hodgkin lymphoid
malignancies in agricultural cohorts from
France, Norway and the USA: a pooled analysis
from the AGRICOH consortium

No

Detailed review performed (D. Miller; 6-
JAN-2020; D455531); summarized in text
above

Lioi, M

1998

Genotoxicity and oxidative stress induced by
pesticide exposure in bovine lymphocyte cultures
in vitro

Yes

--

Lueken, A

2004

Synergistic DNA damage by oxidate stress
(induced by H202) and nongenotoxic
environmental chemicals in human fibroblasts

Yes

--

Manas, F

2009

Genotoxicity of AMPA, the environmental
metabolite of glyphosate, assessed by the Comet
assay and cytogenetics test

Yes

--

Manas, F

2009

Genotoxicity of glyphosate by the comet assay and
cytogenetic test

Yes

--

Manservisi, F

2019

The Ramazzini Institute 13- week pilot study
glyphosate-based herbicides administered at
human-equivalent dose to Sprague Dawley rats:
effects on development and endocrine system

No

Glyphosate and formulation; no blinding
to dose; small sample size for
developmental evaluation (n=8/dose);
generally no adverse effects observed;
only one dose tested; no glyphosate
measurement

Mao, Q

2018

The Ramazzini Institute 13-week pilot study
glyphosate-based herbicides administered at
human-equivalent dose to Sprague Dawley rats:
effects on the microbiome

No

Glyphosate and formulation; no
glyphosate measurement; evaluated
changes in microbiome; no link to adverse
apical outcomes

Martinez, A

2019

Effects of glyphosate and aminomethylphosphonic
acid on an isogeneic model of the human blood-
brain barrier

No

In vitro study; no glyphosate
measurements

Mensah, PK

2015

Ecotoxicology of glyphosate and glyphosate-based
herbicide - toxicity to wildlife and humans

No

Book chapter; all references on human
health effects from glyphosate already
considered

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Part of previous



Author

Year

Title

agency open

literature

reviews?

Comments

Mesnage, R

2016

Multiomics reveal non-alcoholic fatty liver disease
in rats following chronic exposure to an ultra-low
dose of Roundup herbicide

No

Belgium formulation; proteome and
metabolome data; changes may reflect
adaptive liver effects; no link to adverse
apical outcomes

Milesi, MM

2018

Perinatal exposure to a glyphosate-based herbicide
impairs female reproductive outcomes and induces
second-generation adverse effects in Wistar rats

No

Argentina formulation by diet at 2 or 200
mg/kg/day; potential effects at dose
higher than current POD

Milic, M

2018

Oxidative stress, cholinesterase activity, and DNA
damage in the liver, whole blood, and plasma of
Wistar rats following a 28-day exposure to
glyphosate

No

Glyphosate administered via gavage at
0.1, 0.5, 1.75, and 10 mg/kg/day;
glyphosate was not measured in dose
preparations and small sample size (n=5)
used; no change in body and liver
weights; plasma and liver ROS and
plasma GSH levels similar to controls, no
change in GSH activity in blood,
inconsistent choline sterase data and no
neurotoxicity MOA/AOP for glyphosate









Study measured glyphosate/AMPA
metabolite excretion levels in NAFLD

Mills, PJ

2019

Glyphosate excretion is associated with
steatohepatitis and advanced liver fibrosis in
patients with fatty liver disease

No

patients who were either NASH (Non-
Alcoholic Steatohepatitis) or not NASH
patients and compared glyphosate and
AMPA measurement in urine; no subjects
without NAFLD; problems with
temporality; single urine measurement
only; no information collected on dietary
intake or occupation

Mills, PJ

2017

Excretion of the herbicide Glyphosate in older
adults between 1993-2016

No

Exposure/biomonitoring study; no impact
on risk estimates

Niemann, L

2015

A critical review of glyphosate findings in human
urine sample and comparison with the exposure of
operators and consumers

No

Review article for exposure; no impact on
risk estimates; calculations show
glyphosate exposure well below

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Part of previous



Author

Year

Title

agency open

literature

reviews?

Comments









acceptable daily intake (ADI) and
acceptable operator exposure levels
(AOEL)

Parvez, S

2018

Glyphosate exposure in pregnancy and shortened
gestational length: a prospective Indiana birth
cohort study

No

Glyphosate levels in urine and drinking
water; cross-sectional study evaluating
correlation; no AMPA metabolite
measured; small sample size; limited
diversity; no evaluation of other chemical
exposures (except smoking, caffeine, and
alcohol)

Panzacchi, S

2018

The Ramazzini Institute 13-week pilot study
glyphosate-based herbicides administered at
human-equivalent dose to Sprague Dawley rats:
study design and first in-life endpoints evaluation

No

Glyphosate and formulation; no
glyphosate measurement; no adverse
effects observed

Perry, M

2019

Historical evidence of glyphosate exposure from a
US agricultural cohort

No

Exposure/biomonitoring; no impact on
risk estimates

Pham, T

2019

Perinatal exposure to glyphosate and a glyphosate-
based herbicide affect spermatogenesis in mice

No

Glyphosate and Belgium formulation; no
glyphosate measurement; small sample
size for some parameters; no adverse
effect in several parameters; small
magnitude of change and/or lack of dose
response in others; no incidence or
severity scores reported for
histopathological evaluations

Portier, C

2016

Difference in the carcinogenic evaluation of
glyphosate between the IARC and EFSA

Yes

--

Prasad, S

2009

Clastogenic Effects of Glyphosate in Bone Marrow
Cells of Swiss Albino Mice

Yes

--

Richard, S

2005

Differential Effects of Glyphosate and Roundup on
Human Placenta Cells and Aromatase

Yes

--

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Part of previous



Author

Year

Title

agency open

literature

reviews?

Comments

Roustan, A

2014

Genotoxicity of mixtures of glyphosate and
atrazine and their environmental transformation
products before and after photoactivation

Yes

--

Saleh, SM

2018

Hepato-morphology and biochemical studies on the
liver of albino rats after exposure to glyphosate-
Roundup

No

Egypt formulation; no glyphosate
measurements; histopathological findings
cannot be interpreted without incidence or
severity scoring









Authors report chromosomal aberrations









and micronuclei formation in human

Santovito, A

2018

In vitro evaluation of genomic damage induced by
glyphosate on human lymphocytes

No

lymphocytes treated in vitro with 0.025
(ig/mL glyphosate and above; effects
reported are inconsistent with other
findings in the literature; sample size too
small to make reliable conclusions for low
test concentration

Schimpf, MG

2015

Neonatal exposure to a glyphosate based herbicide
alters the development of the rat uterus

No

sc injection (not a relevant route);
formulation not registered in US; no
glyphosate measurements; only one dose
tested

Sena de Souza, J

2019

Maternal glyphosate-based herbicide exposure
alters antioxidant-related genes in the brain and
serum metabolites of male rat offspring

No

Brazil formulation; gene expression
profiles; no link to adverse apical
outcomes









Brazil formulation; no glyphosate

Sena de Souza, J

2017

Perinatal exposure to glyphosate-based herbicide
alters the thyrotrophic axis and causes thyroid
hormone homeostasis imbalance in male rats

No

measurements; no changes in T3 or T4;
TSH decreased rather than increased and
presented a flat dose response; hormone
measurement calibration information not
reported; gene and metabolomic data

Seneff, S

2018

Is Glyphosate a key factor in mesoamerican
nephropathy?

No

Review article; hypothesis generating
paper

Sivikova, K

2005

Cytogenetic effect of technical glyphosate on
cultivated bovine peripheral lymphocytes

Yes

--

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Author

Year

Title

Part of previous
agency open
literature
reviews?

Comments

Stur, E

2019

Glyphosate-based herbicides at low doses affect
canonical pathways in estrogen positive and
negative breast cancer cell lines

No

Brazil formulation testing with breast
cancer cell lines; no glyphosate
measurements

Szepanowksi, F

2018

Differential impact of pure glyphosate and
glyphosate-based herbicide in a model of
peripheral nervous system myelination

No

No differences in myelination in cultures
treated with glyphosate compared to
vehicle control

Teleken, JL

2019

Glyphosate-based herbicide exposure during
pregnancy and lactation malprograms the male
reproductive morphofunction in F1 offspring

No

Brazil formulation; no glyphosate
measurements/only one dose tested that is
above current POD

Thongprakaisang, S

2013

Glyphosate induces human breast cancer cells
growth via estrogen receptors

Yes

--

Townsend, M

2017

Evaluation of various glyphosate concentrations on
DNA damage in human Raji cells and its impact on
cytotoxicity

No

DNA damage in vitro only at
concentrations that the authors report as
several orders of magnitude larger than
those attainable in vivo

Vigfusson, N

1980

The effect of the pesticides, Dexon, Captan and
Roundup, on sister-chromatid exchanges in human
lymphocytes in vitro

Yes

--

Von Ehrenstein, 0

2019

Prenatal and infant exposure to ambient pesticides
and autism spectrum disorder in children:
population based case-control study

No

Assumed exposure from agricultural
application records (no actual
measurements) other exposure pathways
not considered; addresses at time of
enrollment may not reflect an individual's
exposure over pregnancy

Walsh, L

2000

Roundup Inhibits Steroidogenesis by Disrupting
Steroidogenic Acute Regulatory (StAR) Protein
Expression

Yes

--

Wang, L

2019

Glyphosate induces benign monoclonal
gammopathy and promotes multiple myeloma
progression in mice

No

incorrect EPA chronic reference dose
cited; only one dose tested that is above
current POD

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Author

Year

Title

Part of previous
agency open
literature
reviews?

Comments

Wozniak, E

2018

The mechanism of DNA damage induced by
Roundup 360 PLUS, glyphosate and AMP A in
human peripheral blood mononuclear cells -
genotoxic risk assessment (Accepted Manuscript)

No

In vitro study using human lymphocytes;
DNA damage at glyphosate
concentrations of 250 (.iM and higher

Zhang, L

2019

Exposure to glyphosphate-based herbicides and
risk for non-Hodgkin lymphoma: A meta-analysis
and supporting evidence

No

Detailed review performed (D. Miller; 6-
JAN-2020; D455531); summarized in text
above

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